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Abnormalities Caused by Left Bundle Branch Block - Print Article - JAAPA
Marquette University e-Publications@Marquette Physician Assistant Studies Faculty Research and Physician Assistant Studies, Department Publications 12-17-2010 Abnormalities Caused by Left undB le Branch Block James F. Ginter Aurora Cardiovascular Services Patrick Loftis Marquette University, [email protected] Published version. Journal of the American Academy of Physician Assistants, Vol. 23, No. 12 (December 2010). Permalink. © 2010, American Academy of Physician Assistants and Haymarket Media Inc. Useded with permission. Abnormalities caused by left bundle branch block - Print Article - JAAPA http://www.jaapa.com/abnormalities-caused-by-left-bundle-branch-block/... << Return to Abnormalities caused by left bundle branch block James F. Ginter, MPAS, PA-C, Patrick Loftis, PA-C, MPAS, RN December 17 2010 One of the keys to achieving maximal cardiac output is simultaneous contraction of the atria followed by simultaneous contraction of the ventricles. The cardiac conduction system (Figure 1) coordinates the polarization and contraction of the heart chambers. As reviewed in the earlier segment of this department on right bundle branch block (RBBB), the process begins with a stimulus from the sinoatrial (SA) node. The stimulus is then slowed in the atrioventricular (AV) node, allowing complete contraction of the atria. From there, the stimulus proceeds to the His bundle and then to the left and right bundle branches. The bundle branches are responsible for delivering the stimulus to the Purkinje fibers of the left and right ventricles at the same speed, which allows simultaneous contraction of the ventricles. Bundle branch blocks are common disorders of the cardiac conduction system. They can affect the right bundle, the left bundle, or one of its branches (fascicular block), or they may occur in combination. -
LBBB Cardiomyopathy and His- Bundle Pacing
LBBB Cardiomyopathy and His- Bundle Pacing Rajeev Singh General Cardiology Fellow October 2018 Disclosures • No relevant disclosures Goals of this Presentaon • I. Background: Introduce the audience to the concept of LBBB Cardiomyopathy • II. IU Experience with His Bundle Pacing and LeQ Bundle Branch Cardiomyopathy • III. Novel Concepts and Future Work Background: His Bundle Pacing Carlson, Joe. “Making pacemakers easier on the heart may come down to connections.” Star Tribune. May 27, 2017. Background: LBBB Cardiomyopathy • First proposed in 2013; based on JACC arIcle which retrospecIvely analyzed 375 paents form 2007-2010 • Six Paents were idenIfied that fit pre-exisIng criteria which included 1) History of typical LBBB > 5 years 2) LVEF > 50% 3) Decrease LVEF < 40% and development of HF to NYHA II-IV 4) Major mechanical dyssychrony 4) Idiopathic eIology of cardiomyopathy Vaillant, Caroline, et al. "Resolution of left bundle branch block–induced cardiomyopathy by cardiac resynchronization therapy." Journal of the American College of Cardiology 61.10 (2013): 1089-1095. Background: LBBB HFREF Does Not Respond to ConvenIonal Treatment • January 2018 Duke study; QRS duraon, EF, and OMT studied on 659 paents • Highest HF hospitalizaon, mortality for LBBB, worst response to OMT (3.5% improvement in EF vs 10% ) Sze, Edward, et al. "Impaired recovery of left ventricular function in patients with cardiomyopathy and left bundle branch block." Journal of the American College of Cardiology71.3 (2018): 306-317. 72 Paents who underwent His Bundle Pacing -
Resolution of Left Bundle Branch Block–Induced Cardiomyopathy by Cardiac Resynchronization Therapy
Journal of the American College of Cardiology Vol. xx, No. x, 2013 © 2013 by the American College of Cardiology Foundation ISSN 0735-1097/$36.00 Published by Elsevier Inc. http://dx.doi.org/10.1016/j.jacc.2012.10.053 CLINICAL RESEARCH Resolution of Left Bundle Branch Block–Induced Cardiomyopathy by Cardiac Resynchronization Therapy Caroline Vaillant, MD,* Raphaël P. Martins, MD,*† Erwan Donal, MD, PHD,*† Christophe Leclercq, MD, PHD,*† Christophe Thébault, MD,* Nathalie Behar, MD,* Philippe Mabo, MD,*† Claude Daubert, MD, FACC*† Rennes, France Objectives The study sought to describe a specific syndrome characterized by isolated left bundle branch block (LBBB) and a