Osteitis Condensans Iliiand
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Case Report Chondroblastoma of The
CASE REPORT CHONDROBLASTOMA OF THE PATELLA ASSOCIATED WITH AN ANEURYSMAL BONE CYST R. TREBŠE1, A. ROTTER2,V. PIŠOT1 Chondroblastoma is a rare, benign tumor of bone, cartilage germ cells, and they redefined the tumor accounting for about 1% of all bone tumor cases. It as “benign chondroblastoma”. tends to affect the epiphyseal ends of long bones, Chondroblastoma is rare, representing about 1% most often in males during the first and second of all primary bone tumors (1, 5, 9). It is typically decades of life. It has well-characterized radio- centered in an epiphysis. Although it occurs most graphic and histologic features but despite its histo- often in the end of a long tubular bone, it can logically benign appearance a few cases of metastases appear in any secondary center of ossification. It is have been reported. Local recurrences after curet- tage and bone grafting occur in 11% to 25% of cases. most probably a tumor of cartilaginous origin and The features of a patellar chondroblastoma are the is more common in males by a ratio of about 2-to- same as for other locations. In reviewing the litera- 1 (1, 5, 9). Seventy percent of chondroblastomas ture we found an unusually high male-to-female occur during active epiphyseal plate growth, and ratio. It is interesting that the usual treatment of the about two-thirds of the patients are in the second patellar chondroblastoma has been patellectomy, decade of life (5). whereas curettage and bone grafting has predomi- Local pain of several months’ duration and nated in the other locations. -
Osteochondritis Dissecans
Osteochondritis Dissecans John A. Schlechter, DO Pediatric Orthopaedics and Sports Medicine Children’s Hospital Orange County Osteochondritis Dissecans • Developmental condition of the joint − Described by Paget as “quiet necrosis” − Named by Konig 1888 • Lesion of the articular cartilage & subchondral bone before closure of the growth plate Is it OCD? • OCD vs Normal Variant of Ossification • Normal Variants − Tend to be younger patients age <10 − Tend to affect both condyles − Posterior aspect of condyle − Resolves as the child ages OCD Stats • Highest rates − appear among patients aged between 10 and 15 y. Male-to-female ratio ~ 2:1 − ADHD? • Bilaterality − typically in different phases of development, are reported in 15% to 30% of cases Osteochondritis Dissecans • Etiology unknown • Proposed causative factors: − Ischemia − heredity − mechanics (trauma) Osteochondritis Dissecans • Repetitive mechanical trauma or stress, in highly active children & adolescents • Impaction of the tibial spine Osteochondritis Dissecans Symptoms, Signs & Imaging • Nonspecific knee pain • Activity-related • Wilson test • “tunnel view” • MRI - stability of the subchondral bone, arthrography AP view – does not always show OCD Notch view – reveals OCD Location • Cahill described a method of localizing lesions by dividing the knee into 15 distinct alphanumeric zones Am J Sports Med 1983;11: 329-335. Osteochondritis Dissecans Symptoms, Signs & Imaging Osteochondritis Dissecans MRI Staging Hefti et al. JPO-B 1999 • Stage I: Signal change, NO clear margin • Stage II: Clear margin, NO Dissection • Stage III: Partial Dissection of fluid • Stage IV: Complete Dissection, Fragment In Situ • Stage V: Free Fragment I - No Clear margin II- Clear margin III- Partial Dissection Hefti et al. JPO-B 1999 IV- Partial Dissection V- Loose Body Case Example – Hefti 3 MRI Coronal T2 Cartilage Breach Osteochondritis Dissecans Natural History • Patients with open physes fare better than adults. -
Osteochondritis Dissecans (OCD) Results in the Destruction of Subchondral Bone with Secondary Damage to Overlying Articular Cartilage (1,2)
