Cardiovascular Examination 3/02/2013
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1. Intermittent Chest Pain: Angina: • Stable: (Caused By
CVS: 1. Intermittent chest pain: Angina: • Stable: (caused by chronic narrowing in one or more coronary arteries), episodes of pain are precipitated by exertion and may occur more readily when walking in cold or windy weather, after a large meal or while carrying a heavy load; the pain is promptly relieved by rest and/or sublingual glyceryl nitrate (GTN) spray, and typically lasts for less than 10 minutes. • unstable angina (caused by a sudden severe narrowing in a coronary artery), there is usually an abrupt onset or worsening of chest pain episodes that may occur on minimal exertion or at rest. • Retrosternal/ Progressive onset/ increase in intensity over 1–2 minutes/ Constricting, heavy/ Sometimes arm(s), neck, epigastrium/ Associated with breathlessness/ Intermittent, with episodes lasting 2–10 minutes/ Triggered by emotion, exertion, especially if cold, windy/ Relieved by rest, nitrates Mild to moderate. • Aggravated by thyroxine or drug-induced anemia, e.g. aspirin or NSAIDs Esophageal: • Retrosternal or epigastric/ Over 1–2 minutes; can be sudden (spasm)/ C: Gripping, tight or burning/ R: Often to back, sometimes to arms/ A: Heartburn, acid reflux/ T: Intermittent, often at night-time; variable duration/ Lying flat/some foods may trigger/ Not relieved by rest; nitrates sometimes relieve/ Usually mild but esophageal spasm can mimic myocardial infarction. 2. Acute chest pain: MI: • SOCRATES: Retrosternal/ Rapid over a few minutes/ Constricting, heavy/ Often to arm(s), neck, jaw, sometimes epigastrium/ Sweating, nausea, vomiting, breathlessness, feeling of impending death (angor animi)/ Acute presentation; prolonged duration/ ’Stress’ and exercise rare triggers, usually spontaneous/ Not relieved by rest or nitrates/ Usually severe. -
Scoliosis, Alters the Position of the Beat
Br Heart J: first published as 10.1136/hrt.8.3.162 on 1 July 1946. Downloaded from THE HEART IN STERNAL DEPRESSION BY WILLIAM EVANS From the Cardiac Department ofthe London Hospital Received June 25, 1946 The place where the apex beat appears on the chest wall depends as much on the symmetry of the thorax as on the size of the heart. A change in the alignment of the spine, the posterior fulcrum of the thoracic cage, in the form of scoliosis, alters the position of the beat. Local deformity of the ribs which form the walls of the cage will do the same thing. Deformity of the sternum, the anterior fulcrum of the thorax, as a cause of displacement of the apex beat has received less attention. The effects of depression of the sternum (pectus excavatum) on the shape and position of the heart have been studied in sixteen adults examined during the past year. DESCRIPTION OF CASES All sixteen patients had been referred for an explanation of certain signs connected with the heart, with the knowledge that deformity of the chest was present, but without appreciating that the two conditions might be related. In many of them suspicion of heart disease had led to restriction of their physical activities and to a change of design for their future livelihood. The symptoms that had caused the patients to seek medical advice in the first place were http://heart.bmj.com/ TABLE I SUMMARY OF FINDNGS IN 16 HEALTHY SUBJECTS WITH DEPRESSED STERNUM Antero-posterior Radiological findings in anterior view chest measurement _ _ Case Age Sternal in inches No. -
5 Precordial Pulsations
Chapter 5 / Precordial Pulsations 113 5 Precordial Pulsations CONTENTS MECHANICS AND PHYSIOLOGY OF THE NORMAL APICAL IMPULSE PHYSICAL PRINCIPLES GOVERNING THE FORMATION OF THE APICAL IMPULSE NORMAL APICAL IMPULSE AND ITS DETERMINANTS ASSESSMENT OF THE APICAL IMPULSE LEFT PARASTERNAL AND STERNAL MOVEMENTS RIGHT PARASTERNAL MOVEMENT PULSATIONS OVER THE CLAVICULAR HEADS PULSATIONS OVER THE SECOND AND/OR THIRD LEFT INTERCOSTAL SPACES SUBXIPHOID IMPULSE PRACTICAL POINTS IN THE CLINICAL ASSESSMENT OF PRECORDIAL PULSATIONS REFERENCES In this chapter the pulsations of the precordium will be discussed in relation to their identification, the mechanisms of their origin, and their pathophysiological and clinical significance. Precordial