history of progressive left ventricular (LV) dysfunction, successfully treated by cardiac resynchronization ther- apy (CRT). Background Isolated LBBB in animals causes cardiac remodeling due to mechanical dyssynchrony, reversible by biventricular stimulation. However, the existence of LBBB-induced cardiomyopathy in humans remains uncertain. Methods Between 2007 and 2010, 375 candidates for CRT were screened and retrospectively included in this study if they met all criteria of a pre-defined syndrome, including: 1) history of typical LBBB for Ͼ5 years; 2) LV ejection fraction (EF) Ͼ50%; 3) decrease in LVEF to Ͻ40% and development of heart failure (HF) to NYHA functional class II to IV over several years; 4) major mechanical dyssynchrony; 5) no known etiology of cardiomyopathy; and 6) super-response to CRT with LVEF Ͼ45% and decrease in NYHA functional class at 1 year. Results The syndrome was identified in 6 patients (1.6%), 50.5 years of age on average at the time of LBBB diagnosis. -
View Pdf Copy of Original Document
Phenotype definition for the Vanderbilt Genome-Electronic Records project Identifying genetics determinants of normal QRS duration (QRSd) Patient population: • Patients with DNA whose first electrocardiogram (ECG) is designated as “normal” and lacking an exclusion criteria. • For this study, case and control are drawn from the same population and analyzed via continuous trait analysis. The only difference will be the QRSd. Hypothetical timeline for a single patient: Notes: • The study ECG is the first normal ECG. • The “Mildly abnormal” ECG cannot be abnormal by presence of heart disease. It can have abnormal rate, be recorded in the presence of Na-channel blocking meds, etc. For instance, a HR >100 is OK but not a bundle branch block. • Y duration = from first entry in the electronic medical record (EMR) until one month following normal ECG • Z duration = most recent clinic visit or problem list (if present) to one week following the normal ECG. Labs values, though, must be +/- 48h from the ECG time Criteria to be included in the analysis: Criteria Source/Method “Normal” ECG must be: • QRSd between 65-120ms ECG calculations • ECG designed as “NORMAL” ECG classification • Heart Rate between 50-100 ECG calculations • ECG Impression must not contain Natural Language Processing (NLP) on evidence of heart disease concepts (see ECG impression. Will exclude all but list below) negated terms (e.g., exclude those with possible, probable, or asserted bundle branch blocks). Should also exclude normalization negations like “LBBB no longer present.” -
New Emergency Room Requirement for Hospital and Autopay List of Diagnosis Codes
Provider update New emergency room requirement for hospitals Dell Children’s Health Plan reviewed our emergency room (ER) claims data and identified numerous reimbursements for services with diagnoses that are not indicative of urgent or emergent conditions. As a managed care organization, we promote the provision of services in the most appropriate setting and reinforce the need for members to coordinate care with their PCP unless the injury or sudden onset of illness requires immediate medical attention. Effective on or after August 1, 2020, for nonparticipating hospitals and on or after October 1, 2020, for participating hospitals, Dell Children’s Health Plan will only process an ER claim for a hospital as emergent and reimburse at the applicable contracted rate or valid out‐ of‐network Medicaid fee‐for‐service rate when a diagnosis from a designated auto‐pay list is billed as the primary diagnosis on the claim. If the primary diagnosis is not on the auto‐pay list, the provider must submit medical records with the claim. Upon receipt, the claim and records will be reviewed by a prudent layperson standard to determine if the presenting symptoms qualify the patient’s condition as emergent. If the reviewer confirms the visit was emergent, according to the prudent layperson criteria, the claim will pay at the applicable contracted rate or valid out‐of‐network Medicaid fee‐for‐service rate. If it is determined to be nonemergent, the claim will pay a triage fee. In the event a claim from a hospital is submitted without a diagnosis from the auto‐pay list as the primary diagnosis and no medical records are attached, the claim for the ER visit will automatically pay a triage fee. -
