Risk and Protective Factors and Estimates of Substance Use Initiation: Results from the 2015 National Survey on Drug Use and Health Authors SAMHSA: Rachel N
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Fuel Properties Comparison
Alternative Fuels Data Center Fuel Properties Comparison Compressed Liquefied Low Sulfur Gasoline/E10 Biodiesel Propane (LPG) Natural Gas Natural Gas Ethanol/E100 Methanol Hydrogen Electricity Diesel (CNG) (LNG) Chemical C4 to C12 and C8 to C25 Methyl esters of C3H8 (majority) CH4 (majority), CH4 same as CNG CH3CH2OH CH3OH H2 N/A Structure [1] Ethanol ≤ to C12 to C22 fatty acids and C4H10 C2H6 and inert with inert gasses 10% (minority) gases <0.5% (a) Fuel Material Crude Oil Crude Oil Fats and oils from A by-product of Underground Underground Corn, grains, or Natural gas, coal, Natural gas, Natural gas, coal, (feedstocks) sources such as petroleum reserves and reserves and agricultural waste or woody biomass methanol, and nuclear, wind, soybeans, waste refining or renewable renewable (cellulose) electrolysis of hydro, solar, and cooking oil, animal natural gas biogas biogas water small percentages fats, and rapeseed processing of geothermal and biomass Gasoline or 1 gal = 1.00 1 gal = 1.12 B100 1 gal = 0.74 GGE 1 lb. = 0.18 GGE 1 lb. = 0.19 GGE 1 gal = 0.67 GGE 1 gal = 0.50 GGE 1 lb. = 0.45 1 kWh = 0.030 Diesel Gallon GGE GGE 1 gal = 1.05 GGE 1 gal = 0.66 DGE 1 lb. = 0.16 DGE 1 lb. = 0.17 DGE 1 gal = 0.59 DGE 1 gal = 0.45 DGE GGE GGE Equivalent 1 gal = 0.88 1 gal = 1.00 1 gal = 0.93 DGE 1 lb. = 0.40 1 kWh = 0.027 (GGE or DGE) DGE DGE B20 DGE DGE 1 gal = 1.11 GGE 1 kg = 1 GGE 1 gal = 0.99 DGE 1 kg = 0.9 DGE Energy 1 gallon of 1 gallon of 1 gallon of B100 1 gallon of 5.66 lb., or 5.37 lb. -
2012-2015 Ryan White Part B Program Comprehensive Plan
2012-2015 Ryan White Part B Program Comprehensive Plan Table of Contents Acronyms ............................................................................................................................... 1 Acknowledgements ................................................................................................................ 5 Introduction ............................................................................................................................ 6 WV Comprehensive Planning Process .......................................................................... 6 Executive Summary ............................................................................................................... 7 1. Where Are We Now: What is our Current System of Care? ................................... 8 HIV/AIDS In West Virginia-Epidemiologic Trends ..................................................... 8 Unmet Need ................................................................................................................. 24 2010 Unmet Need Framework Report ......................................................................... 27 Early Identification of Individuals with HIV/AIDS (EIIHA) ...................................... 43 Prevention Programs .................................................................................................... 49 Partner Services ........................................................................................................... 50 Continuum of Care ...................................................................................................... -
Automotive Gasoline Cas # 8006-61-9
AUTOMOTIVE GASOLINE CAS # 8006-61-9 Agency for Toxic Substances and Disease Registry ToxFAQs September 1996 This fact sheet answers the most frequently asked health questions (FAQs) about automobile gasoline. For more information, call the ATSDR Information Center at 1-888-422-8737. This fact sheet is one in a series of summaries about hazardous substances and their health effects. This information is important because this substance may harm you. The effects of exposure to any hazardous substance depend on the dose, the duration, how you are exposed, personal traits and habits, and whether other chemicals are present. SUMMARY: Exposure to automotive gasoline most likely occurs from breathing its vapor at a service station while filling a car’s fuel tank. At high levels, automotive gasoline is irritating to the lungs when breathed in and irritating to the lining of the stomach when swallowed. Exposure to high levels may also cause harmful effects to the nervous system. Automotive gasoline has been found in at least 23 of the 1,430 National Priorities List sites identified by the Environmental Protection Agency (EPA). What is automotive gasoline? � Other chemicals in gasoline dissolve in water after spills to surface waters or underground storage tank leaks into (Pronounced ô ) the groundwater. 't;-miftlv gasf;-len' The gasoline discussed in this fact sheet is automotive used � In surface releases, most chemicals in gasoline will prob as a fuel for engines in cars. Gasoline is a colorless, pale brown, or ably evaporate; others may dissolve and be carried away by water; a few will probably stick to soil. -