pulsations include the “apical impulse,” left parasternal movement, right parasternal movement, pulsations of the clavicular heads, pulsations over the second left intercostal space, and subxiphoid impulses. MECHANICS AND PHYSIOLOGY OF THE NORMAL APICAL IMPULSE Since during systole the heart contracts, becoming smaller and therefore moving away from the chest wall, why should one feel a systolic outward movement (the apical impulse) at all? Logically speaking there should not be an apical impulse. Several different methods of recording the precordial motion have been used to study the apical impulse going back to the late 19th century (1,2). Among the more modern methods, the notable ones are the recordings of the apexcardiogram (3–17), the impulse cardiogram (18), and the kinetocardiogram (19–21). While apexcardiography records the relative displacement of the chest wall under the transducer pickup device, which is often held by the examiner’s hands, the proponents of the impulse cardiography and kinetocardiography point out that these methods allow the recording of the absolute movement of the chest wall because the pickup device is anchored to a fixed point held 113 114 Cardiac Physical Examination in space away from the chest. -
Musculoskeletal Clinical Vignettes a Case Based Text
Leading the world to better health MUSCULOSKELETAL CLINICAL VIGNETTES A CASE BASED TEXT Department of Orthopaedic Surgery, RCSI Department of General Practice, RCSI Department of Rheumatology, Beaumont Hospital O’Byrne J, Downey R, Feeley R, Kelly M, Tiedt L, O’Byrne J, Murphy M, Stuart E, Kearns G. (2019) Musculoskeletal clinical vignettes: a case based text. Dublin, Ireland: RCSI. ISBN: 978-0-9926911-8-9 Image attribution: istock.com/mashuk CC Licence by NC-SA MUSCULOSKELETAL CLINICAL VIGNETTES Incorporating history, examination, investigations and management of commonly presenting musculoskeletal conditions 1131 Department of Orthopaedic Surgery, RCSI Prof. John O'Byrne Department of Orthopaedic Surgery, RCSI Dr. Richie Downey Prof. John O'Byrne Mr. Iain Feeley Dr. Richie Downey Dr. Martin Kelly Mr. Iain Feeley Dr. Lauren Tiedt Dr. Martin Kelly Department of General Practice, RCSI Dr. Lauren Tiedt Dr. Mark Murphy Department of General Practice, RCSI Dr Ellen Stuart Dr. Mark Murphy Department of Rheumatology, Beaumont Hospital Dr Ellen Stuart Dr Grainne Kearns Department of Rheumatology, Beaumont Hospital Dr Grainne Kearns 2 2 Department of Orthopaedic Surgery, RCSI Prof. John O'Byrne Department of Orthopaedic Surgery, RCSI Dr. Richie Downey TABLE OF CONTENTS Prof. John O'Byrne Mr. Iain Feeley Introduction ............................................................. 5 Dr. Richie Downey Dr. Martin Kelly General guidelines for musculoskeletal physical Mr. Iain Feeley examination of all joints .................................................. 6 Dr. Lauren Tiedt Dr. Martin Kelly Upper limb ............................................................. 10 Department of General Practice, RCSI Example of an upper limb joint examination ................. 11 Dr. Lauren Tiedt Shoulder osteoarthritis ................................................. 13 Dr. Mark Murphy Adhesive capsulitis (frozen shoulder) ............................ 16 Department of General Practice, RCSI Dr Ellen Stuart Shoulder rotator cuff pathology ................................... -
CARDIOLOGY Section Editors: Dr
2 CARDIOLOGY Section Editors: Dr. Mustafa Toma and Dr. Jason Andrade Aortic Dissection DIFFERENTIAL DIAGNOSIS PATHOPHYSIOLOGY (CONT’D) CARDIAC DEBAKEY—I ¼ ascending and at least aortic arch, MYOCARDIAL—myocardial infarction, angina II ¼ ascending only, III ¼ originates in descending VALVULAR—aortic stenosis, aortic regurgitation and extends proximally or distally PERICARDIAL—pericarditis RISK FACTORS VASCULAR—aortic dissection COMMON—hypertension, age, male RESPIRATORY VASCULITIS—Takayasu arteritis, giant cell arteritis, PARENCHYMAL—pneumonia, cancer rheumatoid arthritis, syphilitic aortitis PLEURAL—pneumothorax, pneumomediasti- COLLAGEN DISORDERS—Marfan syndrome, Ehlers– num, pleural effusion, pleuritis Danlos syndrome, cystic medial necrosis VASCULAR—pulmonary embolism, pulmonary VALVULAR—bicuspid aortic valve, aortic