Tachyarrhythmias

Total Page:16

File Type:pdf, Size:1020Kb

Tachyarrhythmias Patient information factsheet Tachyarrythmias (fast heart rhythms) This factsheet has been written to help you understand more about heart rhythm problems. If there is anything you do not understand, or you would like more information, please ask a member of your healthcare team, who will be happy to explain further. To help you understand this factsheet please refer to our “how the heart works” factsheet first. This is available on: www.uhs.nhs.uk or ask a member of your healthcare team. What is an arrhythmia? Arrhythmia is the term used to describe heart rhythm abnormalities. Sometimes if the heart’s conduction pathway is damaged, blocked, or an extra pathway exists the heart’s rhythm changes. The heart may beat: • too quickly (tachycardia) • too slowly (bradycardia) • irregularly. This may affect the heart’s ability to pump blood around the body. Arrhythmias can occur in the heart’s upper chambers (atria) or lower chambers (ventricles). Causes of an arrhythmia Any interruption in the heart’s electrical system can cause an arrhythmia. Common causes of arrhythmias include stress, caffeine, tobacco, alcohol, diet pills and cough and cold medicines. If your heart tissue is damaged as a result of acquired heart disease, such as myocardial infarction (heart attack) or congenital heart disease you may be at risk of developing arrhythmias. Occasionally it may be an inherited disorder or one that runs in your family. Sometimes it is not possible to identify the cause of the arrhythmia. Diagnosing your arrhythmia If your doctor suspects that you may have an arrhythmia, one or more of the following tests may be performed to determine the cause of your symptoms. Electrocardiogram (ECG) An electrocardiogram is a recording of the electrical activity of your heart. Electrode stickers are placed on your chest and connected by wires to a recording machine. Your heart’s electrical signals produce a pattern on graph paper in the ECG. By analysing the pattern of these waves, your doctor can often determine what type of arrhythmia you have. ECG testing may be done while you are resting, or while you are exercising on a treadmill. www.uhs.nhs.uk Patient information factsheet Holter monitor A Holter monitor shows changes in your heart rhythm over the course of a 24-hour period that may not be detected during a resting or exercise ECG. You will be asked to go about your daily activities as usual (except for showering or bathing) while you wear a small, portable recorder that connects to electrode stickers on your chest. You will then come back to the hospital the next day so that the information can be retrieved and analysed. Cardiac event monitor If your doctor feels you need to be monitored for several days or weeks, you may need to have a cardiac event monitor. This type of recording device is used if your arrhythmias are infrequent. This device is about the size of a large pager, and can be clipped to your belt or waistband or carried in your bag or pocket. When you feel symptoms, you simply hold the recorder against your chest and press a button. The device then records up to 70 seconds of ECG readings. Internal loop recorder (ILR) Your doctor may feel an ILR is appropriate to investigate your symptoms. This is a long-term continuous heart monitor placed under the skin on the chest wall. The ILR comes with an activator, when you have symptoms you place the activator over the ILR and it will store your heart rhythm during your symptoms. If you have potential life-threatening arrhythmias your device will transmit this data automatically to the hospital. The ILR is inserted under local anaesthetic and the battery can last for more than three years. Once the battery has expired the device is usually removed. Types of arrhythmia Arrhythmias that occur in the atria are either ‘atrial’ (in the heart’s upper chambers) or ‘supraventricular’ (above the ventricles) in origin, whereas ventricular arrhythmias start in the ventricles (lower chambers). While some arrhythmias are merely a nuisance, others can be life-threatening. In general, ventricular arrhythmias caused by heart disease are the most serious kind, and require prompt medical attention. Supraventricular tachycardia (SVT) This type of arrhythmia commonly occurs in young, healthy people. Doctors often refer to SVT as ‘re-entry tachycardia’. This is because the electrical impulse does not fade out as with the normal heartbeat, but continues to move in a rapid circle within the conduction system. This is due to an extra electrical pathway that can form a short circuit within the heart’s conduction system. SVT is usually a rapid, regular rhythm. The two most common types of SVT are: • AV-nodal re-entry tachycardia • AV re-entry tachycardia (AVRT), most commonly known as Wolff-Parkinson-White syndrome (WPW). AV nodal re-entry tachycardia (AVNRT) This type of arrhythmia