A Cross-Sectional Descriptive Study of Dermoscopy in Various Nail10.5005/Jp-Journals-10061-0003 Diseases at a Tertiary Care Center Original Article

Total Page:16

File Type:pdf, Size:1020Kb

A Cross-Sectional Descriptive Study of Dermoscopy in Various Nail10.5005/Jp-Journals-10061-0003 Diseases at a Tertiary Care Center Original Article IJDY A Cross-sectional Descriptive Study of Dermoscopy in various Nail10.5005/jp-journals-10061-0003 Diseases at a Tertiary Care Center ORIGINAL ARTICLE A Cross-sectional Descriptive Study of Dermoscopy in various Nail Diseases at a Tertiary Care Center 1Dipali Rathod, 2Meena Bhaskar Makhecha, 3Manas Chatterjee, 4Tishya Singh, 5Shekhar Neema ABSTRACT Clinical significance: Dermoscopy is a noninvasive comple- mentary tool which aids in diagnosis of nail diseases more Introduction: Nail diseases may often possess a challenge for quickly and can also be used for monitoring the evolution, the treating dermatologist, as they may primarily be affected therapeutic response, and prognosis of these diseases. without any skin involvement and sometimes may present with subtle changes which can be easily missed by the naked Keywords: Alopecia areata, Lichen planus, Melanonychia, eye. The macroscopic nail changes can be assessed very Nail dermoscopy, Nail psoriasis, Onychomycosis, Pitted kera- well with a naked eye, however, the important minutiae may tolysis, Scleroderma, Trachyonychia. be assessed with a dermoscope. How to cite this article: Rathod D, Makhecha MB, Chatterjee Aim: To study the characteristic dermoscopic findings in M, Singh T, Neema S. A Cross-sectional Descriptive Study of various nail diseases. Dermoscopy in various Nail Diseases at a Tertiary Care Center. Materials and methods: A total of 250 patients (males-130, Int J Dermoscop 2017;1(1):11-19. females-120) with clinically evident nail diseases were enrolled Source of support: Nil in this cross-sectional descriptive study. After a thorough clinical examination, patients were subjected to dermoscopic Conflict of interest: None examination of all 20 nails. Chi-square test or Fisher’s exact test (in a very few patients of nail diseases) were used for INTRODUCTION statistical analysis, with a significance threshold of p < 0.05. Nails may be involved primarily or secondarily due to Results: Nail psoriasis (n = 45) was the commonest nail disease found in which the most common dermoscopic feature underlying systemic diseases and their examination may was circular punctate depressions (64.5%). Onychomycosis provide clues to establish the diagnosis and guidance (n = 40) was the next common disease in which yellowish regarding the management of these nail diseases. Several discoloration (100%) was commonly seen in all patients and skin, hair, and nail diseases may result in severe nail rough scaly surface (100%) in the total dystrophic type. In pitted keratolysis (n = 19) transverse brown bands (57.9%) were most abnormalities, which if not addressed, may cause func- commonly seen as was the case in eczemas. Lichen planus tional interference and permanent dystrophy. Diseases of (n = 14) was associated with longitudinal fissuring (64.3%) most nail comprise approximately 10% of all the dermatological commonly. Trachyonychia (n = 5) was associated with longitu- conditions.1,2 Clinicopathologic tools are time-consuming dinal fissuring (100%) in all cases. The most common dermo- and give false negative results in up to 35% of patients. scopic feature of alopecia areata (n = 4) was circular punctate depressions (75%). Systemic scleroderma (n = 4) typically Although, the naked eye can appreciate majority of the showed giant capillaries (75%) as the most common feature. macroscopic details of the nail unit apparatus, dermo- Conclusion: A meticulous search with the dermoscope, fur- scope furnishes details which may be easily missed. This nishes minuscule details of the nail unit that can be diagnostic of article provides the diagnostic importance of dermoscope several nail diseases at an early stage before dystrophy sets