Recognizing Drug Use in Adolescents
RecognizingRecognizingRecognizing DrugDrugDrug UseUseUse ininin AdolescentsAAdolescentsdolescents A Quick Guide for Caregivers and Adults 1 RecognizingRecognizing DrugDrug UseUse inin AdolescentsAdolescents A Quick Guide for Caregivers and Adults Concerned caregivers and adults play an important role in ensuring that youth receive adequate help. However, at times it is hard to tell that youth are developing a problem with alcohol and drugs. This guide summarizes the signs of intoxication, use, and abuse commonly reported by substance users. It is important to recognize, however, that some of the behaviors and experiences described in this booklet may also be present among adolescents who are not using substances. For this reason, when deciding on the best course of action to obtain help for your teenager, make sure to talk with your teenager, gather as much information as possible, and consult with health professionals available in your community. i Alcohol and drug use poses significant risks for the healthy development of adolescents, yet substances of abuse are often readily accessible at school, at home, and in the community. This guide has been developed to facilitate early identification of substance use problems in youth. Included is information about common drugs of abuse and key information to help identify youth at risk. Recognizing the signs of use includes how a teenager might look, act, and feel while intoxicated as well as drug para- phernalia and language associated with each drug. g Signs of intoxication vary by type of -
Opioid and Nicotine Use, Dependence, and Recovery: Influences of Sex and Gender
Opioid and Nicotine: Influences of Sex and Gender Conference Report: Opioid and Nicotine Use, Dependence, and Recovery: Influences of Sex and Gender Authors: Bridget M. Nugent, PhD. Staff Fellow, FDA OWH Emily Ayuso, MS. ORISE Fellow, FDA OWH Rebekah Zinn, PhD. Health Program Coordinator, FDA OWH Erin South, PharmD. Pharmacist, FDA OWH Cora Lee Wetherington, PhD. Women & Sex/Gender Differences Research Coordinator, NIH NIDA Sherry McKee, PhD. Professor, Psychiatry; Director, Yale Behavioral Pharmacology Laboratory Jill Becker, PhD. Biopsychology Area Chair, Patricia Y. Gurin Collegiate Professor of Psychology and Research Professor, Molecular and Behavioral Neuroscience Institute, University of Michigan Hendrée E. Jones, Professor, Department of Obstetrics and Gynecology; Executive Director, Horizons, University of North Carolina at Chapel Hill Marjorie Jenkins, MD, MEdHP, FACP. Director, Medical Initiatives and Scientific Engagement, FDA OWH Acknowledgements: We would like to acknowledge and extend our gratitude to the meeting’s speakers and panel moderators: Mitra Ahadpour, Kelly Barth, Jill Becker, Kathleen Brady, Tony Campbell, Marilyn Carroll, Janine Clayton, Wilson Compton, Terri Cornelison, Teresa Franklin, Maciej Goniewcz, Shelly Greenfield, Gioia Guerrieri, Scott Gottlieb, Marsha Henderson, RADM Denise Hinton, Marjorie Jenkins, Hendrée Jones, Brian King, George Koob, Christine Lee, Sherry McKee, Tamra Meyer, Jeffery Mogil, Ann Murphy, Christine Nguyen, Cheryl Oncken, Kenneth Perkins, Yvonne Prutzman, Mehmet Sofuoglu, Jack Stein, Michelle Tarver, Martin Teicher, Mishka Terplan, RADM Sylvia Trent-Adams, Rita Valentino, Brenna VanFrank, Nora Volkow, Cora Lee Wetherington, Scott Winiecki, Mitch Zeller. We would also like to thank those who helped us plan this program. Our Executive Steering Committee included Ami Bahde, Carolyn Dresler, Celia Winchell, Cora Lee Wetherington, Jessica Tytel, Marjorie Jenkins, Pamela Scott, Rita Valentino, Tamra Meyer, and Terri Cornelison. -
Power Point Slides: Slide. One Slide for Long Term & Another for Short
Drug Research Project Drug Project Requirements: . Power Point Slides: Slide #1 - Introduction Slide #2 - Title page – Name of drug {technical and street names}, , & a picture of your drug or something that relates to your drug. Slide #3- The look – describe what your drug looks like in detail. Slide #4 - Information Page - Any background, history, statistics, or interesting facts Slide #7 Slide #8 - Drug Category {Narcotic, Depressant, Stimulant, Hallucinogen) AND DEFINE the category. Some drugs may fit into more than one category! Slide #9- Where does this drug come from {plant, lab, country} describe/explain Slide #10- Medical uses for your drug past & present. Slide #11- How is your drug used? {smoked, injected, sniffed, inhaled…} Slide #12- Long & Short term harmful