coarcta- hypertension tion, Turner syndrome, aortic valve replacement GI—esophagitis, esophageal cancer, GERD, peptic OTHERS—cocaine, trauma ulcer disease, Boerhaave’s, cholecystitis, pancreatitis CLINICAL FEATURES OTHERS—musculoskeletal, shingles, anxiety RATIONAL CLINICAL EXAMINATION SERIES: DOES THIS PATIENT HAVE AN ACUTE THORACIC PATHOPHYSIOLOGY AORTIC DISSECTION? ANATOMY—layers of aorta include intima, media, LR+ LRÀ and adventitia. Majority of tears found in ascending History aorta right lateral wall where the greatest shear force Hypertension 1.6 0.5 upon the artery wall is produced Sudden chest pain 1.6 0.3 AORTIC TEAR AND EXTENSION—aortic tear may Tearing or ripping pain 1.2–10.8 0.4–0.99 produce -
Cardiac Valvular Disease
CARDIOLOGY II: Cardiac Valvular Disease Ischemic Heart Disease Congenital Heart Disease (Adult presentations) Vascular Disease Rutgers PANCE/PANRE Review Course Cardiac Valvular Disease Aortic – Stenosis and Regurgitation Pulmonic Mitral Tricuspid Rutgers PANCE/PANRE Review Course Cardiac Valvular Disease Historically in US – Rheumatic in origin Still true in developing countries Now, atherosclerosis involved ? Genetic markers with AS ? Many patients are s/p surgical intervention ECHO remains best diagnostic tool Rutgers PANCE/PANRE Review Course Valvular Dz Practice Case A 22 y/o waitress presents c/o generalized, sub-sternal chest pain that is worsened with exertion. She appears anxious; she denies ETOH, tobacco, and illicit drug use. You auscultate her heart and diagnose MVP. What did you hear to make this diagnosis? Rutgers PANCE/PANRE Review Course Choices: A. A diastolic rumble B. A holo-systolic murmur C. A midsystolic click D. An opening snap Rutgers PANCE/PANRE Review Course Answer: A. A diastolic rumble B. A holo-systolic murmur C. A midsystolic click D. An opening snap Rutgers PANCE/PANRE Review Course Valvular Disease Basics Four Valves: Two main conditions: Aortic Stenosis Mitral Regurgitation or Tricuspid insufficiency Pulmonic Rutgers PANCE/PANRE Review Course Valvular Disease - localization nd Aortic area: 2 R interspace nd Pulmonic area: 2 L interspace Tricuspid area: LLSB Mitral area: Apex (Think APT. Ment — going from right to left along patient’s chest) Rutgers PANCE/PANRE Review Course The Aortic Valve www.healcentral.org UMDNJ PANCE/PANRERutgers Review CoursePANCE/PANRE (becoming Rutgers Review July 1, 2013) Course Aortic Stenosis (AS) 2 ‘routes of entry’/causes possible: Uni/bicuspid aortic valve (congenital) often presents at 50-65 y/o Degenerative or calcific aortic valve o Results from calcium deposits 2 to atherosclerosis (Genetic markers associated: “Notch 1”) So . -
Cardiology 1
Cardiology 1 SINGLE BEST ANSWER (SBA) a. Sick sinus syndrome b. First-degree AV block QUESTIONS c. Mobitz type 1 block d. Mobitz type 2 block 1. A 19-year-old university rower presents for the pre- e. Complete heart block Oxford–Cambridge boat race medical evaluation. He is healthy and has no significant medical history. 5. A 28-year-old man with no past medical history However, his brother died suddenly during football and not on medications presents to the emergency practice at age 15. Which one of the following is the department with palpitations for several hours and most likely cause of the brother’s death? was found to have supraventricular tachycardia. a. Aortic stenosis Carotid massage was attempted without success. b. Congenital long QT syndrome What is the treatment of choice to stop the attack? c. Congenital short QT syndrome a. Intravenous (IV) lignocaine d. Hypertrophic cardiomyopathy (HCM) b. IV digoxin e. Wolff–Parkinson–White syndrome c. IV amiodarone d. IV adenosine 2. A 65-year-old man presents to the heart failure e. IV quinidine outpatient clinic with increased shortness of breath and swollen ankles. On examination his pulse was 6. A 75-year-old cigarette smoker with known ischaemic 100 beats/min, blood pressure 100/60 mmHg heart disease and a history of cardiac failure presents and jugular venous pressure (JVP) 10 cm water. + to the emergency department with a 6-hour history of The patient currently takes furosemide 40 mg BD, increasing dyspnoea. His ECG shows a narrow complex spironolactone 12.5 mg, bisoprolol 2.5 mg OD and regular tachycardia with a rate of 160 beats/min. -