in. in the evaluation of various nail diseases. However, further studies are required to validate these findings. However, before we move onto the main topic, let us see the difference between nail and skin dermoscopy. Nail dermoscopy is technically difficult owing to 1,2,5 3 4 Assistant Professor, Senior Advisor and Head, Resident nail size, shape, convexity, and hardness. The entire nail 1,3Department of Dermatology, INHS, Asvini, RC Church, Colaba cannot be visualized as a whole at one particular time, Mumbai, Maharashtra, India therefore, cannot be easily interpreted and it is cumber- 2,4Department of Dermatology, Hinduhrudaysamrat Balasaheb some to take pictures of this area. Nail plate surface Thackeray Medical College & Dr. Rustam Narsi Cooper Municipal changes can be appreciated with dry dermoscopy, General Hospital, Juhu, Vile Parle (West), Mumbai, Maharashtra India whereas color abnormalities can be appreciated with large quantities of interface medium (linkage fluid gel) or by 5Department of Dermatology, Command Hospital (EC), Kolkata West Bengal, India use of polarized dermoscopy. Corresponding Author: Dipali Rathod, Assistant Professor Normal Nail Unit under the Dermoscope Department of Dermatology, INHS, Asvini, RC Church, Colaba Mumbai, Maharashtra, India, Phone: +918655638949, e-mail: The nail plate appears pale pink in color with smooth [email protected] and shiny surface. Nail bed below the transparent plate International Journal of Dermoscopy, May-August 2017;1(1):11-19 11 Dipali Rathod et al appears pale pink too. The epithelium of proximal A total of 250 out of 42,105 patients attending der- nailfold (PNF) has a smooth surface with pale pink to matology OPD were enrolled, that accounted for an brown color. Cuticle is easily visible as a transparent trans- incidence of 0.60% during this study period. verse band that seals the nail plate to PNF. Hyponychial The mean age was 37.8 years. The highest number epithelium shows digital creases and capillaries of the (49) of patients belonged to the age group 31 to 40 years dermis appear as red dots (40× magnification). (19.6%) and the lowest number of patients (6) belonged to the age group >70 years (2.4%). The youngest patient AIM was 3-year-old and the oldest was 72-year-old. Our aim was to study the characteristic dermoscopic Among 250 patients, 130 (52%) were males and 120 findings in various nail diseases. (48%) were females and M : F ratio was 1.09 : 1. Right hand fingernails were most commonly involved MATERIALS AND METHODS in 192 patients (78.6%) followed by left hand fingernails After obtaining the approval of institutional ethics com- in 171 patients (68.4%), right foot toenails in 109 (43.6%) mittee, a total of 250 consecutive patients were enrolled and left foot toenails in 102 (40.8%). The finger nails were in the study after written informed consent. Ours was a significantly more commonly involved as compared with cross-sectional descriptive study conducted over a period the toe nails (p = 0.000). of 8 months (October 2014 - May 2015). A total of 140 patients (56%) presented with onset Patients with clinically detectable nail unit involve- of disease ranging from 1 month to 1 year. However, ment and belonging to all age groups of both sexes were the shortest duration observed was 1 week and longest included whereas those already on treatment for the duration was 20 years. primary cause and those not wilfully giving consent were The most common occupations observed were excluded. The diagnosis was established by two senior housewives 77 (30.8%) followed by students 58 (23.2%), consultants, based predominantly on the clinical features. domestic helps 40 (16%), and laborers 29 (11.6%). Each patient was subjected to a detailed history regard- Among 80 patients (32%) with systemic diseases, ing demographic profile, onset and evolution of the nail the most commonly observed conditions were diabetes changes, history pertaining to associated skin, and systemic mellitus in 30 patients (34.5%) followed by hyperten- complaints. This was followed by a complete general physi- sion in 29 patients (33.3%) and ischemic heart disease in cal examination and cutaneous and systemic examination. 