Copy Editor: Selvi S Osteochondritis 45 Dissecans Cecilia Pascual-Garrido, Taylor M. Southworth, Mark A. Slabaugh, Neal B. Naveen, Nicole A. Friel, Ben U. Nwachukwu, and Brian J. Cole prohibited. INTRODUCTION is Osteochondritis dissecans (OCD) results in the destruction of subchondral bone with secondary damage to overlying articular cartilage (1,2). The prevalence of this condition is estimated to contentbe between 11.5 and 21 per 100,000 (3,4). OCD is classically divided into juvenile and adult forms based on the patient’s skeletal maturity (1). Juvenile OCD (JOCD) lesions occur in childrenthe and young adolescents with open growth plates. Although adult OCD lesions may arise de ofnovo, they more commonly result from an incompletely healed and previously asymptomatic JOCD lesion (5). JOCD lesions have a better prognosis and higher rates of spontaneous healing with conservative treatment than do adult OCD lesions (6). Adult OCD lesions have a greater propensity for instabil- ity and, once symptomatic, typically follow a clinical course that is progressive and unremitting (7). While lesions most frequently occur on the femoral condyles, they are also found in the elbow, wrist, ankle, and femoral head (8–12). The highest incidence rates in JOCDreproduction are among patients ages 10 and 15 years old, ranking among the most common causes of knee pain and dysfunction in young Fig. 45-1 adults (6,7,13) (Fig. 45-1). The typical presentation of OCD in the knee includes pain and swelling related to activity. Instability is not usually reported, though mechanical symptoms, such as catching and locking, usually occur in the presence of a loose body and are frequently the initial presenting symptoms. -
Hyperostosis Corticalis Infantalis (Caffey's Disease)* J
754 S.A. TYDSKRIF VIR GENEESKU DE 14 Junie 1969 HYPEROSTOSIS CORTICALIS INFANTALIS (CAFFEY'S DISEASE)* J. M. WAGNER, M.B., B.CH., M.R.e.p., Senior Paediatrician, Edenvale Hospital, AND A. SoLOMON, M.B., B.CH., DIP.MED., D.M.R.(D.), Radiologist, Pneumoconiosis Research Unit, CSIR, Baragwanath Hospital, Johannesburg Infantile cortical hyperostosis is a disorder affecting the skeleton and some of its contiguous fascias and muscles. It is suggested that infantile cortical hyperostosis is a pre natal collagen disease.' The early stage is of acute inflammation and loss of the periosteal and subperiosteal definition. There is a fibrous and osteoblastic reaction and overlying tissue including muscle is involved. No bacteria are seen. Later, there is subperiosteal new lamellar bone formation. The peri osteum is thickened and hyperplastic and the overlying soft tissues are oedematous and sections show round-cell infiltration. The subacute phase re-establishes periosteum as an entity. The later remodelling stage removes the extraperipheral bone from within, resulting in dilatation of the medullary cavity. There is evidence to suggest that infantile cortical hyper ostosis had been recognized in 1930.' Caffey and Silver man first described the disease in 1946." Smyth et al.' also recorded cases in 1946. Altogether 102 cases have been reported, Sidbury and Sidbury' contributing 69 reports and Holrnan' describing 33 cases. Infantile cortical hyper ostosis has been described in Negroes but seems rare in the South African Bantu. CASE REPORT The patient, a 3-year-old Bantu male, had a normal birth weight. Two siblings were in good health. The mother stated that the child's jaw was swollen and that he had Fig. -
Osteomyelitis 78
Osteomyelitis 78 SECTION K: Bone and Joint Infections 78 Osteomyelitis Kathleen Gutierrez Osteomyelitis is inflammation of bone. Bacteria are the usual etio- months, providing a vascular connection between the metaphysis logic agents, but fungal osteomyelitis occurs occasionally. Osteo- and the epiphysis.8 As a result, in infants, infection originating in myelitis in children primarily has hematogenous origin, occurring the metaphysis can spread to the epiphysis and joint space. The less commonly as a result of trauma, surgery, or infected contigu- risk of ischemic damage to the growth plate is greater in the young ous soft tissue. Osteomyelitis due to vascular insufficiency is rare infant with osteomyelitis.9 Before puberty, the periosteum is not in children. firmly anchored to underlying bone. Infection in the metaphysis of a bone can spread to perforate the bony cortex, causing sub- ACUTE HEMATOGENOUS OSTEOMYELITIS periosteal elevation and extension into surrounding soft tissue. Bony destruction can spread to the diaphysis. By age of 2 years, Pathogenesis the cartilaginous growth plate usually prevents extension of infec- tion to the epiphysis and into the joint space. When the metaphy- Acute hematogenous