Sachverzeichnis

Total Page:16

File Type:pdf, Size:1020Kb

Sachverzeichnis Sachverzeichnis A Acne papulopustulosa 329,330, Adstringenzien in der Schwanger­ ABCD-Regel 847 354 schaft 1168 AbszeB, periproktaler 1125 Acne tropicalis (siehe auch Acne Afipia felis (siehe auch Katzenkratz­ ABV-Schema 974 fulminans) 332 krankheit) 167, 168 ABV/ADV-Schema 974 Acne varioliformis 342 Agaven, Hyperpigmentierungen Acanthosis nigricans maligna 911 Acne venenata (siehe auch 792 Accutane, 13cis VAS- 291,292 Kosmetikaakne) 337, 338 Aids (siehe auch HIV-Infektion) ACE-Hemmer, Psoriasis vulgaris Acrivastin 240 197 268 Acrodermatitis Akarophobie 1249 Acetylsalicylsaure, Thrombozyto- - chronica atrophicans 101, 108, Akne (siehe auch Acne) 318 ff. pathie, erworbene 590 713,1192 - AlkoholgenuB 326 Acic10vir 56,57, 64ff. - continua suppurativa Hallopeau - Anabolika 320 - Dosierung 67, 78 302 - Androgene 319 -- bei HIV-Infektion 70 - enteropathica 704,705,915, 1075 - apokrine (siehe auch Acne - Ekzema herpeticatum 69 -- Erbgang 705 inversa) 332 - Nebenwirkung 78 -- intestinale Symptome 705 - Atiologie 318 - vulvovaginitis herpetica 68 -- Klinik 705 - BPO (siehe auch BPO, Akne) - Varizella-Zoster-Infektion -- ResorptionsstOrung 705 327 72, 73 -- Substitution 705 - Chlorakne (siehe auch dort) 338 - Wirkmechanismus 78 -- Zink-DL-Aspartat 705 - Dehydrotestosteron 319 - Wirkspektrum 78 -- Zinkgabe, prophylaktische 705 - Dehydroepiandrosteronsulfat - Zoster-Infektion (siehe auch Actinobacillus actinomycetemcomi­ 319 dort) 73 ff. tans 1054 - Detergensakne 337 Acitretin (siehe auch Retinoide) Actinomadura madurae (siehe auch - Dokumentation 320, 321 290, 291, 302 ~yzetoma) 152, 153 - Einteilung in Schweregrade 321 Acne (siehe auch Akne) 328ff. Actinomyces (siehe auch Aktino- - Emahrungsfaktoren 318,326 Acne comedonica 328,354 mykose) 150 - genetische Untersuchung 318 - Aknetoilette 329 - A. bovis 150 - Graduierung 321 - Azelainsaure 328 - A. israeli 150 - Hyperkeratose, follikulare 319 - Komedonenextraktion 329 - A. meyeri 150 - Hyperkolonisation, mikrobielle - Tretinoin 328 Adamantanamin 79 319 Acne conglobata 330-332 Addison-Krankheit (siehe auch - HyperseborrhO 319 Acne excoriee 335,336, 1258 Hyperpigmentierungen) 785 - Kosmetikaakne (siehe auch dort) Acne fulminans 330- 334 Adenome 830 337,338 - Antibiotikatherapie 332 - A. sebaceum 341 - Linolensaure, Defizit 319 - Isotretinoin 333 AderlaBbehandlung (Phlebotomie) - ~allorca-Akne (siehe auch dort) - isoretinoidinduzierte 331 758, 759 340,341 - Kortikosteroide 332 - Entleerung der Eisenspeicher - Olakne 338 - viszerale Symptomatik 332 758 - pathogenetische Faktoren 319 Acne infantilis 334, 335 - Entnahmemengen 759 - Patientengesprach 321 - Azelainsaure 335 - nach Ippen 758 - Pityrosporon - Behandlung 334 - kombinierte AderlaB- und Chloro- -- P. acnes 325 - BPO 334 quinbehandlung 759 -- P. orbiculare 325 - Komedonen 334 - praktisches Vorgehen 759 - propionibacterium acnes 320 Acne inversa 331, 332 - Serumeisenspiegel 758 - Rauchen 326 Acne juvenilis (siehe auch Acne Adiponecrosis subcutanea neonato- - Schwangerschaft 318 infantilis) 334, 335 rum 645,646 - Schweregrad 318 Acne keloidalis (siehe auch Aknen­ Adiposalgie 658 - Spatakne (siehe auch dort) 335 arben) 333,334 Adipositas dolorosa 658 - Steroidakne 337 Acne necroticans 342 Adipozyten • 644 - Talgdrtisen 319 Acne nodulocystica (siehe