5 patients (5.7%). Both, underlying dermatological disease Later, dermoscopic evaluation of all the nails was done (Table 1) and nail changes (Table 2) were encountered using a handheld polarized Heine’s Delta 20 dermoscope in 181 patients (72.4%), while nail disease alone was whereas digital dermoscopic images were taken with a observed in 69 patients (27.6%). Canon 1200D DSLR camera. Ultrasound gel was used as the Statistically significant dermoscopic nail findings of linkage fluid between the contact plate and the nail plate. the common nail diseases observed in our study were Statistical Methods used for Analysis as follows: After data collection, it was entered in Microsoft Excel and Nail Psoriasis (n = 45) analyzed using STATA version 13. The quantitative data was presented with the help of mean, standard deviation, More common - circular punctate depressions (64.5 vs median, and interquartile range and the qualitative data 5.9%, p = 0.000) (wherein 64.5% stands for percentage was presented with the help of frequency and percent- of this dermoscopic manifestation seen in nail psoriasis age tables. and 5.9%, that of this manifestation seen in other nail We calculated the means and standard deviations diseases), reddish pink border (62.3 vs 1%, p = 0.000), for the linear variables and proportions for the categori- brown-purple streaks (48.9 vs 21.5%, p = 0.000), white cal variables. The proportions across various categories color (35.6 vs 2%, p = 0.000), nail free edge nonruinous were compared using the Chi-square test or the Fisher’s aspect (35.6 vs 3.5%, p = 0.000), irregular edge (33.4 vs exact test (in those conditions where a very few patients 2%, p = 0.000), dilated capillaries (31.2 vs 1%, p = 0.000), were there in the sample). A p-value of less than 0.05 was linear edge (17.8 vs 5.4%, p = 0.010), proximal nail scaling considered to be statistically significant. (17.8 vs 2%, p = 0.000), and orange pink spot (11.2 vs 0%, p = 0.000). RESULTS Less common - brown color (4.5 vs 25.9%, p = 0.001), After enrolling the patients, the data were collected, tabu- gray black color (2.3 vs 19.6%, p = 0.003), and rough scaly lated, analyzd, and the results obtained were as follows: surface (2.3 vs 11.8%, p = 0.057%).
Recommended publications
  • DERMCASE Test Your Knowledge with Multiple-Choice Cases
    DERMCASE Test your knowledge with multiple-choice cases This month—5 cases: Case 1 1. “What’s wrong with my feet?” 2. “Doc...what is this spot?” 3. “My toenail looks funny!” 4. A foot problem 5. “What’s this rash?” “What’s wrong with my feet?” This 60-year-old woman has had a problem with her feet for several years. What can it be? a. Contact dermatitis b. Pustular psoriasis c. Dyshidrotic eczema d. Mycoses fungoides e. Tinea pedis Answer Pustular psoriasis (answer b) frequently involves the palms and soles. While it does The following have also been used with occur in both sexes, it is most common in mid- varying degrees of success: dle-aged women. It is characterized by recur- • oral etretinate, rent sterile pustules on an erythematous base. • tetracycline, As the pustules dry, they form brown spots. • cyclosporin and The cause of pustular psoriasis is unknown • methotrexate. and once it is established, it can last for years. Cigarette smoking has been associated with There is significant morbidity related to chron- this condition and should be discouraged. ic itch, pain and fissuring. Treatment options consists of: • oil soaks, • emollient creams and ointments, • topical steroids and • retinoid gels. Stanley Wine, MD, FRCPC, is a Dermatologist, Toronto, Ontario. The Canadian Journal of CME / June 2006 63 DERMCASE Case 2 “Doc...what is this spot?” A 42-year-old male presents with a well- defined erythematous atrophic patch with a ker- atotic, raised border. What is it? a. Squamous cell carcinoma b. Bowen’s disease c. Porokeratosis of Mibelli d.