Vectorcardiographic Study of Aberrant Conduction' of Intraventricular Block
Br Heart J: first published as 10.1136/hrt.38.6.549 on 1 June 1976. Downloaded from British Heart journal, 1976, 38, 549-557. Vectorcardiographic study of aberrant conduction' Anterior displacement of QRS: another form of intraventricular block H. E. Kulbertus, F. de Leval-Rutten, and P. Casters From the Division of Cardiology, Institute of Medicine, University of Liege School of Medicine, Liege, Belgium Aberrant ventricular conduction was induced in 44 subjects by introduction of atrialpremature beats through a transvenous catheter-electrode. Multiple patterns of aberrant ventricular conduction were obtained in 32 patients and, in the whole group, 116 different configurations were recorded. Of these, 104 showed a classical pattern of mono- or biventricular conduction disturbance. The pattertn frequencies were as follows: right bundle-branch block, 28; left anterior hemiblock combined with right bundle-branch block, 21; left anterior hemiblock, 17; left posterior hemiblock combined with right bundle-branch block, 12; left posterior hemiblock, 10; complete left bundle-branch block, 10; and incomplete left bundle-branch block, 6. The remaining 12 configurations could not be classified into the usual categories of intraventricular blocks. In 7 of them, the alterations only consisted of trivial modifications of the QRS contour. In the other 5 instances, aberrant conduction manifested itself by a conspicuous anterior displacement of the QRS loop, with increased duration of anteriorforces. The latter observation is worthy of notice, as it indicates that, in the differential diagnosis of the vectorcardiographic pattern characterized by prominent anteriorforces, conduction disturbances should http://heart.bmj.com/ be considered a possible aetiological factor in addition to right ventricular hypertrophy, and true posterior wall myocardial infarction. -
Hypercalcemia-Induced New Onset Left Bundle Branch Block Mimicking Acute Myocardial Infarction in a Patient with Primary Hyperparathyroidism
Case Report Acta Cardiol Sin 2013;29:188-191 Hypercalcemia-Induced New Onset Left Bundle Branch Block Mimicking Acute Myocardial Infarction in a Patient with Primary Hyperparathyroidism Yu-Tsung Cheng,1 Chieh-Shou Su,1,2 Wei-Chun Chang,1,2 Meng-Hsia Chiang,1,3 Chih-Tai Ting1,2 and Wei-Win Lin1,4 A 78-year-old women with a recent diagnosis of primary hyperparathyroidism presented with vague chest pain, and new onset left bundle block (LBBB) on the electrocardiogram (ECG) mimicking acute myocardial infarction (AMI). LBBB resolved without abnormal Q waves only after correction of hypercalcemia. The cardiac enzymes, including creatine kinase, creatine kinase-MB, and troponin-I were all within normal range. Hypercalcemia provoking ECG changes that mimic acute myocardial infarction is infrequently reported. To our knowledge, this is the first report of hypercalcemia-induced new onset LBBB mimicking AMI. Emergency physicians should include hypercalcemia-induced new onset LBBB on the ECG in the differential diagnosis of AMI. Key Words: Acute myocardial infarction · Hypercalcemia · Hyperparathyroidism · left bundle branch block INTRODUCTION CASE REPORT Acute myocardial infarction (AMI) is an important A 78-year-old female presented to our facility with a differential diagnosis in patients who present with acute 10-year history of hypertension. The patient denied any chest pain and new onset left bundle branch block (LBBB) history of coronary artery disease, diabetes mellitus, or on the electrocardiogram (ECG). Unnecessary treatment hyperlipidemia. She had been hospitalized in another can be harmful in patients who have new onset LBBB institution for a deteriorating level of consciousness and due to conditions other than AMI. -
Intra-His Bundle Block. Clinical, Electrocardiographic, and Electrophysiologic Characteristics
