PÁLYÁZAT AZ ORSZÁGOS DOKTORI TANÁCS TÁRSELNÖKI MEGBÍZATÁSÁRA 2020-2022

DR. BENYÓ ZOLTÁN SEMMELWEIS EGYETEM

TARTALOM  SZAKMAI ÖNÉLETRAJZ  EDDIGI TEVÉKENYSÉG ISMERTETÉSE  VEZETŐI PROGRAM MELLÉKLETEK  TUDOMÁNYMETRIAI TÁBLÁZAT  PUBLIKÁCIÓS ÉS CITÁCIÓS JEGYZÉK

BUDAPEST 2019. NOVEMBER 11. Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 1. oldal

Szakmai önéletrajz

Név: Dr. Benyó Zoltán Születési hely, idő: Budapest, 1967. június 15. Családi állapot: nős (Dr. Káldi Krisztina), két gyermek (Barnabás, 1996; Franciska, 1999) Munkahely: Semmelweis Egyetem, Transzlációs Medicina Intézet 1094 Budapest, Tűzoltó u. 37-47., Tel.: 1-2100306, 20-6632315, Fax: 1-3343162 e-mail: [email protected]

Beosztás: 1992-1995 TMB ösztöndíjas 1995-2000 egyetemi tanársegéd 2000-2006 egyetemi adjunktus 2006-2010 egyetemi docens 2008-2010 igazgatóhelyettes 2010- egyetemi tanár, igazgató

Egyéb megbízatások: 2006- Magyar Élettani Társaság vezetőségi tagja 2007-2010 Magyar Élettani Társaság ifjúsági titkára és elnökségi tagja 2007-2010 Magyarországi Humboldt Egyesület titkára 2007- Semmelweis Egyetem Rektori Pályamunka Értékelő Bizottság tagja 2008-2010 Semmelweis Egyetem Habilitációs Bizottság tagja 2008- Semmelweis Egyetem Állatkísérleti Bizottság tagja 2009 NKTH-OTKA Élettudományi Vegyes Bizottság tagja 2009-2012 OTKA Kísérletes Orvostudományok Zsűri tagja 2010-2013 Semmelweis Egyetem képviselője a MEDINE2 (Medical Education in Europe 2) Erasmus Academic Network-ben 2011-2016 Semmelweis Egyetem ÁOK Szociális és Esélyegyenlőségi Bizottság elnöke 2011-2015 MTA Elméleti Orvostudományi Bizottság titkára 2013-2015 Semmelweis Egyetem Fogyatékos Hallgatók Ügyeivel Foglalkozó Bizottság elnöke

2013-2016 Semmelweis Egyetem ÁOK, Oktatási és Kreditbizottság tagja 2013-2019 Semmelweis Egyetem Doktori Iskola, Oktatási és Kreditbizottság elnöke Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 2. oldal

2014- Magyar Élettani Alapítvány kuratóriumának elnöke 2014- Semmelweis Egyetem Elméleti és Transzlációs Doktori Iskola programvezetője (1/2. sz. program, „A vérkeringési rendszer normális és kóros működésének mechanizmusai”) 2018- Semmelweis Egyetem Szociális és Esélyegyenlőségi Bizottság elnöke 2019- MTA közgyűlési képviselője 2019- Semmelweis Egyetem Doktori Tanács elnöke 2019- Semmelweis Egyetem Szenátusának választott tagja

Tanulmányok: 1985. középiskolai érettségi bizonyítvány (kitünő), Fazekas Mihály Fővárosi Gyakorló Gimnázium (5-C/1985, 1985. június 14.) 1992. általános orvosi diploma (summa cum laude) Semmelweis Orvostudományi Egyetem, (79/1992, 1992. szeptember) Szakdolgozat címe: "Endogén opiátok szerepe az artériás vérnyomás és az agyi véráramlás szabályozásában"

Tudományos minősítés: 1998. orvostudomány kandidátusa (PhD) Kandidátusi értekezés címe: "Role of opioid peptides, nitric oxide and their interactions in the cardiovascular system" 2006. MTA doktora MTA doktori értekezés címe: " Lokális szabályozási mechanizmusok szerepe és kölcsönhatásai a vérkeringési rendszer élettani és kórélettani folyamataiban " 2008. Dr. med. habil. (Semmelweis Egyetem)

Oktatási tevékenység: Élettan orvostanhallgatók és gyógyszerészhallgatók részére magyar és angol nyelven (gyakorlatok és konzultációk tartása) (1991-) Élettan orvostanhallgatók és gyógyszerészhallgatók részére magyar és angol nyelven (előadások tartása) (2000-) Vizsgáztatás: kollokvium (1998-) és szigorlat (2006-) Tudományos diákkörös hallgatók irányítása (1992-), intézeti TDK-felelős (1999-2002) Részvétel PhD hallgatók szakmai irányításában (1998-), PhD témavezető (2001-), programvezető (2014-)

Nyelvismeret: Angol: Középfokú állami nyelvvizsga (1984) TOEFL 550 pont (1991) Orosz: Középfokú állami nyelvvizsga (1986) Német: Társalgási szint Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 3. oldal

Hosszabb külföldi tanulmányutak: 1990-91: Cerebrovascular Research Center, University of Pennsylvania, Philadelphia, USA (Prof. M. Reivich és Prof. Kovách A.) 1992: Department of Animal Physiology, University of Groningen, Hollandia (Prof. B. Bohus és Prof. P.G.M. Luiten) 1995-98: Department of Physiology, Ludwig Maximilians University, München, Németország (Prof. M. Wahl) 2002-2006: Institute of Pharmacology, Ruprecht-Karls-University, Heidelberg, Németország (Prof. S. Offermanns)

Elismerések és ösztöndíjak: 1989. Rektori pályamunka I. díj 1989, 1990. I. és II. díj a Semmelweis OTE TDK Konferenciáján, Dicsérő oklevél az Országos TDK Konferencián 1992. I. díj az Egészséges Erekért Alapítvány Pályázatán 1995-1997. Alexander von Humboldt Ösztöndíj (2 év) 1997-1998. Magyar Állami Eötvös Ösztöndíj (7 hónap) 1998-2001. Bolyai Kutatási Ösztöndíj (3 év) 1999. II. díj a Sigma fiatal kutatói pályázatán 1999. Richter Gedeon Kutatási Jutalomdíj 2001-2002. Békésy György Posztdoktori Ösztöndíj 2003-2004. Marie Curie Individual Fellowship (2 év) 2006. Kiváló Tudományos Diákköri Nevelő egyetemi kitüntetés 2012. XXX. Jubileumi OTDK Emlékérem

Főbb kutatási területek: A vérkeringési rendszer élettana és kórélettana. Az agyi vérkeringés szabályozása. Az endothelium és a simaizom jelátviteli folyamatai. Lipid mediátorok élettani és kórélettani funkciói a vérkeringési rendszerben.

Témavezetőként elnyert kutatási támogatások:

1994. OTKA W 015186 Világbanki hitelből támogatott fiatal kutatói pályázat (15.400.- USD)

1994-97. OTKA F 013241 "Nitrogén monoxid szabadgyök szintetázok szerepe a cerebro- vaszkuláris simaizomtónus szabályozásában" (1.800 eFt)

1999. Műszeradomány az Alexander von Humboldt Alapítványtól (58.000.- DEM)

1999. Egyetemi kutatási támogatás (500 eFt)

1999. OTKA P31612 Publikációs támogatás (55 eFt) Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 4. oldal

1999-2000. ETT 141/1999 "Lokális szabályozó tényezők szerepe és kölcsönhatásai az agyi érrendszerben" (1.280 eFt)

1999-2001. OTKA F 029801 "Az agyi értónus és véráramlás parakrin szabályozása" (4.500 eFt)

2000. Egyetemi kutatási támogatás (1.400 eFt)

2000-2001. MÖB-DAAD 60/2000 "Az agyi értónus és véráramlás parakrin szabályozása" Magyar-német kutatócsereprogram (600 eFt + 20.800.- DEM)

2000-2002. FKFP 0518/2000 "Endoteliális és neurális tényezők szerepe és kölcsönhatásai az agyi vérkeringés szabályozásában" (3.000 eFt)

2001-2003. ETT 141/1999 "Agyi vazoregulációs mechanizmusok kölcsönhatásainak vizsgálata" (3.000 eFt)

2002-2005. OTKA T 037386 "A hem-oxigenáz és a nitrogén monoxid szintáz reakcióutak szerepe és kölcsönhatásai az agyi vérkeringés szabályozásában" (9.600 eFt)

2006-2010. OTKA K 62375 "A PUMA-G receptor élettani és kórélettani szerepe az agyi vérkeringésben. " ( 17.630 eFt)

2006-2010. European Foundation for the Study of Diabetes (EFSD) and SERVIER "G protein mediated signalling pathways and vascular NADPH oxidases in the development of diabetes-induced cerebrovascular dysfunction. " (100.000,- €)

2007. Egyetemi kutatási támogatás (1.800 eFt)

2008-2009. NKTH-OTKA H07-BEL 74286 "G fehérjék szerepe a húgyhólyag simaizom- működésének szabályozásában." (12.401 eFt)

2009-2011. ETT 429/2009 " A kóros agyi vazomóció és vazospazmus kialakulásának molekuláris alapjai." (4.500 eFt)

2009-2012. NKTH 2009ALAP1-01298/2009 "G fehérjék szerepe az agyi érműködés szabályozásában (EMBO SDIG)" (45.000 eFt)

2012-2015. OTKA K 101775 "A tromboxán receptorok jelátviteli folyamatai érsimaizomban" (28.862 eFt)

2015-2018. OTKA K 112964 "Szfingolipid mediátorok érhatásai" (29.808 eFt)

2016-2019. VEKOP-2.3.3-15-2016-00005 „Nanorészecskék endothelhatásainak és in vivo disztribúciójának vizsgálata multimodális képalkotó rendszerekkel” (295.000 eFt)

2017-2019. NVKP_16-1-2016-0042 Elektromágneses daganat-terápiás készülék hatékonyságának növelése, piaci bevezetése és az onkológiai terápiás gyakorlatba történő integrálásának előkészítése” (projekt összköltsége: 1.655.528 eFt, Semmelweis Egyetemre jutó támogatási összeg: 898.556 eFt)

2017-2021. OTKA K 125174 "A lizofoszfatidsav receptorok élettani és kórélettani funkciói a vérkeringési rendszerben" (48.000 eFt) Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 5. oldal

Szerkesztőbizottsági tagság: Acta Physiologica Hungarica Orvosi Hetilap

Bírálói tevékenység tudományos folyóiratoknál: Acta Physiologica Hungarica Brain Research Bulletin British Journal of Pharmacology Current Medicinal Chemistry Neuroscience Letters Journal of Cerebral Blood Flow and Metabolism Journal of Neuroscience Research Pathophysiology

Bírálói tevékenység tudományos szervezeteknél: Diabetes UK ETT Magyar Ösztöndíj Bizottság National Heart Foundation of New Zealand NKTH OTKA

Tudományos társasági tagság:

1989- Magyar Élettani Társaság 1993- Society for Neuroscience (USA) 1993- Magyar Stroke Társaság 1994- Magyar Biokémiai Egyesület 1995- European Society for Clinical Investigation 1996- Magyarországi Humboldt Egyesület 1997- International Society for Cerebral Blood Flow and Metabolism 2007- European Association for the Study of Diabetes

Válogatott közlemények: 1. Dér B, Molnár PJ, Ruisanchez É, Őrsy P, Kerék M, Faragó B, Nyirády P, Offermanns S, Benyó Z. NK2 receptor-mediated detrusor muscle contraction involves Gq/11-dependent activation of voltage-dependent Ca2+ channels and the RhoA-Rho kinase pathway. American Journal of Physiology - Renal Physiology, 317: F1154-F1163, 2019 (IF: 3,323) Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 6. oldal

2. Dancs PT, Ruisanchez E, Balogh A, Panta CR, Miklós Z, Nüsing RM, Aoki J, Chun J, Offermanns S, Tigyi G, Benyó Z. LPA1 receptor-mediated thromboxane A2 release is responsible for lysophosphatidic acid-induced vascular smooth muscle contraction. FASEB Journal, 31: 1547-1555, 2017 (IF: 5,595) 3. Polycarpou A, Hricisák L, Iring A, Safar D, Ruisanchez E, Horváth B, Sándor P, Benyó Z. Adaptation of the Cerebrocortical Circulation to Carotid Artery Occlusion Involves Blood Flow Redistribution between Cortical Regions and is Independent of eNOS. American Journal of Physiology - Heart and Circulatory Physiology, 311: H972-H980, 2016 (IF: 3,348) 4. Benyó Z, Ruisanchez E, Leszl-Ishiguro M, Sándor P, Pacher P. Endocannabinoids in Cerebrovascular Regulation, American Journal of Physiology - Heart and Circulatory Physiology, 310: H785-H801, 2016 (IF: 3,348) 5. Ruisanchez É, Dancs P, Kerék M, Németh T, Faragó B, Balogh A, Patil R, Jennings BL, Liliom K, Malik KU, Smrcka AV, Tigyi G, Benyó Z. Lysophosphatidic acid induces vasodilation mediated by LPA1 receptors, phospholipase C, and endothelial nitric oxide synthase. FASEB Journal, 28: 880-890, 2014 (IF: 5,043) 6. Iring A, Ruisanchez É, Leszl-Ishiguro M, Horváth B, Benkő R, Lacza Z, Járai Z, Sándor P, Di Marzo V, Pacher P, Benyó Z. Role of endocannabinoids and -1 receptors in cerebrocortical blood flow regulation. PLoS ONE, 8: e53390, 2013 (IF: 3,534) 7. Wirth A, Benyó Z, Lukasova M, Leutgeb B, Wettschureck N, Gorbey S, Őrsy P, Horváth B, Maser-Gluth C, Greiner E, Lemmer B, Schütz G, Gutkind S, Offermanns S. G12/G13- LARG-mediated signalling in vascular smooth muscle is required for salt-induced hypertension. Nature Medicine, 14: 64-68, 2008 (IF: 27,553) 8. Benyó Z, Gille A, Bennett C, Clausen B, Offermanns S. Nicotinic acid-induced flushing is mediated by activation of epidermal Langerhans cells. Molecular Pharmacology, 70: 1844-1849, 2006 (IF: 4,469) 9. Benyó Z, Gille A, Kero J, Csiky M, Suchánková MC, Nüsing RM, Moers A, Pfeffer K, Offermanns S. GPR109A (PUMA-G/HM74A) mediates nicotinic acid-induced flushing. Journal of Clinical Investigation, 115: 3634-3640, 2005 (IF: 15,053) 10. Horváth B, Hrabák A, Káldi K, Sándor P, Benyó Z Contribution of the heme oxygenase pathway to the maintenance of the hypothalamic blood flow during diminished NO synthesis. Journal of Cerebral Blood Flow and Metabolism, 23: 653-657, 2003 (IF: 5,370) 11. Lacza Z, Hermán P, Görlach C, Hortobágyi T, Sándor P, Wahl M, Benyó Z. NO synthase blockade induces chaotic cerebral vasomotion via activation of thromboxane receptors. Stroke, 32: 2609-2614, 2001 (IF: 5,330)

Budapest, 2019. november 11. Dr. Benyó Zoltán

Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 7. oldal

Eddigi tevékenység ismertetése

Doktori képzéssel kapcsolatos tevékenység

- 1998 óta rendszeresen doktori (PhD) értekezések opponensi bírálata, valamint elnöke ill. tagja PhD védések bíráló bizottságának a Semmelweis Egyetemen - 2001 óta PhD témavezető, hallgatói közül eddig négyen szereztek PhD fokozatot (közülük hárman „summa cum laude”, egy pedig „cum laude” minősítéssel), emellett egy „summa cum laude” minősítéssel fokozatot szerzett hallgató társ-témavezetője - 2001 óta rendszeresen a Szegedi Tudományegyetemen folyó PhD és habilitációs eljárások bíráló bizottsági tagja - 2008 óta rendszeresen elnöke ill. tagja PhD szigorlati bizottságoknak a Semmelweis Egyetemen - 2009 óta a Debreceni Egyetemen folyó PhD eljárások és egyetemi tanári pályázatok bíráló bizottsági tagja - 2013 és 2019 között a Semmelweis Egyetem Doktori Tanács, Oktatási és Kreditbizottság elnöke - 2014 óta PhD programvezető (1/2. sz. program, „A vérkeringési rendszer normális és kóros működésének mechanizmusai”) - 2015 óta PhD kurzusvezető („Lipid mediators in health and diseases” és „Klinikai kardiovaszkuláris fiziológia”) - 2019 óta a Semmelweis Egyetem Doktori Tanács elnöke

2016-ban részt vettem a Semmelweis Egyetem (konzorciumvezető), a Szegedi Tudományegyetem, a Pécsi Tudományegyetem, valamint a Debreceni Egyetem által benyújtott „Az orvos-, egészségtudományi- és gyógyszerészképzés tudományos műhelyeinek fejlesztése” című, EFOP-3.6.3-VEKOP-16-2017-00009 azonosítószámú pályázat elkészítésében, melynek kidolgozása során és megvalósításának első évében szakmai vezetőként, azóta pedig társ szakmai vezetőként tevékenykedtem. A projektnek a Semmelweis Egyetemen a doktori képzés fejlesztésére irányuló alprogramjait, és a megvalósításuk során eddig elért eredményeket az alábbiakban foglalom össze.

„Summer School PhD hallgatók számára” című alprogram

Minden évben egy blokkosított PhD kurzust tartunk, részben külföldi előadók részvételével, amelyek térítésmentesen látogathatók a konzorciumban részt vevő társegyetemek hallgatóin túl, más hazai és határon túli magyar tannyelvű

Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 8. oldal

egyetemek, kutatóintézetek PhD hallgatói számára is. Külföldi egyetemek, kutatóintézetek PhD hallgatóit is szívesen várjuk ezekre a rendezvényekre, önköltséges részvételi díj fizetése mellett. A nemzetközileg elismert előadók révén új módszereket, probléma-felvetéseket és -megoldásokat ismerhetnek meg a hallgatók, amelyek hozzájárulnak a doktoranduszok kutatói látásmódjának bővítéséhez is. Az angol nyelvű kurzusok elősegítik a magyar hallgatók szaknyelvi ismereteinek, illetve az idegen nyelvű kommunikációs képességének fejlődését, valamint biztosítják a Semmelweis Egyetem Doktori Iskolájának külföldi PhD hallgatói számára, hogy minél több doktori kurzuson tudjanak részt venni. Célunk az, hogy ezek a rendezvények hozzájáruljanak a doktori képzés és doktori kutatási folyamat hatékonyságának és eredményességének növelésén túl, a tudományos kapcsolati háló fejlődéséhez is.

Az EFOP projekt a Summer School megrendezését alkalmanként 1,5 MFt-al támogatja. Az első Summer School 2018. augusztus 28-29-30.-án került megrendezésre a Semmelweis Egyetem Biofizikai Intézetben az EMC2018 (European Muscle Conference) konferencia bevezető eseményeként, amelyre összesen 35 PhD hallgató regisztrált hazai és külföldi egyetemekről, kutatóintézetekből. Az idei Summer School a FEBS rendezvénysorozatának részeként, 2019. augusztus 25-30. között került megrendezésre Budapesten, „Biochemistry of Membrane proteins – Structure, Trafficking, Regulation” címmel, amelyen 17 PhD hallgató részvételét biztosítottuk.

„Kiválósági PhD-ösztöndíjak létesítése kutatási támogatással” című alprogram

Az EFOP projekt fő célkitűzése és egyben alapvető vállalása a fokozatot szerző PhD hallgatók számának növelése és kutatási kompetenciáinak erősítése. A fokozatszerzési hatékonyság növelésére, valamint a képzésbe bekapcsolódó hallgatók számának a növelésére kidolgozott legfontosabb fokozatszerzést ösztönző rendszerünk a Kiválósági PhD ösztöndíjak létesítése, amely rendszer több részből áll.

A fokozatszerzés hatékonyságának növelésére az egyik lehetőség a doktori fokozatszerzés nélkül lezáruló ösztöndíjas képzések számának csökkentése, amely leginkább a régi típusú, három éves képzési rendszer esetében veszélyezteti/veszélyeztette a doktori képzésbe kerülőket. Ezért a fokozatszerzést ösztönző rendszerünk egyik célcsoportját a régi rendszerű képzésbe bekerült, még aktív hallgatók alkotják. Egyrészt, a doktori értekezésüket a három éves képzési idő lejárta előtt benyújtó régi rendszerű ösztöndíjas hallgatókat 400 eFt „Egyszeri Fokozatszerzési Ösztöndíj”-ban részesítjük, ami az új képzési rendszer szabályai szerint megilleti a sikeresen fokozatot szerző hallgatókat. Továbbá, a régi rendszerű PhD hallgatók közül azoknak, akiknek nem sikerült három év elteltével teljesíteni a fokozatszerzéshez szükséges publikációs elvárásokat, de jelentős

Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 9. oldal

kutatási eredményeket tudtak felmutatni, további 1 évre 180 eFt/hó „Predoktori Ösztöndíj”-at biztosítottunk a PhD fokozat megszerzéséhez. Ha ez sikerül 1 éven belül, akkor 400 eFt Egyszeri Fokozatszerzési Ösztöndíj-at is kap a hallgató. Ezzel a kettő ösztöndíjjal a régi rendszerű PhD hallgatók kapnak azonos lehetőségeket az új rendszerű PhD hallgatókkal. A Predoktori Ösztöndíjhoz szemeszterenként 390 eFt ”bench fee” is társul a kutatócsoportok dologi költségeinek kiegészítésére. Eddig 2 hallgató részesült Egyszeri Fokozatszerzési Ösztöndíjban. Összesen 31 hallgató nyert el Predoktori Ösztöndíj támogatást, akik közül 1 fő 2018-ben és 2 fő 2019-ben szerzett már fokozatot.

A fokozatszerzés nélkül lezárult doktori képzések számának csökkentésre dolgoztuk ki a „PhD Fokozatszerzési Ösztöndíj” pályázatot is, amelynek célcsoportját mindazok a régi rendszerű, már nem aktív ösztöndíjas vagy önköltséges PhD hallgatók alkotják, akik 2016. szeptember 1-ét megelőzően nyertek felvételt a doktori képzésre, és a 3 éves képzési periódust követően az Abszolutóriumot megszerezték, de értekezésüket még nem nyújtották be a Doktori Iskola Hivatalába. A támogatás megítélésénél a döntő szempont a fokozatszerzéshez szükséges publikációs követelmények teljesíthetősége. A támogatást nyert pályázók a PhD értekezés Doktori Iskola Hivatalába történő benyújtásig, de maximum 1 évig, havi 70.000 Ft „PhD Fokozatszerzési Ösztöndíj”- ban részesülnek. Továbbá, ha a PhD értekezés 1 éven belül kerül benyújtásra a Doktori Iskola Hivatalába, akkor ösztöndíj kiegészítésként 400.000 Ft „Egyszeri PhD Fokozatszerzési Ösztöndíj” is illeti a fokozatszerzőt. (Ehhez az ösztöndíjhoz nem kapcsolódik „bench fee” jellegű támogatás.) PhD Fokozatszerzési Ösztöndíjban 50 főt részesítettünk 2019-ben.

A PhD fokozatszerzések számszerű növelésének másik lehetősége a képzésbe bekapcsolódó hallgatók számának a növelésére irányult, azoknak a hallgatóknak a doktori képzésbe való bevonásával, akik már a graduális-, ill. rezidens-képzésük során jelentős tudományos eredményeket értek el. Ez a célcsoport állt a Kiegészítő és ahhoz kapcsolódó Rendszeres Fokozatszerzési Kiválósági Ösztöndíj rendszerünk középpontjában. Lényegében ez egy MD/PhD ösztöndíj rendszer, amelyben a graduális TDK hallgató már az egyetemi évek alatt (utolsó 1-2 évben) elkezdi a PhD munkát, és ekkor 70 eFt/hó Kiegészítő Ösztöndíjat kap. Ez alatt az időszak alatt PhD krediteket is kell szereznie illetve egy közlésre elfogadott publikációt kell produkálnia. Ha ezeket a feltételeket a hallgató teljesíti, akkor az egyetemi diploma megszerzése után egyéni felkészülőként kerül hallgatói jogviszonyba a Doktori Iskolával, és sikeres PhD komplex vizsga letételével 180 eFt Rendszeres PhD Fokozatszerzési Kiválósági Ösztöndíjat kap maximum 2 évig. Ez alatt teljesítenie kell a PhD fokozatszerzés követelményeit, és a doktori értekezését be kell nyújtania a Doktori Iskolához. Mind ezek teljesülése esetén 400 eFt Egyszeri Fokozatszerzési Ösztöndíjban is részesül. Ezt a rendszert a rezidensekre is alkalmaztuk, vagyis a szakképzés első 2 évében a rezidens Kiegészítő Ösztöndíjat, a második 2 évében pedig Rendszeres Ösztöndíjat kap.

Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 10. oldal

Mind az MD/PhD, mind a Rezidens/PhD ösztöndíjakhoz szemeszterenként 390 eFt ”bench fee” is társul a kutatócsoportok anyag- és eszköz-igényének támogatására. Kiegészítő Kutatási Kiválósági Ösztöndíjat 33 graduális hallgató vagy rezidens kap, míg Rendszeres PhD Fokozatszerzési Kiválósági Ösztöndíjban jelenleg 16 fő részesül.

„Nemzetközileg kiemelkedő publikációs aktivitás támogatása” című alprogram

A kutatási eredmények színvonalas, nemzetközileg elismert tudományos folyóiratokban történő publikálása elengedhetetlen feltétele a PhD hallgatók fokozatszerzési folyamatának. Jelentős anyagi terhet jelent a doktoranduszra illetve a kutatócsoportra nézve, hogy jelenleg igen magasak a mérvadó folyóiratokban történő publikációval kapcsolatos költségek. Éppen ennek ellensúlyozására szán jelentős költséget az EFOP projekt a publikációs tevékenység közvetlen támogatásának formájában. Az egyik legkorábban elindult alprogram segítségével, a legjobb nemzetközi folyóiratokban közlésre kerülő, D1 kategóriájú, elsőszerzős vagy megosztott elsőszerzős közlemény megjelenésével kapcsolatos publikációs költséget támogatjuk a PhD hallgatóknak illetve a kutatócsoportjának. A publikációs költség megtérítésének maximális mértéke 500 eFt. Az egyre erősödő támogatási igény is jelzi, hogy ezzel a pályázati rendszerrel jelentősen tudjuk elősegíteni a publikációs aktivitás növekedését. A 2017. szeptembertől folyamatosan meghirdetett pályázaton eddig összesen 25 fő részesült publikációs támogatásban, a kifizetett összeg közel 6 MFt.

„A Doktori Iskola kutatási programjainak bővítése új kutatási témák indításával” című alprogram

A minőségi PhD képzés megvalósulásának egyik legfontosabb feltétele, hogy olyan új PhD kutatási témák kerüljenek akkreditálásra, amelyekben a legmodernebb nemzetközi kutatási irányvonalak, ezeken a szakterületeken elért és ismertté vált eredmények is tükröződnek illetve azokon alapulnak. A PhD kutatási témák ilyen irányú bővítése egyértelműen hozzájárul a PhD kutatómunka színvonalának emeléséhez, segítve a doktoranduszok minőségi képzését, annak érdekében, hogy a fokozatot szerző kutatók és klinikusok szakmai felkészültsége, hozzáértése jelentősen növekedjen a minőségi követelményekkel összhangban. A minőségi kutatómunka eredményeképpen fokozatot szerző hallgatók minden bizonnyal jelentős további szerepet kapnak akár az akadémiai világban, akár a verseny-szférában. Közismert ugyanakkor, hogy a korszerű, legújabb metodikai technikákat alkalmazó kutatási tevékenységek sikeres megvalósulása igen költségigényes, vagyis a minőségi PhD képzés feltétele a megfelelő, eredmény orientált forrásbiztosítás. Ennek a minőségi PhD képzésnek a megvalósítására hirdettük meg azt a pályázatot, amely segítségével az új PhD kutatási témákba

Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 11. oldal

belépő első hallgató szakmai tevékenységével összefüggő dologi kiadások célzott anyagi támogatását kívánjuk elérni. A pályázat kifejezett célja a legújabb tudományos szakterületeket érintő experimentális vagy klinikai PhD programokban résztvevő doktoranduszok tevékenységének közvetlen segítése.

A projekt összesen 10 új kutatási téma indítását támogatta, és mindegyik új témát az első felvett hallgató kutatómunkájának megvalósulása érdekében 3 éven keresztül havi 65.000 Ft kutatási hozzájárulással („bench fee”) segíti. A kutatási projektek folyamatosan hívják le a támogatásaikat, eddig valamivel több mint 6 MFt értékben.

„Új (vagy jelentősen korszerűsített) metodikai és interdiszciplináris PhD kurzusok szervezése, és a gyakorlati képzés erősítése” című alprogram

Az alprogram keretében a doktori képzés és doktori kutatási folyamat hatékonyságának és eredményességének növelése érdekében új metodikai PhD kurzusok szervezését segítettük, amelyek bővítik a PhD hallgatók metodikai ismereteit, valamint segítséget nyújtanak a felmerülő módszertani kérdések és problémák megoldásában. A program elősegíti a doktorandusz kutatások tervezését, valamint eredményes kivitelezését. Jelenleg nagy az igény a kísérleti eredmények reprodukálhatóságának javítására, melynek elengedhetetlen feltétele a helyes kísérlettervezés, majd az eredmények megfelelő statisztikai értékelése. Ennek elérésére célul tűztük ki többek között a PhD hallgatók biostatisztikai ismereteinek bővítését, valamint a klinikai kutatási módszertan oktatását. A PhD kurzusokon a gyakorlati képzés erősítésével a doktori kurzusok gyakorlatorientáltságát kívánjuk növelni. A gyakorlati oktatást előtérbe helyezve olyan készségeket fejlesztünk, amelyeket a hallgatók kutatásuk során hasznosítani tudnak. A program megvalósításában új interdiszciplináris PhD kurzusok szervezése is alapvető célkitűzés, ami lehetővé teszi, hogy a PhD hallgatók naprakész ismereteket kapjanak olyan témákban is, amelyek közvetlenül nem kapcsolódnak a tudományterületükhöz, azonban elengedhetetlenek ahhoz, hogy érvényesülni tudjanak a tudományos életben és sikeresen végezzék kutatómunkájukat.

Az alprogram keretében eddig 27 új PhD kurzus került meghirdetésre, melyek elindításához az EFOP pályázat kurzusonként 400 eFt illetménykiegészítést biztosít a tananyagfejlesztési feladatok ellátására és 700 eFt dologi támogatást az eszközköltség biztosítása a kurzus gyakorlati jellegének erősítésére. A támogatások lehívása folyamatosan történik a kurzusok megvalósulásával párhuzamosan, és eddig valamivel több mint 15 MFt kifizetés történt. A támogatás feltétele, hogy az új PhD kurzust az EFOP projekt futamideje alatt (2017-2021), az oktatási tanév legalább egyik szemeszterében magyar illetve angol nyelven is meg kell hirdetni. Ez utóbbi feltétellel sikerült elérni, hogy jelentősen nőjön az angol

Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 12. oldal

nyelvű PhD kurzusok száma, így a külföldi hallgatók is szélesebb palettáról választhatják ki kurzusaikat, ami az angol nyelvű képzés népszerűségét növeli.

Angol nyelvű PhD kurzusok száma tanévenként (2013-2019) 35 30 25 20 15 10 5 0

„Egyetemi PhD Napok, doktori iskolák PhD hallgatói szimpóziumainak szervezése” című alprogram

A fokozatszerzés alapját képező tudományos közlemények elkészítésének menetében egy fontos állomás, amikor a PhD hallgató az egyetemi doktori iskolák szélesebb közönsége előtt mutatja be saját kutatómunkájának aktuális eredményeit. Az évente megrendezésre kerülő „Egyetemi PhD Napok” rendezvény nyújt erre a prezentációra formális keretet, amelyet az EFOP projekt is támogat. Ez a tudományos fórum több oldalról segíti azt a modernizációs folyamatot, amely a kutatás hatékonyságának növekedését célozza, annak érdekében, hogy a fiatal kutatók sikeresebben publikáljanak és ennek következtében minél hamarabb nyerjenek PhD fokozatot. Egyrészt, a különböző tudományterületeket képviselő PhD hallgatók, doktoranduszok és doktorjelöltek szélesítik egymás tudományos látóterét, új megvilágításba helyezik a tudományos kérdéseket, és egyben segítik az új tudományos felfedezések terjesztését. Másrészt, a szimpóziumon a saját tudományos munka ismertetése egy barátságos környezetben történik, és a közvetlen visszajelzések nagy segítséget nyújtanak az előadói képességek fejlesztéséhez. Végül, a hallgatóknak ezen a fórumon lehetőségük van a kapcsolati hálójukat építeni mind a többi fiatal tehetséggel, mind azok mentoraival, témavezetőivel, valamint az egyetem szakmai életében meghatározó szerepet

Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 13. oldal

betöltő egyetemi oktatókkal, kutatókkal. A legjobb prezentációkat díjakkal jutalmazzuk, a konferencia kiadványban megjelenő absztraktok pedig a fokozatszerzés folyamatát és sikerességét alátámasztó szakmai indikátorok alapvető dokumentumai is egyben a projekt részéről. A 2018. évi PhD napokon összesen 185 PhD hallgató, míg a 2019. évben megrendezett PhD napokon összesen 113 hallgató tartott sikeres poszter vagy előadás prezentációt.

„Doktorandusz hallgatók és predoktorok TDK-témavezetésének elismerése” című alprogram

A minőségi PhD képzés megvalósításának és a kutatói utánpótlás biztosításának folyamatában lényeges előrelépést jelent a doktoranduszok és predoktorok bevonása a fiatalabb hallgatók oktatásába, TDK témavezetésébe. Ez egyrészt a TDK hallgatók számára előnyös, hiszen a kommunikáció lényegesen kötetlenebb és egyszerűbb, ha korban egymáshoz közelebb álló személyek dolgozhatnak együtt. A sikeres TDK kutatómunka feltétele, hogy jó kapcsolat épüljön ki témavezető és a TDK hallgató között, és az azonos kommunikációs csatornák használata jelentősen megkönnyíti, hogy rendszeres, hatékony együttműködés alakuljon ki közöttük. A másik oldalról a TDK témavezetés lehetőséget teremt a PhD hallgatók számára, hogy tapasztalatot szerezzenek az oktatásban, a fiatalabb hallgatókkal való kooperációban, így összességében új kihívások elé kerülnek, amelyek motivációt adnak a folyamatos szakmai fejlődésre. Az EFOP projekt a 2018-2021 években a rendelkezésre álló keret erejéig támogatja az egyetem PhD hallgatóit és a predoktorait TDK-témavezetés vállalására. A pályázat ösztönzi a témavezetőket a TDK-s hallgatóikkal történő szoros kutatási együttműködésre. Ennek keretén belül a PhD hallgató vállalja, hogy a TDK-s hallgatóját szakmailag megfelelően felkészíti az egyetemi és országos TDK konferenciákra a sikeres szereplés érdekében. A legkiválóbb témavezetők szakmai felkészítésének sikerességét, minden évben az egyetemi TDK konferencia után, 150 eFt egyszeri ösztöndíj kiegészítéssel jutalmazza a projekt. Az alprogram keretén belül 2018- ban 6 PhD hallgató, 2019-ben pedig 18 PhD hallgató/predoktor részesült díjazásban, összesen 3,6 MFt értékben.

„PhD és TDK-hallgatók hazai/külföldi konferencián vagy tudományos továbbképzési kurzuson való részvételének támogatása” című alprogram

A fiatal orvosi, fogorvosi, gyógyszerész és egészségtudományi posztgraduális kutatók és sikeres kutatómunkát végző TDK hallgatók bekapcsolása a nemzetközi tudományos életbe elengedhetetlen a minőségi kutatói utánpótlás biztosítása érdekében. A nemzetközi konferenciákon résztvevő hallgatók megismerhetik szűkebb/tágabb szakterületük vezető kutatóit, első kézből értesülhetnek a legfrissebb tudományos eredményekről, és személyes beszélgetések során olyan ismeretekre is szert tehetnek, amelyekre pusztán a publikációk követésével nem lehetne mód (pl. metodikai kihívások, publikálatlan eredmények). A hallgatók saját

Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 14. oldal

eredményeinek bemutatása idegen nyelven, hozzáértő nemzetközi kutatóközösség előtt történik, amely nagyban hozzájárul az előadói és vitakészség fejlődéséhez. Természetesen, a konferencia részvétel a PhD/TDK hallgató tudományos munkájának előrehaladását is jelentősen elősegíti a hallgatóság kritikai észrevételei, ötletei alapján, mint ahogy jövőbeli kutatási együttműködések, munkakapcsolatok kiépítésére, tanulmányutak tervezésére is lehetőség nyílik.

A támogatás feltétele egy elfogadott elsőszerzős vagy megosztott elsőszerzős előadás vagy poszter, amely az utazás céljául választott nemzetközi konferencián bemutatásra kerül. A pályázatok bírálata során, a támogatás megítélésekor a konferencia részvétellel kapcsolatos regisztrációs díj, útiköltség és szállásköltség megtérítése csak abban az esetben haladhatja meg a bruttó 300.000 Ft értéket, ha a pályázó PhD tudományos teljesítménye a pályázat benyújtásakor nagyban valószínűsíti, hogy a konferencián bemutatásra kerülő tudományos eredményekhez kapcsolódó doktori értekezés a PhD képzés eredeti határidejére elkészül. Az alprogram keretén belül eddig 5 alkalommal került meghirdetésre ez a pályázat az EFOP projekt részéről, és összesen 89 fő részesült támogatásban. Az összes igényelt támogatásból eddig 18 MFt kifizetés történt.

„Országos Orvos- és Gyógyszerésztudományi PhD Konferencia” című alprogram

A kutatás minőségi fejlesztését és hatékonyságának növelését szolgálja a Semmelweis Egyetem 250. jubileumi tanévében tervezett országos PhD konferencia, amely az orvos- és gyógyszerész PhD hallgatók számára először kerül megszervezésre, interdiszciplináris jelleggel. A tudományos rendezvényre a konzorciumban részt vevő négy hazai egyetemről érkezhetnek a PhD hallgatók, doktoranduszok és doktorjelöltek, hogy bemutassák tudományos munkájukat, és a jövő kutatói generációja lehetőséget kapjon a saját egyetemi világukon kívüli kapcsolatszerzésre. A tervek szerint, az országos konferencia résztvevői között orvosi és gyógyszerészi témában fokozatot szerzők is lesznek, amely megfelel a modern kutatásban előtérbe került irányvonalaknak, a tudományos témák és technikák sokszínűségének, interdiszciplináris jellegének. A hasonló kutatási területen tevékenykedő PhD hallgatók személyes kapcsolatteremtésén kívül, a határterületeken dolgozó PhD hallgatók és témavezetők ismeretcseréjére is sor kerül, amely révén komplex, több területet átfogó kutatási projektek valósulhatnak meg. Összességében, ez a rendezvény is a minőségi kutatói utánpótlás érdekeit szolgálja a tudományos kutatás színvonalának emelésén keresztül.

Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 15. oldal

Vezetői program

Motiváció A doktori képzés a hazai felsőoktatási rendszer csúcsintézménye, melynek legfontosabb célja a kutatói és oktatói utánpótlás magas színvonalú nevelése. A doktori iskolák legfőbb feladatát abban látom, hogy megszólítsák a legkiválóbb hallgatókat, motiválják őket kutatómunka végzésére, segítsék szakmai fejlődésüket és biztosítsák tovább lépési lehetőségüket a tudományos élet vagy a gazdasági szféra felé. Az Országos Doktori Tanács az alábbi közhasznú tevékenységeket látja el: 1. a tudományos tevékenység, kutatás - ezen belül a doktori képzés és fokozatszerzés - empirikus vizsgálata, az egyetemek gyakorlatának elemzése, a kutatási tevékenység működési feltételeinek elősegítése pályázatok útján és egyéb módon; 2. oktatás, képességfejlesztés, ismeretterjesztés, ezen belül szakmai tudományos fórumok, illetve tudományos közélet szervezése; szakmai kiadványok megjelentetése, a tudományos fokozat megfelelő szintű elismertségének biztosítása, információk terjesztése, PR tevékenység ellátása; 3. működtetheti a fokozatszerzési eljárások és értekezések nyilvántartását. Fentiek alapján az ODT küldetése, hogy segítse a doktori iskolákat feladataik hatékony ellátásában.

Helyzetelemzés és célkitűzések A hazai kutatás, fejlesztés és innováció (K+F+I) színvonala elmarad a kívánatostól, aminek egyik fő oka a magasan képzett szakemberek alacsony száma. Magyarország a Partnerségi Megállapodásban, valamint a Nemzetgazdasági Minisztérium „Befektetés a jövőbe” címet viselő Nemzeti Kutatás-fejlesztési és Innovációs Stratégiájában megfogalmazott célkitűzése a kutatói létszám jelentős, közel 50%-os növelése. E cél elérésének egyik eszköze az államilag finanszírozott PhD ösztöndíjak számának a „Fokozatváltás a felsőoktatásban” című stratégiai program keretében 2019-ben bevezetett másfélszeres növelése, valamint a felsőoktatási hallgatók tudományos műhelyeinek támogatására kiírt EFOP-3.6.3- VEKOP program. Sajnos az utóbbi években folyamatosan csökken az érdeklődés a doktori képzés iránt, aminek okait döntően a következőkben látom: 1. A pályakezdő diplomások javuló anyagi megbecsülése: a közszférában és még inkább a gazdasági szférában a fizetéseknek, a szakképzésben az Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 16. oldal

ösztöndíjaknak és kiegészítő juttatásoknak a PhD ösztöndíjaknál jelentősen gyorsabb emelkedése. 2. A doktori képzés megnyúlása 3-ról 4 évre, főleg azok körében, akik utána szeretnének szakképzésbe is bekapcsolódni, ugyanis az 5-6 év alapképzés, 4 év doktori képzés, majd 3-6 év szakképzés sokak számára nem vállalható anyagi/egzisztenciális szempontból. Különösen igaz ez a nők körében, akik ebben az életszakaszban szeretnének gyermeket is vállalni. 3. A külföld elszívó hatása, ami éppen a legtehetségesebb potenciális PhD hallgatók esetében érvényesül rendkívül erőteljesen. Több éves tapasztalat, hogy a PhD felvételi vizsgán legtöbb pontot szerző hallgatók közül többen végül nem iratkoznak be a képzésbe, hanem külföldre mennek doktorandusznak jobb anyagi és munka-körülményekben bízva. A doktori képzés iránti érdeklődés csökkenése hosszú távon a PhD témavezetői utánpótlás csökkenésén keresztül a folyamat további felerősödésének veszélyét vetíti előre. Ennek megállítása és visszafordítása a következő évek egyik legfontosabb feladata. A doktori képzés alulfinanszírozottsága különösen szembeötlő a kísérletes kutatásokat folytató tudományterületeken. Az egyetemekhez jutó képzési célú támogatás (975 eFt/év) még a kísérletes munka dologi kiadásainak fedezésére sem elég, azt a témavezetők kutatási pályázati forrásokkal kénytelenek kiegészíteni, amit csak részben kompenzál az a tény, hogy a kutatásban részt vevő hallgató állami ösztöndíjban részesül. Ugyanakkor a PhD képzésben dolgozó témavezetők és a kurzusok oktatóinak bér jellegű költségeire egyáltalán nincs állami finanszírozás, ezeket az egyetemek és kutatóintézetek egyéb bevételeikből fedezik. Ez óhatatlanul feszültséget szül a finanszírozást biztosító karok és a doktori iskolák között, ami vélhetőleg tovább fog fokozódni a PhD felvételi keretszámok idén bevezetett másfélszeres megemelése után. A témavezetői és kurzus-oktatói bérek fedezetének hiányához képest kisebb tétel, de a működést jelentősen akadályozó probléma, hogy a doktori iskoláknak nincs lehetőségük a képzéshez szükséges egyéb szakmai tevékenységek (értekezések és belső pályázatok bírálata, vizsga-, védési és felvételi bizottságok feladatai, tananyagfejlesztés, oktatási adminisztráció) ellentételezésére sem.

Vezetői tervek Az előzőekben vázolt problémák megoldásához hozzájárulhat a doktori képzés és az arra való felkészülést biztosító tudományos diákköri rendszer népszerűsítése. Ennek egyik módja a hivatalos és a közösségi médiában való fokozottabb megjelenés, melyhez az ODT az egyes doktori iskolákban már meglévő jó gyakorlat továbbadásával tud hozzájárulni. Kiaknázatlan lehetőségek vannak a Kutatók Éjszakája programsorozatban is, az ezeken a rendezvényeken nagy számban megjelenő, tudomány iránt érdeklődő Dr. Benyó Zoltán, Pályázat Országos Doktori Tanács Társelnöki Megbízatásra 2020-2022, 17. oldal

középiskolások megszólítása fontos feladat. A Semmelweis Egyetem 2020 márciusában az egyetemi TDK konferenciával összefonódva és a társegyetemekkel együttműködve Országos Orvos- és Egészségtudományi PhD Konferenciát szervez, ennek esetleges pozitív tapasztalatai szintén felhasználhatók lesznek a doktori képzés népszerűsítésére más tudományterületeken is. A finanszírozási problémák megoldása részben az állami szerepvállalás fokozásával, illetve a már működő nemzeti pályázati rendszerek bővítésével lenne lehetséges, amihez az ODT-nek a fenntartóval folytatott tárgyalásai vezethetnek el. Másik lehetőség a doktori iskolák saját bevételi forrásainak fokozása, aminek kézenfekvő módja a gazdasági szféra szereplőinek bevonása a doktori képzés finanszírozásába, valamint a külföldi hallgatók számára biztosított önköltséges képzési rendszer fejlesztése. Ennek kézen fekvő „célközönsége” a Magyarországon tanuló nagyszámú külföldi hallgató, akik közül egyre többen kapcsolódnak be a tudományos diákköri munkába, és növekszik az egyetemi és országos TDK konferenciákon díjat szerzők aránya is. Emellett a világ néhány rendkívül dinamikusan fejlődő országában, pl. Kínában, a PhD képzési kapacitás elmarad az igényektől, így reális esély van innen önköltséges vagy a küldő ország által támogatott ösztöndíjas PhD hallgatók toborzására. Az ODT elsősorban a partner országok/egyetemek felkutatásával tudná segíteni a doktori iskolák ilyen irányú aktivitását. A fentieken kívül a doktori képzés iránti érdeklődés fokozása céljából olyan rugalmas képzési struktúrák bevezetését is indokoltnak tartom, amik szigorú feltételek mentén lehetővé teszik, hogy a doktori képzés első (képzési és kutatási) szakasza a graduális, illetve a második (kutatási és disszertációs) szakasza a szakképzéssel párhuzamosan is végezhető legyen. Erre lehetőséget nyújtana az, ha az osztatlan alapképzésben részt vevő hallgatók közül azok, akik már TDK munkájuk során jelentős tapasztalatot szereztek és eredményeket értek el a kutatásban, 1-2 évvel diplomájuk megszerzése előtt bekapcsolódhatnának a PhD képzésbe, hasonlóan a korábban működő MD/PhD rendszerhez. Ezért az osztatlan képzést folytató egyetemi karokkal és a hozzájuk kötődő doktori iskolákkal együttműködve kezdeményezni szeretnék olyan jogszabály-módosítást, mely lehetővé tenné, hogy a kiemelkedő tudományos diákköri eredményekkel rendelkező, graduális képzésben részt vevő hallgató pályázhasson állami PhD ösztöndíjra, ezzel megvalósítva a párhuzamos graduális és doktori képzés lehetőségét. Emellett az is tény, hogy a hallgatók a doktori képzés során sok olyan szakmai ismeretre, készségre tesznek szert, ami a szakképzésüknek is tárgya. Emiatt, azon kiváló tehetségű és szorgalmú PhD hallgatók részére, akik a komplex vizsga idejében már teljesítik a doktori fokozatszerzéshez szükséges feltételeket, indokoltnak tartanám a szakképzésbe való bekapcsolódás lehetőségének biztosítását az állami ösztöndíjas doktori képzéssel párhuzamosan.

Benyó Zoltán (Élettan és kórélettan)

MTMT közlemény és idéző összefoglaló táblázat Benyó Zoltán adatai (2019.11.11)

Közlemény típusok Száma Hivatkozások1 Teljes tudományos közlemények2 Összes Részletezve Független Összes I. Tudományos folyóiratcikk 66 ------nemzetközi szakfolyóiratban --- 62 1402 1701 hazai kiadású szakfolyóiratban idegen --- 2 18 21 nyelven hazai kiadású szakfolyóiratban magyar --- 2 11 12 nyelven II. Könyvek 1 ------a) Könyv, szerzőként 1 ------idegen nyelvű --- 0 0 0 magyar nyelvű --- 1 0 0 b) Könyv, szerkesztőként 0 ------idegen nyelvű --- 0 3------magyar nyelvű --- 0 ------III. Könyvrészlet 1 ------idegen nyelvű --- 1 0 0 magyar nyelvű --- 0 0 0 IV. Konferenciaközlemény 11 ------folyóiratban vagy konferenciakötetben idegen nyelvű --- 11 33 50 magyar nyelvű --- 0 0 0 Tudományos közlemények összesen 79 --- 1464 1784 (I.-IV.) További tudományos művek4 --- 40 0 0

Idézetek száma5 ------1471 1801 Hirsch index5 19 ------

Oktatási művek Felsőoktatási tankönyv 0 ------idegen nyelvű --- 0 0 0 magyar nyelvű --- 0 0 0 Felsőoktatási tankönyv része idegen --- 0 0 0 nyelven Felsőoktatási tankönyv része magyar --- 0 0 0 nyelven További oktatási művek 0 --- 0 0

Oltalmi formák 0 --- 0 0

Alkotás 0 --- 0 0

Ismeretterjesztő művek Könyvek 0 --- 0 0 További ismeretterjesztő művek 0 --- 0 0

Közérdekű és nem besorolt művek 0 --- 0 0

Absztrakt 114 --- 7 17 Egyéb szerzőség 0 --- 0 0 Idézők szerkesztett művekre ------0 0 Idézők disszertációban, egyéb ------9 12 típusban Összes közlemény és összes idézőik 233 --- 1480 1813 Megjegyzések A táblázat számai hivatkozások is. A számra kattintva a program listázza azokat a műveket, amelyeket a cellában összeszámlált. --- : Nem kitölthető cella 1A hivatkozások a disszertáció és egyéb típusú idézők nélkül számolva. A disszertáció és egyéb típusú idézők összesítve a táblázat végén találhatók. 2Teljes tudományos közlemény ebben az adatbázisban: - Folyóiratcikk : szakcikk/tanulmány, összefoglaló cikk, rövid közlemény, sokszerzős vagy csoportos szerzőségű közlemény, forráskiadás, recenzió/kritika, műkritika, esszé - Könyv: szakkönyv, monográa, kézikönyv, tanulmánykötet, forráskiadás, kritikai kiadás, műhelytanulmány, atlasz - Könyvrészlet: szaktanulmány, fejezet, esszé, forráskiadás, recenzió/kritika, műkritika, műtárgyleírás, térkép, műhelytanulmány része - Konferenciaközlemény: folyóiratban, könyvben, egyéb konferenciakötetben megjelent legalább 3 oldal terjedelemben - Oltalmi formák: szabadalmak, mintaoltalmak (részletek) 3Szerkesztőként nem részesedik a könyv idézéséből 4Ide értve a teljes közlemények listájában nem szereplő publikációkat, a nem ismert lektoráltságú folyóiratokban megjelent műveket és minden olyan tudományos művet, amely a táblázat más, nevesített soraiban nem került összeszámlálásra. 5A disszertációk és egyéb típusú idézők nélkül számolva (részletek) A sor értéke a "Tudományos közlemények összesen (I.-IV.)", a "További tudományos művek" és az "Absztrakt" sorok idézettség értékeit összegzi. 2019. nov. 11. 16:52 Benyó Zoltán (Élettan és kórélettan)

1. Besztercei, B. ; Vancsik, T. ; Benedek, A. ; Major, E. ; Thomas, M.J. ; Schvarcz, C.A. ; Krenács, T. ; Benyó, Z. ; Balogh, A. Stress-induced, p53-mediated tumor growth inhibition of melanoma by modulated electrohyperthermia in mouse models without major immunogenic eects INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 20 Paper: 4019 , 16 p. (2019) DOI WoS Scopus PubMed Közlemény:30785596 Jóváhagyott Forrás Idéző Folyóiratcikk (Szakcikk ) Idézett közlemények száma: 2

2. Dér, Bálint ; Molnár, Péter József ; Ruisanchez, Éva ; Őrsy, Petra ; Kerék, Margit ; Faragó, Bernadett ; Nyirády, Péter ; Oermanns, Stefan ; Benyó, Zoltán NK2 receptor-mediated detrusor muscle contraction involves G q/11 -dependent activation of voltage-dependent Ca 2+ channels and the RhoA-Rho kinase pathway AMERICAN JOURNAL OF PHYSIOLOGY: RENAL PHYSIOLOGY 317 : 5 pp. F1154-F1163. (2019) DOI Egyéb URL Közlemény:30877169 Admin láttamozott Forrás Folyóiratcikk (Szakcikk )

3. Hadjadj, Leila ; Pál, Éva ; Monori-Kiss, Anna ; Sziva, Réka Eszter ; Korsós-Novák, Ágnes ; Mária Horváth, Eszter ; Benkő, Rita ; Magyar, Attila ; Magyar, Péter ; Benyó, Zoltán et al. Vitamin D deciency and androgen excess result eutrophic remodeling and reduced myogenic adaptation in small cerebral arterioles in female rats GYNECOLOGICAL ENDOCRINOLOGY 35 : 6 pp. 529-534. , 6 p. (2019) DOI PubMed SE Repozitórium WoS Scopus Zárolt Közlemény:30390754 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 1 Független: 0 Függő: 1 Idézett közlemények száma: 3 1. * Pal Eva et al. Gender, hyperandrogenism and vitamin D deciency related functional and morphological alterations of rat cerebral arteries. (2019) PLOS ONE 1932-6203 1932-6203 14 5

4. Hadjadj, Leila ; Monori-Kiss, Anna ; Horváth, Eszter M. ; Heinzlmann, Andrea ; Magyar, Attila ; Sziva, Réka E. ; Miklós, Zsuzsanna ; Pál, Éva ; Gál, János ; Szabó, István et al. Geometric, elastic and contractile-relaxation changes in coronary arterioles induced by Vitamin D deciency in normal and hyperandrogenic female rats MICROVASCULAR RESEARCH 122 pp. 78-84. , 7 p. (2019) DOI PubMed SE Repozitórium WoS Scopus Zárolt Közlemény:30338457 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Idézett közlemények száma: 6

5. Janovicz, Anna ; Majer, Alíz ; Balogh, Lili ; Balogh, Andrea ; Tigyi, Gábor ; Benyó, Zoltán ; Ruisanchez, Éva Role of the LPC-ATX-LPA Pathway in the Development of Endothelial Dysfunction (2019) The Lysophospholipid and Related Mediators Conference: From Bench to Clinic, Egyéb URL Közlemény:30792704 Admin láttamozott Forrás Egyéb (Nem besorolt )

6. Markovics, Arnold ; Toth, Kinga Fanni ; Sos, Katalin Eszter ; Magi, Jozsef ; Gyongyosi, Adrienn ; Benyo, Zoltan ; Zouboulis, Christos C. ; Biro, Tamas ; Olah, Attila Nicotinic acid suppresses sebaceous lipogenesis of human sebocytes via activating hydroxycarboxylic acid receptor 2 (HCA(2)) JOURNAL OF CELLULAR AND MOLECULAR MEDICINE 9 : 23 pp. 6203-6214. , 12 p. (2019) DOI ISSN: 1582-1838 WoS Scopus PubMed Közlemény:30781513 Admin láttamozott Forrás Idéző Folyóiratcikk (Szakcikk ) Idézett közlemények száma: 2

7. Pal, Eva* ; Hadjadj, Leila* ; Fontanyi, Zoltan ; Monori-Kiss, Anna ; Lippai, Norbert ; Horvath, Eszter M. ; Magyar, Attila ; Horvath, Eszter ; Monos, Emil ; Nadasy, Gyorgy L. et al. Gender, hyperandrogenism and vitamin D deciency related functional and morphological alterations of rat cerebral arteries PLOS ONE 14 : 5 Paper: e0216951 , 13 p. (2019) DOI PubMed SE Repozitórium WoS Scopus Zárolt Közlemény:30701767 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Idézett közlemények száma: 8 8. Pawlak, John B ; Bálint, László ; Lim, Lillian ; Ma, Wanshu ; Davis, Reema B ; Benyo, Zoltan ; Soares, Michael J ; Oliver, Guillermo ; Kahn, Mark L ; Jakus, Zoltán et al. Lymphatic mimicry in maternal endothelial cells promotes placental spiral artery remodeling. JOURNAL OF CLINICAL INVESTIGATION (2019) DOI REAL SE Repozitórium PubMed Közlemény:30767578 Érvényesített Forrás Folyóiratcikk (Szakcikk )

9. Péter, Hamar ; Lea, Danics ; Csaba, Schvarcz ; Zita, Zolcsák ; Zoltán, Benyó ; Tamás, Kaucsár Modulated electro hyperthermia inhibits tumor progression in a triple negative mouse breast cancer model FASEB JOURNAL 33 p. 1 (2019) Közlemény:30625662 Jóváhagyott Forrás Folyóiratcikk (Absztrakt / Kivonat )

10. Ungvari, Zoltan ; Tarantini, Stefano ; Nyúl-Tóth, Ádám ; Kiss, Tamas ; Yabluchanskiy, Andriy ; Csipo, Tamas ; Balasubramanian, Priya ; Lipecz, Agnes ; Benyo, Zoltan ; Csiszar, Anna Nrf2 dysfunction and impaired cellular resilience to oxidative stressors in the aged vasculature: from increased cellular senescence to the pathogenesis of age-related vascular diseases. GEROSCIENCE: OFFICIAL JOURNAL OF THE AMERICAN AGING ASSOCIATION (AGE) 2019 p. In press (2019) DOI PubMed Közlemény:30876980 Jóváhagyott Forrás Folyóiratcikk (Szakcikk )

11. Benyó, Zoltán ; Ruisanchez, Éva ; Tigyi, Gábor LIZOFOSZFOLIPID MEDIÁTOROK SZEREPE A VÉRKERINGÉSI RENDSZER ÉLETTANI ÉS KÓRÉLETTANI FOLYAMATAIBAN (2018) Teljes dokumentum Közlemény:30539745 Jóváhagyott Forrás Egyéb (Nem besorolt )

12. Besztercei, Balázs ; Benedek, Anett ; Major, Enikő ; Hricisák, László ; Fülöp, Ágnes ; Benyó, Zoltán ; Balogh, Andrea Modulált elektrohipertermia hatása endotélsejtekre Paper: P2.1 (2018) Teljes dokumentum Közlemény:30519991 Admin láttamozott Forrás Egyéb (Nem besorolt )

13. Dobrydnio, Laura S ; Gál, Piroska ; Jabronka, Nikolett ; Hricisák, László ; Polycarpou, Andreas ; Benyó, Zoltán Relevance of endothelial and neuronal NOS int he recovery of the cerebrocortical blood ow after unilateral carotid artery occlusion in mice In: Recoop, HST Association - Recoop HST Association - Student Conference - Recoop- Frigyes Korányi Science Forum Budapest, Magyarország : Recoop HST Association, (2018) p. 53 Közlemény:30792892 Admin láttamozott Forrás Könyvrészlet (Absztrakt / Kivonat )

14. Hadjadj, L ; Varbiro, S ; Horvath, EM ; Monori-Kiss, A ; Pal, E ; Karvaly, GB ; Heinzlmann, A ; Magyar, A ; Szabo, I ; Sziva, RE et al. Insulin resistance in an animal model of polycystic ovary disease is aggravated by vitamin D deciency: Vascular consequences DIABETES & VASCULAR DISEASE RESEARCH 15 : 4 pp. 294-301. , 8 p. (2018) DOI WoS PubMed SE Repozitórium Scopus Zárolt Közlemény:3341886 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 4 Független: 2 Függő: 2 Idézett közlemények száma: 1

1. Gunasekar Palanikumar et al. Vitamin D and macrophage polarization in epicardial adipose tissue of atherosclerotic swine. (2018) PLOS ONE 1932-6203 1932-6203 13 10 2. * Pal Eva et al. Gender, hyperandrogenism and vitamin D deciency related functional and morphological alterations of rat cerebral arteries. (2019) PLOS ONE 1932-6203 1932-6203 14 5 3. * Hadjadj Leila et al. Geometric, elastic and contractile-relaxation changes in coronary arterioles induced by Vitamin D deciency in normal and hyperandrogenic female rats. (2019) MICROVASCULAR RESEARCH 0026-2862 1095-9319 122 78-84 4. Shen Qiyang et al. Nontargeted metabolomic analysis of skeletal muscle in a dehydroepiandrosterone-induced mouse model of polycystic ovary syndrome. (2019) MOLECULAR REPRODUCTION AND DEVELOPMENT 1040-452X 1098-2795 86 4 370-378 15. Hricisák, L ; Simoes, Dobrydnio L ; Jabronka, N ; Portörő-Gál, P ; Janovicz, A ; Polycarpou, A ; Ruisanchez, É ; Sándor, P ; Benyó, Z NOS izoenzimek szerepe az egér agykérgi váráramlásának szabályozásában Paper: E11 (2018) Teljes dokumentum Közlemény:30519748 Admin láttamozott Forrás Egyéb (Nem besorolt )

16. Hricisák, L ; Dobrydnio, Simoes L ; Jabronka, N ; Portörő-Gál, P ; Janovicz, A ; Polycarpou, A ; Benyó, Z Relevance of endothelial and neuronal NOS in the autoregulation of cerebrocortical blood ow in mice (2018) Közlemény:30519507 Admin láttamozott Forrás Egyéb (Nem besorolt )

17. Janovicz, Anna ; Kerkovits, Nóra Melinda ; Ruisanchez, Éva ; Benyó, Zoltán A proteáz-aktivált receptor 1 jelátviteli folyamatai endotéliumban és érsimaizomban In: Membrán-Transzport Konferencia : poszter absztraktok Sümeg, Magyarország : Remedicon Kft., (2018) Paper: 28 , 1 p. Teljes dokumentum Közlemény:30548856 Admin láttamozott Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat )

18. Janovicz, Anna ; Kerkovits, Nóra Melinda ; Ruisanchez, Éva ; Benyó, Zoltán A proteáz-aktivált receptor 1 jelátviteli folyamatai az értónus szabályozásában ÉRBETEGSÉGEK / HUNGARIAN JOURNAL OF VASCULAR DISEASES 2018 : 3 p. 114 (2018) Közlemény:30546150 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

19. Janovicz, Anna ; Kerkovits, Nóra Melinda ; Ruisanchez, Éva ; Benyó, Zoltán A proteáz-aktivált receptorok jelátviteli folyamatai endotéliumban és érsimaizomban Paper: P1.32 (2018) Teljes dokumentum Közlemény:30520292 Admin láttamozott Forrás Egyéb (Nem besorolt )

20. Jeremiah, Thomass ; Major, Enikő ; Benedek, Anett ; Besztercei, Balázs ; Fülöp, Ágnes ; Benyó, Zoltán ; Balogh, Andrea Protocol for studying the synergistic therapeutic eect of modulated electro-hyperthermia and aspirin on melanoma lung metastasis in a mouse model Paper: P2.63 (2018) Teljes dokumentum Közlemény:30520811 Jóváhagyott Forrás Egyéb (Nem besorolt )

21. Markovics, Arnold ; Tóth, Kinga Fanni ; Sós, Katalin Eszter ; Magi, József ; Gyöngyösi, Adrienn ; Benyó, Zoltán ; Christos, C. Zouboulis ; Bíró, Tamás ; Oláh, Attila A nikotinsav a „hydoxycarboxylic acid receptor 2” (HCA2) aktiválásával csökkenti a humán szebociták faggyúlipid-termelését Paper: P1.70 (2018) Teljes dokumentum Közlemény:30520496 Jóváhagyott Forrás Egyéb (Nem besorolt )

22. Miklós, Zsuzsanna ; Wafa, Dina ; Gál, Benjámin ; Ruisanchez, Éva ; Kerék, Margit ; Balogh, Andrea ; Oermanns, Stefan ; Chun, Jerold ; Tigyi, Gábor ; Benyó, Zoltán LYSOPHOSPHATIDIC ACID INDUCES STRONG VASOCONSTRICTION IN THE CORONARIES VIA MULTIPLE SIGNALLING PATHWAYS , 1 p. (2018) Teljes dokumentum Közlemény:30539332 Jóváhagyott Forrás Egyéb (Nem besorolt )

23. Molnár, Péter J ; Dér, Bálint ; Borbás, Zsóa ; Molnár, Krisztina ; Ruisanchez, Éva ; Stefan, Oermanns ; Benyó, Zoltán Signal transduction pathways of the thromboxane prostanoid receptor in urinary bladder smooth muscle Paper: P6-1 , 1 p. (2018) Teljes dokumentum Közlemény:30539631 Jóváhagyott Forrás Egyéb (Nem besorolt )

24. Molnár, Péter József ; Dér, Bálint ; Borbás, Zsóa ; Molnár, Krisztina ; Ruisanchez, Éva ; Oermanns, Stefan ; Benyó, Zoltán Signal transduction pathways of prostanoids and isoprostanes in urinary bladder smooth muscle p. 97 Paper: Poster 40 , 1 p. (2018) Teljes dokumentum Közlemény:30539452 Jóváhagyott Forrás Egyéb (Nem besorolt ) 25. Molnár, Péter József ; Dér, Bálint ; Borbás, Zsóa ; Molnár, Krisztina ; Ruisanchez, Éva ; Benyó, Zoltán Tachykinin receptorok szerepe és jelátviteli folyamatai a húgyhólyag simaizom-kontrakció szabályozásában Paper: E1 (2018) Teljes dokumentum Közlemény:30520080 Jóváhagyott Forrás Egyéb (Nem besorolt )

26. Németh, Tamás ; Ruisanchez, Éva ; Hricisák, László ; Fülöp, Ágnes ; Irig, András ; Káldi, Krisztina ; Várnai, Péter ; Smrcka, Alan V ; Merkely, Béla ; Oermanns, Stefan et al. Foszfolipáz C epszilon szerepe a nitrogén-monoxid hiányhoz kapcsolódó hipertóniában [The role of phospholipase C epsilon in nitric oxide deciency associated hypertension] CARDIOLOGIA HUNGARICA 48 : Suppl. C pp. C35-C36. , 2 p. (2018) Közlemény:3380109 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

27. Pal, E ; Hadjadj, L ; Fontanyi, Z ; Monori-Kiss, A ; Mezei, Z ; Lippai, N ; Magyar, A ; Heinzlmann, A ; Karvaly, G ; Monos, E et al. Vitamin D deciency causes inward hypertrophic remodeling and alters vascular reactivity of rat cerebral arterioles. PLOS ONE 13 : 2 Paper: e0192480 , 16 p. (2018) DOI PubMed SE Repozitórium WoS Scopus Zárolt Közlemény:3333552 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 7 Független: 5 Függő: 2 Idézett közlemények száma: 4 1. Братчиков О.И. et al. Vitamin D deciency, metabolic syndrome, and prostate adenoma: current epidemiological trends and pathophysiological mechanisms of interaction. (2018) UROLOGIYA 1728-2985 4 179-185 2. Patel S. Vitamin D in inammation mitigation and role as signaling molecule. (2018) GENE REPORTS 2452-0144 12 74-80 3. * Pal Eva et al. Gender, hyperandrogenism and vitamin D deciency related functional and morphological alterations of rat cerebral arteries. (2019) PLOS ONE 1932-6203 1932-6203 14 5 4. * Hadjadj Leila et al. Geometric, elastic and contractile-relaxation changes in coronary arterioles induced by Vitamin D deciency in normal and hyperandrogenic female rats. (2019) MICROVASCULAR RESEARCH 0026-2862 1095-9319 122 78-84 5. Huang Yue et al. Vitamin D alleviates airway remodeling in asthma by down-regulating the activity of Wnt/beta-catenin signaling pathway. (2019) INTERNATIONAL IMMUNOPHARMACOLOGY 1567-5769 1878-1705 68 88-94 6. Zhou W. et al. Vitamin D attenuates lipopolysaccharide-induced inammatory response in endothelial cells through inhibition of PI3K/Akt/NF-κB signaling pathway. (2019) PHARMAZIE 0031-7144 74 7 412-417 7. Zhou Wen et al. Vitamin D suppresses lipopolysaccharide-induced inammatory response in vascular smooth muscle cells via inhibition of the p38 MAPK signaling pathway. (2019) PHARMAZIE 0031-7144 74 3 168-174

28. Pal, E. ; Hadjadj, L. ; Fontanyi, Z. ; Monori-Kiss, A. ; Mezei, Z. ; Lippai, N. ; Magyar, A. ; Heinzlmann, A. ; Karvaly, G. ; Monos, E. et al. VITAMIN D DEFICIENCY CAUSES INWARD HYPERTROPHIC REMODELING AND ALTERS VASCULAR REACTIVITY OF RAT CEREBRAL ARTERIOLES JOURNAL OF HYPERTENSION 36 pp. e296-e297. (2018) DOI WoS Egyéb URL Zárolt Közlemény:30539089 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

29. Pál, Éva ; Hadjadj, Leila ; Fontányi, Zoltán ; Monori-Kiss, Anna ; Mezei, Zsuzsanna ; Lippai, Norbert ; Magyar, Attila ; Heinzlmann, Andrea ; Karvaly, Gellért ; Makk, Jennifer et al. Vitamin D deciency causes hypertrophic remodeling and impairs vascular reactivity of rat cerebral arteries (2018) MAGYAR TUDÓS IFJÚ HYPERTONOLOGUSOK ÉS IFJÚ NEPHROLOGUSOK ÉRTEKEZLETE - V. MATHINÉ, Egyéb URL Közlemény:30792734 Admin láttamozott Forrás Egyéb (Nem besorolt ) 30. Pál, Éva ; Hadjadj, Leijla ; Fontányi, Zoltán ; Monori-Kiss, Anna ; Mezei, Zsuzsanna ; Lippai, Norbert ; Magyar, Attila ; Heinzlmann, Andrea ; Karvaly, Gellért ; Monos, Emil et al. Vitamin D deciency impairs geometrical structure and vascular reactivity of rat cerebral arterioles. (2018) Zárolt Közlemény:30538950 Admin láttamozott Forrás Egyéb (Nem besorolt )