Ministry of Health of Ukraine Kharkiv National Medical University
Ministry of Health of Ukraine Kharkiv National Medical University PHYSICAL METHODS OF CARDIOVASCULAR SYSTEM EXAMINATION. INQUIRY AND GENERAL INSPECTION OF THE PATIENTS WITH CARDIOVASCULAR PATHOLOGY. INSPECTION AND PALPATION OF PRECORDIAL AREA Methodical instructions for students Рекомендовано Ученым советом ХНМУ Протокол №__от_______2017 г. Kharkiv KhNMU 2017 Physical methods of cardiovascular system examination. Inquiry and general inspection of the patients with cardiovascular pathology. Inspection and palpation of precordial area / Authors: Т.V. Ashcheulova, O.M. Kovalyova, O.V. Honchar. – Kharkiv: KhNMU, 2016. – 16 с. Authors: Т.V. Ashcheulova O.M. Kovalyova O.V. Honchar INQUIRY OF A PATIENT WITH CARDIOVASCULAR PATHOLOGY The main complaints in patients with cardiovascular disease include: 1. Dyspnea, asthma attacks 2. Pain in the heart region 3. Palpitations 4. Intermissions of heart beats 5. Swelling of the lower extremities and accumulation of fluid in cavities 6. Cough, hemoptysis 7. Dyspepsia 8. Asthenovegetative disorders: weakness, fatigue, decline in performance Dyspnea is a painful feeling of lack of air, one of the symptoms of heart failure, predominantly is of inspiratory type and can be associated with physical activity (in the early stages of compensation) or occur at rest (a sign of severe cardiac decompensation). It is a compensatory responsive activation of the respiratory center in case of congestion and decreased blood flow in larger and small circulation due to reduced myocardial contractility. Dyspnea is typical for heart failure on the background of valvular heart disease (especially mitral valve pathology), ischemic heart disease (angina pectoris, myocardial infarction, cardiosclerosis, arrhythmias and heart blockages), essential and symptomatic hypertension (due to chronic kidney disease, pheochromocytoma, Cushing's disease, primary aldosteronism etc.). -
The Cardiovascular Examination
CHAPTER 1 The Cardiovascular Examination KEY POINTS • The cardiovascular examination lends itself to a systematic approach. • The examination should be thorough but should be directed by the history to areas likely to be relevant. • Certain cardiovascular signs are quite sensitive and specific. • When the examination is well performed, many unnecessary investigations can be avoided. CASE 1 SCENARIO: TARA WITH 3. Pick up the patient’s hand. Feel the radial pulse. DYSPNOEA Inspect the patient’s hands for clubbing. Demon- strate Schamroth’s sign (Fig. 1.1). If there is no 34-year-old Tara was referred to the hospital by her general clubbing, opposition of the index finger (nail to practitioner. She presented with increasing dyspnoea for the nail) demonstrates a diamond shape; in clubbing last 2 weeks. She has found it difficult to lie flat in bed and this space is lost. Also look for the peripheral has been waking up frequently feeling breathless. She also stigmata of infective endocarditis. Splinter haemor- has a dry cough and has felt extremely tired for weeks. She rhages are common (and are usually caused by also has high fever with a shake. She is an intravenous drug trauma), whereas Osler’s nodes and Janeway lesions user and her general practitioner (GP) found a loud murmur (Fig. 1.2) are rare. Look quickly, but carefully, at on auscultation. each nail bed, otherwise it is easy to miss key signs. Please examine the cardiovascular system. Note the presence of an intravenous cannula and, if an infusion is running, look at the bag to see The cardiovascular system should be examined in what it is. -
Guide to History Taking and Examination
