Surgery Notes IV OESOPHAGEAL DISEASES 12
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An Atypical Case of Warfarin-Induced Skin Necrosis
CASE REPORT An Atypical Case of Warfarin-Induced Skin Necrosis Lindsay R. Sklar, MD* *Wayne State University, Department of Dermatology, Dearborn, Michigan Anne Messman, MD, FACEP† †Sinai-Grace Hospital, Department of Emergency Medicine, Detroit, Michigan Section Editor: Rick A. McPheeters, DO Submission history: Submitted December 19, 2016; Revision received March 8, 2017; Accepted March 30, 2017 Electronically published October 11, 2017 Full text available through open access at http://escholarship.org/uc/uciem_cpcem DOI: 10.5811/cpcem.2017.3.33373 Skin necrosis is a relatively rare, potentially fatal side effect of warfarin. It is most commonly reported within 10 days of initiation of therapy in warfarin-naïve patients. We report an atypical case of warfarin-induced skin necrosis upon recommencement of warfarin in a non-naïve warfarin patient. [Clin Pract Cases Emerg Med. 2017;1(4):359–361.] INTRODUCTION were significant for a normal platelet level of 180,000/mm,³ Warfarin is currently the most widely prescribed oral partial thromboplastin time of 27.7 sec, prothrombin time of anticoagulant in North America. Thrombosis is a rare, 21.8 sec, and an international normalized ratio (INR) of 1.87. paradoxical, and potentially fatal adverse effect of the drug. Computerized tomography (CT) of the abdomen with and Skin necrosis occurs secondary to the development of without intravenous contrast showed no evidence of microthrombi and endothelial cell damage in the vessels of malignancy or hemorrhage. dermal and subcutaneous tissues. Since this rare The patient had been on the same warfarin regimen (7.5mg complication was first recognized in 1943, there have been three days a week and 10mg four days a week) for two years an estimated 300 cases reported, affecting approximately prior to presentation to our ED; it had been initiated after the 0.01-0.1% of patients on the drug.1 Typically, warfarin- development of a deep vein thrombosis and pulmonary induced skin necrosis presents within three to ten days of embolism. -
Treatment of Equine Gastric Impaction by Gastrotomy R
EQUINE VETERINARY EDUCATION / AE / april 2011 169 Case Reporteve_165 169..173 Treatment of equine gastric impaction by gastrotomy R. A. Parker*, E. D. Barr† and P. M. Dixon Dick Vet Equine Hospital, University of Edinburgh, Easter Bush Veterinary Centre, Midlothian; and †Bell Equine Veterinary Clinic, Mereworth, UK. Keywords: horse; colic; gastric impaction; gastrotomy Summary Edinburgh with a deep traumatic shoulder wound of 24 h duration. Examination showed a mildly contaminated, A 6-year-old Warmblood gelding was referred for treatment of 15 cm long wound over the cranial aspect of the left a traumatic shoulder wound and while hospitalised developed scapula that transected the brachiocephalicus muscle a large gastric impaction which was unresponsive to and extended to the jugular groove. The horse was sound medical management. Gastrotomy as a treatment for gastric at the walk and ultrasonography showed no abnormalities impactions is rarely attempted in adult horses due to the of the bicipital bursa. limited surgical access to the stomach. This report describes The wound was debrided and lavaged under standing the successful surgical treatment of the impaction by sedation and partially closed with 2 layers of 3 metric gastrotomy and management of the post operative polyglactin 910 (Vicryl)1 sutures in the musculature and complications encountered. simple interrupted polypropylene (Prolene)1 skin sutures, leaving some ventral wound drainage. Sodium benzyl Introduction penicillin/Crystapen)2 (6 g i.v. q. 8 h), gentamicin (Gentaject)3 (6.6 mg/kg bwt i.v. q. 24 h), flunixin 4 Gastric impactions are rare in horses but, when meglumine (Flunixin) (1.1 mg/kg bwt i.v. -