occurs when a problem arises in the way the electrical impulses pass through the AV node. Normally, the AV node acts as a gateway, slowing and regulating the impulses as they travel between the atria and the ventricles. In AVNRT there are two pathways, known as dual conduction pathways that can pass impulses to and from the AV node. This type of arrhythmia usually starts following an early beat (ectopic). An electrical short circuit then occurs where the electrical impulse rotates around the circuit and with each cycle pass to the ventricles, resulting in a very fast heartbeat. www.uhs.nhs.uk Patient information factsheet AV re-entry tachycardia (AVRT) or Wolff-Parkinson-White syndrome (WPW) In AVRT an extra electrical pathway exists that bypasses the normal conduction system. The pathway directly connects the atria to the ventricles. This extra pathway is known as an accessory pathway. The electrical impulses travel along the accessory pathway, bypassing the AV node. The tissue in the pathway does not slow the impulse down, as in the AV node. Therefore the electrical impulses reach the ventricles before the normal electrical impulse (this is known as pre-excitation). An ECG recording of a patient with WPW syndrome will often show a ‘delta wave’, which shows the existence of an extra electrical pathway. Very fast heart rates may occur as the electrical impulse bounces between the atria and ventricles. Atrial fibrillation Atrial fibrillation (AF) is one of the most common types of arrhythmia.AF occurs in the atria. The electrical impulse normally originates at the SA node. However, in atrial fibrillation, many electrical impulses are fired rapidly and at random throughout the atria down to the ventricles. The resulting heartbeat is irregular and usually fast. When the atria are beating rapidly and irregularly (fibrillating) they are unable to completely empty all of the blood they receive into the ventricles. This can cause blood clots to form. Therefore, to prevent you being at an increased risk of stroke you will be treated with an anticoagulant (blood thinner). Atrial flutter Atrial flutter also occurs in the atria. The electrical impulses fire rapidly but the resulting rhythm is regular and organised. The rhythm is due to a re-entry circuit within the atria, where the electrical impulse travels in circles leaving and arriving back at the same point. Ventricular tachycardia (VT) VT occurs when the electrical impulses arise in the ventricles. The ventricles start beating at an abnormally fast, irregular rate. When the ventricles are beating rapidly the heart does not work as efficiently, causing symptoms of weakness, dizziness, chest pain, shortness of breath or even collapse. There are several different types of VT and the seriousness of the condition can vary. VT can be a potentially life-threatening heart rhythm as it can progress to ventricular fibrillation and cause the heart to stop beating (cardiac arrest). There are a number of reasons that people may develop VT. For example, in people who have had a previous myocardial infarction (heart attack) the area of the heart muscle damaged by the heart attack forms scar tissue. This can make the heart susceptible to abnormal heart rhythms. Other people who may experience VT are patients with cardiomyopathy, previous corrective congenital heart surgery or inherited arrhythmias. There is also a small group of people who have VT with a structurally normal heart. Ventricular fibrillation Ventricular fibrillation occurs in the ventricles. In ventricular fibrillation, the electrical impulses are fired from multiple sites in the ventricles in a very fast and irregular way, causing the heart to quiver rather than to beat and pump blood. Ventricular fibrillation is a life-threatening emergency requiring urgent medical treatment. Treatments The results of the tests you have had will determine the type and seriousness of your arrhythmia. Your doctor will then discuss your treatment options with you. Many patients with arrhythmias require no further treatment. www.uhs.nhs.uk Patient information factsheet Medicines There are a number of drugs that can be used to treat arrhythmias. Anti-arrhythmic drugs are medicines that change the electrical signals in your heart and help prevent irregular or rapid heart rhythms. Permanent pacemaker If you have atrial fibrillation which has proved difficult to treat, your doctor may recommend you have a pacemaker fitted in conjunction with a procedure called an atrioventricular (AV) node ablation. A pacemaker is a small device used to treat slow heart rhythms. It is implanted beneath the skin below the collarbone and connected to a pacing wire placed inside the heart. The pacemaker delivers a small electrical impulse to stimulate the heart to beat when it is going too slowly. An AV node ablation and permanent pacemaker insertion will regulate the heart rate and provide relief from your symptoms.