    [Show full text]
  • Pediatric and Adolescent Dermatology
    Pediatric and adolescent dermatology Management and referral guidelines ICD-10 guide • Acne: L70.0 acne vulgaris; L70.1 acne conglobata; • Molluscum contagiosum: B08.1 L70.4 infantile acne; L70.5 acne excoriae; L70.8 • Nevi (moles): Start with D22 and rest depends other acne; or L70.9 acne unspecified on site • Alopecia areata: L63 alopecia; L63.0 alopecia • Onychomycosis (nail fungus): B35.1 (capitis) totalis; L63.1 alopecia universalis; L63.8 other alopecia areata; or L63.9 alopecia areata • Psoriasis: L40.0 plaque; L40.1 generalized unspecified pustular psoriasis; L40.3 palmoplantar pustulosis; L40.4 guttate; L40.54 psoriatic juvenile • Atopic dermatitis (eczema): L20.82 flexural; arthropathy; L40.8 other psoriasis; or L40.9 L20.83 infantile; L20.89 other atopic dermatitis; or psoriasis unspecified L20.9 atopic dermatitis unspecified • Scabies: B86 • Hemangioma of infancy: D18 hemangioma and lymphangioma any site; D18.0 hemangioma; • Seborrheic dermatitis: L21.0 capitis; L21.1 infantile; D18.00 hemangioma unspecified site; D18.01 L21.8 other seborrheic dermatitis; or L21.9 hemangioma of skin and subcutaneous tissue; seborrheic dermatitis unspecified D18.02 hemangioma of intracranial structures; • Tinea capitis: B35.0 D18.03 hemangioma of intraabdominal structures; or D18.09 hemangioma of other sites • Tinea versicolor: B36.0 • Hyperhidrosis: R61 generalized hyperhidrosis; • Vitiligo: L80 L74.5 focal hyperhidrosis; L74.51 primary focal • Warts: B07.0 verruca plantaris; B07.8 verruca hyperhidrosis, rest depends on site; L74.52 vulgaris (common warts); B07.9 viral wart secondary focal hyperhidrosis unspecified; or A63.0 anogenital warts • Keratosis pilaris: L85.8 other specified epidermal thickening 1 Acne Treatment basics • Tretinoin 0.025% or 0.05% cream • Education: Medications often take weeks to work AND and the patient’s skin may get “worse” (dry and red) • Clindamycin-benzoyl peroxide 1%-5% gel in the before it gets better.
    [Show full text]
  • Dermatology DDX Deck, 2Nd Edition 65
    63. Herpes simplex (cold sores, fever blisters) PREMALIGNANT AND MALIGNANT NON- 64. Varicella (chicken pox) MELANOMA SKIN TUMORS Dermatology DDX Deck, 2nd Edition 65. Herpes zoster (shingles) 126. Basal cell carcinoma 66. Hand, foot, and mouth disease 127. Actinic keratosis TOPICAL THERAPY 128. Squamous cell carcinoma 1. Basic principles of treatment FUNGAL INFECTIONS 129. Bowen disease 2. Topical corticosteroids 67. Candidiasis (moniliasis) 130. Leukoplakia 68. Candidal balanitis 131. Cutaneous T-cell lymphoma ECZEMA 69. Candidiasis (diaper dermatitis) 132. Paget disease of the breast 3. Acute eczematous inflammation 70. Candidiasis of large skin folds (candidal 133. Extramammary Paget disease 4. Rhus dermatitis (poison ivy, poison oak, intertrigo) 134. Cutaneous metastasis poison sumac) 71. Tinea versicolor 5. Subacute eczematous inflammation 72. Tinea of the nails NEVI AND MALIGNANT MELANOMA 6. Chronic eczematous inflammation 73. Angular cheilitis 135. Nevi, melanocytic nevi, moles 7. Lichen simplex chronicus 74. Cutaneous fungal infections (tinea) 136. Atypical mole syndrome (dysplastic nevus 8. Hand eczema 75. Tinea of the foot syndrome) 9. Asteatotic eczema 76. Tinea of the groin 137. Malignant melanoma, lentigo maligna 10. Chapped, fissured feet 77. Tinea of the body 138. Melanoma mimics 11. Allergic contact dermatitis 78. Tinea of the hand 139. Congenital melanocytic nevi 12. Irritant contact dermatitis 79. Tinea incognito 13. Fingertip eczema 80. Tinea of the scalp VASCULAR TUMORS AND MALFORMATIONS 14. Keratolysis exfoliativa 81. Tinea of the beard 140. Hemangiomas of infancy 15. Nummular eczema 141. Vascular malformations 16. Pompholyx EXANTHEMS AND DRUG REACTIONS 142. Cherry angioma 17. Prurigo nodularis 82. Non-specific viral rash 143. Angiokeratoma 18. Stasis dermatitis 83.
