Documentation Guideline: Lower Limb Assessment (Basic & Advanced) (Paper Version)
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The Foot Angiosomes As Integrated Level of Lower Limb Arterial Perfusion
Research Article iMedPub Journals 2019 www.imedpub.com Journal of Vascular and Endovascular Therapy Vol. 4 No. 1: 7 The Foot Angiosomes as Integrated Level of Alexandrescu VA1* Pottier M1, Lower Limb Arterial Perfusion: Amendments Balthazar S2 and Azdad K3 for Chronic Limb Threatening Ischemia 1Department of Vascular and Thoracic Presentations Surgery, Princess Paola Hospital, Marche- en-Famenne, Belgium 2Department of Anesthesiology, Princess Paola Hospital, Marche-en-Famenne, Belgium Abstract 3Department of Radiology, Princess Paola Introduction: The angiosome concept was initially pioneered by Taylor and Palmer in the Hospital, Marche-en-Famenne, Belgium plastic reconstructive surgery field. The authors described a reproducible model of arterial and venous distribution in humans that follows specific three-dimensional (3D) networks *Corresponding author: Vlad Adrian of tissue. The angiosome model yet represents a specific level among other staged and Alexandrescu graduated levels of harmonious arterial irrigation in the lower extremity. Specific CLTI pathologies enhance characteristic arterial branches affectation, including the angiosomal source arteries. Evaluating main atherosclerotic lesions at peculiar Levels of arterial division [email protected] may afford useful clinical information. Department of Vascular and Thoracic Method: The present study proposes a description of six levels of degressive arterial division Surgery, Princess Paola Hospital, Marche- and collateral distribution in the inferior limb, including the angiosomal stage. Following en-Famenne, Belgium succeeding perioperative 2D angiographic observations over an eight-year period, these levels (I to VI) were analyzed (including the angiosomal Level III) and summarized in attached tables. The medical files of 323 limb-threatening ischemic foot wounds (Rutherford 4-6) in 295 patients (71% men) were retrospectively reviewed. -
Ehrlichiosis and Anaplasmosis Are Tick-Borne Diseases Caused by Obligate Anaplasmosis: Intracellular Bacteria in the Genera Ehrlichia and Anaplasma
Ehrlichiosis and Importance Ehrlichiosis and anaplasmosis are tick-borne diseases caused by obligate Anaplasmosis: intracellular bacteria in the genera Ehrlichia and Anaplasma. These organisms are widespread in nature; the reservoir hosts include numerous wild animals, as well as Zoonotic Species some domesticated species. For many years, Ehrlichia and Anaplasma species have been known to cause illness in pets and livestock. The consequences of exposure vary Canine Monocytic Ehrlichiosis, from asymptomatic infections to severe, potentially fatal illness. Some organisms Canine Hemorrhagic Fever, have also been recognized as human pathogens since the 1980s and 1990s. Tropical Canine Pancytopenia, Etiology Tracker Dog Disease, Ehrlichiosis and anaplasmosis are caused by members of the genera Ehrlichia Canine Tick Typhus, and Anaplasma, respectively. Both genera contain small, pleomorphic, Gram negative, Nairobi Bleeding Disorder, obligate intracellular organisms, and belong to the family Anaplasmataceae, order Canine Granulocytic Ehrlichiosis, Rickettsiales. They are classified as α-proteobacteria. A number of Ehrlichia and Canine Granulocytic Anaplasmosis, Anaplasma species affect animals. A limited number of these organisms have also Equine Granulocytic Ehrlichiosis, been identified in people. Equine Granulocytic Anaplasmosis, Recent changes in taxonomy can make the nomenclature of the Anaplasmataceae Tick-borne Fever, and their diseases somewhat confusing. At one time, ehrlichiosis was a group of Pasture Fever, diseases caused by organisms that mostly replicated in membrane-bound cytoplasmic Human Monocytic Ehrlichiosis, vacuoles of leukocytes, and belonged to the genus Ehrlichia, tribe Ehrlichieae and Human Granulocytic Anaplasmosis, family Rickettsiaceae. The names of the diseases were often based on the host Human Granulocytic Ehrlichiosis, species, together with type of leukocyte most often infected. -
Muscles Connecting the Upper Limb to the Vertebral Column