Recommended publications
  • WPW: WOLFF-PARKINSON-WHITE Syndrome
    WPW: WOLFF-PARKINSON-WHITE Syndrome What is Wolff-Parkinson-White Syndrome? Wolff-Parkinson-White Syndrome, or WPW, is named for three physicians who described a syndrome in 1930 in young people with episodes of heart racing and an abnormal pattern on their electrocardiogram (ECG or EKG). Over the next few decades, it was discovered that this ECG pattern and the heart racing was due to an extra electrical pathway in the heart. Thus, WPW is a syndrome associated with an abnormal heart rhythm, or “arrhythmia”. Most people with WPW do not have any other problems with their heart. Normally, the electrical impulses in the heart originate in the atria or top chambers of the heart and spread across the atria. The electrical impulses are then conducted to the ventricles (the pumping/bottom chambers of the heart) through a group of specialized cells called the atrioventricular node or AV node. This is usually the only electrical pathway between the atria and ventricles. In WPW, there is an additional pathway made up of a few extra cells left over from when the heart formed. The conduction of electricity through the heart causes the contractions which are the “heartbeat”. What is WPW Syndrome as opposed to a WPW ECG? A person has WPW Syndrome if they experience symptoms from abnormal conduction through the heart by the WPW pathway. Most commonly, the symptom is heart racing, or “palpitations”. The particular type of arrhythmia in WPW is called “supraventricular tachycardia” or SVT. “Tachycardia” means fast heart rate; “supraventricular” means the arrhythmia requires the cells above the ventricles to be part of the abnormal circuit.
    [Show full text]
  • Mitral Valve Prolapse, Arrhythmias, and Sudden Cardiac Death: the Role of Multimodality Imaging to Detect High-Risk Features
    diagnostics Review Mitral Valve Prolapse, Arrhythmias, and Sudden Cardiac Death: The Role of Multimodality Imaging to Detect High-Risk Features Anna Giulia Pavon 1,2,*, Pierre Monney 1,2,3 and Juerg Schwitter 1,2,3 1 Cardiac MR Center (CRMC), Lausanne University Hospital (CHUV), 1100 Lausanne, Switzerland; [email protected] (P.M.); [email protected] (J.S.) 2 Cardiovascular Department, Division of Cardiology, Lausanne University Hospital (CHUV), 1100 Lausanne, Switzerland 3 Faculty of Biology and Medicine, University of Lausanne (UniL), 1100 Lausanne, Switzerland * Correspondence: [email protected]; Tel.: +41-775-566-983 Abstract: Mitral valve prolapse (MVP) was first described in the 1960s, and it is usually a benign condition. However, a subtype of patients are known to have a higher incidence of ventricular arrhythmias and sudden cardiac death, the so called “arrhythmic MVP.” In recent years, several studies have been published to identify the most important clinical features to distinguish the benign form from the potentially lethal one in order to personalize patient’s treatment and follow-up. In this review, we specifically focused on red flags for increased arrhythmic risk to whom the cardiologist must be aware of while performing a cardiovascular imaging evaluation in patients with MVP. Keywords: mitral valve prolapse; arrhythmias; cardiovascular magnetic resonance Citation: Pavon, A.G.; Monney, P.; Schwitter, J. Mitral Valve Prolapse, Arrhythmias, and Sudden Cardiac Death: The Role of Multimodality 1. Mitral Valve and Arrhythmias: A Long Story Short Imaging to Detect High-Risk Features. In the recent years, the scientific community has begun to pay increasing attention Diagnostics 2021, 11, 683.