auch Acne Adnexkarzinome 905 - Testosteron, freies 319 conglobata) 330 Adnextumoren 830 - Therapie 322 ff. 1306 Sachverzeichnis -- Aknetherapeutika (siehe auch - Vitamin Bu 337 Allergene (siehe auch Allergie) dort) 322 ff . Aknetherapie 320ff., 338, 339 207,237 -- Aknetherapie (siehe auch dort) - Angriffspunkte 324 - leukozytoklastisches Allergoid 320 ff., 338 - Atopiker 339 (siehe auch leukozytoklastische - UV-Bestrahlungen 326 - Compliance 338 Vaskulitis) 441 ff. - UVA-Strahlung 319 - Dysmorphophobie 339 - Typ I-Reaktion (siehe auch dort) - Wirtsreaktion, immunologische - Hauttyp 339 207,237 319 - Strategie 328 - Typ IV-Reaktion 207 aknegene Medikamente (siehe Akne­ - therapierefraktarer Aknepatient Allergie therapeutika) 337 338 - Blepharoconjunctivitis allergica Aknenarben 333,334,1262,1278 - Therapieplanung 320,321 204 - Dermabrasio (siehe auch dort) Aknetoilette 325, 327, 354 -- Nickelallergie 206 333 Akrodermatitis 574, 1187 - Granulomatose, allergische - Kollagenunterspritzung (siehe auch - papulosa eruptiva infantum (Churg-Strauss) 454 dort) 333 1187 - Hausstaubmilbenallergie 210 - Kortikosteroidinjektion, intra­ - verruciformis 574 - Hyposensibilisierung 213 lasionale 333 Akrokeratose Bazex 912 - Kuhrnilchallergie 212 - Kryotherapie und Kryopeeling Akromegalie 1050 - Nahrungsmittelallergien -intoleran- (siehe auch dort) 333, 334 aktinische Keratosen 798-800,832, zen (siehe auch dort) 209,212, - lokale Exzission 333 1299,1302 213, 374ff. - Rinderkollageninjektion 333 - aromatische Retinoide 799 - Penicillinallergie (siehe auch - Rotationsstanze 333 - Chemotherapie, lokale 799 dort) 4,5, 11, 12, 150, 183 - Steroidcremes, lokale fluorierte - chirurgische Exzision 798 - photoallergische Reaktionen der topische 333 - COz-Laserbehandlung 799 Haut 736, 742, 743 - therapeutisches Vorgehen 333 - Cornua cutanea 1299 - Rhinitis allergica 204 - Tretinoin 333 - Curettage 798 - Vaskulitis, allergische (siehe auch Aknetetrade (siehe auch Acne - Elektrodesikkation 798 leukozytoklastische Vaskulitis) inversa) 331 - Etretinat 799 441 ff. - in der Schwangerschaft 1162 - Kryotherapie 798 Allethrin, Pedikulosis 113 Aknetherapeutika 322 ff. - Rontgentherapie 800 Allopurinol - all-trans-Retinsaure 322 aktinisches - Gicht 690 - Antibiotika, topische 328 - Granulom 1299 - Leishmaniose 127 - Auswahl der Therapeutika 328 - Retikuloid 990 - Necrolysis toxica combustiformis - Azathioprin 337 Aktinomykose 150 ff. 393 - Azelainsaure (siehe auch dort) - Penicillin 151 Allylamine 21, 32 322-324,328 - primare kutane 150 Alopecia mucinosa (siehe auch - Benzoylperoxid 324, 328 - Prognose 150 Mucinosis follicularis) 702, 994 - Chinin 337 - systemische 150 a-Sympathomimetika, Varikosis - Chinolone 324 - zervikale 176 678 - Chlormadinonacetat 324 - zervikofaziale 150 aI-Antitrypsin 650 - Chloroxylenol 325 Aktinomyzeten (siehe auch al-Proteaseninhibitor 650 - Cimetidin 323 Myzetoma) 152, 153 Altershaut, Pflege 1285 - Clindamycin 324 Aktinomyzetome (siehe auch - fettende Externa 1285 - Cyproteronacetat 323, 324 Myzetoma) 152, 153 - Feuchthaltefaktoren 1285 - Dequaliniumdecylenat 325 Aktivkohle, Urokinasetherapie - Mukopolysaccharide, saure 1285 - Erythromycin 324 684 - Pflegekosmetik, vorbeugende - Hexachlorophen 325 Albendazol 1285 - Hormone, antiandrogene 328 - Larva migrans 133 - Riickfetter, langer wirksame 1285 - Isoniacid 337 - Strongyloidiasis, kutane 142 Alterung der Haut 1282 ff. - Isotretinoin (siehe auch dort) -- Dosierung 142 - antioxidative MaBnahmen 1285 323,324,327,328 -- Nebenwirkung 142 - Ascorbinsaure 1285,1287 - Kortikosteroide 337 -- Wirkungsspektrum 142 - ~-Carotin 1287 - Lithium 337 Albinismus, Leukoderm 770 - Blepharoplastik (siehe auch dort) - Nebenwirkungen 326 Alfahydroxysauren 1289 1297 - Phenobarbital 337 - Glattung lichtgealterter Haut - Brauenanhebung durch Kranz­ - Resorcin 325 1289 schnitt (siehe auch dort) 1297 - Rubbelcremes 326 - Hautalterung, Prophylaxe 1289 - Chemopravention 1287 - Salicylspiritlls 323 - Kollagen, Neusynthese 1289 - dermatochirurgische MaBnah- - Schwefel 325 Alfaketosauren 1289 men 1296 - Spironolacton 323 - Glattung lichtgealterter Haut - DNS-Veranderungen 1282 - Tetracycline 324, 328 1289 - "face lifts" 1296 -- systemische 328 - Hautalterung, Prophylaxe 1289 - Isotretinoin, hochdosiertes 1287 - Thiouracil 337 - Kollagen, Neusynthese 1289 - Lebensweise 1284 - Tretinoin 322,324,328 Alimemazin, atopische Dermatitis - Lichtalterung (siehe auch dort) - Vitamin A-Saure 322 214, 220, 238 1284 Sachverzeichnis 1307 - Nicotinamid 1287 Amyloidose 694, 695, 1050 Anamie - Pflege (siehe Altershaut, Pflege) - A. cutis nodulatris atrophicans - perniziose 1050 1285 695 - sideroachrestische 752 - pharmakologische MaBnahmen - bullose 695 - spharozytare 146 1287 - Definition 693 anaphylaktischer Schock (siehe - praventive MaBnahmen 1284 - Lichen amyloidosis (siehe dort) Schock, a.) 362 - Rhytidektomie (siehe auch dort) 695, 708 ANCA-(antineutrophile, zytoplasma­ 1296 - Pathogenese 693 tische Antikorper)-Serologie 452, - Schaden durch freie Radikale -- primare 693 453 (siehe auch Radikale, freie) 1282 - Pseudosklerodermien 507 - cANCA 453 - Tocopherole 1287 - sekundare 693 - diagnostische Bedeutung 453 - Tretinoin (siehe auch dort) 1288 Analekzem 1124 - pANCA 453 - Vitamin A 1285 - akutes 1124 - Wegener-Granulomatose (siehe - Vitamin E 1285 -- Brillantgrun 1124 auch dort) 452, 453 - Xeroderma pigmentosum 1287 -- Ekzem, toxisch-degeneratives ANCA-Autoantikorper 438 - Zeitalterung (siehe auch dort) 1124 Androgene, Akne 319 1284 -- Gentianaviolett 1124 andrologische StOrungen 1205 Aluminiumchloridhexahydrat, -- Hamorrhoiden 1124 - Androgene 1214 Hyperhidrosis 718, 719 -- Kontaktekzem, allergisches - Antiostrogene 1214 Aluminiumhydrochlorid, Hyper- 1124 - Carnitin 1206 hidrosis 719 -- Pruritus 1124 - Clomifen-Test 1205, 1206 Alzianblaufarbung 699 - chronisches 1124 - diagnostisches Vorgehen 1205 Amantadin 56 -- Kortikosteroid 1124 - Fruktose-Wert 1206 Ameisenbisse 145 -- Sitzbader 1124 - FSH 1205 Amikacin 14, 151, 153 -- Teerzusatze 1124 - Funktionsdiagnostik, erweiterte - Myzetoma 153 - Hamorrhoiden, innere 1124 1206, 1207 5-Arninolavulinsaure 815 - Kontaktallergie 1124 -- akrosomale Reaktion 1207 Aminoacyl-tRNS-Synthetasen, Anti- - neoplastische Wucherung 1124 -- Chromatinkondensierung
Recommended publications
  • Diagnostiek En Therapie in De Dermatologie
