Placenta Previa Placental Abruption Antepartum Hemorrhage
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Placental Abruption
Placental Abruption Definition: Placental separation, either partial or complete prior to the birth of the fetus Incidence 0.5 – 1% (4). Risk factors include hypertension, smoking, preterm premature rupture of membranes, cocaine abuse, uterine myomas, and previous abruption (5). Diagnosis: Symptoms (may present with any or all of these) o Vaginal bleeding (usually dark and non-clotting). o Abdominal pain and/or back pain varying from intermittent to severe. o Uterine contractions are usually present and may vary from low amplitude/high frequency to hypertonus. o Fetal distress or fetal death. Ultrasound o Adherent retroplacental clot OR may just appear to be a thick placenta o Resolving hematomas become hypoechoic within one week and sonolucent within 2 weeks May be a diagnosis of exclusion if vaginal bleeding and no other identified etiology Consider in differential with uterine irritability on toco and cat II-III tracing, as small proportion present without bleeding Classification: Grade I: Slight vaginal bleeding and some uterine irritability are usually present. Maternal blood pressure, and fibrinogen levels are unaffected. FHR remains normal. Grade II: Mild to moderate vaginal bleeding seen; tetanic contractions may be present. Blood pressure usually normal, but tachycardia may be present. May be postural hypotension. Decreased fibrinogen; with levels below 250 mg percent; may be evidence of fetal distress. Reviewed 01/16/2020 1 Updated 01/16/2020 Grade III: Bleeding is moderate to severe, but may be concealed. Uterus tetanic and painful. Maternal hypotension usually present. Fetal death has occurred. Fibrinogen levels are less then 150 mg percent with thrombocytopenia and coagulation abnormalities. -
Couvelaire Uterus Manju Rathi,1 Sunil Kumar Rathi,2 Manju Purohit,3,4 Ashish Pathak2,5
BMJ Case Reports: first published as 10.1136/bcr-2014-204211 on 31 March 2014. Downloaded from Images in… Couvelaire uterus Manju Rathi,1 Sunil Kumar Rathi,2 Manju Purohit,3,4 Ashish Pathak2,5 1Department of Obstetrics and DESCRIPTION packed cells during the surgery and two more Gynecology, RD Gardi Medical A 23-year-old primiparous woman with 37 weeks transfusions of 200 mL of packed cells were given College, Ujjain, Madhya Pradesh, India of amenorrhoea was admitted to the Obstetric in the postoperative period. She was given cefazolin 2Department of Peadiatrics, RD ward with symptoms of severe abdominal pain and 1 gm every 8 hours for 5 days in view of leucocyt- Gardi Medical College, Ujjain, non-progression of labour past 20 h. The patient osis. The rest of her postoperative stay was normal. Madhya Pradesh, India 1 fi 3 was registered for antenatal care at a peripheral Couvelaire rst described the entity in 1911. It Department of Pathology, RD health centre (PHC). She had two previous ante- is a rare non-fatal complication of severe abrup- Gardi Medical College, Ujjain, 23 Madhya Pradesh, India natal visits at the PHC. Her last visit was 15 days tion. It is estimated to complicate 5% of all cases 2 4Department of Public Health prior to admission, during which her blood pres- of abruption. The entity is infrequently reported Sciences, Global Health sure was found to be normal. In her second trimes- and the incidence is difficult to estimate because (IHCAR), Stockholm, Sweden 5 ter visit, her blood group was B positive, the diagnosis is made by direct visualisation or Department of Women and 23 Children’s Health, International haemoglobin was found to be 8.5 g/100 mL, urine biopsy. -
PLANNED OUT-OF-HOSPITAL BIRTH Approved 11/12/15
HEALTH EVIDENCE REVIEW COMMISSION (HERC) COVERAGE GUIDANCE: PLANNED OUT-OF-HOSPITAL BIRTH Approved 11/12/15 HERC COVERAGE GUIDANCE Planned out-of-hospital (OOH) birth is recommended for coverage for women who do not have high- risk coverage exclusion criteria as outlined below (weak recommendation). This coverage recommendation is based on the performance of appropriate risk assessments1 and the OOH birth attendant’s compliance with the consultation and transfer criteria as outlined below. Planned OOH birth is not recommended for coverage for women who have high risk coverage exclusion criteria as outlined below, or when appropriate risk assessments are not performed, or where the attendant does not comply with the consultation and