osteomyelitis (AHO) is primarily a disease sis of the proximal femur or humerus is involved, however, of young children, presumably because of the rich vascular supply infection can extend into the hip or shoulder joint at any age, of their rapidly growing bones.1–3 Infecting organisms enter the because at these sites, the metaphysis is intracapsular. bone through the nutrient artery and then travel to the metaphy- Histologic features of acute osteomyelitis include localized sup- seal capillary loops, where they are deposited, replicate, and puration and abscess formation, with subsequent infarction and initiate an inflammatory response (Figure 78-1). -
Osteochondral Injury of the Knee
® Volume 2, Part 3 December 2005 ORTHOPAEDIC SPORTS MEDICINE Board Review Manual Osteochondral Injury of the Knee Endorsed by the Association for Hospital www.turner-white.com Medical Education Your vision is our new bottom line. The company long respected for advancing the science of cartilage repair has more to offer than you ever anticipated. An established leader in the development of biomaterials and cell therapies, Genzyme Biosurgery is excited to now be driving the marketing and distribution of Synvisc® (hylan G-F 20). And our pioneering research into novel OA and cartilage repair solutions is destined to redefine the field of orthobiologics. So take a second look at Genzyme Biosurgery. What you see may surprise you. Genzyme Biosurgery 55 Cambridge Parkway Cambridge, MA 02142 GENZYME and SYNVISC are registered 1-800-901-7251 trademarks of Genzyme Corporation. www.genzymebiosurgery.com ® ORTHOPAEDIC SPORTS MEDICINE BOARD REVIEW MANUAL STATEMENT OF EDITORIAL PURPOSE Osteochondral Injury The Hospital Physician Orthopaedic Sports Medi- cine Board Review Manual is a peer-reviewed of the Knee study guide for orthopaedic sports medicine fellows and practicing orthopaedic surgeons. Contributors: Each manual reviews a topic essential to the current practice of orthopaedic sports medi- Jason M. Scopp, MD cine. Director, Cartilage Restoration Center, Peninsula Orthopaedic Associates, PA, Salisbury, MD PUBLISHING STAFF PRESIDENT, GROUP PUBLISHER Bert R. Mandelbaum, MD Bruce M. White Fellowship Director, Santa Monica Orthopaedic EDITORIAL DIRECTOR and Sports Medicine Group, Santa Monica, CA Debra Dreger ASSOCIATE EDITOR Editor: Tricia Faggioli Andrew J. Cosgarea, MD EDITORIAL ASSISTANT Associate Professor, Department of Orthopaedic Surgery, Farrawh Charles Johns Hopkins University School of Medicine, Baltimore, MD EXECUTIVE VICE PRESIDENT Barbara T. -
Osteochondrosis
Osteochondrosis (Abnormal Bone Formation in Growing Dogs) Basics OVERVIEW • Long bones (such as the humerus, radius and ulna in the foreleg and the femur and tibia in the rear leg) have three sections: the end of the bone, known as the “epiphysis”; the shaft or long portion of the bone, known as the “diaphysis”; and the area that connects the end and the shaft of the bone, known as the “metaphysis” • The metaphysis is the area where bone growth occurs in puppies; the long bones in the body grow in length at specific areas known as “growth plates”; these areas usually continue to produce bone until the bones are fully developed, at which time, no further growth is needed; the growth plates then “close” and become part of the hard bone • Bone is formed by the replacement of calcified cartilage at the growth plates; the bone-forming cells (known as “osteoblasts”) form bone on the cartilage structure; this process is known as “endochondral ossification” • “Osteochondrosis” is a disorder of bone formation in the growth plates (areas where bone grows in length in the young pet) of the bone; it is a disease process in growing cartilage, primarily characterized by a disturbance of the change from cartilage to bone (known as “endochondral ossification”) during bone development that leads to excessive retention of cartilage GENETICS • Multiple genes are involved (known as “polygenetic transmission”)—expression determined by an interaction of genetic and environmental factors • Heritability index—depends on breed, 0.25-0.45 SIGNALMENT/DESCRIPTION -
Anterior Impingement with Exostosis and Fragmentation of the Talus