    Diagnostiek en therapie in de dermatologie Leidraad voor co-assistenten Maatschap Dermatologie St. Antonius Ziekenhuis, locatie Nieuwegein St. Antonius Ziekenhuis, locatie Utrecht Polikliniek Houten Polikliniek Vleuterweide Diagnostiek Het stellen van een dermatologische diagnose De dermatologie onderscheidt zich van de andere specialismen vanwege het feit dat het lichamelijk onderzoek voorafgaat aan de anamnese. De anamnese is vervolgens toegespitst op de bevindingen bij het lichamelijk onderzoek, en kan bestaan uit een speciële, dermatologische anamnese en/of een gerichte, interne anamnese. Het is voor het stellen van een dermatologische diagnose van het grootste belang om systematisch te werk te gaan. Dit geldt vooral voor personen met minder ervaring. Met name het systematisch beschrijven van wat je ziet, en vervolgens dit “plaatje” te verwoorden in een zogenaamde “efflorescentie” zijn bij het stellen van een diagnose onontbeerlijk. Een efflorescentie is een zichtbaar bestanddeel van een huidafwijking. Voor een overzicht van de efflorescenties zij verwezen naar pagina 3. De huidafwijking kan monomorf zijn, dat wil zeggen dat het bestaat uit slechts één efflorescentie,maar het kan ook uit enkele of vele efflorescenties zijn opgebouwd. Het is belangrijk bij iedere huidafwijking de meest kenmerkende efflorescentie of combinatie van efflorescenties te kiezen. Zij bepalen de morfologische diagnose, wat op zijn beurt weer een opstapje vormt voor de differentiële diagnose. Op pagina 4 is hiervan een overzicht weergegeven. Een goede richtlijn bij het beschrijven van een afwijking is het zogenaamde “PROVOKE”-systeem. PROVOKE is een acroniem voor: Plaats, Rangschikking, Omvang (aantal, grootte), Vorm, Omtrek (begrenzing), Kleur en Efflorescentie(s). Valkuilen bij het beschrijven kunnen zijn veranderingen die zich in de loop van de tijd met de huidafwijking hebben voorgedaan.
    [Show full text]
  • Urticaria from Wikipedia, the Free Encyclopedia Jump To: Navigation, Search "Hives" Redirects Here
    Urticaria From Wikipedia, the free encyclopedia Jump to: navigation, search "Hives" redirects here. For other uses, see Hive. Urticaria Classification and external resourcesICD-10L50.ICD- 9708DiseasesDB13606MedlinePlus000845eMedicineemerg/628 MeSHD014581Urtic aria (or hives) is a skin condition, commonly caused by an allergic reaction, that is characterized by raised red skin wheals (welts). It is also known as nettle rash or uredo. Wheals from urticaria can appear anywhere on the body, including the face, lips, tongue, throat, and ears. The wheals may vary in size from about 5 mm (0.2 inches) in diameter to the size of a dinner plate; they typically itch severely, sting, or burn, and often have a pale border. Urticaria is generally caused by direct contact with an allergenic substance, or an immune response to food or some other allergen, but can also appear for other reasons, notably emotional stress. The rash can be triggered by quite innocent events, such as mere rubbing or exposure to cold. Contents [hide] * 1 Pathophysiology * 2 Differential diagnosis * 3 Types * 4 Related conditions * 5 Treatment and management o 5.1 Histamine antagonists o 5.2 Other o 5.3 Dietary * 6 See also * 7 References * 8 External links [edit] Pathophysiology Allergic urticaria on the shin induced by an antibiotic The skin lesions of urticarial disease are caused by an inflammatory reaction in the skin, causing leakage of capillaries in the dermis, and resulting in an edema which persists until the interstitial fluid is absorbed into the surrounding cells. Urticarial disease is thought to be caused by the release of histamine and other mediators of inflammation (cytokines) from cells in the skin.
