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Home , Bradycardia, Chest pain, Sinus bradycardia

San Mateo County Emergency Medical Services (Symptomatic)

History Differential • Past • rate < 50 with associated hypotension, acute • Acute • Heart transplant altered mental status, chest , acute CHF, • • seizures, or secondary to • Pacemaker failure • Beta blockers bradycardia • • Calcium channel blockers • Systolic BP < 90 • Sinus bradycardia • Clonidine • Chest pain • Athletes • Digoxin • Respiratory distress • Head injury (elevated ICP) or • Pacemaker • or shock • Spinal cord lesion • Altered mental status • Sick sinus syndrome • Syncope • AV blocks (e.g., 1o, 2o or 3o) • Overdose

HR < 50 and symptomatic: Exit to Hypotension, acute AMS, , acute CHF, No appropriate TP seizure, syncope, or shock secondary to bradycardia

Yes

Cardiac monitor

P Establish IV/IO

12-Lead ECG

Atropine

If systolic BP < 90 Normal Saline bolus 500ml IV/IO Maximum 2L Consider amnestic properties P if not responsive to . Pacing may be Midazolam considered first line therapy for severe P Consider pain control symptoms. Fentanyl Consider early in 2o or 3o blocks and patients with suspected cardiac EtCO2 monitoring (if available)

Respiratory Distress or Airway Field Dyspnea/increased work of , Yes Procedure especially with hypoxia? if indicated

Notify receiving facility. Consider Base Hospital for medical direction

Treatment Protocol CD01 Page 1 of 2 EffectiveEffective November March 2018 2019 San Mateo County Emergency Medical Services Bradycardia (Symptomatic)

Pearls • Bradycardia causing symptoms is typically < 50/minutes. Rhythm should be interpreted in the context of symptoms and pharmacological treatment given only when symptomatic, otherwise monitor and reassess frequently. • Identifying signs and symptoms of poor perfusion caused by bradycardia is paramount. • Atropine vs. pacing: Caution should be exercised in the setting of a suspected acute MI. The use of Atropine for bradycardia in the presence of an acute MI may worsen heart damage. Providers should NOT DELAY transcutaneous pacing for patients with poor perfusion in the setting of an acute MI or 2o or 3o . • For patients who are not in 2o or 3o heart block, pacing may be considered for bradycardia not responsive to Atropine. Prepare to utilize transcutaneous pacing early if the patient does not respond to Atropine. • For wide complex, bizarre appearance of QRS complexes with slow rhythm, consider . • Consider treatable causes for bradycardia (e.g., OD, OD, etc.) • is a common cause of bradycardia. Be sure to oxygenate the patient and support respiratory effort. • in the absence of key symptoms requires no specific treatment; monitor and observe. • Sinus bradycardia is often seen in patients with STEMI or ischemia. An early 12-Lead ECG should be obtained to assess for STEMI. • A fluid bolus may address hypotension and lessen the need for pacing or treatment with Atropine. • Sedation prior to starting pacing is not required. Patients with urgent needs should be paced first and sedated afterwards. • The objective of amnesia and pain control with pacing is to decrease discomfort, not to decrease level of consciousness. Patients who are in need of pacing are unstable and medications should be used with extreme caution. • Monitor respiratory status closely and support ventilation as necessary. • Atropine is not effective for bradycardia in heart transplant patients as there is no innervation in these patients. • Patients with wide QRS or 2o or 3o heart blocks will not have a response to Atropine because their heart rates are not based on . An increase in ventricular may occur. • If no capture or hypotensive (SBP<90), then start dopamine 5 mcg/kg/min. If inadequate response after 5 minutes, then may increase 5 mcg/kg/min to a maximum dose of 20 mcg/kg/min. Treatment Protocol CD01 Page 2 of 2 EffectiveEffective November March 2018 2019