effects of your drug. {Maybe more than one slide. One slide for long term & another for short term effects} Slide #13- Treatment – how can someone quit using the drug. – REHAB Slide #14- Conclusion about your drug how can we eliminate this drug? Slide #15- Site your sources of where you got your information from if taken from another website. Good site to start with: http://abovetheinfluence.com/drugs/alcohol/ TOPICS: 1. Heroin: ________________________________________________________ 2. Phencyclidine (PCP): _____________________________________________ 3. Alcohol: _______________________________________________________ 4. Smoking (cigarettes): _____________________________________________ 5. Smokeless Tobacco: ______________________________________________ -
Estimates of Nitrous Oxide Emissions from Motor Vehicles and the Effects of Catalyst Composition and Aging
ESTIMATES OF NITROUS OXIDE EMISSIONS FROM MOTOR VEHICLES AND THE EFFECTS OF CATALYST COMPOSITION AND AGING Contract No. 02-313 STATE OF CALIFORNIA AIR RESOURCES BOARD Final Report Prepared by Arthur M. Winer, Ph.D. Principal Investigator Environmental Health Sciences Department and Environmental Science and Engineering Program School of Public Health University of California, Los Angeles, California, 90095 (310) 206-1278 Eduardo Behrentz, D.Env. Co-Investigator Environmental Science and Engineering Program School of Public Health University of California, Los Angeles June 10, 2005 N2O Emissions from Motor Vehicles DISCLAIMER The statements and conclusions in this report are those of the contractor and not necessarily those of the California Air Resources Board. The mention of commercial products, their source, or their use in connection with material reported herein is not to be constructed as actual or implied endorsement of such products. i N2O Emissions from Motor Vehicles ii N2O Emissions from Motor Vehicles ACKNOWLEDGEMENTS The contributions of the California Air Resources Board staff, particularly Paul Rieger, who made invaluable suggestions and contributions throughout the project, and Hector Maldonado who acted as our Project Officer, were greatly appreciated. We also thank Richard Ling of the Monitoring and Laboratory Division who collected all emissions data from the 16th Vehicle Surveillance Program and who provided on-site training and support. We thank Margo Eaddy for her participation during the data collection process. We wish to acknowledge valuable contributions from Jerry Ho and Robin Lang of the Mobile Source Operations Division who provided logistical support during vehicle testing and data collection. We especially appreciated Shane Michael, the project’s Test Engineer, of the Mobile Source Operations Division for his professionalism and willingness to assist us throughout the study. -
Tobacco Harm Reduction
Tobacco Harm Reduction Brad Rodu Professor, Department of Medicine James Graham Brown Cancer Center University of Louisville The Smoking Status Quo: Unacceptable • The American Anti-Smoking Campaign is 45 Years Old • According to the CDC: 45 million smokers in the U.S. 443,000 deaths every year in the U.S. 5,800 in Oklahoma Lung Cancer (ICD 161-162) Mortality in Men and Women Age 35+, Oklahoma and the US, 1979-2009 250 OK Men 200 150 US Men OK Women 100 Deaths per 100,000 py 100,000 Deathsper US Women 50 0 Year If the Status Quo Continues In the next 20 years: • 8 million Americans will die from smoking All are adults over 35 years of age None of them are now children The Failed Anti-Smoking Campaign • The Campaign’s Only Message: Quit Nicotine and Tobacco, or Die • The Campaign’s Only Quitting Tactics: Ineffective Behavioral Therapy Ineffective Use of Nicotine Rodu and Cole. Technology 6: 17-21, 1999. Rodu and Cole. International J Cancer 97: 804-806, 2002. The Anti-Smoking Campaign- Behavioral Therapy • NCI Manual for Physicians- Counsel Patients to: – ”Keep your hands busy- doodle, knit, type a letter” – ”Cut a drinking straw into cigarette-sized pieces and inhale air” – ”Keep a daydream ready to go” Source: How to help your patients stop smoking. NIH Pub. No. 93-3064, 1993 The Anti-Smoking Campaign- Faulted Use of Nicotine • Temporary – 6 to 12 weeks • Expensive – per unit and per box • Very Low Dose – unsatisfying for smokers • 7% Success* – ”Efficacious”, ”Modest” *Hughes et al. Meta-analysis in Tobacco Control, 2003. -
Volatile Solvents As Drugs of Abuse: Focus on the Cortico-Mesolimbic Circuitry