    [Show full text]
  • 5: 1237-1249, 1979 the Naeglerial Causation Of
    Medical Hypotheses 5: 1237-1249, 1979 THE NAEGLERIAL CAUSATION OF RHEUMATOID DISEASE AND MANY HUMAN CANCERS. A NEW CONCEPT IN MEDICINE. R. Wyburn-Mason, 2 Hillbrow, Richmond Hill, Richmond, Surrey, England. ABSTRACT Man and terrestrial animals live in an environment containing free- living amoebae on the surface soil, in pools, fresh water lakes, rivers and streams. They form cysts, which float in the air and which are continually inhaled and found in the nasopharynx and their trophozoites are present in human and animal faeces. Amoebae of the genus, Naegleria, have been demon- strated in all human tissues, both healthy and in larger numbers in those taken from cases of rheumatoid disease, in all human cancers and in the unaffected tissues of cancer patients. They can be killed in vitro by a series of different anti-amoebic substances and treatment of active cases of rheumatoid disease by any of these, either causes cessation of disease activity or a temporary exaggeration of symptoms followed by their lessening or disappearance (Herxheimer reaction), indicating the presence of an amoeba in the affected tissues as the causative organism of the inflammation in this disease in subjects genetically sensitive to the organism. Every internal organ may be involved in the inflammatory response in cases of rheumatoid disease and this also ceases with the above treatments. Many of these internal lesions are premalignant, so that infection with the organism either in sensitive subjects or with pathogenic species, appearsto be the primary cause of cancer in many cases. The presence in the body of Naegleria represents the source of the constant antigenic stimulation thought to be responsible both for rheumatoid disease and for the development of lymphomata and myelomatosis.
    [Show full text]
  • Dr. S. Srinivasa Ravi Dr. Anusha Pilla ABSTRACT KEYWORDS
    ORIGINAL RESEARCH PAPER Volume-6 | Issue-12 | December-2017 | ISSN No 2277 - 8179 | IF : 4.176 | IC Value : 93.98 INTERNATIONAL JOURNAL OF SCIENTIFIC RESEARCH CUTANEOUS MANIFESTATIONS OF INTERNAL MALIGNANCIES – A STUDY OF 217 CASES Oncology Dr. S. Srinivasa D.DLO, MD (DVL), Assistant Professor in DVL, Department of DVL, Rangaraya Ravi Medical College, Kakinada. Dr. Anusha Pilla DDVL, Senior Resident in DVL, Andhra Medical College ,Visakhapatnam. ABSTRACT BACKGROUND : Skin manifestations act as markers for internal malignancy. There may be direct and indirect involvement of skin in various malignancies. Cutaneous manifestations may act as early markers for internal malignancy and various skin manifestations both specific and non specific were seen during the course of malignancy. AIM : The main purpose of this study is to know the incidence and clinical pattern of skin changes both specific and non specific seen in various internal malignancies. MATERIALS AND METHODS : It is an open prospective and observational study. All confirmed malignancy cases attending to the Out patient departments of Radiotherapy and DVL at Government General Hospital, Kakinada were recorded over a period of one year from MAY 2005 to MAY 2006. Total 217 cases of various malignancies were seen during the above period. These cases were clinically evaluated for skin manifestations and the demographic data was noted. These cases were thoroughly clinically examined for various skin manifestations. Relevant haematological, biochemical investigations and skin biopsies were
    [Show full text]
  • 1. Upward Movement of the Thyroid Gland Is Prevented Due To?
    1. Upward movement of the thyroid gland is prevented due to? a) Berry ligament b) Pretracheal fascia c) Sternothyroid muscle d) Thyrohyoid membrane Correct Answer - B Ans: B. Pretracheal fascia The thyroid gland is covered by a thin fibrous capsule, which has an inner and an outer layer. The inner layer extrudes into the gland and forms the septum that divides the thyroid tissue into microscopic lobules. The outer layer is continuous with the pretracheal fascia, attaching the gland to the cricoid and thyroid cartilages via a thickening of the fascia to form the posterior suspensory ligament of the thyroid gland also known as Berry's ligament. This causes the thyroid to move up and down with the movement of these cartilages when swallowing occurs. Gray's Anatomy: The Anatomical Basis of Clinical Practice, 41e ,Page no 470 2. The reason for the long left recurrent laryngeal nerve is due to the persistence of which arch artery? a) 3rd arch b) 4th arch c) 5th arch d) 2nd arch Correct Answer - B Ans: B. 4th arch Left RLN winds around the arch of aorta Arch of aorta is derived from the 4th arch Langmans Medical Embryology 13th edition (Page no 88,239) 3. Ligation of the hepatic artery will impair blood supply in a) Right gastric and Right gastroepiploic artery b) Right gastric and Left gastric artery c) Right gastroepiploic and short gastric vessels d) Right gastric and short gastric vessels Correct Answer - A Ans: A. Right gastric and Right gastroepiploic artery The right gastric artery is a branch of the common hepatic artery The right gastroepiploic artery is a branch of the gastroduodenal artery which is a branch of the common hepatic artery The left gastric artery is a branch of the celiac trunk Short gastric vessels arise from the splenic artery Gray's Anatomy: The Anatomical Basis of Clinical Practice, 41st Edition (Page nos 1116 and 1117) 4.