Muscles Connecting the Upper Limb to the Vertebral Column 1-Trapezius: O: external occiptal protuberance, superior nuchal line, ligamentum nuchae (along dorsal spine of cervical vertebra ), spines of all thoracic vertebrae and their supraspinous ligament " the extention of ligamentum nuchae ". Ins: (opposite to the Ori. Of Deltoid ) Upper fibers : Post. border lateral third of clavicle. Mid.(lateral fibers): inner surface of acromial process. Lower fibers: upper border of dorsal spine of scapula. N.S: Spinal accessory nerve (motor) and C3 and 4 (sensory) XI(11) cranial nerve (spinal part). *Note that accessory nerve = XI(11) cranial nerve which has 2 parts : cranial / spinal Action : Upper fibers :elevate the scapula. middle fibers: pull scapula medially toward the ribs (retracts). lower fibers: pull medial border of scapula downward . *anterior fibers rotates the scapula. Question 1 : to put your hand over your head what are the responsible muscles?? - Supraspinatus for initiation (0-15) or (0-18) - middle fibers of Deltoid (15 or 18 -90) - Trapezius & serratus anterior :after 90, rotation of scapula. Question 2 : to touch the acromial process of the other side which muscle is responsible , and which nerve will stop the movement of the muscle if we cut it?? -muscle: pectorals major , medial & lateral pectoral nerve. *always choose the easiest movement… 2-Latissimus dorsi : O: Iliac crest, lumbar fascia, spines of lower six thoracic vertebrae (T7-T12), lower three or four ribs, and inferior angle of scapula ,then all fibers make conversion to ins. Ins: Floor of bicipital groove of humerus. N.S: Thoracodorsal nerve (branch of post. Cord of post. -
Edema II, Clinical Significance
EDEMA II CLINICAL SIGNIFICANCE F. A. LeFEVRE, M.D., R. H. McDONALD, M.D., AND A. C. CORCORAN, M.D. It is the purpose of this paper to outline the clinical syndromes in which edema significantly appears, to discuss their differentiation, and to comment on the changes to which edema itself may give rise. The frequency with which edema occurs indicates the variety of its origins. Its physiologic bases have been reviewed in a former paper.1 Conditions in which edema commonly appears are summarized in Table 1. Although clinical edema usually involves more than one physiologic mechanism, it is not difficult to determine the predominant disturbance. Table 2 illustrates the physiologic mechanisms of clinical edema. Physiologically, edema is an excessive accumulation of interstitial fluid. Clinically, it may be latent or manifest, and, by its nature, localized or generalizing. These terms, with the exception of generalizing, have been defined, and may be accepted. By generalizing edema is meant a condi- tion in which edema is at first local in its appearance, but in which, as the process extends, edema will become general, causing anasarca. The degree of edema in any area is limited by tissue tension and the sites of its first appearance and later spread are partly determined by gravity. CARDIAC EDEMA Generalizing edema is an early manifestation of cardiac failure. It is usually considered to be evidence of inadequacy of the right ventricu- lar musculature (back pressure theory). Peripheral edema may be accompanied by pulmonary edema in cases where there is simultaneous left ventricular failure. Actually, the genesis of cardiac edema may depend more on sodium retention2,3'4 due to "forward cardiac failure" and renal constriction than on venous back pressure alone. -
Analyzing At-Home Prosthesis Use in Unilateral Upper-Limb Amputees To
2017 International Conference on Rehabilitation Robotics (ICORR) QEII Centre, London, UK, July 17-20, 2017. Analyzing At -Home Prosthesis Use in Unilateral Upper -Limb Amputees to Inform Treatment & Device Design Adam J. Spiers, Member, IEEE, Linda Resnik, and Aaron M. Dollar, Senior Member, IEEE Abstract — New upper limb prosthetic devices are continuously being developed by a variety of industrial, academic, and hobbyist groups. Yet, little research has evaluated the long term use of currently available prostheses in daily life activities, beyond laboratory or survey studies. We seek to objectively measure how experienced unilateral upper limb prosthesis-users employ their prosthetic devices and unaffected limb for manipulation during everyday activities. In particular, our goal is to create a method for evaluating all types of amputee manipulation, including non-prehensile actions