ANTEROIR IMPINGEMENT WITH EXOSTOSIS AND FRAGMENTATION OF THE TALUS, NAVICULAR AND TIBIA IN A MALE COLLEGIATE BASKETBALL PLAYER Belter CA, Lopez RM, McDermott BP; University of Connecticut, Storrs, CT Background: During a regular season game, a 20-year-old male collegiate division I basketball player landed on an opponent’s foot after an offensive rebound. He inverted his right ankle but did not feel or hear a “pop.” He had experienced a grade one ankle sprain on the same ankle during preseason play. Upon evaluation, the athlete showed no signs of deformity. He was point tender over the anterior talofibular, calcaneofibular, and posterior talofibular ligaments. Passive range of motion (ROM) was within normal limits; however, active ROM was limited in plantarflexion and dorsiflexion. Manual muscle testing elicited soreness, but was within normal limits. All neurological tests were normal. An anterior drawer test was positive with increased laxity when compared bilaterally. The talar tilt test was negative. Differential Diagnosis: Ankle syndesmodic sprain, tarsal stress fracture, osteochondritis dissecans (OCD), lateral ankle sprain, anterior impingement with exostosis. Treatment: The team physician ordered an X-ray due to the athlete’s previous history of ankle sprains. Radiographs were negative for fractures but revealed bone spurs that were deemed asymptomatic. The athlete was given a walking boot for two days. He completed rehabilitation for a lateral ankle sprain that consisted of ice and compression, proprioception exercises, calf raises, calf stretches, and stability and agility exercises. Four days post injury, the athlete participated in a competition for approximately twenty minutes. He did not compete in subsequent competitions secondary to pain, weakness and an inability to complete prior practices. -
Chronic Non-Bacterial Osteomyelitis/Osteitis (Or CRMO) Version of 2016
https://www.printo.it/pediatric-rheumatology/IE/intro Chronic non-Bacterial Osteomyelitis/Osteitis (or CRMO) Version of 2016 1. WHAT IS CRMO 1.1 What is it? Chronic Recurrent Multifocal Osteomyelitis (CRMO) is the most severe form of Chronic Non-bacterial Osteomyelitis (CNO). In children and adolescents, the inflammatory lesions predominantly affect the metaphyses of the long bones of the lower limbs. However, lesions can occur at any site of the skeleton. Furthermore, other organs such as the skin, eyes, gastrointestinal tract and joints can be affected. 1.2 How common is it? The frequency of this disease has not been studied in detail. Based on data from European national registries, approximately 1-5 of 10,000 inhabitants might be affected. There is no gender predominance. 1.3 What are the causes of the disease? The causes are unknown. It is hypothesised that this disease is linked to a disturbance in the innate immune system. Rare diseases of bone metabolism might mimic CNO, such as hypophosphatasia, Camurati- Engelman syndrome, benign hyperostosis-pachydermoperiostosis and histiocytosis. 1.4 Is it inherited? 1 / 6 Inheritance has not been proven but is hypothesized. In fact, only a minority of cases is familial. 1.5 Why does my child have this disease? Can it be prevented? The causes are unknown to date. Preventive measures are unknown. 1.6 Is it contagious or infectious? No, it is not. In recent analyses, no causative infectious agent (such as bacteria) has been found. 1.7 What are the main symptoms? Patients usually complain of bone or joint pain; therefore, the differential diagnosis includes juvenile idiopathic arthritis and bacterial osteomyelitis. -
Human Osteology Method Statement N
Human osteology method statement N. Powers (ed) Published online March 2008 Revised February 2012 2 LIST OF CONTRIBUTORS Museum of London Archaeology Centre for Human Bioarchaeology Service (MoLAS) (CHB) Brian Connell HND MSc Jelena Bekvalac BA MSc Amy Gray Jones BSc MSc Lynne Cowal BSc MSc Natasha Powers BSc MSc MIFA RFP Tania Kausmally BSc MSc Rebecca Redfern BA MSc PhD Richard Mikulski BA MSc Don Walker BA MSc AIFA Bill White Dip Arch FRSC FSA 3 CONTENTS Introduction ........................................................................................................................ 8 1 Preservation and archaeological data................................................................... 9 2 Catalogue of completeness ................................................................................... 