    [Show full text]
  • Hypertrichosis in Alopecia Universalis and Complex Regional Pain Syndrome
    NEUROIMAGES Hypertrichosis in alopecia universalis and complex regional pain syndrome Figure 1 Alopecia universalis in a 46-year- Figure 2 Hypertrichosis of the fifth digit of the old woman with complex regional complex regional pain syndrome– pain syndrome I affected hand This 46-year-old woman developed complex regional pain syndrome (CRPS) I in the right hand after distor- tion of the wrist. Ten years before, the diagnosis of alopecia areata was made with subsequent complete loss of scalp and body hair (alopecia universalis; figure 1). Apart from sensory, motor, and autonomic changes, most strikingly, hypertrichosis of the fifth digit was detectable on the right hand (figure 2). Hypertrichosis is common in CRPS.1 The underlying mechanisms are poorly understood and may involve increased neurogenic inflammation.2 This case nicely illustrates the powerful hair growth stimulus in CRPS. Florian T. Nickel, MD, Christian Maiho¨fner, MD, PhD, Erlangen, Germany Disclosure: The authors report no disclosures. Address correspondence and reprint requests to Dr. Florian T. Nickel, Department of Neurology, University of Erlangen-Nuremberg, Schwabachanlage 6, 91054 Erlangen, Germany; [email protected] 1. Birklein F, Riedl B, Sieweke N, Weber M, Neundorfer B. Neurological findings in complex regional pain syndromes: analysis of 145 cases. Acta Neurol Scand 2000;101:262–269. 2. Birklein F, Schmelz M, Schifter S, Weber M. The important role of neuropeptides in complex regional pain syndrome. Neurology 2001;57:2179–2184. Copyright © 2010 by
    [Show full text]
  • Evaluation and Treatment of Subungual Hematoma
    20 EMN I October 2010 Evaluation and Treatment of InFocus Subungual Hematoma By James R. Roberts, MD Author Credentials Finan- cial Disclosure: James R. Roberts, MD, is the Chair- man of the Department of Emergency Medicine and the Director of the Divi- sion of Toxicology at Mercy Catholic Medical Center, and a Professor of Emergency Medicine and Toxicology at the Drexel University College of Medicine, both in Philadelphia. Dr. Roberts has disclosed that he is a member of the Speakers Bureau for Merck Pharmaceuticals. He and all other faculty and staff in a position to 12 control the content of this CME activity have disclosed that they and their spouses/life partners (if any) have no fi- nancial relationships with, or financial interests in, any commercial companies pertaining to this educational activity. Learning Objectives: After participat- ing in this activity, the physician should be better able to: 1. Formulate a plan to identify subun- gual hematomas that require simple nail trephination vs. nail removal. 2. Select the correct method of provid- ing nail trephination. 3. Predict the need for prophylactic an- tibiotics after hematoma evacuation. 5 mergency physicians frequently deal with patients who have suf- Efered trauma to the digits. This month’s column begins a series of discus- sions on a rational approach to fingertip problems by reviewing the ubiquitous subungual hematoma (SUH). SUHs are rather common, and cause incapacitating and throbbing pain, prompting the hardiest of souls to seek relief. Even narcotics may fail to relieve the pain produced by an ex- panding subungual hematoma as it compresses the sensitive nailbed so some method to release the pressure is usually required, and is usually imme- 6 7 diately curative.