31. Pál, Éva ; Hadjadj, Leila ; Monori-Kiss, Anna ; Horváth, Eszter ; Fontányi, Zoltán ; Horváth, Eszter Mária ; Monos, Emil ; Nádasy, György ; Benyó, Zoltán ; Várbíró, Szabolcs Androgén és D-vitamin jelátvitel közötti interakció hatása az agyi erek válaszkészségére Paper: P1.21 (2018) Teljes dokumentum Zárolt Közlemény:30520207 Admin láttamozott Forrás Egyéb (Nem besorolt )

32. Panta, Cecília Rita ; Ruisanchez, Éva ; Móré, Dorottya ; Dancs, Péter Tibor ; Balogh, Andrea ; Oermanns, Stefan ; Tigyi, Gábor ; Benyó, Zoltán Sustained potentiation of α1-adrenergic vasoconstriction by sphingosine-1-phosphate In: Recoop, HST Association - Recoop HST Association - Student Conference - Recoop- Frigyes Korányi Science Forum Budapest, Magyarország : Recoop HST Association, (2018) p. 20 Közlemény:30792891 Admin láttamozott Forrás Könyvrészlet (Absztrakt / Kivonat )

33. Panta, C.R. ; Ruisanchez, É. ; Móré, D. ; Dancs, P. ; Balogh, A. ; Oermanns, S. ; Tigyi, G. ; Benyó, Z. SUSTAINED POTENTIATION OF < ALPHA > 1-ADRENERGIC VASOCONSTRICTION BY SPHINGOSINE 1-PHOSPHATE JOURNAL OF HYPERTENSION 36 p. e291 (2018) DOI Egyéb URL Közlemény:30539192 Jóváhagyott Forrás Folyóiratcikk (Absztrakt / Kivonat )

34. Panta, C. Rita ; Ruisanchez, Éva ; Móré, Dorottya ; Dancs, Péter T. ; Balogh, Andrea ; Stefan, Oermanns ; Tigyi, Gábor ; Benyó, Zoltán A szngozin-1-foszfát potencírozó hatása az α1-adrenoreceptorok által közvetített vazokonstrikcióra Paper: E18 (2018) Teljes dokumentum Közlemény:30520569 Jóváhagyott Forrás Egyéb (Nem besorolt )

35. Szasz, O ; Vincze, G ; Szigeti, GP ; Benyo, Z ; Szasz, A An allometric approach of tumor-angiogenesis MEDICAL HYPOTHESES 116 pp. 74-78. , 5 p. (2018) DOI ISSN: 0306-9877 WoS PubMed Közlemény:3408577 Érvényesített Forrás Folyóiratcikk (Szakcikk )

36. Vancsik, T ; Kovago, C ; Kiss, E ; Papp, E ; Forika, G ; Benyo, Z ; Meggyeshazi, N** ; Krenacs, T** Modulated electro-hyperthermia induced loco-regional and systemic tumor destruction in colorectal cancer allografts JOURNAL OF CANCER 9 : 1 pp. 41-53. , 13 p. (2018) DOI PubMed SE Repozitórium WoS Scopus Közlemény:3322461 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 2 Független: 0 Függő: 2 Idézett közlemények száma: 4 1. * Vancsik Tamas et al. Modulated electro-hyperthermia induced p53 driven apoptosis and cell cycle arrest additively support doxorubicin chemotherapy of colorectal cancer in vitro.. (2019) CANCER MEDICINE 2045-7634 2019 p. In press 2. * Besztercei B. et al. Stress-induced, p53-mediated tumor growth inhibition of melanoma by modulated electrohyperthermia in mouse models without major immunogenic eects. (2019) INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 1661-6596 1422-0067 20

37. Vén, Krisztina ; Kerék, Margit ; Balogh, Andrea ; Tigyi, Gábor ; Benyó, Zoltán ; Ruisanchez, Éva Lysophosphatidic acid (LPA) mediated vasoconstriction depends on fatty acid chain unsaturation and changes with age p. 113 Paper: Poster 56 , 1 p. (2018) Teljes dokumentum Közlemény:30539514 Jóváhagyott Forrás Egyéb (Nem besorolt ) 38. Vén, Krisztina ; Kerék, Margit ; Balogh, Andrea ; Benyó, Zoltán ; Ruisanchez, Éva A lizofoszfatidsav okozta vazokonstrikció erősségének összefüggése a zsírsavlánc telítetlenségi fokával és az életkorral Paper: E21 (2018) Teljes dokumentum Közlemény:30520687 Jóváhagyott Forrás Egyéb (Nem besorolt )

39. Wafa, Dina ; Gaál, Benjámin ; Benyó, Zoltán ; Miklós, Zsuzsanna Endotél eredetű mediátorok és az LPA4 receptor szerepe a lizofoszfatidsav koronáriatónus szabályozó hatásának közvetítésében Paper: P1.17 (2018) Teljes dokumentum Közlemény:30520125 Jóváhagyott Forrás Egyéb (Nem besorolt )

40. Balogh, A ; Besztercei, B ; Benyó, Z ; Tigyi, G Az autotaxin expressziójának szabályozása tumorsejtvonalakban MAGYAR ONKOLÓGIA 61 : Suppl 1. pp. 57-57. , 1 p. (2017) Közlemény:3296878 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

41. Beliczai-Marosi, Gabriella ; Ane, Stensønes Dybvig ; Benyó, Zoltán ; Kiss, Levente A KÉNHIDROGÉN FOKOZOTT VAZORELAXÁCIÓT OKOZ IDŐS DB/DB EGEREKBEN Paper: P1.5.5 (2017) [Poszter], A Magyar Élettani Társaság, A Magyar Kisérletes és Klinikai Farmakológiai Társaság és a Magyar Mikrocirkulációs és Vaszkuláris Biológiai Társaság közös Vándorgyűlése, Debrecen, Egyéb URL Közlemény:30792737 Admin láttamozott Forrás Egyéb (Nem besorolt )

42. Besztercei, B ; Balogh, A ; Vancsik, T ; Major, E ; Krenács, T ; Benyó, Z Modulált elektro-hipertermia hatása az in vivo angiogenezisre MAGYAR ONKOLÓGIA 61 : Suppl 1. pp. 57-58. , 2 p. (2017) Közlemény:3296879 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

43. Dancs, P ; Ruisanchez, É ; Balogh, A ; Panta, CR ; Miklós, Zs ; Nüsing, RM ; Aoki, J ; Chun, J ; Oermanns, S ; Tigyi, G et al. LYSOPHOSPHATIDIC ACID, A JANUS-FACED MEDIATOR OF THE VASCULAR TONE , 1 p. (2017) 12. Sphingolipid Club (SCL) Meeting, Trabia, Olaszország, 2017. Szeptember 6-10., P14, Megjelenés: Olaszország, Egyéb URL Közlemény:3286498 Admin láttamozott Forrás Egyéb (Nem besorolt )

44. Dancs, P ; Ruisanchez, É ; Kerkovits, N ; Iring, A ; Oermanns, S ; Cervenak, L ; Gál, P ; Benyó, Z ENDOTHELIÁLIS PAR2 RECEPTOROK SZEREPE AZ ÉRTÓNUS SZABÁLYOZÁSÁBAN Paper: S13.6 , 1 p. (2017) [Előadás], [Magyar Élettani Társaság és a Magyar Kísérletes és Farmakológiai Társaság és a Magyar Mikrocirkulációs és Vaszkuláris Biológiai Társaság közös vándorgyűlése], [2017 június 13-16, Debrecen], Megjelenés: Magyarország, Teljes dokumentum Közlemény:3243367 Admin láttamozott Forrás Egyéb (Nem besorolt )

45. Dancs, PT ; Ruisanchez, E ; Balogh, A ; Panta, CR ; Miklos, Z ; Nusing, RM ; Aoki, J ; Chun, J ; Oermanns, S ; Tigyi, G et al. LPA1 receptor-mediated thromboxane A2 release is responsible for lysophosphatidic acid-induced vascular smooth muscle contraction FASEB JOURNAL 31 : 4 pp. 1547-1555. , 9 p. (2017) DOI PubMed WoS Scopus Közlemény:3165272 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 7 Független: 6 Függő: 1 Idézett közlemények száma: 8

1. Sakamoto K et al. Eect of ASP6432, A novel type 1 lysophosphatidic acid receptor antagonist, on urethral function and prostate cell proliferations. (2018) JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS 0022-3565 1521-0103 366 2 390-396 2. Chabowski Dawid et al. Lysophosphatidic acid acts on LPA(1) receptor to increase H2O2 during ow-induced dilation in human adipose arterioles. (2018) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 175 22 4266-4280 3. Jaimes Luisauris et al. A Review of the Actions of Endogenous and Exogenous Vasoactive Substances during the Estrous Cycle and Pregnancy in Rats. (2019) ANIMALS 2076-2615 9 6 4. Yang Liping et al. LPA receptor 4 deciency attenuates experimental atherosclerosis. (2019) JOURNAL OF LIPID RESEARCH 0022-2275 1539-7262 60 5 972-980 5. * Iring Andras et al. Shear stress-induced endothelial adrenomedullin signaling regulates vascular tone and blood pressure. (2019) JOURNAL OF CLINICAL INVESTIGATION 0021- 9738 1558-8238 129 7 2775-2791 6. Zhao Yang et al. Targeting the autotaxin - Lysophosphatidic acid receptor axis in cardiovascular diseases. (2019) BIOCHEMICAL PHARMACOLOGY 0006-2952 1873-2968 164 74-81 7. Lei Li et al. The role of lysophosphatidic acid in the physiology and pathology of the skin. (2019) LIFE SCIENCES 0024-3205 220 194-200

46. Forika, G ; Vancsik, T ; Kiss, E ; Hujber, Z ; Sebestyen, A ; Krencz, I ; Benyo, Z ; Hamar, P ; Krenacs, T Correlations between tumour metabolic proles and tumour destruction induced by modulated electro-hyperthermia VIRCHOWS ARCHIV 471 : Suppl. 1. pp. S249-S249. , 1 p. (2017) WoS Közlemény:3322465 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

47. Forika, G ; Kiss, É ; Vancsik, T ; Benyó, Z ; Krenács, T In vitro colorectalis carcinoma modell optimalizálása a modulált elektro-hipertermia által kiváltott tumorstressz és sejthalál tanulmányozásához MAGYAR ONKOLÓGIA 61 : Suppl 1. pp. 59-59. , 1 p. (2017) Közlemény:3296882 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

48. Hadjadj, L ; Várbíró, Sz ; Monori-Kiss, A ; Horváth, EM ; Pál, É ; Sziva, R ; Heinzlmann, A ; Magyar, A ; Nádasy, Gy L ; Benyó, Z Vitamin D deciency and androgen excess causes impaired testosterone and insulin induced relaxation in coronary arterioles of fertile female rats JOURNAL OF VASCULAR RESEARCH 54 : Suppl. 1 pp. 19-19. , 1 p. (2017) DOI WoS Zárolt Közlemény:3429994 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

49. Hadjadj, L ; Várbíró, Sz ; Monori-Kiss, A ; Eszter, M Horváth ; Éva, Pál ; Sziva, Réka ; Heinzlmann, Andrea ; Magyar, Attila ; Nádasy, György L ; Benyó, Zoltán Vitamin D deciency and androgen excess causes impaired testosterone and insulin induced relaxation in coronary arterioles of fertile female rats (2017) ESM-EVBO joint meeting 2017, 2017.05.29-06.01., Geneva, Switzerland, [Poszter], Megjelenés: Svájc, Zárolt Közlemény:3273708 Admin láttamozott Forrás Egyéb (Nem besorolt )

50. Hricisák, L ; Polycarpou, A ; Iring, A ; Safar, D ; Ruisanchez, É ; Horváth, B ; Sándor, P ; Benyó, Z A NITROGÉN-MONOXID SZEREPE A REGIONÁLIS AGYKÉRGI VÉRÁRAMLÁS SZABÁLYOZÁSÁBAN FÉLOLDALI ARTERIA CAROTIS ELZÁRÁS UTÁN Paper: P1.5.4 , 1 p. (2017) [Poszter], [Magyar Élettani Társaság és a Magyar Kísérletes és Farmakológiai Társaság és a Magyar Mikrocirkulációs és Vaszkuláris Biológiai Társaság közös vándorgyűlése], [2017 június 13-16, Debrecen], Megjelenés: Magyarország, Egyéb URL Közlemény:3243370 Admin láttamozott Forrás Egyéb (Nem besorolt )

51. Iring, A ; Hricisák, L ; Benyó, Z CB1 receptor-mediated respiratory depression by endocannabinoids RESPIRATORY PHYSIOLOGY AND NEUROBIOLOGY 240 pp. 48-52. , 5 p. (2017) DOI PubMed WoS Scopus Közlemény:3194182 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 2 Független: 2 Függő: 0 Idézett közlemények száma: 4 1. Wang Yuanqiang et al. An insight into and its metabolites using molecular docking and molecular dynamics simulation. (2018) JOURNAL OF MOLECULAR MODELING 1610- 2940 0948-5023 24 9 2. Wang X. et al. TASK1 and TASK3 are coexpressed with ASIC1 in the ventrolateral medulla and contribute to central chemoreception in rats. (2018) FRONTIERS IN CELLULAR NEUROSCIENCE 1662-5102 12

52. Kaucsár, T ; Danics, L ; Sárközy, H ; Schvarz, Cs ; Bellovits, K ; Balogh, A ; Vancsik, T ; Krenács, T ; Benyó, Z ; Hamar, P Adjuváns kezelés modulált elektro-hipertermiával in vivo egér emlőtumormodellen MAGYAR ONKOLÓGIA 61 : Suppl. 1 pp. 23-24. , 2 p. (2017) Közlemény:3296891 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

53. Miklós, Zs ; Wafa, D ; Hobot, E ; Ruisanchez, É ; Kerék, M ; Balogh, A ; Dancs, P ; Oermanns, S ; Tigyi, G ; Benyó, Z LYSOPHOSPHATIDIC ACID INDUCED VASOCONSTRICTION IN THE CORONARY VASCULAR BED , 1 p. (2017) 12. Sphingolipid Club (SCL) Meeting, Trabia, Olaszország, 2017. Szeptember 6-10., P15, Megjelenés: Olaszország, Egyéb URL Közlemény:3286499 Admin láttamozott Forrás Egyéb (Nem besorolt )

54. Molnár, E ; Molnár, B ; Lohinai, Z ; Tóth, Z ; Benyó, Z ; Hricisák, L ; Windisch, P ; Vág, J Evaluation of Laser Speckle Contrast Imaging for the Assessment of Oral Mucosal Blood Flow following Periodontal Plastic Surgery: An Exploratory Study BIOMED RESEARCH INTERNATIONAL 2017 Paper: 4042902 , 11 p. (2017) DOI PubMed SE Repozitórium WoS Scopus Közlemény:3178732 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 11 Független: 3 Függő: 8 Idézett közlemények száma: 2

1. * Fazekas R et al. A proposed method for assessing the appropriate timing of early implant placements: a case report. (2018) JOURNAL OF ORAL IMPLANTOLOGY 0160-6972 1548-1336 44 5 378-383 2. * Molnar E et al. Assessment of the test-retest reliability of human gingival blood ow measurements by Laser Speckle Contrast Imaging in a healthy cohort.. (2018) MICROCIRCULATION 1073- 9688 1549-8719 25 2 3. Tatarakis Nikolaos et al. Blood ow changes using a 3D xenogeneic collagen matrix or a subepithelial connective tissue graft for root coverage procedures: a pilot study. (2018) CLINICAL ORAL INVESTIGATIONS 1432-6981 1436-3771 22 4 1697-1705 4. Ansson C.D. et al. Blood perfusion in Hewes tarsoconjunctival aps in pigs measured by laser speckle contrast imaging. (2018) JPRAS Open 2352-5878 18 98-103 5. * Fazekas Réka et al. Functional characterization of collaterals in the human gingiva by laser speckle contrast imaging. (2018) MICROCIRCULATION 1073-9688 1549-8719 25 3 6. * Shahbazi A et al. Analysis of blood supply in the hard palate and maxillary tuberosity-clinical implications for ap design and soft tissue graft harvesting (a human cadaver study). (2019) CLINICAL ORAL INVESTIGATIONS 1432-6981 1436-3771 23 3 1153-1160 7. * Fazekas Reka et al. A Novel Approach to Monitoring Graft Neovascularization in the Human Gingiva.. (2019) JOVE-JOURNAL OF VISUALIZED EXPERIMENTS 1940-087X 143 8. * Molnar Balint et al. Assessment of Palatal Mucosal Wound Healing Following Connective-Tissue Harvesting by Laser Speckle Contrast Imaging: An Observational Case Series Study. (2019) INTERNATIONAL JOURNAL OF PERIODONTICS & RESTORATIVE DENTISTRY 0198-7569 1945-3388 39 2 E64-E70 9. * Fazekas Réka et al. Blood ow kinetics of a xenogeneic collagen matrix following a vestibuloplasty procedure in the human gingiva-An explorative study.. (2019) ORAL DISEASES 1354-523X 1601-0825 25 7 1780-1788 10. * Gánti Bernadett et al. Evidence of spreading vasodilation in the human gingiva evoked by nitric oxide.. (2019) JOURNAL OF PERIODONTAL RESEARCH 0022-3484 1600-0765 54 5 499-505 11. Tenland Kajsa et al. Perfusion Monitoring Shows Minimal Blood Flow From the Flap Pedicle to the Tarsoconjunctival Flap. (2019) OPHTHALMIC PLASTIC AND RECONSTRUCTIVE SURGERY 0740- 9303 35 4 346-349

55. Pál, E ; Fontányi, Z ; Hadjadj, L ; Monori-Kiss, A ; Danics, L ; Monos, E ; Nádasy, G ; Benyó, Z ; Várbíró, S Vitamin D Deciency Impairs Geometrical Structure and Physiological Function of Cerebral Arteries In: Duric, Dragan; Jakovljevic, Vladimir (szerk.) Joint Meeting of National Physiological Societies "New Perspectives in Physiological Research - Young Investigator Forum" : Final Program & Abstract Book Beograd, Szerbia : Društvo ziologa Srbije, (2017) pp. 115-115. , 1 p. Zárolt Közlemény:3238968 Admin láttamozott Forrás Könyvrészlet (Absztrakt / Kivonat )

56. Pál, É ; Fontányi, Z ; Hadjadj, L ; Monori-Kiss, A ; Monos, E ; Nádasy, G ; Benyó, Z ; Várbíró, Sz A D-vitamin hiány befolyásolja az arteria cerebri morfológiai jellemzőit és károsítja az érfunkciót (2017) Magyar Élettani Társaság és a Magyar Kísérletes és Farmakológiai Társaság és a Magyar Mikrocirkulációs és Vaszkuláris Biológiai Társaság közös vándorgyűlése, Debrecen, 2017.06 13-2017.06.16, [Előadás], Megjelenés: Magyarország, Zárolt Közlemény:3429971 Admin láttamozott Forrás Egyéb (Nem besorolt )

57. Pál, É ; Fontányi, Z ; Hadjadj, L ; Monori-Kiss, A ; Danics, L ; Monos, E ; Nádasy, G ; Benyó, Z ; Várbíró, Sz Vitamin D deciency impairs geometrical structure and function of cerebral arteries ACTA PHYSIOLOGICA 221 : S713 p. 78 Paper: A02-10 (2017) DOI WoS Zárolt Közlemény:3287932 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

58. Panta, CR ; Móré, D ; Ruisanchez, É ; Dancs, P ; Kerék, M ; Oermanns, S ; Benyó, Z SPHINGOSINE-1-PHOSPHATE ENHANCES α1-ADRENORECEPTOR-MEDIATED VASOCONSTRICTION , 1 p. (2017) 12. Sphingolipid Club (SCL) Meeting, Trabia, Olaszország, 2017. Szeptember 6-10., P11, Megjelenés: Olaszország, Egyéb URL Közlemény:3286497 Admin láttamozott Forrás Egyéb (Nem besorolt )

59. Ruisanchez, E ; Juhász, T ; Harmat, V ; Erdősi, E ; Benyó, Z ; Liliom, K SPHINGOSINE INHIBITS CALMODULIN ACTION: IMPLICATION FOR REGULATION OF ENDOTHELIAL NITRIC OXIDE SYNTHASE-MEDIATED VASORELAXATION , 1 p. (2017) 12. Sphingolipid Club (SCL) Meeting, Trabia, Olaszország, 2017. Szeptember 6-10., P16, Megjelenés: Olaszország, Közlemény:3286500 Admin láttamozott Forrás Egyéb (Nem besorolt )

60. Ruisanchez, É ; Kiss, L ; Strakya, É Zs ; Korda, D ; Párkányi, A ; Liliom, K ; Tigyi, G ; Benyó, Z ENHANCED ENDOTHELIAL NITRIC OXIDE SYNTHASE MEDIATED VASORELAXATION BY SPHINGOMYELINASE IN DB/DB MICE , 1 p. (2017) 12. Sphingolipid Club (SCL) Meeting, Trabia, Olaszország, 2017. Szeptember 6-10., P17, Megjelenés: Olaszország, Közlemény:3286502 Admin láttamozott Forrás Egyéb (Nem besorolt )

61. Vancsik, T ; Major, E ; Kiss, É ; Forika, G ; Krenács, T ; Benyó, Z ; Balogh, A A modulált elektro-hipertermia hatásának vizsgálata C26 colorectalis és B16F10 melanoma tumorsejtvonalakon in vitro MAGYAR ONKOLÓGIA 61 : Suppl 1. pp. 76-76. , 1 p. (2017) Teljes dokumentum Közlemény:3296894 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

62. Vancsik, T ; Kővágó, Cs ; Kiss, É ; Forika, G ; Meggyesházi, N ; Benyó, Z ; Krenács, T A modulált elektro-hipertermia indukálta proliferációgátlás, sejthalál és immunválasz in vivo C26 colorectalis carcinoma modellben MAGYAR ONKOLÓGIA 61 : Suppl 1. pp. 48-48. , 1 p. (2017) Közlemény:3296893 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat ) 63. Wafa, D ; Hobot, E ; Ruisanchez, É ; Kerék, M ; Benyó, Z ; Miklós, Zs A LIZOFOSZFATIDSAV KORONÁRIA SZŰKÍTŐ HATÁSÁT KÖZVETÍTŐ JELÁTVITELI ÚTVONALAK AZONOSÍTÁSA Paper: P2.5.2 , 1 p. (2017) [Poszter], [Magyar Élettani Társaság és a Magyar Kísérletes és Farmakológiai Társaság és a Magyar Mikrocirkulációs és Vaszkuláris Biológiai Társaság közös vándorgyűlése], [2017 június 13-16, Debrecen], Megjelenés: Magyarország, Teljes dokumentum Közlemény:3243379 Admin láttamozott Forrás Egyéb (Nem besorolt )

64. Benyo, Z ; Ruisanchez, E ; Leszl-Ishiguro, M ; Sandor, P ; Pacher, P Endocannabinoids in Cerebrovascular Regulation AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 310 : 7 pp. H785-H801. (2016) DOI WoS Scopus PubMed Közlemény:3011598 Érvényesített Forrás Idéző Folyóiratcikk (Összefoglaló cikk ) Nyilvános idézők összesen: 26 Független: 22 Függő: 4 Idézett közlemények száma: 49 1. * Polycarpou A et al. Adaptation of the Cerebrocortical Circulation to Carotid Artery Occlusion Involves Blood Flow Redistribution between Cortical Regions and is Independent of eNOS.. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 311 4 H972-H980 2. Janero DR et al. Leveraging allostery to improve G protein- coupled receptor (GPCR)-directed therapeutics: 1 as discovery target. (2016) EXPERT OPINION ON DRUG DISCOVERY 1746-0441 1746-045X 11 12 1223-1237 3. Franks Lirit et al. Selective Estrogen Receptor Modulators: Cannabinoid Receptor Inverse Agonists with Dierential CB1 and CB2 Selectivity. (2016) FRONTIERS IN PHARMACOLOGY 1663-9812 7 4. Jayant Shalini et al. Selective Modulator of Cannabinoid Receptor Type 2 Reduces Memory Impairment and Infarct Size During Cerebral Hypoperfusion and Vascular Dementia. (2016) CURRENT NEUROVASCULAR RESEARCH 1567-2026 13 4 289-302 5. Sun Hai-Jing et al. Activation of Endocannabinoid Receptor 2 as a Mechanism of Propofol Pretreatment-Induced Cardioprotection against Ischemia-Reperfusion Injury in Rats. (2017) OXIDATIVE MEDICINE AND CELLULAR LONGEVITY 1942-0900 1942-0994 6. Li Xin et al. A Pulsed Electromagnetic Field Protects against Glutamate-Induced Excitotoxicity by Modulating the in HT22 Cells. (2017) FRONTIERS IN NEUROSCIENCE 1662-4548 1662-453X 11 7. Richards John. Cannabinoid hyperemesis syndrome: A disorder of the HPA axis and sympathetic nervous system?. (2017) MEDICAL HYPOTHESES 0306-9877 1532-2777 103 90-95 8. Ho W.S.V. et al. in the Cardiovascular System. (2017) 80 329-366 9. * Iring A et al. CB1 receptor-mediated respiratory depression by endocannabinoids. (2017) RESPIRATORY PHYSIOLOGY AND NEUROBIOLOGY 1569-9048 1878-1519 240 48-52 10. Chen Jun-Li et al. Ferulic acid attenuates brain microvascular endothelial cells damage caused by oxygen-glucose deprivation via punctate-mitochondria-dependent mitophagy. (2017) BRAIN RESEARCH 0006-8993 1872-6240 1666 17-26 11. Wang Da-Peng. Inhibition of SENP3 by URB597 ameliorates neurovascular unit dysfunction in rats with chronic cerebral hypoperfusion. (2017) BIOMEDICINE & PHARMACOTHERAPY 0753-3322 1950-6007 91 872-879 12. Seleverstov Olga et al. Maternal alcohol exposure during mid- pregnancy dilates fetal cerebral arteries via endocannabinoid receptors. (2017) ALCOHOL 0741-8329 61 51-61 13. Maccarrone Mauro. Metabolism of the Endocannabinoid : Open Questions after 25 Years. (2017) FRONTIERS IN MOLECULAR NEUROSCIENCE 1662-5099 1662-5099 10 14. Wang D et al. URB597 improves cognitive impairment induced by chronic cerebral hypoperfusion by inhibiting mTOR-dependent autophagy. (2017) NEUROSCIENCE 0306-4522 1873-7544 344 293- 304 15. Richter J et al. A Systematic Review of the Complex Eects of Cannabinoids on Cerebral and Peripheral Circulation in Animal Models. (2018) FRONTIERS IN PHYSIOLOGY 1664-042X 9 16. * Pacher P et al. Cardiovascular eects of marijuana and : the good, the bad, and the ugly. (2018) NATURE REVIEWS CARDIOLOGY 1759-5002 1759-5010 15 3 151-166 17. Carswell Hilary et al. Detrimental eects of 2-arachidonoylglycerol on whole blood platelet aggregation and on cerebral blood ow after a focal ischemic insult in rats. (2018) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 314 5 H967-H977 18. Galiazzo Giorgia et al. Localization of cannabinoid receptors CB1, CB2, GPR55, and PPAR alpha in the canine gastrointestinal tract. (2018) HISTOCHEMISTRY AND CELL BIOLOGY 0948-6143 1432- 119X 150 2 187-205 19. * Pacher Pal et al. Neuroprotection in oxidative stress-related neurodegenerative diseases: role of endocannabinoid system modulation. (2018) ANTIOXIDANTS & REDOX SIGNALING 1523- 0864 1557-7716 29 1 75-108 20. Choi Sang-Ho et al. Neuroprotective Eects of MAGL () Inhibitors in Experimental Ischemic Stroke. (2018) STROKE 0039-2499 1524-4628 49 3 718-726 21. Gao Xiaoling et al. Role of the endogenous cannabinoid receptor 1 in brain injury induced by chronic intermittent hypoxia in rats. (2018) INTERNATIONAL JOURNAL OF NEUROSCIENCE 0020-7454 1563-5279 128 9 797-804 22. Pressly J. et al. Activation of the cannabinoid receptor 2 increases renal perfusion. (2019) PHYSIOLOGICAL GENOMICS 1094-8341 1531-2267 51 3 90-96 23. Li Taoxi et al. A novel ABHD12 nonsense variant in Usher syndrome type 3 family with genotype-phenotype spectrum review. (2019) GENE 0378-1119 1879-0038 704 113-120 24. Hai Kerong et al. Monoacylglycerol Lipase Inactivation by Using URB602 Mitigates Myocardial Damage in a Rat Model of Cardiac Arrest. (2019) CRITICAL CARE MEDICINE 0090-3493 1530-0293 47 2 E144-E151 25. Echeverria-Villalobos M. et al. Perioperative care of users: A comprehensive review of pharmacological and anesthetic considerations. (2019) JOURNAL OF CLINICAL ANESTHESIA 0952-8180 57 41-49 26. Ugradar Shoaib et al. Unilateral cilioretinal artery occlusion following cannabis use. (2019) THERAPEUTIC ADVANCES IN OPHTHALMOLOGY 2515-8414 11

65. Dancs, P ; Ruisanchez, É ; Balogh, A ; Panta, C. R ; Miklós, Zs ; Nüsing, R ; Aoki, J ; Chun, J ; Oermanns, S ; Tigyi, G et al. Signaling pathways of lysophosphatidic acid elicited vasoconstriction (2016) [Poszter], 6th European Workshop on Lipid Mediators, Frankfurt, Egyéb URL Közlemény:30792744 Jóváhagyott Forrás Egyéb (Nem besorolt )

66. Hadjadj, L ; Horváth, EM ; Monori-Kiss, A ; Nádasy, GyL ; Benyó, Z ; Várbíró, Sz Insulin resistance of coronary arteries in a rat PCOS model GYNECOLOGICAL ENDOCRINOLOGY 32 : Suppl. 1 pp. 83-84. , 2 p. (2016) DOI WoS Zárolt Közlemény:3085390 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat ) 67. Hadjadj, L ; Monori-Kiss, A ; Nádasy, GyL ; Pál, É ; Benyó, Z ; Várbíró, Sz Biomechanical properties and structural changies of coronary arterioles in chronic rat PCOS model GYNECOLOGICAL ENDOCRINOLOGY 32 : Suppl. 1 p. 83 (2016) DOI WoS Zárolt Közlemény:3085389 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

68. Molnar, E ; Molnar, B ; Lohinai, Z ; Toth, Z ; Benyo, Z ; Hricisak, L ; Windisch, P ; Mikecs, B ; Vag, J Postoperative Microcirculatory Changes In Human Gingiva After Tunnelling Technique JOURNAL OF DENTAL RESEARCH 95 : C Paper: 0075 (2016) Teljes dokumentum Közlemény:3328055 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

69. Pál, É ; Hadjadj, L ; Monori-Kiss, A ; Fontányi, Z ; Nádasy, GyL ; Benyó, Z ; Monos, E ; Várbíró, Sz A D-VITAMIN MEGVONÁS RONTJA A KORONÁRIA ARTERIOLÁK BIOMECHANIKAI JELLEMZŐIT HÍM PATKÁNYOKBAN (2016) FAMÉ konferencia, Pécs, 2016. 06. 1-4, [Előadás absztrakt], Megjelenés: Magyarország, Teljes dokumentum Zárolt Közlemény:3085403 Admin láttamozott Forrás Egyéb (Nem besorolt )

70. Panta, C. Rita ; Móré, D ; Dancs, P ; Ruisanchez, É ; Kerék, M ; Oermanns, S ; Benyó, Z Sphingosine-1-phosphate modulates alpha1-adrenoreceptor-mediated vasoconstriction (2016) [Poszter], 6th European Workshop on Lipid Mediators, Frankfurt, Egyéb URL Közlemény:30792742 Jóváhagyott Forrás Egyéb (Nem besorolt )

71. Polycarpou, A ; Hricisak, L ; Iring, A ; Safar, D ; Ruisanchez, E ; Horvath, B ; Sandor, P ; Benyo, Z Adaptation of the Cerebrocortical Circulation to Carotid Artery Occlusion Involves Blood Flow Redistribution between Cortical Regions and is Independent of eNOS. AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 311 : 4 pp. H972-H980. (2016) DOI SE Repozitórium WoS Scopus PubMed Közlemény:3100748 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 1 Független: 1 Függő: 0 Idézett közlemények száma: 6

1. Wilde Elena et al. Tail-Cu Technique and Its Inuence on Central Blood Pressure in the Mouse. (2017) JOURNAL OF THE AMERICAN HEART ASSOCIATION 2047-9980 2047-9980 6 6

72. Sándor, Péter ; Benyó, Zoltán Az agyi vérkeringés élettani alapjai: Önszabályozó mechanizmusok Budapest, Magyarország : Semmelweis Kiadó és Multimédia Stúdió (2016) , 136 p. ISBN: 9789633313756 Egyéb URL Közlemény:3013894 Jóváhagyott Forrás Idéző Könyv (Szakkönyv ) Idézett közlemények száma: 1

73. Varbiro, S ; Hadjadj, L ; Acs, N ; Horvath, E ; Heinzlmann, A ; Magyar, A ; Tokes, AM ; Patocs, A ; Buday, A ; Fekete, A et al. Investigating the linkage between vitamin D and PCOS: development of new rat model GYNECOLOGICAL ENDOCRINOLOGY 32 : Suppl. 1 pp. 79-79. , 1 p. (2016) DOI WoS Zárolt Közlemény:3064983 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

74. Benyó, Zoltán ; Ruisanchez, Éva ; Iring, András ; Leszl-Ishiguro, Miriam ; Sándor, Péter ; Pacher, Pál Endocannabinoidok szerepe az agyi vérkeringés szabályozásában Paper: S8.6 (2015) [Előadás absztrakt], A MAGYAR ÉLETTANI TÁRSASÁG 79. VÁNDORGYŰLÉSE ÉS A MAGYAR MIKROCIRKULÁCIÓS ÉS VASZKULÁRIS BIOLÓGIAI TÁRSASÁG 2015. ÉVI KONFERENCIÁJA, Teljes dokumentum Közlemény:30792746 Jóváhagyott Forrás Egyéb (Nem besorolt ) 75. Dybvig, AS ; Marosi, G ; Straky, Z ; Korda, D ; Benyo, Z ; Ruisanchez, E ; Kiss, L The role of hydrogen sulde in enhanced sphingomyelinase induced vasorelaxation in db/db mice NITRIC OXIDE-BIOLOGY AND CHEMISTRY 47 : Suppl 1 pp. S26-S27. , 2 p. (2015) DOI WoS Közlemény:2956027 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

76. Hobot, Eszter ; Wafa, Dina ; Ruisanchez, Éva ; Kerék, Margit ; Ivanics, Tamás ; Benyó, Zoltán ; Miklós, Zsuzsanna A lizofoszfatidsav erős vazokonstriktor a koronáriaerekben Paper: P6.5 (2015) [Előadás absztrakt], A MAGYAR ÉLETTANI TÁRSASÁG 79. VÁNDORGYŰLÉSE ÉS A MAGYAR MIKROCIRKULÁCIÓS ÉS VASZKULÁRIS BIOLÓGIAI TÁRSASÁG 2015. ÉVI KONFERENCIÁJA, Szeged, Teljes dokumentum Közlemény:30792757 Jóváhagyott Forrás Egyéb (Nem besorolt )

77. Marosi, Gabriella ; Dybvig, Ane Stensones ; Ruisanchez, Éva ; Benyó, Zoltán ; Kiss, Levente A kénhidrogén fokozott relaxációt okoz 2-es típusú diabéteszes egerek aortáiban Paper: E2.3 (2015) [Előadás absztrakt], A MAGYAR ÉLETTANI TÁRSASÁG 79. VÁNDORGYŰLÉSE ÉS A MAGYAR MIKROCIRKULÁCIÓS ÉS VASZKULÁRIS BIOLÓGIAI TÁRSASÁG 2015. ÉVI KONFERENCIÁJA, Szeged, Teljes dokumentum Közlemény:30792750 Jóváhagyott Forrás Egyéb (Nem besorolt )

78. Móré, Dorottya ; Panta, Rita ; Ruisanchez, Éva ; Stefan, Oermanns ; Benyó, Zoltán A szngozin-1-foszfát jelátviteli útvonalai a simaizom-kontrakcióban Paper: E4.5 (2015) [Előadás absztrakt], A MAGYAR ÉLETTANI TÁRSASÁG 79. VÁNDORGYŰLÉSE ÉS A MAGYAR MIKROCIRKULÁCIÓS ÉS VASZKULÁRIS BIOLÓGIAI TÁRSASÁG 2015. ÉVI KONFERENCIÁJA, Szeged, Teljes dokumentum Közlemény:30792762 Jóváhagyott Forrás Egyéb (Nem besorolt )

79. Szekeres, M ; Nadasy, GL ; Turu, G ; Soltesz-Katona, E ; Benyo, Z ; Oermanns, S ; Ruisanchez, E ; Szabo, E ; Takats, Z ; Batkai, S et al. Endocannabinoid-mediated modulation of G protein-coupled receptor signaling-induced vasoconstriction and hypertension. MOLECULAR AND CELLULAR ENDOCRINOLOGY 403 pp. 46-56. , 11 p. (2015) DOI REAL SE Repozitórium WoS Scopus PubMed Közlemény:2833239 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 17 Független: 14 Függő: 3 Idézett közlemények száma: 18

1. Matthews Anberitha. The role of endocannabinoids in atherosclerosis. (2015) 2. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801 3. Matthews Anberitha et al. Oxyradical stress increases the biosynthesis of 2-arachidonoylglycerol: involvement of NADPH oxidase. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: CELL PHYSIOLOGY 0363-6143 1522-1563 311 6 C960-C974 4. Baranowska-Kuczko Marta et al. Protective role of cannabinoid CB1 receptors and vascular eects of chronic administration of FAAH inhibitor URB597 in DOCA-salt hypertensive rats. (2016) LIFE SCIENCES 0024-3205 151 288-299 5. Willis Monte et al. The alpha-1A adrenergic receptor agonist A61603 reduces cardiac polyunsaturated fatty acid and endocannabinoid metabolites associated with inammation in vivo. (2016) METABOLOMICS 1573-3882 1573-3890 12 10 6. Karpinska Olga et al. Activation of CB1 receptors by 2- arachidonoylglycerol attenuates vasoconstriction induced by U46619 and angiotensin II in human and rat pulmonary arteries. (2017) AMERICAN JOURNAL OF PHYSIOLOGY: REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY 0363-6119 1522- 1490 312 6 R883-R893 7. Ho WSV et al. Cannabinoids in the Cardiovascular System. (2017) Megjelent: ADVANCES IN PHARMACOLOGY pp. 329-366 8. Victor Lamin. Molecular Mechanisms of Sex-Dierences in Vascular Reactivity of Human Internal Mammary Artery. (2017) 9. Aizpurua-Olaizola Oier et al. Targeting the endocannabinoid system: future therapeutic strategies. (2017) DRUG DISCOVERY TODAY 1359-6446 1878-5832 22 1 105-110 10. Yohn S.E. et al. Activation of the mGlu1 metabotropic glutamate receptor has antipsychotic-like eects and is required for ecacy of M4 muscarinic receptor allosteric modulators. (2018) MOLECULAR PSYCHIATRY 1359-4184 1476-5578 11. * Szekeres M et al. Control of myogenic tone and agonist induced contraction of intramural coronary resistance arterioles by cannabinoid type 1 receptors and endocannabinoids.. (2018) PROSTAGLANDINS & OTHER LIPID MEDIATORS 1098-8823 2212- 196X 134 77-83 12. Singh P. et al. Endocannabinoid-mediated modulation of Gq protein-coupled receptor mediates vascular hyporeactivity to nor- adrenaline during polymicrobial sepsis. (2018) POLISH JOURNAL OF PHARMACOLOGY 1230-6002 1734-1140 70 6 1150-1157 13. Karpinska Olga et al. Endocannabinoids modulate G(q/11) protein-coupled receptor agonist-induced vasoconstriction via a negative feedback mechanism. (2018) JOURNAL OF PHARMACY AND PHARMACOLOGY 0022-3573 2042-7158 70 2 214-222 14. * Tóth AD et al. Novel mechanisms of G-protein-coupled receptors functions: AT1 angiotensin receptor acts as a signaling hub and focal point of receptor cross-talk. (2018) BEST PRACTICE & RESEARCH CLINICAL ENDOCRINOLOGY & METABOLISM 1521- 690X 32 2 69-82 15. Berrendero Fernando et al. When orexins meet cannabinoids: Bidirectional functional interactions. (2018) BIOCHEMICAL PHARMACOLOGY 0006-2952 1873-2968 157 43-50 16. Bondarenko A.I.. Cannabinoids and Cardiovascular System. (2019) Megjelent: Advances in Experimental Medicine and Biology pp. 63-87 17. Echeverria-Villalobos M. et al. Perioperative care of cannabis users: A comprehensive review of pharmacological and anesthetic considerations. (2019) JOURNAL OF CLINICAL ANESTHESIA 0952-8180 57 41-49

80. Farago, B ; Dér, B ; Ruisanchez, É ; Őrsy, P ; Oermanns, S ; Benyó, Z Neurokinin A induced contraction of the urinary bladder smooth muscle ACTA PHYSIOLOGICA 211 : Suppl. s697 pp. 76-77. Paper: P3.3 , 2 p. (2014) DOI WoS Közlemény:2797226 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

81. Kiss, L ; Straky, Zs ; Korda, D ; Parkanyi, A ; Ruisanchez, É ; Benyo, Z Sphingomyelinase induces nitric oxide and hydrogen sulde dependent vasorelaxation in db/db mice CARDIOVASCULAR RESEARCH 103 : Suppl. 1 Paper: P186 (2014) DOI WoS Közlemény:2797271 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

82. Masszi, G ; Benko, R ; Csibi, N ; Horvath, EM ; Tokes, A-M ; Novak, A ; Beres, NJ ; Tarszabo, R ; Buday, A ; Repas, C et al. Erratum: Endothelial relaxation mechanisms and nitrative stress are partly restored by vitamin D3 therapy in a rat model of polycystic ovary syndrome (Life Sciences (2013) 93 (133-138)) LIFE SCIENCES 100 : 1 p. 73 (2014) DOI SE Repozitórium WoS Scopus Zárolt Közlemény:2580798 Érvényesített Forrás Idéző Folyóiratcikk (Hozzászólás, helyreigazítás ) Idézett közlemények száma: 1 83. Moré, D ; Ruisanchez, É ; Dancs, P ; Kerék, M ; Oermanns, S ; Benyó, Z Sphingosine-1-phosphate enhances the contractile responsiveness of vascular smooth muscle via distinct S1P2 receptor mediated pathways ACTA PHYSIOLOGICA 211 : Suppl. s697 p. 169 Paper: P12.9 (2014) DOI WoS Közlemény:2797257 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

84. Németh, T ; Ruisanchez, É ; Hricisák, L ; Iring, A ; Merkely, B ; Hunyady, L ; Smrcka, AV ; Oermanns, S ; Benyó, Z Signaling pathways of thromboxane receptor-mediated vasoconstriction: Major role of phospholipase C epsilon ACTA PHYSIOLOGICA 211 : Suppl. s697 pp. 37-38. Paper: S4-B5 , 2 p. (2014) DOI WoS Közlemény:2797263 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

85. Ruisanchez, E ; Dancs, P ; Kerek, M ; Nemeth, T ; Farago, B ; Balogh, A ; Patil, R ; Jennings, BL ; Liliom, K ; Malik, KU et al. Lysophosphatidic acid induces vasodilation mediated by LPA1 receptors, phospholipase C, and endothelial nitric oxide synthase. FASEB JOURNAL 28 : 2 pp. 880-890. , 11 p. (2014) DOI PubMed REAL SE Repozitórium WoS Scopus Közlemény:2470225 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 12 Független: 11 Függő: 1 Idézett közlemények száma: 8

1. Zhao C. Association between endothelial nitric oxide synthase 894G>T polymorphism and prostate cancer risk: a meta-analysis of literature studies. (2014) TUMOR BIOLOGY 1010-4283 1423- 0380 35 12 11727-11733 2. Yung YC et al. LPA receptor signaling: Pharmacology, physiology, and pathophysiology. (2014) JOURNAL OF LIPID RESEARCH 0022- 2275 1539-7262 55 7 1192-1214 3. Binder Bernard et al. Lysophosphatidic Acid and Sphingosine-1- Phosphate: A Concise Review of Biological Function and Applications for Tissue Engineering. (2015) TISSUE ENGINEERING PART B REVIEWS 1937-3368 1937-3376 21 6 531-542 4. Yung Yun et al. Lysophosphatidic Acid Signaling in the Nervous System. (2015) NEURON 0896-6273 85 4 669-682 5. Stoddard Nicole et al. Promising Pharmacological Directions in the World of Lysophosphatidic Acid Signaling. (2015) BIOMOLECULES & THERAPEUTICS 1976-9148 2005-4483 23 1 1-11 6. Chen X et al. Deciency or inhibition of lysophosphatidic acid receptor 1 protects against hyperoxia-induced lung injury in neonatal rats. (2016) ACTA PHYSIOLOGICA 1748-1708 1748-1716 216 3 358-375 7. de Oliveira-Lima Katia. Microcirculation abnormalities provoked by Loxosceles spiders' envenomation. (2016) TOXICON 0041-0101 116 Spec. iss. 35-42 8. * Dancs PT et al. LPA1 receptor-mediated thromboxane A2 release is responsible for lysophosphatidic acid-induced vascular smooth muscle contraction. (2017) FASEB JOURNAL 0892-6638 1530-6860 31 4 1547-1555 9. Ramesh S et al. Autotaxin–lysophosphatidic acid signaling in alzheimer’s disease. (2018) INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 1661-6596 1422-0067 19 7 10. Chabowski Dawid et al. Lysophosphatidic acid acts on LPA(1) receptor to increase H2O2 during ow-induced dilation in human adipose arterioles. (2018) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 175 22 4266-4280 11. Wang Rong et al. Expression prole analysis for epithelial- mesenchymal transition of breast cancer cell line DKTA based on microarray data. (2019) EUROPEAN JOURNAL OF GYNAECOLOGICAL ONCOLOGY 0392-2936 40 4 579-584 12. Zhao Yang et al. Targeting the autotaxin - Lysophosphatidic acid receptor axis in cardiovascular diseases. (2019) BIOCHEMICAL PHARMACOLOGY 0006-2952 1873-2968 164 74-81

86. Ruisanchez, É ; Dancs, P ; Kerék, M ; Németh, T ; Faragó, B ; Panta, R ; Balogh, A ; Tigyi, G ; Benyó, Z Vasoactive actions of lysophosphatidic acid ACTA PHYSIOLOGICA 211 : Suppl. s697 p. 88 Paper: P4.14 (2014) DOI WoS Közlemény:2797241 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

87. Sára, Levente ; Antal, Péter ; Masszi, Gabriella ; Tarszabó, Róbert ; Novák, Ágnes ; Buday, Anna ; Horváth, M Eszter ; Hamar, Péter ; Benkő, Rita ; Monos, Emil et al. Vascular Insulin Resistance of a Rat Model of Polycystic Ovary Syndrome: Eects of parallel vitamin D3 treatment ENDOCRINOLOGY AND METABOLIC SYNDROME 3 : 2 p. 77 (2014) Egyéb URL Zárolt Közlemény:2760453 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

88. Sára, Levente ; Antal, Péter ; Masszi, Gabriella ; Benkő, Rita ; Monos, Emil ; Benyó, Zoltán ; Nádasy, L. György ; Várbíró, Szabolcs Prehypertensive Alteration of Arteriolar Reactivity in a Rat PCOS model - Eects of Parallel Vitamin D3 Administration (2014) ESH - Summer School, Oral Presentation, Varna, Bulgaria, 2014.09.20-26., Zárolt Közlemény:2748531 Admin láttamozott Forrás Egyéb (Nem besorolt )

89. Sára, Levente ; Antal, Péter ; Masszi, Gabriella ; Horváth, Eszter ; Monori-Kiss, Anna ; Nádasy, György L ; Monos, Emil ; Benyó, Zoltán ; Várbíró, Szabolcs A kis- és nagyerek reaktivitás változása patkány policisztás petefészek modellben - A párhuzamos D3-vitamin kezelés hatásai MAGYAR NŐORVOSOK LAPJA 77 : különszám pp. 80-80. , 1 p. (2014) Zárolt Közlemény:2711310 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

90. Straky, Zs ; Korda, D ; Párkányi, A ; Ruisanchez, É ; BenyóZ ; Kiss, L Sphingomyelinase induced vasorelaxations in db/db mice depend on nitric oxide and hydrogen sulde signaling ACTA PHYSIOLOGICA 211 : Suppl. s697 p. 102 Paper: P4.47 (2014) DOI WoS Közlemény:2797253 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

91. Szekeres, M ; Nádasy, Gy ; Soltész-Katona, E ; Benyó, Z ; Tóth, Zs E ; Hunyady, L Endocannabinoid-mediated modulation of GPCR signaling-induced vasoconstriction and hypertension ACTA PHYSIOLOGICA 211 : Supplement s697 Paper: P2.15 (2014) DOI WoS Közlemény:2795110 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

92. Buday, A ; Orsy, P ; Godó, M ; Mózes, M ; Kökény, G ; Lacza, Z ; Koller, A ; Ungvári, Z ; Gross, ML ; Benyó, Z et al. Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) mice In: Semmelweis Symposium 2013 (2013) p. 25 Közlemény:2879428 Admin láttamozott Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat )

93. Dancs, P ; Ruisanchez, É ; Nagy, Z ; Renukadevi, P ; Németh, T ; Liliom, K ; Tigyi, G ; Benyó, Z A lizofoszfatidsav endothelium-függő vazorelaxáns hatása In: Magyar, Élettani Társaság - A Magyar Élettani, Farmakológiai és Mikrocirkulációs Társaságok 2013. évi közös Tudományos Kongresszusa Budapest, Magyarország : Semmelweis Egyetem Testnevelési és Sporttudományi Kar, (2013) pp. 106-106. , 1 p. Közlemény:2704276 Admin láttamozott Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat ) 94. Iring, A* ; Ruisanchez, E* ; Leszl-Ishiguro, M* ; Horvath, B ; Benko, R ; Lacza, Z ; Jarai, Z ; Sandor, P ; Di Marzo, V ; Pacher, P et al. Role of endocannabinoids and cannabinoid-1 receptors in cerebrocortical blood ow regulation PLOS ONE 8 : 1 Paper: e53390 , 11 p. (2013) DOI SE Repozitórium WoS Scopus PubMed Közlemény:2181946 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 12 Független: 9 Függő: 3 Idézett közlemények száma: 24

1. Aso E et al. Cannabinoids for treatment of alzheimer's disease: Moving toward the clinic. (2014) FRONTIERS IN PHARMACOLOGY 1663-9812 2014 MAR5 2. Davis MP. Cannabinoids in pain management: CB1, CB2 and non- classic receptor ligands. (2014) EXPERT OPINION ON INVESTIGATIONAL DRUGS 1354-3784 23 8 1123-1140 3. Lopez-Rodriguez AB et al. Changes in Cannabinoid Receptors, Aquaporin 4 and Vimentin Expression after Traumatic Brain Injury in Adolescent Male Mice. Association with Edema and Neurological Decit. (2015) PLOS ONE 1932-6203 1932-6203 10 6 4. * Altamura Claudia et al. Elevation of Plasma 2- Arachidonoylglycerol Levels in Alzheimer's Disease Patients as a Potential Protective Mechanism against Neurodegenerative Decline. (2015) JOURNAL OF ALZHEIMER'S DISEASE 1387-2877 1875-8908 46 2 497-506 5. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801 6. Libro Rosaliana et al. Natural Phytochemicals in the Treatment and Prevention of Dementia: An Overview. (2016) MOLECULES 1420-3049 21 4 7. Zong Yuan et al. Cannabinoids Regulate the Diameter of Pericyte- Containing Retinal Capillaries in Rats. (2017) CELLULAR PHYSIOLOGY AND BIOCHEMISTRY 1015-8987 1421-9778 43 5 2088-2101 8. * Iring A et al. CB1 receptor-mediated respiratory depression by endocannabinoids. (2017) RESPIRATORY PHYSIOLOGY AND NEUROBIOLOGY 1569-9048 1878-1519 240 48-52 9. Amanullah S et al. Synthetic cannabinoids in dementia. (2017) Megjelent: Handbook of Cannabis and Related Pathologies: Biology, Pharmacology, Diagnosis, and Treatment pp. 828-838 10. Chen Wendong. The inuence of goal-directed uid therapy on perioperative cerebral blood HO-1 and S100-beta of gastrointestinal surgery patients. (2017) Chinese Journal of Critical Care Medicine 1002-1949 37 3 260-264 11. Richter J et al. A Systematic Review of the Complex Eects of Cannabinoids on Cerebral and Peripheral Circulation in Animal Models. (2018) FRONTIERS IN PHYSIOLOGY 1664-042X 9 12. Green S. et al. The eects of on cerebral blood ow and its relationship to memory: an arterial spin labelling study. (2019) EUROPEAN NEUROPSYCHOPHARMACOLOGY 0924-977X 1873-7862 29 S693-S693

95. Kaucsar, T ; Revesz, C ; Godo, M ; Krenacs, T ; Albert, M ; Szalay, C ; Rosivall, L ; Benyo, Z ; Batkai, S ; Thum, T et al. Activation of the miR-17 Family and miR-21 During Murine Kidney Ischemia-Reperfusion Injury. NUCLEIC ACID THERAPEUTICS 23 : 5 pp. 344-354. , 11 p. (2013) DOI PubMed SE Repozitórium WoS Scopus Teljes dokumentum Közlemény:2387875 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 33 Független: 31 Függő: 2 Idézett közlemények száma: 4 1. Du W. By Targeting Stat3 microRNA-17-5p Promotes Cardiomyocyte Apoptosis in Response to Ischemia Followed by Reperfusion. (2014) CELLULAR PHYSIOLOGY AND BIOCHEMISTRY 1015-8987 1421-9778 34 3 955-965 2. Lindoso R. Extracellular vesicles released from mesenchymal stromal cells modulate miRNA in renal tubular cells and inhibit ATP depletion injury. (2014) STEM CELLS AND DEVELOPMENT 1547-3287 1557-8534 23 15 p. 1809-19 3. Collino Federica et al. miRNA Expression in Mesenchymal Stem Cells. (2014) CURRENT PATHOBIOLOGY REPORTS 2167-485X 2 3 101-107 4. Zhou T-B. Role of miR-21 and its signaling pathways in renal diseases. (2014) JOURNAL OF RECEPTOR AND SIGNAL TRANSDUCTION 1079-9893 1532-4281 34 5 335-337 5. Li Zhihui. Elevation of miR-21, through targeting MKK3, may be involved in ischemia pretreatment protection from ischemia– reperfusion induced kidney injury. (2015) JOURNAL OF NEPHROLOGY 1121-8428 29 1 27-36 6. Bhatt Kirti et al. MicroRNA-687 Induced by Hypoxia-Inducible Factor-1 Targets Phosphatase and Tensin Homolog in Renal Ischemia-Reperfusion Injury. (2015) JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY 1046-6673 1533-3450 26 7 1588-1596 7. Yuanyuan WEI. MicroRNA and hepatic brosis of biliary atresia. (2015) TIANJIN YIYAO / TIANJIN MEDICAL JOURNAL 0253-9896 43 11 1334-1338 8. Tang Wei-wei et al. MicroRNA expression proling in male and female model mice after renal ischemia-reperfusion injury. (2015) CHINESE JOURNAL OF TISSUE ENGINEERING RESEARCH 2095- 4344 19 5 772-777 9. Eline van. MicroRNAs in kidney transplantation; living up to their expectations?. (2015) Journal of Transplantation 2090-0007 2015 10. Castoldi Giovanna et al. Dierent regulation of miR-29a-3p in glomeruli and tubules in an experimental model of angiotensin II- dependent hypertension: potential role in renal brosis. (2016) CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY 0305-1870 1440-1681 43 3 335-342 11. Wei Qingqing et al. MicroRNA-489 Induction by Hypoxia-Inducible Factor-1 Protects against Ischemic Kidney Injury. (2016) JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY 1046-6673 1533- 3450 27 9 2784-2796 12. Fan Pei-Chun et al. MicroRNAs in acute kidney injury. (2016) HUMAN GENOMICS 1473-9542 1479-7364 10 13. Liu Zhiwen et al. MicroRNAs in Pathogenesis of Acute Kidney Injury. (2016) NEPHRON CLINICAL PRACTICE 1660-2110 134 3 149-153 14. Bruno Noemi. MicroRNAs relate to early worsening of renal function in patients with acute heart failure. (2016) INTERNATIONAL JOURNAL OF CARDIOLOGY 0167-5273 1874-1754 203 564-569 15. * Kaucsar T et al. Urine/Plasma Neutrophil Gelatinase Associated Lipocalin Ratio Is a Sensitive and Specic Marker of Subclinical Acute Kidney Injury in Mice. (2016) PLOS ONE 1932-6203 1932- 6203 11 1 16. Collino Federica et al. Exosome and Microvesicle-Enriched Fractions Isolated from Mesenchymal Stem Cells by Gradient Separation Showed Dierent Molecular Signatures and Functions on Renal Tubular Epithelial Cells. (2017) STEM CELL REVIEWS AND REPORTS 1550-8943 1558-6804 13 2 226-243 17. Dong Biao. GPR4 knockout improves renal ischemia-reperfusion injury and inhibits apoptosis via suppressing the expression of CHOP. (2017) BIOCHEMICAL JOURNAL 0264-6021 1470-8728 474 4065-4074 18. Hao Jielu. Induction of microRNA-17-5p by p53 protects against renal ischemia-reperfusion injury by targeting death receptor 6. (2017) KIDNEY INTERNATIONAL 0085-2538 1523-1755 91 1 106- 118 19. Dellago Hanna. MicroRNA-17-5p: At the Crossroads of Cancer and Aging - A Mini-Review. (2017) GERONTOLOGY 0304-324X 1423- 0003 63 1 20-28 20. Wang Kun. miR-21 attenuates contrast-induced renal cell apoptosis by targeting PDCD4. (2017) MOLECULAR MEDICINE REPORTS 1791-2997 1791-3004 16 5 6757-6763 21. Jiao XY. miR-21 contributes to renal protection by targeting prolyl hydroxylase domain protein 2 in delayed ischaemic preconditioning. (2017) NEPHROLOGY 1320-5358 1440-1797 22 5 366-373 22. Kota Satya et al. Noncoding RNA and epigenetic gene regulation in renal diseases. (2017) DRUG DISCOVERY TODAY 1359-6446 1878-5832 22 7 1112-1122 23. * Hosszu A et al. Sigma1-Receptor Agonism Protects against Renal Ischemia-Reperfusion Injury. (2017) JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY 1046-6673 1533-3450 28 1 152-165 24. Steichen C. et al. Barriers and Advances in Kidney Preservation. (2018) BIOMED RESEARCH INTERNATIONAL 2314-6133 2314-6141 25. Mohamed Watany et al. Circulating miR-21, miR-210 and miR- 146a as potential biomarkers to dierentiate acute tubular necrosis from hepatorenal syndrome in patients with liver cirrhosis: a pilot study. (2018) CLINICAL CHEMISTRY AND LABORATORY MEDICINE 1434-6621 1437-4331 56 5 739-747 26. Chen Di et al. IRE1 alpha inhibition decreased TXNIP/NLRP3 inammasome activation through miR-17-5p after neonatal hypoxic-ischemic brain injury in rats. (2018) JOURNAL OF NEUROINFLAMMATION 1742-2094 15 27. Xiao Bin et al. Plasma microRNA panel is a novel biomarker for focal segmental glomerulosclerosis and associated with podocyte apoptosis. (2018) CELL DEATH AND DISEASE 2041-4889 9 5 28. Zou Yan-Fang et al. Role of microRNA in the detection, progression, and intervention of acute kidney injury. (2018) EXPERIMENTAL BIOLOGY AND MEDICINE 1535-3702 1535-3699 243 2 129-136 29. Melis Nicolas et al. Emerging therapeutic strategies for transplantation-induced acute kidney injury: protecting the organelles and the vascular bed. (2019) EMERGING THERAPEUTIC TARGETS 1460-0412 1472-8222 23 6 495-509 30. Shunjie Chen et al. MiR-424 is over-expressed and attenuates ischemia-reperfusion kidney injury via p53 and death receptor 6 pathway. (2019) AMERICAN JOURNAL OF TRANSLATIONAL RESEARCH 1943-8141 11 4 1965-1979 31. Ren Gui-Ling et al. Noncoding RNAs in acute kidney injury. (2019) JOURNAL OF CELLULAR PHYSIOLOGY 0021-9541 1097-4652 234 3 2266-2276 32. Hromadnikova Ilona et al. Postnatal Expression Prole of microRNAs Associated with Cardiovascular and Cerebrovascular Diseases in Children at the Age of 3 to 11 Years in Relation to Previous Occurrence of Pregnancy-Related Complications. (2019) INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 1661-6596 1422-0067 20 3 p. 654 33. Shan Hailei et al. The TNF alpha-miR-106b-5p-Caspase 9 axis functions as a key regulator in cerebral infarction via modulating endothelial cell apoptosis. (2019) INTERNATIONAL JOURNAL OF CLINICAL AND EXPERIMENTAL MEDICINE 1940-5901 12 1 618-624

96. Masszi, G* ; Benko, R* ; Csibi, N ; Horvath, EM ; Tokes, AM ; Novak, A ; Beres, NJ ; Tarszabo, R ; Buday, A ; Repas, Cs et al. Endothelial relaxation mechanisms and nitrative stress are partly restored by Vitamin D3 therapy in a rat model of polycystic ovary syndrome LIFE SCIENCES 93 : 4 pp. 133-138. , 6 p. (2013) DOI PubMed REAL SE Repozitórium WoS Scopus Zárolt Közlemény:2320338 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 11 Független: 6 Függő: 5 Idézett közlemények száma: 6

1. * Masszi G et al. Erratum: Endothelial relaxation mechanisms and nitrative stress are partly restored by vitamin D3 therapy in a rat model of polycystic ovary syndrome (Life Sciences (2013) 93 (133- 138)). (2014) LIFE SCIENCES 0024-3205 100 1 p. 73 2. * Varbiro S et al. Lower-limb veins are thicker and vascular reactivity is decreased in a rat PCOS model: concomitant vitamin D3 treatment partially prevents these changes. (2014) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 307 6 H848-H857 3. Maliqueo M et al. Rodent Models of Polycystic Ovary Syndrome: Phenotypic Presentation, Pathophysiology, and the Eects of Dierent Interventions. (2014) SEMINARS IN REPRODUCTIVE MEDICINE 1526-8004 32 3 183-193 4. * Oláh A et al. Cardiac eects of acute exhaustive exercise in a rat model. (2015) INTERNATIONAL JOURNAL OF CARDIOLOGY 0167- 5273 1874-1754 182 258-266 5. Oláh A. Cardiac eects of long - term exercise training and acute exhaustive exercise in rat models. (2015) 6. Tepavcevic S et al. Cardiac Nitric Oxide Synthases and Na+/K+- ATPase in the Rat Model of Polycystic Ovary Syndrome Induced by Dihydrotestosterone. (2015) EXPERIMENTAL AND CLINICAL ENDOCRINOLOGY & DIABETES 0947-7349 1439-3646 123 5 303- 307 7. Haas MJ et al. Inhibition of endoplasmic reticulum stress and oxidative stress by vitamin D in endothelial cells. (2016) FREE RADICAL BIOLOGY AND MEDICINE 0891-5849 1873-4596 99 1-10 8. Tamadon Amin et al. How to choose the suitable animal model of polycystic ovary syndrome?. (2018) Tradit. Med. Mod. Med. 2575- 9000 01 02 95-113 9. * Pal E et al. Vitamin D deciency causes inward hypertrophic remodeling and alters vascular reactivity of rat cerebral arterioles.. (2018) PLOS ONE 1932-6203 1932-6203 13 2 10. * Lajtai Krisztina et al. Eects of Vitamin D Deciency on Proliferation and Autophagy of Ovarian and Liver Tissues in a Rat Model of Polycystic Ovary Syndrome. (2019) BIOMOLECULES 2218-273X 9 11. Hadjadj Leila et al. Vitamin D deciency and androgen excess result eutrophic remodeling and reduced myogenic adaptation in small cerebral arterioles in female rats. (2019) GYNECOLOGICAL ENDOCRINOLOGY 0951-3590 1473-0766 35 6 529-534

97. Masszi, G* ; Horvath, EM* ; Tarszabo, R ; Benko, R ; Novak, A ; Buday, A ; Tokes, AM ; Nadasy, NL ; Hamar, P ; Benyó, Z et al. Reduced Estradiol-Induced Vasodilation and Poly-(ADP-Ribose) Polymerase (PARP) Activity in the Aortas of Rats with Experimental Polycystic Ovary Syndrome (PCOS) PLOS ONE 8 : 3 Paper: e55589 , 7 p. (2013) DOI PubMed SE Repozitórium WoS Scopus Zárolt Közlemény:2169205 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 11 Független: 7 Függő: 4 Idézett közlemények száma: 14 1. * Varbiro S et al. Lower-limb veins are thicker and vascular reactivity is decreased in a rat PCOS model: concomitant vitamin D3 treatment partially prevents these changes. (2014) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 307 6 H848-H857 2. * Masszi G. Reduced Estradiol-Induced Vasodilation and Poly-(ADP- Ribose) Polymerase (PARP) Activity in the Aortas of Rats with Experimental Polycystic Ovary Syndrome (PCOS) (vol 8, e55589, 2013). (2014) PLOS ONE 1932-6203 1932-6203 9 2 3. El Atrash et al. Neuroprotective Role of Vitamin B3 in Experimentally Induced Oxidative Stress. (2015) International Journal of Clinical and Experimental Neurology 3 1 21-25 4. Lambrinoudaki Irene. Non-Hemodynamic Components of EVA: Polycystic Ovary Syndrome (PCOS). (2015) Megjelent: EARLY VASCULAR AGING (EVA): NEW DIRECTIONS IN CARDIOVASCULAR PROTECTION pp. 181-194 5. Örnek N et al. Ocular blood ow in polycystic ovary syndrome. (2015) JOURNAL OF OBSTETRICS AND GYNAECOLOGY RESEARCH 1341-8076 41 7 1080-1086 6. Gao Ling et al. Calcitriol attenuates cardiac remodeling and dysfunction in a murine model of polycystic ovary syndrome. (2016) ENDOCRINE 1355-008X 1559-0100 0969-711X 52 2 363-373 7. Çinar M et al. Experimental models of polycystic ovary syndrome. (2016) Medeniyet Medical Journal 2149-2042 2149-4606 31 1 53- 57 8. Tamadon Amin et al. How to choose the suitable animal model of polycystic ovary syndrome?. (2018) Tradit. Med. Mod. Med. 2575- 9000 01 02 95-113 9. Márton Judit et al. Poly(ADP-ribose) polymerase-2 is a lipid- modulated modulator of muscular lipid homeostasis. (2018) BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR AND CELL BIOLOGY OF LIPIDS 1388-1981 1879-2618 1863 11 1399-1412 10. * Pal Eva et al. Gender, hyperandrogenism and vitamin D deciency related functional and morphological alterations of rat cerebral arteries. (2019) PLOS ONE 1932-6203 1932-6203 14 5 11. * Hadjadj Leila et al. Geometric, elastic and contractile-relaxation changes in coronary arterioles induced by Vitamin D deciency in normal and hyperandrogenic female rats. (2019) MICROVASCULAR RESEARCH 0026-2862 1095-9319 122 78-84

98. Masszi, G* ; Buday, A* ; Novak, A ; Horvath, EM ; Tarszabo, R ; Sara, L ; Revesz, C ; Benko, R ; Nadasy, GL ; Benyo, Z et al. Altered insulin-induced relaxation of aortic rings in a dihydrotestosterone-induced rodent model of polycystic ovary syndrome FERTILITY AND STERILITY 99 : 2 pp. 573-578. , 6 p. (2013) DOI PubMed SE Repozitórium WoS Scopus Zárolt Közlemény:2113444 Hitelesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 7 Független: 5 Függő: 2 Idézett közlemények száma: 3

1. * Masszi G et al. Endothelial relaxation mechanisms and nitrative stress are partly restored by Vitamin D3 therapy in a rat model of polycystic ovary syndrome. (2013) LIFE SCIENCES 0024-3205 93 4 133-138 2. * Buday Anna. Endotél diszfunkció vizsgálata aortán, ateroszklerózis, és policisztás ovárium szindróma kísérleti modelljein. (2014) 3. Ilana B et al. Metabolic, Behavioral, and Reproductive Eects of Vertical Sleeve Gastrectomy in an Obese Rat Model of Polycystic Ovary Syndrome. (2014) OBESITY SURGERY 0960-8923 24 6 866- 876 4. Maliqueo M et al. Rodent Models of Polycystic Ovary Syndrome: Phenotypic Presentation, Pathophysiology, and the Eects of Dierent Interventions. (2014) SEMINARS IN REPRODUCTIVE MEDICINE 1526-8004 32 3 183-193 5. Tian Xiaoyu et al. Eects of Placental Ischemia Are Attenuated by 1,25-Dihydroxyvitamin D Treatment and Associated with Reduced Apoptosis and Increased Autophagy. (2016) DNA AND CELL BIOLOGY 1044-5498 35 2 59-70 6. Ma Su-ling et al. Vitamin D Supplementation Prevents Placental Ischemia Induced Endothelial Dysfunction by Downregulating Placental Soluble FMS-Like Tyrosine Kinase-1. (2017) DNA AND CELL BIOLOGY 1044-5498 36 12 1134-1141 7. Tamadon Amin et al. How to choose the suitable animal model of polycystic ovary syndrome?. (2018) Tradit. Med. Mod. Med. 2575- 9000 01 02 95-113