    [Show full text]
  • 79. Szám Keny Ség Re Pü Lõ Egész Sé Gi Feltételeirõl
    A MAGYAR KÖZTÁRSASÁG HIVATALOS LAPJA Bu da pest, TARTALOMJEGYZÉK Ol dal 2005. jú ni us 14., 2005: XLVI. tv. A ma gyar állam polgár ság ról sz óló 1993. évi LV. törvény és a kedd kül föl di ek be uta zá sá ról és tar tóz ko dá sá ról szóló 2001. évi XXXIX. törvény módosításáról......................... 3736 2005: XLVII. tv. Az igaz ság ügyi szak ér tõi te vé keny ség rõl ................... 3741 2005: XLVIII. tv. Az igaz ság ügyi szak ér tõ nempe res el já rás ban tör té nõ ki ren de lé- sé rõl és ezzel össze füg gés ben a Pol gá ri Per rend tar tás ról szóló 1952. évi III. törvény módosításáról ..................... 3753 22/2005. (VI. 14.) HM–EüM e. r. Az ál lam i célú lé gi köz le ke dés ben foly ta tott szak szol gá la ti te vé - 79. szám keny ség re pü lõ egész sé gi feltételeirõl...................... 3756 75/2005. (VI. 14.) KE h. Po li ti kai ál lam tit kár fel men té sé rõl .......................... 3807 76/2005. (VI. 14.) KE h. Köz igaz ga tá si ál lam tit kár fel men té sé rõl...................... 3807 77/2005. (VI. 14.) KE h. Köz igaz ga tá si ál lam tit kár ki ne ve zé sé rõl ..................... 3807 78/2005. (VI. 14.) KE h. Rek to ri meg bí zá sok ról ................................... 3808 A Föld mû ve lés ügyi és Vi dék fej lesz té si Mi nisz té rium Heves Megyei Föld mû ve lés ügyi Hi va ta lá nak hir det mé nye .........
    [Show full text]
  • Zdravotnícke Štúdie
    OBSAH Príhovor ................................................................................................................................................................................................2 Kubala, J.: Klinická aplikácia vysokosenzitívneho troponínu T v triáži pacientov s hrudným dyskomfortom Clinical Application of High-sensitivity Troponin T in the Triad of Patients with Chest Discomfort .................................................3 Rutowski, J. A.: Leki wpływające na układ hemostazy oraz ich działania i zastosowanie Drugs Acting on the Regulation of Hemostasis, their Actions and Applying .....................................................................................8 Tupý, J., Šemráková, V., Tupá, M., Klementíková, V.: Trombocytopénie v tehotenstve – výberové kazuistiky Thrombocytopenia in Pregnancy - Case Reports ..............................................................................................................................16 Simočková, V., Hlušková, D.: Ošetrovateľstvo cez prizmu legislatív Nursing Throughout the Legislation Prism ......................................................................................................................................28 Kaščáková, M., Majerníková, Ľ., Obročníková, A.: Uplatňovanie preventívnych opatrení pri diabetes melitus u pacientov s diabetickou retinopatiou Application of Preventive Measures in Diabetes Mellitus in Patients with Diabetic Retinopathy ....................................................33 Madarász, Š.: Sú potrebné neurorehabilitačné
    [Show full text]
  • Obligate and Facultative Paraneoplastic Dermatoses: an Overview
    DERMATOLOGY PRACTICAL & CONCEPTUAL www.derm101.com Obligate and facultative paraneoplastic dermatoses: an overview Stefano Caccavale1, Gabriella Brancaccio1, Marina Agozzino1, Paola Vitiello1, Roberto Alfano2, Giuseppe Argenziano1 1 Dermatology Unit, University of Campania Luigi Vanvitelli, Naples, Italy 2 Department of Anesthesiology, Surgery and Emergency, University of Campania Luigi Vanvitelli, Naples, Italy Key words: dermatological paraneoplastic syndromes, obligate paraneoplastic dermatoses, facultative paraneoplastic dermatoses, malignancy, oncological dermatology Citation: Caccavale S, Brancaccio G, Agozzino M, Vitiello P, Alfano R, Argenziano G. Obligate and facultative paraneoplastic dermatoses: an overview. Dermatol Pract Concept. 2018;8(3):191-197. DOI: https://doi.org/10.5826/dpc.0803a09 Received: January 15, 2018; Accepted: March 9, 2018; Published: July 31, 2018 Copyright: ©2018 Caccavale et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Funding: None. Competing interests: The authors have no conflicts of interest to disclose. All authors have contributed significantly to this publication. Corresponding author: Stefano Caccavale, MD, Dermatology Unit, University of Campania Luigi Vanvitelli, Via Sergio Pansini, 5, 80131 Naples, Italy. Email: [email protected] ABSTRACT Dermatological paraneoplastic syndromes are a group of cutaneous diseases associated with malig- nancy, but not directly related to the primary tumor itself or to its metastases. It is of utmost impor- tance for the dermatologist to recognize the major cutaneous paraneoplastic syndromes to diagnose the underlying tumors that trigger them as early as possible. In this overview, skin conditions that are highly correlated with malignancy, whose recognition implies a mandatory investigation of internal cancer, are described.