    [Show full text]
  • Hair That Does Not Grow: Loose Anagen Hair Syndrome Versus Short Anagen Hair Syndrome
    Central Annals of Pediatrics & Child Health Clinical Image *Corresponding author Norma E.Vázquez-Herrera, Hospital Zambrano Hellion Batallón San Patricio 112 Col. Real de San Agustín, San Hair That Does Not Grow: Pedro Garza García, México, Tel: 5281888880650; Email: Submitted: 03 March 2016 Loose Anagen Hair Syndrome Accepted: 29 April 2016 Published: 03 May 2016 Versus Short Anagen Hair Copyright © 2016 Vázquez-Herrera et al. Syndrome OPEN ACCESS Keywords Vázquez-Herrera NE1*, Sharma D2, Tosti A3 • Loose anagen hair syndrome • LAHS 1Departamen of MedicinaInterna, Tecnológico de Monterrey, México • Short anagen hair syndrome 2 Rutgers University, New Jersey Medical School, USA • SAHS 3 Department of Dermatology and Cutaneous Surgery, University of Miami, USA • Trichogram • Alopecia Abstract • Hair disorder • Pediatric hair loss Loose anagen hair syndrome (LAHS) is a hair disorder that is caused by defective • Painless extraction of hair anchorage of the hair shaft to the follicle and primarily affects children. Diagnosis is • Hair that will not grow made clinically by painless plucking of hair that does not grow long and confirmed • Short hair in child by a trichogram with distrophic anagen hairs. Short anagen hair syndrome (SAHS) is another hair condition in which anagen phase has a short duration and as a result, patients present with very short hair and often complain of increased shedding. In this second pathology, pull test shows extraction of telogen hairs with a pointed tip. Both of these diseases must be considered in pediatric patients that present with a complaint of hair that does not grow long. CLINICAL IMAGE encoding for companion layer keratin (K6HF) in patients with LAHS and wooly hair syndrome.
    [Show full text]
  • Alopecia Areata: Evidence-Based Treatments
    Alopecia Areata: Evidence-Based Treatments Seema Garg and Andrew G. Messenger Alopecia areata is a common condition causing nonscarring hair loss. It may be patchy, involve the entire scalp (alopecia totalis) or whole body (alopecia universalis). Patients may recover spontaneously but the disorder can follow a course of recurrent relapses or result in persistent hair loss. Alopecia areata can cause great psychological distress, and the most important aspect of management is counseling the patient about the unpredictable nature and course of the condition as well as the available effective treatments, with details of their side effects. Although many treatments have been shown to stimulate hair growth in alopecia areata, there are limited data on their long-term efficacy and impact on quality of life. We review the evidence for the following commonly used treatments: corticosteroids (topical, intralesional, and systemic), topical sensitizers (diphenylcyclopropenone), psor- alen and ultraviolet A phototherapy (PUVA), minoxidil and dithranol. Semin Cutan Med Surg 28:15-18 © 2009 Elsevier Inc. All rights reserved. lopecia areata (AA) is a chronic inflammatory condition caus- with AA having nail involvement. Recovery can occur spontaneously, Aing nonscarring hair loss. The lifetime risk of developing the although hair loss can recur and progress to alopecia totalis (total loss of condition has been estimated at 1.7% and it accounts for 1% to 2% scalp hair) or universalis (both body and scalp hair). Diagnosis is usu- of new patients seen in dermatology clinics in the United Kingdom ally made clinically, and investigations usually are unnecessary. Poor and United States.1 The onset may occur at any age; however, the prognosis is linked to the presence of other immune diseases, family majority (60%) commence before 20 years of age.2 There is equal history of AA, young age at onset, nail dystrophy, extensive hair loss, distribution of incidence across races and sexes.