99. Masszi, Gabriella ; Novak, Agnes ; Tarszabo, Robert ; Horvath, Eszter Maria ; Buday, Anna ; Ruisanchez, Eva ; Tokes, Anna-Maria ; Sara, Levente ; Benko, Rita ; Nadasy, Gyorgy L et al. Eects of vitamin D3 derivate calcitriol on pharmacological reactivity of aortic rings in a rodent PCOS model PHARMACOLOGICAL REPORTS 65 : 2 pp. 476-483. , 8 p. (2013) DOI PubMed SE Repozitórium WoS Scopus Teljes dokumentum Zárolt Közlemény:2143448 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 9 Független: 6 Függő: 3 Idézett közlemények száma: 3

1. Gao Ling et al. Calcitriol attenuates cardiac remodeling and dysfunction in a murine model of polycystic ovary syndrome. (2016) ENDOCRINE 1355-008X 1559-0100 0969-711X 52 2 363-373 2. Yean Chun et al. Overview of Antagonists Used for Determining the Mechanisms of Action Employed by Potential Vasodilators with Their Suggested Signaling Pathways. (2016) MOLECULES 1420-3049 21 4 3. Zuo T et al. Roles of oxidative stress in polycystic ovary syndrome and cancers. (2016) OXIDATIVE MEDICINE AND CELLULAR LONGEVITY 1942-0900 1942-0994 2016 4. Çelik LS et al. Eects of vitamin D on ovary in DHEA-treated PCOS rat model: A light and electron microscopic study. (2018) ULTRASTRUCTURAL PATHOLOGY 0191-3123 1521-0758 42 1 55- 64 5. Tamadon Amin et al. How to choose the suitable animal model of polycystic ovary syndrome?. (2018) Tradit. Med. Mod. Med. 2575- 9000 01 02 95-113 6. * Hadjadj L et al. Insulin resistance in an animal model of polycystic ovary disease is aggravated by vitamin D deciency: Vascular consequences. (2018) DIABETES & VASCULAR DISEASE RESEARCH 1479-1641 1752-8984 15 4 294-301 7. Alttas Savas et al. Evaluation of the Rho A/Rho-kinase pathway in the uterus of the rat model of polycystic ovary syndrome. (2019) REPRODUCTIVE BIOLOGY 1642-431X 19 1 45-54 8. * Pal Eva et al. Gender, hyperandrogenism and vitamin D deciency related functional and morphological alterations of rat cerebral arteries. (2019) PLOS ONE 1932-6203 1932-6203 14 5 9. * Hadjadj Leila et al. Geometric, elastic and contractile-relaxation changes in coronary arterioles induced by Vitamin D deciency in normal and hyperandrogenic female rats. (2019) MICROVASCULAR RESEARCH 0026-2862 1095-9319 122 78-84 100. Móré, D ; Dancs, P ; Nagy, Z ; Benyó, Z ; Ruisanchez, É A szngozin-1-foszfát tartósan fokozza az erek kontrakciós válaszkészségét In: Magyar, Élettani Társaság - A Magyar Élettani, Farmakológiai és Mikrocirkulációs Társaságok 2013. évi közös Tudományos Kongresszusa Budapest, Magyarország : Semmelweis Egyetem Testnevelési és Sporttudományi Kar, (2013) pp. 99-99. , 1 p. Közlemény:2704277 Admin láttamozott Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat )

101. Ruisanchez, É ; Dancs, P ; Móré, D ; Oermanns, S ; Benyó, Z Sphingosine-1-phosphate permanently enhances the contractile responsiveness of vascular smooth muscle via the S1P2 – G12/13 – Rho- kinase signaling pathway In: 10th Sphingolipid Club Meeting Abstract Book (2013) Paper: OP20 Közlemény:2704284 Admin láttamozott Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat )

102. Tarszabó, R ; Masszi, G ; Horváth, E ; Béres, N J ; Benkő, R ; Novák, Á ; Buday, A ; Tőkés, A -M ; Nádasy, Gy L ; Hamar, P et al. A VAZOKONSTRIKCIÓ ÉS -RELAXÁCIÓ VÁLTOZÁSAI AORTÁN, PATKÁNY POLICISZTÁS OVÁRIUM SZINDRÓMA MODELLBEN pp. 30-31. , 2 p. (2013) Magyar Tudós Ifjú Hypertonologusok és Nephrologusok Értekezlete, II. Aesculap Mesterkurzus (MATHINE), Előadás, 2013. 03.22. Budapest, Zárolt Közlemény:2244919 Admin láttamozott Forrás Egyéb (Nem besorolt )

103. Tóth, G ; Szekeres, M ; Benyó, Z ; Hunyady, L A GFKR-szelektív jelátvitel szerepe az agonista-indukált endokannabinoid-felszabadulás vazokonstrikciót mérséklő hatásában. In: Magyar, Élettani Társaság - A Magyar Élettani, Farmakológiai és Mikrocirkulációs Társaságok 2013. évi közös Tudományos Kongresszusa Budapest, Magyarország : Semmelweis Egyetem Testnevelési és Sporttudományi Kar, (2013) p. A-0162 Közlemény:2565733 Admin láttamozott Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat )

104. Várbíró, Szabolcs ; Masszi, Gabriella ; Sára, Levente ; Buday, Anna ; Horváth, Eszter ; Antal, Péter ; Tarszabó, Róbert ; Novák, Ágnes ; Benkő, Rita ; Ruisanchez, Éva et al. D vitamin hatása kis- és nagyerekre hiperandrogén nőstény patkányokon kialakított policisztás petefészek szindróma (PCOS) modellen - az inzulin rezisztencia regionális különbségei In: Magyar, Élettani Társaság - A Magyar Élettani, Farmakológiai és Mikrocirkulációs Társaságok 2013. évi közös Tudományos Kongresszusa Budapest, Magyarország : Semmelweis Egyetem Testnevelési és Sporttudományi Kar, (2013) Paper: A-0111 Zárolt Közlemény:2708641 Admin láttamozott Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat )

105. Horváth, B ; Iring, A ; Benyó, B ; Hermán, P ; Lenzsér, G ; Lacza, Zs ; Sándor, P ; Benyó, Z Low frequency pial arterial vasomotion and cerebrocortical blood ow oscillations in rodents: role of nitric oxide and thromboxane A2 In: SRBR Society for Research on Biological Rhythms 2012 : 13th Biennial Meeting Society for Research on Biological Rhythms Conference Program (2012) pp. 266-266. Paper: P268 , 1 p. Közlemény:2845095 Érvényesített Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat )

106. Ruisanchez, E ; Cselenyak, A ; Papp, RS ; Nemeth, T ; Kaldi, K ; Sandor, P ; Benyo, Z Perivascular expression and potent vasoconstrictor eect of dynorphin a in cerebral arteries. PLOS ONE 7 : 5 Paper: e37798 , 8 p. (2012) DOI WoS Scopus PubMed Google scholar Google scholar hash Közlemény:1980047 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 3 Független: 1 Függő: 2 Idézett közlemények száma: 7

1. Bodnar Richard. Endogenous opiates and behavior: 2012. (2013) PEPTIDES 0196-9781 1873-5169 50 55-95 2. * Iring A et al. Role of endocannabinoids and cannabinoid-1 receptors in cerebrocortical blood ow regulation. (2013) PLOS ONE 1932-6203 1932-6203 8 1 3. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801

107. Ruisanchez, É ; Cselenyák, A ; Papp, Rege S ; Németh, T ; Káldi, K ; Sándor, P ; Benyó, Z Dynorphin A perivaszkuláris expressziója és erős vazokonstriktor hatása agyi erekben In: Csernoch, László (szerk.) A Magyar Élettani Társaság, a Magyar Anatómusok Társasága, a Magyar Biozikai Társaság és a Magyar Mikrocirkulációs és Vaszkuláris Biológiai Társaság Kongresszusa Magyar Élettani Társaság, (2012) pp. 172-172. , 1 p. Közlemény:2845084 Admin láttamozott Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat )

108. Szekeres, M ; Nádasy, GyL ; Tóth, Zs ; Várnai, P ; Soltész-Katona, E ; Ruisanchez, É ; Benyó, Z ; Hunyady, L Angiotenzin II jelátvitel-függő endokannabinoid hatás heterogenitása az artériás rendszerben In: Csernoch, László (szerk.) A Magyar Élettani Társaság, a Magyar Anatómusok Társasága, a Magyar Biozikai Társaság és a Magyar Mikrocirkulációs és Vaszkuláris Biológiai Társaság Kongresszusa Magyar Élettani Társaság, (2012) pp. 34-34. , 1 p. Közlemény:2087306 Admin láttamozott Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat )

109. Buday, A ; Orsy, P ; Godó, M ; Mózes, M ; Kökény, G ; Lacza, Z ; Koller, A ; Ungvári, Z ; Gross, ML ; Benyó, Z et al. Az emelkedett TGF-béta-szint gyorsult érelmeszesedést és hypertoniát okoz Apo-E knockout egerekben HYPERTONIA ÉS NEPHROLOGIA 16 : Suppl. 3. pp. S36-S36. (2011) Teljes dokumentum Közlemény:2878866 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

110. Hirschberg, K ; Nemeth, T ; Pali, Sz ; Barnucz, E ; Korkmaz, S ; Berta, B ; Nuesing, R ; Benyo, Z ; Merkely, B ; Szabo, Sz Reduced wire-injury induced stenosis in the femoral artery of aging thromboxane receptor knockout mice EUROPEAN HEART JOURNAL 32 : Suppl. 1 pp. 32-32. , 1 p. (2011) Kiadónál WoS Közlemény:2013928 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

111. Masszi, G ; Buday, A ; Novák, Á ; Horváth, EM ; Tarszabó, R ; Sára, L ; Révész, Cs ; Benkő, R ; Nádasy, Gy ; Benyó, Z et al. Inzulin rezisztencia endothel funkcióra gyakorolt hatása policisztás ovárium szindróma patkány modelljén HYPERTONIA ÉS NEPHROLOGIA 15 : Suppl. 3. p. s36 (2011) Közlemény:2878869 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

112. Buday, A ; Őrsy, P ; Godó, M ; Kökény, G ; Mózes, M ; Gross, ML ; Ungvári, Z ; Benyó, Z ; Hamar, P A TGF-b kardiovaszkuláris hatásainak vizsgálata kettős génmodikált egereken In: Magyar Élettani Társaság 2010. évi Vándorgyűlése, és a Magyar Kísérletes és Klinikai Farmakológiai Társaság tudományos konferenciája (2010) p. 6 Közlemény:2879009 Admin láttamozott Forrás Egyéb konferenciaközlemény (Absztrakt / Kivonat )

113. Buday, A ; Orsy, P ; Godo, M ; Mozes, M ; Kokeny, G ; Lacza, Z ; Koller, A ; Ungvari, Z ; Gross, ML ; Benyo, Z et al. Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) mice AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 299 : 2 pp. H386-H395. (2010) DOI WoS Scopus PubMed Közlemény:1394350 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 50 Független: 47 Függő: 3 Idézett közlemények száma: 6

1. Gil-Bernabe P et al. Atherosclerosis Amelioration by Moderate Alcohol Consumption Is Associated With Increased Circulating Levels of Stromal Cell-Derived Factor-1. (2011) CIRCULATION JOURNAL: OFFICIAL JOURNAL OF THE JAPANESE CIRCULATION SOCIETY 1346-9843 1347-4820 75 9 2269-2279 2. Reilko RA et al. Fibrocytes: emerging eector cells in chronic inammation. (2011) NATURE REVIEWS IMMUNOLOGY 1474-1733 11 6 427-435 3. Blasig Ingolf et al. Occludin Protein Family: Oxidative Stress and Reducing Conditions. (2011) ANTIOXIDANTS & REDOX SIGNALING 1523-0864 1557-7716 15 5 1195-1219 4. Mitra S et al. Oxidized LDL, LOX-1 and Atherosclerosis. (2011) CARDIOVASCULAR DRUGS AND THERAPY 0920-3206 1573-7241 25 5 419-429 5. WANG Xiaoxing. Pirfenidone improves advanced atherosclerosis in apolipoprotein E decient mice. (2011) Military Medical Sciences 1674-9960 35 6 410-415 6. Afanas'ev I. ROS and RNS Signaling in Heart Disorders: Could Antioxidant Treatment Be Successful?. (2011) OXIDATIVE MEDICINE AND CELLULAR LONGEVITY 1942-0900 1942-0994 2011 7. Miguel-Carrasco JL et al. Blockade of TGF-beta 1 Signalling Inhibits Cardiac NADPH Oxidase Overactivity in Hypertensive Rats. (2012) OXIDATIVE MEDICINE AND CELLULAR LONGEVITY 1942-0900 1942-0994 2012 8. Russell TM. Flow cytometric identication of brocytes in scleroderma lung disease. (2012) Megjelent: Autoimmunity pp. 327-346 9. Ekuni Daisuke et al. Hydrogen-rich water prevents lipid deposition in the descending aorta in a rat periodontitis model. (2012) ARCHIVES OF ORAL BIOLOGY 0003-9969 57 12 1615-1622 10. Meyer A et al. Platelet TGF-beta 1 contributions to plasma TGF- beta 1, cardiac brosis, and systolic dysfunction in a mouse model of pressure overload. (2012) BLOOD 0006-4971 1528-0020 119 4 1064-1074 11. Toma I et al. Transforming growth factor-beta and atherosclerosis: interwoven atherogenic and atheroprotective aspects. (2012) CELL AND TISSUE RESEARCH 0302-766X 1432- 0878 347 1 155-175 12. Guo W et al. AAV-Mediated Administration of Myostatin Pro- Peptide Mutant in Adult Ldlr Null Mice Reduces Diet-Induced Hepatosteatosis and Arteriosclerosis. (2013) PLOS ONE 1932- 6203 1932-6203 8 8 13. * Masszi G et al. Altered insulin-induced relaxation of aortic rings in a dihydrotestosterone-induced rodent model of polycystic ovary syndrome. (2013) FERTILITY AND STERILITY 0015-0282 99 2 573- 578 14. Baltanas A et al. A Synthetic Peptide from Transforming Growth Factor-beta(1) Type III Receptor Inhibits NADPH Oxidase and Prevents Oxidative Stress in the Kidney of Spontaneously Hypertensive Rats. (2013) ANTIOXIDANTS & REDOX SIGNALING 1523-0864 1557-7716 19 14 1607-1618 15. Droujinine IA et al. Dening the interorgan communication network: systemic coordination of organismal cellular processes under homeostasis and localized stress. (2013) FRONTIERS IN CELLULAR AND INFECTION MICROBIOLOGY 2235-2988 3 16. * Masszi Gabriella et al. Eects of vitamin D3 derivate calcitriol on pharmacological reactivity of aortic rings in a rodent PCOS model. (2013) PHARMACOLOGICAL REPORTS 1734-1140 65 2 476-483 17. Jiang Hui et al. Genetic polymorphisms of cytokines associated with Kawasaki disease. (2013) JOURNAL OF CLINICAL PEDIATRICS 1000-3606 31 1 85-88 18. Mazagova M et al. Growth dierentiation factor 15 impairs aortic contractile and relaxing function through altered caveolar signaling of the endothelium. (2013) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 304 5 H709-H718 19. He Aina et al. Mast Cell Chymase and Tryptase as Targets for Cardiovascular and Metabolic Diseases. (2013) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873-4286 19 6 1114-1125 20. Tang T et al. Prevention of TGF beta induction attenuates angII- stimulated vascular biglycan and atherosclerosis in Ldlr(-/-) mice. (2013) JOURNAL OF LIPID RESEARCH 0022-2275 1539-7262 54 8 2255-2264 21. * Masszi G et al. Reduced Estradiol-Induced Vasodilation and Poly- (ADP-Ribose) Polymerase (PARP) Activity in the Aortas of Rats with Experimental Polycystic Ovary Syndrome (PCOS). (2013) PLOS ONE 1932-6203 1932-6203 8 3 22. Fei AH et al. Salvianolate inhibits reactive oxygen species production in H2O2-treated mouse cardiomyocytes in vitro via the TGF beta pathway. (2013) ACTA PHARMACOLOGICA SINICA 1671-4083 1745-7254 34 4 496-500 23. Samarakoon R et al. TGF-beta signaling in tissue brosis: Redox controls, target genes and therapeutic opportunities. (2013) CELLULAR SIGNALLING 0898-6568 1873-3913 25 1 264-268 24. Roda Julie et al. The Cellular Component of Chronic Inammation. (2013) Megjelent: CHRONIC INFLAMMATION: MOLECULAR PATHOPHYSIOLOGY, NUTRITIONAL AND THERAPEUTIC INTERVENTIONS pp. 21-33 25. SHANG Ruru. The role of Cd4 T lymphocytes in the development of high blood pressure. (2013) Chinese Journal of Hypertension 1673-7245 21 12 1155-1158 26. Montero-Vega MT. Are oxidised low-density lipoproteins the true inducers of inammation in atherosclerosis?. (2014) Inammasome 2300-102X 1 1 55-74 27. Cappelletti A et al. Association of LOXIN, a new functional splicing isoform of the OLR1 gene, with severity and prognostic localization of critical coronary artery stenoses. (2014) JOURNAL OF CARDIOVASCULAR MEDICINE 1558-2027 15 5 391-396 28. Yan F et al. Nox4 and redox signaling mediate TGF-beta-induced endothelial cell apoptosis and phenotypic switch. (2014) CELL DEATH AND DISEASE 2041-4889 5 29. Ekuni Daisuke et al. Occlusal disharmony accelerates the initiation of atherosclerosis in apoE knockout rats. (2014) LIPIDS IN HEALTH AND DISEASE 1476-511X 13 30. Ekuni Daisuke et al. Periodontitis and Atherosclerosis. (2014) Megjelent: Studies on Periodontal Disease pp. 183-195 31. Byun HO et al. PKC delta phosphorylation is an upstream event of GSK3 inactivation-mediated ROS generation in TGF-beta 1- induced senescence. (2014) FREE RADICAL RESEARCH 1071-5762 1029-2470 48 9 1100-1108 32. Marín-García J et al. Post-Genomic Cardiology: Second Edition. (2014) ISBN:9780124045996 33. Camara AKS et al. Reactive oxygen species and cardiac ischemia and reperfusion injury. (2014) Megjelent: Systems Biology of Free Radicals and Antioxidants pp. 889-949 34. GAO Zuo. The role of reactive oxygen in the phenotype transformation, proliferation, migration and collagen secretion of vascular broblasts in vitro. (2014) ZHONGGUO LAONIANXUE ZAZHI / CHINESE JOURNAL OF GERONTOLOGY 1005-9202 34 22 6523-6526 35. Mao S et al. Association of transforming growth factor-beta 1 polymorphisms with the risk of chronic kidney diseases. (2015) RENAL FAILURE 0886-022X 1525-6049 37 10 304-311 36. Kusters PascalJH et al. Cytokines and Immune Responses in Murine Atherosclerosis. (2015) Megjelent: Methods in Mouse Atherosclerosis pp. 17-40 37. Iwata Hiroshi et al. Dynamic Interplay Between Smooth Muscle Cells and Macrophages in Vascular Disease. (2015) Megjelent: Muscle Cell and Tissue pp. 233-257 38. Hu X et al. Flow cytometric identication of brocytes in the human circulation. (2015) METHODS IN MOLECULAR BIOLOGY 1064-3745 1940-6029 1343 19-33 39. Gisterå A. T - CELL SPECIFICITY AND REGULATION IN ATHEROSCLEROSIS. (2016) 40. Sfyri Peggy et al. Crossroads between peripheral atherosclerosis, western-type diet and skeletal muscle pathophysiology: emphasis on apolipoprotein E deciency and peripheral arterial disease. (2017) JOURNAL OF BIOMEDICAL SCIENCE 1021-7770 1423-0127 24 41. Randell Amy et al. Elastin microbril interface-located protein 1, transforming growth factor beta, and implications on cardiovascular complications. (2017) JOURNAL OF THE AMERICAN SOCIETY OF HYPERTENSION 1933-1711 1878-7436 11 7 437-448 42. Gistera Anton et al. The immunology of atherosclerosis. (2017) NATURE REVIEWS NEPHROLOGY 1759-5061 1759-507X 13 6 368- 380 43. Vallee Alexandre et al. Thermodynamic Aspects and Reprogramming Cellular Energy Metabolism during the Fibrosis Process. (2017) INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 1661-6596 1422-0067 18 12 44. Maria C et al. Eect of the Antioxidant Lipoic Acid in Aortic Phenotype in a Marfan Syndrome Mouse Model. (2018) OXIDATIVE MEDICINE AND CELLULAR LONGEVITY 1942-0900 1942-0994 2018 45. Kekulandara Dilini. Investigating Redox-mediated Protein Glutathionylation and Thioredoxin 1 Signaling Pathway. (2018) 46. Bonafede R et al. Nitric oxide and related factors linked to oxidation and inammation as possible biomarkers of heart failure. (2018) CLINICA E INVESTIGACION EN ARTERIOSCLEROSIS 0214-9168 30 2 84-94 47. P.J.H. Kusters. Evaluating the therapeutic potential of CD40 in low-grade chronic inammation. (2019) 48. Ma Si-Ming et al. Gene-Level Regulation of Acupuncture Therapy in Spontaneously Hypertensive Rats: A Whole Transcriptome Analysis. (2019) EVIDENCE-BASED COMPLEMENTARY AND ALTERNATIVE MEDICINE 1741-427X 1741-4288 49. Zhang Xiaodong et al. Morin attenuates oxidized low‐density lipoprotein‐mediated injury by inducing autophagy via activating AMPK signaling in HUVECs. (2019) CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY 0305-1870 1440-1681 46 11 1053-1060 50. Han‐Seok Jeong et al. Retinal pigment epithelium‐derived transforming growth factor‐β2 inhibits the angiogenic response of endothelial cells by decreasing vascular endothelial growth factor receptor‐2 expression. (2019) JOURNAL OF CELLULAR PHYSIOLOGY 0021-9541 1097-4652 234 4 3837-3849

114. Donko, A ; Ruisanchez, E ; Orient, A ; Enyedi, B ; Kapui, R ; Peter, Z ; de Deken, X ; Benyo, Z ; Geiszt, M Urothelial cells produce hydrogen peroxide through the activation of Duox1 FREE RADICAL BIOLOGY AND MEDICINE 49 : 12 pp. 2040-2048. , 9 p. (2010) DOI WoS Scopus PubMed Közlemény:1468050 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 45 Független: 29 Függő: 16 Idézett közlemények száma: 4

1. * Grasberger H et al. Mice Decient in Dual Oxidase Maturation Factors Are Severely Hypothyroid. (2012) MOLECULAR ENDOCRINOLOGY 0888-8809 1944-9917 26 3 481-492 2. Suzuki M et al. The molecular mechanism of multifunctional mechano-gated channel TRPV4. (2012) Megjelent: Mechanically Gated Channels and their Regulation pp. 103-157 3. Li L et al. Activation of Transient Receptor Potential Vanilloid 4 Increases NMDA-Activated Current in Hippocampal Pyramidal Neurons. (2013) FRONTIERS IN CELLULAR NEUROSCIENCE 1662- 5102 7 4. Russo J et al. Comparative Eects of the Preventive Eect of Pregnancy, Steroidal Hormones, and hCG in the Transcriptomic Prole of the Rat Mammary Gland. (2013) Megjelent: Role of the transcriptome in breast cancer prevention pp. 73-189 5. Zhang XZ et al. Evolution of the Ferric Reductase Domain (FRD) Superfamily: Modularity, Functional Diversication, and Signature Motifs. (2013) PLOS ONE 1932-6203 1932-6203 8 3 6. Sobhakumari A et al. NOX4 mediates cytoprotective autophagy induced by the EGFR inhibitor erlotinib in head and neck cancer cells. (2013) TOXICOLOGY AND APPLIED PHARMACOLOGY 0041- 008X 1096-0333 272 3 736-745 7. Russo J et al. Role of the transcriptome in breast cancer prevention. (2013) ISBN:9781461448846; 9781461448839; 1461448832 8. * Enyedi B et al. Spatial and Temporal Analysis of NADPH Oxidase- Generated Hydrogen Peroxide Signals by Novel Fluorescent Reporter Proteins.. (2013) ANTIOXIDANTS & REDOX SIGNALING 1523-0864 1557-7716 19 6 523-534 9. Chaturvedi KS et al. Cupric yersiniabactin is a virulence-associated superoxide dismutase mimic. (2014) ACS CHEMICAL BIOLOGY 1554-8929 1554-8937 9 2 551-561 10. Brommage Robert et al. High-throughput screening of mouse gene knockouts identies established and novel skeletal phenotypes. (2014) BONE RESEARCH 2095-4700 2095-6231 2 11. Choi H et al. Hydrogen peroxide generated by DUOX1 regulates the expression levels of specic dierentiation markers in normal human keratinocytes. (2014) JOURNAL OF DERMATOLOGICAL SCIENCE 0923-1811 1873-569X 74 1 56-63 12. * Donkó A et al. Hypothyroidism-associated missense mutation impairs NADPH oxidase activity and intracellular tracking of Duox2. (2014) FREE RADICAL BIOLOGY AND MEDICINE 0891-5849 1873-4596 73 190-200 13. * Hristova M et al. Identication of DUOX1-dependent redox signaling through protein S-glutathionylation in airway epithelial cells. (2014) REDOX BIOLOGY 2213-2317 2 1 436-446 14. * Rada B et al. NLRP3 inammasome activation and interleukin- 1beta release in macrophages require calcium but are independent of calcium-activated NADPH oxidases.. (2014) INFLAMMATION RESEARCH 1023-3830 1420-908X 63 10 821-830 15. * De Deken X et al. Roles of DUOX-mediated hydrogen peroxide in metabolism, host defense, and signaling. (2014) ANTIOXIDANTS & REDOX SIGNALING 1523-0864 1557-7716 20 17 2776-2793 16. Fortunato RS et al. Sexual dimorphism and thyroid dysfunction: a matter of oxidative stress?. (2014) JOURNAL OF ENDOCRINOLOGY 0022-0795 1479-6805 221 2 R31-R40 17. Zhang Eric et al. Brain transient receptor potential channels and stroke. (2015) JOURNAL OF NEUROSCIENCE RESEARCH 0360-4012 1097-4547 93 8 1165-1183 18. Lu H et al. DUOX2 promotes the elimination of the Klebsiella pneumoniae strain K5 from T24 cells through the reactive oxygen species pathway. (2015) INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE 1107-3756 1791-244X 36 2 551-558 19. Altenhofer Sebastian et al. Evolution of NADPH Oxidase Inhibitors: Selectivity and Mechanisms for Target Engagement. (2015) ANTIOXIDANTS & REDOX SIGNALING 1523-0864 1557-7716 23 5 406-427 20. O'Neill S et al. Genetic disorders coupled to ROS deciency. (2015) REDOX BIOLOGY 2213-2317 6 135-156 21. Ding Hao et al. Research progress of TRPV4 and cerebral ischemic reperfusion injury. (2015) ACTA PHYSIOLOGICA SINICA 0371-0874 67 5 527-532 22. * Ueyama T et al. The extracellular A-loop of dual oxidases aects the specicity of reactive oxygen species release. (2015) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 290 10 6495- 6506 23. * Hristova M et al. Airway epithelial dual oxidase 1 mediates allergen-induced IL-33 secretion and activation of type 2 immune responses. (2016) JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY 0091-6749 1097-6825 137 5 1545-1556e11 24. * Péter K et al. Complement MASP-1 enhances adhesion between endothelial cellsand neutrophils by up-regulating E-selectin expression. (2016) MOLECULAR IMMUNOLOGY 0161-5890 75 38- 47 25. * Habibovic A et al. DUOX1 mediates persistent epithelial EGFR activation, mucous cell metaplasia, and airway remodeling during allergic asthma. (2016) JCI INSIGHT 2379-3708 1 18 26. Hong Zhiwen et al. Enhanced Oxidative Stress Is Responsible for TRPV4-Induced Neurotoxicity. (2016) FRONTIERS IN CELLULAR NEUROSCIENCE 1662-5102 10 27. * Sirokmany G et al. Epidermal growth factor-induced hydrogen peroxide production is mediated by dual oxidase 1.. (2016) FREE RADICAL BIOLOGY AND MEDICINE 0891-5849 1873-4596 97 204- 211 28. Ameziane-El-Hassani R et al. NADPH oxidases: New actors in thyroid cancer?. (2016) NATURE REVIEWS ENDOCRINOLOGY 1759- 5029 1759-5037 12 8 485-494 29. * Sirokmány G et al. Nox/Duox Family of NADPH Oxidases: Lessons from Knockout Mouse Models. (2016) TRENDS IN PHARMACOLOGICAL SCIENCES 0165-6147 37 4 318-327 30. * Eskalli Zineb et al. Overexpression of Interleukin-4 in the Thyroid of Transgenic Mice Upregulates the Expression of Duox1 and the Anion Transporter Pendrin. (2016) THYROID 1050-7256 1557- 9077 26 10 1499-1512 31. Heppner DE et al. The NADPH oxidases DUOX1 and NOX2 play distinct roles in redox regulation of epidermal growth factor receptor signaling. (2016) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 291 44 23282-23293 32. Wu Qiong-Feng et al. Activation of transient receptor potential vanilloid 4 involves in hypoxia/reoxygenation injury in cardiomyocytes. (2017) CELL DEATH AND DISEASE 2041-4889 8 33. Muzza Marina et al. Disorders of H2O2 generation. (2017) BEST PRACTICE & RESEARCH CLINICAL ENDOCRINOLOGY & METABOLISM 1521-690X 31 2 225-240 34. Kizys Marina et al. DUOX2 Mutations Are Associated With Congenital Hypothyroidism With Ectopic Thyroid Gland. (2017) JOURNAL OF CLINICAL ENDOCRINOLOGY AND METABOLISM 0021-972X 1945-7197 102 11 4060-4071 35. Sarr Demba et al. Antimicrobial actions of dual oxidases and lactoperoxidase. (2018) JOURNAL OF MICROBIOLOGY 1225-8873 56 6 373-386 36. Peters C. et al. DIAGNOSIS OF ENDOCRINE DISEASE Congenital hypothyroidism: update and perspectives. (2018) EUROPEAN JOURNAL OF ENDOCRINOLOGY 0804-4643 1479-683X 179 6 R297- R317 37. van der Vliet Albert et al. Dual oxidase: a novel therapeutic target in allergic disease. (2018) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 175 9 1401-1418 38. * Sirokmány G et al. Peroxidasin-mediated crosslinking of collagen IV is independent of NADPH oxidases. (2018) REDOX BIOLOGY 2213-2317 16 314-321 39. Iguchi Nao et al. Doxorubicin induces detrusor smooth muscle impairments through myosin dysregulation, leading to a risk of lower urinary tract dysfunction. (2019) AMERICAN JOURNAL OF PHYSIOLOGY: RENAL PHYSIOLOGY 1931-857X 317 1 F197-F206 40. * Sugamata Ryuichi et al. Duox1 Regulates Primary B Cell Function under the Inuence of IL-4 through BCR-Mediated Generation of Hydrogen Peroxide. (2019) JOURNAL OF IMMUNOLOGY 0022- 1767 1550-6606 202 2 428-440 41. Szanto I. et al. H2O2 metabolism in normal thyroid cells and in thyroid tumorigenesis: Focus on NADPH oxidases. (2019) ANTIOXIDANTS 2076-3921 8 5 42. Liu Shiguo et al. Identication of Two Missense Mutations in DUOX1 (p.R1307Q) and DUOXA1 (p.R56W) That Can Cause Congenital Hypothyroidism Through Impairing H2O2 Generation. (2019) FRONTIERS IN ENDOCRINOLOGY 1664-2392 10 43. Herb Marc et al. Mitochondrial reactive oxygen species enable proinammatory signaling through disulde linkage of NEMO. (2019) SCIENCE SIGNALING 1945-0877 1937-9145 12 568 44. Park Jong-Su et al. Targeted knockout of duox causes defects in zebrash growth, thyroid development, and social interaction. (2019) JOURNAL OF GENETICS AND GENOMICS 1673-8527 1873- 5533 46 2 101-104 45. * Sirokmany Gabor et al. The Relationship of NADPH Oxidases and Heme Peroxidases: Fallin' in and Out. (2019) FRONTIERS IN IMMUNOLOGY 1664-3224 1664-3224 10

115. Donko, A ; Ruisanchez, E ; Orient, A ; Enyedi, B ; Kapui, R ; Peter, Z ; de Deken, X ; Benyo, Z ; Geiszt, M Urothelial cells produce hydrogen peroxide through the activation of Duox1 ACTA PHYSIOLOGICA HUNGARICA 97 : 4 pp. 436-436. , 1 p. (2010) WoS Közlemény:1465953 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

116. Horvath, B* ; Lenzser, G* ; Benyo, B ; Nemeth, T ; Benko, R ; Iring, A ; Herman, P ; Komjati, K ; Lacza, Z ; Sandor, P et al. Hypersensitivity to thromboxane receptor mediated cerebral vasomotion and CBF oscillations during acute NO-deciency in rats. PLOS ONE 5 : 12 Paper: e14477 , 6 p. (2010) DOI WoS Scopus PubMed Pubmed Central Google scholar Google scholar hash Közlemény:1465909 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 11 Független: 5 Függő: 6 Idézett közlemények száma: 7

1. * Homlok József et al. Model based analysis of cerebrovascular oscillation using the system Circle of Willis. (2012) Megjelent: Proceedings of the IEEE 16th International Conference on Intelligent Engineering Systems 2012 pp. 543-547 2. * Ruisanchez E et al. Perivascular expression and potent vasoconstrictor eect of dynorphin a in cerebral arteries.. (2012) PLOS ONE 1932-6203 1932-6203 7 5 3. Wang Qingsong et al. Recent advance in pathogenesis of cerebral vasospasm after subarachnoid hemorrhage WA NG. (2013) ZHONGHUA SHENJING YIXUE ZAZHI / CHINESE JOURNAL OF NEUROMEDICINE 1671-8925 12 4 422-425 4. * Iring A et al. Role of endocannabinoids and cannabinoid-1 receptors in cerebrocortical blood ow regulation. (2013) PLOS ONE 1932-6203 1932-6203 8 1 5. Mima Akira et al. Use of a polysulfone hemodialysis membrane may prevent recurrent posterior reversible encephalopathy syndrome in a patient undergoing hemodialysis. (2014) INTERNATIONAL UROLOGY AND NEPHROLOGY 0301-1623 1573- 2584 46 1 255-260 6. Carlstrom Mattias et al. RENAL AUTOREGULATION IN HEALTH AND DISEASE. (2015) PHYSIOLOGICAL REVIEWS 0031-9333 1522- 1210 95 2 405-511 7. * Polycarpou A et al. Adaptation of the Cerebrocortical Circulation to Carotid Artery Occlusion Involves Blood Flow Redistribution between Cortical Regions and is Independent of eNOS.. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 311 4 H972-H980 8. * Vácz G et al. Eects of amniotic epithelial cell transplantation in endothelial injury. (2016) INTERVENTIONAL MEDICINE AND APPLIED SCIENCE 2061-1617 2061-5094 8 4 164-171 9. Moreira Aline et al. L-arginine supplementation and thromboxane synthase inhibition increases cerebral blood ow in experimental cerebral malaria. (2019) SCIENTIFIC REPORTS 2045-2322 9 10. Lagier David et al. Thromboxane-prostaglandin receptor antagonist, terutroban, prevents neurovascular events after subarachnoid haemorrhage: a nanoSPECT study in rats. (2019) CRITICAL CARE 1364-8535 1466-609X 23 11. * Hadjadj Leila et al. Vitamin D deciency and androgen excess result eutrophic remodeling and reduced myogenic adaptation in small cerebral arterioles in female rats. (2019) GYNECOLOGICAL ENDOCRINOLOGY 0951-3590 1473-0766 35 6 529-534

117. Iring, A ; Horváth, B ; Lenzsér, G ; Tillson, M ; Bakken, B ; Benyó, Z Acute eects of unilateral carotid artery occlusion on the cerebrocortical microcirculation in mice. FRONTIERS IN NEUROSCIENCE 2010 Paper: 2010.10.00232 (2010) DOI Közlemény:1856291 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

118. Iring, A ; Horvath, B ; Benyo, B ; Tillson, M ; Bakken, B ; Benyo, Z Inuence of unilateral carotid artery occlusion on the cerebrocortical microcirculation in mice: a laser-speckle study ACTA PHYSIOLOGICA HUNGARICA 97 : 4 pp. 447-447. , 1 p. (2010) WoS Közlemény:1465954 Jóváhagyott Forrás Folyóiratcikk (Absztrakt / Kivonat )

119. Lenzsér, G ; Horváth, B ; Iring, A ; Hermán, P ; Komjáti, K ; Sándor, P ; Benyó, Z Thromboxane receptor-mediated vasomotion and cerebrocortical blood ow oscillations in NO-deciency. FRONTIERS IN NEUROSCIENCE 2010 Paper: 2010.10.00026 (2010) DOI Közlemény:1856294 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

120. Nemeth, T ; Wirth, A ; Ruisanchez, E ; Oermanns, S ; Benyo, Z G protein mediated signaling pathways of thromboxane A2 in the vascular smooth muscle EUROPEAN HEART JOURNAL 31 : S1 pp. 702-702. , 1 p. (2010) WoS Közlemény:1626625 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

121. Lacza, Z ; Hortobágyi, L ; Horváth, B ; Horváth, E M ; Sándor, P ; Benyó, Z Additive eect of cyclooxygenase and nitric oxide synthase blockade on the cerebrocortical microcirculation NEUROREPORT 20 : 11 pp. 1027-1031. , 5 p. (2009) DOI WoS Scopus PubMed Google scholar Google scholar hash Közlemény:1475866 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 4 Független: 3 Függő: 1 Idézett közlemények száma: 10

1. Toth P et al. Isolated human and rat cerebral arteries constrict to increases in ow: role of 20-HETE and TP receptors. (2011) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271- 678X 1559-7016 31 10 2096-2105 2. Vuletic Vladimira et al. Eect of indomethacin on cerebrovascular reactivity in patients with type 2 diabetes mellitus. (2013) DIABETES RESEARCH AND CLINICAL PRACTICE 0168-8227 1872- 8227 101 1 81-87 3. Leger Pierre-Louis et al. Cyclooxygenase-2-Derived Prostaglandins Mediate Cerebral Microcirculation in a Juvenile Ischemic Rat Model. (2016) STROKE 0039-2499 1524-4628 47 12 3048-3052 4. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801

122. Magyar, A ; Orsy, P ; Horvath, B ; Donko, A ; Benyo, Z Vascular eects of sphingolipid mediators ACTA PHYSIOLOGICA HUNGARICA 96 : 1 pp. 98-100. , 3 p. (2009) WoS Közlemény:1626636 Érvényesített Forrás Idéző Folyóiratcikk (Absztrakt / Kivonat ) Idézett közlemények száma: 1

123. Parkanyi, A ; Ruisanchez, E ; Fekete, K ; Horvath, B ; Sandor, P ; Benyo, Z Vascular eects of sphingomyelinase DIABETOLOGIA 52 : Suppl. 1 pp. S515-S516. (2009) WoS Közlemény:1626635 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

124. Ruisanchez, E ; Benko, R ; Leszl-Ishiguro, M ; Lacza, Z ; Jarai, Z ; Sandor, P ; Benyo, Z Cerebrovascular eects of endocannabinoids DIABETOLOGIA 52 : Suppl. 1 pp. S258-S258. (2009) WoS Közlemény:1626634 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

125. Benyo, Z ; Magyar, A ; Orsy, P ; Donko, A ; Horvath, B Role of reactive oxygen species in mediation of sphingomyelinase induced vascular dysfunction DIABETOLOGIA 51 : Suppl. 1 pp. S111-S112. (2008) WoS Közlemény:1628730 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

126. Horvath, B ; Hortobagyi, L ; Lenzser, G ; Schweer, H ; Hrabak, A ; Sandor, P ; Benyo, Z Carbon monoxide-prostaglandin E-2 interaction in the hypothalamic circulation NEUROREPORT 19 : 16 pp. 1601-1604. , 4 p. (2008) DOI WoS Scopus PubMed Közlemény:1151561 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 5 Független: 2 Függő: 3 Idézett közlemények száma: 5

1. * Lacza Z et al. Additive eect of cyclooxygenase and nitric oxide synthase blockade on the cerebrocortical microcirculation. (2009) NEUROREPORT 0959-4965 20 11 1027-1031 2. * Iring A et al. Role of endocannabinoids and cannabinoid-1 receptors in cerebrocortical blood ow regulation. (2013) PLOS ONE 1932-6203 1932-6203 8 1 3. Katada K et al. Therapeutic roles of carbon monoxide in intestinal ischemia-reperfusion injury. (2015) JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY 0815-9319 1440-1746 30 Suppl. 1 46-52 4. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801 5. Robison Christopher et al. Increased expression of carbon monoxide-producing enzymes in the MPOA after sexual experience in male rats. (2017) PHYSIOLOGY & BEHAVIOR 0031- 9384 171 149-157

127. Wirth, A ; Benyo, Z ; Lukasova, M ; Leutgeb, B ; Wettschureck, N ; Gorbey, S ; Orsy, P ; Horvath, B ; Maser-Gluth, C ; Greiner, E et al. G(12)-G(13)-LARG-mediated signaling in vascular smooth muscle is required for salt-induced hypertension NATURE MEDICINE 14 : 1 pp. 64-68. , 5 p. (2008) DOI WoS Scopus PubMed Közlemény:1628738 Érvényesített Forrás Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 354 Független: 296 Függő: 58