transfer criteria as outlined below (strong recommendation). High-risk coverage exclusion criteria: Complications in a previous pregnancy: Maternal surgical history Cesarean section or other hysterotomy Uterine rupture Retained placenta requiring surgical removal Fourth-degree laceration without satisfactory functional recovery Maternal medical history Pre-eclampsia requiring preterm birth Eclampsia HELLP syndrome Fetal Unexplained stillbirth/neonatal death or previous death related to intrapartum difficulty Baby with neonatal encephalopathy Placental abruption with adverse outcome Complications of current pregnancy: Maternal Induction of labor Prelabor rupture of membranes > 24 hours 1 Pre-existing chronic hypertension; Pregnancy-induced hypertension with diastolic blood pressure greater than -
Aspirin Use to Prevent Preeclampsia and Related Morbidity and Mortality: an Evidence Update for the U.S
Evidence Synthesis Number 205 Aspirin Use to Prevent Preeclampsia and Related Morbidity and Mortality: An Evidence Update for the U.S. Preventive Services Task Force Prepared for: Agency for Healthcare Research and Quality U.S. Department of Health and Human Services 5600 Fishers Lane Rockville, MD 20857 www.ahrq.gov Contract No. HHSA-290-2015-00007-I-EPC5, Task Order No. 6 Prepared by: Kaiser Permanente Research Affiliates Evidence-based Practice Center Kaiser Permanente Center for Health Research Portland, OR Investigators: Jillian T. Henderson, PhD, MPH Kimberly K. Vesco, MD, MPH Caitlyn A. Senger, MPH Rachel G. Thomas, MPH Nadia Redmond, MSPH AHRQ Publication No. 21-05274-EF-1 February 2021 This report is based on research conducted by the Kaiser Permanente Research Affiliates Evidence-based Practice Center (EPC) under contract to the Agency for Healthcare Research and Quality (AHRQ), Rockville, MD (Contract No. HHSA-290-2015-00007-I-EPC5, Task Order No. 6). The findings and conclusions in this document are those of the authors, who are responsible for its contents; the findings and conclusions do not necessarily represent the views of AHRQ. Therefore, no statement in this report should be construed as an official position of AHRQ or of the U.S. Department of Health and Human Services. The information in this report is intended to help health care decisionmakers—patients and clinicians, health system leaders, and policymakers, among others—make well-informed decisions and thereby improve the quality of healthcare services. This report is not intended to be a substitute for the application of clinical judgment. Anyone who makes decisions concerning the provision of clinical care should consider this report in the same way as any medical reference and in conjunction with all other pertinent information (i.e., in the context of available resources and circumstances presented by individual patients). -
Antepartum Haemorrhage
OBSTETRICS AND GYNAECOLOGY CLINICAL PRACTICE GUIDELINE Antepartum haemorrhage Scope (Staff): WNHS Obstetrics and Gynaecology Directorate staff Scope (Area): Obstetrics and Gynaecology Directorate clinical areas at KEMH, OPH and home visiting (e.g. Community Midwifery Program) This document should be read in conjunction with this Disclaimer Contents Initial management: MFAU APH QRG ................................................. 2 Subsequent management of APH: QRG ............................................. 5 Management of an APH ........................................................................ 7 Key points ............................................................................................................... 7 Background information .......................................................................................... 7 Causes of APH ....................................................................................................... 7 Defining the severity of an APH .............................................................................. 8 Initial assessment ................................................................................................... 8 Emergency management ........................................................................................ 9 Maternal well-being ................................................................................................. 9 History taking ....................................................................................................... -
Rotation Objectives