    [Show full text]
  • Constrictive Pericarditis Causing Ventricular Tachycardia.Pdf
    EP CASE REPORT ....................................................................................................................................................... A visually striking calcific band causing monomorphic ventricular tachycardia as a first presentation of constrictive pericarditis Kian Sabzevari 1*, Eva Sammut2, and Palash Barman1 1Bristol Heart Institute, UH Bristol NHS Trust UK, UK; and 2Bristol Heart Institute, UH Bristol NHS Trust UK & University of Bristol, UK * Corresponding author. Tel: 447794900287; fax: 441173425926. E-mail address: [email protected] Introduction Constrictive pericarditis (CP) is a rare condition caused by thickening and stiffening of the pericar- dium manifesting in dia- stolic dysfunction and enhanced interventricu- lar dependence. In the developed world, most cases are idiopathic or are associated with pre- vious cardiac surgery or irradiation. Tuberculosis remains a leading cause in developing areas.1 Most commonly, CP presents with symptoms of heart failure and chest discomfort. Atrial arrhythmias have been described as a rare pre- sentation, but arrhyth- mias of ventricular origin have not been reported. Figure 1 (A) The 12 lead electrocardiogram during sustained ventricular tachycardia is shown; (B and C) Case report Different projections of three-dimensional reconstructions of cardiac computed tomography demonstrating a A 49-year-old man with a striking band of calcification around the annulus; (D) Carto 3DVR mapping—the left hand panel (i) demonstrates a background of diabetes, sinus beat with late potentials at the point of ablation in the coronary sinus, the right hand panel (iii) shows the hypertension, and hyper- pacemap with a 89% match to the clinical tachycardia [matching the morphology seen on 12 lead ECG (A)], and cholesterolaemia and a the middle panel (ii) displays the three-dimensional voltage map.
    [Show full text]
  • J Wave Syndromes
    Review Article http://dx.doi.org/10.4070/kcj.2016.46.5.601 Print ISSN 1738-5520 • On-line ISSN 1738-5555 Korean Circulation Journal J Wave Syndromes: History and Current Controversies Tong Liu, MD1, Jifeng Zheng, MD2, and Gan-Xin Yan, MD3,4 1Tianjin Key Laboratory of Ionic-Molecular Function of Cardiovascular disease, Department of Cardiology, Tianjin Institute of Cardiology, The Second Hospital of Tianjin Medical University, Tianjin, 2Department of cardiology, The Second Hospital of Jiaxing, Jiaxing, China, 3Lankenau Institute for Medical Research and Lankenau Medical Center, Wynnewood, Pennsylvania, USA, 4The First Affiliated Hospital, Medical School of Xi'an Jiaotong University, Xi'an, China The concept of J wave syndromes was first proposed in 2004 by Yan et al for a spectrum of electrocardiographic (ECG) manifestations of prominent J waves that are associated with a potential to predispose affected individuals to ventricular fibrillation (VF). Although the concept of J wave syndromes is widely used and accepted, there has been tremendous debate over the definition of J wave, its ionic and cellular basis and arrhythmogenic mechanism. In this review article, we attempted to discuss the history from which the concept of J wave syndromes (JWS) is evolved and current controversies in JWS. (Korean Circ J 2016;46(5):601-609) KEY WORDS: Brugada syndrome; Sudden cardiac death; Ventricular fibrillation. Introduction History of J wave and J wave syndromes The concept of J wave syndromes was first proposed in 2004 The J wave is a positive deflection seen at the end of the QRS by Yan et al.1) for a spectrum of electrocardiographic (ECG) complex; it may stand as a distinct “delta” wave following the QRS, manifestations of prominent J waves that are associated with a or be partially buried inside the QRS as QRS notching or slurring.