1. Ohtsu H. Central role of G(q) in the hypertrophic signal transduction of angiotensin II in vascular smooth muscle cells. (2008) ENDOCRINOLOGY 0013-7227 1945-7170 149 7 3569-3575 2. * Wirth A. G(12)-G(13)-LARG-mediated signaling in vascular smooth muscle is required for salt-induced hypertension (vol 14, pg 64, 2008). (2008) NATURE MEDICINE 1078-8956 1546-170X 14 2 222- 222 3. * Worzfeld T. G(12)/G(13)-mediated signalling in mammalian physiology and disease. (2008) TRENDS IN PHARMACOLOGICAL SCIENCES 0165-6147 29 11 582-589 4. * Wirth A. G-protein-mediated signalling in salt-dependent hypertension. (2008) DRUG DISCOVERY TODAY: DISEASE MECHANISMS 1740-6765 5 3-4 e307-e314 5. Foley JF. Pass on the salt. (2008) SCIENCE SIGNALING 1945-0877 1937-9145 1 3 6. Riise J et al. Prostanoid F receptors elicit an inotropic eect in rat left ventricle by enhancing myosin light chain phosphorylation. (2008) CARDIOVASCULAR RESEARCH 0008-6363 1755-3245 80 3 407-415 7. * Gavard J. Protein Kinase C-related Kinase and ROCK Are Required for Thrombin-induced Endothelial Cell Permeability Downstream from G alpha(12/13) and G alpha(11/q). (2008) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 283 44 29888- 29896 8. Schoner W. Salt abuse: the path to hypertension. (2008) NATURE MEDICINE 1078-8956 1546-170X 14 1 16-17 9. Ziomber A. Sodium-, potassium-, chloride-, and bicarbonate- related eects on blood pressure and electrolyte homeostasis in deoxycorticosterone acetate-treated rats. (2008) AMERICAN JOURNAL OF PHYSIOLOGY: RENAL PHYSIOLOGY 1931-857X 295 6 F1752-F1763 10. Eguchi S. Triple twist theory of Rho inhibition by the angiotensin II type 2 receptor. (2008) CIRCULATION RESEARCH 0009-7330 1524- 4571 102 10 1143-1145 11. Yang E et al. 17 beta-Estradiol attenuates vascular contraction through inhibition of RhoA/Rho kinase pathway. (2009) NAUNYN- SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 0028-1298 1432-1912 380 1 35-44 12. Ito K. Acquisition of Brain Na Sensitivity Contributes to Salt- Induced Sympathoexcitation and Cardiac Dysfunction in Mice With Pressure Overload. (2009) CIRCULATION RESEARCH 0009- 7330 1524-4571 104 8 1004-U200 13. Suzuki N. Activation of Leukemia-associated RhoGEF by G alpha(13) with Signicant Conformational Rearrangements in the Interface. (2009) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 284 8 5000-5009 14. * Korhonen H. Anaphylactic shock depends on endothelial G(q)/G(11). (2009) JOURNAL OF EXPERIMENTAL MEDICINE 0022- 1007 1540-9538 206 2 411-420 15. Bregeon J. Angiotensin II induces RhoA activation through SHP2- dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells. (2009) AMERICAN JOURNAL OF PHYSIOLOGY: CELL PHYSIOLOGY 0363-6143 1522-1563 297 5 C1062-C1070 16. Kimura K. Angiotensin II type-1 receptor regulates RhoA and Rho- kinase/ROCK activation via multiple mechanisms. Focus on "Angiotensin II induces RhoA activation through SHP2-dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells". (2009) AMERICAN JOURNAL OF PHYSIOLOGY: CELL PHYSIOLOGY 0363-6143 1522-1563 297 5 C1059-C1061 17. Suzuki H. Distinct Roles of Protease-Activated Receptors in Signal Transduction Regulation of Endothelial Nitric Oxide Synthase. (2009) HYPERTENSION 0194-911X 53 2 182-U139 18. Suzuki H et al. Endothelial Nitric Oxide Synthase Inhibits G(12/13) and Rho-Kinase Activated by the Angiotensin II Type-1 Receptor Implication in Vascular Migration. (2009) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 29 2 217-224 19. Baek I. Flavone Attenuates Vascular Contractions by Inhibiting RhoA/Rho Kinase Pathway. (2009) KOREAN JOURNAL OF PHYSIOLOGY & PHARMACOLOGY 1226-4512 2093-3827 13 3 201- 207 20. Evelyn CR. High-Throughput Screening for Small-Molecule Inhibitors of LARG-Stimulated RhoA Nucleotide Binding via a Novel Fluorescence Polarization Assay. (2009) JOURNAL OF BIOMOLECULAR SCREENING 1087-0571 1552-454X 14 2 161-172 21. Middeke M. Hypertensiology 2009. (2009) DEUTSCHE MEDIZINISCHE WOCHENSCHRIFT 0012-0472 1439-4413 134 25-26 1312-1315 22. * Sprossmann F. Inducible knockout mutagenesis reveals compensatory mechanisms elicited by constitutive BK channel deciency in overactive murine bladder. (2009) FEBS JOURNAL 1742-464X 1742-4658 276 6 1680-1697 23. Beetz N. Phosducin inuences sympathetic activity and prevents stress-induced hypertension in humans and mice. (2009) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 119 12 3597-3612 24. Suzuki N. Regulation and Physiological Functions of G12/13- Mediated Signaling Pathways. (2009) NEUROSIGNALS 1424-862X 1424-8638 17 1 55-70 25. Siehler S. Regulation of RhoGEF proteins by G12/13-coupled receptors. (2009) BRITISH JOURNAL OF PHARMACOLOGY 0007- 1188 1476-5381 158 1 41-49 26. Puetz Sandra et al. Regulation of Smooth Muscle Contraction by Small GTPases. (2009) PHYSIOLOGY 1548-9213 1548-9221 24 6 342-356 27. Juneja J. Role of G12 proteins in oncogenesis and metastasis. (2009) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476- 5381 158 1 32-40 28. Ponsioen B. Spatiotemporal Regulation of Chloride Intracellular Channel Protein CLIC4 by RhoA. (2009) MOLECULAR BIOLOGY OF THE CELL 1059-1524 1939-4586 20 22 4664-4672 29. Blaustein MP. The Pump, the Exchanger, and Endogenous Ouabain Signaling Mechanisms That Link Salt Retention to Hypertension. (2009) HYPERTENSION 0194-911X 53 2 291-U341 30. Wynne BM. Vascular smooth muscle cell signaling mechanisms for contraction to angiotensin II and endothelin-1. (2009) JOURNAL OF THE AMERICAN SOCIETY OF HYPERTENSION 1933- 1711 1878-7436 3 2 84-95 31. * Ahmed K. An Autocrine Lactate Loop Mediates Insulin-Dependent Inhibition of Lipolysis through GPR81. (2010) CELL METABOLISM 1550-4131 11 4 311-319 32. Yang E. Calcium Sensitization Induced by Sodium Fluoride in Permeabilized Rat Mesenteric Arteries. (2010) KOREAN JOURNAL OF PHYSIOLOGY & PHARMACOLOGY 1226-4512 2093-3827 14 1 51-57 33. Van Agtmael. Col4a1 mutation in mice causes defects in vascular function and low blood pressure associated with reduced red blood cell volume. (2010) HUMAN MOLECULAR GENETICS 0964- 6906 1460-2083 19 6 1119-1128 34. Tsang S. Deregulation of RGS2 in cardiovascular diseases. (2010) FRONTIERS IN BIOSCIENCE (SCHOLAR EDITION) 1945-0516 1945- 0524 2 S 2 547-557 35. Song MJ. Eects of 3',4'-dihydroxyavonol on vascular contractions of rat aortic rings. (2010) CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY 0305-1870 1440-1681 37 8 803-810 36. * Oermanns S. G protein functions identied using genetic mouse models. (2010) Megjelent: G Protein-Coupled Receptors: Structure, Signaling, and Physiology pp. 125-144 37. Nishida M. et al. Heterotrimeric g proteins in heart failure. (2010) Megjelent: Heart Failure: Symptoms, Causes and Treatment Options pp. 51-72 38. * Horvath B et al. Hypersensitivity to thromboxane receptor mediated cerebral vasomotion and CBF oscillations during acute NO-deciency in rats.. (2010) PLOS ONE 1932-6203 1932-6203 5 12 39. Zhang J. In vivo assessment of artery smooth muscle [Ca2+](i) and MLCK activation in FRET-based biosensor mice. (2010) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 299 3 H946-H956 40. * Oermanns S. In vivo functions of heterotrimeric g proteins. (2010) Megjelent: Handbook of Cell Signaling, 2/e pp. 1621-1627 41. Zhang J. Knockout of Na+/Ca2+ exchanger in smooth muscle attenuates vasoconstriction and L-type Ca2+ channel current and lowers blood pressure. (2010) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 298 5 H1472-H1483 42. * Albinsson S. MicroRNAs Are Necessary for Vascular Smooth Muscle Growth, Dierentiation, and Function. (2010) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 30 6 1118-U80 43. Nishida M. Molecular mechanism underlying the development of heart failure mediated by heterotrimeric G protein signaling. (2010) FOLIA PHARMACOLOGICA JAPONICA 0015-5691 135 5 179- 183 44. * Wang L. Mutations in Myosin Light Chain Kinase Cause Familial Aortic Dissections. (2010) AMERICAN JOURNAL OF HUMAN GENETICS 0002-9297 1537-6605 87 5 701-707 45. * Hanson J. Nicotinic acid- and monomethyl fumarate-induced ushing involves GPR109A expressed by keratinocytes and COX- 2-dependent prostanoid formation in mice. (2010) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 120 8 2910-2919 46. Prager-Khoutorsky M. Osmosensation in vasopressin neurons: changing actin density to optimize function. (2010) TRENDS IN NEUROSCIENCES 0166-2236 33 2 76-83 47. Wuertz CM et al. p63RhoGEF - A key mediator of angiotensin II- dependent signaling and processes in vascular smooth muscle cells. (2010) FASEB JOURNAL 0892-6638 1530-6860 24 12 4865- 4876 48. Carretero-Ortega J. Phosphatidylinositol 3,4,5-Triphosphate- Dependent Rac Exchanger 1 (P-Rex-1), a Guanine Nucleotide Exchange Factor for Rac, Mediates Angiogenic Responses to Stromal Cell-Derived Factor-1/Chemokine Stromal Cell Derived Factor-1 (SDF-1/CXCL-12) Linked to Rac Activation, Endothelial Cell Migration, and in Vitro Angiogenesis. (2010) MOLECULAR PHARMACOLOGY 0026-895X 1521-0111 77 3 435-442 49. Gurley SB. Renal Actions of RGS2 Control Blood Pressure. (2010) JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY 1046- 6673 1533-3450 21 11 1847-1851 50. * Vázquez-Prado J. RGS-RhoGEFs and other RGS multidomain proteins as eector molecules in GPCR-dependent and GPCR- independent cell signaling. (2010) Megjelent: G Protein-Coupled Receptors: Structure, Signaling, and Physiology pp. 159-188 51. * Wirth A. Rho kinase and hypertension. (2010) BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE 0925-4439 1879-260X 1802 12 1276-1284 52. Miano JM. Role of serum response factor in the pathogenesis of disease. (2010) LABORATORY INVESTIGATION 0023-6837 1530- 0307 90 9 1274-1284 53. Blaustein MP. Signaling mechanisms that link salt retention to hypertension: Endogenous ouabain, the Na+ pump, the Na+/Ca2+ exchanger and TRPC proteins. (2010) BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE 0925-4439 1879-260X 1802 12 1219-1229 54. Hajicek N. Signaling through Galpha12/13 and RGS-RhoGEFs. (2010) Megjelent: The Rho GTPases in Cancer pp. 59-76 55. * Groneberg D. Smooth Muscle-Specic Deletion of Nitric Oxide- Sensitive Guanylyl Cyclase Is Sucient to Induce Hypertension in Mice. (2010) CIRCULATION 0009-7322 1524-4539 121 3 401-U91 56. Medlin MD. Sphingosine 1-Phosphate Receptor 2 Signals Through Leukemia-Associated RhoGEF (LARG), to Promote Smooth Muscle Cell Dierentiation. (2010) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 30 9 1779-1786 57. Aittaleb M. Structure and Function of Heterotrimeric G Protein- Regulated Rho Guanine Nucleotide Exchange Factors. (2010) MOLECULAR PHARMACOLOGY 0026-895X 1521-0111 77 2 111- 125 58. Loirand G. The exchange factor Arhgef1: a new target in hypertension. (2010) M S MEDECINE SCIENCE 0767-0974 1958- 5381 26 6-7 561-563 59. Haba M. The Modulation of Vascular ATP-Sensitive K+ Channel Function via the Phosphatidylinositol 3-Kinase-Akt Pathway Activated by Phenylephrine. (2010) JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS 0022-3565 1521-0103 334 2 673-678 60. * Guilluy C. The Rho exchange factor Arhgef1 mediates the eects of angiotensin II on vascular tone and blood pressure. (2010) NATURE MEDICINE 1078-8956 1546-170X 16 2 183-U89 61. Toumaniantz G. The Rho protein exchange factor Vav3 regulates vascular smooth muscle cell proliferation and migration. (2010) CARDIOVASCULAR RESEARCH 0008-6363 1755-3245 86 1 131-140 62. Loirand G. The role of rho protein signaling in hypertension. (2010) NATURE REVIEWS CARDIOLOGY 1759-5002 1759-5010 7 11 637-647 63. Willard MD et al. The superfamily of "regulator of g-protein signaling" (rgs) proteins. (2010) Megjelent: Handbook of Cell Signaling, 2/e pp. 1683-1703 64. Savoia C et al. Angiotensin II and the vascular phenotype in hypertension. (2011) EXPERT REVIEWS IN MOLECULAR MEDICINE 1462-3994 13 65. Gurley SB et al. AT(1A) Angiotensin Receptors in the Renal Proximal Tubule Regulate Blood Pressure. (2011) CELL METABOLISM 1550-4131 13 4 469-475 66. Campbell Norm. Canadian Initiatives to Prevent Hypertension by Reducing Dietary Sodium. (2011) NUTRIENTS 2072-6643 2072- 6643 3 8 756-764 67. Aurelie NDC. Cell Signaling of Angiotensin II on Vascular Tone: Novel Mechanisms. (2011) CURRENT HYPERTENSION REPORTS 1522-6417 1534-3111 13 2 122-128 68. Seok YM. Eects of gomisin A on vascular contraction in rat aortic rings. (2011) NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 0028-1298 1432-1912 383 1 45-56 69. Seok YM et al. attenuates vascular contraction through the inhibition of the RhoA/Rho-kinase signalling pathway in rat aortic rings. (2011) JOURNAL OF PHARMACY AND PHARMACOLOGY 0022-3573 2042-7158 63 9 1244-1251 70. Bastin G. Intracellular regulation of heterotrimeric G-protein signaling modulates vascular smooth muscle cell contraction. (2011) ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS 0003-9861 1096-0384 510 2 182-189 71. Balakumar P. Multifaceted cardiac signal transduction mediated by G protein-coupled receptors: Potential target sites where an unambiguous attention is needed for exploring new drugs for cardiovascular disorders. (2011) Biomedicine and Aging Pathology 2210-5220 1 4 197-202 72. Groneberg D. Nitric Oxide-Sensitive Guanylyl Cyclase Is Dispensable for Nitrergic Signaling and Gut Motility in Mouse Intestinal Smooth Muscle. (2011) GASTROENTEROLOGY 0016- 5085 1528-0012 140 5 1608-1617 73. Momotani K et al. P63RhoGEF couples G αq/11-mediated signaling to Ca 2+ sensitization of vascular smooth muscle contractility. (2011) CIRCULATION RESEARCH 0009-7330 1524- 4571 109 9 993-1002 74. Surma M. Rho kinase as a therapeutic target in cardiovascular disease. (2011) FUTURE CARDIOLOGY 1479-6678 1744-8298 7 5 657-671 75. He WQ. Role of myosin light chain kinase in regulation of basal blood pressure and maintenance of salt-induced hypertension. (2011) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 301 2 H584- H591 76. * Lob HE. Role of Vascular Extracellular Superoxide Dismutase in Hypertension. (2011) HYPERTENSION 0194-911X 58 2 232-U213 77. * Nemeno RA. SDF-1 alpha Induction in Mature Smooth Muscle Cells by Inactivation of PTEN Is a Critical Mediator of Exacerbated Injury-Induced Neointima Formation. (2011) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 31 6 1300-U138 78. Kozasa Tohru. Signalling mechanisms of RhoGTPase regulation by the heterotrimeric G proteins G12 and G13. (2011) JOURNAL OF BIOCHEMISTRY 0021-924X 150 4 357-369 79. * Albinsson S. Smooth Muscle miRNAs Are Critical for Post-Natal Regulation of Blood Pressure and Vascular Function. (2011) PLOS ONE 1932-6203 1932-6203 6 4 80. Cox Brian. Translational Analysis of Mouse and Human Placental Protein and mRNA Reveals Distinct Molecular Pathologies in Human Preeclampsia. (2011) MOLECULAR & CELLULAR PROTEOMICS 1535-9476 1535-9484 10 12 p. $ 81. Coman Thomas. Under pressure: the search for the essential mechanisms of hypertension. (2011) NATURE MEDICINE 1078- 8956 1546-170X 17 11 1402-1409 82. Bergaya Sonia. WNK1 Regulates Vasoconstriction and Blood Pressure Response to alpha(1)-Adrenergic Stimulation in Mice. (2011) HYPERTENSION 0194-911X 58 3 439-U223 83. Chiu Wei-chiao. Angiotensin II regulates the LARG/RhoA/MYPT1 axis in rat vascular smooth muscle in vitro. (2012) ACTA PHARMACOLOGICA SINICA 1671-4083 1745-7254 33 12 1502- 1510 84. Troidl K. Arteriogenesis. Genetic model for understanding the molecular mechanisms. (2012) GEFAESSCHIRURGIE 0948-7034 17 8 727-730 85. * Tunaru Sorin. Castor oil induces laxation and uterus contraction via ricinoleic acid activating prostaglandin EP3 receptors. (2012) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 0027-8424 1091-6490 109 23 9179- 9184 86. Pelham Christopher. Cullin-3 Regulates Vascular Smooth Muscle Function and Arterial Blood Pressure via PPAR gamma and RhoA/Rho-Kinase. (2012) CELL METABOLISM 1550-4131 16 4 462- 472 87. Chiu Wei-Chiao. Dierential baseline expression and angiotensin II-stimulation of leukemia-associated RhoGEF in vascular smooth muscle cells of spontaneously hypertensive rats. (2012) INTERNATIONAL JOURNAL OF NANOMEDICINE 1176-9114 1178- 2013 7 5929-5939 88. Tang Zhenyu. Dierentiation of multipotent vascular stem cells contributes to vascular diseases. (2012) NATURE COMMUNICATIONS 2041-1723 3 89. Seok Young. Eects of on vascular contraction in rat aortic rings. (2012) CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY 0305-1870 1440-1681 39 1 28-36 90. Kauenstein Gilles. Emerging role of G protein-coupled receptors in microvascular myogenic tone. (2012) CARDIOVASCULAR RESEARCH 0008-6363 1755-3245 95 2 223-232 91. Rautureau Yohann. Endothelin in hypertension: an update. (2012) CURRENT OPINION IN NEPHROLOGY AND HYPERTENSION 1062- 4821 1473-6543 21 2 128-136 92. Chen Songcang. Essential hypertension: perspectives and future directions. (2012) JOURNAL OF HYPERTENSION 0263-6352 1473- 5598 30 1 42-45 93. * Tappe-Theodor A et al. Galpha(q/11) signaling tonically modulates nociceptor function and contributes to activity-dependent sensitization. (2012) PAIN 0304-3959 153 1 184-196 94. * Wirth A et al. G-protein-coupled receptors in smooth muscle. (2012) Megjelent: Muscle pp. 1145-1153 95. Blaustein Mordecai. How NaCl raises blood pressure: a new paradigm for the pathogenesis of salt-dependent hypertension. (2012) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 302 5 H1031- H1049 96. Heximer SP. Intracellular regulation of heterotrimeric G-protein signaling in normal and diseased tissues of the cardiovascular system. (2012) DRUG DISCOVERY TODAY: DISEASE MODELS 1740- 6757 9 3 e91-e96 97. Nanda Vivek. Leiomodin 1, a New Serum Response Factor- dependent Target Gene Expressed Preferentially in Dierentiated Smooth Muscle Cells. (2012) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 287 4 2459-2467 98. Turczynska Karolina. MicroRNAs Are Essential for Stretch-induced Vascular Smooth Muscle Contractile Dierentiation via MicroRNA (miR)-145-dependent Expression of L-type Calcium Channels. (2012) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083- 351X 287 23 19199-19206 99. Momotani K et al. P63RhoGEF: A New Switch for G q-Mediated Activation of Smooth Muscle. (2012) TRENDS IN CARDIOVASCULAR MEDICINE 1050-1738 22 5 122-127 100. * Altho Till. Procontractile G protein-mediated signaling pathways antagonistically regulate smooth muscle dierentiation in vascular remodeling. (2012) JOURNAL OF EXPERIMENTAL MEDICINE 0022-1007 1540-9538 209 12 2277-2290 101. * Greif Daniel. Radial Construction of an Arterial Wall. (2012) DEVELOPMENTAL CELL 1534-5807 1878-1551 23 3 482-493 102. Osei-Owusu Patrick. Regulator of G Protein Signaling 2 Deciency Causes Endothelial Dysfunction and Impaired Endothelium- derived Hyperpolarizing Factor-mediated Relaxation by Dysregulating G(i/o) Signaling. (2012) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 287 15 12541-12549 103. Miller Nichol. Rgnef (p190RhoGEF) Knockout Inhibits RhoA Activity, Focal Adhesion Establishment, and Cell Motility Downstream of Integrins. (2012) PLOS ONE 1932-6203 1932-6203 7 5 104. Cario-Toumaniantz Chrystelle. RhoA guanine exchange factor expression prole in arteries: evidence for a Rho kinase- dependent negative feedback in angiotensin II-dependent hypertension. (2012) AMERICAN JOURNAL OF PHYSIOLOGY: CELL PHYSIOLOGY 0363-6143 1522-1563 302 9 C1394-C1404 105. Ma Mingming. Rho-kinase in hypertension and its relationship with volume regulated chloride channel. (2012) CHINESE PHARMACOLOGICAL BULLETIN 1001-1978 28 6 741-744 106. Seeland U et al. Sex and gender dierences in cardiovascular drug therapy. (2012) HANDBOOK OF EXPERIMENTAL PHARMACOLOGY 0171-2004 214 211-236 107. Kitazawa Toshio. Size-dependent heterogeneity of contractile Ca2+ sensitization in rat arterial smooth muscle. (2012) JOURNAL OF PHYSIOLOGY-LONDON 0022-3751 1469-7793 590 21 5401- 5423 108. Rawlins Emma. The a"MAZE"ing World of Lung-Specic Transgenic Mice. (2012) AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY 1044-1549 1535-4989 46 3 269- 282 109. Chen Daian. The Kidney and Hypertension: Lessons From Mouse Models. (2012) CANADIAN JOURNAL OF CARDIOLOGY 0828-282X 28 3 305-310 110. Mao Xia. Vascular Smooth Muscle Cell Smad4 Gene Is Important for Mouse Vascular Development. (2012) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 32 9 2171-+ 111. * Vaque Jose. A Genome-wide RNAi Screen Reveals a Trio- Regulated Rho GTPase Circuitry Transducing Mitogenic Signals Initiated by G Protein-Coupled Receptors. (2013) MOLECULAR CELL 1097-2765 1097-4164 49 1 94-108 112. * Artamonov MV et al. Agonist-induced Ca2+ sensitization in smooth muscle: Redundancy of Rho guanine nucleotide exchange factors (RhoGEFs) and response kinetics, a caged compound study. (2013) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 288 47 34030-34040 113. Lies Barbara. Correlation of cellular expression with function of NO-sensitive guanylyl cyclase in the murine lower urinary tract. (2013) JOURNAL OF PHYSIOLOGY-LONDON 0022-3751 1469-7793 591 21 5365-5375 114. Gomez Delphine et al. Detection of histone modications at specic gene loci in single cells in histological sections. (2013) NATURE METHODS 1548-7091 1548-7105 10 2 171-177 115. Boedtkjer E. Disturbed acid-base transport: An emerging cause of hypertension. (2013) FRONTIERS IN PHYSIOLOGY 1664-042X 4 DEC 116. Horinouchi Takahiro. Endothelin Receptor Signaling: New Insight Into Its Regulatory Mechanisms. (2013) JOURNAL OF PHARMACOLOGICAL SCIENCES 1347-8613 1347-8648 123 2 85- 101 117. Rautureau Y et al. Endothelin system: Roles in hypertension. (2013) Megjelent: Hypertension pp. 103-115 118. Friebe Andreas. Evidence for Dual Pathway for Nitrergic Neuromuscular Transmission in Doubt: Evidence Favors Lack of Role of ICC Reply. (2013) GASTROENTEROLOGY 0016-5085 1528- 0012 145 5 1161-1161 119. Kitchen Chad. Expression and promoter analysis of a highly restricted integrin alpha gene in vascular smooth muscle. (2013) GENE 0378-1119 1879-0038 513 1 82-89 120. * Jiang Ming. Gpr177 regulates pulmonary vasculature development. (2013) DEVELOPMENT 0950-1991 1477-9129 140 17 3589-3594 121. Derewenda U et al. Identication of quercitrin as an inhibitor of the p90 S6 ribosomal kinase (RSK): structure of its complex with the N-terminal domain of RSK2 at 1.8 angstrom resolution. (2013) ACTA CRYSTALLOGRAPHICA SECTION D-BIOLOGICAL CRYSTALLOGRAPHY 0907-4449 1399-0047 69 266-275 122. Arteaga-Solis Emilio et al. Inhibition of Leptin Regulation of Parasympathetic Signaling as a Cause of Extreme Body Weight- Associated Asthma. (2013) CELL METABOLISM 1550-4131 17 1 35- 48 123. Joseph Biny. Ion channel remodeling in vascular smooth muscle during hypertension: Implications for novel therapeutic approaches. (2013) PHARMACOLOGICAL RESEARCH 1043-6618 1096-1186 70 1 126-138 124. Salmon M. KLF4 regulates abdominal aortic aneurysm morphology and deletion attenuates aneurysm formation. (2013) CIRCULATION 0009-7322 1524-4539 128 SUPPL.1 S163-S174 125. Zhang J. New insights into the contribution of arterial NCX to the regulation of myogenic tone and blood pressure. (2013) ADVANCES IN EXPERIMENTAL MEDICINE AND BIOLOGY 0065- 2598 961 329-343 126. * Mikelis Constantinos. PDZ-RhoGEF and LARG Are Essential for Embryonic Development and Provide a Link between Thrombin and LPA Receptors and Rho Activation. (2013) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 288 17 12232- 12243 127. * Marchesi Chiara et al. Protective role of vascular smooth muscle cell PPAR gamma in angiotensin II-induced vascular disease. (2013) CARDIOVASCULAR RESEARCH 0008-6363 1755-3245 97 3 562-570 128. Takefuji Mikito. RhoGEF-mediated vasoconstriction in hypertension. (2013) HYPERTENSION RESEARCH 0916-9636 1348- 4214 36 11 930-931 129. Kather Jakob. Rho guanine exchange factors in blood vessels: Fine-tuners of angiogenesis and vascular function. (2013) EXPERIMENTAL CELL RESEARCH 0014-4827 1090-2422 319 9 1289-1297 130. Amin Ehsan. Rho-kinase: regulation, (dys) function, and inhibition. (2013) BIOLOGICAL CHEMISTRY 1431-6730 1437-4315 394 11 1399-1410 131. Shi J. Rho kinases in cardiovascular physiology and pathophysiology: The eect of fasudil. (2013) JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 0160-2446 1533-4023 62 4 341-354 132. Nagase Miki. Role of Rac1 GTPase in salt-sensitive hypertension. (2013) CURRENT OPINION IN NEPHROLOGY AND HYPERTENSION 1062-4821 1473-6543 22 2 148-155 133. Gao Ning. Signaling through Myosin Light Chain Kinase in Smooth Muscles. (2013) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 288 11 7596-7605 134. Loirand Gervaise. SMALL G PROTEINS IN THE CARDIOVASCULAR SYSTEM: PHYSIOLOGICAL AND PATHOLOGICAL ASPECTS. (2013) PHYSIOLOGICAL REVIEWS 0031-9333 1522-1210 93 4 1659-1717 135. Nguyen Anh. Smooth Muscle Cell Plasticity Fact or Fiction?. (2013) CIRCULATION RESEARCH 0009-7330 1524-4571 112 1 17-22 136. Su Wen. Smooth muscle-selective CPI-17 expression increases vascular smooth muscle contraction and blood pressure. (2013) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 305 1 H104-H113 137. Waypa Gregory. Superoxide Generated at Mitochondrial Complex III Triggers Acute Responses to Hypoxia in the Pulmonary Circulation. (2013) AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE 1073-449X 1535-4970 187 4 424-432 138. Bai X. The smooth muscle-selective RhoGAP GRAF3 is a critical regulator of vascular tone and hypertension. (2013) NATURE COMMUNICATIONS 2041-1723 4 139. Fouillade Charles. Transcriptome Analysis for Notch3 Target Genes Identies Grip2 as a Novel Regulator of Myogenic Response in the Cerebrovasculature. (2013) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 33 1 76-+ 140. Bae YH. A FAK-Cas-Rac-lamellipodin signaling module transduces extracellular matrix stiness into mechanosensitive cell cycling. (2014) SCIENCE SIGNALING 1945-0877 1937-9145 7 330 141. Cavin S. A-kinase anchoring protein-Lbc promotes pro-brotic signaling in cardiac broblasts. (2014) BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH 0167-4889 1879- 2596 1843 2 335-345 142. Osei-Owusu P. Altered reactivity of resistance vasculature contributes to hypertension in elastin insuciency. (2014) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 306 5 H654-H666 143. Zhou J. An emerging role for Hippo-YAP signaling in cardiovascular development. (2014) JOURNAL OF BIOMEDICAL RESEARCH 1674-8301 1007-4376 28 4 251-254 144. Cognat E. Archetypal Arg169Cys Mutation in NOTCH3 Does Not Drive the Pathogenesis in Cerebral Autosomal Dominant Arteriopathy With Subcortical Infarcts and Leucoencephalopathy via a Loss-of-Function Mechanism. (2014) STROKE 0039-2499 1524-4628 45 3 842-849 145. Gao J. Automated NMR fragment based screening identied a novel interface blocker to the LARG/RhoA complex. (2014) PLOS ONE 1932-6203 1932-6203 9 2 146. Hu Y. Bkca opener, NS1619 pretreatment protects against shock- induced vascular hyporeactivity through PDZ-Rho GEF-RhoA-Rho kinase pathway in rats. (2014) JOURNAL OF TRAUMA AND ACUTE CARE SURGERY 2163-0755 2163-0763 76 2 394-401 147. Chia CY. Breast cancer cell invasion mediated by Gα12 signaling involves expression of interleukins-6 and -8, and matrix metalloproteinase-2. (2014) JOURNAL OF MOLECULAR SIGNALING 1750-2187 9 1 148. Garrido-Martin EM. Common and Distinctive Pathogenetic Features of Arteriovenous Malformations in Hereditary Hemorrhagic Telangiectasia 1 and Hereditary Hemorrhagic Telangiectasia 2 Animal Models-Brief Report. (2014) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 34 10 2232-2236 149. Tsai Ming-Ho et al. Constitutive phosphorylation of myosin phosphatase targeting subunit-1 in smooth muscle. (2014) JOURNAL OF PHYSIOLOGY-LONDON 0022-3751 1469-7793 592 14 3031-3051 150. Maruhashi T. Critical Role of Exogenous Nitric Oxide in ROCK Activity in Vascular Smooth Muscle Cells. (2014) PLOS ONE 1932- 6203 1932-6203 9 10 151. Miyagawa K. Current state of endothelin receptor antagonism in hypertension and pulmonary hypertension. (2014) Therapeutic Advances in Cardiovascular Disease 1753-9447 8 5 202-216 152. Lei Yan. [Cyclosporin A improves the vascular and organ function after traumatic hemorrhagic shock]. (2014) JICHU YIXUE YU LINCHUANG / BASIC AND CLINICAL MEDICINE 1001-6325 34 9 1171-1174 153. Kumar Maya et al. Dening a mesenchymal progenitor niche at single-cell resolution. (2014) SCIENCE 0036-8075 1095-9203 346 6211 154. Heinze C et al. Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.. (2014) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 124 2 675-686 155. Bhattachariya A. Expression of microRNAs is essential for arterial myogenic tone and pressure-induced activation of the PI3- kinase/Akt pathway. (2014) CARDIOVASCULAR RESEARCH 0008- 6363 1755-3245 101 2 288-296 156. Yu Meifang. [Galphaq-Mediated Signaling Pathway Stimulates RGS4 Expression]. (2014) Acta Biophysica Sinica 1000-6737 30 5 336-348 157. * Fabbiano S. Genetic Dissection of the Vav2-Rac1 Signaling Axis in Vascular Smooth Muscle Cells. (2014) MOLECULAR AND CELLULAR BIOLOGY 0270-7306 1098-5549 34 24 4404-4419 158. * Lei XF. Identication of Hic-5 as a Novel Scaold for the MKK4/p54 JNK Pathway in the Development of Abdominal Aortic Aneurysms. (2014) JOURNAL OF THE AMERICAN HEART ASSOCIATION 2047-9980 2047-9980 3 3 159. Lies B. Interstitial cells of Cajal mediate nitrergic inhibitory neurotransmission in the murine gastrointestinal tract. (2014) AMERICAN JOURNAL OF PHYSIOLOGY: GASTROINTESTINAL AND LIVER PHYSIOLOGY 0193-1857 1522-1547 307 1 G98-G106 160. Loirand G. et al. Involvement of Rho GTPases and their regulators in the pathogenesis of hypertension. (2014) SMALL GTPASES 2154-1248 2154-1256 5 4 161. Loirand G. Involvement of Rho GTPases and their regulators in the pathogenesis of hypertension. (2014) SMALL GTPASES 2154- 1248 2154-1256 5 MAY 162. Schumacker PT. Mitochondria in lung biology and pathology: more than just a powerhouse. (2014) AMERICAN JOURNAL OF PHYSIOLOGY: LUNG CELLULAR AND MOLECULAR PHYSIOLOGY 1040-0605 1522-1504 306 11 L962-L974 163. Qiao Yan-Ning et al. Myosin Phosphatase Target Subunit 1 (MYPT1) Regulates the Contraction and Relaxation of Vascular Smooth Muscle and Maintains Blood Pressure. (2014) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 289 32 22512- 22523 164. Reho JJ et al. Neural programming of mesenteric and renal arteries. (2014) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 307 4 H563-H573 165. * Herring BP. Previously dierentiated medial vascular smooth muscle cells contribute to neointima formation following vascular injury. (2014) VASCULAR CELL 2045-824X 6 1 166. Ball MK. Regulation of hypoxia-induced pulmonary hypertension by vascular smooth muscle hypoxia-inducible factor-1α. (2014) AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE 1073-449X 1535-4970 189 3 314-324 167. Valdez CN. Retinal Microangiopathy in a Mouse Model of Inducible Mural Cell Loss. (2014) AMERICAN JOURNAL OF PATHOLOGY 0002-9440 1525-2191 184 10 2618-2626 168. Van Buul. Rho GAPs and GEFs: Controling switches in endothelial cell adhesion. (2014) CELL ADHESION & MIGRATION 1933-6918 1933-6926 8 2 108-124 169. Miller NLG. RhoGEFs in cell motility: Novel links between Rgnef and focal adhesion kinase. (2014) CURRENT MOLECULAR MEDICINE 1566-5240 14 2 221-234 170. Cook DR. Rho guanine nucleotide exchange factors: Regulators of Rho GTPase activity in development and disease. (2014) ONCOGENE 0950-9232 1476-5594 33 31 4021-4035 171. * André G et al. Smooth muscle specic rac1 deciency induces hypertension by preventing p116RIP3-dependent RhoA Inhibition. (2014) JOURNAL OF THE AMERICAN HEART ASSOCIATION 2047- 9980 2047-9980 3 3 172. Schleifenbaum J. Stretch-activation of angiotensin II Type 1a receptors contributes to the myogenic response of mouse mesenteric and renal arteries. (2014) CIRCULATION RESEARCH 0009-7330 1524-4571 115 2 263-272 173. Albinsson S. Stretch-Dependent Smooth Muscle Dierentiation in the Portal Vein-Role of Actin Polymerization, Calcium Signaling, and microRNAs. (2014) MICROCIRCULATION 1073-9688 1549- 8719 21 3 230-238 174. * Li Wei. Tgfbr2 disruption in postnatal smooth muscle impairs aortic wall homeostasis. (2014) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 124 2 755-767 175. * Wong MSK. Vitamin D Promotes Vascular Regeneration. (2014) CIRCULATION 0009-7322 1524-4539 130 12 976-986 176. San Roman et al. Wnt secretion from epithelial cells and subepithelial myobroblasts is not required in the mouse intestinal stem cell niche in vivo. (2014) STEM CELL REPORTS 2213-6711 2213-6711 2 2 127-134 177. Shimokawa Hiroaki et al. 2015 ATVB Plenary Lecture Translational Research on Rho-Kinase in Cardiovascular Medicine. (2015) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 35 8 1756-1769 178. Billaud M. A molecular signature in the pannexin1 intracellular loop confers channel activation by the alpha 1 adrenoreceptor in smooth muscle cells. (2015) SCIENCE SIGNALING 1945-0877 1937- 9145 8 364 179. Hartmann DA. A Murine Toolbox for Imaging the Neurovascular Unit. (2015) MICROCIRCULATION 1073-9688 1549-8719 22 3 168- 182 180. Carbone Maria. Angiotensin II Activates the RhoA Exchange Factor Arhgef1 in Humans. (2015) HYPERTENSION 0194-911X 65 6 1273- 1278 181. Bettaga N. Cell-specic impact of nitric oxide-dependent guanylyl cyclase on arteriogenesis and angiogenesis in mice. (2015) ANGIOGENESIS 0969-6970 1573-7209 18 3 245-254 182. * Sumida T. Complement C1q-induced activation of β-catenin signalling causes hypertensive arterial remodelling. (2015) NATURE COMMUNICATIONS 2041-1723 6 183. * Tang HY. Deciency of Akt1, but not Akt2, attenuates the development of pulmonary hypertension. (2015) AMERICAN JOURNAL OF PHYSIOLOGY: LUNG CELLULAR AND MOLECULAR PHYSIOLOGY 1040-0605 1522-1504 308 2 L208-L220 184. Groneberg D. Dominant role of interstitial cells of Cajal in nitrergic relaxation of murine lower oesophageal sphincter. (2015) JOURNAL OF PHYSIOLOGY-LONDON 0022-3751 1469-7793 593 2 403-414 185. * Szekeres M et al. Endocannabinoid-mediated modulation of G protein-coupled receptor signaling-induced vasoconstriction and hypertension.. (2015) MOLECULAR AND CELLULAR ENDOCRINOLOGY 0303-7207 1872-8057 403 46-56 186. * Rotllan Noemi. Genetic Evidence Supports a Major Role for Akt1 in VSMCs During Atherogenesis. (2015) CIRCULATION RESEARCH 0009-7330 1524-4571 116 11 1744-1752 187. * Altho Till et al. G-protein-mediated signaling in vascular smooth muscle cells - implications for vascular disease. (2015) JOURNAL OF MOLECULAR MEDICINE-JMM 0946-2716 1432-1440 93 9 973- 981 188. Ibeawuchi SRC et al. Hypertension-causing Mutations in Cullin3 Protein Impair RhoA Protein Ubiquitination and Augment the Association with Substrate Adaptors. (2015) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 290 31 19208- 19217 189. Ganss R. Keeping the Balance Right: Regulator of G Protein Signaling 5 in Vascular Physiology and Pathology. (2015) Megjelent: RGS Protein Physiology and Pathophysiology pp. 93- 121 190. Park C et al. Loss of serum response factor induces microRNA- mediated apoptosis in intestinal smooth muscle cells. (2015) CELL DEATH AND DISEASE 2041-4889 6 191. Kondo J. LRIG1 regulates ontogeny of smooth muscle-derived subsets of interstitial cells of cajal in mice. (2015) GASTROENTEROLOGY 0016-5085 1528-0012 149 2 407-419.e8 192. Huang JH. Myocardin is required for maintenance of vascular and visceral smooth muscle homeostasis during postnatal development. (2015) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 0027-8424 1091-6490 112 14 4447-4452 193. Lies Barbara et al. Nitrergic signalling via interstitial cells of Cajal regulates motor activity in murine colon. (2015) JOURNAL OF PHYSIOLOGY-LONDON 0022-3751 1469-7793 593 20 4589-4601 194. Behuliak Michal et al. Ontogenetic changes in contribution of calcium sensitization and calcium entry to blood pressure maintenance of Wistar-Kyoto and spontaneously hypertensive rats. (2015) JOURNAL OF HYPERTENSION 0263-6352 1473-5598 33 12 2443-2454 195. * Wang SP et al. P2Y(2) and G(q)/G(11) control blood pressure by mediating endothelial mechanotransduction. (2015) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 125 8 3077-3086 196. * Retailleau Kevin et al. Piezo1 in Smooth Muscle Cells Is Involved in Hypertension-Dependent Arterial Remodeling. (2015) CELL REPORTS 2211-1247 13 6 1161-1171 197. Hu Jie et al. Postnatal Deletion of the Type II Transforming Growth Factor-beta Receptor in Smooth Muscle Cells Causes Severe Aortopathy in Mice. (2015) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 35 12 2647-2656 198. Reho John. Redox signaling and splicing dependent change in myosin phosphatase underlie early versus late changes in NO vasodilator reserve in a mouse LPS model of sepsis. (2015) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 308 9 H1039-H1050 199. Osei-Owusu P et al. Regulation of Renal Hemodynamics and Function by RGS2. (2015) PLOS ONE 1932-6203 1932-6203 10 7 200. Holobotovskyy Vasyl. Regulator of G protein signaling 5 is a determinant of gestational hypertension and preeclampsia. (2015) SCIENCE TRANSLATIONAL MEDICINE 1946-6234 7 290 201. Pang P. RGS4 inhibits angiotensin II signaling and macrophage localization during renal reperfusion injury independent of vasospasm. (2015) KIDNEY INTERNATIONAL 0085-2538 1523-1755 87 4 771-783 202. * Masuda Masashi et al. Saturated phosphatidic acids mediate saturated fatty acid-induced vascular calcication and lipotoxicity. (2015) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558- 8238 125 12 4544-4558 203. Artamonov MV. Signaling Pathways That Control Rho Kinase Activity Maintain the Embryonic Epicardial Progenitor State. (2015) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083- 351X 290 16 10353-10367 204. Lee MY et al. Smooth Muscle Cell Genome Browser: Enabling the Identication of Novel Serum Response Factor Target Genes. (2015) PLOS ONE 1932-6203 1932-6203 10 8 205. Kleinhenz Jennifer et al. Smooth Muscle-Targeted Overexpression of Peroxisome Proliferator Activated Receptor-gamma Disrupts Vascular Wall Structure and Function. (2015) PLOS ONE 1932- 6203 1932-6203 10 10 206. Climent Montserrat. TGF beta Triggers miR-143/145 Transfer From Smooth Muscle Cells to Endothelial Cells, Thereby Modulating Vessel Stabilization. (2015) CIRCULATION RESEARCH 0009-7330 1524-4571 116 11 1753-1764 207. Hartmann Svenja et al. The Function of Rho-Associated Kinases ROCK1 and ROCK2 in the Pathogenesis of Cardiovascular Disease. (2015) FRONTIERS IN PHARMACOLOGY 1663-9812 6 208. * Yagi K. Therapeutically Targeting Tumor Necrosis Factor- alpha/Sphingosine-1-Phosphate Signaling Corrects Myogenic Reactivity in Subarachnoid Hemorrhage. (2015) STROKE 0039- 2499 1524-4628 46 8 2260-2270 209. Chang Ying-Ju et al. The Rho-guanine nucleotide exchange factor PDZ-RhoGEF governs susceptibility to diet-induced obesity and type 2 diabetes. (2015) ELIFE 2050-084X 4 210. Miano JM et al. The short and long of noncoding sequences in the control of vascular cell phenotypes. (2015) CELLULAR AND MOLECULAR LIFE SCIENCES 1420-682X 1420-9071 72 18 3457- 3488 211. Zheng XX et al. TRA2 beta controls Mypt1 exon 24 splicing in the developmental maturation of mouse mesenteric artery smooth muscle. (2015) AMERICAN JOURNAL OF PHYSIOLOGY: CELL PHYSIOLOGY 0363-6143 1522-1563 308 4 C289-C296 212. Chen Songcang et al. Vitamin D deciency and essential hypertension. (2015) JOURNAL OF THE AMERICAN SOCIETY OF HYPERTENSION 1933-1711 1878-7436 9 11 885-901 213. Masuda Masashi et al. Activating transcription factor-4 promotes mineralization in vascular smooth muscle cells. (2016) JCI INSIGHT 2379-3708 1 18 214. Cherepanova Olga et al. Activation of the pluripotency factor OCT4 in smooth muscle cells is atheroprotective. (2016) NATURE MEDICINE 1078-8956 1546-170X 22 6 657-665 215. * Wirth Angela et al. Age-dependent blood pressure elevation is due to increased vascular smooth muscle tone mediated by G- protein signalling. (2016) CARDIOVASCULAR RESEARCH 0008-6363 1755-3245 109 1 131-140 216. * Retailleau Kevin et al. Arterial Myogenic Activation through Smooth Muscle Filamin A. (2016) CELL REPORTS 2211-1247 14 9 2050-2058 217. Reho John et al. A splice variant of the myosin phosphatase regulatory subunit tunes arterial reactivity and suppresses response to salt loading. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 11 H1715-H1724 218. * Schreier Barbara et al. Consequences of postnatal vascular smooth muscle EGFR deletion on acute angiotensin II action. (2016) CLINICAL SCIENCE 0143-5221 1470-8736 130 1 19-33 219. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801 220. * Wang S et al. Endothelial cation channel PIEZO1 controls blood pressure by mediating ow-induced ATP release.. (2016) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 126 12 4527-4536 221. Houde M et al. Endothelin-1: Biosynthesis, Signaling and Vasoreactivity. (2016) Megjelent: Endothelium pp. 143-175 222. Chappell J et al. Extensive Proliferation of a Subset of Dierentiated, yet Plastic, Medial Vascular Smooth Muscle Cells Contributes to Neointimal Formation in Mouse Injury and Atherosclerosis Models. (2016) CIRCULATION RESEARCH 0009- 7330 1524-4571 119 12 1313-1323 223. Roulis Manolis et al. Fibroblasts and myobroblasts of the intestinal lamina propria in physiology and disease. (2016) DIFFERENTIATION 0301-4681 1432-0436 92 3 116-131 224. Kanisicak O et al. Genetic lineage tracing denes myobroblast origin and function in the injured heart. (2016) NATURE COMMUNICATIONS 2041-1723 7 225. Lin Qingsong et al. IP3 receptors regulate vascular smooth muscle contractility and hypertension. (2016) JCI INSIGHT 2379- 3708 1 17 226. Peterson SM et al. Ligation of the mouse common carotid artery. (2016) Megjelent: Mouse Models of Vascular Diseases pp. 43-68 227. * Albarran-Juarez Julian et al. Lineage tracing of cells involved in atherosclerosis. (2016) ATHEROSCLEROSIS 0021-9150 251 445- 453 228. Ragot H et al. Loss of Notch3 Signaling in Vascular Smooth Muscle Cells Promotes Severe Heart Failure Upon Hypertension. (2016) HYPERTENSION 0194-911X 68 2 392-400 229. Brozovich F et al. Mechanisms of Vascular Smooth Muscle Contraction and the Basis for Pharmacologic Treatment of Smooth Muscle Disorders. (2016) PHARMACOLOGICAL REVIEWS 0031-6997 1521-0081 68 2 476-532 230. Chen Tianji et al. miR-17/20 Controls Prolyl Hydroxylase 2 (PHD2)/Hypoxia-Inducible Factor 1 (HIF1) to Regulate Pulmonary Artery Smooth Muscle Cell Proliferation. (2016) JOURNAL OF THE AMERICAN HEART ASSOCIATION 2047-9980 2047-9980 5 12 231. Mutlu Merve et al. miR-564 acts as a dual inhibitor of PI3K and MAPK signaling networks and inhibits proliferation and invasion in breast cancer. (2016) SCIENTIFIC REPORTS 2045-2322 6 232. Nakanishi N et al. MURC deciency in smooth muscle attenuates pulmonary hypertension. (2016) NATURE COMMUNICATIONS 2041-1723 7 233. Houtz J et al. Neurotrophin Signaling Is Required for Glucose- Induced Insulin Secretion. (2016) DEVELOPMENTAL CELL 1534- 5807 1878-1551 39 3 329-345 234. Evans Ann et al. New Insights into Mechanisms and Functions of Chemokine (C-X-C Motif) Receptor 4 Heteromerization in Vascular Smooth Muscle. (2016) INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 1661-6596 1422-0067 17 6 235. Khandekar Aditi et al. Notch-Mediated Epigenetic Regulation of Voltage-Gated Potassium Currents. (2016) CIRCULATION RESEARCH 0009-7330 1524-4571 119 12 1324-1338 236. Gao Ning et al. Physiological signalling to myosin phosphatase targeting subunit-1 phosphorylation in ileal smooth muscle. (2016) JOURNAL OF PHYSIOLOGY-LONDON 0022-3751 1469-7793 594 12 3209-3225 237. Ye Weiping et al. Preeclampsia is Associated with Decreased Methylation of the GNA12 Promoter. (2016) ANNALS OF HUMAN GENETICS 0003-4800 1469-1809 80 1 7-10 238. Yang Pu et al. Preexisting smooth muscle cells contribute to neointimal cell repopulation at an incidence varying widely among individual lesions. (2016) SURGERY 0039-6060 159 2 602- 612 239. Shen Yan et al. Responses of of adventitial CD34(+) vascular wall- resident stem/progenitor cells and medial smooth muscle cells to carotid injury in rats. (2016) EXPERIMENTAL AND MOLECULAR PATHOLOGY 0014-4800 1096-0945 101 3 332-340 240. Jie Li et al. RGS2 squelches vascular G(i/o) and G(q) signaling to modulate myogenic tone and promote uterine blood ow. (2016) PHYSIOLOGICAL REPORTS 2051-817X 4 2 241. Shimokawa Hiroaki et al. RhoA/Rho-Kinase in the Cardiovascular System. (2016) CIRCULATION RESEARCH 0009-7330 1524-4571 118 2 352-366 242. Evans Roger et al. Role of the kidney in the pathogenesis of hypertension: time for a neo-Guytonian paradigm or a paradigm shift?. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY 0363-6119 1522- 1490 310 3 R217-R229 243. Liao Mingmei et al. Smooth muscle cell-specic Tgfbr1 deciency attenuates neointimal hyperplasia but promotes an undesired vascular phenotype for injured arteries. (2016) PHYSIOLOGICAL REPORTS 2051-817X 4 23 244. Yang Pu et al. Smooth muscle cell-specic Tgfbr1 deciency promotes aortic aneurysm formation by stimulating multiple signaling events. (2016) SCIENTIFIC REPORTS 2045-2322 6 245. Morris R et al. Vasodysfunction That Involves Renal Vasodysfunction, Not Abnormally Increased Renal Retention of Sodium, Accounts for the Initiation of Salt-Induced Hypertension. (2016) CIRCULATION 0009-7322 1524-4539 133 9 881-893 246. Pi Jingjiang et al. A MicroRNA302-367-Erk1/2-Klf2-S1pr1 Pathway Prevents Tumor Growth via Restricting Angiogenesis and Improving Vascular Stability. (2017) CIRCULATION RESEARCH 0009-7330 1524-4571 120 1 85-98 247. Lee S et al. Analysis of CCN protein expression and activities in vasoproliferative retinopathies. (2017) Megjelent: METHODS IN MOLECULAR BIOLOGY pp. 543-556 248. * Liao Mingmei et al. An X-linked Myh11-CreER(T2) mouse line resulting from Y to X chromosome-translocation of the Cre allele. (2017) GENESIS: THE JOURNAL OF GENETICS AND DEVELOPMENT 1526-954X 1526-968X 55 9 249. Bai Xue et al. Blood pressure-associated polymorphism controls ARHGAP42 expression via serum response factor DNA binding. (2017) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558- 8238 127 2 670-680 250. Saddouk FZ et al. Ca2 +/Calmodulin-Dependent Protein Kinase II in Vascular Smooth Muscle. (2017) Megjelent: Vascular Pharmacology, Smooth Muscle pp. 171-202 251. Heusch Gerd. Cardioprotection Is Alive But Remains Enigmatic The Nitric Oxide-Protein Kinases-Mitochondria Signaling Axis. (2017) CIRCULATION 0009-7322 1524-4539 136 24 2356-2358 252. * Kroetsch Jerey et al. Constitutive smooth muscle tumour necrosis factor regulates microvascular myogenic responsiveness and systemic blood pressure. (2017) NATURE COMMUNICATIONS 2041-1723 8 253. Park Arick et al. Decits in Col5a2 Expression Result in Novel Skin and Adipose Abnormalities and Predisposition to Aortic Aneurysms and Dissections. (2017) AMERICAN JOURNAL OF PATHOLOGY 0002-9440 1525-2191 187 10 2300-2311 254. * Zahedi Farima et al. Dicer generates a regulatory microRNA network in smooth muscle cells that limits neointima formation during vascular repair. (2017) CELLULAR AND MOLECULAR LIFE SCIENCES 1420-682X 1420-9071 74 2 359-372 255. Asada Noboru et al. Dierential cytokine contributions of perivascular haematopoietic stern cell niches. (2017) NATURE CELL BIOLOGY 1465-7392 1476-4679 19 3 214-223 256. Jacobsen Kevin et al. Diverse cellular architecture of atherosclerotic plaque derives from clonal expansion of a few medial SMCs. (2017) JCI INSIGHT 2379-3708 2 19 257. Zhao J et al. Dual function for mature vascular smooth muscle cells during arteriovenous stula remodeling. (2017) JOURNAL OF THE AMERICAN HEART ASSOCIATION 2047-9980 2047-9980 6 4 258. Kamato Danielle et al. Ga-q proteins: molecular pharmacology and therapeutic potential. (2017) CELLULAR AND MOLECULAR LIFE SCIENCES 1420-682X 1420-9071 74 8 1379-1390 259. Rautureau Y et al. Generation of a mouse model with smooth muscle cell specic loss of the expression of PPARγ. (2017) Megjelent: METHODS IN MOLECULAR BIOLOGY pp. 381-407 260. Kim Jee. High fat diet confers vascular hyper-contractility against angiotensin II through upregulation of MLCK and CPI-17. (2017) KOREAN JOURNAL OF PHYSIOLOGY & PHARMACOLOGY 1226- 4512 2093-3827 21 1 99-106 261. Sontake Vishwaraj et al. Hsp90 regulation of broblast activation in pulmonary brosis. (2017) JCI INSIGHT 2379-3708 2 4 262. Herring B et al. Inammation and vascular smooth muscle cell dedierentiation following carotid artery ligation. (2017) PHYSIOLOGICAL GENOMICS 1094-8341 1531-2267 49 3 115-126 263. Riascos-Bernal Dario et al. Inhibition of Smooth Muscle beta- Catenin Hinders Neointima Formation After Vascular Injury. (2017) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 37 5 879-888 264. Tykocki NR et al. Inhibition of vascular smooth muscle inward- rectier K+ channels restores myogenic tone in mouse urinary bladder arterioles. (2017) AMERICAN JOURNAL OF PHYSIOLOGY: RENAL PHYSIOLOGY 1931-857X 312 5 F836-F847 265. Koliaraki Vasiliki et al. Mesenchymal Cells in Colon Cancer. (2017) GASTROENTEROLOGY 0016-5085 1528-0012 152 5 964-979 266. McManus David et al. Messenger RNA and MicroRNA transcriptomic signatures of cardiometabolic risk factors. (2017) BMC GENOMICS 1471-2164 18 267. Dahan Diana et al. MicroRNA-Dependent Control of Serotonin- Induced Pulmonary Arterial Contraction. (2017) JOURNAL OF VASCULAR RESEARCH 1018-1172 1423-0135 54 4 246-256 268. Anderson Preston et al. Multipotent Myoepithelial Progenitor Cells Are Born Early during Airway Submucosal Gland Development. (2017) AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY 1044-1549 1535-4989 56 6 716-726 269. Casas Ana et al. NOX4-dependent neuronal autotoxicity and BBB breakdown explain the superior sensitivity of the brain to ischemic damage. (2017) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 0027-8424 1091-6490 114 46 12315-12320 270. Singh Nikhlesh et al. p115 RhoGEF activates the Rac1 GTPase signaling cascade in MCP1 chemokine-induced vascular smooth muscle cell migration and proliferation. (2017) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 292 34 14080- 14091 271. Gao Ning et al. Physiological vs. pharmacological signalling to myosin phosphorylation in airway smooth muscle. (2017) JOURNAL OF PHYSIOLOGY-LONDON 0022-3751 1469-7793 595 19 6231-6247 272. de Carcer Guillermo et al. Plk1 regulates contraction of postmitotic smooth muscle cells and is required for vascular homeostasis. (2017) NATURE MEDICINE 1078-8956 1546-170X 23 8 964-+ 273. Hubert Astrid et al. Selective Deletion of Leptin Signaling in Endothelial Cells Enhances Neointima Formation and Phenocopies the Vascular Eects of Diet-Induced Obesity in Mice. (2017) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 37 9 1683-1697 274. Lee Moon et al. Serum response factor regulates smooth muscle contractility via myotonic dystrophy protein kinases and L-type calcium channels. (2017) PLOS ONE 1932-6203 1932-6203 12 2 275. * Kaur H et al. Single-cell proling reveals heterogeneity and functional patterning of GPCR expression in the vascular system. (2017) NATURE COMMUNICATIONS 2041-1723 8 276. Durgin Brittany et al. Smooth muscle cell-specic deletion of Col15a1 unexpectedly leads to impaired development of advanced atherosclerotic lesions. (2017) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 312 5 H943-H958 277. Fisher SA. Smooth Muscle Phenotypic Diversity: Eect on Vascular Function and Drug Responses. (2017) Megjelent: Vascular Pharmacology, Smooth Muscle pp. 383-415 278. Matthey Michaela et al. Targeted inhibition of G(q) signaling induces airway relaxation in mouse models of asthma. (2017) SCIENCE TRANSLATIONAL MEDICINE 1946-6234 9 407 279. Da Ros et al. Targeting Interleukin-1 beta Protects from Aortic Aneurysms Induced by Disrupted Transforming Growth Factor beta Signaling. (2017) IMMUNITY 1074-7613 1097-4180 47 5 959-+ 280. Angelov Stoyan et al. TGF-beta (Transforming Growth Factor-beta) Signaling Protects the Thoracic and Abdominal Aorta From Angiotensin II-Induced Pathology by Distinct Mechanisms. (2017) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 37 11 2102-2113 281. Ha Se et al. Transcriptome analysis of PDGFR alpha(+) cells identies T-type Ca2+ channel CACNA1G as a new pathological marker for PDGFR alpha(+) cell hyperplasia. (2017) PLOS ONE 1932-6203 1932-6203 12 8 282. Misra Ashish et al. Using In Vivo and Tissue and Cell Explant Approaches to Study the Morphogenesis and Pathogenesis of the Embryonic and Perinatal Aorta. (2017) JOVE-JOURNAL OF VISUALIZED EXPERIMENTS 1940-087X 127 283. Doring Yvonne et al. Vascular CXCR4 Limits Atherosclerosis by Maintaining Arterial Integrity Evidence From Mouse and Human Studies. (2017) CIRCULATION 0009-7322 1524-4539 136 4 388-403 284. Trehan N et al. Vitamin D Deciency, Supplementation, and Cardiovascular Health. (2017) Critical Pathways in Cardiology 1535-282X 16 3 109-118 285. Cotton Jennifer et al. YAP/TAZ and Hedgehog Coordinate Growth and Patterning in Gastrointestinal Mesenchyme. (2017) DEVELOPMENTAL CELL 1534-5807 1878-1551 43 1 35-47.e4 286. Mastop Marieke et al. A FRET-based biosensor for measuring G alpha 13 activation in single cells. (2018) PLOS ONE 1932-6203 1932-6203 13 3 287. Forrester S.J. et al. Angiotensin II signal transduction: An update on mechanisms of physiology and pathophysiology. (2018) PHYSIOLOGICAL REVIEWS 0031-9333 1522-1210 98 3 1627-1738 288. Schwartz S.M. et al. An update on clonality: What smooth muscle cell type makes up the atherosclerotic plaque? [version 1; referees: 2 approved]. (2018) F1000RESEARCH 2046-1402 2046- 1402 7 289. Fong Valerie et al. Arhgef12 drives IL17A-induced airway contractility and airway hyperresponsiveness in mice. (2018) JCI INSIGHT 2379-3708 3 21 290. Nguyen Emily et al. CaMKII (Ca2+/Calmodulin-Dependent Kinase II) in Mitochondria of Smooth Muscle Cells Controls Mitochondrial Mobility, Migration, and Neointima Formation. (2018) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 38 6 1333-1345 291. Voussen B et al. Comparison of nitrergic signaling in circular and longitudinal smooth muscle of murine ileum. (2018) NEUROGASTROENTEROLOGY AND MOTILITY 1350-1925 1365- 2982 30 3 292. * Villahoz Silvia et al. Conditional deletion of Rcan1 predisposes to hypertension-mediated intramural hematoma and subsequent aneurysm and aortic rupture. (2018) NATURE COMMUNICATIONS 2041-1723 9 293. Lyu Qing et al. CRISPR-Cas9-Mediated Epitope Tagging Provides Accurate and Versatile Assessment of MyocardinBrief Report. (2018) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 38 9 2184-2190 294. Berthiaume Andree-Anne et al. Dynamic Remodeling of Pericytes In Vivo Maintains Capillary Coverage in the Adult Mouse Brain. (2018) CELL REPORTS 2211-1247 22 1 8-16 295. Pi Jingjiang et al. Elevated Expression of miR302-367 in Endothelial Cells Inhibits Developmental Angiogenesis via CDC42/CCND1 Mediated Signaling Pathways. (2018) THERANOSTICS 1838-7640 8 6 1511-1526 296. Singh P. et al. Endocannabinoid-mediated modulation of Gq protein-coupled receptor mediates vascular hyporeactivity to nor- adrenaline during polymicrobial sepsis. (2018) POLISH JOURNAL OF PHARMACOLOGY 1230-6002 1734-1140 70 6 1150-1157 297. Dai Zhiyu et al. Endothelial and Smooth Muscle Cell Interaction via FoxM1 Signaling Mediates Vascular Remodeling and Pulmonary Hypertension. (2018) AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE 1073-449X 1535- 4970 198 6 788-802 298. Song Min-Kyoung et al. G alpha 12 regulates osteoclastogenesis by modulating NFATc1 expression. (2018) JOURNAL OF CELLULAR AND MOLECULAR MEDICINE 1582-1838 1582-4934 22 2 849-860 299. Huang Jian et al. Genetic approaches to identify pathological limitations in aortic smooth muscle contraction. (2018) PLOS ONE 1932-6203 1932-6203 13 3 300. Li Yan et al. Genetic Lineage Tracing of Nonmyocyte Population by Dual Recombinases. (2018) CIRCULATION 0009-7322 1524- 4539 138 8 793-805 301. Zou Qing-yun et al. GNA11 dierentially mediates broblast growth factor 2-and vascular endothelial growth factor A-induced cellular responses in human fetoplacental endothelial cells. (2018) JOURNAL OF PHYSIOLOGY-LONDON 0022-3751 1469-7793 596 12 2333-2344 302. Misra Ashish et al. Integrin beta3 regulates clonality and fate of smooth muscle-derived atherosclerotic plaque cells. (2018) NATURE COMMUNICATIONS 2041-1723 9 303. DeLalio Leon et al. Interaction Between Pannexin 1 and Caveolin- 1 in Smooth Muscle Can Regulate Blood Pressure. (2018) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 38 9 2065-2078 304. Gomez D et al. Interleukin-1β has atheroprotective eects in advanced atherosclerotic lesions of mice. (2018) NATURE MEDICINE 1078-8956 1546-170X 24 9 1418-1429 305. Carnevale Daniela et al. Loss of EMILIN-1 Enhances Arteriolar Myogenic Tone Through TGF- (Transforming Growth Factor-)- Dependent Transactivation of EGFR (Epidermal Growth Factor Receptor) and Is Relevant for Hypertension in Mice and Humans. (2018) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 38 10 2484-2497 306. Ramadan A et al. Loss of vascular smooth muscle cell autophagy exacerbates angiotensin II-associated aortic remodeling. (2018) JOURNAL OF VASCULAR SURGERY 0741-5214 1097-6809 68 3 859- 871 307. Beck K et al. Nitrergic signaling via interstitial cells of Cajal and smooth muscle cells inuences circular smooth muscle contractility in murine colon. (2018) NEUROGASTROENTEROLOGY AND MOTILITY 1350-1925 1365-2982 30 6 308. Peterson Sarah et al. Notch2 and Proteomic Signatures in Mouse Neointimal Lesion Formation. (2018) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 38 7 1576-1593 309. Basu Sanchita et al. Notch signaling regulates arterial vasoreactivity through opposing functions of Jagged1 and Dll4 in the vessel wall. (2018) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 315 6 H1835-H1850 310. Bonniaud Philippe et al. Optimising experimental research in respiratory diseases: an ERS statement. (2018) EUROPEAN RESPIRATORY JOURNAL 0903-1936 1399-3003 51 5 311. Good Miranda et al. Pannexin 1 Channels as an Unexpected New Target of the Anti-Hypertensive Drug Spironolactone. (2018) CIRCULATION RESEARCH 0009-7330 1524-4571 122 4 606-615 312. Duennes Sarah et al. Phosphodiesterase 3A expression and activity in the murine vasculature is inuenced by NO-sensitive guanylyl cyclase. (2018) PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY 0031-6768 1432-2013 470 4 693-702 313. Crnkovic Slaven et al. Resident cell lineages are preserved in pulmonary vascular remodeling. (2018) JOURNAL OF PATHOLOGY 0022-3417 1096-9896 244 4 485-498 314. Chen Felicia et al. Retinoic acid signaling is essential for airway smooth muscle homeostasis. (2018) JCI INSIGHT 2379-3708 3 16 315. Bjorling Karl et al. Role of age, Rho-kinase 2 expression, and G protein-mediated signaling in the myogenic response in mouse small mesenteric arteries. (2018) PHYSIOLOGICAL REPORTS 2051- 817X 6 17 316. Lian Xiaoming et al. RXFP1 Receptor Activation by Relaxin-2 Induces Vascular Relaxation in Mice via a G alpha(i2)-Protein/PI3K beta/gamma/Nitric Oxide-Coupled Pathway. (2018) FRONTIERS IN PHYSIOLOGY 1664-042X 9 317. Morikawa Yoshifumi et al. Sibutramine facilitates apoptosis and contraction of aortic smooth muscle cells through elevating production of reactive oxygen species. (2018) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 841 113-121 318. Dai J et al. Smooth muscle cell-specic FoxM1 controls hypoxia- induced pulmonary hypertension. (2018) CELLULAR SIGNALLING 0898-6568 1873-3913 51 119-129 319. Shiozaki Yuji et al. The CDK9-cyclin T1 complex mediates saturated fatty acid-induced vascular calcication by inducing expression of the transcription factor CHOP. (2018) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 293 44 17008- 17020 320. Chang Audrey et al. The dominant protein phosphatase PP1c isoform in smooth muscle cells, PP1c, is essential for smooth muscle contraction. (2018) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 293 43 16677-16686 321. Divorty Nina et al. The Orphan Receptor GPR35 Contributes to Angiotensin II-Induced Hypertension and Cardiac Dysfunction in Mice. (2018) AMERICAN JOURNAL OF HYPERTENSION 0895-7061 1941-7225 1879-1905 31 9 1049-1058 322. * Yin Wenguang et al. The potassium channel KCNJ13 is essential for smooth muscle cytoskeletal organization during mouse tracheal tubulogenesis. (2018) NATURE COMMUNICATIONS 2041- 1723 9 323. Brown Isola et al. Vascular Smooth Muscle Remodeling in Conductive and Resistance Arteries in Hypertension. (2018) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 38 9 1969-1985 324. Sontake Vishwaraj et al. Wilms' tumor 1 drives broproliferation and myobroblast transformation in severe brotic lung disease. (2018) JCI INSIGHT 2379-3708 3 16 325. Zawieja Scott et al. Ano1 mediates pressure-sensitive contraction frequency changes in mouse lymphatic collecting vessels. (2019) JOURNAL OF GENERAL PHYSIOLOGY 0022-1295 151 4 532-554 326. Meleka Matthew et al. Anti-hypertensive mechanisms of cyclic depsipeptide inhibitor ligands for G(q/11) class G proteins. (2019) PHARMACOLOGICAL RESEARCH 1043-6618 1096-1186 141 264- 275 327. Amin Fakhra et al. An Update on the Association of Protein Kinases with Cardiovascular Diseases. (2019) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873-4286 25 2 174-183 328. Wirka Robert et al. Atheroprotective roles of smooth muscle cell phenotypic modulation and the TCF21 disease gene as revealed by single-cell analysis. (2019) NATURE MEDICINE 1078-8956 1546- 170X 25 8 1280-+ 329. Lubomirov Lubomir et al. Augmented contractility of murine femoral arteries in a streptozotocin diabetes model is related to increased phosphorylation of MYPT1. (2019) PHYSIOLOGICAL REPORTS 2051-817X 7 3 330. Zhuang Tao et al. Cell-Specic Eects of GATA (GATA Zinc Finger Transcription Factor Family)-6 in Vascular Smooth Muscle and Endothelial Cells on Vascular Injury Neointimal Formation. (2019) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 39 5 888-901 331. Dee Rachel et al. Druggable targets in the Rho pathway and their promise for therapeutic control of blood pressure. (2019) PHARMACOLOGY & THERAPEUTICS 0163-7258 1879-016X 193 121-134 332. Herring Brian et al. Gastroparesis is associated with decreased FOXF1 and FOXF2 in humans, and loss of FOXF1 and FOXF2 results in gastroparesis in mice. (2019) NEUROGASTROENTEROLOGY AND MOTILITY 1350-1925 1365- 2982 31 3 333. Arnold Caroline et al. Genetic ablation of NFAT5/TonEBP in smooth muscle cells impairs ow- and pressure-induced arterial remodeling in mice. (2019) FASEB JOURNAL 0892-6638 1530-6860 33 3 3364-3377 334. Lakhal-Littleton Samira et al. Intracellular iron deciency in pulmonary arterial smooth muscle cells induces pulmonary arterial hypertension in mice. (2019) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 0027-8424 1091-6490 116 26 13122-13130 335. Cai Weibin et al. Limited Regeneration Potential with Minimal Epicardial Progenitor Conversions in the Neonatal Mouse Heart after Injury. (2019) CELL REPORTS 2211-1247 28 1 190-+ 336. Durgin Brittany et al. Loss of smooth muscle CYB5R3 amplies angiotensin II-induced hypertension by increasing sGC heme oxidation. (2019) JCI INSIGHT 2379-3708 4 19 337. Gabani Mohanad et al. MiR-204 regulates type 1 IP3R to control vascular smooth muscle cell contractility and blood pressure. (2019) CELL CALCIUM 0143-4160 80 18-24 338. * Chennupati Ramesh et al. Myogenic vasoconstriction requires G(12)/G(13) and LARG to maintain local and systemic vascular resistance. (2019) ELIFE 2050-084X 8 339. Shvetso Anastasia et al. Negative feedback regulation of vasocontraction by potassium channels in 10-to 15-day-old rats: Dominating role of K(v)7 channels. (2019) ACTA PHYSIOLOGICA 1748-1708 1748-1716 225 2 340. Jeong Kyuho et al. Nuclear Focal Adhesion Kinase Controls Vascular Smooth Muscle Cell Proliferation and Neointimal Hyperplasia Through GATA4-Mediated Cyclin D1 Transcription. (2019) CIRCULATION RESEARCH 0009-7330 1524-4571 125 2 152- 166 341. Hess Daniel et al. Perivascular cell-specic knockout of the stem cell pluripotency gene Oct4 inhibits angiogenesis. (2019) NATURE COMMUNICATIONS 2041-1723 10 342. Quijada Pearl et al. Pre-existing broblasts of epicardial origin are the primary source of pathological brosis in cardiac ischemia and aging. (2019) JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY 0022-2828 1095-8584 129 92-104 343. Chakraborty Raja et al. Promoters to Study Vascular Smooth Muscle Mistaken Identity?. (2019) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 39 4 603-612 344. Mahan Sidney et al. Quantitative Analysis of Cellular Composition in Advanced Atherosclerotic Lesions of Smooth Muscle Cell Lineage-Tracing Mice. (2019) JOVE-JOURNAL OF VISUALIZED EXPERIMENTS 1940-087X 144 345. Chiu W.-C. et al. Reduction of blood pressure elevation by losartan in spontaneously hypertensive rats through suppression of LARG expression in vascular smooth muscle cells. (2019) JOURNAL OF THE FORMOSAN MEDICAL ASSOCIATION 0929-6646 2019 p. In press 346. Strassheim Derek et al. RhoGTPase in Vascular Disease. (2019) CELLS 2073-4409 8 6 347. Kaßmann M. et al. Role of Ryanodine Type 2 Receptors in Elementary Ca2+ Signaling in Arteries and Vascular Adaptive Responses. (2019) JOURNAL OF THE AMERICAN HEART ASSOCIATION 2047-9980 2047-9980 8 9 348. Liu Mingjun et al. Smooth Muscle Cell Phenotypic Diversity At the Crossroads of Lineage Tracing and Single-Cell Transcriptomics. (2019) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 39 9 1715-1723 349. Mahmoud Marwa et al. Smooth muscle cell-specic knockout of neuropilin-1 impairs postnatal lung development and pathological vascular smooth muscle cell accumulation. (2019) AMERICAN JOURNAL OF PHYSIOLOGY: CELL PHYSIOLOGY 0363- 6143 1522-1563 316 3 C424-C433 350. Osman Islam et al. TEAD1 (TEA Domain Transcription Factor 1) Promotes Smooth Muscle Cell Proliferation Through Upregulating SLC1A5 (Solute Carrier Family 1 Member 5)-Mediated Glutamine Uptake. (2019) CIRCULATION RESEARCH 0009-7330 1524-4571 124 9 1309-1322 351. Deng Guorui et al. The renin - angiotensin system in the arcuate nucleus controls resting metabolic rate. (2019) CURRENT OPINION IN NEPHROLOGY AND HYPERTENSION 1062-4821 1473- 6543 28 2 120-127 352. Basatemur G.L. et al. Vascular smooth muscle cells in atherosclerosis. (2019) NATURE REVIEWS CARDIOLOGY 1759-5002 1759-5010 16 724-744 353. Wu Wen et al. Vascular smooth muscle-MAPK14 is required for neointimal hyperplasia by suppressing VSMC dierentiation and inducing proliferation and inammation. (2019) REDOX BIOLOGY 2213-2317 22 354. Salmon Morgan et al. ZFP148 (Zinc-Finger Protein 148) Binds Cooperatively With NF-1 (Neurobromin 1) to Inhibit Smooth Muscle Marker Gene Expression During Abdominal Aortic Aneurysm Formation. (2019) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 39 1 73-88

128. Gille, A ; Benyó, Z ; Oermanns, S Mechanism of nicotinic acid-induced ushing in mice NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 375 : suppl 1 p. 19 Paper: 37 (2007) DOI WoS Közlemény:1856289 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat ) 129. Hortobagyi, L ; Kis, B ; Hrabak, A ; Horvath, B ; Huszty, G ; Schweer, H ; Benyo, B ; Sandor, P ; Busija, DW ; Benyo, Z Adaptation of the hypothalamic blood ow to chronic nitric oxide deciency is independent of vasodilator prostanoids BRAIN RESEARCH 1131 : 1 pp. 129-137. , 9 p. (2007) DOI WoS Scopus PubMed Google scholar Google scholar hash Közlemény:1151559 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 18 Független: 16 Függő: 2 Idézett közlemények száma: 15

1. Mersich T. Szomatikus fájdalominger hatása az agy regionális véráramlására és az agyi vértérfogatra altatott patkányokon. (2007) 2. 曹非 et al. 急性脑血管病患者血清一氧化氮和一氧化氮合酶变化规律 及临床意义. (2007) ZHONGHUA SHEN JING KE ZA ZHI / CHINESE JOURNAL OF NEUROLOGY 1006-7876 40 10 690-691 3. Pena Dias. Étude du rôle des kinines dans les neuropathies et l'hypertention [ie hypertension] artérielle chez le rat diabétique. (2008) 4. * Lacza Z et al. Additive eect of cyclooxygenase and nitric oxide synthase blockade on the cerebrocortical microcirculation. (2009) NEUROREPORT 0959-4965 20 11 1027-1031 5. Toda N et al. Cerebral Blood Flow Regulation by Nitric Oxide: Recent Advances. (2009) PHARMACOLOGICAL REVIEWS 0031- 6997 1521-0081 61 1 62-97 6. Porre A et al. Evaluation of antiinamatory activity of liposome encapsulated superoxide dismutase in rats peritonitis. (2009) FARMACIA 0367-3014 57 4 412-423 7. Gyurkovics M et al. Venodilatory eect of vascular endothelial growth factor (VEGF) on rat gingiva.. (2009) JOURNAL OF PERIODONTOLOGY 0022-3492 80 9 1518-1523 8. Fujita Y et al. Early Protective Eect of Bone Marrow Mononuclear Cells Against Ischemic White Matter Damage Through Augmentation of Cerebral Blood Flow. (2010) STROKE 0039-2499 1524-4628 41 12 2938-2943 9. Ainslie PN et al. Regulation of cerebral blood ow in mammals during chronic hypoxia: a matter of balance. (2010) EXPERIMENTAL PHYSIOLOGY 0958-0670 1469-445X 95 2 251-262 10. Shukla R et al. Cognitive function and hypertension: Pathophysiology and mechanisms. (2011) Megjelent: Vascular Dementia: Risk Factors, Diagnosis and Treatment pp. 257-285 11. Ainslie P.N. et al. Cerebral circulation and brain. (2013) Megjelent: High Altitude: Human Adaptation to Hypoxia pp. 141-170 12. * Iring A et al. Role of endocannabinoids and cannabinoid-1 receptors in cerebrocortical blood ow regulation. (2013) PLOS ONE 1932-6203 1932-6203 8 1 13. Islam MdZ et al. Bradykinin induces NO and PGF2α production via B 2 receptor activation from cultured porcine basilar arterial endothelial cells. (2014) NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 0028-1298 1432-1912 387 7 697-702 14. Chen Q et al. Long-Term aerobic exercise increases redox-Active iron through nitric oxide in rat hippocampus. (2014) NITRIC OXIDE-BIOLOGY AND CHEMISTRY 1089-8603 1089-8611 36 1-10 15. Tabatabaei Seyed et al. Nitric Oxide and Cerebrovascular Regulation. (2014) Megjelent: Nitric Oxide pp. 347-385 16. Desai Mitul et al. Molecular imaging with engineered physiology. (2016) NATURE COMMUNICATIONS 2041-1723 7 17. Toth P et al. Functional Vascular Contributions to Cognitive Impairment and Dementia (VCID): Mechanisms and Consequences of Cerebral Microvascular Dysfunction in Aging.. (2017) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 312 1 H1-H20 18. Czigler Andras et al. Hypertension exacerbates cerebrovascular oxidative stress induced by mild traumatic brain injury : protective eects of the mitochondria-targeted antioxidative peptide SS-31. (2019) JOURNAL OF NEUROTRAUMA 0897-7151 2019 p. In press

130. Horvath, B ; Hortobagyi, L ; Hrabak, A ; Sandor, P ; Benyo, Z Interactions between the heme oxygenase, cyclooxygenase and nitric oxide synthase pathways in the regulation of the resting hypothalamic blood ow ACTA PHYSIOLOGICA HUNGARICA 94 : 4 pp. 352-353. , 2 p. (2007) WoS Közlemény:1151560 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

131. Leszl-Ishiguro, M ; Horváth, B ; Johnson, R A ; Johnson, F K ; Lenzsér, G ; Hermán, P ; Horváth, E M ; Benyó, Z Inuence of the heme-oxygenase pathway on cerebrocortical blood ow NEUROREPORT 18 : 11 pp. 1193-1197. , 5 p. (2007) DOI WoS Scopus PubMed Közlemény:1475873 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 5 Független: 1 Függő: 4 Idézett közlemények száma: 3

1. * Horvath B et al. Carbon monoxide-prostaglandin E-2 interaction in the hypothalamic circulation. (2008) NEUROREPORT 0959-4965 19 16 1601-1604 2. * Johnson Robert et al. Heme oxygenase-derived endogenous carbon monoxide impairs ow-induced dilation in resistance vessels. (2008) SHOCK 1073-2322 1540-0514 29 4 526-530 3. * Lacza Z et al. Additive eect of cyclooxygenase and nitric oxide synthase blockade on the cerebrocortical microcirculation. (2009) NEUROREPORT 0959-4965 20 11 1027-1031 4. * Horvath B et al. Hypersensitivity to thromboxane receptor mediated cerebral vasomotion and CBF oscillations during acute NO-deciency in rats.. (2010) PLOS ONE 1932-6203 1932-6203 5 12 5. Lu X et al. Upregulation of heme oxygenase-1 protected against brain damage induced by transient cerebral ischemia-reperfusion injury in rats. (2018) EXPERIMENTAL AND THERAPEUTIC MEDICINE 1792-0981 1792-1015 15 6 4629-4636

132. Benyo, Z* ; Gille, A* ; Bennett, CL ; Clausen, BE ; Oermanns, S Nicotinic acid-induced ushing is mediated by activation of epidermal langerhans cells MOLECULAR PHARMACOLOGY 70 : 6 pp. 1844-1849. , 6 p. (2006) DOI WoS Scopus PubMed Közlemény:1628750 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 168 Független: 154 Függő: 14 Idézett közlemények száma: 1