beyond conventional grasp functions, as well as to examine the relative use of both limbs in unilateral and bilateral cases. This study employs a head-mounted video camera to record participant’s hands and arms as they Figure 1: A video screenshot from the head-mounted camera (for complete unstructured domestic tasks within their own homes. participant P2). A new ‘Unilateral Prosthesis-User Manipulation Taxonomy’ is presented based observations from 10 hours of recorded videos. has been a wide variety of prosthetic TDs (e.g. [4]–[6]). The taxonomy addresses manipulation actions of the intact However, follow-ups of how such devices’ are practically hand, prostheses, bilateral activities, and environmental and specifically used has been limited outside of the feature-use (affordances). Our preliminary results involved laboratory. Further motivation for the need of such tagging 23 minute segments of the full videos from 3 amputee understanding comes from well-known high prevalence rates participants using the taxonomy. -
Historical Aspects of Powered Limb Prostheses by Dudley S
Historical Aspects of Powered Limb Prostheses by Dudley S. Childress, Ph.D. INTRODUCTION People involved in work on powered limb from the viewpoint of important meetings and prostheses may wonder if the history of this events. Control approaches, another viewpoint, field is important. My answer is that one can are considered but not emphasized. Also, the learn a lot from history. Nevertheless, Hegel perspective is from America. has said, "What history teaches us is that men never learned anything from it." Unfortunately, PROLOGUE (1915-1945) it sometimes does seem true in prosthetics that we have not always profited from past experi The first powered prosthesis, of which I am ences. Too many aspects of the work are never aware, was a pneumatic hand patented in Ger published, and the multidisciplinary nature of many in 1915.13 A drawing of an early pneu the field produces papers in a broad spectrum of matic hand is shown in Figure 1. Figure 2 journals that are difficult to track. Books on the shows a drawing of what I believe to be the first field are, unfortunately, not numerous. electric powered hand. These drawings were The brief history that follows is by no means published in 1919 in Ersatzglieder und Arbeit complete, and since some of it involves years shilfen (Substitute Limbs and Work Aids).35 that are within readers' memories, I apologize This German publication illustrates the impor in advance for omissions that anyone may con tance of history in prosthetics, containing ideas sider significant. The history is intended to en that are still being discovered today. -
Human Glans and Preputial Development
Differentiation xxx (xxxx) xxx–xxx Contents lists available at ScienceDirect Differentiation journal homepage: www.elsevier.com/locate/diff ☆ Human glans and preputial development Xin Liu1, Ge Liu1, Joel Shen, Aaron Yue, Dylan Isaacson, Adriane Sinclair, Mei Cao, Aron Liaw, ⁎ Gerald R. Cunha, Laurence Baskin UCSF, USA ARTICLE INFO ABSTRACT Keywords: The urethra within the human penile shaft develops via (1) an “Opening Zipper” that facilitates distal canali- Development zation of the solid urethral plate to form a wide urethral groove and (2) a “Closing Zipper” that facilitates fusion Penis of the epithelial surfaces of the urethral folds. Herein, we extend our knowledge by describing formation of the Urethra human urethra within the glans penis as well as development of the prepuce. Forty-eight normal human fetal Human penile specimens were examined using scanning electron microscopy and optical projection tomography. Serial Glans histologic sections were evaluated for morphology and immunohistochemical localization for epithelial differ- Prepuce Canalization entiation markers: Cytokeratins 6, 7, 10, FoxA1, uroplakin and the androgen receptor. As the closing zipper completes fusion of the urethral folds within the penile shaft to form a tubular urethra (~ 13 weeks), canali- zation of the urethral plate continues in proximal to distal fashion into the glans penis to directly form the urethra within the glans without forming an open urethral groove. Initially, the urethral plate is attached ventrally to the epidermis via an epithelial seam, which is remodeled and eliminated, thus establishing me- senchymal confluence ventral to the glanular urethra. The morphogenetic remodeling involves the strategic expression of cytokeratin 7, FoxA1 and uroplakin in endodermal epithelial cells as the tubular glanular urethra forms. -
