Dermatology-Guidelines.Pdf
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
Photoaging & Skin Damage
Use_for_Revised_OFC_Only_2006_PhotoagingSkinDamage 5/21/13 9:11 AM Page 2 PEORIA (309) 674-7546 MORTON (309) 263-7546 GALESBURG (309) 344-5777 PERU (815) 224-7400 NORMAL (309) 268-9980 CLINTON, IA (563) 242-3571 DAVENPORT, IA (563) 344-7546 SoderstromSkinInstitute.comsoderstromskininstitute.com FROMFrom YOUR Your DERMATOLOGISTDermatologist [email protected]@skinnews.com PHOTOAGING & SKIN DAMAGE Before You Worship The Sun Who’s At Risk? Today, many researchers and dermatologists Skin types that burn easily and tan rarely are believe that wrinkling and aging changes of the skin much more susceptible to the ravages of the sun on the are much more related to sun damage than to age! skin than are those that tan easily, rather than burn. Many of the signs of skin damage from the sun are Light complected, blue-eyed, red-haired people such as pictured on these pages. The decrease in the ozone Swedish, Irish, and English, are usually more suscep- layer, increasing the sun’s intensity, and the increasing tible to photo damage, and their skin shows the signs sun exposure among our population – through work, of photo damage earlier in life and in a more pro- sports, sunbathing and tanning parlors – have taken a nounced manner. Dark complexions give more protec- tremendous toll on our skin. Sun damage to the skin tion from light and the sun. ranks with other serious health dangers of smoking, alcohol, and increased cholesterol, and is being seen in younger and younger people. NO TAN IS A SAFE TAN! Table of Contents Sun Damage .............................................Pg. 1 Skin Cancer..........................................Pgs. 2-3 Mohs Micrographic Surgery ......................Pg. -
Skin Care, Insect Bites and Stings
DEPARTMENT KLINISCHE WETENSCHAPPEN | MEDISCHE DIENSTEN Kronenburgstraat 43/3, 2000 Antwerpen | Fax: +32 3 247 64 10 Updated version (20/05/2015 – AVG) see: www.travelhealth.be SKIN CARE, INSECT BITES AND STINGS Sun The closer you get to the Equator, the more intense sunlight becomes. Sunbathing in the tropics has to be done in moderation. Protective clothing and hats are recommended. Apply sun cream with high sun protection factor (30 or more) to exposed skin regularly (every two hours) and carefully. Apply sun cream after bathing and avoid long water exposure since sun stroke will be imminent in spite of reduced heat feeling. Avoid perfumed sun creams and check whether or not used creams or medication can cause "sun allergy" (photo-toxic or photo-allergic reactions). We would like to refer to point 5 of the European cancer code: avoid excessive exposure to sun and sunburn during childhood (increased risk of melanomas in later life). Do not take a course of sunbed sessions before going on holiday as the sun tan obtained through UV-A does not give any extra protection against the natural UV-rays. When using sun creams and insect repellents based on DEET, recent studies have shown that DEET reduces the effectiveness of the sun cream, but that sun creams do not have a negative influence on the effectiveness of DEET. It is advisable, therefore, to apply the insect repellent (DEET or another repellent) with the sun lotion and then to take additional precautions to protect against UV (e.g. a sun cream with a higher protection factor). -
Allergic Reactions to Bites and Stings
Allergic Reactions to Bites and Stings ASCIA EDUCATION RESOURCES (AER) PATIENT INFORMATION Most insect bites and stings result in a localised itch and swelling that settles within a few days. Severe allergic reactions (anaphylaxis) to insects are relatively uncommon, and are usually due to bees, wasps or the Australian Jack Jumper ant. Fortunately, effective treatments are available to treat allergic reactions to bites and stings. Stinging insects are a common cause of anaphylaxis Allergies to venoms from stinging insects are one of the most common causes of severe allergic reactions (anaphylaxis) in Australia. Symptoms include an all over rash, swelling of tongue or throat, trouble breathing, gut cramps, diarrhoea, vomiting or even a drop in blood pressure (shock). Although the insects are all hymenoptera (which means membranous winged