1. Skinner PJ. 3-Nitro-4-amino benzoic acids and 6-amino nicotinic acids are highly selective agonists of GPR109b. (2007) BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 0960-894X 1464- 3405 17 23 6619-6622 2. Jacobson EL. A topical lipophilic niacin derivative increases NAD, epidermal dierentiation and barrier function in photodamaged skin. (2007) EXPERIMENTAL DERMATOLOGY 0906-6705 1600-0625 16 6 490-499 3. Smesny S. Endophenotype properties of niacin sensitivity as marker of impaired prostaglandin signalling in Schizophrenia. (2007) PROSTAGLANDINS LEUKOTRIENES AND ESSENTIAL FATTY ACIDS 0952-3278 77 2 79-85 4. Stone NJ. Metabolic syndrome management. (2007) EXPERT OPINION ON PHARMACOTHERAPY 1465-6566 1744-7666 8 13 2059-2075 5. Fyfe MCT. New Nonpeptide-Binding GPCRs as Targets for Diabetes and the Metabolic Syndrome. (2007) Megjelent: ANNUAL REPORTS IN MEDICINAL CHEMISTRY, VOL 42 pp. 129-145 6. Nicholls SJ. New targets of high-density lipoprotein therapy. (2007) CURRENT OPINION IN LIPIDOLOGY 0957-9672 1473-6535 18 4 421-426 7. Guyton JR. Niacin in cardiovascular prevention: mechanisms, ecacy, and safety. (2007) CURRENT OPINION IN LIPIDOLOGY 0957-9672 1473-6535 18 4 415-420 8. Kamanna VS. Nicotinic acid (niacin) receptor agonists: Will they be useful therapeutic agents?. (2007) AMERICAN JOURNAL OF CARDIOLOGY 0002-9149 1879-1913 100 11, Suppl. S53-S61 9. Richman JG. Nicotinic acid receptor agonists dierentially activate downstream eectors. (2007) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 282 25 18028-18036 10. Schmitz G. Pharmacogenetics and pharmacogenomics of cholesterol-lowering therapy. (2007) CURRENT OPINION IN LIPIDOLOGY 0957-9672 1473-6535 18 2 164-173 11. Carballo-Jane E. Potential role for epidermal Langerhans cells in nicotinic acid-induced vasodilatation in the mouse. (2007) INFLAMMATION RESEARCH 1023-3830 1420-908X 56 6 254-261 12. Semple G. Recent progress in the discovery of niacin receptor agonists. (2007) CURRENT OPINION IN DRUG DISCOVERY & DEVELOPMENT 1367-6733 10 4 452-459 13. Stern RH. The role of nicotinic acid metabolites in ushing and hepatotoxicity. (2007) JOURNAL OF CLINICAL LIPIDOLOGY 1933- 2874 1 3 191-193 14. Semple G. 3-(1H-tetrazol-5-yl)-1,4,5,6-tetrahydro- cyclopentapyrazole (MK-0354): A partial agonist of the nicotinic acid receptor, G-protein coupled receptor 109a, with antilipolytic but no vasodilatory activity in mice. (2008) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 51 16 5101-5108 15. Regard JB. Anatomical Proling of G Protein-Coupled Receptor Expression. (2008) CELL 0092-8674 1097-4172 135 3 561-571 16. Shen HC. Discovery of pyrazolopyrimidines as the rst class of allosteric agonists for the high anity nicotinic acid receptor GPR109A. (2008) BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 0960-894X 1464-3405 18 18 4948-4951 17. Lai E et al. Eects of a niacin receptor partial agonist, MK-0354, on plasma free fatty acids, lipids, and cutaneous ushing in humans. (2008) JOURNAL OF CLINICAL LIPIDOLOGY 1933-2874 2 5 375-383 18. Vo AN et al. Fixed-dose combination of extended-release niacin plus simvastatin for lipid disorders. (2008) EXPERT REVIEW OF CARDIOVASCULAR THERAPY 1477-9072 6 10 1303-1310 19. Taylor AJ. Flushing and the HDL-C response to extended-release niacin. (2008) JOURNAL OF CLINICAL LIPIDOLOGY 1933-2874 2 4 285-288 20. Nicholls SJ. HDL: Still a target for new therapies?. (2008) CURRENT OPINION IN INVESTIGATIONAL DRUGS 1472-4472 2040-3429 9 9 950-956 21. Nicholls SJ. High-density lipoprotein and atheroma monitoring. (2008) CURRENT OPINION IN CARDIOLOGY 0268-4705 1531-7080 23 4 386-392 22. Leithauser B et al. Inuence of xantinole nicotinic acid on cutaneous microcirculation in patients with coronary artery disease and hyperlipoproteinemia. (2008) CLINICAL HEMORHEOLOGY AND MICROCIRCULATION 1386-0291 1875- 8622 39 1-4 287-292 23. Majeed F. Low high-density lipoprotein cholesterol: an important consideration in coronary heart disease risk assessment. (2008) CURRENT OPINION IN ENDOCRINOLOGY DIABETES AND OBESITY 1752-296X 1752-2978 15 2 175-181 24. Kamanna VS. Mechanism of action of niacin. (2008) AMERICAN JOURNAL OF CARDIOLOGY 0002-9149 1879-1913 101 8, Suppl. S20-S26 25. Kostner K et al. Niacin: A lipid polypill?. (2008) EXPERT OPINION ON PHARMACOTHERAPY 1465-6566 1744-7666 9 16 2911-2920 26. Papaliodis D. Niacin-induced "Flush" Involves Release of Prostaglandin D-2 from Mast Cells and Serotonin from Platelets: Evidence from Human Cells in Vitro and an Animal Model. (2008) JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS 0022-3565 1521-0103 327 3 665-672 27. Davidson Michael. Niacin use and cutaneous ushing: Mechanisms and strategies for prevention. (2008) AMERICAN JOURNAL OF CARDIOLOGY 0002-9149 1879-1913 101 8 Suppl S14-S19 28. * Bodor ET. Nicotinic acid: an old drug with a promising future. (2008) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476- 5381 153 Suppl. 1 S68-S75 29. Bogan Katrina et al. Nicotinic acid nicotinamide and nicotinamide riboside: A molecular evaluation of NAD(+) precursor vitamins in human nutrition. (2008) ANNUAL REVIEW OF NUTRITION 0199- 9885 1545-4312 28 115-130 30. * Gille A. Nicotinic acid: Pharmacological eects and mechanisms of action. (2008) ANNUAL REVIEW OF PHARMACOLOGY AND TOXICOLOGY 0362-1642 48 79-106 31. Kamanna VS. Nicotinic acid: recent developments. (2008) CURRENT OPINION IN CARDIOLOGY 0268-4705 1531-7080 23 4 393-398 32. Boatman PD. Nicotinic Acid Receptor Agonists. (2008) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 51 24 7653- 7662 33. Nickolo BJ. Saving the skin from drug-induced detachment. (2008) NATURE MEDICINE 1078-8956 1546-170X 14 12 1311-1313 34. Papaliodis D. The avonoid luteolin inhibits niacin-induced ush. (2008) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476- 5381 153 7 1382-1387 35. Tang H. The psoriasis drug monomethylfumarate is a potent nicotinic acid receptor agonist. (2008) BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS 0006-291X 1090- 2104 375 4 562-565 36. Gülcü N et al. Topikal nikotinat katkili etofenamat kullanilmasinin periferik venöz kanülasyon başansina etkisi [The eect of topical etofenamate with nicotinate on the success of peripheric venous cannulation]. (2008) TURK ANESTEZIYOLOJI VE REANIMASYON DERNEGI DERGISI 1304-0871 36 4 244-249 37. Skinner Philip et al. 5-N,N-Disubstituted 5-aminopyrazole-3- carboxylic acids are highly potent agonists of GPR109b. (2009) BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 0960-894X 1464- 3405 19 15 4207-4209 38. Thakkar RB. Acetylsalicylic Acid Reduces Niacin Extended-Release- Induced Flushing in Patients with Dyslipidemia. (2009) AMERICAN JOURNAL OF CARDIOVASCULAR DRUGS 1175-3277 1179-187X 9 2 69-79 39. Walters RW. beta-Arrestin1 mediates nicotinic acid-induced ushing, but not its antilipolytic eect, in mice. (2009) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 119 5 1312- 1321 40. Manzato E et al. Colesterolo-HDL e rischio cardiovascolare residuo. La terapia combinata statina/niacina [HDL cholesterol and residual cardiovascular risk. Combined statin/niacin treatment]. (2009) Trends in Medicine 1594-2848 9 4 195-204 41. Shen HC. Discovery of Novel Tricyclic Full Agonists for the G- Protein-Coupled Niacin Receptor 109A with Minimized Flushing in Rats. (2009) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 52 8 2587-2602 42. Schwartz JI. Eect of Laropiprant, a PGD(2) Receptor 1 Antagonist, on Estradiol and Norgestimate Pharmacokinetics After Oral Contraceptive Administration in Women. (2009) AMERICAN JOURNAL OF THERAPEUTICS 1075-2765 16 6 487-495 43. Martin PM. Expression and localization of GPR109A (PUMA- G/HM74A) mRNA and protein in mammalian retinal pigment epithelium. (2009) MOLECULAR VISION 1090-0535 15 37 362-372 44. * Ahmed Kashan et al. GPR109A, GPR109B and GPR81, a family of hydroxy-carboxylic acid receptors. (2009) TRENDS IN PHARMACOLOGICAL SCIENCES 0165-6147 30 11 557-562 45. Whayne TF. High-density Lipoprotein Cholesterol: Current Perspective for Clinicians. (2009) ANGIOLOGY 0003-3197 60 5 644-649 46. Martres P. HM74a Agonists: Will they be the New Generation of Nicotinic Acid?. (2009) CURRENT TOPICS IN MEDICINAL CHEMISTRY 1568-0266 1873-4294 9 5 428-435 47. Schwartz JI. Inuence of Laropiprant, a Selective Prostaglandin D- 2 Receptor 1 Antagonist, on the Pharmacokinetics and Pharmacodynamics of Warfarin. (2009) AMERICAN JOURNAL OF THERAPEUTICS 1075-2765 16 3 215-223 48. Schweikart J. Investigation of niacin on parameters of metabolism in a physiologic dose: randomized, double-blind clinical trial with three dierent dosages. (2009) INTERNATIONAL JOURNAL OF FOOD SCIENCES AND NUTRITION 0963-7486 1465-3478 60 Suppl. 5 192-202 49. Liu Changlu et al. Lactate Inhibits Lipolysis in Fat Cells through Activation of an Orphan G-protein-coupled Receptor, GPR81. (2009) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083- 351X 284 5 2811-2822 50. Vesely DL. Langerhans Cell Deciency Impairs Ixodes scapularis Suppression of Th1 Responses in Mice. (2009) INFECTION AND IMMUNITY 0019-9567 1098-5522 77 5 1881-1887 51. Sood A. Mechanisms of Flushing Due to Niacin and Abolition of These Eects. (2009) JOURNAL OF CLINICAL HYPERTENSION 1524- 6175 11 11 685-689 52. Kamanna VS. Niacin: An Old Drug Rejuvenated. (2009) CURRENT ATHEROSCLEROSIS REPORTS 1523-3804 11 1 45-51 53. Vosper Helen. Niacin: a re-emerging pharmaceutical for the treatment of dyslipidaemia. (2009) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 158 2 429-441 54. Brown BG et al. Nicotinic Acid. (2009) Megjelent: Clinical Lipidology: A Companion to Braunwald's Heart Disease pp. 298- 314 55. Farmer JA. Nicotinic acid: A new look at an old drug. (2009) CURRENT ATHEROSCLEROSIS REPORTS 1523-3804 11 2 87-92 56. Penberthy W. Nicotinic Acid-Mediated Activation of Both Membrane and Nuclear Receptors towards Therapeutic Glucocorticoid Mimetics for Treating Multiple Sclerosis. (2009) PPAR RESEARCH 1687-4757 1687-4765 2009 57. Ren N. Phenolic acids suppress adipocyte lipolysis via activation of the nicotinic acid receptor GPR109A (HM74a/PUMA-G). (2009) JOURNAL OF LIPID RESEARCH 0022-2275 1539-7262 50 5 908-914 58. Descamps OS. Renaissance de la niacine sous une nouvelle forme Galénique, le Tredaptive™ [Rebirth of niacin under a new Galenic form, Tredaptive™]. (2009) LOUVAIN MEDICAL 0024-6956 128 9 308-318 59. Kamanna V et al. The mechanism and mitigation of niacin- induced ushing. (2009) INTERNATIONAL JOURNAL OF CLINICAL PRACTICE 1368-5031 63 9 1369-1377 60. Jacobson TA. A "Hot" Topic in Dyslipidemia Management-"How to Beat a Flush": Optimizing Niacin Tolerability to Promote Long- term Treatment Adherence and Coronary Disease Prevention. (2010) MAYO CLINIC PROCEEDINGS 0025-6196 85 4 365-379 61. Zimbelman RB. Eects of encapsulated niacin on evaporative heat loss and body temperature in moderately heat-stressed lactating Holstein cows. (2010) JOURNAL OF DAIRY SCIENCE 0022- 0302 1525-3198 93 6 2387-2394 62. Lai E. Eects of extended release niacin/laropiprant, laropiprant, extended release niacin and placebo on platelet aggregation and bleeding time in healthy subjects. (2010) PLATELETS 0953-7104 1369-1635 21 3 191-198 63. Liu F. Eects of Laropiprant, a Selective Prostaglandin D-2 Receptor 1 Antagonist, on the Steady-State Pharmacokinetics of Digoxin in Healthy Adult Subjects. (2010) JOURNAL OF CLINICAL PHARMACOLOGY 0091-2700 1552-4604 50 7 823-828 64. Shen HC et al. High-Anity Niacin Receptor GPR109A Agonists. (2010) Megjelent: ANNUAL REPORTS IN MEDICINAL CHEMISTRY, VOL 45 pp. 72-94 65. Oates E. Interaction Between Enfuvirtide, an Injectable Fusion Inhibitor, and Niacin in an HIV-Infected Patient. (2010) ANNALS OF PHARMACOTHERAPY 1060-0280 1542-6270 44 12 2014-2017 66. * Romani N. Langerhans cells and more: langerin-expressing dendritic cell subsets in the skin. (2010) IMMUNOLOGICAL REVIEWS 0105-2896 234 1 120-141 67. Sanyal S et al. Niacin and laropiprant. (2010) DRUGS OF TODAY 1699-3993 46 6 371-378 68. Benhalima K. Niacin, an old drug with new perspectives for the management of dyslipidaemia. (2010) ACTA CLINICA BELGICA: INTERNATIONAL JOURNAL OF CLINICAL AND LABORATORY MEDICINE 1784-3286 2295-3337 65 1 23-28 69. Al-Mohaissen MA. Niacin: From Mechanisms of Action to Therapeutic Uses. (2010) MINI-REVIEWS IN MEDICINAL CHEMISTRY 1389-5575 1875-5607 10 3 204-217 70. Messamore E. Niacin sensitivity and the arachidonic acid pathway in schizophrenia. (2010) SCHIZOPHRENIA RESEARCH 0920-9964 1573-2509 122 1-3 248-256 71. * Hanson J. Nicotinic acid- and monomethyl fumarate-induced ushing involves GPR109A expressed by keratinocytes and COX- 2-dependent prostanoid formation in mice. (2010) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 120 8 2910-2919 72. Boatman PD. Potent tricyclic pyrazole tetrazole agonists of the nicotinic acid receptor (GPR109a). (2010) BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 0960-894X 1464-3405 20 9 2797- 2800 73. Dunbar RL. Seeing red: ushing out instigators of niacin- associated skin toxicity. (2010) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 120 8 2651-2655 74. Luo WL. Single Therapeutic and Supratherapeutic Doses of Laropiprant, a Selective Prostaglandin D-2 Receptor 1 Antagonist, Do Not Prolong the QTcF Interval in Healthy Volunteers. (2010) JOURNAL OF CLINICAL PHARMACOLOGY 0091-2700 1552-4604 50 11 1273-1279 75. * Blad CC et al. Biological and Pharmacological Roles of HCA Receptors. (2011) Megjelent: Pharmacology of G Protein Coupled Receptors pp. 219-250 76. Ahmed K. Biological roles and therapeutic potential of hydroxy- carboxylic acid receptors. (2011) FRONTIERS IN ENDOCRINOLOGY 1664-2392 2 77. Li Guo et al. Distinct Kinetic and Spatial Patterns of Protein Kinase C (PKC)- and Epidermal Growth Factor Receptor (EGFR)- dependent Activation of Extracellular Signal-regulated Kinases 1 and 2 by Human Nicotinic Acid Receptor GPR109A. (2011) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 286 36 31199-31212 78. Wanders D. Future of GPR109A agonists in the treatment of dyslipidaemia. (2011) DIABETES OBESITY & METABOLISM 1462- 8902 1463-1326 13 8 685-691 79. Kwong Alan et al. High Dietary Niacin May Increase Prostaglandin Formation but Does Not Increase Tumor Formation in Apc(Min/+) Mice. (2011) NUTRITION AND CANCER : AN INTERNATIONAL JOURNAL 0163-5581 1532-7914 63 6 950-959 80. Kammermann M et al. Impedance measurement: A new method to detect ligand-biased receptor signaling. (2011) BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS 0006-291X 1090-2104 412 3 419-424 81. Backes JM. Important Considerations for Treatment with Dietary Supplement Versus Prescription Niacin Products. (2011) POSTGRADUATE MEDICINE 0032-5481 123 2 70-83 82. * Oermanns Stefan et al. International Union of Basic and Clinical Pharmacology. LXXXII: Nomenclature and Classication of Hydroxy-carboxylic Acid Receptors (GPR81, GPR109A, and GPR109B). (2011) PHARMACOLOGICAL REVIEWS 0031-6997 1521- 0081 63 2 269-290 83. Mullangi R. Niacin and its metabolites: role of LC-MS/MS bioanalytical methods and update on clinical pharmacology. An overview. (2011) BIOMEDICAL CHROMATOGRAPHY 0269-3879 1099-0801 25 1-2 218-237 84. Wierzbicki AS. Niacin: the only vitamin that reduces cardiovascular events. (2011) INTERNATIONAL JOURNAL OF CLINICAL PRACTICE 1368-5031 65 4 380-385 85. Li HM. Nicotinic Acid Inhibits Glucose-Stimulated Insulin Secretion Via the G Protein-Coupled Receptor PUMA-G in Murine Islet beta Cells. (2011) PANCREAS 0885-3177 1536-4828 40 4 615-621 86. * Lukasova M. Nicotinic acid inhibits progression of atherosclerosis in mice through its receptor GPR109A expressed by immune cells. (2011) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 121 3 1163-1173 87. * Lukasova Martina et al. Nicotinic acid (niacin): new lipid- independent mechanisms of action and therapeutic potentials. (2011) TRENDS IN PHARMACOLOGICAL SCIENCES 0165-6147 32 12 700-707 88. Bermudez Y. Nicotinic Acid Receptor Abnormalities in Human Skin Cancer: Implications for a Role in Epidermal Dierentiation. (2011) PLOS ONE 1932-6203 1932-6203 6 5 89. Babcock MJ. Persistent erythema with niacin is not attributable to aspirin resistance. (2011) JOURNAL OF CLINICAL LIPIDOLOGY 1933-2874 5 2 114-119 90. Wang YH. Pharmacokinetics of Laropiprant, a Selective Prostaglandin D-2 Receptor 1 Antagonist, in Patients With Moderate Hepatic Impairment. (2011) JOURNAL OF CLINICAL PHARMACOLOGY 0091-2700 1552-4604 51 3 406-412 91. van Veldhoven JPD. Structure-activity relationships of trans- substituted-propenoic acid derivatives on the nicotinic acid receptor HCA2 (GPR109A). (2011) BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 0960-894X 1464-3405 21 9 2736-2739 92. Kuei Chester. Study of GPR81, the Lactate Receptor, from Distant Species Identies Residues and Motifs Critical for GPR81 Functions. (2011) MOLECULAR PHARMACOLOGY 0026-895X 1521- 0111 80 5 848-858 93. Dallob Aimee et al. The eects of laropiprant, a selective prostaglandin D-2 receptor 1 antagonist, on the antiplatelet activity of clopidogrel or aspirin. (2011) PLATELETS 0953-7104 1369-1635 22 7 495-503 94. Boatman P. (1aR,5aR)1a,3,5,5a-Tetrahydro-1H-2,3-diaza- cyclopropa[a]pentalene-4-carb oxylic Acid (MK-1903): A Potent GPR109a Agonist that Lowers Free Fatty Acids in Humans. (2012) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 55 8 3644-3666 95. Liu Changlu et al. 3,5-Dihydroxybenzoic Acid, a Specic Agonist for Hydroxycarboxylic Acid 1, Inhibits Lipolysis in Adipocytes. (2012) JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS 0022-3565 1521-0103 341 3 794-801 96. VanHorn J et al. Attenuation of niacin-induced prostaglandin D 2 generation by omega-3 fatty acids in THP-1 macrophages and Langerhans dendritic cells. (2012) JOURNAL OF INFLAMMATION RESEARCH 1178-7031 5 1 37-50 97. Romani Nikolaus. Changing Views of the Role of Langerhans Cells. (2012) JOURNAL OF INVESTIGATIVE DERMATOLOGY 0022-202X 132 3 872-881 98. Yadav Rahul. Extended-release niacin with laropiprant: a review on ecacy, clinical eectiveness and safety. (2012) EXPERT OPINION ON PHARMACOTHERAPY 1465-6566 1744-7666 13 9 1345-1362 99. * Blad CC et al. G protein-coupled receptors for energy metabolites as new therapeutic targets. (2012) NATURE REVIEWS DRUG DISCOVERY 1474-1776 11 8 603-619 100. Mureddu Gian et al. High-density lipoprotein levels and risk of cardiovascular events: a review. (2012) JOURNAL OF CARDIOVASCULAR MEDICINE 1558-2027 13 9 575-586 101. Dvorak Curt et al. Identication of Hydroxybenzoic Acids as Selective Lactate Receptor (GPR81) Agonists with Antilipolytic Eects. (2012) ACS MEDICINAL CHEMISTRY LETTERS 1948-5875 3 8 637-639 102. Mason Carol. Niacin-A Critical Component to the Management of Atherosclerosis: Contemporary Management of Dyslipidemia to Prevent, Reduce, or Reverse Atherosclerotic Cardiovascular Disease. (2012) JOURNAL OF CARDIOVASCULAR NURSING 0889- 4655 27 4 303-316 103. Creider Julia et al. Niacin: another look at an underutilized lipid- lowering medication. (2012) NATURE REVIEWS ENDOCRINOLOGY 1759-5029 1759-5037 8 9 517-528 104. Ingersoll Molly. Niacin inhibits skin dendritic cell mobilization in a GPR109A independent manner but has no impact on monocyte tracking in atherosclerosis. (2012) IMMUNOBIOLOGY 0171-2985 1878-3279 217 5 548-557 105. Lauring Brett et al. Niacin Lipid Ecacy Is Independent of Both the Niacin Receptor GPR109A and Free Fatty Acid Suppression. (2012) SCIENCE TRANSLATIONAL MEDICINE 1946-6234 4 148 106. Messamore Erik. Niacin subsensitivity is associated with functional impairment in schizophrenia. (2012) SCHIZOPHRENIA RESEARCH 0920-9964 1573-2509 137 1-3 180-184 107. Villines Todd. Niacin: The Evidence, Clinical Use, and Future Directions. (2012) CURRENT ATHEROSCLEROSIS REPORTS 1523- 3804 14 1 49-59 108. Kei Anastazia et al. Nicotinic acid: clinical considerations. (2012) EXPERT OPINION ON DRUG SAFETY 1474-0338 11 4 551-564 109. Huang Yan et al. Progess in mechanism of nicotinic acid mediated by nicotinic acid receptor GPR109A. (2012) CHINESE JOURNAL OF PHARMACOLOGY AND TOXICOLOGY / ZHONG GUO YAO LI XUE YU DU LI XUE ZA ZHI 1000-3002 26 1 105-107 110. * Hanson Julien et al. Role of HCA(2) (GPR109A) in nicotinic acid and fumaric acid ester-induced eects on the skin. (2012) PHARMACOLOGY & THERAPEUTICS 0163-7258 1879-016X 136 1 1-7 111. Yadav Rahul. Safety and tolerability of extended-release niacin with laropiprant. (2012) EXPERT OPINION ON DRUG SAFETY 1474- 0338 11 1 151-159 112. Drummond Peter. The eect of facial blood ow on ratings of blushing and negative aect during an embarrassing task: Preliminary ndings. (2012) JOURNAL OF ANXIETY DISORDERS 0887-6185 26 2 305-310 113. Drummond Peter. The eect of niacin on facial blood ow in people with an elevated fear of negative evaluation. (2012) EUROPEAN NEUROPSYCHOPHARMACOLOGY 0924-977X 1873- 7862 22 3 200-204 114. Penberthy WT. The niacin ush pathway in recovery from schizophrenia and how arginine and glutamine may provide added benet. (2012) JOURNAL OF ORTHOMOLECULAR MEDICINE 0834-4825 27 1 29-38 115. Cantó C et al. Crosstalk between poly(ADP-ribose) polymerase and sirtuin enzymes. (2013) MOLECULAR ASPECTS OF MEDICINE 0098-2997 34 6 1168-1201 116. Gaidarov Ibragim et al. Dierential tissue and ligand-dependent signaling of GPR109A receptor: Implications for anti- atherosclerotic therapeutic potential. (2013) CELLULAR SIGNALLING 0898-6568 1873-3913 25 10 2003-2016 117. Zimbelman R et al. Eects of utilizing rumen protected niacin on core body temperature as well as milk production and composition in lactating dairy cows during heat stress. (2013) ANIMAL FEED SCIENCE AND TECHNOLOGY 0377-8401 180 1-4 26- 33 118. * Tonack Sarah et al. Endogenous metabolites as ligands for G protein-coupled receptors modulating risk factors for metabolic and cardiovascular disease. (2013) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 304 4 H501-H513 119. Gauly M et al. Future consequences and challenges for dairy cow production systems arising from climate change in Central Europe - a review. (2013) ANIMAL: THE INTERNATIONAL JOURNAL OF ANIMAL BIOSCIENCES 1751-7311 1571-732X 7 5 843-859 120. Kaore SN et al. Niacin and niacin/laropiprant combination: Opening new hopes for dyslipidemia and cardiovascular diseases with few novel actions. (2013) INTERNATIONAL JOURNAL OF PHARMA AND BIO SCIENCES 0975-6299 4 3 P1053-P1078 121. Strack Alison et al. Nicotinic acid and DP1 blockade: studies in mouse models of atherosclerosis. (2013) JOURNAL OF LIPID RESEARCH 0022-2275 1539-7262 54 1 177-188 122. Nadalin Sergej et al. The impact of PLA2G4A and PTGS2 gene polymorphisms, and red blood cell PUFAs decit on niacin skin ush response in schizophrenia patients. (2013) PROSTAGLANDINS LEUKOTRIENES AND ESSENTIAL FATTY ACIDS 0952-3278 88 2 185-190 123. Gouni-Berthold Ioanna et al. The Role of Niacin in Lipid-lowering Treatment: Are we Aiming Too High?. (2013) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873-4286 19 17 3094- 3106 124. Sheikh Sarah et al. Tolerability and Pharmacokinetics of Delayed- Release Dimethyl Fumarate Administered With and Without Aspirin in Healthy Volunteers. (2013) CLINICAL THERAPEUTICS 0149-2918 1879-114X 35 10 1582-1594 125. Drummond PD et al. Topical ibuprofen inhibits blushing during embarrassment and facial ushing during aerobic exercise in people with a fear of blushing. (2013) EUROPEAN NEUROPSYCHOPHARMACOLOGY 0924-977X 1873-7862 23 12 1747-1753 126. Rungruang S et al. A dose-response evaluation of rumen- protected niacin in thermoneutral or heat-stressed lactating Holstein cows. (2014) JOURNAL OF DAIRY SCIENCE 0022-0302 1525-3198 97 8 5023-5034 127. Yu AL et al. Constitutive Expression of HCA(2) in Human Retina and Primary Human Retinal Pigment Epithelial Cells. (2014) CURRENT EYE RESEARCH 0271-3683 1460-2202 39 5 487-492 128. * Oermanns S. Free fatty acid (FFA) and hydroxy carboxylic acid (HCA) receptors. (2014) ANNUAL REVIEW OF PHARMACOLOGY AND TOXICOLOGY 0362-1642 54 407-434 129. Verma N et al. HDL Cholesterol: All Hope Is Not Lost After the Torcetrapib Setback- Emerging Therapeutic Strategies on the Horizon. (2014) AMERICAN JOURNAL OF THERAPEUTICS 1075- 2765 21 3 222-232 130. Sakurai T et al. Identication of a novel GPR81-selective agonist that suppresses lipolysis in mice without cutaneous ushing. (2014) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 727 1-7 131. Promsote W et al. L-2-oxothiazolidine-4-carboxylic acid attenuates oxidative stress and inammation in retinal pigment epithelium. (2014) MOLECULAR VISION 1090-0535 20 73-88 132. Ma Linlin et al. Nicotinic Acid Activates the Capsaicin Receptor TRPV1 Potential Mechanism for Cutaneous Flushing. (2014) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 34 6 1272-1280 133. Bosier B et al. Promises of Biased Signaling in the Development of Improved Therapeutics. (2014) Megjelent: Biased Signaling in Physiology, Pharmacology and Therapeutics pp. 251-292 134. McNamara Martin et al. A randomized pilot study on the eect of niacin on pulmonary arterial pressure. (2015) TRIALS 1745-6215 16 135. Sprecher D et al. Discovery and characterization of GSK256073, a non-ushing hydroxy-carboxylic acid receptor 2 (HCA2) agonist. (2015) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 756 1-7 136. Chen L et al. Niacin-induced hyperglycemia is partially mediated via niacin receptor GPR109a in pancreatic islets. (2015) MOLECULAR AND CELLULAR ENDOCRINOLOGY 0303-7207 1872- 8057 404 56-66 137. Guyton JR. Niacin: Risk benets and role in treating dyslipidemias. (2015) Megjelent: Dyslipidemias: Pathophysiology, Evaluation and Management pp. 439-452 138. Sinthupoom N et al. Nicotinic acid and derivatives as multifunctional pharmacophores for medical applications. (2015) EUROPEAN FOOD RESEARCH AND TECHNOLOGY 1438-2377 1438-2385 240 1 1-17 139. Ma L et al. Nicotinic acid is a common regulator of heat-sensing TRPV1-4 ion channels. (2015) SCIENTIFIC REPORTS 2045-2322 5 140. Salvati G et al. Response of lactating cows to live yeast supplementation during summer. (2015) JOURNAL OF DAIRY SCIENCE 0022-0302 1525-3198 98 6 4062-4073 141. Rezq Samar et al. Central GPR109A Activation Mediates Glutamate-Dependent Pressor Response in Conscious Rats. (2016) JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS 0022-3565 1521-0103 356 2 456-465 142. Pineda A et al. Eects of rumen-protected niacin on milk production and body temperature of middle and late lactation Holstein cows. (2016) LIVESTOCK SCIENCE 1871-1413 1878-0490 187 16-23 143. Selleri Silvia et al. Human mesenchymal stromal cell-secreted lactate induces M2-macrophage dierentiation by metabolic reprogramming. (2016) ONCOTARGET 1949-2553 1949-2553 7 21 30193-30210 144. Kuei C et al. Molecular and pharmacological characterization of hamster HCA1 and HCA2. (2016) IMMUNOLOGY ENDOCRINE AND METABOLIC AGENTS IN MEDICINAL CHEMISTRY 1871-5222 1875- 6115 16 1 61-69 145. Messamore Erik et al. Phospholipid, arachidonate and eicosanoid signaling in schizophrenia. (2016) OCL-OLEAGINEUX CORPS GRAS LIPIDES 1258-8210 23 1 146. Aragona K et al. Prepartum supplementation of nicotinic acid: Eects on health of the dam, colostrum quality, and acquisition of immunity in the calf. (2016) JOURNAL OF DAIRY SCIENCE 0022- 0302 1525-3198 99 5 3529-3538 147. Yao JK et al. Prevalence and specicity of the abnormal niacin response: A potential endophenotype marker in schizophrenia. (2016) SCHIZOPHRENIA BULLETIN 0586-7614 1745-1701 42 2 369- 376 148. de Oliveira CA et al. Safety and ecacy evaluation of gelatin- based nanoparticles associated with UV lters. (2016) COLLOIDS AND SURFACES B: BIOINTERFACES 0927-7765 140 531-537 149. Deepa B et al. Enhanced NLO and antibacterial properties of nicotinic acid-doped KDP crystals: synthesis, growth and characterisation. (2017) MATERIALS RESEARCH INNOVATIONS 1432-8917 21 2 86-90 150. Moro Christian et al. Enhancing capillary blood collection: The inuence of nicotinic acid and nonivamide. (2017) JOURNAL OF CLINICAL LABORATORY ANALYSIS 0887-8013 1098-2825 31 6 151. Katsyuba Elena et al. Modulating NAD(+) metabolism, from bench tobedside. (2017) EMBO JOURNAL 0261-4189 1460-2075 36 18 2670-2683 152. Puchalska Patrycja et al. Multi-dimensional Roles of Ketone Bodies in Fuel Metabolism, Signaling, and Therapeutics. (2017) CELL METABOLISM 1550-4131 25 2 262-284 153. Li Juanjuan et al. Niacin ameliorates ulcerative colitis via prostaglandin D-2-mediated D prostanoid receptor 1 activation. (2017) EMBO MOLECULAR MEDICINE 1757-4676 1757-4684 9 5 571-588 154. El-Zaatari Mohamad et al. Role of Dietary Metabolites in Regulating the Host Immune Response in Gastrointestinal Disease. (2017) FRONTIERS IN IMMUNOLOGY 1664-3224 1664- 3224 8 155. Elhassan Yasir et al. Targeting NAD+ in Metabolic Disease: New Insights Into an Old Molecule. (2017) JOURNAL OF THE ENDOCRINE SOCIETY 2472-1972 1 7 816-835 156. Martens Christopher et al. Chronic nicotinamide riboside supplementation is well-tolerated and elevates NAD(+) in healthy middle-aged and older adults. (2018) NATURE COMMUNICATIONS 2041-1723 9 157. Singh LR et al. Discovery of coumarin-dihydroquinazolinone analogs as niacin receptor 1 agonist with in-vivo anti-obesity ecacy. (2018) EUROPEAN JOURNAL OF MEDICINAL CHEMISTRY 0223-5234 1768-3254 152 208-222 158. Gaidarov I et al. Embelin and its derivatives unravel the signaling, proinammatory and antiatherogenic properties of GPR84 receptor. (2018) PHARMACOLOGICAL RESEARCH 1043-6618 1096- 1186 131 185-198 159. Almquist Joachim et al. Overexpressing cell systems are a competitive option to primary adipocytes when predicting in vivo potency of dual GPR81/GPR109A agonists. (2018) EUROPEAN JOURNAL OF PHARMACEUTICAL SCIENCES 0928-0987 1879-0720 114 155-165 160. Masuda Yusuke et al. Overexpressing human GPR109A leads to pronounced reduction in plasma triglyceride levels in BAC transgenic rats. (2018) ATHEROSCLEROSIS 0021-9150 272 182- 192 161. Langbein Kerstin et al. State marker properties of niacin skin sensitivity in ultra-high risk groups for psychosis - An optical reection spectroscopy study. (2018) SCHIZOPHRENIA RESEARCH 0920-9964 1573-2509 192 377-384 162. Recio Carlota et al. The Role of Metabolite-Sensing G Protein- Coupled Receptors in Inammation and Metabolic Disease. (2018) ANTIOXIDANTS & REDOX SIGNALING 1523-0864 1557-7716 29 3 237-256 163. Xu Chuangye et al. An investigation of calcium-independent phospholipase A2 (iPLA2) and cytosolic phospholipase A2 (cPLA2) in schizophrenia. (2019) PSYCHIATRY RESEARCH 0165-1781 273 782-787 164. Gafson Arie et al. Breaking the cycle Reversal of ux in the tricarboxylic acid cycle by dimethyl fumarate. (2019) NEUROLOGY-NEUROIMMUNOLOGY & NEUROINFLAMMATION 2332-7812 6 3 165. Strømland Øyvind et al. Keeping the balance in NAD metabolism. (2019) BIOCHEMICAL SOCIETY TRANSACTIONS 0300-5127 1470- 8752 47 1 119-130 166. Connell Niels et al. NAD(+) metabolism as a target for metabolic health: have we found the silver bullet?. (2019) DIABETOLOGIA 0012-186X 1432-0428 62 6 888-899 167. * Markovics Arnold et al. Nicotinic acid suppresses sebaceous lipogenesis of human sebocytes via activating hydroxycarboxylic acid receptor 2 (HCA(2)). (2019) JOURNAL OF CELLULAR AND MOLECULAR MEDICINE 1582-1838 1582-4934 9 23 6203-6214 168. Trotta Maria et al. The activation of retinal HCA2 receptors by systemic beta-hydroxybutyrate inhibits diabetic retinal damage through reduction of endoplasmic reticulum stress and the NLRP3 inammasome. (2019) PLOS ONE 1932-6203 1932-6203 14 1

133. Benyó, Z ; Gille, A ; Kero, J ; Csiky, M ; Suchánková, MC ; Nüsing, RM ; Moers, A ; Pfeer, K ; Oermanns, S Mechanism of nicotinic acid-induced cutaneous vasodilation FASEB JOURNAL 20 : 4 p. A665 (2006) WoS Teljes dokumentum Közlemény:1856281 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

134. Ruisanchez, É ; Őrsy, P ; Németh, T ; Sándor, P ; Benyó, Z Eects of sphingolipid mediators on the vascular reactivity. ACTA PHYSIOLOGICA HUNGARICA 93 pp. 225-226. , 2 p. (2006) Közlemény:1856282 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat ) 135. Wirth, A ; Benyó, Z ; Moers, A ; Leutgeb, B ; Gorbey, S ; Lemmer, B ; Wettschureck, N ; Oermanns, S Gq/11 is required for maintenance of basal blood pressure whereas Gq/11 and G12/13 are important in DOCA/salt-induced hypertension. NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 372 : Suppl. 1 Paper: 155 (2006) DOI WoS Közlemény:1856286 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

136. Benyó, B ; Benyó, Z ; Benyó, Z jr ; Somogyi, P ; Paláncz, B Classication of cerebral blood ow oscillation using SVM classiers with dierent kernels. In: Elmenreich, W; Tenreiro, Machado JA; Rudas, IJ (szerk.) Intelligent Systems at the Service of Mankind. Volume II Augsburg, Németország : UBooks, (2005) pp. 417-427. , 11 p. Közlemény:1847932 Admin láttamozott Forrás Könyvrészlet (Szaktanulmány )

137. Benyo, Z ; Gille, A ; Kero, J ; Csiky, M ; Suchankova, MC ; Nusing, RM ; Moers, A ; Pfeer, K ; Oermanns, S GPR109A (PUMA-G/HM74A) mediates nicotinic acid-induced ushing JOURNAL OF CLINICAL INVESTIGATION 115 : 12 pp. 3634-3640. , 7 p. (2005) DOI WoS Scopus PubMed Teljes dokumentum Közlemény:1949002 Érvényesített Forrás Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 250 Független: 231 Függő: 19

1. Pike NB. Flushing out the role of GPR109A (HM74A) in the clinical ecacy of nicotinic acid. (2005) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 115 12 3400-3403 2. Cheng K et al. Antagonism of the prostaglandin D-2 receptor 1 suppresses nicotinic acid-induced vasodilation in mice and humans. (2006) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 0027-8424 1091- 6490 103 17 6682-6687 3. Oberwittler H et al. Clinical evidence for use of acetyl salicylic acid in control of ushing related to nicotinic acid treatment. (2006) INTERNATIONAL JOURNAL OF CLINICAL PRACTICE 1368-5031 60 6 707-715 4. Ganji Sbobba et al. Eect of niacin on lipoproteins and atherosclerosis. (2006) FUTURE LIPIDOLOGY 1746-0875 1746- 0883 1758-4299 1758-4302 1 5 549-557 5. Tang YT et al. Enhancement of arachidonic acid signaling pathway by nicotinic acid receptor HM74A. (2006) BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS 0006-291X 1090- 2104 345 1 29-37 6. Holinstat Michael et al. G-protein-coupled receptors: evolving views on physiological signalling - Symposium on G-protein- coupled receptors: Evolving concepts and new techniques. (2006) EMBO REPORTS 1469-221X 1469-3178 7 9 866-869 7. Pelton P. GW-501516 - GlaxoSmithKline/Ligand. (2006) CURRENT OPINION IN INVESTIGATIONAL DRUGS 1472-4472 2040-3429 7 4 360-370 8. Sirtori Cesare. HDL and the progression of atherosclerosis: new insights. (2006) EUROPEAN HEART JOURNAL SUPPLEMENTS 1520- 765X 1554-2815 8 F F4-F9 9. Maciejewski-Lenoir Dominique et al. Langerhans cells release prostaglandin D-2 in response to nicotinic acid. (2006) JOURNAL OF INVESTIGATIVE DERMATOLOGY 0022-202X 1523-1747 126 12 2637-2646 10. Roosterman Dirk et al. Neuronal control of skin function: The skin as a neuroimmunoendocrine organ. (2006) PHYSIOLOGICAL REVIEWS 0031-9333 1522-1210 86 4 1309-1379 11. Carlson LA. Nicotinic acid and other therapies for raising high- density lipoprotein. (2006) CURRENT OPINION IN CARDIOLOGY 0268-4705 1531-7080 21 4 336-344 12. * Benyo Z et al. Nicotinic acid-induced ushing is mediated by activation of epidermal langerhans cells. (2006) MOLECULAR PHARMACOLOGY 0026-895X 1521-0111 70 6 1844-1849 13. * Oermanns Stefan. The nicotinic acid receptor GPR109A (HM74A or PUMA-G) as a new therapeutic target. (2006) TRENDS IN PHARMACOLOGICAL SCIENCES 0165-6147 27 7 384-390 14. Chlopicki S et al. 1-methylnicotinamide (MNA), a primary metabolite of nicotinamide, exerts anti-thrombotic activity mediated by a cyclooxygenase-2/prostacyclin pathway. (2007) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 152 2 230-239 15. Skinner Philip et al. 3-Nitro-4-amino benzoic acids and 6-amino nicotinic acids are highly selective agonists of GPR109b. (2007) BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 0960-894X 1464- 3405 17 23 6619-6622 16. Yu Bi-lian et al. Anti-inammatory eect is an important property of niacin on atherosclerosis beyond its lipid-altering eects. (2007) MEDICAL HYPOTHESES 0306-9877 1532-2777 69 1 90-94 17. Torhan April et al. Cloning and characterization of the hamster and guinea pig nicotinic acid receptors. (2007) JOURNAL OF LIPID RESEARCH 0022-2275 1539-7262 48 9 2065-2071 18. Carballo-Jane Ester et al. Comparison of rat and dog models of vasodilatation and lipolysis for the calculation of a therapeutic index for GPR109A agonists. (2007) JOURNAL OF PHARMACOLOGICAL AND TOXICOLOGICAL METHODS 1056-8719 56 3 308-316 19. Shen Hong et al. Discovery of orally bioavailable and novel urea agonists of the high anity niacin receptor GPR109A. (2007) BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 0960-894X 1464- 3405 17 24 6723-6728 20. McKenney James. Eect of drugs on high-density lipoprotein. (2007) JOURNAL OF CLINICAL LIPIDOLOGY 1933-2874 1 1 74-87 21. Brown B. Expert commentary: Niacin safety. (2007) AMERICAN JOURNAL OF CARDIOLOGY 0002-9149 1879-1913 99 6 Suppl 1 32C-S34 22. Skinner Philip et al. Fluorinated pyrazole acids are agonists of the high anity niacin receptor GPR109a. (2007) BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 0960-894X 1464-3405 17 20 5620-5623 23. Pal Manojit et al. HDL elevators and mimetics - Emerging therapies for atherosclerosis. (2007) CARDIOVASCULAR AND HEMATOLOGICAL AGENTS IN MEDICINAL CHEMISTRY 1871-5257 1875-6182 5 1 55-66 24. Zhou Lubing et al. Human epidermoid A431 cells express functional nicotinic Acid receptor HM74a. (2007) MOLECULAR AND CELLULAR BIOCHEMISTRY 0300-8177 1573-4919 294 1-2 243-248 25. Rader Daniel. Mechanisms of disease: HDL metabolism as a target for novel therapies. (2007) NATURE CLINICAL PRACTICE CARDIOVASCULAR MEDICINE 1743-4297 1743-4300 1759-5010 4 2 102-109 26. Belenky P et al. NAD(+) metabolism in health and disease. (2007) TRENDS IN BIOCHEMICAL SCIENCES 0968-0004 32 1 12-19 27. Guyton John. Niacin in cardiovascular prevention: mechanisms, ecacy, and safety. (2007) CURRENT OPINION IN LIPIDOLOGY 0957-9672 1473-6535 18 4 415-420 28. Mueller Daniela et al. Niacin lowers serum phosphate and increases HDL cholesterol in dialysis patients. (2007) CLINICAL JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY 1555- 9041 1555-905X 2 6 1249-1254 29. Belenky Peter et al. Nicotinamide riboside promotes Sir2 silencing and extends lifespan via Nrk and Urh1/Pnp1/Meu1 pathways to NAD(+). (2007) CELL 0092-8674 1097-4172 129 3 473-484 30. * Ahmed K. Nicotinic acid in disorders of lipid metabolism. (2007) PHARMAZIE IN UNSERER ZEIT 0048-3664 36 2 120-125 31. Meyers C et al. Nicotinic acid induces secretion of prostaglandin D2 in human macrophages: An in vitro model of the niacin ush. (2007) ATHEROSCLEROSIS 0021-9150 192 2 253-258 32. Kamanna Vaijinath et al. Nicotinic acid (niacin) receptor agonists: Will they be useful therapeutic agents?. (2007) AMERICAN JOURNAL OF CARDIOLOGY 0002-9149 1879-1913 100 11 Suppl S53-S61 33. Richman Jeremy et al. Nicotinic acid receptor agonists dierentially activate downstream eectors. (2007) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 282 25 18028- 18036 34. Soudijn Willem et al. Nicotinic acid receptor subtypes and their ligands. (2007) MEDICINAL RESEARCH REVIEWS 0198-6325 1098- 1128 27 3 417-433 35. Schmitz Gerd et al. Pharmacogenetics and pharmacogenomics of cholesterol-lowering therapy. (2007) CURRENT OPINION IN LIPIDOLOGY 0957-9672 1473-6535 18 2 164-173 36. Penberthy W. Pharmacological targeting of IDO-mediated tolerance for treating autoimmune disease. (2007) CURRENT DRUG METABOLISM 1389-2002 1875-5453 8 3 245-266 37. Carballo-Jane E et al. Potential role for epidermal Langerhans cells in nicotinic acid-induced vasodilatation in the mouse. (2007) INFLAMMATION RESEARCH 1023-3830 1420-908X 56 6 254-261 38. Semple Graeme et al. Recent progress in the discovery of niacin receptor agonists. (2007) CURRENT OPINION IN DRUG DISCOVERY & DEVELOPMENT 1367-6733 10 4 452-459 39. Lai E et al. Suppression of niacin-induced vasodilation with an antagonist to prostaglandin D-2 receptor subtype 1. (2007) CLINICAL PHARMACOLOGY & THERAPEUTICS 0009-9236 1532- 6535 81 6 849-857 40. Steinberg D. The Cholesterol Wars. (2007) ISBN:9780123739797 41. Stern Ralph. The role of nicotinic acid metabolites in ushing and hepatotoxicity. (2007) JOURNAL OF CLINICAL LIPIDOLOGY 1933- 2874 1 3 191-193 42. Semple Graeme et al. 3-(1H-tetrazol-5-yl)-1,4,5,6-tetrahydro- cyclopentapyrazole (MK-0354): A partial agonist of the nicotinic acid receptor, G-protein coupled receptor 109a, with antilipolytic but no vasodilatory activity in mice. (2008) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 51 16 5101-5108 43. Ge Hongfei et al. Activation of G protein-coupled receptor 43 in adipocytes leads to inhibition of lipolysis and suppression of plasma free fatty acids. (2008) ENDOCRINOLOGY 0013-7227 1945-7170 149 9 4519-4526 44. Lafontan M. Advances in adipose tissue metabolism. (2008) INTERNATIONAL JOURNAL OF OBESITY 0307-0565 1476-5497 32 S39-S51 45. Regard Jean et al. Anatomical Proling of G Protein-Coupled Receptor Expression. (2008) CELL 0092-8674 1097-4172 135 3 561-571 46. Tall A. Cholesterol eux pathways and other potential mechanisms involved in the athero-protective eect of high density lipoproteins. (2008) JOURNAL OF INTERNAL MEDICINE 0954-6820 1365-2796 263 3 256-273 47. Tiwari A. Current and emerging paradigms in the therapeutic management of atherosclerosis. (2008) EXPERT OPINION ON THERAPEUTIC TARGETS 1472-8222 1744-7631 12 12 1523-1546 48. Volek Je et al. Dietary carbohydrate restriction induces a unique metabolic state positively aecting atherogenic dyslipidemia, fatty acid partitioning, and metabolic syndrome. (2008) PROGRESS IN LIPID RESEARCH 0163-7827 47 5 307-318 49. Valencia C. Efectividad y seguridad del ácido nicotínico en el tratamiento de la hiperlipidemia asociada a hiperfosfatemia en pacientes con enfermedad renal crónica. (2008) NEFROLOGIA 0211-6995 28 1 61-66 50. Ge H et al. Elucidation of signaling and functional activities of an orphan GPCR, GPR81. (2008) JOURNAL OF LIPID RESEARCH 0022- 2275 1539-7262 49 4 797-803 51. Vo AN et al. Fixed-dose combination of extended-release niacin plus simvastatin for lipid disorders. (2008) EXPERT REVIEW OF CARDIOVASCULAR THERAPY 1477-9072 6 10 1303-1310 52. Taylor Allen. Flushing and the HDL-C response to extended- release niacin. (2008) JOURNAL OF CLINICAL LIPIDOLOGY 1933- 2874 2 4 285-288 53. Ferns Gordon. HDL-cholesterol modulation and its impact on the management of cardiovascular risk. (2008) ANNALS OF CLINICAL BIOCHEMISTRY 0004-5632 1758-1001 45 2 122-128 54. Pessler Frank et al. Identication of novel monosodium urate crystal regulated mRNAs by transcript proling of dissected murine air pouch membranes. (2008) ARTHRITIS RESEARCH & THERAPY 1478-6354 1478-6362 10 3 55. Ferns G et al. Investigation and management of hypertriglyceridaemia. (2008) JOURNAL OF CLINICAL PATHOLOGY 0021-9746 1472-4146 61 11 1174-1183 56. Kamanna Vaijinath et al. Mechanism of action of niacin. (2008) AMERICAN JOURNAL OF CARDIOLOGY 0002-9149 1879-1913 101 8 Suppl S20-S26 57. Kostylina G. Neutrophil apoptosis mediated by nicotinic acid receptors (GPR109A). (2008) CELL DEATH AND DIFFERENTIATION 1350-9047 1476-5403 15 1 134-142 58. van der Hoorn Jose. Niacin Increases HDL by Reducing Hepatic Expression and Plasma Levels of Cholesteryl Ester Transfer Protein in APOE*3Leiden.CETP Mice. (2008) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 28 11 2016-2022 59. Papaliodis Dean. Niacin-induced "Flush" Involves Release of Prostaglandin D-2 from Mast Cells and Serotonin from Platelets: Evidence from Human Cells in Vitro and an Animal Model. (2008) JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS 0022-3565 1521-0103 327 3 665-672 60. Davidson Michael. Niacin use and cutaneous ushing: Mechanisms and strategies for prevention. (2008) AMERICAN JOURNAL OF CARDIOLOGY 0002-9149 1879-1913 101 8 Suppl S14-S19 61. * Bodor E. Nicotinic acid: an old drug with a promising future. (2008) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476- 5381 153 Suppl 1 S68-S75 62. * Gille Andreas. Nicotinic acid: Pharmacological eects and mechanisms of action. (2008) ANNUAL REVIEW OF PHARMACOLOGY AND TOXICOLOGY 0362-1642 48 79-106 63. Kamanna Vaijinath. Nicotinic acid: recent developments. (2008) CURRENT OPINION IN CARDIOLOGY 0268-4705 1531-7080 23 4 393-398 64. Boatman P. Nicotinic Acid Receptor Agonists. (2008) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 51 24 7653-7662 65. Hausenloy D. Targeting residual cardiovascular risk: raising high- density lipoprotein cholesterol levels. (2008) HEART 1355-6037 94 6 706-714 66. Hausenloy D et al. Targeting residual cardiovascular risk: raising high-density lipoprotein cholesterol levels (Reprinted from Heart, vol 94, pg 706-14, 2008). (2008) POSTGRADUATE MEDICAL JOURNAL 0032-5473 1469-0756 84 997 590-598 67. Papaliodis D. The avonoid luteolin inhibits niacin-induced ush. (2008) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476- 5381 153 7 1382-1387 68. Miller Christine. The high-anity niacin receptor HM74A is decreased in the anterior cingulate cortex of individuals with schizophrenia. (2008) BRAIN RESEARCH BULLETIN 0361-9230 1873-2747 77 1 33-41 69. Suryadevara RS et al. Use of extended-release niacin in clinical practice. (2008) FUTURE LIPIDOLOGY 1746-0875 1746-0883 1758- 4299 1758-4302 3 1 9-16 70. Skinner Philip. 5-N,N-Disubstituted 5-aminopyrazole-3-carboxylic acids are highly potent agonists of GPR109b. (2009) BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 0960-894X 1464-3405 19 15 4207-4209 71. Walters Robert. beta-Arrestin1 mediates nicotinic acid-induced ushing, but not its antilipolytic eect, in mice. (2009) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 119 5 1312- 1321 72. Shen Hong. Discovery of Novel Tricyclic Full Agonists for the G- Protein-Coupled Niacin Receptor 109A with Minimized Flushing in Rats. (2009) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 52 8 2587-2602 73. Dishy Victor. Eects of Aspirin When Added to the Prostaglandin D-2 Receptor Antagonist Laropiprant on Niacin-Induced Flushing Symptoms. (2009) JOURNAL OF CLINICAL PHARMACOLOGY 0091- 2700 1552-4604 49 4 416-422 74. Linke Axel. Eects of extended-release niacin on lipid prole and adipocyte biology in patients with impaired glucose tolerance. (2009) ATHEROSCLEROSIS 0021-9150 205 1 207-213 75. Greenfeder Scott. Emerging Strategies and Agents to Lower Cardiovascular Risk by Increasing High Density Lipoprotein Cholesterol Levels. (2009) CURRENT MEDICINAL CHEMISTRY 0929-8673 1875-533X 16 2 144-156 76. Hausenloy Derek. Enhancing cardiovascular disease risk reduction: raising high-density lipoprotein levels. (2009) CURRENT OPINION IN CARDIOLOGY 0268-4705 1531-7080 24 5 473-482 77. * Ahmed Kashan. GPR109A, GPR109B and GPR81, a family of hydroxy-carboxylic acid receptors. (2009) TRENDS IN PHARMACOLOGICAL SCIENCES 0165-6147 30 11 557-562 78. Martres Paul. HM74a Agonists: Will they be the New Generation of Nicotinic Acid?. (2009) CURRENT TOPICS IN MEDICINAL CHEMISTRY 1568-0266 1873-4294 9 5 428-435 79. Schweikart Jennifer. Investigation of niacin on parameters of metabolism in a physiologic dose: randomized, double-blind clinical trial with three dierent dosages. (2009) INTERNATIONAL JOURNAL OF FOOD SCIENCES AND NUTRITION 0963-7486 1465- 3478 60 192-202 80. Goumas Georgios. Is There Evidence-based Hypolipidemic Treatment With Clinical Benet Beyond Statins?. (2009) ANGIOLOGY 0003-3197 60 1 93-98 81. Liu Changlu et al. Lactate Inhibits Lipolysis in Fat Cells through Activation of an Orphan G-protein-coupled Receptor, GPR81. (2009) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083- 351X 284 5 2811-2822 82. Lauring Brett. Laropiprant in Combination With Extended-Release Niacin Does Not Alter Urine 11-Dehydrothromboxane B-2, a Marker of In Vivo Platelet Function, in Healthy, Hypercholesterolemic, and Diabetic Subjects. (2009) JOURNAL OF CLINICAL PHARMACOLOGY 0091-2700 1552-4604 49 12 1426- 1435 83. Sood Aditya. Mechanisms of Flushing Due to Niacin and Abolition of These Eects. (2009) JOURNAL OF CLINICAL HYPERTENSION 1524-6175 11 11 685-689 84. Benavente Claudia. NAD in Skin: Therapeutic Approaches for Niacin. (2009) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873-4286 15 1 29-38 85. Kamanna Vaijinath. Niacin: An Old Drug Rejuvenated. (2009) CURRENT ATHEROSCLEROSIS REPORTS 1523-3804 11 1 45-51 86. Vosper Helen. Niacin: a re-emerging pharmaceutical for the treatment of dyslipidaemia. (2009) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 158 2 429-441 87. Brown BG et al. Nicotinic Acid. (2009) Megjelent: Clinical Lipidology: A Companion to Braunwald's Heart Disease pp. 298- 314 88. Shen Hong. Novel patent publications on high-anity nicotinic acid receptor agonists. (2009) EXPERT OPINION ON THERAPEUTIC PATENTS 1354-3776 19 7 957-967 89. Raal Frederick. Pathogenesis and Management of the Dyslipidemia of the Metabolic Syndrome. (2009) METABOLIC SYNDROME AND RELATED DISORDERS 1540-4196 7 2 83-88 90. Ren Ning. Phenolic acids suppress adipocyte lipolysis via activation of the nicotinic acid receptor GPR109A (HM74a/PUMA- G). (2009) JOURNAL OF LIPID RESEARCH 0022-2275 1539-7262 50 5 908-914 91. Parhofer KG. Review of extended-release niacin/laropiprant xed combination in the treatment of mixed dyslipidemia and primary hypercholesterolemia. (2009) VASCULAR HEALTH AND RISK MANAGEMENT 1176-6344 1178-2048 5 901-908 92. Ewald Nils. Severe hypertriglyceridemia and pancreatitis: presentation and management. (2009) CURRENT OPINION IN LIPIDOLOGY 0957-9672 1473-6535 20 6 497-504 93. Nilsson B. Test-retest stability of the oral niacin test and electrodermal activity in patients with schizophrenia. (2009) PROSTAGLANDINS LEUKOTRIENES AND ESSENTIAL FATTY ACIDS 0952-3278 81 5-6 367-372 94. Miller Christine. The Evolution of Schizophrenia: A Model for Selection by Infection, with a Focus on NAD. (2009) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873-4286 15 1 100-109 95. Penberthy W et al. The Importance of NAD in Multiple Sclerosis. (2009) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873- 4286 15 1 64-99 96. Kamanna V et al. The mechanism and mitigation of niacin- induced ushing. (2009) INTERNATIONAL JOURNAL OF CLINICAL PRACTICE 1368-5031 63 9 1369-1377 97. Isaksson Christine. Turnover modeling of non-esteried fatty acids in rats after multiple intravenous infusions of nicotinic acid. (2009) DOSE-RESPONSE 1559-3258 7 3 247-269 98. Duy Danielle. Update on strategies to increase HDL quantity and function. (2009) NATURE REVIEWS CARDIOLOGY 1759-5002 1759- 5010 6 7 455-463 99. Jacobson Terry. A "Hot" Topic in Dyslipidemia Management-"How to Beat a Flush": Optimizing Niacin Tolerability to Promote Long- term Treatment Adherence and Coronary Disease Prevention. (2010) MAYO CLINIC PROCEEDINGS 0025-6196 85 4 365-379 100. Shen Hong. Discovery of a Biaryl Cyclohexene Carboxylic Acid (MK-6892): A Potent and Selective High Anity Niacin Receptor Full Agonist with Reduced Flushing Proles in Animals as a Preclinical Candidate. (2010) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 53 6 2666-2670 101. Mandrika Ilona. Evidence for constitutive dimerization of niacin receptor subtypes. (2010) BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS 0006-291X 1090-2104 395 2 281- 287 102. Tiwari Atul. GPR43: An emerging target for the potential treatment of type 2 diabetes, obesity and insulin resistance. (2010) CURRENT OPINION IN INVESTIGATIONAL DRUGS 1472- 4472 2040-3429 11 4 385-393 103. Olsson A. HDL and LDL as therapeutic targets for cardiovascular disease prevention: The possible role of niacin. (2010) NUTRITION METABOLISM AND CARDIOVASCULAR DISEASES 0939-4753 1590- 3729 20 8 553-557 104. Bruckert E et al. HDL-cholestérol et risque cardiovasculaire: Au- delà du LDL! [HDL cholesterol and cardiovascular risk: Beyond LDL!]. (2010) MEDECINE DES MALADIES METABOLIQUES 1957- 2557 4 4 379-388 105. Shen HC et al. High-Anity Niacin Receptor GPR109A Agonists. (2010) Megjelent: ANNUAL REPORTS IN MEDICINAL CHEMISTRY, VOL 45 pp. 72-94 106. Natarajan Pradeep. High-Density Lipoprotein and Coronary Heart Disease Current and Future Therapies. (2010) JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY 0735-1097 1558-3597 55 13 1283-1299 107. Meurs I. High-Density Lipoprotein: Key Molecule in Cholesterol Eux and the Prevention of Atherosclerosis. (2010) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873-4286 16 13 1445- 1467 108. Spillmann Frank et al. High-Density Lipoprotein-Raising Strategies: Update 2010. (2010) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873-4286 16 13 1517-1530 109. Li Guo. Internalization of the Human Nicotinic Acid Receptor GPR109A Is Regulated by G(i), GRK2, and Arrestin3. (2010) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 285 29 22605-22618 110. Olsson Anders. Laropiprant plus niacin for dyslipidemia and prevention of cardiovascular disease. (2010) EXPERT OPINION ON PHARMACOTHERAPY 1465-6566 1744-7666 11 10 1715-1726 111. Li Xiaoyu. Modulation of HDL metabolism by the niacin receptor GPR109A in mouse hepatocytes. (2010) BIOCHEMICAL PHARMACOLOGY 0006-2952 1873-2968 80 9 1450-1457 112. Luthi Andrea. Nanotechnology for synthetic high-density lipoproteins. (2010) TRENDS IN MOLECULAR MEDICINE 1471-4914 1471-499X 16 12 553-560 113. Montecucco F et al. New evidence for nicotinic acid treatment to reduce atherosclerosis. (2010) EXPERT REVIEW OF CARDIOVASCULAR THERAPY 1477-9072 8 10 1457-1467 114. Chapman M. Niacin and brates in atherogenic dyslipidemia: Pharmacotherapy to reduce cardiovascular risk. (2010) PHARMACOLOGY & THERAPEUTICS 0163-7258 1879-016X 126 3 314-345 115. Sanyal S et al. Niacin and laropiprant. (2010) DRUGS OF TODAY 1699-3993 46 6 371-378 116. Messamore Erik. Niacin sensitivity and the arachidonic acid pathway in schizophrenia. (2010) SCHIZOPHRENIA RESEARCH 0920-9964 1573-2509 122 1-3 248-256 117. * Hanson Julien. Nicotinic acid- and monomethyl fumarate-induced ushing involves GPR109A expressed by keratinocytes and COX- 2-dependent prostanoid formation in mice. (2010) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 120 8 2910-2919 118. Vaadaki Elizabeth et al. Pharmacogenetically Tailored Treatments for Heart Disease. (2010) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873-4286 16 20 2194- 2213 119. Peters Jens-Uwe. Pyrido pyrimidinones as selective agonists of the high anity niacin receptor GPR109A: Optimization of in vitro activity. (2010) BIOORGANIC & MEDICINAL CHEMISTRY LETTERS 0960-894X 1464-3405 20 18 5426-5430 120. Hemandez Carlos. Regulation of Hepatic ApoC3 Expression by PGC-1 beta Mediates Hypolipidemic Eect of Nicotinic Acid. (2010) CELL METABOLISM 1550-4131 12 4 411-419 121. Dunbar Richard. Seeing red: ushing out instigators of niacin- associated skin toxicity. (2010) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 120 8 2651-2655 122. Inamadugu Jaswanth. Simultaneous determination of niacin and its metabolites-nicotinamide, nicotinuric acid and N-methyl-2- pyridone-5-carboxamide-in human plasma by LC-MS/MS and its application to a human pharmacokinetic study. (2010) BIOMEDICAL CHROMATOGRAPHY 0269-3879 1099-0801 24 10 1059-1074 123. Zheng Xiaowan. Synthesis and in vitro evaluation of potential sustained release prodrugs via targeting ASBT. (2010) INTERNATIONAL JOURNAL OF PHARMACEUTICS 0378-5173 1873- 3476 396 1-2 111-118 124. Failor RA. Use of Lipid-Lowering Agents in the Prevention of Cardiovascular Disease. (2010) Megjelent: Evidence-Based Cardiology: Third Edition pp. 147-164 125. DeWire Scott et al. Biased Ligands for Better Cardiovascular Drugs Dissecting G-Protein-Coupled Receptor Pharmacology. (2011) CIRCULATION RESEARCH 0009-7330 1524-4571 109 2 205- 216 126. * Blad CC et al. Biological and Pharmacological Roles of HCA Receptors. (2011) ADVANCES IN PHARMACOLOGY 1054-3589 62 219-250 127. Ahmed K. Biological roles and therapeutic potential of hydroxy- carboxylic acid receptors. (2011) FRONTIERS IN ENDOCRINOLOGY 1664-2392 2 128. Saggini A. Cholesterol and vitamins: Revisited study. (2011) JOURNAL OF BIOLOGICAL REGULATORS AND HOMEOSTATIC AGENTS 0393-974X 1724-6083 25 4 505-515 129. Li Guo. Distinct Kinetic and Spatial Patterns of Protein Kinase C (PKC)- and Epidermal Growth Factor Receptor (EGFR)-dependent Activation of Extracellular Signal-regulated Kinases 1 and 2 by Human Nicotinic Acid Receptor GPR109A. (2011) JOURNAL OF BIOLOGICAL CHEMISTRY 0021-9258 1083-351X 286 36 31199- 31212 130. Vosper H. Extended release niacin-laropiprant in patients with hypercholesterolemia or mixed dyslipidemias improves clinical parameters. (2011) CLINICAL MEDICINE INSIGHTS: CARDIOLOGY 1179-5468 5 85-101 131. Wanders D. Future of GPR109A agonists in the treatment of dyslipidaemia. (2011) DIABETES OBESITY & METABOLISM 1462- 8902 1463-1326 13 8 685-691 132. Bruckert E et al. HDL-cholestérol et approche thérapeutique [HDL-cholesterol and treatment pathway]. (2011) ARCHIVES OF CARDIOVASCULAR DISEASES SUPPLEMENTS 1878-6480 3 4 273- 279 133. Kwong Alan. High Dietary Niacin May Increase Prostaglandin Formation but Does Not Increase Tumor Formation in Apc(Min/+) Mice. (2011) NUTRITION AND CANCER : AN INTERNATIONAL JOURNAL 0163-5581 1532-7914 63 6 950-959 134. * Oermanns Stefan et al. International Union of Basic and Clinical Pharmacology. LXXXII: Nomenclature and Classication of Hydroxy-carboxylic Acid Receptors (GPR81, GPR109A, and GPR109B). (2011) PHARMACOLOGICAL REVIEWS 0031-6997 1521- 0081 63 2 269-290 135. Marar D et al. Mechanism of Action of Niacin: Implications for Atherosclerosis and Drug Discovery. (2011) Megjelent: Metabolic Syndrome: Underlying Mechanisms and Drug Therapies pp. 235- 251 136. Sharma Anjali. Multivalent niacin nanoconjugates for delivery to cytoplasmic lipid droplets. (2011) BIOMATERIALS 0142-9612 1878- 5905 32 5 1419-1429 137. Cantó C et al. NAD + as a signaling molecule modulating metabolism. (2011) COLD SPRING HARBOR SYMPOSIA ON QUANTITATIVE BIOLOGY 0091-7451 76 291-298 138. Mullangi Ramesh. Niacin and its metabolites: role of LC-MS/MS bioanalytical methods and update on clinical pharmacology. An overview. (2011) BIOMEDICAL CHROMATOGRAPHY 0269-3879 1099-0801 25 1-2 218-237 139. Li Hong-ming. Nicotinic Acid Inhibits Glucose-Stimulated Insulin Secretion Via the G Protein-Coupled Receptor PUMA-G in Murine Islet beta Cells. (2011) PANCREAS 0885-3177 1536-4828 40 4 615- 621 140. * Lukasova Martina et al. Nicotinic acid (niacin): new lipid- independent mechanisms of action and therapeutic potentials. (2011) TRENDS IN PHARMACOLOGICAL SCIENCES 0165-6147 32 12 700-707 141. Bermudez Yira. Nicotinic Acid Receptor Abnormalities in Human Skin Cancer: Implications for a Role in Epidermal Dierentiation. (2011) PLOS ONE 1932-6203 1932-6203 6 5 142. Babcock Michael. Persistent erythema with niacin is not attributable to aspirin resistance. (2011) JOURNAL OF CLINICAL LIPIDOLOGY 1933-2874 5 2 114-119 143. Prousky J et al. Pharmacologic use of Niacin. (2011) JOURNAL OF EVIDENCE-BASED COMPLEMENTARY AND ALTERNATIVE MEDICINE 2156-5872 16 2 91-101 144. Kuei Chester. Study of GPR81, the Lactate Receptor, from Distant Species Identies Residues and Motifs Critical for GPR81 Functions. (2011) MOLECULAR PHARMACOLOGY 0026-895X 1521- 0111 80 5 848-858 145. Kei Anastazia. What Restricts the Clinical Use of Nicotinic Acid?. (2011) CURRENT VASCULAR PHARMACOLOGY 1570-1611 9 4 521- 530 146. Liu Changlu et al. 3,5-Dihydroxybenzoic Acid, a Specic Agonist for Hydroxycarboxylic Acid 1, Inhibits Lipolysis in Adipocytes. (2012) JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS 0022-3565 1521-0103 341 3 794-801 147. Zhou Q et al. Activated human hydroxy-carboxylic acid receptor-3 signals to MAP kinase cascades via the PLC-dependent PKC and MMP-mediated EGFR pathways. (2012) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 166 6 1756-1773 148. Dunatchik Andrew. A systematic review on evidence of the eectiveness and safety of a wax-matrix niacin formulation. (2012) JOURNAL OF CLINICAL LIPIDOLOGY 1933-2874 6 2 121-131 149. VanHorn J et al. Attenuation of niacin-induced prostaglandin D 2 generation by omega-3 fatty acids in THP-1 macrophages and Langerhans dendritic cells. (2012) JOURNAL OF INFLAMMATION RESEARCH 1178-7031 5 1 37-50 150. Ibrahim Islam. beta-Arrestin-Mediated Signaling Improves the Ecacy of Therapeutics. (2012) JOURNAL OF PHARMACOLOGICAL SCIENCES 1347-8613 1347-8648 118 4 408-412 151. Li Zhaosha et al. Eects of pyrazole partial agonists on HCA2- mediated ushing and VLDL-triglyceride levels in mice. (2012) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 167 4 818-825 152. Yadav Rahul. Extended-release niacin with laropiprant: a review on ecacy, clinical eectiveness and safety. (2012) EXPERT OPINION ON PHARMACOTHERAPY 1465-6566 1744-7666 13 9 1345-1362 153. * Blad CC et al. G protein-coupled receptors for energy metabolites as new therapeutic targets. (2012) NATURE REVIEWS DRUG DISCOVERY 1474-1776 11 8 603-619 154. Mureddu Gian et al. High-density lipoprotein levels and risk of cardiovascular events: a review. (2012) JOURNAL OF CARDIOVASCULAR MEDICINE 1558-2027 13 9 575-586 155. Li Guo et al. Identication and Characterization of Distinct C- Terminal Domains of the Human Hydroxycarboxylic Acid Receptor-2 That Are Essential for Receptor Export, Constitutive Activity, Desensitization, and Internalization. (2012) MOLECULAR PHARMACOLOGY 0026-895X 1521-0111 82 6 1150-1161 156. Dvorak Curt et al. Identication of Hydroxybenzoic Acids as Selective Lactate Receptor (GPR81) Agonists with Antilipolytic Eects. (2012) ACS MEDICINAL CHEMISTRY LETTERS 1948-5875 3 8 637-639 157. Ledwidge Mark. In vivo impact of prodrug isosorbide-5- nicotinate-2-aspirinate on lipids and prostaglandin D-2: Is this a new immediate-release therapeutic option for niacin?. (2012) ATHEROSCLEROSIS 0021-9150 221 2 478-483 158. Wrinkle S et al. Niacin delivery to the intestinal absorptive site impacts heat stress and productivity responses of high producing dairy cows during hot conditions. (2012) ANIMAL FEED SCIENCE AND TECHNOLOGY 0377-8401 175 1-2 33-47 159. Ingersoll Molly. Niacin inhibits skin dendritic cell mobilization in a GPR109A independent manner but has no impact on monocyte tracking in atherosclerosis. (2012) IMMUNOBIOLOGY 0171-2985 1878-3279 217 5 548-557 160. Lauring Brett et al. Niacin Lipid Ecacy Is Independent of Both the Niacin Receptor GPR109A and Free Fatty Acid Suppression. (2012) SCIENCE TRANSLATIONAL MEDICINE 1946-6234 4 148 161. Li Zhaosha et al. Niacin reduces plasma CETP levels by diminishing liver macrophage content in CETP transgenic mice. (2012) BIOCHEMICAL PHARMACOLOGY 0006-2952 1873-2968 84 6 821-829 162. Villines Todd. Niacin: The Evidence, Clinical Use, and Future Directions. (2012) CURRENT ATHEROSCLEROSIS REPORTS 1523- 3804 14 1 49-59 163. Kei Anastazia et al. Nicotinic acid: clinical considerations. (2012) EXPERT OPINION ON DRUG SAFETY 1474-0338 11 4 551-564 164. Huang Yan et al. Progess in mechanism of nicotinic acid mediated by nicotinic acid receptor GPR109A. (2012) CHINESE JOURNAL OF PHARMACOLOGY AND TOXICOLOGY / ZHONG GUO YAO LI XUE YU DU LI XUE ZA ZHI 1000-3002 26 1 105-107 165. Liang Dawei. Progress on cholesterol synthesis inhibitors as anti- atherosclerosis agents. (2012) Chinese Journal of Medicinal Chemistry 22 5 432-441 166. * Hanson Julien et al. Role of HCA(2) (GPR109A) in nicotinic acid and fumaric acid ester-induced eects on the skin. (2012) PHARMACOLOGY & THERAPEUTICS 0163-7258 1879-016X 136 1 1-7 167. Yadav Rahul. Safety and tolerability of extended-release niacin with laropiprant. (2012) EXPERT OPINION ON DRUG SAFETY 1474- 0338 11 1 151-159 168. Canto Carles. Targeting Sirtuin 1 to Improve Metabolism: All You Need Is NAD(+)?. (2012) PHARMACOLOGICAL REVIEWS 0031-6997 1521-0081 64 1 166-187 169. Canto Caries et al. The NAD(+) Precursor Nicotinamide Riboside Enhances Oxidative Metabolism and Protects against High-Fat Diet-Induced Obesity. (2012) CELL METABOLISM 1550-4131 15 6 838-847 170. Penberthy WT. The niacin ush pathway in recovery from schizophrenia and how arginine and glutamine may provide added benet. (2012) JOURNAL OF ORTHOMOLECULAR MEDICINE 0834-4825 27 1 29-38 171. Cantó C et al. Crosstalk between poly(ADP-ribose) polymerase and sirtuin enzymes. (2013) MOLECULAR ASPECTS OF MEDICINE 0098-2997 34 6 1168-1201 172. Gaidarov Ibragim et al. Dierential tissue and ligand-dependent signaling of GPR109A receptor: Implications for anti- atherosclerotic therapeutic potential. (2013) CELLULAR SIGNALLING 0898-6568 1873-3913 25 10 2003-2016 173. Lohoelter Malte et al. Eects of niacin supplementation and dietary concentrate proportion on body temperature, ruminal pH and milk performance of primiparous dairy cows. (2013) ARCHIVES OF ANIMAL NUTRITION 1745-039X 1477-2817 67 3 202- 218 174. * Tonack Sarah et al. Endogenous metabolites as ligands for G protein-coupled receptors modulating risk factors for metabolic and cardiovascular disease. (2013) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 304 4 H501-H513 175. Ananth Sudha et al. Induction of the Cystine/Glutamate Exchanger SLC7A11 in Retinal Pigment Epithelial Cells by the Antipsoriatic Drug Monomethylfumarate. (2013) INVESTIGATIVE OPHTHALMOLOGY AND VISUAL SCIENCE 0146-0404 1552-5783 54 3 1592-1602 176. Kaore SN et al. Niacin and niacin/laropiprant combination: Opening new hopes for dyslipidemia and cardiovascular diseases with few novel actions. (2013) INTERNATIONAL JOURNAL OF PHARMA AND BIO SCIENCES 0975-6299 4 3 P1053-P1078 177. Song W-L et al. Niacin, an old drug with a new twist. (2013) JOURNAL OF LIPID RESEARCH 0022-2275 1539-7262 54 10 2586- 2594 178. Parson Henri et al. Role of prostaglandin D-2 and the autonomic nervous system in niacin-induced ushing. (2013) JOURNAL OF DIABETES 1753-0393 1753-0407 5 1 59-67 179. Landmesser Ulf. The dicult search for a 'partner' of statins in lipid-targeted prevention of vascular events: the re-emergence and fall of niacin. (2013) EUROPEAN HEART JOURNAL 0195-668X 1522-9645 34 17 1254-1257 180. * Zandi-Nejad K et al. The role of HCA2 (GPR109A) in regulating macrophage function. (2013) FASEB JOURNAL 0892-6638 1530- 6860 27 11 4366-4374 181. Gouni-Berthold Ioanna et al. The Role of Niacin in Lipid-lowering Treatment: Are we Aiming Too High?. (2013) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873-4286 19 17 3094- 3106 182. Sheikh Sarah et al. Tolerability and Pharmacokinetics of Delayed- Release Dimethyl Fumarate Administered With and Without Aspirin in Healthy Volunteers. (2013) CLINICAL THERAPEUTICS 0149-2918 1879-114X 35 10 1582-1594 183. Marcovina Santica et al. Translating the basic knowledge of mitochondrial functions to metabolic therapy: role of L-carnitine. (2013) TRANSLATIONAL RESEARCH 1931-5244 1878-1810 161 2 73-84 184. Wakade C et al. A novel treatment target for Parkinson's disease. (2014) JOURNAL OF THE NEUROLOGICAL SCIENCES 0022-510X 1878-5883 347 1-2 34-38 185. Vadivel G. Butyrate-Mediated Protection Against Colonic Inammation And Colon Carcinogenesis: Role Of Butyrate Transporters And Butyrate Receptors. (2014) Megjelent: Butyrate: Food Sources, Functions and Health Benets pp. 157-176 186. Yu AL et al. Constitutive Expression of HCA(2) in Human Retina and Primary Human Retinal Pigment Epithelial Cells. (2014) CURRENT EYE RESEARCH 0271-3683 1460-2202 39 5 487-492 187. * Oermanns S. Free fatty acid (FFA) and hydroxy carboxylic acid (HCA) receptors. (2014) ANNUAL REVIEW OF PHARMACOLOGY AND TOXICOLOGY 0362-1642 54 407-434 188. Docherty KF. Genomics and Pharmacogenomics of Lipid- Lowering Therapies. (2014) Megjelent: Handbook of Pharmacogenomics and Stratied Medicine pp. 715-746 189. * Oermanns S. Heating Up the Cutaneous Flushing Response. (2014) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 34 6 1122-1123 190. Sakurai T et al. Identication of a novel GPR81-selective agonist that suppresses lipolysis in mice without cutaneous ushing. (2014) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 727 1-7 191. Feingold KR et al. Inammation stimulates niacin receptor (GPR109A/HCA2) expression in adipose tissue and macrophages. (2014) JOURNAL OF LIPID RESEARCH 0022-2275 1539-7262 55 12 2501-2508 192. Zhou E et al. Niacin attenuates the production of pro- inammatory cytokines in LPS-induced mouse alveolar macrophages by HCA2 dependent mechanisms. (2014) INTERNATIONAL IMMUNOPHARMACOLOGY 1567-5769 1878- 1705 23 1 121-126 193. Couturier A et al. Niacin in Pharmacological Doses Alters MicroRNA Expression in Skeletal Muscle of Obese Zucker Rats. (2014) PLOS ONE 1932-6203 1932-6203 9 5 194. Ma Linlin et al. Nicotinic Acid Activates the Capsaicin Receptor TRPV1 Potential Mechanism for Cutaneous Flushing. (2014) ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY 1079-5642 1524-4636 34 6 1272-1280 195. Li JJ et al. Nicotinic acid modulates intracellular calcium concentration and disassembles the cytoskeleton. (2014) MOLECULAR MEDICINE REPORTS 1791-2997 1791-3004 10 6 2805-2810 196. Elangovan S et al. The Niacin/Butyrate Receptor GPR109A Suppresses Mammary Tumorigenesis by Inhibiting Cell Survival. (2014) CANCER RESEARCH 0008-5472 1538-7445 74 4 1166-1178 197. McNamara Martin et al. A randomized pilot study on the eect of niacin on pulmonary arterial pressure. (2015) TRIALS 1745-6215 16 198. Sprecher D et al. Discovery and characterization of GSK256073, a non-ushing hydroxy-carboxylic acid receptor 2 (HCA2) agonist. (2015) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 756 1-7 199. Kim HY et al. Discovery of 4-(phenyl)thio-1H-pyrazole derivatives as agonists of GPR109A, a high anity niacin receptor. (2015) ARCHIVES OF PHARMACAL RESEARCH 0253-6269 38 6 1019-1032 200. Tienken R et al. Eects of prepartum dietary energy level and nicotinic acid supplementation on immunological, hematological and biochemical parameters of periparturient dairy cows diering in parity. (2015) ANIMALS 2076-2615 5 3 910-933 201. van de Weijer Tineke et al. Evidence for a Direct Eect of the NAD(+) Precursor Acipimox on Muscle Mitochondrial Function in Humans. (2015) DIABETES 0012-1797 1939-327X 64 4 1193-1201 202. Parodi B et al. Fumarates modulate microglia activation through a novel HCAR2 signaling pathway and rescue synaptic dysregulation in inamed CNS. (2015) ACTA NEUROPATHOLOGICA 0001-6322 1432-0533 130 2 279-295 203. Freeman M.W. et al. Lipoprotein Metabolism and the Treatment of Lipid Disorders. (2015) Megjelent: Endocrinology: Adult and Pediatric pp. 715-736.e7 204. Sharma D et al. Multifaceted role of beta-arrestins in inammation and disease. (2015) GENES AND IMMUNITY 1466- 4879 1476-5470 16 8 499-513 205. Canto Carles et al. NAD(+) Metabolism and the Control of Energy Homeostasis: A Balancing Act between Mitochondria and the Nucleus. (2015) CELL METABOLISM 1550-4131 22 1 31-53 206. Sinthupoom N et al. Nicotinic acid and derivatives as multifunctional pharmacophores for medical applications. (2015) EUROPEAN FOOD RESEARCH AND TECHNOLOGY 1438-2377 1438-2385 240 1 1-17 207. Liu DW et al. Nicotinic Acid Regulates Glucose and Lipid Metabolism Through Lipid-independent Pathways. (2015) CURRENT PHARMACEUTICAL BIOTECHNOLOGY 1389-2010 1873- 4316 16 1 3-10 208. Krishna R et al. Non-obligatory role of prostaglandin D2 receptor subtype 1 in rosacea: Laropiprant in Comparison to a placebo did not alleviate the symptoms of erythematoelangiectaic rosacea. (2015) JOURNAL OF CLINICAL PHARMACOLOGY 0091-2700 1552- 4604 55 2 137-143 209. * Oermanns Stefan et al. Nutritional or pharmacological activation of HCA(2) ameliorates neuroinammation. (2015) TRENDS IN MOLECULAR MEDICINE 1471-4914 1471-499X 21 4 245-255 210. Yang R et al. Activation of the niacin receptor HCA2 reduces demyelination and neurolament loss, and promotes functional recovery after spinal cord injury in mice. (2016) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 791 124-136 211. Morigny Pauline et al. Adipocyte lipolysis and insulin resistance. (2016) BIOCHIMIE 0300-9084 125 259-266 212. Fukano Y et al. A label-free impedance-based whole cell assay revealed a new G protein-coupled receptor ligand for mouse microglial cell migration. (2016) BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS 0006-291X 1090-2104 478 2 624- 630 213. Rezq Samar et al. Central GPR109A Activation Mediates Glutamate-Dependent Pressor Response in Conscious Rats. (2016) JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS 0022-3565 1521-0103 356 2 456-465 214. Preyat Nicolas et al. Complex role of nicotinamide adenine dinucleotide in the regulation of programmed cell death pathways. (2016) BIOCHEMICAL PHARMACOLOGY 0006-2952 1873-2968 101 13-26 215. Pineda A et al. Eects of rumen-protected niacin on milk production and body temperature of middle and late lactation Holstein cows. (2016) LIVESTOCK SCIENCE 1871-1413 1878-0490 187 16-23 216. More Sandeep et al. Emerging preclinical pharmacological targets for Parkinson's disease. (2016) ONCOTARGET 1949-2553 1949- 2553 7 20 29835-29863 217. Kuei C et al. Molecular and pharmacological characterization of hamster HCA1 and HCA2. (2016) IMMUNOLOGY ENDOCRINE AND METABOLIC AGENTS IN MEDICINAL CHEMISTRY 1871-5222 1875- 6115 16 1 61-69 218. Park JH et al. Nicotinamide mononucleotide inhibits post-ischemic NAD(+) degradation and dramatically ameliorates brain damage following global cerebral ischemia. (2016) NEUROBIOLOGY OF DISEASE 0969-9961 1095-953X 95 102-110 219. Messamore Erik et al. Phospholipid, arachidonate and eicosanoid signaling in schizophrenia. (2016) OCL-OLEAGINEUX CORPS GRAS LIPIDES 1258-8210 23 1 220. Aragona K et al. Prepartum supplementation of nicotinic acid: Eects on health of the dam, colostrum quality, and acquisition of immunity in the calf. (2016) JOURNAL OF DAIRY SCIENCE 0022- 0302 1525-3198 99 5 3529-3538 221. Mangoni Arduino. Repurposing Niacin as Antiplatelet Drug?. (2016) CURRENT CLINICAL PHARMACOLOGY 1574-8847 11 1 2-3 222. Conze DB et al. Safety assessment of nicotinamide riboside, a form of Vitamin B3. (2016) HUMAN & EXPERIMENTAL TOXICOLOGY 0960-3271 1477-0903 35 11 1149-1160 223. Bleeker Jeannette et al. Sirtuin activation as a therapeutic approach against inborn errors of metabolism. (2016) JOURNAL OF INHERITED METABOLIC DISEASE 0141-8955 1573-2665 39 Lyon 565-572 224. Montserrat-de la Paz et al. Exogenous fatty acids and niacin on acute prostaglandin D2 production in human myeloid cells. (2017) JOURNAL OF NUTRITIONAL BIOCHEMISTRY 0955-2863 39 22-31 225. Husted Anna et al. GPCR-Mediated Signaling of Metabolites. (2017) CELL METABOLISM 1550-4131 25 4 777-796 226. * Oermanns Stefan. Hydroxy-Carboxylic Acid Receptor Actions in Metabolism. (2017) TRENDS IN ENDOCRINOLOGY AND METABOLISM 1043-2760 1879-3061 28 3 227-236 227. Dunbar RL et al. Measuring niacin-associated skin toxicity (NASTy) stigmata along with symptoms to aid development of niacin mimetics. (2017) JOURNAL OF LIPID RESEARCH 0022-2275 1539- 7262 58 4 783-797 228. Pugazhenthi S. Metabolic Syndrome and the Cellular Phase of Alzheimer's Disease. (2017) PROGRESS IN MOLECULAR BIOLOGY AND TRANSLATIONAL SCIENCE 1877-1173 1878-0814 146 243- 258 229. Puchalska Patrycja et al. Multi-dimensional Roles of Ketone Bodies in Fuel Metabolism, Signaling, and Therapeutics. (2017) CELL METABOLISM 1550-4131 25 2 262-284 230. Kong D et al. Niacin promotes cardiac healing after myocardial infarction through activation of the myeloid prostaglandin D2receptor subtype 1. (2017) JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS 0022-3565 1521-0103 360 3 435- 444 231. El-Zaatari Mohamad et al. Role of Dietary Metabolites in Regulating the Host Immune Response in Gastrointestinal Disease. (2017) FRONTIERS IN IMMUNOLOGY 1664-3224 1664- 3224 8 232. Elhassan Yasir et al. Targeting NAD+ in Metabolic Disease: New Insights Into an Old Molecule. (2017) JOURNAL OF THE ENDOCRINE SOCIETY 2472-1972 1 7 816-835 233. Diaz Gwendoline et al. Dimethyl fumarate treatment in multiple sclerosis: Recent advances in clinical and immunological studies. (2018) AUTOIMMUNITY REVIEWS 1568-9972 17 12 1240-1250 234. Talele Tanaji. Natural-Products-Inspired Use of the gem-Dimethyl Group in Medicinal Chemistry. (2018) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 61 6 2166-2210 235. Almquist Joachim et al. Overexpressing cell systems are a competitive option to primary adipocytes when predicting in vivo potency of dual GPR81/GPR109A agonists. (2018) EUROPEAN JOURNAL OF PHARMACEUTICAL SCIENCES 0928-0987 1879-0720 114 155-165 236. Masuda Yusuke et al. Overexpressing human GPR109A leads to pronounced reduction in plasma triglyceride levels in BAC transgenic rats. (2018) ATHEROSCLEROSIS 0021-9150 272 182- 192 237. Priyadarshini Medha et al. Role of Short Chain Fatty Acid Receptors in Intestinal Physiology and Pathophysiology. (2018) COMPREHENSIVE PHYSIOLOGY 2040-4603 8 3 1091-1115 238. Abdullah KM et al. Therapeutic eect of vitamin B3 on hyperglycemia, oxidative stress and DNA damage in alloxan induced diabetic rat model. (2018) BIOMEDICINE & PHARMACOTHERAPY 0753-3322 1950-6007 105 1223-1231 239. Recio Carlota et al. The Role of Metabolite-Sensing G Protein- Coupled Receptors in Inammation and Metabolic Disease. (2018) ANTIOXIDANTS & REDOX SIGNALING 1523-0864 1557-7716 29 3 237-256 240. Olson E.J. et al. A Randomized, Placebo-Controlled Trial to Assess the Eects of 8 Weeks of Administration of GSK256073, a Selective GPR109A Agonist, on High-Density Lipoprotein Cholesterol in Subjects With Dyslipidemia. (2019) CLINICAL PHARMACOLOGY IN DRUG DEVELOPMENT 2160-763X 2160-7648 2019 p. In press 241. Giroud-Gerbetant J. et al. A reduced form of nicotinamide riboside denes a new path for NAD+ biosynthesis and acts as an orally bioavailable NAD+ precursor. (2019) MOLECULAR METABOLISM 2212-8778 30 192-202 242. Gafson Arie et al. Breaking the cycle Reversal of ux in the tricarboxylic acid cycle by dimethyl fumarate. (2019) NEUROLOGY-NEUROIMMUNOLOGY & NEUROINFLAMMATION 2332-7812 6 3 243. Song Seon et al. Diverse therapeutic ecacies and more diverse mechanisms of nicotinamide. (2019) METABOLOMICS 1573-3882 1573-3890 15 10 244. Aday Aaron et al. Impact of Acipimox Therapy on Free Fatty Acid Eux and Endothelial Function in the Metabolic Syndrome: A Randomized Trial. (2019) OBESITY 1930-7381 1930-739X 2019 p. In press 245. Takakura Ayumi et al. Lactate-induced activation of HCA2 improves survival in mice with sepsis. (2019) FASEB JOURNAL 0892-6638 1530-6860 33 6 7625-7634 246. * Markovics Arnold et al. Nicotinic acid suppresses sebaceous lipogenesis of human sebocytes via activating hydroxycarboxylic acid receptor 2 (HCA(2)). (2019) JOURNAL OF CELLULAR AND MOLECULAR MEDICINE 1582-1838 1582-4934 9 23 6203-6214 247. Csiszar Anna et al. Role of endothelial NAD+ deciency in age- related vascular dysfunction.. (2019) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 316 6 H1253-H1266 248. Chandrasekaran K. et al. Role of mitochondria in diabetic peripheral neuropathy: Inuencing the NAD+-dependent SIRT1– PGC-1α–TFAM pathway. (2019) Megjelent: International Review of Neurobiology pp. 177-209 249. Trotta Maria et al. The activation of retinal HCA2 receptors by systemic beta-hydroxybutyrate inhibits diabetic retinal damage through reduction of endoplasmic reticulum stress and the NLRP3 inammasome. (2019) PLOS ONE 1932-6203 1932-6203 14 1 250. Bridges Allison et al. Vitamin B3: niacin and transcriptome analysis in relation to the GPR109A receptor. (2020) Megjelent: MOLECULAR NUTRITION: VITAMINS pp. 673-690