Rotation Objectives Obstetrical Ward (Caseroom) Medical Expert 1. Be able to diagnose and manage the following antenatal complications a. Gestational Hypertension and preeclampsia b. Gestational diabetes c. Premature rupture of membranes d. Antepartum Bleeding (after 20 weeks gestation) e. Maternal hypothyroidism f. Intrauterine growth retardation 2. Demonstrate the following skills during antenatal visits: a. Fundal height measurements, b. Leopold manoeuvres, c. Fetal Doppler heart monitoring, d. Cervical exam 3. Be able to assess and triage risk associated with a trial of labour after Cesarian 4. Be able to assess the factors that will influence the decision to induce labour 5. Be able to perform normal risk labour and vaginal deliveries a. Demonstrate accurate cervical exams to determine the stage of labour b. Be able to evaluate for spontaneous rupture of membranes c. Fetal surveillance during labour d. Be able to diagnose non-cephalic fetal presentation using appropriate techniques 6. Be able to recognize and manage the following complications of labour and delivery a. Labour dystocia b. Shoulder dystocia c. Postpartum hemorrhage d. Perineal lacerations e. Vacuum assisted delivery f. Non-cephalic presentation g. Peripartum fever 7. Be able to manage pain in labour Postnatal and neonatal Care *The following objectives are relevant to services in which the resident is responsible for newborn assessments 8. Be able to describe and perform neonatal resuscitation 9. Demonstrate a comprehensive evaluation of a newborn in the first few days of life that will identify complications specific to the newborn period a. Hypoglycemia b. Hyperbilirubinemia c. Neonatal respiratory distress d. Congenital anomalies 10. Be able to provide support for breastfeeding, and anticipatory guidance for new mothers 11. -
Antepartum Haemorhhage(Aph)
ANTEPARTUM HAEMORHHAGE(APH) MAJ SAMIA NASREEN CLASSIFIED GYNAECOLOGIST CMH OBJECTIVES OF PRESENTATION • Identify serious causes of vaginal bleeding in the second half of pregnancy • Describe a systematic approach to identify the cause of bleeding • Describe specific treatment options based on diagnosis DEFINITION • Vaginal bleeding after 24weeks and before the delivery of the fetus. • It complicates (3-4%) of all pregnancies. • It is an obstetric emergency because it endanger the life of both the mother and fetus. • Hemorrhage remain the most frequent cause of maternal deaths. • Mild= <50 mL loss of blood, Major= 50-1000mL loss, Massive= >1000mL loss. • Bleeding >1 occasion regarded as recurrent APH. Causes of Late Pregnancy Bleeding • Placenta previa • Placental abruption • Uterine scar disruption Life Threatening • Ruptured vasa previa • Cervical polyp • Bloody show/cervical change • Cervicitis or cervical ectropion • Vaginal trauma • Cervical cancer INITIAL MANAGEMENT • Same initial steps regardless of etiology in all cases of APH • Assess vital signs, circulatory stability • Secure intravenous access, administer fluids HISTORY & EXAMINATION • Targeted history and physical • Abdominal examination for estimated fetal weight (EFW), estimated gestational age (EGA), and fetal presentation Localize tenderness Palpate for uterine contractions • Gentle speculum examination is safe • NO digital vaginal exam unless placental location is known DIAGNOSTIC EVALUATION • Continuous electronic fetal monitoring (CTG) and tocodynamometry • Ultrasound for placental location, presence of clots, and fetal presentation • Obtain baseline laboratory tests Complete blood count Blood type and antibody screen Consider: coagulation studies, blood urea nitrogen (BUN), creatinine, liver function testing, and type and cross • Prepare for possible emergent cesarean delivery PLACENTA PREVIA Grading of placenta previa Grade .1 (lateral placenta): The placenta implanted in the lower uterine segment but not reach the internal os. -
OBGYN-Study-Guide-1.Pdf