insects), their venoms are very different. Allergy to one type of stinging insect does not usually increase the risk of reaction to another. The Honey Bee is the most common cause of allergic reactions in Australia. Paper Wasps and European Wasps can sting multiple times. The European Wasp is becoming an increasing problem in Australia, is particularly aggressive and likes to get inside drink cans at barbeques, although the more familiar Paper Wasp is responsible for the majority of serious stings. The Australian Jack Jumper Ant (Myrmecia pilosula) is a medium sized black bull ant prevalent down the eastern side of Australia and Tasmania. It can be recognised by its characteristic hopping motion when it walks. It is a very aggressive ant and its sting can cause severe local pain. Severe allergic reactions are much more common than is seen with more common bull ants. -
61497191.Pdf
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Repositório Institucional dos Hospitais da Universidade de Coimbra Metadata of the chapter that will be visualized online Chapter Title Phototoxic Dermatitis Copyright Year 2011 Copyright Holder Springer-Verlag Berlin Heidelberg Corresponding Author Family Name Gonçalo Particle Given Name Margarida Suffix Division/Department Clinic of Dermatology, Coimbra University Hospital Organization/University University of Coimbra Street Praceta Mota Pinto Postcode P-3000-175 City Coimbra Country Portugal Phone 351.239.400420 Fax 351.239.400490 Email [email protected] Abstract • Phototoxic dermatitis from exogenous chemicals can be polymorphic. • It is not always easy to distinguish phototoxicity from photoallergy. • Phytophotodermatitis from plants containing furocoumarins is one of the main causes of phototoxic contact dermatitis. • Topical and systemic drugs are a frequent cause of photosensitivity, often with phototoxic aspects. • The main clinical pattern of acute phototoxicity is an exaggerated sunburn. • Subacute phototoxicity from systemic drugs can present as pseudoporphyria, photoonycholysis, and dyschromia. • Exposure to phototoxic drugs can enhance skin carcinogenesis. Comp. by: GDurga Stage: Proof Chapter No.: 18 Title Name: TbOSD Page Number: 0 Date:1/11/11 Time:12:55:42 1 18 Phototoxic Dermatitis 2 Margarida Gonc¸alo Au1 3 Clinic of Dermatology, Coimbra University Hospital, University of Coimbra, Coimbra, Portugal 4 Core Messages photoallergy, both photoallergic contact dermatitis and 45 5 ● Phototoxic dermatitis from exogenous chemicals can systemic photoallergy, and autoimmunity with photosen- 46 6 be polymorphic. sitivity, as in drug-induced photosensitive lupus 47 7 ● It is not always easy to distinguish phototoxicity from erythematosus in Ro-positive patients taking terbinafine, 48 8 photoallergy. -
Avoiding and Treating Work-Related Insect Bites and Stings
FACT SHEET Avoiding and Treating Work-Related Insect Bites and Stings This WorkCare Fact Sheet describes work-related bite and sting risks, symptoms, treatment and preventive measures. Bites and stings are a relatively common occurrence for people who work outdoors and in enclosed environments where bees and wasps, fire ants, insects and arachnids (spiders, scorpions, ticks and mites) feel at home. Employers and workers are encouraged to understand exposure risks, how to recognize and respond to stings and bites, and what they can do to prevent them. Under 29 CFR Part 1904 – Recording and Reporting Occupational Injuries and Illnesses, the Occupational Safety and Health Administration (OSHA) considers bites and stings to be recordable when an employee who is bitten or stung while working receives medical treatment beyond first aid. First aid is defined in1904.7 (b)(5)(ii). Exposure Risk Thousands of people in the U.S. are stung or bitten each year. Exposed arms and hands tend to be more susceptible to stings and bites among workers than legs and feet, which are usually protected by clothing and enclosed shoes. In a study of occupationally related bites and stings, the head, one of the most exposed body parts, accounted for one-tenth of cases involving insects and arachnids; a third of those cases affected the eyes. An estimated 90 to 100 people die each year in the U.S. as a result of allergic reactions to bites and stings. Overall, bite- and sting-related injuries and fatalities may be misdiagnosed as heart attack or sunstroke, or attributed to other causes, according to the National Institute for Occupational Safety and Health (NIOSH). -