138. Benyó, Z ; Kero, J ; Csiky, M ; Gille, A ; Moers, A ; Pfeer, K ; Oermanns, S Mechanism of nicotinic acid-induced ushing in mice NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 371 : Suppl. 1 pp. R9-R9. Paper: 36 (2005) WoS Közlemény:1856260 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

139. Hilpert, A ; Benyó, Z ; Schilling, L Isometric force measurement in mouse cerebral arteries: establishing reference values and characterizing functional consequences of endothelial nitric oxide synthase knock-out in the basilar artery. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 25 : Suppl. 1 p. S546 (2005) DOI Közlemény:1856277 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

140. Hortobágyi, L ; Kis, B ; Hrabák, A ; Horváth, B ; Sándor, P ; Busija, DW ; Benyó, Z Adaptation of the hypothalamic blood ow to chronic nitric oxide synthase blockade JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 25 : Suppl. 1 p. S206 (2005) DOI Közlemény:1856275 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

141. Horvath, B ; Orsy, P ; Benyo, Z Endothelial NOS-mediated relaxations of isolated thoracic aorta of the C57BL/6J mouse a methodological study JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 45 : 3 pp. 225-231. , 7 p. (2005) DOI WoS Scopus PubMed Közlemény:1628759 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 15 Független: 8 Függő: 7 Idézett közlemények száma: 3

1. Boittin FX. Ca2+ -independent PLA(2) controls endothelial store- operated Ca2+ entry and vascular tone in intact aorta. (2008) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 295 6 H2466-H2474 2. Xu ML. Characterization of agonist-induced vasoconstriction in mouse pulmonary artery. (2008) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 294 1 H220-H228 3. * Magyar A et al. Vascular eects of sphingolipid mediators. (2009) ACTA PHYSIOLOGICA HUNGARICA 0231-424X 1588-2683 2498- 602X 96 1 98-100 4. Li Y-Z. Carbachol inhibits TNF-α-induced endothelial barrier dysfunction through alpha 7 nicotinic receptors. (2010) ACTA PHARMACOLOGICA SINICA 1671-4083 1745-7254 31 10 1389- 1394 5. * Buday A et al. Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) mice. (2010) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 299 2 H386- H395 6. * Masszi G et al. Altered insulin-induced relaxation of aortic rings in a dihydrotestosterone-induced rodent model of polycystic ovary syndrome. (2013) FERTILITY AND STERILITY 0015-0282 99 2 573- 578 7. * Masszi Gabriella et al. Eects of vitamin D3 derivate calcitriol on pharmacological reactivity of aortic rings in a rodent PCOS model. (2013) PHARMACOLOGICAL REPORTS 1734-1140 65 2 476-483 8. Prendergast C. Atherosclerosis dierentially aects calcium signaling in endothelial cells from aortic arch and thoracic aorta in Apolipoprotein E knockout mice. (2014) PHYSIOLOGICAL REPORTS 2051-817X 2 10 9. Szijártó IA. Increase in insulin-induced relaxation of consecutive arterial segments toward the periphery: Role of vascular oxidative state.. (2014) FREE RADICAL RESEARCH 1071-5762 1029-2470 48 7 749-757 10. * Ruisanchez E et al. Lysophosphatidic acid induces vasodilation mediated by LPA1 receptors, phospholipase C, and endothelial nitric oxide synthase.. (2014) FASEB JOURNAL 0892-6638 1530- 6860 28 2 880-890 11. * Szekeres M et al. Endocannabinoid-mediated modulation of G protein-coupled receptor signaling-induced vasoconstriction and hypertension.. (2015) MOLECULAR AND CELLULAR ENDOCRINOLOGY 0303-7207 1872-8057 403 46-56 12. Krishnan Manickam. Enhancing eNOS activity with simultaneous inhibition of IKK beta restores vascular function in Ins2(Akita+/-) type-1 diabetic mice. (2015) LABORATORY INVESTIGATION 0023- 6837 1530-0307 95 10 1092-1104 13. Del Campo L. Wire myography to study vascular tone and vascular structure of isolated mouse arteries. (2015) Megjelent: Methods in Mouse Atherosclerosis pp. 255-276 14. * Dancs PT et al. LPA1 receptor-mediated thromboxane A2 release is responsible for lysophosphatidic acid-induced vascular smooth muscle contraction. (2017) FASEB JOURNAL 0892-6638 1530-6860 31 4 1547-1555 15. Tanaka Hiroki. Elevated Plasma Levels of LDL Cholesterol Promote Dissecting Thoracic Aortic Aneurysms in Angiotensin II- Induced Mice. (2018) ANNALS OF VASCULAR SURGERY 0890-5096 48 204-213

142. Horváth, B ; Hortobágyi, L ; Sándor, P ; Benyó, Z Interactions between the heme oxygenase, cyclooxygenase and nitric oxide synthase pathways in the regulation of the resting hypothalamic blood ow. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 25 : Suppl. 1 p. S187 (2005) DOI Közlemény:1856272 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

143. Horváth, BA ; Őrsy, P ; Benyó, Z Endothelial-dependent relaxations of the mouse thoracic aorta FASEB JOURNAL 19 : 5 p. A1553 (2005) WoS Közlemény:1856268 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

144. Ishiguro, M ; Lacza, Z ; Horváth, EM ; Járai, Z ; Sándor, P ; Benyó, Z Inhibition of the cannabinoid-1 receptor enhances the cerebrocortical hyperemic response to hypoxia/hypercapnia. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 25 : Suppl. 1 p. S189 (2005) DOI Közlemény:1856274 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat ) 145. Wirth, A ; Benyó, Z ; Leutgeb, B ; Wettschureck, N ; Oermanns, S The role of Gq/G11-family of G-proteins in vascular tone and blood pressure regulation. NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 371 : suppl 1 pp. R85-R86. Paper: 361 (2005) WoS Közlemény:1856265 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

146. Hilpert, A ; Benyó, Z ; Schilling, L Studies on contraction and relaxation behaviour in ring segments of mouse basilar artery JOURNAL OF VASCULAR RESEARCH 41 : 5 pp. 468-468. , 1 p. (2004) WoS Közlemény:1856259 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

147. Horvath, B ; Ishiguro, M ; Lenzser, G ; Herman, P ; Hrabak, A ; Kaldi, K ; Sandor, P ; Benyo, Z Interaction between the heme oxygenase and nitric oxide pathways in the regulation of the resting CBF. FASEB JOURNAL 18 : 4 pp. A268-A268. (2004) WoS Közlemény:1162429 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

148. Horváth, B ; Ishiguro, M ; Hortobágyi, L ; Lenzsér, G ; Hermán, P ; Hrabák, A ; Káldi, K ; Sándor, P ; Benyó, Z Role of the heme oxygenase pathway in the regulation of the resting CBF. ACTA PHYSIOLOGICA HUNGARICA 91 pp. 302-303. , 2 p. (2004) Közlemény:1856254 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

149. Ishiguro, M ; Lacza, Z ; Horváth, EM ; Járai, Z ; Sándor, P ; Benyó, Z Role of cannabinoid 1 receptor in the modulation of the cerebrocortical hyperemic response to hypoxia/hypercapnia. ACTA PHYSIOLOGICA HUNGARICA 91 pp. 311-312. , 2 p. (2004) Közlemény:1856256 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

150. Ishiguro, M ; Lacza, Z ; Horvath, EM ; Jarai, Z ; Sandor, P ; Benyo, Z The cannabinoid receptor antagonist AM-251 enhances the cerebrocortical hyperemic response to hypoxia/hypercapnia FASEB JOURNAL 18 : 4 pp. A267-A267. (2004) WoS Közlemény:1630982 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

151. Lenzsér, G ; Hermán, P ; Komjáti, K ; Sándor, P ; Benyó, Z Nitric oxide synthase blockade sensitizes the cerebrocortical circulation to thromboxane-induced CBF oscillations. FASEB JOURNAL 18 : 5 p. A1009 (2004) WoS Közlemény:1856252 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

152. Őrsy, P ; Horváth, B ; Oermanns, S ; Benyó, Z Endothelium-dependent relaxation of the mouse thoracic aorta. ACTA PHYSIOLOGICA HUNGARICA 91 pp. 343-344. , 2 p. (2004) Közlemény:1856258 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

153. Zador, Z ; Benyo, Z ; Lacza, Z ; Hortobagyi, T Sr ; Harkany, T ; Hortobagyi, T Neuroprotekció agyi ischaemiában: kételyek és remények IDEGGYOGYASZATI SZEMLE / CLINICAL NEUROSCIENCE 57 : 3-4 pp. 81-93. , 13 p. (2004) Scopus PubMed Teljes dokumentum Közlemény:1632812 Érvényesített Forrás Folyóiratcikk (Összefoglaló cikk ) Nyilvános idézők összesen: 8 Független: 8 Függő: 0

1. Boedker M et al. Budesonide epimer R, LAU-8080 and phenyl butyl nitrone synergistically repress cyclooxygenase-2 induction in [IL-1 beta + A beta 42]-stressed human neural cells. (2005) NEUROSCIENCE LETTERS 0304-3940 1872-7972 380 1-2 176-180 2. CAI Zhiyou et al. Minocycline inhibits COX-2 and NF-kappaB expressions in rats of focal cerebral ischemia-reperfusion. (2007) JOURNAL OF THIRD MILITARY MEDICAL UNIVERSITY 1000-5404 29 21 2067-2069 3. Boetkjaer A et al. Synergism in the repression of COX-2- and TNF alpha-induction in platelet activating factor-stressed human neural cells. (2007) NEUROSCIENCE LETTERS 0304-3940 1872- 7972 426 1 59-63 4. FAN Xiao-nong et al. Study on optimum stimulation parameter for Shuigou (GV 26) in Xingnao Kaiqiao needling method. (2008) Chinese Acupuncture and Moxibustion 0255-2930 28 12 913-917 5. Joshi S et al. The proposed role of optical sensing in translational stroke research. (2010) ANNALS OF THE NEW YORK ACADEMY OF SCIENCES 0077-8923 1749-6632 1199 149-157 6. Duan Shiwan et al. Eect of musk,borneol,diosgenin and geniposide on inammatory injury in cerebral ischemia and reperfusion rats. (2012) ZHONGYAO YAOLI YU LINCHUANG / PHARMACOLOGY OF CLINICS OF CHINESE MATERIA MEDICA 1001-859X 28 3 43-46 7. Song X-Y et al. Neurons apoptosis and cerebral ischemia. (2012) CHINESE PHARMACOLOGICAL BULLETIN 1001-1978 28 3 307-310 8. Tian C-Y. Eect of Gridi-13 on expression of NSE, TNF-α, and IL-6 after focal cerebral ischemia in rats. (2015) CHINESE TRADITIONAL AND HERBAL DRUGS 0253-2670 46 5 716-720

154. Hortobagyi, T ; Gorlach, C ; Benyo, Z ; Lacza, Z ; Hortobagyi, S ; Wahl, M ; Harkany, T Inhibition of neuronal nitric oxide synthase-mediated activation of poly(ADP-ribose) polymerase in traumatic brain injury: neuroprotection by 3-aminobenzamide. NEUROSCIENCE 121 : 4 pp. 983-990. , 8 p. (2003) DOI WoS Scopus PubMed Közlemény:1518200 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 24 Független: 22 Függő: 2 Idézett közlemények száma: 7

1. McArthur DL et al. Moderate and severe traumatic brain injury: Epidemiologic, imaging and neuropathologic perspectives. (2004) BRAIN PATHOLOGY 1015-6305 1750-3639 14 2 185-194 2. Tse Y-H. Cerebral protection in traumatic brain injury. (2005) SURGICAL PRACTICE 1744-1625 9 4 122-125 3. Chiarugi A. Intrinsic mechanisms of poly(ADP-ribose) neurotoxicity: Three hypotheses. (2005) NEUROTOXICOLOGY 0161-813X 26 5 847-855 4. Komjáti K et al. Poly (ADP-Ribose) polymerase inhibitors as potential therapeutic agents in stroke and neurotrauma. (2005) CURRENT DRUG TARGETS: CNS AND NEUROLOGICAL DISORDERS 1568-007X 2212-5299 1871-5273 4 2 179-194 5. Lau A et al. Inhibition of caspase-mediated apoptosis by peroxynitrite in traumatic brain injury. (2006) JOURNAL OF NEUROSCIENCE 0270-6474 1529-2401 26 45 11540-11553 6. Klawitter V et al. Plasticity of the central nervous system (CNS) following perinatal asphyxia: Does nicotinamide provide neuroprotection?. (2006) AMINO ACIDS 0939-4451 1438-2199 31 4 377-384 7. Paul R et al. Dierential regulation of blood-brain barrier permeability in brain trauma and pneumococcal meningitis - role of Src kinases. (2007) EXPERIMENTAL NEUROLOGY 0014-4886 1090-2430 203 1 158-167 8. Klawitter V et al. Plasticity of basal ganglia neurocircuitries following perinatal asphyxia: eect of nicotinamide. (2007) EXPERIMENTAL BRAIN RESEARCH 0014-4819 1432-1106 180 1 139-152 9. Barr TL et al. Poly(ADP-ribose) polymerase-1 and its clinical applications in brain injury. (2007) JOURNAL OF NEUROSCIENCE NURSING 0888-0395 39 5 278-284 10. Kovesdi E et al. Rescuing neurons and glia: is inhibition of apoptosis useful?. (2007) PROGRESS IN BRAIN RESEARCH 0079- 6123 1875-7855 161 81-95 11. Cipriani S et al. Adenosine A(2A) receptor antagonism increases nNOS-immunoreactive neurons in the striatum of Huntington transgenic mice. (2008) EXPERIMENTAL NEUROLOGY 0014-4886 1090-2430 213 1 163-170 12. Ansari MA. A time course of contusion-induced oxidative stress and synaptic proteins in cortex in a rat model of TBI. (2008) JOURNAL OF NEUROTRAUMA 0897-7151 25 5 513-526 13. Hu J-J et al. Correlation between nNOS expression and erectile dysfunction in experimental diabetes mellitus rats of dierent ages. (2008) Neuroembryology and Aging 1661-3406 1661-3414 5 4 144-150 14. Besson Valerie. Drug targets for traumatic brain injury from poly(ADP-ribose)polymerase pathway modulation. (2009) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 157 5 695- 704 15. Morales P et al. Pathophysiology of perinatal asphyxia: Can we predict and improve individual outcomes?. (2011) EPMA JOURNAL 1878-5077 1878-5085 2 2 211-230 16. Herrera-Marschitz Mario et al. Perinatal asphyxia: current status and approaches towards neuroprotective strategies, with focus on sentinel proteins. (2011) NEUROTOXICITY RESEARCH 1029- 8428 1476-3524 19 4 603-627 17. Cabezas Ricardo. Mitochondrial functions in astrocytes: Neuroprotective implications from oxidative damage by rotenone. (2012) NEUROSCIENCE RESEARCH 0168-0102 74 2 80- 90 18. Ansari MA et al. A time course of NADPH-oxidase up-regulation and endothelial nitric oxide synthase activation in the hippocampus following neurotrauma. (2014) FREE RADICAL BIOLOGY AND MEDICINE 0891-5849 1873-4596 77 21-29 19. Neira-Pena T et al. Molecular, cellular, and behavioural eects produced by perinatal asphyxia: Protection by poly (ADP-Ribose) polymerase 1 (PARP-1) inhibition. (2014) Megjelent: Handbook of Neurotoxicity pp. 2075-2098 20. * Csonka T et al. Poly(ADP-ribose) polymerase-1 (PARP1) and p53 labelling index correlates with tumour grade in meningiomas. (2014) FOLIA NEUROPATHOLOGICA 1641-4640 1509-572X 52 2 111-120 21. Abdul-Muneer P et al. Interactions of Oxidative Stress and Neurovascular Inammation in the Pathogenesis of Traumatic Brain Injury. (2015) MOLECULAR NEUROBIOLOGY 0893-7648 1559-1182 51 3 966-979 22. Boone DR et al. Eects of AAV-mediated knockdown of nNOS and GPx-1 gene expression in rat hippocampus after traumatic brain injury. (2017) PLOS ONE 1932-6203 1932-6203 12 10 23. * Murnyák B et al. PARP1 expression and its correlation with survival is tumour molecular subtype dependent in glioblastoma. (2017) ONCOTARGET 1949-2553 1949-2553 8 28 46348-46362 24. Surikow SY et al. Nitrosative Stress as a Modulator of Inammatory Change in a Model of Takotsubo Syndrome. (2018) JACC: Basic to Translational Science 2452-302X 3 2 213-226 155. Horvath, B ; Hrabak, A ; Kaldi, K ; Sandor, P ; Benyo, Z Contribution of the heme oxygenase pathway to the maintenance of the hypothalamic blood ow during diminished nitric oxide synthesis JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 23 : 6 pp. 653-657. , 5 p. (2003) DOI WoS Scopus PubMed Közlemény:1151550 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 15 Független: 10 Függő: 5 Idézett közlemények száma: 3

1. Oermann E et al. Eects of 1 alpha,25 dihydroxyvitamin D-3 on the expression of HO-1 and GFAP in glial cells of the photothrombotically lesioned cerebral cortex. (2004) JOURNAL OF CHEMICAL NEUROANATOMY 0891-0618 1873-6300 28 4 225-238 2. Chan CY et al. Lack of detrimental eects of nitric oxide inhibition in bile duct-ligated rats with hepatic encephalopathy. (2004) EUROPEAN JOURNAL OF CLINICAL INVESTIGATION 0014-2972 1365-2362 34 2 122-128 3. Leer CW et al. Carbon monoxide and hydrogen sulde: gaseous messengers in cerebrovascular circulation. (2006) JOURNAL OF APPLIED PHYSIOLOGY 8750-7587 1522-1601 100 3 1065-1076 4. De Gottardi A et al. Sensitivity to endothelin-1 is decreased in isolated livers of endothelial constitutive nitric oxide synthase knockout mice. (2006) Comparative Hepatology 1476-5926 5 5. Gao M et al. 1-aminocyclopropanecarboxylic acid, an antagonist of N-methyl-D-aspartate receptors, causes hypotensive and antioxidant eects with Upregulation of heme oxygenase-1 in stroke-prone spontaneously hypertensive rats. (2007) HYPERTENSION RESEARCH 0916-9636 1348-4214 30 3 249-257 6. * Hortobagyi L et al. Adaptation of the hypothalamic blood ow to chronic nitric oxide deciency is independent of vasodilator prostanoids. (2007) BRAIN RESEARCH 0006-8993 1872-6240 1131 1 129-137 7. Holt DC et al. Age and species dependence of pial arteriolar responses to topical carbon monoxide in vivo. (2007) EXPERIMENTAL BIOLOGY AND MEDICINE 1535-3702 1535-3699 232 11 1465-1469 8. * Leszl-Ishiguro M et al. Inuence of the heme-oxygenase pathway on cerebrocortical blood ow. (2007) NEUROREPORT 0959-4965 18 11 1193-1197 9. Li AL et al. Astrocyte-derived CO is a diusible messenger that mediates glutamate-induced cerebral arteriolar dilation by activating smooth muscle cell K-Ca channels. (2008) CIRCULATION RESEARCH 0009-7330 1524-4571 102 2 234-241 10. * Horvath B et al. Carbon monoxide-prostaglandin E-2 interaction in the hypothalamic circulation. (2008) NEUROREPORT 0959-4965 19 16 1601-1604 11. * Lacza Z et al. Additive eect of cyclooxygenase and nitric oxide synthase blockade on the cerebrocortical microcirculation. (2009) NEUROREPORT 0959-4965 20 11 1027-1031 12. Toda N et al. Cerebral Blood Flow Regulation by Nitric Oxide: Recent Advances. (2009) PHARMACOLOGICAL REVIEWS 0031- 6997 1521-0081 61 1 62-97 13. Leer Charles et al. Carbon monoxide as an endogenous vascular modulator. (2011) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 301 1 H1-H11 14. * Iring A et al. Role of endocannabinoids and cannabinoid-1 receptors in cerebrocortical blood ow regulation. (2013) PLOS ONE 1932-6203 1932-6203 8 1 15. Park Sarah et al. The real-time in vivo electrochemical measurement of nitric oxide and carbon monoxide release upon direct epidural electrical stimulation of the rat neocortex. (2015) ANALYST 0003-2654 1364-5528 140 10 3415-3421

156. Horváth, B ; Ishiguro, M ; Hrabák, A ; Káldi, K ; Lacza, Z ; Sándor, P ; Benyó, Z Interaction between the heme oxygenase and nitric oxide pathways in the regulation of the resting CBF. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 23 p. S77 (2003) Közlemény:1856245 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

157. Lacza, Z ; Dézsi, L ; Káldi, K ; Horváth, E M ; Sándor, P ; Benyó, Z Prostacyclin-mediated compensatory mechanism in the coronary circulation during acute NO synthase blockade LIFE SCIENCES 73 : 9 pp. 1141-1149. , 9 p. (2003) DOI WoS Scopus PubMed Közlemény:1475884 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 10 Független: 8 Függő: 2 Idézett közlemények száma: 4

1. Zima E et al. Nitrogen oxide blockade does not aggravate the endothelin-1-induced myocardial ischemia and release of purine metabolites from the dog heart.. (2004) JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 0160-2446 1533-4023 44 S1 S313-S317 2. * Hortobagyi L et al. Adaptation of the hypothalamic blood ow to chronic nitric oxide deciency is independent of vasodilator prostanoids. (2007) BRAIN RESEARCH 0006-8993 1872-6240 1131 1 129-137 3. Hirao A et al. Cyclooxygenase-dependent vasoconstricting factor(s) in remodelled rat femoral arteries. (2008) CARDIOVASCULAR RESEARCH 0008-6363 1755-3245 79 1 161-168 4. * Lacza Z et al. Additive eect of cyclooxygenase and nitric oxide synthase blockade on the cerebrocortical microcirculation. (2009) NEUROREPORT 0959-4965 20 11 1027-1031 5. Yoshino J et al. Diabetes-associated Alterations in Volatile Anesthetic Actions on Contractile Response to Norepinephrine in Isolated Mesenteric Resistance Arteries. (2010) ANESTHESIOLOGY 0003-3022 1528-1175 112 3 595-606 6. Lee A-W et al. Ursolic Acid attenuates HMGB1-induced LOX-1 expression in vascular endothelial cells in vitro and inhibits atherogenesis in hypercholesterolemic mice in vivo. (2012) IMMUNOLOGY ENDOCRINE AND METABOLIC AGENTS IN MEDICINAL CHEMISTRY 1871-5222 1875-6115 12 4 317-329 7. Vuletic Vladimira et al. Eect of indomethacin on cerebrovascular reactivity in patients with type 2 diabetes mellitus. (2013) DIABETES RESEARCH AND CLINICAL PRACTICE 0168-8227 1872- 8227 101 1 81-87 8. Chai Qiang et al. Hydrogen sulde impairs shear stress-induced vasodilation in mouse coronary arteries. (2014) PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY 0031-6768 1432- 2013 466 2 392-340 9. Kij A et al. Simultaneous quantication of PGI2 and TXA2 metabolites in plasma and urine in NO-decient mice by a novel UHPLC/MS/MS method. (2016) JOURNAL OF PHARMACEUTICAL AND BIOMEDICAL ANALYSIS 0731-7085 1873-264X 129 148-154 10. Pérez-Cremades D et al. Extracellular histones disarrange vasoactive mediators release through a COX-NOS interaction in human endothelial cells. (2017) JOURNAL OF CELLULAR AND MOLECULAR MEDICINE 1582-1838 1582-4934 21 8 1584-1592 158. Lenzsér, G ; Hermán, P ; Komjáti, K ; Sándor, P ; Benyó, Z Nitric oxide synthase blockade sensitizes the cerebrocortical circulation to thromboxane-induced CBF oscillations. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 23 p. S88 (2003) Közlemény:1856249 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat ) Nyilvános idézők összesen: 5 Független: 3 Függő: 2

1. * Benyó B. Characterization of the temporal pattern of cerebral blood ow oscillations. (2004) 2. * Balázs Benyó. CHARACTERIZATION OF CEREBRAL BLOOD FLOW OSCILLATIONS USING DIFFERENT CLASSIFICATION METHODS. (2005) 3. Benyó B. Classication of time series using singular values and wavelet subband analysis with ANN and SVM classiers. (2005) 4. Peter Somogyi. Articial neural network based classication of cerebral blood owsignals. (2006) PERIODICA POLYTECHNICA SER. EL. ENG. 50 63-68 5. B Paláncz. Classication of time series via wavelet subband analysis using support vector machine classier. (2006) PERIODICA POLYTECHNICA SER. EL. ENG. 50 129-140

159. Zador, Z ; Lacza, Z ; Benyo, Z ; Harkany, T ; Hortobagyi, T Apoptozis fokalis agyi ischaemiában IDEGGYOGYASZATI SZEMLE / CLINICAL NEUROSCIENCE 56 : 7-8 pp. 216-228. , 13 p. (2003) Scopus PubMed MOB Közlemény:1518201 Érvényesített Forrás Folyóiratcikk (Összefoglaló cikk ) Nyilvános idézők összesen: 4 Független: 3 Függő: 1

1. Luo Z-D et al. Inuence of electroacupuncture on apoptosis and nerve growth factor of focal cerebral ischemia of rats. (2004) CHINESE JOURNAL OF CLINICAL REHABILITATION 1671-5926 8 7 1382-1383 2. Xiao XH et al. Protective eect of protopine on the focal cerebral ischaemic injury in rats. (2007) BASIC & CLINICAL PHARMACOLOGY & TOXICOLOGY 1742-7835 1742-7843 101 2 85- 89 3. * Csonka T et al. A herpesencephalitis diagnosztikajarol egy esetismertetes kapcsan.. (2013) IDEGGYOGYASZATI SZEMLE / CLINICAL NEUROSCIENCE 0019-1442 2498-6208 66 9-10 337-342 4. Jinzhu Y et al. Aluminum and benzo[a]pyrene co-operate to induce neuronal apoptosis in vitro. (2015) JOURNAL OF TOXICOLOGICAL SCIENCES 0388-1350 1880-3989 40 3 365-373

160. Benyo, Z ; Lacza, Z ; Hortobagyi, T ; Gorlach, C ; Sandor, P ; Wahl, M Neuronal nitric oxide synthase in the cerebrovascular endothelium In: Tomita, M; Kanno, I; Hamel, E (szerk.) BRAIN ACTIVATION AND CBF CONTROL, PROCEEDINGS : International Symposium on Brain Activation and Cerebral Blood Flow Control, JUN 05-08, 2001. TOKYO, JAPAN Amsterdam, Hollandia * : Elsevier Science Publishers, (2002) pp. 369-377. , 9 p. DOI WoS Scopus Közlemény:1631128 Érvényesített Forrás Idéző Könyvrészlet (Konferenciaközlemény ) Idézett közlemények száma: 3

161. Benyó, Z ; Lacza, Z ; Hermán, P ; Görlach, C ; Hortobágyi, T ; Sándor, P ; Wahl, M Interplay between cyclic GMP- and thromboxane receptor-mediated mechanisms in the rat middle cerebral artery (MCA). ACTA PHYSIOLOGICA HUNGARICA 89 p. 56 (2002) Közlemény:1856239 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

162. Horváth, B ; Hrabák, A ; Káldi, K ; Johnson, FK ; Sándor, P ; Benyó, Z The role of endogenous carbon monoxide (CO) and its interaction with nitric oxide (NO) in the cerebral circulation. ACTA PHYSIOLOGICA HUNGARICA 89 p. 58 (2002) Közlemény:1856240 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat ) 163. Sandor, P ; Benyo, Z ; Erdos, B ; Lacza, Z ; Komjati, K The Roy-Sherrington hypothesis: facts and surmises In: Tomita, M; Kanno, I; Hamel, E (szerk.) BRAIN ACTIVATION AND CBF CONTROL, PROCEEDINGS : International Symposium on Brain Activation and Cerebral Blood Flow Control, JUN 05-08, 2001. TOKYO, JAPAN Amsterdam, Hollandia : Elsevier Science Publishers, (2002) pp. 325-335. , 11 p. DOI ISSN: 0531-5131 WoS Scopus Közlemény:1631127 Admin láttamozott Forrás Idéző Könyvrészlet (Konferenciaközlemény ) Nyilvános idézők összesen: 3 Független: 2 Függő: 1 Idézett közlemények száma: 5

1. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801 2. Javed Ehtasham et al. Removal of BCG artefact from concurrent fMRI-EEG recordings based on EMD and PCA. (2017) JOURNAL OF NEUROSCIENCE METHODS 0165-0270 291 150-165 3. Liu Xiuyun et al. Cross-Frequency Coupling Between Cerebral Blood Flow Velocity and EEG in Ischemic Stroke Patients With Large Vessel Occlusion. (2019) FRONTIERS IN NEUROLOGY 1664- 2295 10

164. Benyó, Z ; Lacza, Z ; Görlach, C ; Sándor, P ; Wahl, M Cerebrovascular eects of the inhibition of soluble guanylyl cyclase. EUROPEAN JOURNAL OF CLINICAL INVESTIGATION 31 : Suppl. 1 p. 6 Paper: 25 (2001) DOI Közlemény:1856233 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

165. Gorlach, C ; Hortobagyi, T ; Hortobagyi, S ; Benyo, Z ; Wahl, M Inhibition of endothelin-1 by the competitive ET(A) receptor antagonist Ro 61-1790 reduces lesion volume after cold injury in the rat. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY 441 : 6 pp. 844-849. , 6 p. (2001) DOI WoS Scopus PubMed Közlemény:1518207 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 12 Független: 12 Függő: 0 Idézett közlemények száma: 3

1. Calvani M. L'ipotermia [Hypothermia]. (2002) Pediatria Oggi Medica e Chirurgica 0391-898X 22 3 67-76 2. Petrov T et al. Sources of endothelin-1 in hippocampus and cortex following traumatic brain injury. (2002) NEUROSCIENCE 0306-4522 1873-7544 115 1 275-283 3. Vatter H et al. Experimental approaches to evaluate endothelin-A receptor antagonists. (2004) METHODS AND FINDINGS IN EXPERIMENTAL AND CLINICAL PHARMACOLOGY 0379-0355 26 4 277-286 4. Vatter H et al. Cerebrovascular characterization of clazosentan, the rst nonpeptide endothelin receptor antagonist clinically eective for the treatment of cerebral vasospasm. Part I: Inhibitory eect on endothelin(A) receptor-mediated contraction. (2005) JOURNAL OF NEUROSURGERY 0022-3085 102 6 1101-1107 5. Ostrow LW et al. Mechanosensation and endothelin in astrocytes - hypothetical roles in CNS pathophysiology. (2005) BRAIN RESEARCH REVIEWS 0165-0173 48 3 488-508 6. Uhlmann D. Clazosentan (Actelion). (2006) CURRENT OPINION IN INVESTIGATIONAL DRUGS 1472-4472 2040-3429 7 3 272-281 7. Macdonald RL. Clazosentan: A review of subarachnoid hemorrhage data. (2009) FUTURE NEUROLOGY 1479-6708 4 1 13- 21 8. Salonia R et al. Endothelin-1 Is Increased in Cerebrospinal Fluid and Associated with Unfavorable Outcomes in Children after Severe Traumatic Brain Injury. (2010) JOURNAL OF NEUROTRAUMA 0897-7151 27 10 1819-1825 9. Sang N et al. SO2 Inhalation Contributes to the Development and Progression of Ischemic Stroke in the Brain. (2010) TOXICOLOGICAL SCIENCES 1096-6080 1096-0929 114 2 226-236 10. Kreipke CW et al. Clazosentan, a novel endothelin A antagonist, improves cerebral blood ow and behavior after traumatic brain injury. (2011) NEUROLOGICAL RESEARCH 0161-6412 33 2 208-213 11. Kreipke C.W. et al. New frontiers in clinical trials aimed at improving outcome following traumatic brain injury. (2013) Megjelent: Cerebral Blood Flow, Metabolism, and Head Trauma: The Pathotrajectory of Traumatic Brain Injury pp. 155-163 12. Kropinski A et al. Situating the endothelin system in the pathotrajectory of TBI-induced changes in hemodynamics. (2013) Megjelent: Cerebral Blood Flow, Metabolism, and Head Trauma: The Pathotrajectory of Traumatic Brain Injury pp. 95-133

166. Gorlach, C ; Hortobagyi, T ; Hortobagyi, S ; Benyo, Z ; Relton, J ; Whalley, ET ; Wahl, M Bradykinin B2, but not B1, receptor antagonism has a neuroprotective eect after brain injury. JOURNAL OF NEUROTRAUMA 18 : 8 pp. 833-838. , 6 p. (2001) DOI WoS Scopus PubMed Közlemény:1518205 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 27 Független: 25 Függő: 2 Idézett közlemények száma: 1

1. Zausinger S. Eects of LF 16-0687 Ms, a bradykinin B-2 receptor antagonist, on brain edema formation and tissue damage in a rat model of temporary focal cerebral ischemia. (2002) BRAIN RESEARCH 0006-8993 1872-6240 950 1-2 268-278 2. Zausinger S. Bradykinin receptor antagonists in cerebral ischemia and trauma. (2003) IDRUGS 1369-7056 6 10 970-975 3. Hellal F et al. Detrimental role of bradykinin B-2 receptor in a murine model of diuse brain injury. (2003) JOURNAL OF NEUROTRAUMA 0897-7151 20 9 841-851 4. Li DZ et al. LF 16-0687 Ms, a bradykinin B-2 receptor antagonist, reduces ischemic brain injury in a murine modelof transient focal cerebral ischemia. (2003) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 139 8 1539-1547 5. Kaplanski J et al. LF 16-0687 Ms, a new bradykinin B-2 receptor antagonist, decreases ex vivo brain tissue prostaglandin E-2 synthesis after closed head trauma in rats. (2003) RESUSCITATION 0300-9572 56 2 207-213 6. * Gröger M. Release of bradykinin and expression of kinin B2 receptors in the brain: Role for cell death and brain edema formation after focal cerebral ischemia in mice. (2005) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559- 7016 25 8 978-989 7. Ongali R. Autoradiographic analysis of mouse brain kinin B-1 and B-2 receptors after closed head trauma and ability of anatibant mesylate to cross the blood-brain barrier. (2006) JOURNAL OF NEUROTRAUMA 0897-7151 23 5 696-707 8. Ivashkova Y et al. Bradykinin B2 receptor antagonism with LF 18- 1505T reduces brain edema and improves neurological outcome after closed head trauma in rats. (2006) JOURNAL OF TRAUMA: INJURY INFECTION AND CRITICAL CARE 0022-5282 1529-8809 2163-0763 1079-6061 61 4 879-885 9. Juhasz Vedres et al. Dehydroepiandrosterone sulfate (DHEAS) is neuroprotective when administered either before or after injury in a focal cortical cold lesion model. (2006) ENDOCRINOLOGY 0013-7227 1945-7170 147 2 683-686 10. Weiler CR et al. Genetic test indications and interpretations in patients with hereditary angioedema. (2006) MAYO CLINIC PROCEEDINGS 0025-6196 81 7 958-972 11. Klasner B. Therapeutic window of bradykinin B-2 receptor inhibition after focal cerebral ischemia in rats. (2006) NEUROCHEMISTRY INTERNATIONAL 0197-0186 1872-9754 49 5 442-447 12. Zhang Lei et al. Caeic acid attenuates neuronal damage, astrogliosis and glial scar formation in mouse brain with cryoinjury. (2007) LIFE SCIENCES 0024-3205 80 6 530-537 13. * Rákos G et al. Evans blue uorescence permits the rapid visualization of non-intact cells in the perilesional rim of cold- injured rat brain. (2007) ACTA NEUROBIOLOGIAE EXPERIMENTALIS 0065-1400 1689-0035 67 2 149-154 14. Dos Santos et al. Kinin B2 receptor regulates chemokines CCL2 and CCL5 expression and modulates leukocyte recruitment and pathology in experimental autoimmune encephalomyelitis (EAE) in mice. (2008) JOURNAL OF NEUROINFLAMMATION 1742-2094 5 15. Raslan F. Inhibition of bradykinin receptor B1 protects mice from focal brain injury by reducing blood-brain barrier leakage and inammation. (2010) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 30 8 1477-1486 16. Schoeller Karsten et al. Contribution of Bradykinin Receptors to the Development of Secondary Brain Damage After Experimental Subarachnoid Hemorrhage. (2011) NEUROSURGERY 0148-396X 1524-4040 68 4 1118-1123 17. Kleinschnitz C. Kallikrein-kinin system in ischemic stroke and brain trauma. (2011) Megjelent: Kinins pp. 337-354 18. Albert-Weissenberger Christiane et al. Blocking of bradykinin receptor B1 protects from focal closed head injury in mice by reducing axonal damage and astroglia activation. (2012) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559- 7016 32 9 1747-1756 19. Kunz Mathias et al. Bradykinin in Blood and Cerebrospinal Fluid after Acute Cerebral Lesions: Correlations with Cerebral Edema and Intracranial Pressure. (2013) JOURNAL OF NEUROTRAUMA 0897-7151 30 19 1638-1644 20. Albert-Weissenberger Christiane et al. Ischemic stroke and traumatic brain injury: The role of the kallikrein-kinin system. (2013) PROGRESS IN NEUROBIOLOGY: AN INTERNATIONAL REVIEW JOURNAL 0301-0082 1873-5118 101 65-82 21. Ferreira APO. HOE-140, an antagonist of B2 receptor, protects against memory decits and brain damage induced by moderate lateral uid percussion injury in mice. (2014) PSYCHOPHARMACOLOGY 0033-3158 1432-2072 231 9 1935-1948 22. Albert-Weissenberger C. Role of the kallikrein–kinin system in traumatic brain injury. (2014) FRONTIERS IN CELLULAR NEUROSCIENCE 1662-5102 8 23. Arbo Bruno et al. Astrocytes as a target for neuroprotection: Modulation by progesterone and dehydroepiandrosterone. (2016) PROGRESS IN NEUROBIOLOGY: AN INTERNATIONAL REVIEW JOURNAL 0301-0082 1873-5118 144 27-47 24. Li Youhai et al. Enhancement of bradykinin-induced relaxation by focal brain ischemia in the rat middle cerebral artery: Receptor expression upregulation and activation of multiple pathways. (2018) PLOS ONE 1932-6203 1932-6203 13 6 25. Li Y. et al. Enhancement of bradykinin-induced relaxation by focal brain ischemia in the rat middle cerebral artery: Receptor expression upregulation and activation of multiple pathways. (2018) PLOS ONE 1932-6203 1932-6203 13 6 26. Nokkari A et al. Implication of the Kallikrein-Kinin system in neurological disorders: Quest for potential biomarkers and mechanisms. (2018) PROGRESS IN NEUROBIOLOGY: AN INTERNATIONAL REVIEW JOURNAL 0301-0082 1873-5118 165-167 26-50 27. Sharma H.S. et al. Neuroprotective eects of a potent bradykinin B2 receptor antagonist HOE-140 on microvascular permeability, blood ow disturbances, edema formation, cell injury and nitric oxide synthase upregulation following trauma to the spinal cord. (2019) Megjelent: International Review of Neurobiology pp. 103- 152

167. Horváth ; Hrabák, A ; Johnson, RA ; Sándor, P ; Benyó, Z Heme oxygenase blockade increases nitric oxide synthase activity without changing blood ow or vascular resistance in the hypothalamus of rats. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 21 pp. S205-S205. (2001) Közlemény:1161862 Jóváhagyott Forrás Folyóiratcikk (Absztrakt / Kivonat )

168. Lacza, Z ; Hermán, P ; Görlach, C ; Hortobágyi, T ; Sándor, P ; Wahl, M ; Benyó, Z Characterization of cerebral vasomotion following the blocade of the NO-cGMP pathway. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 21 p. S213 (2001) Közlemény:1856235 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

169. Lacza, Z ; Dézsi, L ; Káldi, K ; Horváth, E ; Sándor, P ; Benyó, Z Prostacyclin maintains coronary vasodilation in the absence of nitric oxide. EUROPEAN JOURNAL OF CLINICAL INVESTIGATION 31 : Suppl. 1 p. 6 Paper: 26 (2001) DOI Közlemény:1856231 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

170. Lacza, Z ; Dezsi, L ; Kaldi, K ; Horvath, E ; Monos, E ; Sandor, P ; Benyo, Z Increased prostacyclin production maintains coronary arterial tone during nitric oxide synthase blockade in the rat FASEB JOURNAL 15 : 5 pp. A764-A764. (2001) WoS Közlemény:1628768 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

171. Lacza, Z ; Herman, P ; Gorlach, C ; Hortobagyi, T ; Sandor, P ; Wahl, M ; Benyo, Z NO synthase blockade induces chaotic cerebral vasomotion via activation of thromboxane receptors. STROKE 32 : 11 pp. 2609-2614. , 6 p. (2001) DOI WoS Scopus PubMed Közlemény:1518204 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 33 Független: 21 Függő: 12 Idézett közlemények száma: 5

1. Tsoukias NM et al. A theoretical model of nitric oxide transport in arterioles: frequency- vs. amplitude-dependent control of cGMP formation. (2004) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 286 3 H1043-H1056 2. B Benyó et al. Characterization of the Temporal Pattern of Cerebral Blood Flow Oscillations. (2004) Megjelent: Neural Networks 2004. Proceedings of the IEEE International Joint Conference Vols 1-4 pp. 2467-2470 3. * B Benyó et al. Characterization of Cerebral Blood Flow Oscillations Using Dierent Classication Methods. (2005) Megjelent: Proceedings of the 16th IFAC World Congress pp. 214- 219 4. B Benyó et al. Classication of Time Series using Singular Values and Wavelet Subband Analysis with ANN and SVM Classiers. (2005) Megjelent: Proceedings of the 9th IEEE International Conference on Intelligent Engineering Systems (INES 2005) pp. 239-243 5. Rahman A et al. Eects of cGMP on coordination of vascular smooth muscle cells of rat mesenteric small arteries. (2005) JOURNAL OF VASCULAR RESEARCH 1018-1172 1423-0135 42 4 301-311 6. Gonzales RJ et al. Testosterone treatment increases thromboxane function in rat cerebral arteries. (2005) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 289 2 H578-H585 7. B Benyó. Analysis of Temporal Patterns of Physiological Parameters. (2006) Megjelent: Neural Networks in Healthcare : Potential and Challenges pp. 285-317 8. Haddock RE et al. Endothelial coordination of cerebral vasomotion via myoendothelial gap junctions containing connexins 37 and 40. (2006) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 291 5 H2047-H2056 9. * Eke A et al. Fractal and noisy CBV dynamics in humans: inuence of age and gender. (2006) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 26 7 891-898 10. * Herman P et al. Nonlinear analysis of blood cell ux uctuations in the rat brain cortex during stepwise hypotension challenge. (2006) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 26 9 1189-1197 11. * Hortobagyi L et al. Adaptation of the hypothalamic blood ow to chronic nitric oxide deciency is independent of vasodilator prostanoids. (2007) BRAIN RESEARCH 0006-8993 1872-6240 1131 1 129-137 12. * Leszl-Ishiguro M et al. Inuence of the heme-oxygenase pathway on cerebrocortical blood ow. (2007) NEUROREPORT 0959-4965 18 11 1193-1197 13. McNeish AJ et al. Thromboxane A(2) inhibition of SKCa after NO synthase block in rat middle cerebral artery. (2007) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 151 4 441- 449 14. Hernanz R et al. Ouabain treatment increases nitric oxide bioavailability and decreases superoxide anion production in cerebral vessels. (2008) JOURNAL OF HYPERTENSION 0263-6352 1473-5598 26 10 1944-1954 15. * Lacza Z et al. Additive eect of cyclooxygenase and nitric oxide synthase blockade on the cerebrocortical microcirculation. (2009) NEUROREPORT 0959-4965 20 11 1027-1031 16. Abatay H. A three-state non-linear model of vascular nitric oxide transport. (2009) Megjelent: Proceedings of the 31st Annual International Conference of the IEEE Engineering in Medicine and Biology Society: Engineering th... pp. 4917-4920 17. Toda N et al. Cerebral Blood Flow Regulation by Nitric Oxide: Recent Advances. (2009) PHARMACOLOGICAL REVIEWS 0031- 6997 1521-0081 61 1 62-97 18. Sharma V. Deterministic chaos and fractal complexity in the dynamics of cardiovascular behavior: Perspectives on a new frontier. (2009) OPEN CARDIOVASCULAR MEDICINE JOURNAL 1874-1924 3 110-123 19. Abatay H. Analysis of the Eects of Nitric Oxide on Vasomotion. (2010) Megjelent: 6TH WORLD CONGRESS OF BIOMECHANICS (WCB 2010) pp. 863-866 20. Nguyen T-U. A second-order biomechanical model of ow- mediated dilation response in obese post-menopausal women. (2010) Megjelent: 6th World Congress of Biomechanics, WCB 2010 pp. 867-870 21. * Horvath B et al. Hypersensitivity to thromboxane receptor mediated cerebral vasomotion and CBF oscillations during acute NO-deciency in rats.. (2010) PLOS ONE 1932-6203 1932-6203 5 12 22. Matchkov VV. Mechanisms of cellular synchronization in the vascular wall. (2010) DANISH MEDICAL BULLETIN 0907-8916 1603-9629 2245-1919 57 10 23. Parthimos D et al. Deterministic nonlinear features of cutaneous perfusion are lost in diabetic subjects with neuropathy. (2011) MICROVASCULAR RESEARCH 0026-2862 1095-9319 82 1 42-51 24. * Kiss L et al. Human internal thoracic artery grafts exhibit severe morphological and functional damage and spasmic vasomotion due to oxidative stress.. (2011) MEDICAL SCIENCE MONITOR 1234-1010 1643-3750 17 7 CR411-CR416 25. Aalkjaer C et al. Vasomotion - what is currently thought?. (2011) ACTA PHYSIOLOGICA 1748-1708 1748-1716 202 3 253-269 26. * Ruisanchez E et al. Perivascular expression and potent vasoconstrictor eect of dynorphin a in cerebral arteries.. (2012) PLOS ONE 1932-6203 1932-6203 7 5 27. * Lacar B et al. S Phase Entry of Neural Progenitor Cells Correlates with Increased Blood Flow in the Young Subventricular Zone. (2012) PLOS ONE 1932-6203 1932-6203 7 2 28. McNeish Alister et al. Statins and Selective Inhibition of Rho Kinase Protect Small Conductance Calcium-Activated Potassium Channel Function (K(Ca)2.3) in Cerebral Arteries. (2012) PLOS ONE 1932-6203 1932-6203 7 10 29. Vuletic Vladimira et al. Eect of indomethacin on cerebrovascular reactivity in patients with type 2 diabetes mellitus. (2013) DIABETES RESEARCH AND CLINICAL PRACTICE 0168-8227 1872- 8227 101 1 81-87 30. Di Marco LY et al. Is Vasomotion in Cerebral Arteries Impaired in Alzheimer's Disease?. (2015) JOURNAL OF ALZHEIMER'S DISEASE 1387-2877 1875-8908 46 1 35-53 31. * Polycarpou A et al. Adaptation of the Cerebrocortical Circulation to Carotid Artery Occlusion Involves Blood Flow Redistribution between Cortical Regions and is Independent of eNOS.. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 311 4 H972-H980 32. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801 33. Addison M et al. NO synthase inhibition attenuates EDHF- mediated relaxation induced by TRPV4 channel agonist GSK1016790A in the rat pulmonary artery: Role of TxA(2). (2016) PHARMACOLOGICAL REPORTS 1734-1140 68 3 620-626

172. Lacza, Z ; Káldi, K ; Horváth, E ; Balla, T ; Mouzarou, A ; Sándor, P ; Benyó, Z Interaction between nitric oxide and prostacyclin in the cerebral microcirculation of rats. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 21 : suppl1 Paper: S91 (2001) Közlemény:1476038 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat ) Nyilvános idézők összesen: 1 Független: 0 Függő: 1

1. * Hortobagyi L et al. Adaptation of the hypothalamic blood ow to chronic nitric oxide deciency is independent of vasodilator prostanoids. (2007) BRAIN RESEARCH 0006-8993 1872-6240 1131 1 129-137 173. Lacza, Z ; Kaldi, K ; Kovecs, K ; Gorlach, C ; Nagy, Z ; Sandor, P ; Benyo, Z ; Wahl, M Involvement of prostanoid release in the mediation of UTP-induced cerebrovascular contraction in the rat BRAIN RESEARCH 896 : 1-2 pp. 169-174. , 6 p. (2001) DOI WoS Scopus PubMed Közlemény:1052608 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 7 Független: 5 Függő: 2 Idézett közlemények száma: 3

1. * Lacza Z et al. NO synthase blockade induces chaotic cerebral vasomotion via activation of thromboxane receptors.. (2001) STROKE 0039-2499 1524-4628 32 11 2609-2614 2. Burnstock Georey et al. Cellular distribution and functions of P2 receptor subtypes in dierent systems. (2004) INTERNATIONAL REVIEW OF CYTOLOGY-A SURVEY OF CELL BIOLOGY 0074-7696 1937-6448 2004 31-304 3. Gonzales RJ et al. Testosterone treatment increases thromboxane function in rat cerebral arteries. (2005) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 289 2 H578-H585 4. Burnstock G et al. Adenosine and ATP Receptors in the Brain. (2011) CURRENT TOPICS IN MEDICINAL CHEMISTRY 1568-0266 1873-4294 11 8 973-1011 5. Burnstock Georey et al. Purinergic Signaling and Blood Vessels in Health and Disease. (2014) PHARMACOLOGICAL REVIEWS 0031- 6997 1521-0081 66 1 102-192 6. Ralevic Vera et al. Purinergic transmission in blood vessels. (2015) AUTONOMIC NEUROSCIENCE-BASIC & CLINICAL 1566-0702 1872- 7484 191 48-66 7. * Pal E et al. Vitamin D deciency causes inward hypertrophic remodeling and alters vascular reactivity of rat cerebral arterioles.. (2018) PLOS ONE 1932-6203 1932-6203 13 2

174. Sandor, P ; Komjati, K ; De Jong, W ; Erdos, B ; Lacza, Z ; Benyo, Z ; Reivich, M Hemorrhage-induced brain ischemia: Modulatory role of endogenous opioids and antiopioids in the cerebrovascular bed In: Fukuuchi, Y; Tomita, M; Koto, A (szerk.) ISCHEMIC BLOOD FLOW IN THE BRAIN Tokyo, Japán : Springer-Verlag, (2001) pp. 178-194. , 17 p. DOI WoS Közlemény:1631129 Érvényesített Forrás Idéző Könyvrészlet (Konferenciaközlemény ) Nyilvános idézők összesen: 1 Független: 0 Függő: 1 Idézett közlemények száma: 14

1. * Komjáti K. Interactions between the endothelium-derived relaxing factor/nitric oxide system and the endogenous opiate system in the modulation of cerebral and spinal vascular CO2 responsiveness. (2002) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 21 8 937-944

175. Benyo, Z ; Lacza, Z ; Hortobagyi, T ; Gorlach, C ; Wahl, M Contribution of neuronal nitric oxide synthase to the relaxant eect of acetylcholine but not to the maintenance of the resting tone in rat basilar arteries JOURNAL OF PHYSIOLOGY-LONDON 526 : Suppl. S pp. 126P-126P. (2000) WoS Közlemény:1628774 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

176. Benyo, Z ; Lacza, Z ; Hortobagyi, T ; Gorlach, C ; Wahl, M Functional importance of neuronal nitric oxide synthase in the endothelium of rat basilar arteries. BRAIN RESEARCH 877 : 1 pp. 79-84. Paper: 10980246 , 6 p. (2000) DOI WoS Scopus PubMed Közlemény:1518209 Érvényesített Forrás Idéző Folyóiratcikk (Rövid közlemény ) Nyilvános idézők összesen: 18 Független: 13 Függő: 5 Idézett közlemények száma: 1 1. Lindauer U et al. Nitric oxide from perivascular nerves modulates cerebral arterial pH reactivity. (2001) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 281 3 H1353-H1363 2. * Lacza Z et al. Hydrogen peroxide acts as an EDHF in the piglet pial vasculature in response to bradykinin. (2002) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 283 1 H406-H411 3. * Benyo Z et al. Neuronal nitric oxide synthase in the cerebrovascular endothelium. (2002) Megjelent: BRAIN ACTIVATION AND CBF CONTROL, PROCEEDINGS pp. 369-377 4. * Hortobagyi T et al. Inhibition of neuronal nitric oxide synthase- mediated activation of poly(ADP-ribose) polymerase in traumatic brain injury: neuroprotection by 3-aminobenzamide.. (2003) NEUROSCIENCE 0306-4522 1873-7544 121 4 983-990 5. Wang WH et al. NOS isoenzyme content in brain nuclei as related to food intake in experimental cancer cachexia. (2005) MOLECULAR BRAIN RESEARCH 0169-328X 134 2 205-214 6. Andresen J et al. Endothelial inuences on cerebrovascular tone. (2006) JOURNAL OF APPLIED PHYSIOLOGY 8750-7587 1522-1601 100 1 318-327 7. Coutard M et al. High-ow-induced arterial remodeling in rats with dierent susceptibilities to cerebral aneurysms. (2006) JOURNAL OF VASCULAR RESEARCH 1018-1172 1423-0135 43 3 217-228 8. Teixeira CE et al. Vasorelaxing eect of BAY 41-2272 in rat basilar artery - Involvement of cGMP-dependent and independent mechanisms. (2006) HYPERTENSION 0194-911X 47 3 596-602 9. Miyamoto Atsushi et al. Ibuprofen or ozagrel increases NO release and L-nitro, arginine induces TXA(2) release from cultured porcine basilar arterial endothelial cells. (2007) VASCULAR PHARMACOLOGY 1537-1891 1879-3649 46 2 85-90 10. Sedlakova B et al. Eect of 7-nitroindazole on the expression of intracellular calcium channels in the kidney of spontaneously hypertensive rats. (2009) GENERAL PHYSIOLOGY AND BIOPHYSICS 0231-5882 28 3 225-232 11. Cacanyiova S et al. Eect of chronic nNOS inhibition on blood pressure, vasoactivity, and arterial wall structure in Wistar rats. (2009) NITRIC OXIDE-BIOLOGY AND CHEMISTRY 1089-8603 1089- 8611 20 4 304-310 12. Mayhan WG et al. Nitric oxide synthase-dependent responses of the basilar artery during acute infusion of nicotine. (2009) NICOTINE AND TOBACCO RESEARCH 1462-2203 1469-994X 11 3 270-277 13. Institoris A et al. Impaired vascular responses of insulin-resistant rats after mild subarachnoid hemorrhage. (2011) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 300 6 H2080-H2087 14. Peterson EC et al. Regulation of cerebral blood ow. (2011) INTERNATIONAL JOURNAL OF VASCULAR MEDICINE 2090-2824 2090-2832 2011 15. Cacanyiova Sona. The Vasoactive Role of Nitric Oxide: Physiological and Morphological Aspects. (2011) CURRENT PHARMACEUTICAL BIOTECHNOLOGY 1389-2010 1873-4316 12 9 1294-1304 16. * Ruisanchez E et al. Perivascular expression and potent vasoconstrictor eect of dynorphin a in cerebral arteries.. (2012) PLOS ONE 1932-6203 1932-6203 7 5 17. Cacanyiova S et al. Dierent vasoactive eects of chronic endothelial and neuronal NO-synthase inhibition in young Wistar rats. (2014) JOURNAL OF PHYSIOLOGY AND BIOCHEMISTRY 1138- 7548 70 3 749-760 18. * Polycarpou A et al. Adaptation of the Cerebrocortical Circulation to Carotid Artery Occlusion Involves Blood Flow Redistribution between Cortical Regions and is Independent of eNOS.. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 311 4 H972-H980

177. Gorlach, C ; Hortobagyi, T ; Benyo, Z ; Wahl, M Aminoguanidine reduces brain lesion volume after cold injury in the rat. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY 440 : 2 pp. 309-314. , 6 p. (2000) DOI WoS Scopus PubMed Közlemény:1518210 Érvényesített Forrás Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 20 Független: 16 Függő: 4

1. * Gorlach C et al. Neuronal nitric oxide synthase inhibitor has a neuroprotective eect in a rat model of brain injury.. (2000) RESTORATIVE NEUROLOGY AND NEUROSCIENCE 0922-6028 17 2- 3 71-76 2. Laurer HL et al. Pharmacologic therapy in traumatic brain injury: Update on experimental treatment strategies. (2001) CURRENT PHARMACEUTICAL DESIGN 1381-6128 1873-4286 7 15 1505-1516 3. Nag S. The blood-brain barrier and cerebral angiogenesis: lessons from the cold-injury model. (2002) TRENDS IN MOLECULAR MEDICINE 1471-4914 1471-499X 8 1 38-44 4. * Hortobagyi T et al. Inhibition of neuronal nitric oxide synthase- mediated activation of poly(ADP-ribose) polymerase in traumatic brain injury: neuroprotection by 3-aminobenzamide.. (2003) NEUROSCIENCE 0306-4522 1873-7544 121 4 983-990 5. * Lacza Z et al. PARP inhibition improves the eectiveness of neural stem cell transplantation in experimental brain trauma.. (2003) INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE 1107-3756 1791-244X 12 2 153-159 6. Rinecker M. Secondary growth of a cortical necrosis: eect of NOS inhibition by aminoguanidine post insult. (2003) ACTA NEUROCHIRURGICA 0001-6268 0942-0940 145 11 977-981 7. Jones NC et al. A detrimental role for nitric oxide synthase-2 in the pathology resulting from acute cerebral injury. (2004) JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY 0022-3069 63 7 708-720 8. Atan MS et al. Eects of aminoguanidine and L-arginine methyl ester resuscitation following induction of uid-percussion injury and severe controlled hemorrhagic shock in the rat brain. (2004) JOURNAL OF NEUROSURGERY 0022-3085 101 1 138-144 9. Cherian L. Nitric oxide in traumatic brain injury. (2004) BRAIN PATHOLOGY 1015-6305 1750-3639 14 2 195-201 10. Bayir H et al. Enhanced oxidative stress in iNOS-decient mice after traumatic brain injury: support for a neuroprotective role of iNOS. (2005) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 25 6 673-684 11. Gahm Caroline et al. Neuroprotection by selective inhibition of inducible nitric oxide synthase after experimental brain contusion. (2006) JOURNAL OF NEUROTRAUMA 0897-7151 23 9 1343-1354 12. Louin G et al. Selective inhibition of inducible nitric oxide synthase reduces neurological decit but not cerebral edema following traumatic brain injury. (2006) NEUROPHARMACOLOGY 0028-3908 1873-7064 50 2 182-190 13. Lescot T. Stratégies anti-inammatoires et traumatisme crânien. (2006) ANNALES FRANCAISES D ANESTHESIE ET DE REANIMATION 0750-7658 25 7 755-760 14. * Rákos G et al. Evans blue uorescence permits the rapid visualization of non-intact cells in the perilesional rim of cold- injured rat brain. (2007) ACTA NEUROBIOLOGIAE EXPERIMENTALIS 0065-1400 1689-0035 67 2 149-154 15. Rangel-Castilla L. Nitric oxide metabolism after traumatic brain injury. (2007) Megjelent: Intensive Care Medicine: Annual Update 2007 pp. 739-747 16. Besson Valerie. Drug targets for traumatic brain injury from poly(ADP-ribose)polymerase pathway modulation. (2009) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 157 5 695- 704 17. Covey Matthew. Dening the Critical Period for Neocortical Neurogenesis after Pediatric Brain Injury. (2010) DEVELOPMENTAL NEUROSCIENCE 0378-5866 32 5-6 488-498 18. Stevanovic I et al. The eect of aminoguanidine, an inducible nitric oxide synthase inhibitor, on ALCL3 toxicity in the rat hippocampus. (2010) ARCHIVES OF BIOLOGICAL SCIENCES 0354- 4664 1821-4339 62 4 981-991 19. Schiavone Stefania et al. The NADPH oxidase NOX2 mediates loss of parvalbumin interneurons in traumatic brain injury: human autoptic immunohistochemical evidence. (2017) SCIENTIFIC REPORTS 2045-2322 7 1 20. Che Xiaoru et al. The Role of Gaseous Molecules in Traumatic Brain Injury: An Updated Review. (2018) FRONTIERS IN NEUROSCIENCE 1662-4548 1662-453X 12

178. Gorlach, C ; Hortobagyi, T ; Hortobagyi, S ; Benyo, Z Neuronal nitric oxide synthase inhibitor has a neuroprotective eect in a rat model of brain injury. RESTORATIVE NEUROLOGY AND NEUROSCIENCE 17 : 2-3 pp. 71-76. , 6 p. (2000) WoS Scopus PubMed Teljes dokumentum Közlemény:1518206 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 8 Független: 7 Függő: 1 Idézett közlemények száma: 1

1. Seiler N. The polyamine oxidase inactivator MDL 72527. (2002) Megjelent: Progress in Drug Research pp. 1-40 2. * Hortobagyi T et al. Inhibition of neuronal nitric oxide synthase- mediated activation of poly(ADP-ribose) polymerase in traumatic brain injury: neuroprotection by 3-aminobenzamide.. (2003) NEUROSCIENCE 0306-4522 1873-7544 121 4 983-990 3. Bharatam PV. Electron delocalization in aminoguanidine: A computational study. (2004) JOURNAL OF PHYSICAL CHEMISTRY A 1089-5639 1520-5215 108 47 10509-10517 4. Li RC. Nitric oxide synthase and intermittent hypoxia-induced spatial learning decits in the rat. (2004) NEUROBIOLOGY OF DISEASE 0969-9961 1095-953X 17 1 44-53 5. Mollace V et al. Modulation of prostaglandin biosynthesis by nitric oxide and nitric oxide donors. (2005) PHARMACOLOGICAL REVIEWS 0031-6997 1521-0081 57 2 217-252 6. Prabhakaran K. Molecular mechanism of manganese exposure- induced dopaminergic toxicity. (2008) BRAIN RESEARCH BULLETIN 0361-9230 1873-2747 76 4 361-367 7. Srinivasan Krishnamoorthy. 3-Bromo-7-nitroindazole attenuates brain ischemic injury in diabetic stroke via inhibition of endoplasmic reticulum stress pathway involving CHOP. (2012) LIFE SCIENCES 0024-3205 90 3-4 154-160 8. Ozdemir Namik. Synthesis, spectroscopic (FT-IR/NMR) characterization, X-ray structure and DFT studies on (E)-2-(1- phenylethylidene) hydrazinecarboximidamide nitrate hemimethanol. (2013) SPECTROCHIMICA ACTA PART A- MOLECULAR AND BIOMOLECULAR SPECTROSCOPY 1386-1425 114 175-182

179. Görlach, C ; Hortobágyi, T ; Hortobágyi, S ; Benyó, Z ; Relton, JK ; Whalley, ET ; Wahl, M Application of bradykinin B2 receptor antagonist is protective after brain injury, while antagonism of B1 receptor has no eect. RESTORATIVE NEUROLOGY AND NEUROSCIENCE 16 p. 260 (2000) Közlemény:1856225 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