OBSTETRICS PREGNANCY Physiology of Pregnancy: • CO input increases 30-50% (max 20-24 weeks) (mostly due to increase in stroke volume) • SVR anD arterial bp Decreases (likely due to increase in progesterone) o decrease in systolic blood pressure of 5 to 10 mm Hg and in diastolic blood pressure of 10 to 15 mm Hg that nadirs at week 24. • Increase tiDal volume 30-40% and total lung capacity decrease by 5% due to diaphragm • IncreaseD reD blooD cell mass • GI: nausea – due to elevations in estrogen, progesterone, hCG (resolve by 14-16 weeks) • Stomach – prolonged gastric emptying times and decreased GE sphincter tone à reflux • Kidneys increase in size anD ureters dilate during pregnancy à increaseD pyelonephritis • GFR increases by 50% in early pregnancy anD is maintaineD, RAAS increases = increase alDosterone, but no increaseD soDium bc GFR is also increaseD • RBC volume increases by 20-30%, plasma volume increases by 50% à decreased crit (dilutional anemia) • Labor can cause WBC to rise over 20 million • Pregnancy = hypercoagulable state (increase in fibrinogen anD factors VII-X); clotting and bleeding times do not change • Pregnancy = hyperestrogenic state • hCG double 48 hours during early pregnancy and reach peak at 10-12 weeks, decline to reach stead stage after week 15 • placenta produces hCG which maintains corpus luteum in early pregnancy • corpus luteum produces progesterone which maintains enDometrium • increaseD prolactin during pregnancy • elevation in T3 and T4, slight Decrease in TSH early on, but overall euthyroiD state • linea nigra, perineum, anD face skin (melasma) changes • increase carpal tunnel (median nerve compression) • increased caloric need 300cal/day during pregnancy and 500 during breastfeeding • shoulD gain 20-30 lb • increaseD caloric requirements: protein, iron, folate, calcium, other vitamins anD minerals Testing: In a patient with irregular menstrual cycles or unknown date of last menstruation, the last Date of intercourse shoulD be useD as the marker for repeating a urine pregnancy test. -
Are Maternal and Neonatal Outcomes Different in Placental Abruption
Placenta 85 (2019) 69–73 Contents lists available at ScienceDirect Placenta journal homepage: www.elsevier.com/locate/placenta Are maternal and neonatal outcomes different in placental abruption T between women with and without preeclampsia? Min Hana, Dan Liua, Shahn Zebb, Chunfang Lia, Mancy Tongc, Xuelan Lia,**, Qi Chend,e,* a Department of Obstetrics & Gynaecology, First affiliated hospital of Xi'an Jiaotong University, China b Xi'an Jiaotong University, Xi'an, China c Department of Obstetrics, Gynecology and Reproductive Sciences, Yale School of Medicine, New Haven, CT, USA d The Hospital of Obstetrics & Gynaecology, Fudan University China, China e Department of Obstetrics & Gynaecology, The University of Auckland, New Zealand ARTICLE INFO ABSTRACT Keywords: Introduction: Placental abruption is a serious pregnancy complication that causes maternal and neonatal mor- Placental abruption tality and morbidity. Whether maternal and neonatal outcomes differ between patients who concurrently pre- Preeclampsia sented with preeclampsia and those who did not, have not been fully investigated. Maternal fetal and neonatal outcomes Methods: A total number of 158 patients with placental abruption were included. Of them, 66 concurrently had preeclampsia. Maternal and neonatal characteristics including delivery weeks, time of onset and birthweight as well as the grade of placental abruption were collected and analysed. Results: The time at diagnosis of placental abruption in patients who concurrently presented preeclampsia was significantly earlier than that in patients who did not. The number of patients with grade III placental abruption was significantly higher in patients who concurrently presented with preeclampsia, compared to patients who did not. The odds ratio of an increase in grade III placental abruption in patients who concurrently presented preeclampsia was 5.27 (95%CL: 2.346, 12.41), compared to patients who did not. -
Third Trimester Bleeding
Third Trimester Bleeding Nicole Sprawka, MD Maternal Fetal Medicine Causes of antepartum bleeding Labor Cervical bleeding Placental abruption Placenta previa Bleeding from a site above the cervix Vasa previa Bleeding in Labor Bleeding during labor is common Effacement and dilation of the cervix causes tearing of small vessels Often called “bloody show” Cervical bleeding Cervix my be friable due pregnancy or infection Cervical polyps Ask about recent intercourse Placental abruption Separation from it’s site of implantation before delivery Occurs 1 in 200 deliveries Cause of fetal death 1 in 1600 10% of all 3rd trimester stillbirth due to abruption Pathology of abruption Initiated by hemorrhage in to the decidua basalis The decidual hematoma leads to separation, compression, and destruction of the adjacent placenta In the early stages there may be no clinical symptoms The blood may dissect the membranes from the uterine wall and escape through the cervix Placental abruption The blood can be retained between the placenta and uterus resulting in a concealed abruption Concealed abruption likely go undiagnosed till delivery and are associated with a higher rate of DIC Risk factors for abruption Prior abruption 10 - 25 Preterm ruptured membranes 2.4 – 4.9 Preeclampsia 2.1 – 4.0 Chronic hypertension 1.8 – 3.0 Multifetal gestation 2.1 Polyhydramnios 2.0 Cigarette smoking 1.4 – 1.9 Increased age and parity 1.3 – 1.5 Cocaine use Uterine fibroids (esp. if behind placental implantation site) Hypertension and abruption Most common -