Stretching and Positioning Regime for Upper Limb
Information for patients and visitors Stretching and Positioning Regime for Upper Limb Physiotherapy Department This leaflet has been designed to remind you of the exercises you Community & Therapy Services have been taught, the correct techniques and who to contact with any queries. For more information about our Trust and the services we provide please visit our website: www.nlg.nhs.uk Information for patients and visitors Muscle Tone Muscle tone is an unconscious low level contraction of your muscles while they are at rest. The purpose of this is to keep your muscles primed and ready to generate movement. Several neurological causes may change a person’s muscle tone to increase or decrease resulting in a lack of movement. Over time, a lack of movement can cause stiffness, pain, and spasticity. In severe cases this may also lead to contractures. Spasticity Spasticity can be defined as a tightening or stiffness of the muscle due to increased muscle tone. It can interfere with normal functioning. It can also greatly increase fatigue. However, exercise, properly done, is vital in managing spasticity. The following tips may prove helpful: • Avoid positions that make the spasticity worse • Daily stretching of muscles to their full length will help to manage the tightness of spasticity, and allow for optimal movement • Moving a tight muscle to a new position may result in an increase in spasticity. If this happens, allow a few minutes for the muscles to relax • When exercising, try to keep head straight • Sudden changes in spasticity may -
Bone Limb Upper
Shoulder Pectoral girdle (shoulder girdle) Scapula Acromioclavicular joint proximal end of Humerus Clavicle Sternoclavicular joint Bone: Upper limb - 1 Scapula Coracoid proc. 3 angles Superior Inferior Lateral 3 borders Lateral angle Medial Lateral Superior 2 surfaces 3 processes Posterior view: Acromion Right Scapula Spine Coracoid Bone: Upper limb - 2 Scapula 2 surfaces: Costal (Anterior), Posterior Posterior view: Costal (Anterior) view: Right Scapula Right Scapula Bone: Upper limb - 3 Scapula Glenoid cavity: Glenohumeral joint Lateral view: Infraglenoid tubercle Right Scapula Supraglenoid tubercle posterior anterior Bone: Upper limb - 4 Scapula Supraglenoid tubercle: long head of biceps Anterior view: brachii Right Scapula Bone: Upper limb - 5 Scapula Infraglenoid tubercle: long head of triceps brachii Anterior view: Right Scapula (with biceps brachii removed) Bone: Upper limb - 6 Posterior surface of Scapula, Right Acromion; Spine; Spinoglenoid notch Suprspinatous fossa, Infraspinatous fossa Bone: Upper limb - 7 Costal (Anterior) surface of Scapula, Right Subscapular fossa: Shallow concave surface for subscapularis Bone: Upper limb - 8 Superior border Coracoid process Suprascapular notch Suprascapular nerve Posterior view: Right Scapula Bone: Upper limb - 9 Acromial Clavicle end Sternal end S-shaped Acromial end: smaller, oval facet Sternal end: larger,quadrangular facet, with manubrium, 1st rib Conoid tubercle Trapezoid line Right Clavicle Bone: Upper limb - 10 Clavicle Conoid tubercle: inferior -
Assessing Acute Collapse for Presentation Powerpoint and Handouts
“I’ve Fallen and [email protected] I Can’t Get Up” Assessing Acute Collapse For Presentation PowerPoint and Handouts: http://wendyblount.com Wendy Blount, DVM Kinds of Shock [email protected] Anaphylactic Shock •Obstructed airway •Acute allergic reaction •Lung Disease •Mast Cell Tumor Degranulation •Pleural air or effusion Cardiovascular Shock Neurogenic shock •Arrhythmia •Forebrain and brainstem - For Presentation PowerPoint •Left Heart Failure decreased consciousness •Right Heart Failure •Spinal cord – flaccid paralysis and Handouts: •Pericardial Disease Septic Shock http://wendyblount.com Hypovolemic Shock •Overwhelming infection •Dehydration Traumatic Shock •Hemorrhage •Due to pain •Hypoproteinemia Toxic Shock Hypoxic Shock •Due to inflammatory mediators, •Anemia endogenous and exogenous •Hemoglobin Pathology toxins Collapse Other Than Shock Assessment of Inability or Unwillingness to get up Collapse Profound Weakness Ataxia – lack of coordination •Metabolic weakness •Vestibular ataxia Quick Assessment •Hypercalcemia •Cerebellar ataxia Life Saving Treatment •Hypokalemia •Sensory ataxia •Hypoglycemia Paresis - loss of voluntary Physical Exam •Neurotoxins motor Emergency Diagnostics •Polyneuropathy •Lower Motor Neuron •Junctionopathy •CNS Lesion at level of History •Myopathy paresis Pain •Flaccid paresis In House Diagnostics •Spinal Cord/Nerve Pain •Upper Motor Neuron •Orthopedic Pain •CNS Lesion above paresis •Muscular Pain •Spastic paresis 1 Assessment of Assessment of Collapse Collapse Quick Assessment Life Saving Treatment -