A CASE REPORT Introduction Acute Pancreatitis Is an Inflammato
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Archivio istituzionale della ricerca - Università di Palermo Acta Medica Mediterranea, 2014, 30: 33 THE UNSOLVED QUESTION OF ACUTE PANCREATITIS. PROGNOSTIC CRITERIA: A CASE REPORT CLAUDIO ENNA, GIUSEPPE TAORMINA, AURELIO SEIDITA, ALBERTO D’ALCAMO, MIRIAM CARTA, FLORIANA ADRAGNA, PASQUALE MANSUETO Internal Medicine, Biomedical Department of Internal and Specialist Medicine (DIBIMIS), University Hospital of Palermo, Italy ABSTRACT Acute pancreatitis is a severe pathologic condition that requires an early identification of patients at risk of developing poten- tially lethal complications. To date, the use of clinical scoring systems and evaluation of biochemical parameters are, by far, the most widely used means to stratify the risk, even if they seem approximate and inadequate. The computed tomography severity index has proved to be superior in predicting acute pancreatitis outcome. Here we report the case of an adult male, admitted to our Department with the initial diagnosis of acute edematous pancreatitis, which was proved later to be a necrotizing pancreatitis, in which the clini- cal and laboratory prognostic scores were inadequate and discrepant with the more accurate computed tomography severity index. Key words: Acute pancreatitis, Ranson’s criteria Glasgow score APACHE II score Balthazar’s score. Received November 18, 2013; Accepted December 31, 2013 Introduction Patients with pancreatic necrosis require to be close- ly monitored in intensive care unit, with a strict clini- Acute pancreatitis is an inflammatory process cal and laboratory follow-up, associated with CT and of pancreas characterized by sudden onset and is, to MRI examination(3). -
Surgery Notes IIIII a PPROACH to ABDOMINAL MASSES 1111 IV IV OESOPHAGEAL DISEASES 1212
CONTENTS Page I TRAUMA (MULTI-SPECIALTY APPROACH) 22 IIII APPROACH TO ABDOMINAL PAIN 1100 Surgery Notes IIIII A PPROACH TO ABDOMINAL MASSES 1111 IIVV OESOPHAGEAL DISEASES 1122 For the M.B.B.S. VV UPPER BLEEDING GIT AND ITS CAUSES 2211 VVII COLORECTAL DISEASES 1199 By Andre Tan VII LIVER DISEASES 3399 VIII PANCREA TIC DISEASES 4455 IIXX BILIARY TRACT DISEASES 5511 XX BREAST DISEASES 6600 XXII HEAD AND NECK MASSES 6699 XII SALIVARY GLAND SWELLINGS 7744 XIII THYROID DISEASES 7788 XIV PERIPHERAL ARTERIAL DISEASE 8855 XV ABDOMINAL AORTIC ANEURYSM 9933 XVI PERIPHERAL VENOUS DISEASE 9955 XVII UROLOGICAL DISEASES 9999 XVIII SURGICAL INSTRUMENTS 111100 TRAUMA (MULTI-SPECIALTY APPROACH) Management o f breathing -- Supplemental oxygen -- Ventilate as required if patient requires assistance with breathing AADVANCED TTRAUMA LLIFEIFE SSUPPORT ALGORITHM -- Needle thoracotomy for tension pneumothorax, followed by chest tube MAIN PRINCIPLES: -- Occlusive dressing for open pneumothorax -- Treat greatest threat to life first -- Definitive diagnosis is less important 3.3. CIRCULATION -- Time is important – – the “golden hour” after trauma is when 30% of trauma deaths Assessment of organ perfusion occur, and are preventable by ATLS -- Level of consciousness -- Skin colour and temperature, capillary refill -- Pulse rate and character – – all major pulses APPROACH -- Blood pressure 1.1. Primary survey and Resuscitation with adjuncts 2.2. Re-evaluation of the patient Classes of haemorrhagic shock 3.3. Secondary survey with adjuncts I II III IVIV 4.4. Post-resuscitation monitoring and re-evaluation Bld loss 5.5. Optimise for transfer and definitive care Amt (ml) <750 750-1500 1500-2000 >2000 Percentage <15<15 15-30 30-40 >40>40 Ht rate <100 >100 >120 >140 PRIMARY SURVEY – – ABCDE BPBP Normal Normal Decreased Decreased Cap refill Normal Prolonged Prolonged Prolonged 1.1. -