Andréa et al OriginalArq Bras Article Cardiol Intra-His bundle 2002; 79: 532-7. Intra-His Bundle Block. Clinical, Electrocardiographic, and Electrophysiologic Characteristics Eduardo M. Andréa, Jacob Atié, Washington A. Maciel, Nilson A. de Oliveira Jr, Luiz Eduardo Camanho, Luís Gustavo Belo, Hecio Affonso de Carvalho, Leonardo Siqueira, Eduardo Saad, Ana Claudia Venancio Rio de Janeiro, RJ - Brazil Objective - To assess the clinical, electrocardiogra- Atrioventricular block is a conduction disturbance at phic, and electrophysiologic characteristics of patients the axis formed by the atrium, AV node, His bundle, and its (pt) with intra-His bundle block undergoing an electro- branches, which may vary from a mild delay in conduction physiologic study (EPS). (first-degree atrioventricular block) to a conduction block between atria and ventricles (second- and third-degree Methods - We analyzed the characteristics of 16 pt with atrioventricular blocks) 1. second-degree atrioventricular block and symptoms of Atrioventricular block may occur at the 3 following syncope or dyspnea, or both, undergoing conventional EPS. electrophysiological levels: atrioventricular node, within 2 Results - Intra-His bundle block was documented in 16 the His bundle, and below the His bundle . These levels pt during an EPS. In 15 (94%) pt, the atrioventricular block have anatomical correlation with, respectively, the atrioven- was recorded in sinus rhythm; 4 (25%) pt had intra-His tricular node, the penetrating His bundle (within the central Wenckebach phenomenon, which correlated with Mobitz I fibrous body), and nonpenetrating His bundle (out of the (MI) atrioventricular block on the electrocardiogram. Seven central fibrous body). First-degree atrioventricular block (44%) pt had 2:1 atrioventricular block, 2 of whom were usually has a delay in the conduction within the atrioventri- asymptomatic (12.5%). -
Revisiting Electrocardiographic Wolff-Parkinson-White Pattern
The Journal of Medical Research 2020; 6(4): 114-116 Review Article Revisiting Electrocardiographic Wolff-Parkinson-White Pattern JMR 2020; 6(4): 114-116 1 2 July- August Pradnya Brijmohan Bhattad , Vinay Jain ISSN: 2395-7565 1 Resident, Department of Internal Medicine, East Tennessee State University, Tennessee (TN), USA © 2020, All rights reserved 2 Attending Radiologist, Department of Radiology, James H. Quillen Veterans Affairs Medical Center, Mountain www.medicinearticle.com Home, Tennessee (TN), USA Received: 14-06-2020 Accepted: 18-07-2020 Abstract Electrocardiographic features of a short PR interval, a delta wave, and a wide QRS complex constitutes a Wolff- Parkinson-White (WPW) pattern. Asymptomatic electrocardiographic findings are defined as a WPW pattern. Symptomatic patients with these electrocardiographic features have WPW syndrome. WPW syndrome may predispose to arrhythmias such as paroxysmal atrial tachycardia, atrial fibrillation, ventricular tachycardia. Patient’s with WPW syndrome at risk for sudden cardiac death. It is important to recognize the common electrocardiographic characteristics of WPW pattern. The advent of electrophysiological studies (EPS) and radiofrequency ablation has revolutionized the management of WPW syndrome. Keywords: Wolff-Parkinson-White Syndrome, Wolff-Parkinson-White Pattern, Pre-excitation, Accessory pathway. INTRODUCTION Wolff, Parkinson, and White described a patient series in the year 1930 who suffered from paroxysms of tachycardia with classic electrocardiographic findings which has been described as WPW pattern [1]. It is a congenital abnormality in the cardiac conduction system. WPW pattern is a ventricular pre-excitation entity wherein an accessory bypass tract known as the bundle of Kent serves as the connection between the atrial to the ventricular myocardium bypassing the atrioventricular (AV) node [2,3]. -
IHEU-IHES-IHP Front Matter
IHEU unique 7/13/06 12:03 PM Page i 2007 ICD-9-CM Expert for Hospitals Volumes 1, 2 & 3 International Classification of Diseases 9th Revision Clinical Modification Sixth Edition Edited by: Anita C. Hart, RHIA, CCS, CCS-P Beth Ford, RHIT, CCS Ingenix is committed to providing you with the ICD-9-CM code update information you need to code accurately and to be in compliance with HIPAA regulations. In the case of adoption of additional ICD-9-CM code changes effective April 1, 2007, Ingenix will provide these code changes to you at no additional cost! Just check back at http://www.IngenixOnline.com and look for the ICD-9, CPT® and HCPCS Alerts link under the Quick Access Resources menu to review the latest information concerning any new code changes. Codes Valid October 1, 2006, through September 30, 2007 October 2006 Update 