Comparison of the Upper and Lower Limbs-A Phylogenetic Concept
IOSR Journal of Dental and Medical Sciences (IOSR-JDMS) e-ISSN: 2279-0853, p-ISSN: 2279-0861.Volume 14, Issue 8 Ver. I (Aug. 2015), PP 14-16 www.iosrjournals.org Comparison of the Upper and Lower Limbs-A Phylogenetic Concept Dr.Vandana Sinam1, Dr.Thonthon Daimei2, I Deven Singh3, N Damayanti Devi4 1.Medical officer,2. Senior Resident,3. Assistant Proffessor,4. Professor and Head, Department of Anatomy Regional Institute of Medical Sciences, Imphal, Manipur Abstract: The upper and lower limbs of the human body are phylogenetically homologues of the forelimbs and hind limbs of the quadrupeds. Primates started lifting of the forelimbs off the ground for various functional adaptations as evolution progressed and this led to the deviation of the forelimbs from lower limbs. With the gradual diversification of the functions, morphological evolution of the two limbs follows closely leading to the differences in upper and the lower limbs in the human. Since the inception of the Anatomy as one of the curriculum in medical subjects, the anatomical position has been termed as one that the body stand erect with the eyes looking straight forward and the two upper limbs hanging by the side of the body with the palm facing forward. Therefore in this position the upper limb looked forward with the palm also accordingly faced forward too whilst with the thumb on the lateral side whilst in the lower limb, the big toe which is homologous to the thumb, is placed on the medial side. The anatomical position in the upper limb is not normally a comfortable position as it is kept in this position with effort. -
Two Intriguing Cases of Cutibacterium Acnes Endocarditis
Open Access Case Report DOI: 10.7759/cureus.8532 Acne on the Valve: Two Intriguing Cases of Cutibacterium Acnes Endocarditis Samra Haroon Lodhi 1, 2 , Ayesha Abbasi 1, 2 , Taha Ahmed 3 , Albert Chan 4 1. Internal Medicine, King Edward Medical University, Lahore, PAK 2. Internal Medicine, Mayo Hospital, Lahore, PAK 3. Internal Medicine, Cleveland Clinic Foundation, Cleveland, USA 4. Cardiovascular Medicine, Cleveland Clinic Fairview Hospital, Cleveland, USA Corresponding author: Taha Ahmed, [email protected] Abstract Cutibacterium acnes is a skin commensal which is most often regarded as a contaminant when detected on blood cultures. In rare instances, it may be the causative pathogen in severe systemic illnesses. Subacute endocarditis, especially of prosthetic valves and devices, is an important grave pathology caused by Cutibacterium acnes. Herein we report two cases of prosthetic valve endocarditis with varied presentations as valve dehiscence with a “rocking” prosthetic valve apparatus in one encounter and as a septic embolic stroke in the second encounter. Although a rare cause of endocarditis, it becomes an especially important entity in patients with prosthetic devices and should be high in the list of differentials. Categories: Cardiology, Internal Medicine, Infectious Disease Keywords: cutibacterium acnes, endocarditis, rocking valve, prosthetic valves, paravalvular leak, vegetations, aortic root abscess Introduction Prosthetic cardiac valves have been labeled as a predisposing cardiac condition for infectious endocarditis in the guidelines provided by the American Heart Association [1]. Cutibacterium acnes (C. acnes) is a micro- aerophilic, non-spore-forming, gram-positive coccobacillus which constitutes a part of the normal skin flora. Although known as a causative agent of acne, it has been associated with various severe infections including endocarditis, osteomyelitis, arthritis, spondylodiscitis, endophthalmitis, post-craniotomy, and ventriculoperitoneal shunt infections [2].