Most Common Robotic Bariatric Procedures: Review and Technical Aspects Pablo A
Acquafresca et al. Ann Surg Innov Res (2015) 9:9 DOI 10.1186/s13022-015-0019-9 REVIEW Open Access Most common robotic bariatric procedures: review and technical aspects Pablo A. Acquafresca1, Mariano Palermo1*, Tomasz Rogula2, Guillermo E. Duza1 and Edgardo Serra1 Abstract Since its appear in the year 1997, when Drs. Cadiere and Himpens did the first robotic cholecystectomy in Brus- sels, not long after the first cholecystectomy, they performed the first robotic bariatric procedure. It is believed that robotically-assisted surgery’s most notable contributions are reflected in its ability to extend the benefits of minimally invasive surgery to procedures not routinely performed using minimal access techniques. We describe the 3 most common bariatric procedures done by robot. The main advantages of the robotic system applied to the gastric bypass appear to be better control of stoma size, avoidance of stapler costs, elimination of the potential for oro- pharyngeal and esophageal trauma, and a potential decrease in wound infection. While in the sleeve gastrectomy and adjustable gastric banding its utility is more debatable, giving a bigger advantage during surgery on patients with a very large BMI or revisional cases. Keywords: Bariatric surgery, Robotic surgery, Gastric by pass, Sleeve gastrectomy, Gastric band Background laparoscopy and robotic laparoscopy is now a controver- Since its appear in the year 1997, when Drs. Cadiere and sial topic that concerns patients and surgeons alike. Himpens did the first robotic cholecystectomy in Brussels To date, the robotic technique is reported to be at least [1], the da Vinci™ Robotic Surgical System from Intuitive as safe and effective as the conventional approach for sev- Surgical, Inc., Sunny Vale, California (Fig. -
Sample Chapter CHAPTER 16: Liver, Biliary Tract, & Pancreas Disorders BUY
Sample Chapter CHAPTER 16: Liver, Biliary Tract, & Pancreas Disorders BUY NOW mhprofessional.com 728129654 – ©2021 McGraw Hill LLC. All Rights Reserved. CMDT 2022 677 Liver, Biliary Tract, & Pancreas Disorders Lawrence S. Friedman, MD 16 JAUNDICE & EVALUATION OF ABNORMAL hepatic uptake of bilirubin due to certain drugs; or impaired LIVER BIOCHEMICAL TESTS conjugation of bilirubin by glucuronide, as in Gilbert syn- drome, due to mild decreases in uridine diphosphate (UDP) glucuronyl transferase, or Crigler-Najjar syndrome, ESSENTIALS OF DIAGNOSIS caused by moderate decreases (type II) or absence (type I) of UDP glucuronyl transferase. Hemolysis alone rarely elevates the serum bilirubin level to more than 7 mg/dL » Jaundice results from accumulation of bilirubin in (119.7 mcmol/L). Predominantly conjugated hyperbiliru- body tissues; the cause may be hepatic or binemia may result from impaired excretion of bilirubin nonhepatic. from the liver due to hepatocellular disease, drugs, sepsis, » Hyperbilirubinemia may be due to abnormalities or hereditary hepatocanalicular transport defects (such as in the formation, transport, metabolism, or excre- Dubin-Johnson syndrome, progressive familial intrahe- tion of bilirubin. patic cholestasis syndromes, and intrahepatic cholestasis of » Persistent mild elevations of the aminotransferase pregnancy) or from extrahepatic biliary obstruction. Fea- levels are common in clinical practice and caused tures of some hyperbilirubinemic syndromes are summa- most often by nonalcoholic fatty liver disease rized in Table 16–2. The term “cholestasis” denotes (NAFLD). retention of bile in the liver, and the term “cholestatic jaundice” is often used when conjugated hyperbilirubine- » Evaluation of obstructive jaundice begins with ultrasonography and is usually followed by mia results from impaired bile formation or flow. -