Unit 3 Bites and Stings
First Aid in Common and Environmental Emergencies UNIT 3 BITES AND STINGS Structure 3.0 Introduction 3.1 Objectives 3.2 Bites and Stings 3.2.1 Definition, Causes, Types and Recognition of Bites and Stings 3.2.2 Assessment of the Victim and General First Aid 3.3 Various Bites/Stings 3.3.1 Scorpion Bite and Spider Bite 3.3.2 Snake Bite 3.3.3 Insect Bite 3.3.4 Animal Bites (Dog Bite/Monkey Bites) 3.3.5 Human Bites 3.4 Let Us Sum Up 3.5 Keywords 3.6 Answers to Check Your Progress 3.7 References and Further Readings 3.0 INTRODUCTION Bites and stings are commonly seen in the rural and remote areas. Nowadays, however, they can occur in urban areas also. Lakhs of people every year are bitten or stung by someone or something. These emergencies include bites and stings due to various reasons. These bites or stings need to be identified and treated early as they affect some part or the whole of the body which can cause mild, moderate or severe reaction and can even be life-threatening. Most are not medical emergencies but however, treatment is usually required if there is bleeding, wounds or infection. All bites and stings are not same. Different First Aid treatment and care is needed depending on the type of insect or animal that has caused the bite. Some species are more dangerous and cause more harm compared to others. Hence, in this unit we shall discuss the different types of bites and stings, causes, recognition and first aid in these situations. -
Triamcinolone Acetonide Cream USP, 0.025%, 0.1%, 0.5% for Dermatologic Use Only Not for Ophthalmic Use Rx Only
TRIAMCINOLONE ACETONIDE- triamcinolone acetonide cream Padagis Israel Pharmaceuticals Ltd ---------- Triamcinolone Acetonide Cream USP, 0.025%, 0.1%, 0.5% For Dermatologic Use Only Not For Ophthalmic Use Rx Only DESCRIPTION The topical corticosteroids constitute a class of primarily synthetic steroids used as anti- inflammatory and anti-pruritic agents. Triamcinolone acetonide is designated chemically as pregna-1,4-diene-3,20-dione,9-fluoro-11,21-dihydroxy-16,17-[(1-methylethylidene) bis (oxy)]-,(11ß,16α)-. C24H31FO6, and M.W. of 434.51; CAS Reg. No. 76-25-5. Each gram of 0.025%, 0.1% and 0.5% Triamcinolone Acetonide Cream USP contains 0.25 mg, 1 mg, or 5 mg triamcinolone acetonide respectively, in a washable cream base of cetyl alcohol, cetyl esters wax, glycerin, glyceryl monostearate, isopropyl palmitate, polysorbate-60, propylene glycol, purified water, sorbic acid, and sorbitan monostearate. CLINICAL PHARMACOLOGY Topical corticosteroids share anti-inflammatory, anti-pruritic and vasoconstrictive actions. The mechanism of anti-inflammatory activity of the topical corticosteroids is unclear. Various laboratory methods, including vasoconstrictor assays, are used to compare and predict potencies and/or clinical efficacies of the topical corticosteroids. There is some evidence to suggest that a recognizable correlation exists between vasoconstrictor potency and therapeutic efficacy in man. Pharmacokinetics - The extent of percutaneous absorption of topical corticosteroids is determined by many factors including the vehicle, the integrity of the epidermal barrier, and the use of occlusive dressings. Topical corticosteroids can be absorbed from normal intact skin. Inflammation and/or other disease processes in the skin increase percutaneous absorption. Occlusive dressings substantially increase the percutaneous absorption of topical corticosteroids. -
Summary of Product Characteristics