    [Show full text]
  • Cutaneous Symptoms and Syndromes in Pathology of Digestive Organs Y
    Этот документ создан нерегистрированной версией Document2PDF Pilot 2.19. Cutaneous symptoms and syndromes in pathology of digestive organs Y. S. Tsimmerman, I. Y. Tsimmerman Perm State Medical Academy n. a. acad. E. A. Vagner, Perm, Russia Keywords: skin symptoms and syndromes, pathogenesis, disease of the digestive system, liver disease, biliary tract and pancreas, the diagnostic value It is worth admitting that modern doctors insufficiently use classical methods of clinical studies in the diagnosis of patients, due to having poor knowledge of them. In particular, little attention is paid to examining of skin, the diagnostics value of the skin characteristic changes and its appendages (hair, nails) in various internal diseases. Meanwhile, the skin is a kind of screen or mirror, reflecting various pathological processes occurring in the human body, especially in the digestive system [9, 10, 17, 41, 43, 52, 57, 58]. Inspection of skin allows to diagnose certain diseases and pathological processes already at the first meeting with the patient, much earlier than the results of modern, highly labor-intensive and expensive instrumentation and laboratory research. Moreover, on the basis of changes in the skin, identified during the examination, it is possible, suspecting a specific disease, to guide further diagnostic search in a certain way to purposefully seek confirmation of the existence of the disease assumed, using only methods and laboratory diagnosis, which will quickly and reliably establish a final diagnosis. In this article, we give a brief review of the details, making it possible to summarize the known facts on the different changes of the skin, reflecting a greater or lesser degree of pathological processes in the gastro-intestinal tract, hepatobiliary system, and pancreas (pancreatic) .
    [Show full text]
  • British Journal of Dermatology
    British Journal of Dermatology December 2007 - Vol. 157 Issue 6 Page I-1335 Contents pages I–XI Snippets Research Snippets pages xxi–xxi Topical review Paraneoplastic hypertrichosis lanuginosa acquisita: uncommon or overlooked? P.H.T.J. Slee, R.I.F. van der Waal, J.H. Schagen van Leeuwen, R.A. Tupker, R. Timmer, C.A. Seldenrijk and M.A.M. van Steensel pages 1087–1092 Review articles The relationships between exposure dose and response in induction and elicitation of contact hypersensitivity in humans P.S. Friedmann pages 1093–1102 Psoriasis: evolution of pathogenic concepts and new therapies through phases of translational research E. Guttman-Yassky and J.G. Krueger pages 1103–1115 Guidelines Guidelines for evaluation and management of urticaria in adults and children C.E.H. Grattan and F. Humphreys on behalf of the British Association of Dermatologists Therapy Guidelines and Audit Subcommittee pages 1116–1123 Original articles Cutaneous biology Cathelicidin LL-37 induces the generation of reactive oxygen species and release of human α- defensins from neutrophils Y. Zheng, F. Niyonsaba, H. Ushio, I. Nagaoka, S. Ikeda, K. Okumura and H. Ogawa pages 1124–1131 Microarray analysis of aberrant gene expression in actinic keratosis: effect of the Toll-like receptor-7 agonist imiquimod A. Torres, L. Storey, M. Anders, R.L. Miller, B.J. Bulbulian, J. Jin, S. Raghavan, J. Lee, H.B. Slade and W. Birmachu pages 1132–1147 Biphasic expression of stromal cell-derived factor-1 during human wound healing A. Toksoy, V. Müller, R. Gillitzer and M. Goebeler pages 1148–1154 Original articles Clinical and laboratory investigations CTACK /CCL27 expression in psoriatic skin and its modification after administration of etanercept A.