Journal of Clinical Toxicology Iwai Et Al., J Clin Toxicol 2014, 4:6 ISSN: 2161-0495 DOI: 10.4172/2161-0495.1000218
linica f C l To o x l ic a o n r l o u g o y J Journal of Clinical Toxicology Iwai et al., J Clin Toxicol 2014, 4:6 ISSN: 2161-0495 DOI: 10.4172/2161-0495.1000218 Case Report Open Access Utility of Upper Gastrointestinal Endoscopy for Management of Patients with Roundup® Poisoning Kenji Iwai1, Masato Miyauchi2, Daisuke Komazawa1, Ryoko Murao1, Hiroyuki Yokota2, and Atushi Koyama1 1Department of Emergency and Critical Care Medicine, Iwaki Kyoritu General Hospital, Fukushima, Japan 2Department of Emergency and Critical Care Medicine, Nippon Medical School, Tokyo, Japan *Corresponding author: Masato Miyauchi, Department of Emergency and Critical Care Medicine, Nippon Medical School, Tokyo, Japan, Tel: +81-3-3822-2131; E-mail: [email protected] Received date: Nov 03, 2014, Accepted date: Dec 05, 2014, Published date: Dec 08, 2014 Copyright: © 2014, Miyauchi M, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Abstract Introduction: Roundup® is a herbicide widely used in Japan in gardening and agriculture. When ingested, Roundup is highly toxic, but gastrointestinal decontamination, including gastric lavage, is not routinely performed after ingestion. Endoscopy may be useful in managing individuals with liquid herbicide poisoning, by identifying gastric residual contents, assessing mucosal damage and retrieving herbicide directly by aspiration. Case report: A 73 year old, 40 kg female with a history of depression was transported to our emergency room by ambulance 1 h after attempting suicide by ingesting 100 ml Roundup, which contains 48% glyphosate-potassium, and 52% surfactant and water. -
Ministry of Healthcare of Ukraine Danylo Halytsky Lviv National Medical University
MINISTRY OF HEALTHCARE OF UKRAINE DANYLO HALYTSKY LVIV NATIONAL MEDICAL UNIVERSITY DEPARTMENT OF SURGERY № 1 ACUTE PANCREATITIS Guidelines for Medical Students LVIV – 2019 2 Approved at the meeting of the surgical methodological commission of Danylo Halytsky Lviv National Medical University (Meeting report № ___ on __________ ____, 2019) Guidelines prepared: CHOOKLIN Sergiy Mykolayovych – PhD, professor of Department of Surgery №1 at Danylo Halytsky Lviv National Medical University KHOMYAK Volodymyr Vsevolodovych – PhD, associate professor of Department of Surgery №1 at Danylo Halytsky Lviv National Medical University Referees: ANDRYUSHCHENKO Viktor Petrovych – PhD, professor of Department of General Surgery at Danylo Halytsky Lviv National Medical University OREL Yuriy Glibovych - PhD, professor of Department of Surgery №1 at Danylo Halytsky Lviv National Medical University Responsible for the issue first vice-rector on educational and pedagogical affairs at Danylo Halytsky Lviv National Medical University, corresponding member of National Academy of Medical Sciences of Ukraine, PhD, professor M.R. Gzegotsky 3 I. Background The incidence of acute pancreatitis (AP) has increased during the past 20 years. Most patients develop a mild and self-limited course; however, 10% to 20% of patients have a rapidly progressive inflammatory response associated with prolonged length of hospital stay and significant morbidity and mortality. Patients with mild pancreatitis have a mortality rate of less than 1% but, in severe pancreatitis, this increases up to 10% to 30%.3 The most common cause of death in this group of patients is multiorgan dysfunction syndrome. Mortality in pancreatitis has a bimodal distribution; in the first 2 weeks, also known as the early phase, the multiorgan dysfunction syndrome is the final result of an intense inflammatory cascade triggered initially by pancreatic inflammation. -