180. Görlach, C ; Hortobágyi, T ; Hortobágyi, S ; Benyó, Z ; Relton, JK ; Whalley, ET ; Wahl, M Neuroprotection by a bradykinin B2 receptor antagonist after traumatic brain injury is not attenuated by the simultaneous application of a bradykinin B1 receptor antagonist. JOURNAL OF PHYSIOLOGY-LONDON 526 pp. 50-51. , 2 p. (2000) Közlemény:1856219 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

181. Hortobagyi, T ; Hortobagyi, S ; Gorlach, C ; Kovecs, K ; Benyo, Z ; Wahl, M Traumatic brain injury in the mouse: characterization of a novel cold lesion model for studying brain oedema JOURNAL OF PHYSIOLOGY-LONDON 526 : Suppl. pp. 48P-48P. (2000) WoS Közlemény:1628772 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

182. Hortobagyi, T ; Hortobagyi, S ; Gorlach, C ; Harkany, T ; Benyo, Z ; Gorogh, T ; Nagel, W ; Wahl, M A novel brain trauma model in the mouse: eects of dexamethasone treatment. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY 441 : 2-3 pp. 409-415. , 7 p. (2000) DOI WoS Scopus PubMed Közlemény:1518208 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 19 Független: 14 Függő: 5 Idézett közlemények száma: 2

1. * Ábrahám IM et al. Action of glucocorticoids on survival of nerve cells: promoting neurodegeneration or neuroprotection?. (2001) JOURNAL OF NEUROENDOCRINOLOGY 0953-8194 1365-2826 13 9 749-760 2. * Gorlach C et al. Bradykinin B2, but not B1, receptor antagonism has a neuroprotective eect after brain injury.. (2001) JOURNAL OF NEUROTRAUMA 0897-7151 18 8 833-838 3. * Hortobagyi T et al. Inhibition of neuronal nitric oxide synthase- mediated activation of poly(ADP-ribose) polymerase in traumatic brain injury: neuroprotection by 3-aminobenzamide.. (2003) NEUROSCIENCE 0306-4522 1873-7544 121 4 983-990 4. Lacza Z et al. Neural stem cell transplantation in cold lesion: A novel approach for the investigation of brain trauma and repair. (2003) BRAIN RESEARCH PROTOCOLS 1385-299X 11 3 145-154 5. * Lacza Z et al. PARP inhibition improves the eectiveness of neural stem cell transplantation in experimental brain trauma.. (2003) INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE 1107-3756 1791-244X 12 2 153-159 6. Vink R et al. Recent advances in the development of multifactorial therapies for the treatment of traumatic brain injury. (2004) EXPERT OPINION ON INVESTIGATIONAL DRUGS 1354-3784 13 10 1263-1274 7. Ekin S. Eects of dexamethasone on trace elements and serum protein patterns following brain trauma in rats. (2005) BIOLOGICAL TRACE ELEMENT RESEARCH 0163-4984 1559-0720 107 1 53-60 8. Gahm C et al. Neuronal degeneration and iNOS expression in experimental brain contusion following treatment with colchicine, dexamethasone, tirilazad mesylate and nimodipine. (2005) ACTA NEUROCHIRURGICA 0001-6268 0942-0940 147 10 1071-1084 9. Cho YH. The behavioral eect of human mesenchymal stem cell transplantation in cold brain injured rats. (2006) ACTA NEUROCHIRURGICA SUPPLEMENT 0065-1419 13 99 125-132 10. Zhang Lei et al. Caeic acid attenuates neuronal damage, astrogliosis and glial scar formation in mouse brain with cryoinjury. (2007) LIFE SCIENCES 0024-3205 80 6 530-537 11. Ágoston VA et al. Dierent behaviour of implanted stem cells in intact and lesioned forebrain cortices. (2007) NEUROPATHOLOGY AND APPLIED NEUROBIOLOGY 0305-1846 1365-2990 33 5 510- 522 12. * Rákos G et al. Evans blue uorescence permits the rapid visualization of non-intact cells in the perilesional rim of cold- injured rat brain. (2007) ACTA NEUROBIOLOGIAE EXPERIMENTALIS 0065-1400 1689-0035 67 2 149-154 13. Demir Oezguer et al. Investigation of neuroprotective eect of dexamethasone by using nitric oxide and leukocyte levels in experimental head trauma. (2007) INTERNATIONAL JOURNAL OF NEUROSCIENCE 0020-7454 1563-5279 117 12 1719-1730 14. Grasso Giovanni. Neuroprotection by erythropoietin administration after experimental traumatic brain injury. (2007) BRAIN RESEARCH 0006-8993 1872-6240 1182 99-105 15. Ichikawa-Shindo Yuka et al. The GPCR modulator protein RAMP2 is essential for angiogenesis and vascular integrity. (2008) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 118 1 29-39 16. Moorthy Ranjith et al. Intralesional mesenchymal stromal cell transplant in a rodent model of cortical cryoinjury. (2011) NEUROLOGY INDIA 0028-3886 1998-4022 59 4 573-578 17. Shi Wen-Zhen et al. Aquaporin-4 deciency attenuates acute lesions but aggravates delayed lesions and microgliosis after cryoinjury to mouse brain. (2012) NEUROSCIENCE BULLETIN 1673-7067 1995-8218 28 1 61-68 18. Turel MK et al. Eect of pretreatment with a tyrosine kinase inhibitor (PP1) on brain oedema and neurological function in an automated cortical cryoinjury model in mice. (2013) JOURNAL OF CLINICAL NEUROSCIENCE 0967-5868 20 4 593-596 19. Michinaga Shotaro et al. Pathogenesis of Brain Edema and Investigation into Anti-Edema Drugs. (2015) INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 1661-6596 1422-0067 16 5 9949-9975

183. Hortobágyi, T ; Hortobágyi, S ; Benyó, Z ; Wahl, M ; Görlach, C Poly (ADP-ribose) synthase is an important eector after experimental traumatic brain injury. JOURNAL OF VASCULAR RESEARCH 38 p. 507 (2000) Közlemény:1856227 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

184. Hortobágyi, T ; Hortobágyi, S ; Görlach, C ; Harkány, T ; Benyó, Z ; Wahl, M Characterization of a novel brain trauma model in the mouse. RESTORATIVE NEUROLOGY AND NEUROSCIENCE 16 p. 239 (2000) Közlemény:1856222 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

185. Hortobágyi, T ; Görlach, C ; Hortobágyi, S ; Benyó, Z ; Relton, J ; Whalley, ET ; Wahl, M Neuroprotection by a bradykinin B2 receptor antagonist after brain injury is not attenuated by coapplication of a B1 antagonist. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY 439 : suppl 6 p. 437 (2000) Közlemény:1856217 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat ) Nyilvános idézők összesen: 1 Független: 0 Függő: 1 1. * Hortobágyi T. A novel brain trauma model in the mouse: Eects of dexamethasone treatment. (2000) PFLÜGERS ARCHIV FÜR DIE GESAMTE PHYSIOLOGIE DES MENSCHEN UND DER TIERE 0365- 267X 441 409-415

186. Lacza, Z ; Erdős, B ; Komjáti, K ; Benyó, Z ; Sándor, P Baseline Laser Doppler ow carries information regarding the microcirculatory eects of the L-arginine – nitric oxide pathway. SOCIETY FOR NEUROSCIENCE ABSTRACTS 26 p. 1738 (2000) Közlemény:1856226 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

187. Lacza, Z ; Erdos, B ; Gorlach, C ; Wahl, M ; Sandor, P ; Benyo, Z The cerebrocortical microcirculatory eect of nitric oxide synthase blockade is dependent upon baseline red blood cell ow in the rat NEUROSCIENCE LETTERS 291 : 2 pp. 65-68. , 4 p. (2000) DOI WoS Scopus PubMed Közlemény:1628776 Érvényesített Forrás Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 12 Független: 6 Függő: 6

1. Tuettenberg J. Nitric oxide modulates cerebral blood ow stimulation by acetazolamide in the rat cortex: a laser Doppler scanning study. (2001) NEUROSCIENCE LETTERS 0304-3940 1872- 7972 315 1-2 65-68 2. * Lacza Z et al. NO synthase blockade induces chaotic cerebral vasomotion via activation of thromboxane receptors.. (2001) STROKE 0039-2499 1524-4628 32 11 2609-2614 3. Rassaf T. Evidence for in vivo transport of bioactive nitric oxide in human plasma. (2002) JOURNAL OF CLINICAL INVESTIGATION 0021-9738 1558-8238 109 9 1241-1248 4. * Horvath B et al. Contribution of the heme oxygenase pathway to the maintenance of the hypothalamic blood ow during diminished nitric oxide synthesis. (2003) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 23 6 653- 657 5. B Benyó et al. Characterization of the Temporal Pattern of Cerebral Blood Flow Oscillations. (2004) Megjelent: Neural Networks 2004. Proceedings of the IEEE International Joint Conference Vols 1-4 pp. 2467-2470 6. * B Benyó et al. Characterization of Cerebral Blood Flow Oscillations Using Dierent Classication Methods. (2005) Megjelent: Proceedings of the 16th IFAC World Congress pp. 214- 219 7. B Benyó et al. Classication of Time Series using Singular Values and Wavelet Subband Analysis with ANN and SVM Classiers. (2005) Megjelent: Proceedings of the 9th IEEE International Conference on Intelligent Engineering Systems (INES 2005) pp. 239-243 8. B Benyó. Analysis of Temporal Patterns of Physiological Parameters. (2006) Megjelent: Neural Networks in Healthcare : Potential and Challenges pp. 285-317 9. * Lacza Z et al. Urotensin-II is a nitric oxide-dependent vasodilator in the pial arteries of the newborn pig. (2006) LIFE SCIENCES 0024-3205 78 23 2763-2766 10. * Leszl-Ishiguro M et al. Inuence of the heme-oxygenase pathway on cerebrocortical blood ow. (2007) NEUROREPORT 0959-4965 18 11 1193-1197 11. * Horvath B et al. Hypersensitivity to thromboxane receptor mediated cerebral vasomotion and CBF oscillations during acute NO-deciency in rats.. (2010) PLOS ONE 1932-6203 1932-6203 5 12 12. Ances BM. Interaction between nitric oxide synthase inhibitor induced oscillations and the activation ow coupling response. (2010) BRAIN RESEARCH 0006-8993 1872-6240 1309 19-28 188. Lacza, Z ; Kaldi, K ; Kovecs, K ; Gorlach, C ; Nagy, Z ; Sandor, P ; Benyo, Z ; Wahl, M Thromboxane A2 mediates the cerebrovascular contractile eect of uridine-5 '-triphosphate JOURNAL OF PHYSIOLOGY-LONDON 526 : Suppl. pp. 141P-142P. (2000) WoS Közlemény:1628775 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

189. Benyo, Z ; Gorlach, C ; Wahl, M Interaction between nitric oxide and thromboxane A2 in the regulation of the resting cerebrovascular tone. ADVANCES IN EXPERIMENTAL MEDICINE AND BIOLOGY 471 pp. 373-379. , 7 p. (1999) DOI WoS Scopus PubMed Közlemény:1848226 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 5 Független: 3 Függő: 2 Idézett közlemények száma: 4

1. * Benyo Z et al. Functional importance of neuronal nitric oxide synthase in the endothelium of rat basilar arteries.. (2000) BRAIN RESEARCH 0006-8993 1872-6240 877 1 79-84 2. * Benyo Z et al. Neuronal nitric oxide synthase in the cerebrovascular endothelium. (2002) Megjelent: BRAIN ACTIVATION AND CBF CONTROL, PROCEEDINGS pp. 369-377 3. Dandona P et al. Endothelial dysfunction and hypertension in diabetes mellitus. (2004) MEDICAL CLINICS OF NORTH AMERICA 0025-7125 1557-9859 88 4 911-931 4. Dandona P et al. Vascular reactivity and thiazolidinediones. (2004) AMERICAN JOURNAL OF MEDICINE 0002-9343 1555-7162 115 8 Suppl. 81-86 5. Miyamoto Atsushi et al. Ibuprofen or ozagrel increases NO release and L-nitro, arginine induces TXA(2) release from cultured porcine basilar arterial endothelial cells. (2007) VASCULAR PHARMACOLOGY 1537-1891 1879-3649 46 2 85-90

190. Benyo, Z ; Szabo, C ; Kovach, AGB ; Sandor, P Prevention of the hemorrhagic hypotension-induced hepatic arterial vasoconstriction by L-arginine and naloxone SHOCK 11 : 5 pp. 342-346. , 5 p. (1999) DOI WoS Scopus PubMed Közlemény:1628782 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) | Hibás/hiányos Nyilvános idézők összesen: 3 Független: 3 Függő: 0 Idézett közlemények száma: 6

1. Vaccarino AL et al. Endogenous opiates: 1999. (2000) PEPTIDES 0196-9781 1873-5169 21 12 1975-2034 2. Faruk EM. Comparative study on the eect of N-acetyl-cysteine versus sulpiride on experimentally induced stress in ovary of albino rats: A histological, immunohistochemical and biochemical study. (2015) EGYPTIAN JOURNAL OF HISTOLOGY 1110-0559 2090-2417 38 4 756-765 3. Yuvaraj Babu. Antistress potential of convolvulus pluricaulis choisy in chronic cold swimming stress rat model. (2018) INTERNATIONAL JOURNAL OF RESEARCH IN PHARMACEUTICAL SCIENCES 0975-7538 9 2 349-352

191. Benyó, Z ; Lacza, Zs ; Görlach, Ch ; Wahl, M Selective inhibition of neuronal nitric oxide synthase fails to alter the resting tension and the relaxant eect of bradykinin in isolated rat middle cerebral arteries ACTA PHYSIOLOGICA HUNGARICA 86 : 2 pp. 161-165. , 5 p. (1999) PubMed Scopus Teljes dokumentum Közlemény:2520389 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 3 Független: 2 Függő: 1 Idézett közlemények száma: 4

1. Hayashi T et al. Neuronal nitric oxide has a role as a perfusion regulator and a synaptic modulator in cerebellum but not in neocortex during somatosensory stimulation - An animal PET study. (2002) NEUROSCIENCE RESEARCH 0168-0102 44 155-165 2. * Benyo Z et al. Neuronal nitric oxide synthase in the cerebrovascular endothelium. (2002) Megjelent: BRAIN ACTIVATION AND CBF CONTROL, PROCEEDINGS pp. 369-377 3. Toda N et al. Cerebral Blood Flow Regulation by Nitric Oxide: Recent Advances. (2009) PHARMACOLOGICAL REVIEWS 0031- 6997 1521-0081 61 1 62-97

192. Benyó, Z ; Lacza, Z ; Görlach, C ; Wahl, M Role of soluble guanylyl cyclase in the regulation of the resting cerebrovascular tone. FUNDAMENTAL & CLINICAL PHARMACOLOGY 31 : suppl 1 p. 165 (1999) Közlemény:1856212 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

193. Benyó, Z ; Gallatz, K ; SirenA-L ; Görlach, C ; Sándor, P ; Wahl, M Eect of dynorphins on the cerebrovascular tone. FUNDAMENTAL & CLINICAL PHARMACOLOGY 31 : suppl 1 p. 58 (1999) Közlemény:1856209 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

194. Benyó, Z ; Gallatz, K ; Siren, A-L ; Görlach, C ; Sándor, P ; Wahl, M Dynorphinergic control of the cerebrovascular tone. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 19 p. S347 (1999) Közlemény:1856198 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

195. Benyó, Z ; Görlach, C ; Lacza, Z ; Wahl, M Involvement of thromboxane A2 in the mediation of the cerebrovascular constriction induced by inhibition of the soluble guanylyl cyclase. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 19 p. S257 (1999) Közlemény:1856191 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

196. Gorlach, C ; Benyo, Z ; Wahl, M Reduced dilator and constrictor response of the middle cerebral artery after cold lesion of the rat parietal cortex. ADVANCES IN EXPERIMENTAL MEDICINE AND BIOLOGY 471 pp. 83-89. , 7 p. (1999) DOI WoS Scopus PubMed Közlemény:1848206 Érvényesített Forrás Folyóiratcikk (Szakcikk )

197. Görlach, C ; Hortobágyi, T ; Benyó, Z ; Wahl, M Inhibition of iNOS reduces lesion volume only after less severe parietotemporal cold injury in rats. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 19 p. S382 (1999) Közlemény:1856206 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

198. Hortobágyi, T ; Görlach, C ; Benyó, Z ; Wahl, M Neuroprotection by iNOS inhibitor aminoguanidine in focal brain injury. FUNDAMENTAL & CLINICAL PHARMACOLOGY 31 : suppl 1 p. 298 (1999) Közlemény:1856215 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

199. Hortobágyi, T ; Görlach, C ; Benyó, Z ; Wahl, M In vitro characterization and in vivo action after brain trauma of a new endothelin-A receptor antagonist: RO 61-1790. FUNDAMENTAL & CLINICAL PHARMACOLOGY 31 : suppl 1 p. 167 (1999) Közlemény:1856213 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat ) Nyilvános idézők összesen: 4 Független: 3 Függő: 1

1. * Gorlach C et al. Inhibition of endothelin-1 by the competitive ET(A) receptor antagonist Ro 61-1790 reduces lesion volume after cold injury in the rat.. (2001) PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY 0031-6768 1432-2013 441 6 844-849 2. Sorbera L. Ambrisentan: Treatment of pulmonary arterial hypertension endothelin ETA receptor antagonist. (2005) DRUGS OF THE FUTURE 0377-8282 30 8 765-770 3. Uhlmann D. Clazosentan (Actelion). (2006) CURRENT OPINION IN INVESTIGATIONAL DRUGS 1472-4472 2040-3429 7 3 272-281 4. Wu C. Recent developments of small molecule endothelin modulators. (2006) EXPERT OPINION ON THERAPEUTIC PATENTS 1354-3776 16 10 1337-1345

200. Wahl, M ; Gorlach, C ; Hortobagyi, T ; Benyo, Z Eects of bradykinin in the cerebral circulation. ACTA PHYSIOLOGICA HUNGARICA 86 : 2 pp. 155-160. , 6 p. (1999) Scopus PubMed Közlemény:1518211 Érvényesített Forrás Folyóiratcikk (Összefoglaló cikk ) Nyilvános idézők összesen: 18 Független: 16 Függő: 2

1. * Benyó Z et al. Selective inhibition of neuronal nitric oxide synthase fails to alter the resting tension and the relaxant eect of bradykinin in isolated rat middle cerebral arteries. (1999) ACTA PHYSIOLOGICA HUNGARICA 0231-424X 1588-2683 2498-602X 86 2 161-165 2. * Hortobagyi T et al. A novel brain trauma model in the mouse: eects of dexamethasone treatment.. (2000) PFLUGERS ARCHIV- EUROPEAN JOURNAL OF PHYSIOLOGY 0031-6768 1432-2013 441 2-3 409-415 3. Yamazaki J et al. Cell-to-cell communication via nitric oxide modulation of oscillatory Cl- currents in rat intact cerebral arterioles. (2001) JOURNAL OF PHYSIOLOGY-LONDON 0022-3751 1469-7793 536 1 67-78 4. Lacza Z et al. Hydrogen peroxide acts as an EDHF in the piglet pial vasculature in response to bradykinin. (2002) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 283 1 H406-H411 5. Lomez ESL et al. Tonin and kallikrein in the brain of transgenic rat line expressing human tissue kallikrein. (2002) HYPERTENSION 0194-911X 39 2 229-232 6. Norman MU et al. Metalloendopeptidases EC 3.4.24.15/16 regulate bradykinin activity in the cerebral microvasculature. (2003) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 284 6 H1942- H1948 7. Wolka AM et al. Pain and the blood-brain barrier: Obstacles to drug delivery. (2003) ADVANCED DRUG DELIVERY REVIEWS 0169- 409X 1872-8294 55 8 987-1006 8. Shcherbakova IV. [Activation of innate immunity in schizophrenia]. (2006) ZHURNAL NEVROLOGII I PSIKHIATRII IMENI S S KORSAKOVA 1997-7298 2309-4729 106 10 79-82 9. Kis B et al. Autocrine peptide mediators of cerebral endothelial cells and their role in the regulation of blood-brain barrier. (2006) PEPTIDES 0196-9781 1873-5169 27 1 211-222 10. Horváth E et al. Graft derived cells with double nuclei in the penumbral region of experimental brain trauma. (2006) NEUROSCIENCE LETTERS 0304-3940 1872-7972 396 3 182-186 11. Kis B et al. Peptide mediators of the brain endothelium. (2007) Megjelent: Handbook of Neurochemistry and Molecular Neurobiology: Neural Membranes and Transport pp. 191-208 12. Watts SW et al. Receptor-Mediated Events in the Microcirculation. (2008) Megjelent: Microcirculation pp. 285-348 13. Zweckberger Klaus et al. Anatibant (R), a selective non-peptide bradykinin B-2 receptor antagonist, reduces intracranial hypertension and histopathological damage after experimental traumatic brain injury. (2009) NEUROSCIENCE LETTERS 0304-3940 1872-7972 454 2 115-117 14. Côté J et al. Selective tumor blood-brain barrier opening with the kinin B2 receptor agonist [Phe8ψ(CH2NH)Arg9]-BK in a F98 glioma rat model: An MRI study. (2010) NEUROPEPTIDES 0143- 4179 1532-2785 44 2 177-185 15. Noda M. Kallikrein-kinin system in the brain. (2011) Megjelent: Kinins pp. 85-102 16. Jor O et al. Mozková (mikro)cirkulace-základní struktura a funkce. (2014) Anesteziologie a Intenzivni Medicina 1214-2158 25 2 137- 139 17. Pal E et al. Vitamin D deciency causes inward hypertrophic remodeling and alters vascular reactivity of rat cerebral arterioles.. (2018) PLOS ONE 1932-6203 1932-6203 13 2 18. Sharma H.S. et al. Neuroprotective eects of a potent bradykinin B2 receptor antagonist HOE-140 on microvascular permeability, blood ow disturbances, edema formation, cell injury and nitric oxide synthase upregulation following trauma to the spinal cord. (2019) Megjelent: International Review of Neurobiology pp. 103- 152

201. Benyo, Z ; Szabo, C ; Csaki, C ; Wahl, M ; Kovach, AGB ; Sandor, P Eect of L-arginine on adrenal and renal blood ows during hemorrhage in cats KIDNEY INTERNATIONAL 54 : Suppl. 67 pp. S221-S223. (1998) DOI WoS Scopus PubMed Közlemény:2520346 Admin láttamozott Forrás Idéző Folyóiratcikk (Konferenciaközlemény ) Nyilvános idézők összesen: 3 Független: 2 Függő: 1 Idézett közlemények száma: 2

1. * Benyo Z et al. Prevention of the hemorrhagic hypotension- induced hepatic arterial vasoconstriction by L-arginine and naloxone. (1999) SHOCK 1073-2322 1540-0514 11 5 342-346 2. Baylis C. Acute and long-term eects of modied hemoglobin (HBOC-201) in a rat model of hypertension and chronic kidney disease. (2006) TRANSFUSION 0041-1132 1537-2995 46 7 1104- 1111 3. Loehe F et al. The role of L-arginine following trauma and blood loss. (2007) CURRENT OPINION IN CLINICAL NUTRITION AND METABOLIC CARE 1363-1950 10 1 80-87

202. Benyo, Z ; Gorlach, C ; Wahl, M Role of nitric oxide and thromboxane in the maintenance of cerebrovascular tone KIDNEY INTERNATIONAL SUPPLEMENT (1974-2011) 54 : Suppl. 67 pp. S218-S220. , 3 p. (1998) DOI WoS Scopus PubMed Közlemény:2520267 Érvényesített Forrás Idéző Folyóiratcikk (Konferenciaközlemény ) Nyilvános idézők összesen: 10 Független: 7 Függő: 3 Idézett közlemények száma: 1

1. * Benyo Z et al. Interaction between nitric oxide and thromboxane A2 in the regulation of the resting cerebrovascular tone.. (1999) ADVANCES IN EXPERIMENTAL MEDICINE AND BIOLOGY 0065- 2598 471 373-379 2. Hosokawa A. Role of ET(B) receptors and nitric oxide in adrenal catecholamine secretion in anesthetized dogs. (1999) AMERICAN JOURNAL OF PHYSIOLOGY: REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY 0363-6119 1522-1490 277 4 R1051- R1056 3. * Lacza Z et al. Involvement of prostanoid release in the mediation of UTP-induced cerebrovascular contraction in the rat. (2001) BRAIN RESEARCH 0006-8993 1872-6240 896 1-2 169-174 4. * Lacza Z et al. NO synthase blockade induces chaotic cerebral vasomotion via activation of thromboxane receptors.. (2001) STROKE 0039-2499 1524-4628 32 11 2609-2614 5. Matsuno H. Pharmacokinetic and pharmacodynamic properties of a new thromboxane receptor antagonist (Z-335) after single and multiple oral administrations to healthy volunteers. (2002) JOURNAL OF CLINICAL PHARMACOLOGY 0091-2700 1552-4604 42 7 782-790 6. Pster SL. Role of contaminating platelets in thromboxane synthesis in primary cultures of human umbilical vein endothelial cells. (2002) PROSTAGLANDINS 0090-6980 70 1-2 39-49 7. Miyamoto Atsushi. Ibuprofen or ozagrel increases NO release and L-nitro, arginine induces TXA(2) release from cultured porcine basilar arterial endothelial cells. (2007) VASCULAR PHARMACOLOGY 1537-1891 1879-3649 46 2 85-90 8. Gordon GRJ. Astrocyte control of blood ow. (2009) Megjelent: Astrocytes in (Patho)Physiology of the Nervous System pp. 461- 486 9. Craici IM. Advances in the pathophysiology of pre-eclampsia and related podocyte injury. (2014) KIDNEY INTERNATIONAL 0085- 2538 1523-1755 86 2 275-285 10. Tabatabaei Seyed et al. Nitric Oxide and Cerebrovascular Regulation. (2014) Megjelent: Nitric Oxide pp. 347-385

203. Benyo, Z ; Gorlach, C ; Wahl, M Involvement of thromboxane A(2) in the mediation of the contractile eect induced by inhibition of nitric oxide synthesis in isolated rat middle cerebral arteries JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 18 : 6 pp. 616-618. , 3 p. (1998) DOI WoS Scopus PubMed Közlemény:1628793 Érvényesített Forrás Folyóiratcikk (Rövid közlemény ) Nyilvános idézők összesen: 28 Független: 20 Függő: 8

1. * Benyo Z et al. Interaction between nitric oxide and thromboxane A2 in the regulation of the resting cerebrovascular tone.. (1999) ADVANCES IN EXPERIMENTAL MEDICINE AND BIOLOGY 0065- 2598 471 373-379 2. * Benyó Z et al. Selective inhibition of neuronal nitric oxide synthase fails to alter the resting tension and the relaxant eect of bradykinin in isolated rat middle cerebral arteries. (1999) ACTA PHYSIOLOGICA HUNGARICA 0231-424X 1588-2683 2498-602X 86 2 161-165 3. Wallis SJ et al. Conditions permitting suppression of stretch- induced and vasoconstrictor tone by basal nitric oxide activity in porcine cerebral artery. (2000) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 130 3 567-574 4. Lowe DT. Nitric oxide dysfunction in the pathophysiology of preeclampsia. (2000) NITRIC OXIDE-BIOLOGY AND CHEMISTRY 1089-8603 1089-8611 4 4 441-458 5. Page NM. Emerging molecular targets for the treatment of pre- eclampsia. (2001) EXPERT OPINION ON THERAPEUTIC TARGETS 1472-8222 1744-7631 5 3 395-413 6. * Lacza Z. Involvement of prostanoid release in the mediation of UTP-induced cerebrovascular contraction in the rat. (2001) BRAIN RESEARCH 0006-8993 1872-6240 896 1-2 169-174 7. * Lacza Z et al. NO synthase blockade induces chaotic cerebral vasomotion via activation of thromboxane receptors.. (2001) STROKE 0039-2499 1524-4628 32 11 2609-2614 8. Loichot U. Shear stress modulates vasopressin-induced renal vasoconstriction in rats. (2002) NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 0028-1298 1432-1912 366 6 555- 561 9. Page NM. The endocrinology of pre-eclampsia. (2002) CLINICAL ENDOCRINOLOGY 0300-0664 1365-2265 57 4 413-423 10. Cheikh RB. Thromboxane A2 in vasomotor eects of phenylephrine, acetylcholine, and bradykinin in rat mesenteric bed. (2002) JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 0160-2446 1533-4023 40 2 255-264 11. Page NM. Therapeutic patents for the treatment of pre- eclampsia. (2004) EXPERT OPINION ON THERAPEUTIC PATENTS 1354-3776 14 11 1579-1591 12. Gonzales RJ et al. Testosterone treatment increases thromboxane function in rat cerebral arteries. (2005) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 289 2 H578-H585 13. * Hortobagyi L et al. Adaptation of the hypothalamic blood ow to chronic nitric oxide deciency is independent of vasodilator prostanoids. (2007) BRAIN RESEARCH 0006-8993 1872-6240 1131 1 129-137 14. Michel FS. Increased spontaneous tone in renal arteries of spontaneously hypertensive rats. (2007) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 293 3 H1673-H1681 15. McNeish AJ. Thromboxane A(2) inhibition of SKCa after NO synthase block in rat middle cerebral artery. (2007) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 151 4 441- 449 16. Hernanz R. Ouabain treatment increases nitric oxide bioavailability and decreases superoxide anion production in cerebral vessels. (2008) JOURNAL OF HYPERTENSION 0263-6352 1473-5598 26 10 1944-1954 17. * Lacza Z et al. Additive eect of cyclooxygenase and nitric oxide synthase blockade on the cerebrocortical microcirculation. (2009) NEUROREPORT 0959-4965 20 11 1027-1031 18. Gordon GRJ et al. Astrocyte control of blood ow. (2009) Megjelent: Astrocytes in (Patho)Physiology of the Nervous System pp. 461-486 19. * Horvath B et al. Hypersensitivity to thromboxane receptor mediated cerebral vasomotion and CBF oscillations during acute NO-deciency in rats.. (2010) PLOS ONE 1932-6203 1932-6203 5 12 20. McNeish Alister. Statins and Selective Inhibition of Rho Kinase Protect Small Conductance Calcium-Activated Potassium Channel Function (K(Ca)2.3) in Cerebral Arteries. (2012) PLOS ONE 1932- 6203 1932-6203 7 10 21. Vuletic Vladimira et al. Eect of indomethacin on cerebrovascular reactivity in patients with type 2 diabetes mellitus. (2013) DIABETES RESEARCH AND CLINICAL PRACTICE 0168-8227 1872- 8227 101 1 81-87 22. Csébi Péter et al. Hogyan reagál az autológ rectushüvelyből képzett érgraft vazoaktív anyagok hatására? [How does the autologous internal rectus fascia sheath vascular graft react upon dierent vasoactive substances? Experimental myographic examination in a dog model]. (2014) MAGYAR ÁLLATORVOSOK LAPJA 0025-004X 136 12 743-749 23. Tabatabaei Seyed et al. Nitric Oxide and Cerebrovascular Regulation. (2014) Megjelent: Nitric Oxide pp. 347-385 24. Dorniak-Wall T. The role of L-arginine in the prevention and treatment of pre-eclampsia: a systematic review of randomised trials. (2014) JOURNAL OF HUMAN HYPERTENSION 0950-9240 28 4 230-235 25. Gordon G.R.J. et al. Bidirectional control of blood ow by astrocytes: A role for tissue oxygen and other metabolic factors. (2016) Megjelent: Advances in Experimental Medicine and Biology pp. 209-219 26. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801 27. Addison M et al. NO synthase inhibition attenuates EDHF- mediated relaxation induced by TRPV4 channel agonist GSK1016790A in the rat pulmonary artery: Role of TxA(2). (2016) PHARMACOLOGICAL REPORTS 1734-1140 68 3 620-626 28. Grafka A. et al. Study of the role of L-arginine in the diagnosis of pregnancy-induced hypertension. (2016) NADCIŚNIENIE TĘTNICZE / ARTERIAL HYPERTENSION 1428-5851 1644-3101 20 3 113-118

204. Benyo, Z ; Wahl, M Kappa opiate receptor-mediated constriction of the rat middle cerebral artery NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 358 : Suppl. 1 Paper: P3717 (1998) WoS Közlemény:1628792 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

205. Benyo, Z ; Gorlach, C ; Wahl, M Involvement of prostanoid release in the mediation of the contractile eect of NG-nitro-L-arginine in rat middle cerebral arteries NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 358 : Suppl. 1 Paper: P3716 (1998) WoS Közlemény:1628791 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

206. Gorlach, C ; Benyo, Z ; Wahl, M Dilator eect of bradykinin and acetylcholine in cerebral vessels after brain lesion KIDNEY INTERNATIONAL SUPPLEMENT (1974-2011) 54 : Suppl. 67. pp. S226-S227. , 2 p. (1998) DOI WoS Scopus PubMed Közlemény:1823845 Érvényesített Forrás Folyóiratcikk (Konferenciaközlemény ) Nyilvános idézők összesen: 3 Független: 3 Függő: 0

1. Parfenova H et al. Developmental changes in endothelium- derived vasorelaxant factors in cerebral circulation. (2000) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 278 3 H780-H788 2. Willis AP. Endothelial NO and prostanoid involvement in newborn and juvenile pig pial arteriolar vasomotor responses. (2001) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 281 6 H2366-H2377 3. Sharma H.S. et al. Neuroprotective eects of a potent bradykinin B2 receptor antagonist HOE-140 on microvascular permeability, blood ow disturbances, edema formation, cell injury and nitric oxide synthase upregulation following trauma to the spinal cord. (2019) Megjelent: International Review of Neurobiology pp. 103- 152

207. Gorlach, C ; Benyo, Z ; Wahl, M Endothelin-1-induced contraction in cerebral vessels mediated by phospholipase C/protein kinase C cascade. KIDNEY INTERNATIONAL SUPPLEMENT (1974-2011) 54 : Suppl. 67. pp. S224-S225. , 2 p. (1998) DOI WoS Scopus PubMed Közlemény:1816856 Érvényesített Forrás Folyóiratcikk (Konferenciaközlemény ) Nyilvános idézők összesen: 22 Független: 18 Függő: 4

1. * Gorlach C. Delayed loss of ETB receptor-mediated vasorelaxation after cold lesion of the rat parietal cortex. (1998) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559- 7016 18 12 1357-1364 2. * Gorlach C et al. Reduced reactivity of the middle cerebral artery and its large branches after cold lesion. (1998) JOURNAL OF NEUROTRAUMA 0897-7151 15 12 1067-1075 3. Gomazkov OA. [The system of the endothelin peptides: Mechanisms of the cardio-vascular pathologies]. (1999) VOPROSY MEDITSINSKOI KHIMII 0042-8809 2310-6905 45 4 301-303 4. Bagrov AY. Cicletanine reverses vasoconstriction induced by the endogenous sodium pump ligand, marinobufagenin, via a protein kinase C dependent mechanism. (2000) JOURNAL OF HYPERTENSION 0263-6352 1473-5598 18 2 209-215 5. Ungvari Z et al. Endothelin and nitric oxide modulate arteriolar smooth muscle Ca2+ sensitivity and myogenic tone in hypertension. (2000) Megjelent: 21ST EUROPEAN CONFERENCE ON MICROCIRCULATION pp. 105-110 6. Ungvari Z et al. Endothelin and Prostaglandin Hˇˇ2ˇˇˇ/Thromboxane A2 Enhance Myogenic Constriction in Hypertension by Increasing Ca^^2+^^^ Sensitivity of Arteriolar Smooth Muscle. (2000) HYPERTENSION 0194-911X 36 5 856-861 7. Zubkov AY. Mechanism of endothelin-1-induced contraction in rabbit basilar artery. (2000) STROKE 0039-2499 1524-4628 31 2 526-533 8. Gomazkov OA. Endothelin in Cardiology: Molecular, Physiological, and Pathological Aspects. (2001) KARDIOLOGIYA 0022-9040 41 2 50-58 9. Thorin E. Inuence of nitric oxide synthase inhibition and endothelin-1 receptor blockade on acetylcholine-induced coronary artery contraction in vitro in dilated and ischemic cardiomyopathies. (2001) JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 0160-2446 1533-4023 38 1 90-98 10. * Gorlach C. Inhibition of endothelin-1 by the competitive ET(A) receptor antagonist Ro 61-1790 reduces lesion volume after cold injury in the rat.. (2001) PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY 0031-6768 1432-2013 441 6 844-849 11. Koller A. Signaling pathways of mechanotransduction in arteriolar endothelium and smooth muscle cells in hypertension. (2002) MICROCIRCULATION 1073-9688 1549-8719 9 4 277-294 12. Kleeberg J. ET-1 induces cortical spreading depression via activation of the ET A receptor/phospholipase C pathway in vivo. (2004) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 286 4 H1339- H1346 13. Ko EA. Extracellular Mg2+ blocks endothelin-1-induced contraction through the inhibition of non-selective cation channels in coronary smooth muscle. (2004) PFLUGERS ARCHIV- EUROPEAN JOURNAL OF PHYSIOLOGY 0031-6768 1432-2013 449 2 195-204 14. Schwindt W. Functional magnetic resonance imaging and somatosensory evoked potentials in rats with a neonatally induced freeze lesion of the somatosensory cortex. (2004) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271- 678X 1559-7016 24 12 1409-1418 15. Van Woerkom. Inuence of morphine and naloxone on endothelin and its receptors in newborn piglet brain vascular endothelial cells: Clinical Implications in Neonatal Care. (2004) PEDIATRIC RESEARCH 0031-3998 1530-0447 55 1 147-151 16. Erdös B et al. Vasoconstrictor mechanisms in the cerebral circulation are unaected by insulin resistance. (2004) AMERICAN JOURNAL OF PHYSIOLOGY: REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY 0363-6119 1522-1490 287 6 R1456- R1461 17. Tong XK et al. Vascular remodeling versus amyloid beta-induced oxidative stress in the cerebrovascular dysfunctions associated with Alzheimer's disease. (2005) JOURNAL OF NEUROSCIENCE 0270-6474 1529-2401 25 48 11165-11174 18. Yu J. Propofol inhibits phorbol 12, 13-dibutyrate-induced, protein kinase C-mediated contraction of rat aortic smooth muscle. (2006) ACTA ANAESTHESIOLOGICA SCANDINAVICA 0001-5172 1399-6576 50 9 1131-1138 19. Bouallegue A. Endothelin-1-induced signaling pathways in vascular smooth muscle cells. (2007) CURRENT VASCULAR PHARMACOLOGY 1570-1611 5 1 45-52 20. Bouallegue A. Involvement of insulin-like growth factor 1 receptor transactivation in endothelin-1-induced signaling in vascular smooth muscle cells. (2010) CANADIAN JOURNAL OF PHYSIOLOGY AND PHARMACOLOGY 0008-4212 1205-7541 88 5 501-509 21. Little PJ. The paradigm of G protein receptor transactivation: A mechanistic denition and novel example. (2011) SCIENTIFIC WORLD JOURNAL 1537-744X 2356-6140 11 709-714 22. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801

208. Gorlach, C ; Benyo, Z ; Schilling, L ; Wahl, M Reduced vascular reactivity of middle cerebral artery after cold lesion NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY 358 : Suppl. 1 Paper: P3715 (1998) WoS Közlemény:1628784 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat ) Nyilvános idézők összesen: 1 Független: 0 Függő: 1

1. * Gorlach C et al. Reduced reactivity of the middle cerebral artery and its large branches after cold lesion. (1998) JOURNAL OF NEUROTRAUMA 0897-7151 15 12 1067-1075

209. Gorlach, C ; Benyo, Z ; Wahl, M Reduced reactivity of the middle cerebral artery and its large branches after cold lesion JOURNAL OF NEUROTRAUMA 15 : 12 pp. 1067-1075. , 9 p. (1998) DOI WoS Scopus PubMed Közlemény:1628783 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 8 Független: 3 Függő: 5 Idézett közlemények száma: 2

1. * Görlach C. Loss of ETB-receptor-mediated relaxation in basilar artery after cold lesion of the rat parietal cortex. (1999) Megjelent: OXYGEN TRANSPORT TO TISSUE XXI pp. 91-97 2. * Gorlach C. Reduced dilator and constrictor response of the middle cerebral artery after cold lesion of the rat parietal cortex. (1999) Megjelent: OXYGEN TRANSPORT TO TISSUE XXI pp. 83-89 3. * Gorlach C. Aminoguanidine reduces brain lesion volume after cold injury in the rat. (2000) PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY 0031-6768 1432-2013 440 2 309-314 4. * Gorlach C. Neuronal nitric oxide synthase inhibitor has a neuroprotective eect in a rat model of brain injury. (2000) RESTORATIVE NEUROLOGY AND NEUROSCIENCE 0922-6028 17 2- 3 71-76 5. Barth M. Time course of apoptotic cell death after experimental neurotrauma. (2000) Megjelent: BRAIN EDEMA XI pp. 121-124 6. Klusek J. Changes in some lipid concentrations in mice under high and low temperatures. (2001) ACTA BIOLOGICA CRACOVIENSIA SERIES ZOOLOGIA 0001-530X 43 45-48 7. * Gorlach C. Inhibition of endothelin-1 by the competitive ET(A) receptor antagonist Ro 61-1790 reduces lesion volume after cold injury in the rat.. (2001) PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY 0031-6768 1432-2013 441 6 844-849 8. Trabold R. Arterial hypotension triggers perifocal depolarizations and aggravates secondary damage in focal brain injury. (2006) BRAIN RESEARCH 0006-8993 1872-6240 1071 1 237-244

210. Benyó, Z ; Wahl, M Potent contractile eect of dynorphin A 1-13 in isolated rat cerebral arteries. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 17 p. 346 (1997) Közlemény:1856159 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat ) 211. Benyó, Z ; Görlach, C ; Wahl, M Neuronal nitric oxide synthase is involved in agonist-induced but not in basal nitric oxide release in isolated rat basilar arteries. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 17 : Suppl. 1 p. S345 (1997) Közlemény:1856144 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat ) Nyilvános idézők összesen: 4 Független: 1 Függő: 3

1. Rosenblum W. A review of vasomotor responses of arterioles on the surface of the mouse brain: The necessary prelude to studies using genetically manipulated mice. (1998) MICROCIRCULATION 1073-9688 1549-8719 5 2-3 129-138 2. * Benyo Z et al. Role of nitric oxide and thromboxane in the maintenance of cerebrovascular tone. (1998) KIDNEY INTERNATIONAL SUPPLEMENT (1974-2011) 0098-6577 54 Suppl. 67 S218-S220 3. * Benyo Z et al. Interaction between nitric oxide and thromboxane A2 in the regulation of the resting cerebrovascular tone.. (1999) ADVANCES IN EXPERIMENTAL MEDICINE AND BIOLOGY 0065- 2598 471 373-379 4. * Benyó Z et al. Selective inhibition of neuronal nitric oxide synthase fails to alter the resting tension and the relaxant eect of bradykinin in isolated rat middle cerebral arteries. (1999) ACTA PHYSIOLOGICA HUNGARICA 0231-424X 1588-2683 2498-602X 86 2 161-165

212. Benyo, Z ; Szabo, C ; Velkei, MH ; Bohus, B ; Wahl, M ; Sandor, P Intravenous beta-endorphin administration fails to alter hypothalamic blood ow in rats expressing normal or reduced nitric oxide synthase activity PEPTIDES 17 : 4 pp. 733-736. , 4 p. (1996) DOI WoS Scopus PubMed Közlemény:1628795 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 5 Független: 4 Függő: 1 Idézett közlemények száma: 5

1. * Benyo Z et al. Opiate receptor-mediated mechanisms in the regulation of cerebral blood ow. (1996) CEREBROVASCULAR AND BRAIN METABOLISM REVIEWS 1040-8827 8 4 326-357 2. Olson GA. Endogenous opiates: 1996. (1997) PEPTIDES 0196-9781 1873-5169 18 10 1651-1688 3. Kampa M. Opioids are non-competitive inhibitors of nitric oxide synthase in T47D human breast cancer cells. (2001) CELL DEATH AND DIFFERENTIATION 1350-9047 1476-5403 8 9 943-952 4. Uetsuki N. The role of CRF1 receptors for sympathetic nervous response to laparotomy in anesthetized rats. (2005) BRAIN RESEARCH 0006-8993 1872-6240 1044 1 107-115 5. Shariftabrizi A. Matrix metalloproteinase 2 secretion in WEHI 164 brosarcoma cells is nitric oxide-related and modied by morphine. (2006) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 530 1-2 33-39

213. Benyo, Z ; Wahl, M Opiate receptor-mediated mechanisms in the regulation of cerebral blood ow CEREBROVASCULAR AND BRAIN METABOLISM REVIEWS 8 : 4 pp. 326-357. , 32 p. (1996) WoS Scopus PubMed Közlemény:1628794 Érvényesített Forrás Idéző Folyóiratcikk (Összefoglaló cikk ) Nyilvános idézők összesen: 37 Független: 33 Függő: 4 Idézett közlemények száma: 14

1. Hauerberg J et al. Eects of morphine and naloxone on cerebral blood ow and metabolism in experimental subarachnoid hemorrhage. (1997) ACTA NEUROLOGICA SCANDINAVICA 0001- 6314 1600-0404 96 3 187-193 2. Olson GA. Endogenous opiates: 1996. (1997) PEPTIDES 0196-9781 1873-5169 18 10 1651-1688 3. Danos P. Pathological regional blood ow in opiate-dependent patients during withdrawal: A HMPAO-SPECT study. (1998) NEUROPSYCHOBIOLOGY 0302-282X 37 4 194-199 4. Lever JR et al. Selective in-Vivo Binding of (H-3)Naltriben to Delta- Opioid Receptors in Mouse-Brain.. (1998) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 350 335-344 5. Low PA. Cerebrovascular regulation in the postural orthostatic tachycardia syndrome (POTS). (1999) AMERICAN JOURNAL OF THE MEDICAL SCIENCES 0002-9629 1538-2990 317 2 124-133 6. Ma XD et al. Eects of morphine on cerebral blood ow autoregulation and CO2-reactivity in experimental subarachnoid hemorrhage. (1999) JOURNAL OF NEUROSURGICAL ANESTHESIOLOGY 0898-4921 11 4 264-272 7. Harrison JM. Eects of target-controlled infusion of propofol on the transient hyperaemic response and carbon dioxide reactivity in the middle cerebral artery. (1999) BRITISH JOURNAL OF ANAESTHESIA 0007-0912 1471-6771 83 6 839-844 8. Hock C. Heroin-induced deoxygenation of cerebral hemoglobin: Monitoring by means of near-infrared spectroscopy. (1999) ANNALS OF NEUROLOGY 0364-5134 1531-8249 45 1 134-135 9. Sandor P. Nervous control of the cerebrovascular system: doubts and facts. (1999) NEUROCHEMISTRY INTERNATIONAL 0197-0186 1872-9754 35 3 237-259 10. Wagner K. Regional cerebral blood ow in humans during remifentanil infusion. (1999) ANESTHESIA AND ANALGESIA 0003- 2999 1526-7598 88 2 Suppl. 11. Lorenz IH. Low-dose remifentanil increases regional cerebral blood ow and regional cerebral blood volume, but decreases regional mean transit time and regional cerebrovascular resistance in volunteers. (2000) BRITISH JOURNAL OF ANAESTHESIA 0007-0912 1471-6771 85 2 199-204 12. Zamani R. Systemic naloxone enhances cerebral blood ow in anesthetized morphine-dependent rats. (2000) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 408 3 299- 304 13. * Sandor P et al. Hemorrhage-induced brain ischemia: Modulatory role of endogenous opioids and antiopioids in the cerebrovascular bed. (2001) Megjelent: ISCHEMIC BLOOD FLOW IN THE BRAIN pp. 178-194 14. Komjati K et al. Interactions between the endothelium-derived relaxing factor/nitric oxide system and the endogenous opiate system in the modulation of cerebral and spinal vascular CO2 responsiveness. (2001) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 21 8 937-944 15. Harrison JM. Eects of propofol and nitrous oxide on middle cerebral artery ow velocity and cerebral autoregulation. (2002) ANAESTHESIA 0003-2409 57 1 27-32 16. Lorenz IH. The inuence of nitrous oxide and remifentanil on cerebral hemodynamics in conscious human volunteers. (2002) NEUROIMAGE 1053-8119 1095-9572 17 2 1056-1064 17. Stohler R. Opioids and neurological sequelae. (2003) Megjelent: BASIC AND CLINICAL SCIENCE OF OPIOID ADDICTION pp. 55-58 18. Hancock SM. Nitrous oxide or remifentanil for the 'at risk' brain. (2004) ANAESTHESIA 0003-2409 59 4 313-315 19. Lyoo IK. White matter hyperintensities in subjects with cocaine and opiate dependence and healthy comparison subjects. (2004) PSYCHIATRY RESEARCH-NEUROIMAGING 0925-4927 131 2 135- 145 20. Buttner A. Alterations of the vascular basal lamina in the cerebral cortex in drug abuse: a combined morphometric and immunohistochemical investigation. (2005) DRUG AND ALCOHOL DEPENDENCE 0376-8716 79 1 63-70 21. Borne J. Neuroimaging in drug and substance abuse part II: Opioids and solvents. (2005) TOPICS IN MAGNETIC RESONANCE IMAGING 0899-3459 16 3 239-245 22. Collins LR. Prolonged morphine exposure in utero causes fetal and placental vasoconstriction: A case report. (2005) JOURNAL OF MATERNAL-FETAL & NEONATAL MEDICINE 1476-7058 1476-4954 17 6 417-421 23. Beydon L. Traitement de la douleur des hémorragies méningées graves [Pain management in severe subarachnoid haemorrhage]. (2005) ANNALES FRANCAISES D ANESTHESIE ET DE REANIMATION 0750-7658 24 7 782-786 24. Eskandary H. The eects of morphine and Cerebral Blood Flow and its neuroprotective or cell damage before and after brain ischemia reperfusion in rabbits. (2006) INTERNATIONAL JOURNAL OF PHARMACOLOGY 1811-7775 1812-5700 2 6 613-617 25. Pattinson KTS. Pharmacological FMRI: measuring opioid eects on the BOLD response to hypercapnia. (2007) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559- 7016 27 2 414-423 26. Hagan IG. Radiology of recreational drug abuse. (2007) RADIOGRAPHICS 0271-5333 1527-1323 27 4 919-940 27. Serban DN. Vascular eects and mechanisms of action for certain enkephalin-related peptides. (2007) Megjelent: JOINT MEETING OF THE SLOVAK PHYSIOLOGICAL SOCIETY, THE PHYSIOLOGICAL SOCIETY AND THE FEDERATION OF EUROPEAN PHYSIOLOGICAL SOCIE... pp. 59-62 28. Lee M. Beta-Endorphin. (2010) Megjelent: Encyclopedia of Stress pp. 332-335 29. Przewlocki R. Stress, opioid peptides, and their receptors. (2010) Megjelent: Hormones, Brain and Behavior Online pp. 289-332 30. Veenith T. The cerebral circulation. (2011) Megjelent: Core Topics in Neuroanaesthesia and Neurointensive Care pp. 17-32 31. * Ruisanchez E et al. Perivascular expression and potent vasoconstrictor eect of dynorphin a in cerebral arteries.. (2012) PLOS ONE 1932-6203 1932-6203 7 5 32. * Iring A et al. Role of endocannabinoids and cannabinoid-1 receptors in cerebrocortical blood ow regulation. (2013) PLOS ONE 1932-6203 1932-6203 8 1 33. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801 34. Przewlocki R et al. Stress and Opioid Systems. (2016) Megjelent: Hormones, Brain and Behavior: Third Edition pp. 225-260 35. Someya Eriko. Stimulation of mu-opioid receptors dilates retinal arterioles by neuronal nitric oxide synthase-derived nitric oxide in rats. (2017) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 803 124-129 36. Solis Ernesto et al. Opposing mechanisms underlying dierential changes in brain oxygen an temperature induced by intravenous morphine. (2018) JOURNAL OF NEUROPHYSIOLOGY 0022-3077 1522-1598 120 5 2513-2521 37. Vlah V. Role of central versus peripheral opioid system in antinociceptive and anti-inammatory eect of botulinum toxin type A in trigeminal region. (2018) EUROPEAN JOURNAL OF PAIN 1090-3801 1532-2149 22 3 583-591 214. Benyó, Z ; Önnerfált, J ; Tjernström, F ; Sándor, P Eect of L-arginine on hypothalamic blood ow in rats expressing normal or reduced nitric oxide synthase activity JOURNAL OF VASCULAR RESEARCH 33 : Suppl. 2 p. 22 (1996) Közlemény:1856133 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

215. Benyó, Z ; Urbanics, R ; Nyakas, C ; Sándor, P ; Luiten, PGM In situ blotting of NADPH diaphorase in the rodent brain: a novel method for the detection of ischemic damage. CEREBROVASCULAR DISEASES 6 : suppl 2 pp. 135-136. , 2 p. (1996) Közlemény:1856124 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

216. BENYO, Z ; DEJONG, GI ; LUITEN, PGM Nimodipine prevents early loss of hippocampal CA1 parvalbumin immunoreactivity after focal cerebral ischemia in the rat BRAIN RESEARCH BULLETIN 36 : 6 pp. 569-572. , 4 p. (1995) DOI WoS Scopus PubMed Közlemény:1628805 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 13 Független: 11 Függő: 2 Idézett közlemények száma: 2

1. Winsky L. Antibody recognition of calcium-binding proteins depends on their calcium-binding status. (1996) JOURNAL OF NEUROCHEMISTRY 0022-3042 1471-4159 66 2 764-771 2. States BA. DNA fragmentation and HSP70 protein induction in hippocampus and cortex occurs in separate neurons following permanent middle cerebral artery occlusions. (1996) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559- 7016 16 6 1165-1175 3. Pauls TL. The Ca2+-binding proteins parvalbumin and oncomodulin and their genes: New structural and functional ndings. (1996) BIOCHIMICA ET BIOPHYSICA ACTA-GENE STRUCTURE AND EXPRESSION 0167-4781 1874-9399 1306 1 39-54 4. Honkaniemi J. Expression of zinc nger immediate early genes in rat brain after permanent middle cerebral artery occlusion. (1997) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 17 6 636-646 5. Scotti AL et al. Loss of Perikaryal Parvalbumin Immunoreactivity from Surviving GABAergic Neurons in the Ca1 Field of Epileptic Gerbils.. (1997) HIPPOCAMPUS 1050-9631 1098-1063 7 5 524-535 6. Kwon YB. Eects of dizocilpine pretreatment on parvalbumin immunoreactivity and Fos expression after cerebral ischemia in the hippocampus of the mongolian gerbil. (2000) JOURNAL OF VETERINARY MEDICAL SCIENCE 0916-7250 1347-7439 62 2 141- 146 7. * Harkany T et al. Increased amyloid precursor protein expression and serotonergic sprouting following excitotoxic lesion of the rat magnocellular nucleus basalis: neuroprotection by Ca²⁺ antagonist nimodipine. (2000) NEUROSCIENCE 0306-4522 1873- 7544 101 101-114 8. Taya K. Nimodipine improves the disruption of spatial cognition induced by cerebral ischemia. (2000) PHYSIOLOGY & BEHAVIOR 0031-9384 70 1-2 19-25 9. Wang JH. Short-term cerebral ischemia causes the dysfunction of interneurons and more excitation of pyramidal neurons in rats. (2003) BRAIN RESEARCH BULLETIN 0361-9230 1873-2747 60 1-2 53-58 10. Frahm C. GABA neurons survive focal ischemic injury. (2004) NEUROSCIENCE 0306-4522 1873-7544 127 2 341-346 11. * Berends A et al. A review of the neuroprotective properties of the 5-HT1A receptor agonist repinotan HCl (BAY x 3702) in ischemic stroke. (2005) CNS DRUG REVIEWS 1080-563X 11 4 379-402 12. Holmberg P. Secondary Brain Injuries in Thalamus and Hippocampus after Focal Ischemia Caused by Mild, Transient Extradural Compression of the Somatosensori Cortex in the Rat. (2009) CURRENT NEUROVASCULAR RESEARCH 1567-2026 6 1 1-11 13. Muller HD. Pravastatin treatment causes a shift in the balance of hippocampal neurotransmitter binding densities towards inhibition. (2010) BRAIN RESEARCH 0006-8993 1872-6240 1316 17-26

217. BENYO, Z ; SZABO, C ; SANDOR, P ; KOVACH, AGB INFLUENCE OF ENDOGENOUS OPIATES ON HEPATIC ARTERIAL BLOOD-FLOW IN NORMOTENSIVE STATE AND DURING HEMORRHAGIC HYPOTENSION AND RETRANSFUSION FASEB JOURNAL 9 : 4 pp. A904-A904. (1995) WoS Közlemény:1628804 Érvényesített Forrás Folyóiratcikk (Absztrakt / Kivonat )

218. BENYO, Z ; SZABO, C ; STUIVER, BT ; BOHUS, B ; SANDOR, P Hypothalamic blood ow remains unaltered following chronic nitric oxide synthase blockade in rats NEUROSCIENCE LETTERS 198 : 2 pp. 127-130. , 4 p. (1995) DOI WoS Scopus PubMed Közlemény:1628796 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 14 Független: 7 Függő: 7 Idézett közlemények száma: 4

1. * Benyo Z et al. Intravenous beta-endorphin administration fails to alter hypothalamic blood ow in rats expressing normal or reduced nitric oxide synthase activity. (1996) PEPTIDES 0196-9781 1873-5169 17 4 733-736 2. * Szabó C. Physiological and pathophysiological roles of nitric oxide in the central nervous system. (1996) BRAIN RESEARCH BULLETIN 0361-9230 1873-2747 41 131-141 3. Ryan KL. Circulatory failure induced by 35 GHz microwave heating: Eects of chronic nitric oxide synthesis inhibition. (1997) SHOCK 1073-2322 1540-0514 7 1 70-76 4. Vincent VAM. Production, regulation and role of nitric oxide in glial cells. (1998) MEDIATORS OF INFLAMMATION 0962-9351 1466-1861 7 4 239-255 5. Vag J et al. Role of nitric oxide in the regulation of blood ow in the rat submandibular gland during carotid artery occlusion. (2001) ARCHIVES OF ORAL BIOLOGY 0003-9969 46 3 261-267 6. * Horvath B et al. Contribution of the heme oxygenase pathway to the maintenance of the hypothalamic blood ow during diminished nitric oxide synthesis. (2003) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 23 6 653- 657 7. * Szelke E et al. Eects of estrogen and progestin on hypothalamic blood ow autoregulation. (2005) JOURNAL OF THE SOCIETY FOR GYNECOLOGIC INVESTIGATION 1071-5576 1556-7117 1933-7191 12 8 604-609 8. Raai G et al. Experimental orthostasis elicits sustained hypertension, which can be prevented by sympathetic blockade in the rat. (2005) JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 0160-2446 1533-4023 45 4 354-361 9. Raai G et al. Inverse-orthostasis may induce elevation of blood pressure due to sympathetic activation. (2006) JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 0160-2446 1533-4023 47 2 287-294 10. * Hortobagyi L et al. Adaptation of the hypothalamic blood ow to chronic nitric oxide deciency is independent of vasodilator prostanoids. (2007) BRAIN RESEARCH 0006-8993 1872-6240 1131 1 129-137 11. * Horvath B et al. Carbon monoxide-prostaglandin E-2 interaction in the hypothalamic circulation. (2008) NEUROREPORT 0959-4965 19 16 1601-1604 12. Fujita Y et al. Early Protective Eect of Bone Marrow Mononuclear Cells Against Ischemic White Matter Damage Through Augmentation of Cerebral Blood Flow. (2010) STROKE 0039-2499 1524-4628 41 12 2938-2943 13. * Horvath B et al. Hypersensitivity to thromboxane receptor mediated cerebral vasomotion and CBF oscillations during acute NO-deciency in rats.. (2010) PLOS ONE 1932-6203 1932-6203 5 12 14. Das UN. Metabolic Syndrome Pathophysiology: The Role of Essential Fatty Acids. (2010) ISBN:9780813815534

219. Benyó, Z ; Velkei, MH ; Sándor, P Eect of intravenous ß-endorphin administration on the hypothalamic blood ow in rats. SOCIETY FOR NEUROSCIENCE ABSTRACTS 21 p. 156 (1995) Közlemény:1852754 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

220. Benyó, Z ; Donkó, I ; Székely, E ; Sándor, P Eect of chronic nitric oxide blockade on systemic arterial pressure and on the acetylcholine-induced hypotensive response in rats. EUROPEAN JOURNAL OF CLINICAL INVESTIGATION 25 p. A3 (1995) Közlemény:1852744 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

221. Benyó, Z ; Velkei, M ; Sándor, P L-arginine reverses hypothalamic vasoconstriction induced by chronic nitric oxide synthase inhibition in rats. SOCIETY FOR NEUROSCIENCE ABSTRACTS 20 p. 998 (1994) Közlemény:1852740 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

222. SZABO, C ; CSAKI, C ; BENYO, Z ; MARCZIS, J ; REIVICH, M ; KOVACH, AGB Eect of superoxide dismutase on hemorrhagic hypotension and retransfusion-evoked middle cerebral artery endothelial dysfunction CIRCULATORY SHOCK 44 : 3 pp. 104-110. , 7 p. (1994) PubMed WoS Scopus Közlemény:1628797 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 9 Független: 7 Függő: 2 Idézett közlemények száma: 5

1. Busija DW. Induction of Cyclooxygenase-2 Following Anoxic Stress in Piglet Cerebral Arteries. (1996) MICROCIRCULATION 1073-9688 1549-8719 3 4 379-386 2. Dewitt DS. L-arginine and superoxide dismutase prevent or reverse cerebral hypoperfusion after uid-percussion traumatic brain injury. (1997) JOURNAL OF NEUROTRAUMA 0897-7151 14 4 223-233 3. * Kilbourn RG. Nitric oxide and shock. (1997) DM DISEASE-A- MONTH 0011-5029 1557-8194 43 5 279-348 4. Flynn WJ et al. Xanthine oxidase inhibition prevents mesenteric blood ow decits after resuscitated hemorrhagic shock by preserving endothelial function. (1997) JOURNAL OF SURGICAL RESEARCH 0022-4804 1095-8673 68 2 175-180 5. * Szabó C. Molecular Mechanisms of the Nitric Oxide Induced Vessel Wall Dysfunction in Sepsis. (1998) SEPSIS (WARSZAWA) 1898-6307 1899-2013 1 2 107-114 6. Hartig W et al. Functional recovery of cholinergic basal forebrain neurons under disease conditions: Old problems, new solutions?. (2002) REVIEWS IN THE NEUROSCIENCES 0334-1763 13 2 95-165 7. Zakaria ER. Role of neutrophils on shock/resuscitation-mediated intestinal arteriolar derangements. (2004) SHOCK 1073-2322 1540-0514 21 3 248-253 8. Prough DS. Eects of hypertonic arginine on cerebral blood ow and intracranial pressure after traumatic brain injury combined with hemorrhagic hypotension. (2006) SHOCK 1073-2322 1540- 0514 26 3 290-295 9. Chuaiphichai Surawee. Endothelial cell tetrahydrobiopterin deciency attenuates LPS-induced vascular dysfunction and hypotension. (2016) VASCULAR PHARMACOLOGY 1537-1891 1879-3649 77 69-79

223. Benyó, Z ; Velkei, MH ; Sándor, P Eect of chronic nitric oxide synthase inhibition on hypothalamic blood ow in rat. SOCIETY FOR NEUROSCIENCE ABSTRACTS 19 p. 1660 (1993) Közlemény:1852737 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

224. Benyó, Z ; Okabe, M ; Lenkei, Z ; Nyakas, C ; Luiten, PGM Reduced nitric oxide synthase content in the rodent cerebral cortex after middle cerebral artery occlusion. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 13 p. S169 (1993) Közlemény:1852735 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

225. CSAKI, C ; SZABO, C ; BENYO, Z ; KOVACH, AGB Role of platelet-activating factor in the development of endothelial dysfunction in hemorrhagic hypotension and retransfusion THROMBOSIS RESEARCH 66 : 1 pp. 23-31. , 9 p. (1992) DOI WoS Scopus PubMed Közlemény:1628807 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 17 Független: 15 Függő: 2 Idézett közlemények száma: 1

1. SCHMIDSCHONBEIN GW. RELEASE OF PROINFLAMMATORY AND PROTHROMBOTIC MEDIATORS IN THE BRAIN AND PERIPHERAL- CIRCULATION IN SPONTANEOUSLY HYPERTENSIVE AND NORMOTENSIVE WISTAR-KYOTO RATS - COMMENT. (1992) STROKE 0039-2499 1524-4628 23 11 1650-1651 2. * SZABO C et al. INVITED OPINION - ROLE OF NITRIC-OXIDE IN HEMORRHAGIC, TRAUMATIC, AND ANAPHYLACTIC SHOCK AND THERMAL-INJURY. (1994) SHOCK 1073-2322 1540-0514 2 2 145- 155 3. KAPOOR R et al. ROLE OF OXYRADICALS IN CARDIOVASCULAR DEPRESSION AND CELLULAR INJURY IN HEMORRHAGIC-SHOCK AND REINFUSION - EFFECT OF SOD AND CATALASE. (1994) CIRCULATORY SHOCK 0092-6213 43 2 79-94 4. KAPOOR R et al. Benecial Eects of Antioxidants in Hemorrhagic Shock. (1995) ANGIOLOGY 0003-3197 46 4 281-297 5. KUBES P et al. EXCESS NITRIC-OXIDE DOES NOT CAUSE CELLULAR, VASCULAR, MUCOSAL DYSFUNCTION IN THE CAT SMALL- INTESTINE. (1995) AMERICAN JOURNAL OF PHYSIOLOGY: GASTROINTESTINAL AND LIVER PHYSIOLOGY 0193-1857 1522- 1547 269 1 G34-G41 6. WANG P et al. PENTOXIFYLLINE ATTENUATES THE DEPRESSED ENDOTHELIAL-CELL FUNCTION AND VASCULAR MUSCLE CONTRACTILITY FOLLOWING TRAUMA AND HEMORRHAGIC- SHOCK. (1995) JOURNAL OF TRAUMA: INJURY INFECTION AND CRITICAL CARE 0022-5282 1529-8809 2163-0763 1079-6061 39 1 121-127 7. Summers JB et al. Platelet Activating Factor Antagonists. (1995) ADVANCES IN PHARMACOLOGY 1054-3589 32 C 67-168 8. ABUZIDAN FM et al. ROLE OF PLATELET-ACTIVATING-FACTOR ANTAGONISM IN HEMORRHAGIC-SHOCK IN PIGS. (1995) EUROPEAN SURGICAL RESEARCH 0014-312X 1421-9921 27 6 379- 388 9. Kapoor R et al. Cardiac depression and cellular injury in hemorrhagic shock and reinfusion: Role of free radicals. (1997) MOLECULAR AND CELLULAR BIOCHEMISTRY 0300-8177 1573- 4919 176 1-2 291-301 10. Flynn WJ et al. Xanthine oxidase inhibition prevents mesenteric blood ow decits after resuscitated hemorrhagic shock by preserving endothelial function. (1997) JOURNAL OF SURGICAL RESEARCH 0022-4804 1095-8673 68 2 175-180 11. * Szabó C. Molecular Mechanisms of the Nitric Oxide Induced Vessel Wall Dysfunction in Sepsis. (1998) SEPSIS (WARSZAWA) 1898-6307 1899-2013 1 2 107-114 12. Katircioglu SF et al. Benecial eects of iloprost during experimentally induced hemorrhagic shock. (2000) PANMINERVA MEDICA 0031-0808 42 2 109-117 13. Childs EW et al. Eect of WEB 2086 on leukocyte adherence in response to hemorrhagic shock in rats. (2000) JOURNAL OF TRAUMA: INJURY INFECTION AND CRITICAL CARE 0022-5282 1529-8809 2163-0763 1079-6061 49 6 1102-1107 14. Hartig W et al. Functional recovery of cholinergic basal forebrain neurons under disease conditions: Old problems, new solutions?. (2002) REVIEWS IN THE NEUROSCIENCES 0334-1763 13 2 95-165 15. Zakaria ER et al. Impairment of endothelium-dependent dilation response after resuscitation from hemorrhagic shock involved postreceptor mechanisms. (2004) SHOCK 1073-2322 1540-0514 21 2 175-181 16. Victorino GP et al. Modulation of microvascular hydraulic permeability by platelet-activating factor. (2004) JOURNAL OF TRAUMA: INJURY INFECTION AND CRITICAL CARE 0022-5282 1529-8809 2163-0763 1079-6061 56 2 379-384 17. Zakaria ER et al. Role of neutrophils on shock/resuscitation- mediated intestinal arteriolar derangements. (2004) SHOCK 1073- 2322 1540-0514 21 3 248-253

226. Kovach, AGB ; Szabó, C ; Faragó, M ; Horváth, I ; Lohinai, Zs ; Balla, I ; Benyó, Z ; Ottlakán, A ; Csáki, C ; Vass, Z et al. Nitric oxide and global cerebral ischemia In: Krieglstein, J; Oberpichler-Schwenk, H (szerk.) Pharmacology of Cerebral Ischemia Stuttgart, Németország : Wissenschaftliche Verlagsgesellschaft Stuttgart, (1992) pp. 395-408. , 14 p. WoS Közlemény:1537911 Érvényesített Forrás Könyvrészlet (Konferenciaközlemény ) Nyilvános idézők összesen: 7 Független: 1 Függő: 6

1. * SZABO C et al. Eect of superoxide dismutase on hemorrhagic hypotension and retransfusion-evoked middle cerebral artery endothelial dysfunction. (1994) CIRCULATORY SHOCK 0092-6213 44 3 104-110 2. * Benyo Z et al. Intravenous beta-endorphin administration fails to alter hypothalamic blood ow in rats expressing normal or reduced nitric oxide synthase activity. (1996) PEPTIDES 0196-9781 1873-5169 17 4 733-736 3. * Benyo Z et al. Opiate receptor-mediated mechanisms in the regulation of cerebral blood ow. (1996) CEREBROVASCULAR AND BRAIN METABOLISM REVIEWS 1040-8827 8 4 326-357 4. * Komjáti K et al. Cerebrocortical and medullary blood ow changes after general opiate receptor blockade during hemorrhagic shock in cats. (1997) CIRCULATORY SHOCK 0092- 6213 7 4 288-293 5. * Benyo Z et al. Prevention of the hemorrhagic hypotension- induced hepatic arterial vasoconstriction by L-arginine and naloxone. (1999) SHOCK 1073-2322 1540-0514 11 5 342-346 6. * Komjati K et al. Interactions between the endothelium-derived relaxing factor/nitric oxide system and the endogenous opiate system in the modulation of cerebral and spinal vascular CO2 responsiveness. (2001) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 21 8 937-944 7. Hartig W et al. Functional recovery of cholinergic basal forebrain neurons under disease conditions: Old problems, new solutions?. (2002) REVIEWS IN THE NEUROSCIENCES 0334-1763 13 2 95-165