    [Show full text]
  • The Syndrome of Alternating Bradycardia and Tachycardia by D
    Br Heart J: first published as 10.1136/hrt.16.2.208 on 1 April 1954. Downloaded from THE SYNDROME OF ALTERNATING BRADYCARDIA AND TACHYCARDIA BY D. S. SHORT From the National Heart Hospita. Received September 15, 1953 Among the large number of patients suffering from syncopal attacks who attended the National Heart Hospital during a four-year period, there were four in whom examination revealed sinus bradycardia alternating with prolonged phases of auricular tachycardia. These patients presented a difficult problem in treatment. Each required at least one admission to hospital and in one case the symptoms were so intractable as to necessitate six admissions in five years. Two patients had mitral valve disease, one of them with left bundle branch block. One had aortic valve sclerosis while the fourth had no evidence of heart disease. THE HEART RATE The sinus rate usually lay between 30 and 50 a minute, a rate as slow as 22 a minute being observed in one patient (Table I). Sinus arrhythmia was noted in all four patients, wandering of TABLE I http://heart.bmj.com/ RATE IN SINus RHYTHM AND IN AURICULAR TACHYCARDIA Rate in Case Age Sex Associated Rate in auricular tachycardia heart disease sinus rhythm Auricular Venliicular 1 65 M Aortic valve sclerosis 28-48 220-250 60-120 2 47 F Mitral valve disease 35-75 180-130 90-180 on September 26, 2021 by guest. Protected copyright. 3 38 F Mitral valve disease 22-43 260 50-65 4 41 F None 35-45 270 110 the pacemaker in three, and periods of sinus standstill in two (Fig.
    [Show full text]
  • Unstable Angina with Tachycardia: Clinical and Therapeutic Implications
    Unstable angina with tachycardia: Clinical and therapeutic implications We prospectively evaluated 19 patients with prolonged chest pain not evolving to myocardiai infarction and accompanied with reversible ST-T changes and tachycardia (heart rate >lOO beats/min) in order to correlate heart rate reduction with ischemic electrocardiographic (ECG) changes. Fourteen patients (74%) received previous long-term combined treatment with nifedipine and nitrates. Continuous ECG monitoring was carried out until heart rate reduction and at least one of the following occurred: (1) relief of pain or (2) resolution of ischemic ECG changes. The study protocol consisted of carotid massage in three patients (IS%), intravenous propranolol in seven patients (37%), slow intravenous amiodarone infusion in two patients (lo%), and intravenous verapamil in four patients (21%) with atrial fibrillation. In three patients (16%) we observed a spontaneous heart rate reduction on admission. Patients responded with heart rate reduction from a mean of 126 + 10.4 beats/min to 64 k 7.5 beats/min (p < 0.005) and an ST segment shift of 4.3 k 2.13 mm to 0.89 k 0.74 mm (p < 0.005) within a mean interval of 13.2 + 12.7 minutes. Fifteen (79%) had complete response and the other four (21%) had partial relief of pain. A significant direct correlation was observed for heart rate reduction and ST segment deviation (depression or elevation) (f = 0.7527 and 0.8739, respectively). These patients represent a unique subgroup of unstable angina, in which the mechanism responsible for ischemia is excessive increase in heart rate. Conventional vasodilator therapy may be deleterious, and heart rate reduction Is mandatory.
    [Show full text]
  • Basic Arrhythmia Review Guide-Advanced
    BASIC ARRHYTHMIA REVIEW GUIDE ADVANCED The following study guide provides a review of information covered in the basic arrhythmia competency. Preparation with this guide will help to achieve success on the exam. Sample questions and websites are provided at the end of this guide. DESCRIPTION OF THE HEART The adult heart is a muscular organ weighing less than a pound and about the size of a clenched fist. It lies between the right and Left left lung in an area called the mediastinal cavity behind the sternum of the breast bone. Approximately two-thirds of the heart Atrium lies to the left of the sternum and one-third to the right of the sternum. Right HEART MUSCLES Atrium The heart is composed of three layers each with its own special function. The outermost layer is called the pericardium, essentially a sac around the heart. The middle and thickest layer of the heart is called the Left myocardium. This layer contains all the atrial and ventricular Ventricle muscle fibers needed for contraction as well as the blood supply Right and electrical conduction system. Ventricle The innermost layer of the heart is the endocardium and is composed of endothelium and connective tissue. Any disruption or injury to this endothelium can lead to infection, which in turn can cause valve damage, sepsis, or death. CHAMBERS A normal human heart contains four separate chambers: right atrium, left atrium, right ventricle, and left ventricle. The right and left sides of the heart are divided by a septum. The right atrium (RA) receives oxygen-poor (venous) blood from the body’s organs via the superior and inferior vena cava (SVC and IVC).