Multidisciplinary Approach to Treating Severe Acute Pancreatitis in a Low-Volume Hospital
World J Surg (2019) 43:2994–3002 https://doi.org/10.1007/s00268-019-05114-8 ORIGINAL SCIENTIFIC REPORT Multidisciplinary Approach to Treating Severe Acute Pancreatitis in a Low-Volume Hospital 1 2 1 Alvaro Robin-Lersundi • Ana Abella Alvarez • Carlos San Miguel Mendez • 1 1 1 Almudena Moreno Elalo-Olaso • Arturo Cruz Cidoncha • Asuncio´n Aguilera Velardo • 2,3 1,4 Federico Gordo Vidal • Miguel-Angel Garcı´a-Uren˜a Published online: 22 August 2019 Ó Socie´te´ Internationale de Chirurgie 2019 Abstract Background Up to 25% of patients with acute pancreatitis develop severe complications and are classified as severe pancreatitis with a high death rate. To improve outcomes, patients may require interventional measures including surgical procedures. Multidisciplinary approach and best practice guidelines are important to decrease mortality. Methods We have conducted a retrospective analysis from a prospectively maintained database in a low-volume hospital. A total of 1075 patients were attended for acute pancreatitis over a ten-year period. We have analysed 44 patients meeting the criteria for severe acute pancreatitis and for intensive care unit (ICU) admittance. Demographics and clinical data were analysed. Patients were treated according to international guidelines and a multidisciplinary flowchart for acute pancreatitis and a step-up approach for pancreatic necrosis. Results Forty-four patients were admitted to the ICU due to severe acute pancreatitis. Twenty-five patients needed percutaneous drainage of peri-pancreatic or abdominal fluid collections or cholecystitis. Eight patients underwent endoscopic retrograde cholangiopancreatography for choledocholithiasis and biliary sepsis or pancreatic leakage, and one patient received endoscopic trans-gastric endoscopic prosthesis for pancreatic necrosis. -
Clinical Biliary Tract and Pancreatic Disease
Clinical Upper Gastrointestinal Disorders in Urgent Care, Part 2: Biliary Tract and Pancreatic Disease Urgent message: Upper abdominal pain is a common presentation in urgent care practice. Narrowing the differential diagnosis is sometimes difficult. Understanding the pathophysiology of each disease is the key to making the correct diagnosis and providing the proper treatment. TRACEY Q. DAVIDOFF, MD art 1 of this series focused on disorders of the stom- Pach—gastritis and peptic ulcer disease—on the left side of the upper abdomen. This article focuses on the right side and center of the upper abdomen: biliary tract dis- ease and pancreatitis (Figure 1). Because these diseases are regularly encountered in the urgent care center, the urgent care provider must have a thorough understand- ing of them. Biliary Tract Disease The gallbladder’s main function is to concentrate bile by the absorption of water and sodium. Fasting retains and concentrates bile, and it is secreted into the duodenum by eating. Impaired gallbladder contraction is seen in pregnancy, obesity, rapid weight loss, diabetes mellitus, and patients receiving total parenteral nutrition (TPN). About 10% to 15% of residents of developed nations will form gallstones in their lifetime.1 In the United States, approximately 6% of men and 9% of women 2 have gallstones. Stones form when there is an imbal- ©Phototake.com ance in the chemical constituents of bile, resulting in precipitation of one or more of the components. It is unclear why this occurs in some patients and not others, Tracey Q. Davidoff, MD, is an urgent care physician at Accelcare Medical Urgent Care in Rochester, New York, is on the Board of Directors of the although risk factors do exist. -