    [Show full text]
  • Finger Injuries in Primary Care
    FINGER INJURIES IN PRIMARY CARE www.orthoedu.com © 2020 ORTHOPAEDIC EDUCATIONAL SERVICES, INC. ALL RIGHTS RESERVED Faculty Disclosures • Orthopaedic Educational Services, Inc. Financial Intellectual Property No off-label product discussions American Academy of Physician Assistants Financial Splinting/Casting Workshop Director, Guide to the MSK Galaxy Course • JBJS- JOPA Journal of Orthopaedics for Physician Assistants- Associate Editor • Americian Academy of Surgical Physician Assistants – Editorial Review Board • © 2020 ORTHOPAEDIC EDUCATIONAL SERVICES, INC ALL RIGHTS RESERVED LEARNING OBJECTIVES Attendees will be able to…………… • Recognize and treat Mallet finger injuries • Recognize and treat adult Trigger finger • Recognize and treat Subungual Hematoma & Nail bed injuries • Recognize and treat Superficial Finger infections • Paronychia • Felon • Abscess • Recognize and treat Herpetic Whitlow © 2020 ORTHOPAEDIC EDUCATIONAL SERVICES, INC ALL RIGHTS RESERVED MALLET FINGER DEFORMITY © 2020 ORTHOPAEDIC EDUCATIONAL SERVICES, INC ALL RIGHTS RESERVED Epidemiology MALLET FINGER • “Baseball Finger” • 2 types injury: Soft tissue tendinous vs. Bone avulsion fracture • Pathophysiology • Occurs 2nd to disruption of terminal extensor tendon @ insertion into distal phalanx • Traumatic blow tip of finger causing eccentric flexion @ DIP jt. • Laceration dorsal finger over area to EDC insertion into distal phalanx • All injury mechanisms result in droop at DIP jt. Wieschhoff GG, Sheehan Se, Wortman JR, Et Al, Traumatic Finger Injuries: What the Orthopaedic Surgeon Wants to Know, RadioGraphics, 2016; 36(4):1106-1128 Wang QC, Johnson BA, Fingertip Injuries, Am Fam Physician 2001;63(10): 1961-6 © 2020 ORTHOPAEDIC EDUCATIONAL SERVICES, INC ALL RIGHTS RESERVED MALLET FINGER DEFORMITY Presentation: • obvious droop deformity DIP jt. • Swelling & tenderness dorsal Pictures courtesy T Gocke, PA-C DIP jt. region • Inability to actively extend finger @ DIP jt.
    [Show full text]
  • Hair and Nail Disorders
    Hair and Nail Disorders E.J. Mayeaux, Jr., M.D., FAAFP Professor of Family Medicine Professor of Obstetrics/Gynecology Louisiana State University Health Sciences Center Shreveport, LA Hair Classification • Terminal (large) hairs – Found on the head and beard – Larger diameters and roots that extend into sub q fat LSUCourtesy Health of SciencesDr. E.J. Mayeaux, Center Jr., – M.D.USA Hair Classification • Vellus hairs are smaller in length and diameter and have less pigment • Intermediate hairs have mixed characteristics CourtesyLSU Health of E.J. Sciences Mayeaux, Jr.,Center M.D. – USA Life cycle of a hair • Hair grows at 0.35 mm/day • Cycle is typically as follows: – Anagen phase (active growth) - 3 years – Catagen (transitional) - 2-3 weeks – Telogen (preshedding or rest) about 3 Mon. • > 85% of hairs of the scalp are in Anagen – Lose 75 – 100 hairs a day • Each hair follicle’s cycle is usually asynchronous with others around it LSU Health Sciences Center – USA Alopecia Definition • Defined as partial or complete loss of hair from where it would normally grow • Can be total, diffuse, patchy, or localized Courtesy of E.J. Mayeaux, Jr., M.D. CourtesyLSU of Healththe Color Sciences Atlas of Family Center Medicine – USA Classification of Alopecia Scarring Nonscarring Neoplastic Medications Nevoid Congenital Injury such as burns Infectious Systemic illnesses Genetic (male pattern) (LE) Toxic (arsenic) Congenital Nutritional Traumatic Endocrine Immunologic PhysiologicLSU Health Sciences Center – USA General Evaluation of Hair Loss • Hx is
    [Show full text]
  • Case of Persistent Regrowth of Blond Hair in a Previously Brunette Alopecia Areata Totalis Patient