    [Show full text]
  • Us 2018 / 0305689 A1
    US 20180305689A1 ( 19 ) United States (12 ) Patent Application Publication ( 10) Pub . No. : US 2018 /0305689 A1 Sætrom et al. ( 43 ) Pub . Date: Oct. 25 , 2018 ( 54 ) SARNA COMPOSITIONS AND METHODS OF plication No . 62 /150 , 895 , filed on Apr. 22 , 2015 , USE provisional application No . 62/ 150 ,904 , filed on Apr. 22 , 2015 , provisional application No. 62 / 150 , 908 , (71 ) Applicant: MINA THERAPEUTICS LIMITED , filed on Apr. 22 , 2015 , provisional application No. LONDON (GB ) 62 / 150 , 900 , filed on Apr. 22 , 2015 . (72 ) Inventors : Pål Sætrom , Trondheim (NO ) ; Endre Publication Classification Bakken Stovner , Trondheim (NO ) (51 ) Int . CI. C12N 15 / 113 (2006 .01 ) (21 ) Appl. No. : 15 /568 , 046 (52 ) U . S . CI. (22 ) PCT Filed : Apr. 21 , 2016 CPC .. .. .. C12N 15 / 113 ( 2013 .01 ) ; C12N 2310 / 34 ( 2013. 01 ) ; C12N 2310 /14 (2013 . 01 ) ; C12N ( 86 ) PCT No .: PCT/ GB2016 /051116 2310 / 11 (2013 .01 ) $ 371 ( c ) ( 1 ) , ( 2 ) Date : Oct . 20 , 2017 (57 ) ABSTRACT The invention relates to oligonucleotides , e . g . , saRNAS Related U . S . Application Data useful in upregulating the expression of a target gene and (60 ) Provisional application No . 62 / 150 ,892 , filed on Apr. therapeutic compositions comprising such oligonucleotides . 22 , 2015 , provisional application No . 62 / 150 ,893 , Methods of using the oligonucleotides and the therapeutic filed on Apr. 22 , 2015 , provisional application No . compositions are also provided . 62 / 150 ,897 , filed on Apr. 22 , 2015 , provisional ap Specification includes a Sequence Listing . SARNA sense strand (Fessenger 3 ' SARNA antisense strand (Guide ) Mathew, Si Target antisense RNA transcript, e . g . NAT Target Coding strand Gene Transcription start site ( T55 ) TY{ { ? ? Targeted Target transcript , e .
    [Show full text]
  • Dijagnoze.Pdf
    Str.: 1 18.09.2020 Dijagnoze Oznaka Latinski Naziv Dijagnoze Srpski Naziv Dijagnoze 0 Nepoznata Nepoznata A00 Cholera Kolera A000 Cholera classica Kolera, uzrocnik Vibrio cholerae 01,biotip cholerae A001 Cholera El Tor Kolera, uzrocnik Vibrio cholerae 01,biotip El Tor A009 Cholera, non specificata Kolera, neoznacena A01 Febris typhica et febris paratyphica Tifusna groznica i paratifusna groznica A010 Typhus abdominalis Trbusni tifus A011 Paratyphus A Paratifus A A012 Paratyphus B Paratifus B A013 Paratyphus C Paratifus C A014 Paratyphus, non specificatus Paratifus, neoznacen A02 Salmonelloses aliae Druge infekcije uzrokovane salmonelama A020 Enteritis salmonellosa Zapaljenje tankog creva uzrokovano salmonelama A021 Salmonellosis septica Sepsa uzrokovana salmonelama A022 Infectio per salmonellam localisata Lokalizovana infekcija salmonelama A028 Infectio per salmonellam alia, specificata Druga oznacena infekcija salmonelama A029 Infectio per salmonellam, non specificata Salmonelozna infekcija, neoznacena A03 Shigellosis Dizenterija, uzrocnik Shigellae A030 Dysenteria bacillaris per Shigellam dysenteriae Dizenterija, uzrocnik Shigella dysenteriae A031 Dysenteria bacillaris per Shigellam flexneri Dizenterija, uzrocnik Shigell flexneri A032 Dysenteria bacillaris per Shigellam boydi Dizenterija, uzrocnik Shigella boydi A033 Dysenteria bacillaris per Shigellam sonnei Dizenterija,uzrocnik Shigella sonnei Shigellosis D A038 Shigellosis alia Druga dizenterija A039 Shigellosis, non specifiata Dizenterija, neoznacena A04 Infectiones intestinales
    [Show full text]