MBBS Year 4 GUIDE TO HISTORY TAKING AND EXAMINATION 2015-16 Copyright University College London Medical School University College London 1 Contents The Medical History ................................................................................ 3 How to take a Respiratory History ..................................................... 12 How to take a Cardiovascular History ............................................... 13 How to take a Locomotor History ....................................................... 14 How to take a history of pain ............................................................... 14 Presenting patients Workshop ............................................................. 15 General Tips on How to Perform an Examination ........................... 16 Dress and Behaviour Expected in Clinical Area ............................... 18 Cardiovascular Examination ................................................................ 19 Respiratory Examination ...................................................................... 24 Abdominal Examination ....................................................................... 26 Musculoskeletal Examination- GALS Screen ................................... 30 Motor Examination of Lower Limbs ................................................... 32 Examination of the Upper Limbs ........................................................ 35 Sensory Examination of Lower Limbs ................................................ 38 Cranial Nerve Examination ................................................................. -
Peripheral Vascular System Examination
06/11/1431 Ra'eda Almashaqba 1 Arteries: in the arms there are 3 arterial pulses: 1. Brachial pulse 2. Radial pulse 3. Ulnar pulse Ra'eda Almashaqba 2 1 06/11/1431 In the legs: 4 pulses 1. Femoral 2. Popliteal 3. Dorsalis pedis 4. Posterior tibialis Ra'eda Almashaqba 3 Veins Superficial veins:- - Great saphenous - Small saphenous Deep Vein: Femoral Ra'eda Almashaqba 4 2 06/11/1431 In the arms epitrochlear node: on the medial surface of the arm, 3cm above the elbow In the legs superficial inguinal node: 2 group 1. Horizontal group : lies in a chain in the anterior thigh below the inguinal ligament 2. Vertical group: cluster near the upper part of the saphenous vein. Ra'eda Almashaqba 5 Ask pt about pain In the arms or legs to assess for peripheral vascular diseases Ask if there is an intermittent claudication - Have you ever had any pain or cramping in your leg when you walk or exercise? - how far can you walk without stopping to rest? - dose the pain get better with rest? coldness, numbness, pallor in leg or feet Hair loss over the anterior tibial surface Ra'eda Almashaqba 6 3 06/11/1431 Assess pt risk factors ( HTN, Diabetes, tobacco use, hyperlipedemia, MI, CVA) Elicit symptoms of arterial spasm: - Do your fingertips ever change color in cold weather or when you handle cold objects - what color changes do you notice? ask for symptoms of venous peripheral vascular diseases: swelling of feet and leg, ulcer on lower leg near the ankle Ask if there is swelling with redness or tenderness unilateral or bilateral Ra'eda Almashaqba 7 Arms Inspect both arms from the fingertips to the shoulder. -
Common Station
COMMON STATION SPECIALIST IN PLAB 2 PREPARATION EXAMINATIONS In PLAB 2 Dr Elmira Yaghmaei Dr Hamed Salehi 1 Copyrights © 2017 Common Stations PLAB Academy – Dr Hamed Salehi. All Rights Reserved Table of Contents 1. Abdominal Examination (GI Examination) ............................................................................. 3 2. Thyroid Examination ................................................................................................................... 16 3. Unconscious Patient Examination .......................................................................................... 26 4. Meningitis Examination (Headache) ....................................................................................... 33 5. Alcoholic/Diabetic foot Examination ...................................................................................... 39 6. Hip Examination ........................................................................................................................... 51 7. Knee Examination ....................................................................................................................... 60 8. Elbow Examination ..................................................................................................................... 71 9. Whiplash Injury (Cervical Examination) ................................................................................ 80 10. Primary Survey..........................................................................................................................