Neuropsychopharmacology (2013) 38, 2555–2567 & 2013 American College of Neuropsychopharmacology. All rights reserved 0893-133X/13 www.neuropsychopharmacology.org Review Volatile Solvents as Drugs of Abuse: Focus on the Cortico-Mesolimbic Circuitry 1,2 ,1,2 Jacob T Beckley and John J Woodward* 1 2 Department of Neurosciences, Medical University of South Carolina, Charleston, SC, USA; Center for Drug and Alcohol Programs, Department of Psychiatry/Neurosciences, Medical University of South Carolina, Charleston, SC, USA Volatile solvents such as those found in fuels, paints, and thinners are found throughout the world and are used in a variety of industrial applications. However, these compounds are also often intentionally inhaled at high concentrations to produce intoxication. While solvent use has been recognized as a potential drug problem for many years, research on the sites and mechanisms of action of these compounds lags behind that of other drugs of abuse. In this review, we first discuss the epidemiology of voluntary solvent use throughout the world and then consider what is known about their basic pharmacology and how this may explain their use as drugs of abuse. We next present data from preclinical and clinical studies indicating that these substances induce common addiction sequelae such as dependence, withdrawal, and cognitive impairments. We describe how toluene, the most commonly studied psychoactive volatile solvent, alters synaptic transmission in key brain circuits such as the mesolimbic dopamine system and medial prefrontal cortex (mPFC) that are thought to underlie addiction pathology. Finally, we make the case that activity in mPFC circuits is a critical regulator of the mesolimbic dopamine system’s ability to respond to volatile solvents like toluene. -
Tobacco Fact Sheet What Is Smokeless Tobacco?
Tobacco Fact Sheet Smokeless Tobacco Keep Tobacco Sacred, Honor Your Health, Honor Your Nation Numbers What is Smokeless Tobacco? Smokeless tobacco products contain tobacco or tobacco blends that are ei- at a Glance ther chewed, sucked, or sniffed. Most smokeless tobacco products are placed 80% between the cheek or lips and gums for a few minutes to hours. They have many Higher risk of smokeless names, such as spit tobacco, chew, tobacco users developing oral pinch, or dip, and fall into several cate- cancer. gories. Chewing tobacco is in the form of loose 60% leaves, leaves pressed together that is commonly known as “plug,” to resem- Higher risk of smokeless ble a rope that is commonly known as tobacco users developing “twist.” Chewing tobacco is held be- pancreatic and esophageal tween the cheek and gum. Usually the tobacco juices are spit out, but long- cancer. Smokeless Tobacco is NOT a safe alternative to smoking time users tend to swallow some of the cigarettes juices. Public health advocates worry that laws 28 Snuff is finely ground tobacco that banning smoking in certain public places will comes in dry or moist forms and is Number of cancer causing not effectively encourage people to quit sometimes packaged in ready-to-use agents in smokeless tobacco. using tobacco products as long as snus is pouches. Dry snuff is usually sniffed or available. swallowed, whereas moist snuff— similar to snus (see below)—is placed Dissolvable tobacco is powdered $354 million between the gum and the lip or cheek tobacco that is compressed to resemble a and slowly absorbed. -
Tobacco and E-Cigarette Use in Youth with Autism Spectrum Disorders By
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Locating and Estimating Sources of Methyl Chloroform
EPA-454/R-93-045 LOCATING AND ESTIMATING AIR EMISSIONS FROM SOURCES OF METHYL CHLOROFORM Office of Air Quality Planning and Standards U.S. Environmental Protection Agency Research Triangle Park, North Carolina 27711 February 1994 DISCLAIMER This report has been reviewed by the Office of Air Quality Planning and Standards, and has been approved for publication. Any mention of tradenames or commercial products is not intented to constitute endorsement or recommendation for use. ii CONTENTS Section Page DISCLAIMER ..................................................... ii LIST OF FIGURES .................................................. v LIST OF TABLES ................................................... vi 1.0 PURPOSE OF DOCUMENT ..................................... 1-1 1.1 Reference for Section 1.0 ................................... 1-5 2.0 OVERVIEW OF DOCUMENT CONTENTS .......................... 2-1 2.1 References for Section 2.0 .................................. 2-5 3.0 BACKGROUND .............................................. 3-1 3.1 Nature of Pollutant ....................................... 3-1 3.2 Regulatory Actions Affecting Methyl Chloroform Production and Use .... 3-4 3.3 Overview of Production and Use .............................. 3-7 3.4 References for Section 3.0 ................................. 3-10 4.0 EMISSIONS FROM METHYL CHLOROFORM PRODUCTION ............ 4-1 4.1 Production Process Descriptions .............................. 4-2 4.1.1 Hydrochlorination of Vinyl Chloride ...................... 4-4 4.1.2 Hydrochlorination