Health Products Regulatory Authority Summary of Product Characteristics 1 NAME OF THE MEDICINAL PRODUCT Audaval 0.1% Ointment 2 QUALITATIVE AND QUANTITATIVE COMPOSITION One gram of ointment contains 1 mg of betamethasone (0.1% w/w) as valerate. For a full list of excipients, see section 6.1. 3 PHARMACEUTICAL FORM Ointment Opaque ointment. 4 CLINICAL PARTICULARS 4.1 Therapeutic Indications Audaval preparations are indicated for the treatment of: eczema in children over 1 year elderly and adults; including atopic and discoid eczemas; prurigo nodularis; psoriasis (excluding widespread plaque psoriasis); neurodermatoses, including lichen simplex, lichen planus; seborrhoeic dermatitis; contact sensitivity reactions; discoid lupus erythematosus and they may be used as an adjunct to systemic steroid therapy in generalised erythroderma. In general, ointment preparations are particularly appropriate for dry, lichenified or scaly skin conditions whereas a cream preparation may be more suitable in the case of moist or weeping lesions. 4.2 Posology and method of administration For topical use only. If no improvement is seen after two to four weeks, the diagnosis should be reconsidered and specialist referral may be necessary. Adults, adolescents and the elderly A small quantity of Audaval should be applied to the affected area one to three times daily as directed by physician until improvement occurs. It may then be possible to maintain improvement by applying once a day, or even less often, or by using the appropriate ready diluted (1 in 4) preparation, Audaval RD 0.025% Ointment. Allow adequate time for absorption after each application before applying an emollient. If no improvement is seen within two to four weeks, reassessment of the diagnosis, or referral, may be necessary. -
Photodermatoses Update Knowledge and Treatment of Photodermatoses Discuss Vitamin D Levels in Photodermatoses
Ashley Feneran, DO Jenifer Lloyd, DO University Hospitals Regional Hospitals AMERICAN OSTEOPATHIC COLLEGE OF DERMATOLOGY Objectives Review key points of several photodermatoses Update knowledge and treatment of photodermatoses Discuss vitamin D levels in photodermatoses Types of photodermatoses Immunologically mediated disorders Defective DNA repair disorders Photoaggravated dermatoses Chemical- and drug-induced photosensitivity Types of photodermatoses Immunologically mediated disorders Polymorphous light eruption Actinic prurigo Hydroa vacciniforme Chronic actinic dermatitis Solar urticaria Polymorphous light eruption (PMLE) Most common form of idiopathic photodermatitis Possibly due to delayed-type hypersensitivity reaction to an endogenous cutaneous photo- induced antigen Presents within minutes to hours of UV exposure and lasts several days Pathology Superficial and deep lymphocytic infiltrate Marked papillary dermal edema PMLE Treatment Topical or oral corticosteroids High SPF Restriction of UV exposure Hardening – natural, NBUVB, PUVA Antimalarial PMLE updates Study suggests topical vitamin D analogue used prophylactically may provide therapeutic benefit in PMLE Gruber-Wackernagel A, Bambach FJ, Legat A, et al. Br J Dermatol, 2011. PMLE updates Study seeks to further elucidate the pathogenesis of PMLE Found a decrease in Langerhans cells and an increase in mast cell density in lesional skin Wolf P, Gruber-Wackernagel A, Bambach I, et al. Exp Dermatol, 2014. Actinic prurigo Similar to PMLE Common in native -
Urticaria from Wikipedia, the Free Encyclopedia Jump To: Navigation, Search "Hives" Redirects Here
Urticaria From Wikipedia, the free encyclopedia Jump to: navigation, search "Hives" redirects here. For other uses, see Hive. Urticaria Classification and external resourcesICD-10L50.ICD- 9708DiseasesDB13606MedlinePlus000845eMedicineemerg/628 MeSHD014581Urtic aria (or hives) is a skin condition, commonly caused by an allergic reaction, that is characterized by raised red skin wheals (welts). It is also known as nettle rash or uredo. Wheals from urticaria can appear anywhere on the body, including the face, lips, tongue, throat, and ears. The wheals may vary in size from about 5 mm (0.2 inches) in diameter to the size of a dinner plate; they typically itch severely, sting, or burn, and often have a pale border. Urticaria is generally caused by direct contact with an allergenic substance, or an immune response to food or some other allergen, but can also appear for other reasons, notably emotional stress. The rash can be triggered by quite innocent events, such as mere rubbing or exposure to cold. Contents [hide] * 1 Pathophysiology * 2 Differential diagnosis * 3 Types * 4 Related conditions * 5 Treatment and management o 5.1 Histamine antagonists o 5.2 Other o 5.3 Dietary * 6 See also * 7 References * 8 External links [edit] Pathophysiology Allergic urticaria on the shin induced by an antibiotic The skin lesions of urticarial disease are caused by an inflammatory reaction in the skin, causing leakage of capillaries in the dermis, and resulting in an edema which persists until the interstitial fluid is absorbed into the surrounding cells. Urticarial disease is thought to be caused by the release of histamine and other mediators of inflammation (cytokines) from cells in the skin. -