3539/IHEU/U0515 07 ICD9 v1 H 7/11/06 2:24 PM Page 127 Tabular List CIRCULATORY SYSTEM 425.8–426.89 Circulatory System 425.8–426.89 Nerve Conduction of the Heart Normal and Long QT Electrocardiogram QRS Interval ST Interval Normal Long QT R Syndrome R PR Sinoatrial node Segment QT Interval QT Interval (pacemaker) Bachmannʼs bundle PR ST Interval Segment Internodal tracts: Anterior T Middle P T P Posterior Left bundle branch block Left bundle branch: Atrioventricular Q node Anterior fascicle Q Common bundle Posterior fascicle S (of His) S Left bundle branch Atrioventricular block hemiblock 0.2.4.6 0.2.4.6 Accessory bundle MC (of Kent) 426.3 ¥>Other left bundle branch block Right bundle branch Left bundle branch block: Right -
Atrioventricular Conduction in Patients with Clinical Indications for Transvenous Cardiac Pacing1
British Heart Journal, 1975, 37, 583-592. Atrioventricular conduction in patients with clinical indications for transvenous cardiac pacing1 Stafford I. Cohen, L. Kent Smith, Julian M. Aoresty, Panagiotis Voukydis, and Eugene Morkin From the Cardiac Unit, Department of Medicine, Beth Israel Hospital and Harvard Medical School, Boston, Massachusetts, U.S.A. Eighty patients with clinical indications for cardiac pacing had atrioventricular conduction analysed by His bundle study. The indicationsfor cardiac pacing included high grade atrioventricular block, sick sinus node syndrome without tachycardia, bradycardia-tachycardia syndrome, unstable bilateral bundle-branch block, and uncontrolled ventricular irritability. Complete heart block, Wenckebach block (Mobitz I), and 2:i block were notedproximal and distal to the His bundle. Mobitz II block only occurred distal to the His bundle. Ofspecial interest were the high incidence ofdistal conduction abnormalities by His bundle analysis (40/80, 5o%), the re-establishment ofnormal atrio- ventricular conduction in acutely ill patients with recent evidence of heart block, and the high incidence of intraventricular conduction disturbances on standard electrocardiogram (48/8o, 60%). Intensive study of atrioventricular conduction by occurring electrophysiological data in this large His bundle analysis has been performed in a variety group of patients in clinical need of pacemakers of patient populations. In many instances studies constitutes the substance of this report. The data were electively undertaken in patients who had should be representative of the cardiac conduction never been threatened by a compromising cardiac abnormalities which present in a general hospital. arrhythmia. In addition, abnormalities of atrio- ventricular conduction were frequently achieved by Subjects and methods pacemaker-induced acceleration of the atrial rate. -
CMS Limitations Guide - Cardiovascular Services
CMS Limitations Guide - Cardiovascular Services Starting October 1, 2015, CMS will update their It is the responsibility of the provider to code to the existing medical necessity limitations on tests and highest level specified in the ICD-10-CM. The correct procedures to correspond to ICD-10 codes. This use of an ICD-10-CM code listed below does not limitations guide provides you with the latest assure coverage of a service. The service must be changes. reasonable and necessary in the specific case and must meet the criteria specified in this This guide is not an all-inclusive list of National determination. Coverage Documents (NCD) and Local Coverage Documents (LCD). You can search by LCD or NCD or We will continue to update this list as new CMS keyword and region on the CMS website at: limitations are announced. You can always find the https://www.cms.gov/medicare-coverage- most current list at: database/overview-and-quick- www.munsonhealthcare.org/medicalnecessity. search.aspx?clickon=search. If you have any questions, please contact Kari Smith, CMS will deny payment if the correct diagnosis Office Coordinator, at (231) 935-2296, or Karen codes are not entered on the order form, and your Popa, Director, Patient Access Services, at (231) 935- 7493. patient’s test or procedure will not be covered. We compiled this information in one location to make it easier for you to find the proper codes for medically necessary diagnoses. CMS Limitations Guide – Cardiovascular Services (L34636) Electrocardiographic (EKG or ECG) Monitoring (Holter