Couvelaire Uterus ISSN: 2394-0026 (P) ISSN: 2394-0034 (O) Case Report
Couvelaire uterus ISSN: 2394-0026 (P) ISSN: 2394-0034 (O) Case Report Couvelaire uterus - A case report Mahendra G 1*, Ravindra S. Pukale 2, Vijayalakshmi S 3, Priya 4 1Assistant Professor, 2Associate professor, 3Professor and Head, 4Junior Resident Department of Obstetrics and Gynecology , Adichunchanagiri Institute of Medical S ciences, B.G. Nagara, India *Corresponding author email: [email protected] How to cite this article: Mahendra G , Ravindra S. Pukale, Vijayalakshmi S , Priya . Couvelaire uterus - A case report. IAIM, 2015; 2(3): 142 -145. Available online at www.iaimjournal.com Received on: 03-01-2015 Accepted on: 16-01-2015 Abstract “Couvelaire uterus” or “Utero-placental apoplexy” is a rare complication of severe forms of placental abruption. It occurs when vascular damage within the placenta causes hemorrhage that progresses to and infiltrates the wall of the uterus . We presented here rare case of 23 years old f emale with Couvelaire uterus. Key words Couvelaire uterus, Utero-placental apoplexy, Placental abruption. Introduction pregnancy induced hypertension ( PIH) in “Couvelaire uterus” or “Utero -placental previous pregnancy. Her personal and family apoplexy” is a rare complication of severe forms history was not significant. of placen tal abruption. It occurs when vascular damage within the placenta causes hemorrhage General examination • that progresses to and infiltrates the wall of the Pallor +++ uterus [1]. It is a syndrome that can only be • BP - 116/80 mm Hg in t he supine left diagnosed by direct visualization or biopsy (or lateral position both). For this reason, its occurrence is perhaps • Pulse rate - 108/min underreported and underestimated in the literature [2]. -
Management of Obstetric Hemorrhage
MANAGEMENT OF OBSTETRIC HEMORRHAGE AMITAVA RUDRA*, SUMAN CHATTERJEE**, SAIKAT SENGUPTA***, RAVI WANKHEDE***, BIswaJIT NANDI****, GAURAB MAITRA***, AND JAYANTA MITRA**** Abstract Major obstetric hemorrhage is an extremely challenging obstetric emergency associated with significant morbidity and mortality. Pharmacological treatment of uterine atony has not altered much in recent years apart from the increasing use of misoprostol, although controversy surrounds its advantages over other uterotonics. Placenta accreta is becoming more common, a sequel to the rising caesarean section rate. Interventional radiology may reduce blood loss in these cases. Uterine compression sutures, intrauterine tamponade balloons and cell salvage have been introduced in the last decade. Keywords: Antepartum hemorrhage, postpartum hemorrhage, uterotonics. Obstetric hemorrhage is the world’s leading cause of maternal mortality1. Postpartum hemorrhage (PPH) accounts for the majority of these deaths1. The global maternal ratio of 402 deaths per 100,000 live births2 obscures the fact that 99% of these deaths occur in the developing world3. In addition, even in many developed countries, it is also the maternal complication for which the highest rate of substandard care is observed4. Furthermore, Zeeman’s5 study of obstetric critical care provision identifies hemorrhage as one of the most frequent reasons for admission to intensive care unit. Major obstetric hemorrhage accounts for 30% of cases6. Obstetric hemorrhage is often sudden, unexpected, and may be associated with coagulopathy. Blood loss can be notoriously difficult to assess in obstetric bleeds. Bleeding may sometimes be concealed and the presence of amniotic fluid makes accurate estimation challenging. Hence, early recognition and treatment are essential to ensure the best outcome. Therefore, it is important to have a thorough knowledge of the pathophysiology, etiology, and management strategies of obstetric hemorrhage.