227. KOVACH, AGB ; SZABO, C ; BENYO, Z ; CSAKI, C ; GREENBERG, JH ; REIVICH, M Eects of NG-nitro-L-arginine and L-arginine on regional cerebral blood ow in the cat JOURNAL OF PHYSIOLOGY-LONDON 449 pp. 183-196. , 14 p. (1992) DOI WoS Scopus PubMed Közlemény:1628812 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 143 Független: 125 Függő: 18 Idézett közlemények száma: 1

1. MONCADA C et al. EFFECT OF NO SYNTHASE INHIBITION ON NMDA-INDUCED AND ISCHEMIA-INDUCED HIPPOCAMPAL- LESIONS. (1992) NEUROREPORT 0959-4965 3 6 530-532 2. SYMINGTON PA. PROTECTIVE ACTIONS OF S-NITROSO-N- ACETYLPENICILLAMINE (SNAP) IN A RAT MODEL OF HEMORRHAGIC-SHOCK. (1992) METHODS AND FINDINGS IN EXPERIMENTAL AND CLINICAL PHARMACOLOGY 0379-0355 14 10 789-797 3. FARACI FM. REGULATION OF THE CEREBRAL-CIRCULATION BY ENDOTHELIUM. (1992) PHARMACOLOGY & THERAPEUTICS 0163- 7258 1879-016X 56 1 1-22 4. Fernández N. et al. Cerebral blood ow and cerebrovascular reactivity after inhibition of nitric oxide synthesis in conscious goats. (1993) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 110 1 428-434 5. Lefroy D.C. et al. Eect of inhibition of nitric oxide synthesis on epicardial coronary artery caliber and coronary blood ow in humans. (1993) CIRCULATION 0009-7322 1524-4539 88 1 43-54 6. NISHIKAWA T. EFFECT OF NITRIC-OXIDE SYNTHASE INHIBITION ON CEREBRAL BLOOD-FLOW AND INJURY VOLUME DURING FOCAL ISCHEMIA IN CATS. (1993) STROKE 0039-2499 1524-4628 24 11 1717-1724 7. * KOVACH AGB et al. ENDOTHELIAL DYSFUNCTION IN SHOCK STATES. (1993) NEWS IN PHYSIOLOGICAL SCIENCES 0886-1714 1522-161X 1548-9221 8 145-148 8. FARACI FM. ENDOTHELIAL MODULATION OF CONTRACTIONS CAUSED BY OXYHEMOGLOBIN AND N(G)-NITRO-L-ARGININE IN ISOLATED DOG AND MONKEY CEREBRAL-ARTERIES - COMMENT. (1993) STROKE 0039-2499 1524-4628 24 10 1589-1589 9. FARACI FM. ENDOTHELIUM-DERIVED VASOACTIVE FACTORS AND REGULATION OF THE CEREBRAL-CIRCULATION. (1993) NEUROSURGERY 0148-396X 1524-4040 33 4 648-659 10. WANG Q. NITRIC-OXIDE DOES NOT ACT AS A MEDIATOR COUPLING CEREBRAL BLOOD-FLOW TO NEURAL ACTIVITY FOLLOWING SOMATOSENSORY STIMULI IN RATS. (1993) NEUROLOGICAL RESEARCH 0161-6412 15 1 33-36 11. GARTHWAITE J. NITRIC-OXIDE SIGNALING IN THE NERVOUS- SYSTEM. (1993) SEMINARS IN THE NEUROSCIENCES 1044-5765 5 3 171-180 12. ZHANG FY. NITROPRUSSIDE IMPROVES BLOOD-FLOW AND REDUCES BRAIN-DAMAGE AFTER FOCAL ISCHEMIA. (1993) NEUROREPORT 0959-4965 4 5 559-562 13. MCPHERSON RW et al. N-OMEGA-NITRO-L-ARGININE METHYL- ESTER PREVENTS CEREBRAL HYPEREMIA BY INHALED ANESTHETICS IN DOGS. (1993) ANESTHESIA AND ANALGESIA 0003-2999 1526-7598 77 5 891-897 14. WAHL M. REGULATION OF CEREBRAL BLOOD-FLOW - A BRIEF REVIEW. (1993) ACTA NEUROCHIRURGICA 0001-6268 0942-0940 3-10 15. IADECOLA C. REGULATION OF THE CEREBRAL MICROCIRCULATION DURING NEURAL ACTIVITY - IS NITRIC-OXIDE THE MISSING LINK. (1993) TRENDS IN NEUROSCIENCES 0166- 2236 16 6 206-214 16. MURPHY S et al. SYNTHESIS OF NITRIC-OXIDE IN CNS GLIAL- CELLS. (1993) TRENDS IN NEUROSCIENCES 0166-2236 16 8 323- 328 17. GRANSTAM E et al. VASCULAR EFFECTS OF ENDOTHELIN-1 IN THE CAT - MODIFICATION BY INDOMETHACIN AND L-NAME. (1993) ACTA PHYSIOLOGICA SCANDINAVICA 0001-6772 1365-201X 1748- 1716 148 2 165-176 18. STURAITIS MK et al. A CHOLINERGIC AGONIST INDUCES CEREBRAL HYPEREMIA IN ISOFLURANE-ANESTHETIZED BUT NOT PENTOBARBITAL-ANESTHETIZED DOGS. (1994) ANESTHESIA AND ANALGESIA 0003-2999 1526-7598 78 5 876-883 19. MCPHERSON RW et al. CEREBRAL BLOOD-FLOW IN PRIMATES IS INCREASED BY ISOFLURANE OVER TIME AND IS DECREASED BY NITRIC-OXIDE SYNTHASE INHIBITION. (1994) ANESTHESIOLOGY 0003-3022 1528-1175 80 6 1320-1327 20. CLAVIER N. CEREBRAL BLOOD-FLOW IS REDUCED BY N-OMEGA- NITRO-L-ARGININE METHYL-ESTER DURING DELAYED HYPOPERFUSION IN CATS. (1994) AMERICAN JOURNAL OF PHYSIOLOGY 0002-9513 267 1 H174-H181 21. KELLY PAT et al. CEREBROVASCULAR AUTOREGULATION IN RESPONSE TO HYPERTENSION INDUCED BY N-G-NITRO-L- ARGININE METHYL-ESTER. (1994) NEUROSCIENCE 0306-4522 1873-7544 59 1 13-20 22. PIOTROVSKIJ V et al. DOSE-RANGING STUDY OF N-G-NITRO-L- ARGININE PHARMACOKINETICS IN RATS AFTER BOLUS INTRAVENOUS ADMINISTRATION. (1994) XENOBIOTICA: THE FATE OF FOREIGN COMPOUNDS IN BIOLOGICAL SYSTEMS 0049-8254 1366-5928 24 7 663-669 23. * KOVACH AGB et al. EFFECT OF CEREBRAL OR ENDOTHELIAL NITRIC-OXIDE SYNTHASE (NOS) INHIBITION ON REGIONAL CEREBRAL BLOOD-FLOW (RCBF) AND CEREBRAL NITRIC-OXIDE (NO) CONTENT. (1994) FASEB JOURNAL 0892-6638 1530-6860 8 4 A291-A291 24. * SZABO C et al. Eect of superoxide dismutase on hemorrhagic hypotension and retransfusion-evoked middle cerebral artery endothelial dysfunction. (1994) CIRCULATORY SHOCK 0092-6213 44 3 104-110 25. FABRICIUS M. EXAMINATION OF THE ROLE OF NITRIC-OXIDE FOR THE HYPERCAPNIC RISE OF CEREBRAL BLOOD-FLOW IN RATS. (1994) AMERICAN JOURNAL OF PHYSIOLOGY 0002-9513 266 4 H1457-H1464 26. Rothman S.M.. Excitotoxic neuronal death: Mechanisms and clinical relevance. (1994) SEMINARS IN THE NEUROSCIENCES 1044-5765 6 5 315-322 27. BUCHAN AM. FAILURE TO PREVENT SELECTIVE CA1 NEURONAL DEATH AND REDUCE CORTICAL INFARCTION FOLLOWING CEREBRAL-ISCHEMIA WITH INHIBITION OF NITRIC-OXIDE SYNTHASE. (1994) NEUROSCIENCE 0306-4522 1873-7544 61 1 1- 11 28. AKGOREN N et al. IMPORTANCE OF NITRIC-OXIDE FOR LOCAL INCREASES OF BLOOD-FLOW IN RAT CEREBELLAR CORTEX DURING ELECTRICAL-STIMULATION. (1994) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 0027-8424 1091-6490 91 13 5903-5907 29. VANGELDEREN EM et al. INHIBITION OF NITRIC-OXIDE BIOSYNTHESIS AND CAROTID ARTERIOVENOUS ANASTOMOTIC SHUNTING IN THE PIG. (1994) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 111 3 961-967 30. MCPHERSON RW. INHIBITION OF NITRIC-OXIDE SYNTHASE DOES NOT AFFECT ALPHA(2)-ADRENERGIC-MEDIATED CEREBRAL VASOCONSTRICTION. (1994) ANESTHESIA AND ANALGESIA 0003- 2999 1526-7598 78 1 67-72 31. * SANDOR P et al. MAJOR ROLE OF NITRIC-OXIDE IN THE MEDIATION OF REGIONAL CO2 RESPONSIVENESS OF THE CEREBRAL AND SPINAL-CORD VESSELS OF THE CAT. (1994) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271- 678X 1559-7016 14 1 49-58 32. EDVINSSON L et al. NEUROPEPTIDES IN MIGRAINE AND CLUSTER HEADACHE. (1994) CEPHALALGIA 0333-1024 1468-2982 14 5 320- 327 33. GOLANOV EV. NITRIC-OXIDE AND PROSTANOIDS PARTICIPATE IN CEREBRAL VASODILATION ELICITED BY ELECTRICAL-STIMULATION OF THE ROSTRAL VENTROLATERAL MEDULLA. (1994) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559- 7016 14 3 492-502 34. FARACI FM et al. Nitric oxide and the cerebral circulation. (1994) STROKE 0039-2499 1524-4628 25 3 692-703 35. IADECOLA C. NITRIC OXIDE-DEPENDENT AND NITRIC OXIDE- INDEPENDENT COMPONENTS OF CEREBROVASODILATION ELICITED BY HYPERCAPNIA. (1994) AMERICAN JOURNAL OF PHYSIOLOGY 0002-9513 266 2 R546-R552 36. Sancesario G et al. Nitric oxide inhibition aggravates ischemic damage of hippocampal but not of nadph neurons in gerbils. (1994) STROKE 0039-2499 1524-4628 25 2 436-443 37. Goadsby PJ. Nitric oxide is not the sole determinant of hypercapnic or metabolically driven vasodilation in the cerebral circulation. (1994) JOURNAL OF THE AUTONOMIC NERVOUS SYSTEM 0165-1838 1872-7484 49 SUPPL. 67-72 38. KIMURA M et al. NITRIC-OXIDE REGULATES CEREBRAL ARTERIOLAR TONE IN RATS. (1994) STROKE 0039-2499 1524-4628 25 11 2227-2233 39. IADECOLA C et al. NITRIC-OXIDE SYNTHASE INHIBITION AND CEREBROVASCULAR REGULATION. (1994) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 14 2 175- 192 40. GREENBERG RS et al. NITRIC-OXIDE SYNTHASE INHIBITION WITH N-G-MONO-METHYL-L-ARGININE REVERSIBLY DECREASES CEREBRAL BLOOD BOW IN PIGLETS. (1994) CRITICAL CARE MEDICINE 0090-3493 1530-0293 22 3 384-392 41. DIETRICH HH. N-OMEGA-NITRO-L-ARGININE CONSTRICTS CEREBRAL ARTERIOLES WITHOUT INCREASING INTRACELLULAR CALCIUM LEVELS. (1994) AMERICAN JOURNAL OF PHYSIOLOGY 0002-9513 266 4 H1681-H1686 42. TAGAWA T et al. ROLE OF NITRIC-OXIDE IN REACTIVE HYPEREMIA IN HUMAN FOREARM VESSELS. (1994) CIRCULATION 0009-7322 1524-4539 90 5 2285-2290 43. Horváth I et al. Role of nitric oxide in regulatin cerebrocortical oxygen consumption and blood ow during hypercapnia. (1994) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271- 678X 1559-7016 14 3 503-509 44. MONTAGUE PR et al. ROLE OF NO PRODUCTION IN NMDA RECEPTOR-MEDIATED NEUROTRANSMITTER RELEASE IN CEREBRAL-CORTEX. (1994) SCIENCE 0036-8075 1095-9203 263 5149 973-977 45. BLOT S et al. SPINAL-CORD INFARCTS DURING LONG-TERM INHIBITION OF NITRIC-OXIDE SYNTHASE IN RATS. (1994) STROKE 0039-2499 1524-4628 25 8 1666-1673 46. GOLANOV EV. SPONTANEOUS WAVES OF CEREBRAL BLOOD- FLOW ASSOCIATED WITH A PATTERN OF ELECTROCORTICAL ACTIVITY. (1994) AMERICAN JOURNAL OF PHYSIOLOGY 0002-9513 266 1 R204-R214 47. TODD MM. THE DOSE-RELATED EFFECTS OF NITRIC-OXIDE SYNTHASE INHIBITION ON CEREBRAL BLOOD-FLOW DURING ISOFLURANE AND PENTOBARBITAL-ANESTHESIA. (1994) ANESTHESIOLOGY 0003-3022 1528-1175 80 5 1128-1136 48. * Kovach A et al. The eect of hemorrhagic hypotension and retransfusion and 7-nitro-indazole on rCBF, NOS catalytic activity, and cortical NO content in the cat. (1994) ANNALS OF THE NEW YORK ACADEMY OF SCIENCES 0077-8923 1749-6632 738 348-368 49. * Southan GJ. Amidines are potent inhibitors of nitric oxide synthases: Preferential inhibition of the inducible isoform. (1995) EUROPEAN JOURNAL OF PHARMACOLOGY-MOLECULAR PHARMACOLOGY SECTION 0922-4106 291 3 311-318 50. MARIN J. ANGIOTENSIN-CONVERTING ENZYME-INHIBITORS AND THE ROLE OF NITRIC-OXIDE AND EXCITATORY AMINO-ACIDS IN IMPROVEMENT OF COGNITION AND MEMORY. (1995) JOURNAL OF AUTONOMIC PHARMACOLOGY 0144-1795 1365-2680 1474- 8673 15 2 129-149 51. McCrabb G.J. et al. Cerebral blood ow is increased throughout 12 h of hypoxaemia in the mid-gestation ovine fetus. (1995) REPRODUCTION FERTILITY AND DEVELOPMENT 1031-3613 7 3 463-467 52. KELLY PAT. CEREBROVASCULAR CONSEQUENCES OF REPEATED EXPOSURE TO N-G-NITRO-L-ARGININE METHYL-ESTER. (1995) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 116 6 2771-2777 53. MCPHERSON RW et al. EFFECT OF NITRIC-OXIDE SYNTHASE INHIBITION ON THE CEREBRAL VASCULAR-RESPONSE TO HYPERCAPNIA IN PRIMATES. (1995) STROKE 0039-2499 1524-4628 26 4 682-687 54. * HAMADA J. EFFECTS OF CENTRAL INHIBITION OF NITRIC-OXIDE SYNTHASE ON FOCAL CEREBRAL-ISCHEMIA IN RATS. (1995) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271- 678X 1559-7016 15 5 779-786 55. Nogawa S.. Eects of intravenous arginine administration on cerebral circulation in the rat. (1995) KEIO JOURNAL OF MEDICINE 0022-9717 44 1 19-29 56. * Lohinai Z et al. Evidence for the role of nitric oxide in the circulation of the dental pulp.. (1995) JOURNAL OF DENTAL RESEARCH 0022-0345 74 8 1501-1506 57. * BENYO Z et al. Hypothalamic blood ow remains unaltered following chronic nitric oxide synthase blockade in rats. (1995) NEUROSCIENCE LETTERS 0304-3940 1872-7972 198 2 127-130 58. KELLY PAT. INHIBITION OF NEURONAL NITRIC-OXIDE SYNTHASE BY 7-NITROINDAZOLE - EFFECTS UPON LOCAL CEREBRAL BLOOD- FLOW AND GLUCOSE USE IN THE RAT. (1995) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559- 7016 15 5 766-773 59. NGAI AC. L-NNA SUPPRESSES CEREBROVASCULAR RESPONSE AND EVOKED-POTENTIALS DURING SOMATOSENSORY STIMULATION IN RATS. (1995) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 269 5 H1803-H1810 60. SIENKIEWICZ W. NADPH-DIAPHORASE-CONTAINING CEREBROVASCULAR NERVE-FIBERS AND THEIR POSSIBLE ORIGIN IN THE PIG. (1995) JOURNAL OF BRAIN RESEARCH 0944-8160 36 3 353-363 61. HAXHIU MA. NITRIC-OXIDE AND VENTILATORY RESPONSE TO HYPOXIA. (1995) RESPIRATION PHYSIOLOGY 0034-5687 1872- 7611 1569-9048 101 3 257-266 62. DEVASCONCELOS AP. NITRIC-OXIDE MEDIATES THE INCREASE IN LOCAL CEREBRAL BLOOD-FLOW DURING FOCAL SEIZURES. (1995) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 0027-8424 1091-6490 92 8 3175- 3179 63. KAJITA Y. REGIONAL DIFFERENCES IN CEREBRAL VASOMOTOR CONTROL BY NITRIC-OXIDE. (1995) BRAIN RESEARCH BULLETIN 0361-9230 1873-2747 38 4 365-369 64. TIAN R. ROLE OF EXTRACELLULAR AND INTRACELLULAR ACIDOSIS FOR HYPERCAPNIA-INDUCED INHIBITION OF TENSION OF ISOLATED RAT CEREBRAL-ARTERIES. (1995) CIRCULATION RESEARCH 0009-7330 1524-4571 76 2 269-275 65. SKUJINS A. THE NO SYNTHASE INHIBITOR L-NNA DEPRESSES NEUROHYPOPHYSEAL VASOPRESSIN BUT NOT ITS PRECURSOR AMIDATING ENZYMES IN SALT-LOADED RATS. (1995) ACTA PHYSIOLOGICA SCANDINAVICA 0001-6772 1748-1716 155 3 243- 250 66. SMITH JJ. THE ROLE OF NITRIC-OXIDE IN CEREBROCORTICAL LASER-DOPPLER FLOW RESPONSE TO HALOTHANE IN THE RAT. (1995) JOURNAL OF NEUROSURGICAL ANESTHESIOLOGY 0898- 4921 7 3 187-195 67. FERGUS A et al. VASODILATORY ACTIONS OF CALCITONIN-GENE- RELATED PEPTIDE AND NITRIC-OXIDE IN PARENCHYMAL MICROVESSELS OF THE RAT HIPPOCAMPUS. (1995) BRAIN RESEARCH 0006-8993 1872-6240 694 1-2 78-84 68. Cock ML. Alterations in fetal urine production during prolonged hypoxaemia induced by reduced uterine blood ow in sheep: Mechanisms. (1996) CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY 0305-1870 1440-1681 23 1 57-63 69. Goadsby PJ. Cerebral vasodilatation in the cat involves nitric oxide from parasympathetic nerves. (1996) BRAIN RESEARCH 0006- 8993 1872-6240 707 1 110-118 70. Miyagi Y et al. Dual regulation of cerebrovascular tone by UTP: P- 2U receptor-mediated contraction and endothelium-dependent relaxation. (1996) BRITISH JOURNAL OF PHARMACOLOGY 0007- 1188 1476-5381 118 4 847-856 71. Preckel MP et al. Eect of nitric oxide blockade on the lower limit of the cortical cerebral autoregulation in pentobarbital- anaesthetized rats. (1996) INTERNATIONAL JOURNAL OF MICROCIRCULATION-CLINICAL AND EXPERIMENTAL 0167-6865 16 6 277-283 72. * Vromen A et al. Eects of S-isopropyl isothiourea, a potent inhibitor of nitric oxide synthase, in severe hemorrhagic shock. (1996) JOURNAL OF APPLIED PHYSIOLOGY 8750-7587 1522-1601 81 2 707-715 73. Wang RJ et al. Human dermal broblasts produce nitric oxide and express both constitutive and inducible nitric oxide synthase isoforms. (1996) JOURNAL OF INVESTIGATIVE DERMATOLOGY 0022-202X 106 3 419-427 74. Ohta D. Inhibition of nitric oxide synthase in the brain suppresses colonic motor activity. (1996) AMERICAN JOURNAL OF PHYSIOLOGY: GASTROINTESTINAL AND LIVER PHYSIOLOGY 0193- 1857 1522-1547 270 4 G717-G724 75. Tanaka K. Is nitric oxide really important for regulation of the cerebral circulation? Yes or no?. (1996) KEIO JOURNAL OF MEDICINE 0022-9717 45 1 14-27 76. Patel J et al. Limited role for nitric oxide in mediating cerebrovascular control of newborn piglets. (1996) ARCHIVES OF DISEASE IN CHILDHOOD 0003-9888 1468-2044 75 2 F82-F86 77. Ayata C et al. L-NA-sensitive rCBF augmentation during vibrissal stimulation in type III nitric oxide synthase mutant mice. (1996) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271- 678X 1559-7016 16 4 539-541 78. Thompson BG et al. Nitric oxide mediation of chemoregulation but not autoregulation of cerebral blood ow in primates. (1996) JOURNAL OF NEUROSURGERY 0022-3085 84 1 71-78 79. Tsui S.S.L. et al. Nitric oxide production aects cerebral perfusion and metabolism after deep hypothermic circulatory arrest. (1996) ANNALS OF THORACIC SURGERY 0003-4975 1552-6259 61 6 1699- 1707 80. Shiode N. Nitric oxide production by coronary conductance and resistance vessels in hypercholesterolemia patients. (1996) AMERICAN HEART JOURNAL 0002-8703 1097-6744 131 6 1051- 1057 81. Pajewski TN et al. Nitric oxide synthase inhibitors, 7-nitro indazole and nitro(G)-L-arginine methyl ester, dose dependently reduce the threshold for isourane anesthesia. (1996) ANESTHESIOLOGY 0003-3022 1528-1175 85 5 1111-1119 82. * Szabó C. Physiological and pathophysiological roles of nitric oxide in the central nervous system. (1996) BRAIN RESEARCH BULLETIN 0361-9230 1873-2747 41 131-141 83. Brian JE. Recent insights into the regulation of cerebral circulation. (1996) CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY 0305-1870 1440-1681 23 6-7 449-457 84. Ma JY et al. Regional cerebral blood ow response to vibrissal stimulation in mice lacking type I NOS gene expression. (1996) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 270 3 H1085-H1090 85. Hitchon PW et al. Response of spinal cord blood ow to the nitric oxide inhibitor nitroarginine. (1996) NEUROSURGERY 0148-396X 1524-4040 39 4 795-803 86. Yang ST. Role of nitric oxide in the maintenance of resting cerebral blood ow during chronic hypertension. (1996) LIFE SCIENCES 0024-3205 58 15 1231-1238 87. McCrabb GJ. Role of nitric oxide in the regulation of cerebral blood ow in the ovine foetus. (1996) CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY 0305-1870 1440-1681 23 10-11 855-860 88. Schottler F et al. Structural interactions between NOS-positive neurons and blood vessels in the hippocampus. (1996) NEUROREPORT 0959-4965 7 4 966-968 89. * Lohinai Z et al. The eect of a nitric oxide donor and an inhibitor of nitric oxide synthase on blood ow and vascular resistance in feline submandibular, parotid and pancreatic glands. (1996) ARCHIVES OF ORAL BIOLOGY 0003-9969 41 7 699-704 90. Green LR et al. The role of nitric oxide synthesis in cardiovascular responses to acute hypoxia in the late gestation sheep fetus. (1996) JOURNAL OF PHYSIOLOGY-LONDON 0022-3751 1469-7793 497 1 271-277 91. Fergus A et al. Tonic protein kinase C-mediated vasoconstriction is unmasked when nitric oxide synthase is inhibited in cerebral microvessels. (1996) NEUROSCIENCE 0306-4522 1873-7544 74 3 927-934 92. Ohta D. Central inhibition of nitric oxide synthase modulates upper gastrointestinal motor activity. (1997) AMERICAN JOURNAL OF PHYSIOLOGY: GASTROINTESTINAL AND LIVER PHYSIOLOGY 0193-1857 1522-1547 272 3 G417-G424 93. McCrabb GJ et al. Cerebral oxygen delivery is reduced during the acidaemia associated with prolonged hypoxaemia in the immature ovine fetus. (1997) BIOLOGY OF THE NEONATE 0006- 3126 1661-7800 71 6 385-394 94. Teppema L. Eect of N-omega-nitro-L-arginine on ventilatory response to hypercapnia in anesthetized cats. (1997) JOURNAL OF APPLIED PHYSIOLOGY 8750-7587 1522-1601 82 1 292-297 95. Kwan AL. Inhibition of nitric oxide generation and lipid peroxidation attenuates hemolysate-induced injury to cerebrovascular endothelium. (1997) ACTA NEUROCHIRURGICA 0001-6268 0942-0940 139 3 240-247 96. Reutens DC. L-arginine infusion increases basal but not activated cerebral blood ow in humans. (1997) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 17 3 309- 315 97. Schmetterer L. Nitric oxide synthase inhibition in the histamine headache model. (1997) CEPHALALGIA 0333-1024 1468-2982 17 3 175-182 98. Raevskii KS. Possible role of nitric oxide in pathologies of the central nervous system. (1997) BULLETIN OF EXPERIMENTAL BIOLOGY AND MEDICINE 0007-4888 1573-8221 123 5 419-423 99. Ayers NA. The inhibitory eects of N omega-nitro-L-arginine methyl ester on nitric oxide synthase activity vary among brain regions in vivo but not in vitro. (1997) NEUROCHEMICAL RESEARCH 0364-3190 1573-6903 22 1 81-86 100. Smith JJ. The role of nitric oxide in the cerebrovascular response to hypercapnia. (1997) ANESTHESIA AND ANALGESIA 0003-2999 1526-7598 84 2 363-369 101. Higashino H et al. Tissue variation of acute haemodynamic changes by N-G-nitro-L-arginine in stroke-prone spontaneously hypertensive and Wistar-Kyoto rats. (1997) CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY 0305-1870 1440-1681 24 3-4 249-255 102. Dieguez G. Adrenergic reactivity after inhibition of nitric oxide synthesis in the cerebral circulation of awake goats. (1998) BRAIN RESEARCH 0006-8993 1872-6240 813 2 381-389 103. Stepien A. Level of nitric oxide-dependent cGMP in patients with migraine. (1998) CEPHALALGIA 0333-1024 1468-2982 18 9 631- 634 104. Caramia F. Measurement of changes in cerebral blood volume in spontaneously hypertensive rats following L-arginine infusion using dynamic susceptibility contrast MRI. (1998) MAGNETIC RESONANCE IN MEDICINE 0740-3194 1522-2594 39 1 160-163 105. Bashkatova VG. Nitric oxide in mechanisms of brain damage induced by neurotoxic eect of glutamate. (1998) BIOCHEMISTRY- MOSCOW 0006-2979 1608-3040 63 7 866-873 106. White RP. Nitric oxide synthase inhibition in humans reduces cerebral blood ow but not the hyperemic response to hypercapnia. (1998) STROKE 0039-2499 1524-4628 29 2 467-472 107. Faraci FM. Regulation of the cerebral circulation: Role of endothelium and potassium channels. (1998) PHYSIOLOGICAL REVIEWS 0031-9333 1522-1210 78 1 53-97 108. Kiss B. Acetazolamide-induced cerebral and ocular vasodilation in humans is independent of nitric oxide. (1999) AMERICAN JOURNAL OF PHYSIOLOGY: REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY 0363-6119 1522-1490 276 6 R1661- R1667 109. Hussain MB. Autoregulation of nitric oxide-soluble guanylate cyclase-cyclic GMP signalling in mouse thoracic aorta. (1999) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 128 5 1082-1088 110. Plochl W. Cerebral response to haemodilution during cardiopulmonary bypass in dogs: the role of nitric oxide synthase. (1999) BRITISH JOURNAL OF ANAESTHESIA 0007-0912 1471-6771 82 2 237-243 111. Sandor P. Nervous control of the cerebrovascular system: doubts and facts. (1999) NEUROCHEMISTRY INTERNATIONAL 0197-0186 1872-9754 35 3 237-259 112. Budinger TF et al. Physiological changes in brain microcirculation during activation. (1999) Megjelent: MAGNETIC RESONANCE AND BRAIN FUNCTION: APPROACHES FROM PHYSICS pp. 395-410 113. Gautier H. Role of nitric oxide in hypoxic hypometabolism in rats. (1999) JOURNAL OF APPLIED PHYSIOLOGY 8750-7587 1522-1601 87 1 104-110 114. White RP. The eect of the nitric oxide synthase inhibitor L- NMMA on basal CBF and vasoneuronal coupling in man: a PET study. (1999) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 19 6 673-678 115. Wallis SJ et al. Conditions permitting suppression of stretch- induced and vasoconstrictor tone by basal nitric oxide activity in porcine cerebral artery. (2000) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 130 3 567-574 116. Hilz MJ. Mechanisms of cerebral autoregulation, assessment and interpretation by means of transcranial Doppler sonography. (2000) FORTSCHRITTE DER NEUROLOGIE PSYCHIATRIE 0720-4299 1439-3522 68 9 398-412 117. Gahm C. Temporal proles and cellular sources of three nitric oxide synthase isoforms in the brain after experimental contusion. (2000) NEUROSURGERY 0148-396X 1524-4040 46 1 169-177 118. Sasaki M. The eect of ultraviolet B irradiation on nitric oxide synthase expression in murine keratinocytes. (2000) EXPERIMENTAL DERMATOLOGY 0906-6705 1600-0625 9 6 417- 422 119. Nilsson SFE. The signicance of nitric oxide for parasympathetic vasodilation in the eye and other orbital tissues in the cat. (2000) EXPERIMENTAL EYE RESEARCH 0014-4835 1096-0007 70 1 61-72 120. Fernandez N. Adrenergic vasoconstrictor activity in the cerebral circulation after inhibition of nitric oxide synthesis in conscious goats. (2001) AUTONOMIC NEUROSCIENCE-BASIC & CLINICAL 1566-0702 1872-7484 89 1-2 16-23 121. Strecker T et al. Nitric oxide releases calcitonin-gene-related peptide from rat dura mater encephali promoting increases in meningeal blood ow. (2002) JOURNAL OF VASCULAR RESEARCH 1018-1172 1423-0135 39 6 489-496 122. Kruse A. Nitric oxide synthase activity in human pituitary adenomas. (2002) ACTA NEUROLOGICA SCANDINAVICA 0001- 6314 1600-0404 106 6 361-366 123. * Lacza Z et al. PARP inhibition improves the eectiveness of neural stem cell transplantation in experimental brain trauma.. (2003) INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE 1107-3756 1791-244X 12 2 153-159 124. Cuzzocrea S. Eect of inhibitors of nitric oxide in animal models and future directions for therapy in inammatory disorders. (2004) CURRENT MEDICINAL CHEMISTRY - ANTI-INFLAMMATORY & ANTI-ALLERGY AGENTS 1568-0142 3 3 261-270 125. Mollace V et al. Modulation of prostaglandin biosynthesis by nitric oxide and nitric oxide donors. (2005) PHARMACOLOGICAL REVIEWS 0031-6997 1521-0081 57 2 217-252 126. de Vasconcelos AP. Role of endothelial nitric oxide synthase in cerebral blood ow changes during kainate seizures: A genetic approach using knockout mice. (2006) NEUROBIOLOGY OF DISEASE 0969-9961 1095-953X 23 1 219-227 127. * Hortobagyi L et al. Adaptation of the hypothalamic blood ow to chronic nitric oxide deciency is independent of vasodilator prostanoids. (2007) BRAIN RESEARCH 0006-8993 1872-6240 1131 1 129-137 128. * Szabo C. Hydrogen sulphide and its therapeutic potential. (2007) NATURE REVIEWS DRUG DISCOVERY 1474-1776 6 11 917-935 129. * Lacza Z et al. Additive eect of cyclooxygenase and nitric oxide synthase blockade on the cerebrocortical microcirculation. (2009) NEUROREPORT 0959-4965 20 11 1027-1031 130. Zhou D. Design and Synthesis of 2-Amino-4-methylpyridine Analogues as Inhibitors for Inducible Nitric Oxide Synthase and in Vivo Evaluation of [(18)F]6-(2-Fluoropropyl)-4-methyl-pyridin-2- amine as a Potential PET Tracer for Inducible Nitric Oxide Synthase. (2009) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 52 8 2443-2453 131. Berg RMG et al. Static cerebral blood ow autoregulation in humans. (2009) CURRENT HYPERTENSION REVIEWS 1573-4021 5 2 140-157 132. Perko D et al. Associations between cerebral and systemic endothelial function in migraine patients: a post-hoc study. (2011) BMC NEUROLOGY 1471-2377 11 133. Perko D. Dierences between Cerebrovascular Reactivity to L- Arginine in the Anterior and Posterior Cerebral Circulation. (2011) CEREBROVASCULAR DISEASES 1015-9770 1421-9786 31 4 358-364 134. Yoon SH. pCO 2 and pH regulation of cerebral blood ow. (2012) FRONTIERS IN PHYSIOLOGY 1664-042X 3 SEP 135. Kelly PAT. Pharmacology of the cerebral circulation. (2012) Megjelent: The Pharmacology of Vascular Smooth Muscle pp. 252-275 136. * Iring A et al. Role of endocannabinoids and cannabinoid-1 receptors in cerebrocortical blood ow regulation. (2013) PLOS ONE 1932-6203 1932-6203 8 1 137. Tabatabaei Seyed et al. Nitric Oxide and Cerebrovascular Regulation. (2014) Megjelent: Nitric Oxide pp. 347-385 138. Gralinski M et al. Cardiovascular Analysis In Vivo. (2015) Megjelent: Drug Discovery and Evaluation: Pharmacological Assay, Fourth Edition pp. 41-133 139. * Benyo Z et al. Endocannabinoids in Cerebrovascular Regulation. (2016) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 310 7 H785- H801 140. Tran Hai. Inhibition of MMP-13 with modied polymer particles. (2016) SURFACE SCIENCE 0039-6028 648 371-375 141. Reid Caroline et al. Long Term Amperometric Recordings in the Brain Extracellular Fluid of Freely Moving Immunocompromised NOD SCID Mice. (2017) SENSORS 1424-8220 17 2 142. Koerdt Steen et al. NOS1-, NOS3-, PIK3CA-, and MAPK-pathways in skin following radiation therapy. (2017) BIOMARKER RESEARCH 2050-7771 5 143. Czarnecka Anna et al. Cerebrovascular reactivity and cerebral perfusion of rats with acute liver failure: role of L-glutamine and asymmetric dimethylarginine in L-arginine-induced response. (2018) JOURNAL OF NEUROCHEMISTRY 0022-3042 1471-4159 147 5 692-704

228. Kovách, AGB ; Szabó, C ; Benyó, Z ; Csáki, C Mechanism of the development of endothelial dysfunction in hemorrhagic hypotensionand retransfusion. CIRCULATORY SHOCK 37 p. 25 (1992) Közlemény:1852731 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat ) Nyilvános idézők összesen: 1 Független: 0 Függő: 1

1. * Kovách AGB. Nitric oxide and global cerebral ischemia. (1992) Pharmacology of Cerebral Ischemia 395-408

229. Kovách, AGB ; Szabó, C ; Benyó, Z ; Csáki, C ; Greenberg, HJ ; Reivich, M Role of nitric oxide in regulation of cerebral blood ow. In: Krieglstein, J; Oberpichler-Schwenk, H (szerk.) Pharmacology of Cerebral Ischemia Stuttgart, Németország : Wissenschaftliche Verlagsgesellschaft Stuttgart, (1992) pp. 359-370. , 12 p. WoS Közlemény:1847876 Érvényesített Forrás Könyvrészlet (Konferenciaközlemény ) Nyilvános idézők összesen: 1 Független: 0 Függő: 1

1. * Kovach A et al. The eect of hemorrhagic hypotension and retransfusion and 7-nitro-indazole on rCBF, NOS catalytic activity, and cortical NO content in the cat. (1994) ANNALS OF THE NEW YORK ACADEMY OF SCIENCES 0077-8923 1749-6632 738 348-368

230. SZABO, C ; CSAKI, C ; BENYO, Z ; REIVICH, M ; KOVACH, AGB Role of the L-arginine-nitric oxide pathway in the changes in cerebrovascular reactivity following hemorrhagic hypotension and retransfusion CIRCULATORY SHOCK 37 : 4 pp. 307-316. , 10 p. (1992) PubMed WoS Scopus Közlemény:1628806 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 48 Független: 39 Függő: 9 Idézett közlemények száma: 3

1. FARACI FM. REGULATION OF THE CEREBRAL-CIRCULATION BY ENDOTHELIUM. (1992) PHARMACOLOGY & THERAPEUTICS 0163- 7258 1879-016X 56 1 1-22 2. PARKER JL et al. CORONARY VASCULAR FUNCTION AFTER HEMORRHAGIC HYPOTENSION IN DOGS. (1993) CIRCULATORY SHOCK 0092-6213 41 2 119-129 3. CHI OZ. Diminished eect of inhibition of nitric oxide synthase on regional cerebral vascular resistance in conscious and in isourane anesthetized rats during hemorrhage. (1993) BRAIN RESEARCH 0006-8993 1872-6240 630 1-2 214-220 4. * KOVACH AGB et al. ENDOTHELIAL DYSFUNCTION IN SHOCK STATES. (1993) NEWS IN PHYSIOLOGICAL SCIENCES 0886-1714 1522-161X 1548-9221 8 145-148 5. FARACI FM. ENDOTHELIUM-DERIVED VASOACTIVE FACTORS AND REGULATION OF THE CEREBRAL-CIRCULATION. (1993) NEUROSURGERY 0148-396X 1524-4040 33 4 648-659 6. ANEMAN A et al. Accumulation of an endogenous inhibitor of nitric oxide synthase during graded hemorrhagic shock. (1994) CIRCULATORY SHOCK 0092-6213 44 3 111-114 7. * SZABO C et al. Eect of superoxide dismutase on hemorrhagic hypotension and retransfusion-evoked middle cerebral artery endothelial dysfunction. (1994) CIRCULATORY SHOCK 0092-6213 44 3 104-110 8. * SZABO C et al. INVITED OPINION - ROLE OF NITRIC-OXIDE IN HEMORRHAGIC, TRAUMATIC, AND ANAPHYLACTIC SHOCK AND THERMAL-INJURY. (1994) SHOCK 1073-2322 1540-0514 2 2 145- 155 9. FARACI FM et al. Nitric oxide and the cerebral circulation. (1994) STROKE 0039-2499 1524-4628 25 3 692-703 10. KAPOOR R et al. ROLE OF OXYRADICALS IN CARDIOVASCULAR DEPRESSION AND CELLULAR INJURY IN HEMORRHAGIC-SHOCK AND REINFUSION - EFFECT OF SOD AND CATALASE. (1994) CIRCULATORY SHOCK 0092-6213 43 2 79-94 11. KERWIN JF et al. The arginine-nitric oxide pathway: A target for new drugs. (1994) MEDICINAL RESEARCH REVIEWS 0198-6325 1098-1128 14 1 23-74 12. * Kovach A et al. The eect of hemorrhagic hypotension and retransfusion and 7-nitro-indazole on rCBF, NOS catalytic activity, and cortical NO content in the cat. (1994) ANNALS OF THE NEW YORK ACADEMY OF SCIENCES 0077-8923 1749-6632 738 348-368 13. Thiemermann C. The Role of the L-Arginine: Nitric Oxide Pathway in Circulatory Shock. (1994) ADVANCES IN PHARMACOLOGY 1054- 3589 28 C 45-79 14. BAKER CH. Arteriolar reactivity of endotoxin-tolerant rats after hemorrhage and reinfusion. (1995) SHOCK 1073-2322 1540-0514 4 6 455-460 15. KAPOOR R et al. Benecial Eects of Antioxidants in Hemorrhagic Shock. (1995) ANGIOLOGY 0003-3197 46 4 281-297 16. Chiu T et al. Eect of Inhibition of Nitric Oxide Synthesis on the Cardiovascular and Endocrine Responses to Hemorrhage in Conscious Rabbits. (1995) HYPERTENSION RESEARCH 0916-9636 1348-4214 18 1 55-61 17. PEITZMAN AB. Hemorrhagic shock. (1995) CURRENT PROBLEMS IN SURGERY 0011-3840 32 11 930-1002 18. WANG P et al. PENTOXIFYLLINE ATTENUATES THE DEPRESSED ENDOTHELIAL-CELL FUNCTION AND VASCULAR MUSCLE CONTRACTILITY FOLLOWING TRAUMA AND HEMORRHAGIC- SHOCK. (1995) JOURNAL OF TRAUMA: INJURY INFECTION AND CRITICAL CARE 0022-5282 1529-8809 2163-0763 1079-6061 39 1 121-127 19. * Vromen A et al. Eects of S-isopropyl isothiourea, a potent inhibitor of nitric oxide synthase, in severe hemorrhagic shock. (1996) JOURNAL OF APPLIED PHYSIOLOGY 8750-7587 1522-1601 81 2 707-715 20. Daughters K et al. Increasing nitric oxide production improves survival in experimental hemorrhagic shock. (1996) RESUSCITATION 0300-9572 31 2 141-144 21. Kapoor R et al. Role of polymorphonuclear leukocytes in cardiovascular depression and cellular injury in hemorrhagic shock and reinfusion. (1996) FREE RADICAL BIOLOGY AND MEDICINE 0891-5849 1873-4596 21 5 609-618 22. Kapoor R et al. Cardiac depression and cellular injury in hemorrhagic shock and reinfusion: Role of free radicals. (1997) MOLECULAR AND CELLULAR BIOCHEMISTRY 0300-8177 1573- 4919 176 1-2 291-301 23. Kilbourn R. Nitric oxide and shock - Foreword. (1997) DM DISEASE-A-MONTH 0011-5029 1557-8194 43 5 281-348 24. Kelly E et al. Physiologic and molecular characterization of the role of nitric oxide in hemorrhagic shock: Evidence that type II nitric oxide synthase does not regulate vascular decompensation. (1997) SHOCK 1073-2322 1540-0514 7 3 157-163 25. Krausz MM. The eect of nitric oxide synthase inhibition on hypertonic saline treatment of controlled hemorrhagic shock. (1997) SHOCK 1073-2322 1540-0514 8 6 422-426 26. Flynn WJ et al. Xanthine oxidase inhibition prevents mesenteric blood ow decits after resuscitated hemorrhagic shock by preserving endothelial function. (1997) JOURNAL OF SURGICAL RESEARCH 0022-4804 1095-8673 68 2 175-180 27. * Benyo Z et al. Eect of L-arginine on adrenal and renal blood ows during hemorrhage in cats. (1998) KIDNEY INTERNATIONAL 0085-2538 1523-1755 54 Suppl. 67 S221-S223 28. Smail N et al. Gut and liver - The organs responsible for increased nitric oxide production after trauma-hemorrhage and resuscitation. (1998) ARCHIVES OF SURGERY 0004-0010 1538- 3644 2168-6262 133 4 399-404 29. * Szabó C. Molecular Mechanisms of the Nitric Oxide Induced Vessel Wall Dysfunction in Sepsis. (1998) SEPSIS (WARSZAWA) 1898-6307 1899-2013 1 2 107-114 30. Sato S. [The role of nitric oxide in hemorrhagic shock]. (1998) MASUI/JAPANESE JOURNAL OF ANESTHESIOLOGY 0021-4892 47 4 392-403 31. Price SA et al. Altered vasoconstrictor and dilator responses after a "two-hit" model of sequential hemorrhage and bacteremia. (1999) JOURNAL OF SURGICAL RESEARCH 0022-4804 1095-8673 81 1 59-64 32. Duran-Reyes G et al. Nitric oxide synthesis inhibition suppresses implantation and decreases cGMP concentration and protein peroxidation. (1999) LIFE SCIENCES 0024-3205 65 21 2259-2268 33. * Benyo Z et al. Prevention of the hemorrhagic hypotension- induced hepatic arterial vasoconstriction by L-arginine and naloxone. (1999) SHOCK 1073-2322 1540-0514 11 5 342-346 34. Katircioglu SF et al. Benecial eects of iloprost during experimentally induced hemorrhagic shock. (2000) PANMINERVA MEDICA 0031-0808 42 2 109-117 35. Jeroukhimov I. Eect of methylene blue on resuscitation after haemorrhagic shock. (2001) EUROPEAN JOURNAL OF SURGERY 1102-4151 1741-9271 167 10 742-747 36. Hartig W et al. Functional recovery of cholinergic basal forebrain neurons under disease conditions: Old problems, new solutions?. (2002) REVIEWS IN THE NEUROSCIENCES 0334-1763 13 2 95-165 37. Liu LM et al. Hemorrhage-induced vascular hyporeactivity to norepinephrine in select vasculatures of rats and the roles of nitric oxide and endothelin. (2003) SHOCK 1073-2322 1540-0514 19 3 208-214 38. Guan Xiangdong et al. 血浆一氧化氮浓度与失血性低血容量休克 MODS发生的研究 [Nitric oxide involving in the development of multiple organ dysfunction syndrome after hemorrhagic hypovolemic shock]. (2003) ZHONGGUO SHIYONG WAIKE ZAZHI / CHINESE JOURNAL OF PRACTICAL SURGERY 1005-2208 23 6 355- 357 39. Feng Haomiao et al. L-精氨酸对创伤性休克大鼠的保护作用研究 [Protective eect of L- arginine on a rat model of traumatic shock]. (2004) Chinese Journal of Critical Care Medicine 1002- 1949 24 1 1-3 40. Huang Zonghai et al. 氧化氮合酶抑制剂L-精氨酸对创伤性休克大鼠 存活率的影响 [Inuence of inhibitors of nitric oxide synthase and L-arginine on the survival rate in a rat model of traumatic shock]. (2004) ZHONGGUO WEIZHONGBING JIJIU YIXUE / CHINESE CRITICAL CARE MEDICINE 1003-0603 16 1 45-48 41. Prough DS et al. Eects of hypertonic arginine on cerebral blood ow and intracranial pressure after traumatic brain injury combined with hemorrhagic hypotension. (2006) SHOCK 1073- 2322 1540-0514 26 3 290-295 42. Li D-L. [Eects of progesterone on the content of nitric oxide in the hypoxic brain tissue]. (2006) CHINESE JOURNAL OF CLINICAL REHABILITATION 1671-5926 10 12 71-73 43. Evora PRB et al. The Guanylyl cyclase inhibition by MB as vasoplegic circulatory shock therapeutical target. (2006) CURRENT DRUG TARGETS 1389-4501 7 9 1195-1204 44. Yokoyama Y et al. Trauma-hemorrhage and its eects on the endothelium. (2007) Megjelent: Endothelial Biomedicine pp. 1513-1522 45. Groeneveld ABJ. Hypovolemic Shock. (2008) Megjelent: Critical Care Medicine: Principles of Diagnosis and Management in the Adult pp. 485-520 46. * Szabo C et al. Role of Nitric Oxide in the Development of Vascular Contractile Dysfunction in Circulatory Shock. (2011) JOURNAL OF MEDICAL SCIENCES 1682-4474 1812-5727 31 1 1-16 47. Nin N et al. Vascular Dysfunction in Sepsis: Eects of the Peroxynitrite Decomposition Catalyst MnTMPyP. (2011) SHOCK 1073-2322 1540-0514 36 2 156-161 48. Krumrei NJ. Practical understanding of the pathophysiology of hemorrhagic shock. (2017) Megjelent: Hemorrhagic Shock: Recognition, Pathophysiology and Management pp. 23-40

231. Szabó, C ; Benyó, Z ; Csáki, C ; Kovách, AGB Mechanism of the development of endothelial dysfunction in hemorrhagic hypotensionand retransfusion: Role of the L-arginine - nitric oxide (NO) pathway, superoxide anion, platelet-activating factor (PAF) and opiate receptors. JOURNAL OF VASCULAR RESEARCH 29 p. 204 (1992) Közlemény:1852724 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )

232. BENYO, Z ; KISS, G ; SZABO, C ; CSAKI, C ; KOVACH, AGB IMPORTANCE OF BASAL NITRIC-OXIDE SYNTHESIS IN REGULATION OF MYOCARDIAL BLOOD-FLOW CARDIOVASCULAR RESEARCH 25 : 8 pp. 700-703. , 4 p. (1991) DOI WoS Scopus PubMed Közlemény:1628814 Érvényesített Forrás Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 60 Független: 51 Függő: 9

1. * SZABO C et al. Eect of small changes in extracellular magnesium concentration on the tone of feline mesenteric arteries: involvement of endothelium.. (1992) ACTA PHYSIOLOGICA HUNGARICA 0231-424X 1588-2683 2498-602X 79 3 295-303 2. SEARLE NR. ENDOTHELIAL VASOMOTOR REGULATION IN HEALTH AND DISEASE. (1992) CANADIAN JOURNAL OF ANESTHESIA- JOURNAL CANADIEN D ANESTHESIE 0832-610X 1496-8975 39 8 838-857 3. GREENBLATT EP. ENDOTHELIUM-DEPENDENT CIRCULATORY CONTROL - A MECHANISM FOR THE DIFFERING PERIPHERAL VASCULAR EFFECTS OF ISOFLURANE VERSUS HALOTHANE. (1992) ANESTHESIOLOGY 0003-3022 1528-1175 77 6 1178-1185 4. KLABUNDE RE. N(G)-METHYL-L-ARGININE DECREASES CONTRACTILITY, CGMP AND CAMP IN ISOPROTERENOL- STIMULATED RAT HEARTS INVITRO. (1992) EUROPEAN JOURNAL OF PHARMACOLOGY 0014-2999 1879-0712 223 1 1-7 5. JONES CJH. ENDOTHELIUM-DEPENDENT RELAXATION COMPETES WITH ALPHA-1-ADRENERGIC AND ALPHA-2-ADRENERGIC CONSTRICTION IN THE CANINE EPICARDIAL CORONARY MICROCIRCULATION. (1993) CIRCULATION 0009-7322 1524-4539 87 4 1264-1275 6. GREENBLATT EP. MARKED REGIONAL HETEROGENEITY IN THE MAGNITUDE OF EDRF/NO-MEDIATED VASCULAR TONE IN AWAKE RATS. (1993) JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 0160-2446 1533-4023 21 2 235-240 7. * SZABO C. NITRIC OXIDE-MEDIATED HYPOREACTIVITY TO NORADRENALINE PRECEDES THE INDUCTION OF NITRIC-OXIDE SYNTHASE IN ENDOTOXIN-SHOCK. (1993) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 108 3 786-792 8. ROBERTSON FM. DETRIMENTAL HEMODYNAMIC-EFFECTS OF NITRIC-OXIDE SYNTHASE INHIBITION IN SEPTIC SHOCK. (1994) ARCHIVES OF SURGERY 0004-0010 1538-3644 2168-6262 129 2 149-156 9. GIBSON RL. EFFECT OF NITRIC-OXIDE SYNTHASE INHIBITION DURING GROUP-B STREPTOCOCCAL SEPSIS IN NEONATAL PIGLETS. (1994) PEDIATRIC RESEARCH 0031-3998 1530-0447 36 6 776-783 10. WOODMAN OL. ENHANCEMENT OF NORADRENERGIC CONSTRICTION OF LARGE CORONARY-ARTERIES BY INHIBITION OF NITRIC-OXIDE SYNTHESIS IN ANESTHETIZED DOGS. (1994) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 112 2 443-448 11. LOWENSTEIN CJ. NITRIC-OXIDE - A PHYSIOLOGICAL MESSENGER. (1994) ANNALS OF INTERNAL MEDICINE 0003-4819 1539-3704 120 3 227-237 12. KAM PCA. NITRIC-OXIDE - BASIC SCIENCE AND CLINICAL- APPLICATIONS. (1994) ANAESTHESIA 0003-2409 49 6 515-521 13. BOCZKOWSKI J. ROLE OF NITRIC-OXIDE AND PROSTAGLANDINS IN THE REGULATION OF DIAPHRAGMATIC ARTERIOLAR TONE IN THE RAT. (1994) JOURNAL OF APPLIED PHYSIOLOGY 8750-7587 1522-1601 77 2 590-596 14. * Southan GJ. Amidines are potent inhibitors of nitric oxide synthases: Preferential inhibition of the inducible isoform. (1995) EUROPEAN JOURNAL OF PHARMACOLOGY-MOLECULAR PHARMACOLOGY SECTION 0922-4106 291 3 311-318 15. Orgül S et al. Basic sciences in clinical glaucoma: Physiology of blood ow regulation and mechanisms involved in optic nerve perfusion. (1995) JOURNAL OF GLAUCOMA 1057-0829 1536-481X 4 6 427-443 16. Jones CJ et al. Regulation of coronary blood ow: Coordination of heterogeneous control mechanisms in vascular microdomains. (1995) CARDIOVASCULAR RESEARCH 0008-6363 1755-3245 29 5 585-596 17. * Vromen A et al. Eects of S-isopropyl isothiourea, a potent inhibitor of nitric oxide synthase, in severe hemorrhagic shock. (1996) JOURNAL OF APPLIED PHYSIOLOGY 8750-7587 1522-1601 81 2 707-715 18. Werdan K. Elucidating molecular mechanisms of septic cardiomyopathy - The cardiomyocyte model. (1996) MOLECULAR AND CELLULAR BIOCHEMISTRY 0300-8177 1573-4919 164 291- 303 19. Laughlin M. Endothelium-mediated control of the coronary circulation. Exercise training-induced vascular adaptations. (1996) SPORTS MEDICINE 0112-1642 22 4 228-250 20. Wang RJ. Human dermal broblasts produce nitric oxide and express both constitutive and inducible nitric oxide synthase isoforms. (1996) JOURNAL OF INVESTIGATIVE DERMATOLOGY 0022-202X 106 3 419-427 21. Ellwood AJ. Mechanism of 5-hydroxytryptamine-induced coronary vasodilatation assessed by direct detection of nitric oxide production in guinea-pig isolated heart. (1996) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 119 4 721-729 22. Stowe DF. Neural and endothelial control of the peripheral circulation - Implications for anesthesia .2. Endothelium-mediated eects in the normal and diseased circulation. (1996) JOURNAL OF CARDIOTHORACIC AND VASCULAR ANESTHESIA 1053-0770 1532-8422 10 1 159-171 23. Kevelaitis E. Poststorage diastolic abnormalities of heart transplants: Is vascular dysfunction or myocardial contracture the culprit?. (1996) JOURNAL OF HEART AND LUNG TRANSPLANTATION 1053-2498 15 5 461-469 24. Curtis J. Production of pulmonary vasodilation by tolazoline, independent of nitric oxide production in neonatal lambs. (1996) JOURNAL OF PEDIATRICS 0022-3476 1097-6833 128 1 118-124 25. deLarminat V. Role of prostaglandins and nitric oxide on halothane-induced arteriolar dilatation in rat diaphragm. (1996) BRITISH JOURNAL OF ANAESTHESIA 0007-0912 1471-6771 77 2 232-237 26. Mankad P. Inuence of basal release of nitric oxide on systolic and diastolic function of both ventricles. (1997) JOURNAL OF THORACIC AND CARDIOVASCULAR SURGERY 0022-5223 1097- 685X 113 4 770-776 27. Wang QD. L-arginine enhances functional recovery and Ca2+- dependent nitric oxide synthase activity after ischemia and reperfusion in the rat heart. (1997) JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 0160-2446 1533-4023 29 2 291-296 28. * Kilbourn RG et al. Nitric oxide and shock. (1997) DM DISEASE-A- MONTH 0011-5029 1557-8194 43 5 281-348 29. Wang RJ. Nitric oxide synthase expression and nitric oxide production are reduced in hypertrophic scar tissue and broblasts. (1997) JOURNAL OF INVESTIGATIVE DERMATOLOGY 0022-202X 108 4 438-444 30. Danialou G. Predominant role of A(1) adenosine receptors in mediating adenosine induced vasodilatation of rat diaphragmatic arterioles: Involvement of nitric oxide and the ATP-dependent K+ channels. (1997) BRITISH JOURNAL OF PHARMACOLOGY 0007- 1188 1476-5381 121 7 1355-1363 31. Mankad PS. Relative susceptibility of endothelium and myocardial cells to ischaemia-reperfusion injury. (1997) ACTA PHYSIOLOGICA SCANDINAVICA 0001-6772 1748-1716 161 1 103-112 32. Vag J et al. Blood ow of the submandibular gland in sodium- depleted and -loaded rats: eect of nitric oxide synthase inhibition. (1998) RESEARCH IN EXPERIMENTAL MEDICINE 0300- 9130 198 2 101-108 33. Paulsen S. Expression of inducible nitric oxide synthase in human burn wounds. (1998) WOUND REPAIR AND REGENERATION 1067- 1927 6 2 142-148 34. Pearson PJ. Hypoxia increases vasodilator release from internal mammary artery and saphenous vein grafts. (1998) ANNALS OF THORACIC SURGERY 0003-4975 1552-6259 65 5 1220-1225 35. Bund SJ. Inuence of mode of contraction on the mechanism of acetylcholine-mediated relaxation of coronary arteries from normotensive and spontaneously hypertensive rats. (1998) CLINICAL SCIENCE 0143-5221 1470-8736 94 3 231-238 36. RIJIAN WANG. Interferons and dermal brotic disorders: mtric oxide production and transforming growth factor-PL gene expression by normal and hypertrophic dermal broblasts and tissues after interferon treatment.. (1998) DOCTOR OF PHILOSOPHY p. 252 37. Chandran S. Nitric oxide: Concepts, current perspectives and future therapeutic implications. (1998) INDIAN JOURNAL OF PHARMACOLOGY 0253-7613 1998-3751 30 6 351-366 38. Garcia SR. Nitric oxide modulation of coronary artery myogenic tone in spontaneously hypertensive and Wistar-Kyoto rats. (1998) CLINICAL SCIENCE 0143-5221 1470-8736 94 3 225-229 39. Hably C et al. Nitric oxide synthase inhibition increases vascular resistance in sodium and water loaded rats. (1998) RESEARCH IN EXPERIMENTAL MEDICINE 0300-9130 198 3 145-156 40. Danialou G. Theophylline dilates rat diaphragm arterioles via the prostaglandins pathway. (1998) BRITISH JOURNAL OF PHARMACOLOGY 0007-1188 1476-5381 124 7 1355-1362 41. Mária Szekeres. Control of the tone of coronary resistance arteries. (2000) PhD tézis, Budapest 159 42. * Yang ZQ. Crucial role of endogenous interleukin-10 production in myocardial ischemia/reperfusion injury. (2000) CIRCULATION 0009-7322 1524-4539 101 9 1019-1026 43. * Yang ZQ. Eect of genetic disruption of poly (ADP-ribose) synthetase on delayed production of inammatory mediators and delayed necrosis during myocardial ischemia-reperfusion injury. (2000) SHOCK 1073-2322 1540-0514 13 1 60-66 44. Shinde U. Nitric oxide: A molecule of the millennium. (2000) INDIAN JOURNAL OF EXPERIMENTAL BIOLOGY 0019-5189 0975- 1009 38 3 201-210 45. Sasaki M. The eect of ultraviolet B irradiation on nitric oxide synthase expression in murine keratinocytes. (2000) EXPERIMENTAL DERMATOLOGY 0906-6705 1600-0625 9 6 417- 422 46. Setty S. Endogenous nitric oxide modulates myocardial oxygen consumption in canine right ventricle. (2001) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363- 6135 1522-1539 281 2 H831-H837 47. Waaler B. Fordelingen av blodstrømmen fra venstre hijertekammer. (2001) TIDSSKRIFT FOR DEN NORSKE LEGEFORENING 0029-2001 0807-7096 121 7 817-821 48. Tarr FI. Quantitative measurement of endothelium derived nitric oxide production of the internal mammary artery bypass graft during extracorporeal circulation.. (2001) EUROPEAN JOURNAL OF CARDIO-THORACIC SURGERY 1010-7940 1873-734X 19 5 653-656 49. Yamashita T. Role of endogenous nitric oxide generation in the regulation of vascular tone and reactivity in small vessels as investigated in transgenic mice using synchrotron radiation microangiography. (2001) NITRIC OXIDE-BIOLOGY AND CHEMISTRY 1089-8603 1089-8611 5 5 494-503 50. Setty S. Nitric oxide modulates right ventricular ow and oxygen consumption during norepinephrine infusion. (2002) AMERICAN JOURNAL OF PHYSIOLOGY: HEART AND CIRCULATORY PHYSIOLOGY 0363-6135 1522-1539 282 2 H696-H703 51. * Pacher P et al. Potent metalloporphyrin peroxynitrite decomposition catalyst protects against the development of doxorubicin-induced cardiac dysfunction. (2003) CIRCULATION 0009-7322 1524-4539 107 6 896-904 52. * Lacza Z et al. Prostacyclin-mediated compensatory mechanism in the coronary circulation during acute NO synthase blockade. (2003) LIFE SCIENCES 0024-3205 73 9 1141-1149 53. Chan SL. Vasorelaxations induced by calcitonin gene-related peptide, vasoactive intestinal peptide, and acetylcholine in aortic rings of endothelial and inducible nitric oxide synthase-knockout mice. (2003) JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 0160-2446 1533-4023 41 3 434-443 54. Cuzzocrea S. Eect of inhibitors of nitric oxide in animal models and future directions for therapy in inammatory disorders. (2004) CURRENT MEDICINAL CHEMISTRY - ANTI-INFLAMMATORY & ANTI-ALLERGY AGENTS 1568-0142 3 3 261-270 55. Zong P. Mechanisms of oxygen demand/supply balance in the right ventricle. (2005) EXPERIMENTAL BIOLOGY AND MEDICINE 1535-3702 1535-3699 230 8 507-519 56. Mollace V et al. Modulation of prostaglandin biosynthesis by nitric oxide and nitric oxide donors. (2005) PHARMACOLOGICAL REVIEWS 0031-6997 1521-0081 57 2 217-252 57. Zhou D. Design and Synthesis of 2-Amino-4-methylpyridine Analogues as Inhibitors for Inducible Nitric Oxide Synthase and in Vivo Evaluation of [(18)F]6-(2-Fluoropropyl)-4-methyl-pyridin-2- amine as a Potential PET Tracer for Inducible Nitric Oxide Synthase. (2009) JOURNAL OF MEDICINAL CHEMISTRY 0022-2623 1520-4804 52 8 2443-2453 58. Wang JF. Role of dermal brobasts in post-burn hypertrophic scarring. (2013) Megjelent: Dermal Fibroblasts: Histological Perspectives, Characterization and Role in Disease pp. 57-81 59. Koerdt Steen. NOS1-, NOS3-, PIK3CA-, and MAPK-pathways in skin following radiation therapy. (2017) BIOMARKER RESEARCH 2050-7771 5 60. Mechelinck Mare. Adaptation to acute pulmonary hypertension in pigs. (2018) PHYSIOLOGICAL REPORTS 2051-817X 6 5

233. Benyó, Z ; Kövecs, K ; Kovách, AG ; Sándor, P Inuence of opiate receptor blockade on blood ow and cerebrovascular CO2-sensitivity of dierent brain regions. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 11 p. S272 (1991) Közlemény:1852713 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat ) Nyilvános idézők összesen: 3 Független: 1 Függő: 2

1. * Benyo Z et al. Opiate receptor-mediated mechanisms in the regulation of cerebral blood ow. (1996) CEREBROVASCULAR AND BRAIN METABOLISM REVIEWS 1040-8827 8 4 326-357 2. * Komjati K et al. Regional heterogeneity and dierential vulnerability of cerebral and spinal vascular CO2-responsiveness during graded haemorrhagic hypotension.. (1996) ACTA PHYSIOLOGICA HUNGARICA 0231-424X 1588-2683 2498-602X 84 3 229-249 3. Hauerberg J et al. Eects of morphine and naloxone on cerebral blood ow and metabolism in experimental subarachnoid hemorrhage. (1997) ACTA NEUROLOGICA SCANDINAVICA 0001- 6314 1600-0404 96 3 187-193

234. CSAKI, C ; SZABO, C ; BENYO, Z ; REIVICH, M ; KOVACH, AGB Activated neutrophils inhibit cerebrovascular endothelium-dependent relaxations in vitro LIFE SCIENCES 49 : 15 pp. 1087-1094. , 8 p. (1991) DOI WoS Scopus PubMed Közlemény:1628813 Érvényesített Forrás Idéző Folyóiratcikk (Szakcikk ) Nyilvános idézők összesen: 20 Független: 13 Függő: 7 Idézett közlemények száma: 2 1. FARACI FM. REGULATION OF THE CEREBRAL-CIRCULATION BY ENDOTHELIUM. (1992) PHARMACOLOGY & THERAPEUTICS 0163- 7258 1879-016X 56 1 1-22 2. * SZABO C et al. Role of the L-arginine-nitric oxide pathway in the changes in cerebrovascular reactivity following hemorrhagic hypotension and retransfusion. (1992) CIRCULATORY SHOCK 0092-6213 37 4 307-316 3. FARACI FM. ENDOTHELIUM-DERIVED VASOACTIVE FACTORS AND REGULATION OF THE CEREBRAL-CIRCULATION. (1993) NEUROSURGERY 0148-396X 1524-4040 33 4 648-659 4. * SZABO C et al. Eect of superoxide dismutase on hemorrhagic hypotension and retransfusion-evoked middle cerebral artery endothelial dysfunction. (1994) CIRCULATORY SHOCK 0092-6213 44 3 104-110 5. AKOPOV SE. ENDOTHELIAL DYSFUNCTION IN CEREBRAL VESSELS FOLLOWING CAROTID-ARTERY INFUSION OF PHORBOL ESTER IN RABBITS - THE ROLE OF POLYMORPHONUCLEAR LEUKOCYTES. (1994) JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM 0271-678X 1559-7016 14 6 1078-1087 6. * SZABO C. INVITED OPINION - ROLE OF NITRIC-OXIDE IN HEMORRHAGIC, TRAUMATIC, AND ANAPHYLACTIC SHOCK AND THERMAL-INJURY. (1994) SHOCK 1073-2322 1540-0514 2 2 145- 155 7. Thiemermann C. The Role of the L-Arginine: Nitric Oxide Pathway in Circulatory Shock. (1994) ADVANCES IN PHARMACOLOGY 1054- 3589 28 C 45-79 8. Akopov S. Cerebrovascular reactivity: Role of endothelium/platelet/leukocyte interactions. (1996) CEREBROVASCULAR AND BRAIN METABOLISM REVIEWS 1040- 8827 8 1 11-94 9. Oshiro EM. Inhibition of experimental vasospasm with anti- intracellular adhesion molecule-1 monoclonal antibody in rats. (1997) STROKE 0039-2499 1524-4628 28 10 2031-2037 10. * Kilbourn RG et al. Nitric oxide and shock. (1997) DM DISEASE-A- MONTH 0011-5029 1557-8194 43 5 281-348 11. * Szabó C. Molecular Mechanisms of the Nitric Oxide Induced Vessel Wall Dysfunction in Sepsis. (1998) SEPSIS (WARSZAWA) 1898-6307 1899-2013 1 2 107-114 12. Faraci FM. Regulation of the cerebral circulation: Role of endothelium and potassium channels. (1998) PHYSIOLOGICAL REVIEWS 0031-9333 1522-1210 78 1 53-97 13. Thai QA. Inhibition of experimental vasospasm in rats with the periadventitial administration of ibuprofen using controlled- release polymers. (1999) STROKE 0039-2499 1524-4628 30 1 140- 146 14. * Benyo Z et al. Prevention of the hemorrhagic hypotension- induced hepatic arterial vasoconstriction by L-arginine and naloxone. (1999) SHOCK 1073-2322 1540-0514 11 5 342-346 15. Choi JM. Rebamipide prevents periarterial blood-induced vasospasm in the rat femoral artery model. (2001) PHARMACOLOGICAL RESEARCH 1043-6618 1096-1186 43 5 489- 495 16. Clatterbuck RE. Inhibition of vasospasm with lymphocyte function-associated antigen-1 monoclonal antibody in a femoral artery model in rats. (2002) JOURNAL OF NEUROSURGERY 0022- 3085 97 3 676-682 17. Zakaria ER. Role of neutrophils on shock/resuscitation-mediated intestinal arteriolar derangements. (2004) SHOCK 1073-2322 1540-0514 21 3 248-253 18. * Horvath B et al. Endothelial NOS-mediated relaxations of isolated thoracic aorta of the C57BL/6J mouse a methodological study. (2005) JOURNAL OF CARDIOVASCULAR PHARMACOLOGY 0160- 2446 1533-4023 45 3 225-231 19. Bocheva G. Epigenetic regulation of fetal bone development and placental transfer of nutrients: Progress for osteoporosis. (2011) INTERDISCIPLINARY TOXICOLOGY 1337-6853 1337-9569 4 4 167- 172 20. Bauer V. Endothelium and the eect of activated neutrophils on arterial smooth muscle. (2015) INTERDISCIPLINARY TOXICOLOGY 1337-6853 1337-9569 8 1 44-47 235. Kovách, AGB ; Szabó, C ; Csáki, C ; Benyó, Z ; Kiss, G ; Reivich, M Hemorrhagic hypotension and retransfusion depresses endothelium-dependent responses of feline middle cerebral artery: restoration of normal responses by L-arginine treatment. CIRCULATORY SHOCK 34 pp. 111-112. , 2 p. (1991) Közlemény:1852719 Admin láttamozott Forrás Folyóiratcikk (Absztrakt / Kivonat )