Endoscopic Management of Pancreatitis
2001 년 도 대한쉐당도연구회 학술대회 口특 강口 Endoscopic Management of Pancreatitis Joseph W. Leung, M.D., FRCP., FACP., FACG C.W. Law Professor of Medicine, Chief, Division of Gastroenterology, University of California, Davis Health System and VA Northem Califomia Health Care System Leaming objectives: distal common duct. Characteristic EUS appearance 1. Understand the role of ERCP in the diagnosis has also been described in the diagnosis of pancreas and treatment of pancreatic diseases divisum. In addition, EUS guided fine needle aspi 2. Discuss the pathophysiology of acute and chro ration cyto10gy or biopsy is an effective too1 for the mc pancreatltls diagnosis of underlying pancreatic malignancy. Des 3. Discuss the management of pain associated with pite the advances in pancreaticobili앙y imaging, thera pancreatic diseases peutic ERCP continues to play an important role in 4. Endoscopic management of acute biliary pancre the treatment of pancreatic diseases. Endoscopy is atitis, idiopathic pancreatitis and pancreas divisum useful to rule out the presence of an ampullary 5. Endoscopic management of chronic pancreatitis tumor as a cause of pancreatitis. Endoscopic brush in c1uding strictures, stones and pseudocysts cytology, aspiration bile cytology and needle biopsy 6. Discuss parasitic infestation of the pancreatic also aid in the diagnosis of under1ying pancreatic system malignancies. INTRODUCTION DEFINITIONS Diagnostic and therapeutic ERCP play an im Acute pancreatitis is defined as inflammation of portant role in the management of patients with pan the pancreas with no residual damage upon reco creaticobiliaη problems. Diagnostic pancreatogram veη , except in cases complicated by strictures or compliments ultrasound scan and CT scan in the pseudocyst formation. -
CPT Code Description Charge Amount 83498 17-Alpha
CPT Code Description Charge Amount 83498 17-alpha-Hydroxyprogester 308.41 83497 5-HIAA, SO 125.99 83516 A MYELOPEROX (MPO) AB QL 74.1 86021 AB ID LEUKOCYTE AB/SO 610.25 86022 AB ID, PLATELET ABS;SRA U 1318 86720 AB LEPTOSPIRA/SO 166.12 86850 AB SCREEN (IDC) 207.83 86850 AB SCREEN RBC EA SRM TECH 195.25 86793 AB, YERSINIA/SO 149 74018 ABDOMEN 1 VIEW 348.75 74018 ABDOMEN 1 VIEW PORTABLE 321.36 74022 ABDOMEN ACUTE COMP WSGL V 398.36 74019 ABDOMEN COMPLETE 398.36 74018 ABDOMEN SGL ANTEROPOSTERI 475.8 49083 ABDOMINAL PARACENTESIS W/ 1216.89 86870 ABID,WNJ 294.85 ABLATOR APOLLORF XL90 ASP 877.8 86900 ABO BLOOD TYPE 370 86900 ABO,BBSO 176.5 73050 AC JOINTS W/WO WEIGHTS BI 297.94 ACCUGRID RADIOGRAPH BREAS 121.36 82164 ACE, CSF SO 144.38 83519 ACHR BIND AB QT,RIA/SO MA 258 83519 ACHR BIND QNT MGP/SO 181.37 83519 ACHR BLOC QNT MGP/SO 181.37 83519 ACHR GANGL NEUR AB,RIA/SO 258 83519 ACHR MOD QNT MGP/SO 201.16 87116 ACID FAST CULTURE SO 227.33 83519 ACR BLOCKING QNT SO 181.37 83519 ACR RECEPTOR QNT SO 108.61 82024 ACTH,SO 459.3 86602 ACTINOMYCES AB/SO 64 85347 ACTIVATED CLOTTING TIME 126.93 85307 Activated Protein C Resis 216.04 97535GO ACTIVITY DAILY LIVING 15 265.91 78278 ACUTE GI BLOOD LOSS IMAGI 1326.15 82017 ACYLCARNITINES; QUANT, EA 574 85397 ADAMSTS 13 ACTIVITY/SO 796.62 ADAPTER CATH LUER 8.69 ADAPTER CONFIDENCE CEMENT 743.66 ADAPTER DLP PERFUS Y W/6 47.54 ADAPTER FIBEROPTIC SWIVEL 73.16 ADAPTER LUER LOC SHORT 3/ 2.2 ADAPTER LUER TO COLDER 15.29 ADAPTER MALE-MALE 4.57 C1776 ADAPTER PFC SIGMA FEMORAL 8474.76 ADAPTER PLUG MALE CLAVE 5.02 ADAPTER PRODIGY EXTENSION 2340 ADAPTER UROSTOMY DRAIN TU 9.09 ADAPTER VERSO AIRWAY ADUL 33.51 82952 ADDL GLUCOSE > 3 SPEC 136.24 87260 ADENOV/ RSPFAC / SO 141.75 ADHESIVE DEMABOND .07 PEN 193.48 ADHESIVE DEMABOND .07 PEN 193.48 ADHESIVE DERMABOND PEN 0.