    [Show full text]
  • Tachycardia (Fast Heart Rate)
    Tachycardia (fast heart rate) Working together to improve the diagnosis, treatment and quality of life for all those aff ected by arrhythmias www.heartrhythmalliance.org Registered Charity No. 1107496 Glossary Atrium Top chambers of the heart that receive Contents blood from the body and from the lungs. The right atrium is where the heart’s natural pacemaker (sino The normal electrical atrial node) can be found system of the heart Arrhythmia An abnormal heart rhythm What are arrhythmias? Bradycardia A slow heart rate, normally less than 60 beats per minute How do I know what arrhythmia I have? Cardiac Arrest the abrupt loss of heart function, breathing and consciousness Types of arrhythmia Cardioversion a procedure used to return an abnormal What treatments are heartbeat to a normal rhythm available to me? Defi brillation a treatment for life-threatening cardiac arrhythmias. A defibrillator delivers a dose of electric current to the heart Important information This booklet is intended for use by people who wish to understand more about Tachycardia. The information within this booklet comes from research and previous patients’ experiences. The booklet off ers an explanation of Tachycardia and how it is treated. This booklet should be used in addition to the information given to you by doctors, nurses and physiologists. If you have any questions about any of the information given in this booklet, please ask your nurse, doctor or cardiac physiologist. 2 Heart attack A medical emergency in which the blood supply to the heart is blocked, causing serious damage or even death of heart muscle Tachycardia Fast heart rate, more than 100 beats per minute Ventricles The two lower chambers of the heart.
    [Show full text]
  • The Example of Short QT Syndrome Jules C
    Hancox et al. Journal of Congenital Cardiology (2019) 3:3 Journal of https://doi.org/10.1186/s40949-019-0024-7 Congenital Cardiology REVIEW Open Access Learning from studying very rare cardiac conditions: the example of short QT syndrome Jules C. Hancox1,4* , Dominic G. Whittaker2,3, Henggui Zhang4 and Alan G. Stuart5,6 Abstract Background: Some congenital heart conditions are very rare. In a climate of limited resources, a viewpoint could be advanced that identifying diagnostic criteria for such conditions and, through empiricism, effective treatments should suffice and that extensive mechanistic research is unnecessary. Taking the rare but dangerous short QT syndrome (SQTS) as an example, this article makes the case for the imperative to study such rare conditions, highlighting that this yields substantial and sometimes unanticipated benefits. Genetic forms of SQTS are rare, but the condition may be under-diagnosed and carries a risk of sudden death. Genotyping of SQTS patients has led to identification of clear ion channel/transporter culprits in < 30% of cases, highlighting a role for as yet unidentified modulators of repolarization. For example, recent exome sequencing in SQTS has identified SLC4A3 as a novel modifier of ventricular repolarization. The need to distinguish “healthy” from “unhealthy” short QT intervals has led to a search for additional markers of arrhythmia risk. Some overlap may exist between SQTS, Brugada Syndrome, early repolarization and sinus bradycardia. Genotype-phenotype studies have led to identification of arrhythmia substrates and both realistic and theoretical pharmacological approaches for particular forms of SQTS. In turn this has increased understanding of underlying cardiac ion channels.