Dear Editor, Photo-Onycholysis Due to Tetracyclines
Dear EditOr, Photo-onycholysis Due to Tetracyclines: a Case Report (Tetrasiklin Kulammma Baglz Fotoonikolizis: Olgu Sunumu) Mehmet Kose Assist. Prof.1 M.D. Department of Pediatrics Erciyes University Medical Faculty Photo-onycholysis refers to separation of the nail plate from the nail bed after [email protected] exposure to ultraviolet light. Drug induced photo-onycholysis is seen most commonly with tetracycline. Harun Y1lmaz M.D. Department of Pediatrics A-12-years old boy was admitted to our pediatric department because of fever and Erciyes University Medical Faculty arthralgia. He was diagnosed as Brucellosis and treated with rifampin (20 mglkg/24hr) Kaz•m Ozum and tetracycline (30mg/kg/24hr in 3 divided oral doses). Seven days later pinkish Prof., M.D. purple discoloration and onycholysis developed on his fingernails. Two weeks later Department of Pediatrics Erciyes University Medical Faculty his fingernails darkened to a purple-black color and onycholysis became evident [email protected] (Picture 1). He was admitted again. Toenails were normal. Tetracycline was discontinued after 14 days of treatment and trimethoprim- sulfamethoxizole was Selim Kurtoglu Prof., M.D. added to therapy. Two months later, the nail changes resolved spontaneously. Department of Pedi atrics Erciyes University Medica l Facu lty Photosensitivity is a well-recognized complication of tetracyclines. Photo-onycholysis selimk@e rciyes.edu.tr has been observed with many of the tetracyclines usually appearing more than 2 weeks commencing drug administration (I, 2). It was first described by Segal as photosensitivity fo llowed by discoloration of the nails and onycholysis (3). Tetracyclines were reported to cause pseudoporphyria, cutaneous pigmentation, erythema multiforme, toxic epidermal necrolysis, Steven-Johnson Syndrome, fixed drug eruptions, pruritis, urticaria and vasculitis (2, 4). -
St John's Institute of Dermatology
St John’s Institute of Dermatology Topical steroids This leaflet explains more about topical steroids and how they are used to treat a variety of skin conditions. If you have any questions or concerns, please speak to a doctor or nurse caring for you. What are topical corticosteroids and how do they work? Topical corticosteroids are steroids that are applied onto the skin and are used to treat a variety of skin conditions. The type of steroid found in these medicines is similar to those produced naturally in the body and they work by reducing inflammation within the skin, making it less red and itchy. What are the different strengths of topical corticosteroids? Topical steroids come in a number of different strengths. It is therefore very important that you follow the advice of your doctor or specialist nurse and apply the correct strength of steroid to a given area of the body. The strengths of the most commonly prescribed topical steroids in the UK are listed in the table below. Table 1 - strengths of commonly prescribed topical steroids Strength Chemical name Common trade names Mild Hydrocortisone 0.5%, 1.0%, 2.5% Hydrocortisone Dioderm®, Efcortelan®, Mildison® Moderate Betamethasone valerate 0.025% Betnovate-RD® Clobetasone butyrate 0.05% Eumovate®, Clobavate® Fluocinolone acetonide 0.001% Synalar 1 in 4 dilution® Fluocortolone 0.25% Ultralanum Plain® Fludroxycortide 0.0125% Haelan® Tape Strong Betamethasone valerate 0.1% Betnovate® Diflucortolone valerate 0.1% Nerisone® Fluocinolone acetonide 0.025% Synalar® Fluticasone propionate 0.05% Cutivate® Hydrocortisone butyrate 0.1% Locoid® Mometasone furoate 0.1% Elocon® Very strong Clobetasol propionate 0.1% Dermovate®, Clarelux® Diflucortolone valerate 0.3% Nerisone Forte® 1 of 5 In adults, stronger steroids are generally used on the body and mild or moderate steroids are used on the face and skin folds (armpits, breast folds, groin and genitals).