10 2.1 Cranial elements ..................................................................................................... 10 2.2 Post-cranial elements.............................................................................................. 10 2.3 Cartilage.................................................................................................................. 11 2.4 Dentition ................................................................................................................. 11 3 Age at death estimation........................................................................................ 12 3.1 Subadult age at death............................................................................................. -
Tuberculosis of the Pubic Symphysis Masquerading As Osteitis Pubis: a Case Report
CASE REPORT Acta Orthop Traumatol Turc 2012;46(3):223-227 doi:10.3944/AOTT.2012.2696 Tuberculosis of the pubic symphysis masquerading as osteitis pubis: a case report Shailendra SINGH, Sumit ARORA, Sumit SURAL, Anil DHAL Department of Orthopedic Surgery, Maulana Azad Medical College & Associated Lok Nayak Hospital, New Delhi, India Tuberculosis is one of the oldest diseases affecting mankind and is known for its ability to present in various forms and guises. Pubic symphysis is an uncommon site for tuberculous affliction; hence very few cases have been reported in the English-language literature. We present a rare case of pubic sym- physis tuberculosis diagnosed as osteitis pubis before presentation to our institution. The patient made an uneventful recovery following antitubercular chemotherapy. Key words: Antitubercular chemotherapy; osteitis pubis; pubic symphysis; tuberculosis. Tuberculosis has been recorded in Egyptian mummies Case report aging back to 3000 B.C. It commonly affects the pul- A 35-year-old male presented with a history of supra- monary system but extrapulmonary involvement is pubic pain for six months following a fall while playing seen in approximately 14% of patients, with 1% to 8 % football. The pain was insidious in onset, dull aching in [1] having osseous involvement. The major areas of nature and localized in the suprapubic area. It predilection in order of occurrence are: spine, hip, increased on exertion and relieved with rest and anti- knee, foot, elbow and hand. Tuberculosis of the pubic inflammatory medications and was not aggravated by symphysis is rare and few cases have been presented in coughing, sneezing, voiding or straining during stool. -
Infantile Peri-Osteitis Postgrad Med J: First Published As 10.1136/Pgmj.74.871.307 on 1 May 1998
Self-assessment questions 307 Infantile peri-osteitis Postgrad Med J: first published as 10.1136/pgmj.74.871.307 on 1 May 1998. Downloaded from Alaric Aroojis, Harold D'Souza, M G Yagnik A 14-week-old girl was brought in with a history of painful swelling of both legs since the age of one month. The onset was insidious and was not associated with trauma or fall. There was no his- tory of fever or associated constitutional symp- toms. The birth history was normal and the infant was apparently asymptomatic until the age of one month. Examination revealed a healthy and alert infant. Both legs were bowed anteriorly and a uniform bony thickening of both tibiae was palpable throughout their lengths (figure 1). Both legs were extremely ten- der and the infant would withdraw both lower limbs and cry incessantly if any attempt was made to touch them. There was no increase in local temperature nor redness of the overlying skin. Knees and ankle joints were normal and demonstrated a full range of motion. Regional lymph nodes were not enlarged and other bones and joints were normal on examination. X-Rays of both legs revealed peri-osteitis of both tibiae with extensive subperiosteal new bone forma- tion involving the entire diaphysis (figure 2). Questions 1 What is the differential diagnosis of peri- osteitis in an infant? 2 What further investigations are required? Figure 1 Clinical photograph showing bony swelling 3 What is the likely diagnosis and treatment? with anterior bowing of both legs http://pmj.bmj.com/ on October 5, 2021 by guest.