    [Show full text]
  • Common Types of Supraventricular Tachycardia: Diagnosis and Management RANDALL A
    Common Types of Supraventricular Tachycardia: Diagnosis and Management RANDALL A. COLUCCI, DO, MPH, Ohio University College of Osteopathic Medicine, Athens, Ohio MITCHELL J. SILVER, DO, McConnell Heart Hospital, Columbus, Ohio JAY SHUBROOK, DO, Ohio University College of Osteopathic Medicine, Athens, Ohio The most common types of supraventricular tachycardia are caused by a reentry phenomenon producing acceler- ated heart rates. Symptoms may include palpitations (including possible pulsations in the neck), chest pain, fatigue, lightheadedness or dizziness, and dyspnea. It is unusual for supraventricular tachycardia to be caused by structurally abnormal hearts. Diagnosis is often delayed because of the misdiagnosis of anxiety or panic disorder. Patient history is important in uncovering the diagnosis, whereas the physical examination may or may not be helpful. A Holter moni- tor or an event recorder is usually needed to capture the arrhythmia and confirm a diagnosis. Treatment consists of short-term or as-needed pharmacotherapy using calcium channel or beta blockers when vagal maneuvers fail to halt or slow the rhythm. In those who require long-term pharmacotherapy, atrioventricular nodal blocking agents or class Ic or III antiarrhythmics can be used; however, these agents should generally be managed by a cardiologist. Catheter ablation is an option in patients with persistent or recurrent supraventricular tachycardia who are unable to tolerate long-term pharmacologic treatment. If Wolff-Parkinson-White syndrome is present, expedient referral
    [Show full text]
  • Acute Non-Specific Pericarditis R
    Postgrad Med J: first published as 10.1136/pgmj.43.502.534 on 1 August 1967. Downloaded from Postgrad. med. J. (August 1967) 43, 534-538. CURRENT SURVEY Acute non-specific pericarditis R. G. GOLD * M.B., B.S., M.RA.C.P., M.R.C.P. Senior Registrar, Cardiac Department, Brompton Hospital, London, S.W.3 Incidence neck, to either flank and frequently through to the Acute non-specific pericarditis (acute benign back. Occasionally pain is experienced on swallow- pericarditis; acute idiopathic pericarditis) has been ing (McGuire et al., 1954) and this was the pre- recognized for over 100 years (Christian, 1951). In senting symptom in one of our own patients. Mild 1942 Barnes & Burchell described fourteen cases attacks of premonitory chest pain may occur up to of the condition and since then several series of 4 weeks before the main onset of symptoms cases have been published (Krook, 1954; Scherl, (Martin, 1966). Malaise is very common, and is 1956; Swan, 1960; Martin, 1966; Logue & often severe and accompanied by listlessness and Wendkos, 1948). depression. The latter symptom is especially com- Until recently Swan's (1960) series of fourteen mon in patients suffering multiple relapses or patients was the largest collection of cases in this prolonged attacks, but is only partly related to the country. In 1966 Martin was able to collect most length of the illness and fluctuates markedly from of his nineteen cases within 1 year in a 550-bed day to day with the patient's general condition. hospital. The disease is thus by no means rare and Tachycardia occurs in almost every patient at warrants greater attention than has previously some stage of the illness.
    [Show full text]
  • Inappropriate Sinus Tachycardia Following Viral Illness
    Case Report Inappropriate Sinus Tachycardia Following Viral Illness Khalid Sawalha 1,* , Fuad Habash 2 , Srikanth Vallurupalli 2 and Hakan Paydak 3 1 Internal Medicine Division, White River Health System, Batesville, AR 72501, USA 2 Cardiology Division, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA; [email protected] (F.H.); [email protected] (S.V.) 3 Electrophysiology Division, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA; [email protected] * Correspondence: [email protected]; Tel.: +1-984-364-1158 Abstract: A 67-year-old female patient with a past medical history of menopause, migraines, and gastro-esophageal disease presented with palpitation, fatigue, and shortness of breath. One month prior to her presentation, she reported having flu-like symptoms. Her EKG showed sinus tachycar- dia with no other abnormality. Laboratory findings, along with imaging, showed normal results. The event monitor failed to detect any arrythmias. We report a case of inappropriate sinus tachycardia secondary to viral infection as a diagnosis of exclusion. Keywords: inappropriate sinus tachycardia; viral infection; palpitations 1. Introduction Inappropriate sinus tachycardia, also called chronic non-paroxysmal sinus tachycardia, is an unusual condition that occurs in individuals without apparent heart disease or other cause of sinus tachycardia, such as hyperthyroidism or fever, and is generally considered a diagnosis of exclusion [1–4]. Inappropriate sinus tachycardia is defined as a resting heart Citation: Sawalha, K.; Habash, F.; rate >100 beats per minute associated with highly symptomatic palpitations [5,6]. Vallurupalli, S.; Paydak, H. Commonly used criteria to define inappropriate sinus tachycardia include [7] P-wave Inappropriate Sinus Tachycardia axis and morphology similar to sinus rhythm, and a resting heart rate of 100 beats per Following Viral Illness.
    [Show full text]