    Case of Persistent Regrowth of Blond Hair in a Previously Brunette Alopecia Areata Totalis Patient Karla Snider, DO,* John Young, MD** *PGYIII, Silver Falls Dermatology/Western University, Salem, OR **Program Director, Dermatology Residency Program, Silver Falls Dermatology, Salem, OR Abstract We present a case of a brunette, 64-year-old female with no previous history of alopecia areata who presented to our clinic with diffuse hair loss over the scalp. She was treated with triamcinolone acetonide intralesional injections and experienced hair re-growth of initially white hair that then partially re-pigmented to blond at the vertex. Two years following initiation of therapy, she continued to have blond hair growth on her scalp with no dark hair re-growth and no recurrence of alopecia areata. Introduction (CBC), comprehensive metabolic panel (CMP), along the periphery of the occipital, parietal and Alopecia areata (AA) is a fairly common thyroid stimulating hormone (TSH) test and temporal scalp), sisaipho pattern (loss of hair in autoimmune disorder of non-scarring hair loss. antinuclear antibody (ANA) test. All values were the frontal parietotemporal scalp), patchy hair unremarkable, and the ANA was negative. The loss (reticular variant) and a diffuse thinning The disease commonly presents as hair loss from 2 any hair-bearing area of the body. Following patient declined a biopsy. variant. Often, “exclamation point hairs” can be hair loss, it is not rare to see initial growth of A clinical diagnosis of alopecia areata was seen in and around the margins of the hair loss. depigmented or hypopigmented hair in areas made. The patient was treated with 5.0 mg/mL The distal ends of these hairs are thicker than the proximal ends, and they are a marker of active of regrowth in the first anagen cycle.
    [Show full text]
  • Poudre School District Ssn# 21-690-002 Exhibit A
    POUDRE SCHOOL DISTRICT SSN# 21-690-002 EXHIBIT A Poudre School District Employee Health Clinic Service Provision Matrix Included Acute Provided Services Billed to Excluded Illness/Physical Exam Included Acute Injury Services Medical TPA Services Services Professional services in this category Items in this category are not covered include evaluation and treatment of Services in this category are common injuries under the capitated payment and will Specifically common illnesses typical to any typically treated in a primary care office or UC. be billed to the Employee's normal excluded services. primary care practice. Initial visit Visit codes using CPT codes. Initial visit plus PSD medical insurance. All charges This list is not plus any clinically appropriate short- any clinically appropriate short-term f/u will be applied by the medical TPA exhaustive or all- term f/u appointments are also appointments are also included. E.g. Suture according to the applicable plan inclusive. included. 30- day initial pharmacy removal after a laceration repair. benefits. prescription. Acute respiratory illness: URI's, All DME supplies such as crutches, influenza, bronchitis, otitis media, Joint sprains and muscle strains; commercial splints, air casts, wheel Obstetrical Care sinus infections, pharyngitis, strep Minor burn care chairs, walkers. throat, mononucleosis. Initial evaluation, stabilization and treatment Genitourinary problems: Urinary of fractures. (Complex fractures will be Radiology imaging: Technical tract infections, STD's, Vaginitis. referred to PCP or orthopedics at the component (obtaining image) is Hospital Skin disorders: Acne, insect bites, discretion of the provider.) Non-commercial included in capitated rate. Professional Rounding or bee stings, rashes, sunburn, DME supplies needed for fracture care component (interpretation of image) inpatient care of evaluation and initial treatment of including fiberglass and plaster splinting will be performed by a radiologist and any kind.
    [Show full text]
  • Loose Anagen Syndrome in a 2-Year-Old Female: a Case Report and Review of the Literature
    Loose Anagen Syndrome in a 2-year-old Female: A Case Report and Review of the Literature Mathew Koehler, DO,* Anne Nguyen, MS,** Navid Nami, DO*** * Dermatology Resident, 2nd year, Opti-West/College Medical Center, Long Beach, CA ** Medical Student, 4th Year, Western University of Health Sciences, College of Osteopathic Medicine, Pomona, CA *** Dermatology Residency Program Director, Opti-West/College Medical Center, Long Beach, CA Abstract Loose anagen syndrome is a rare condition of abnormal hair cornification leading to excessive and painless loss of anagen hairs from the scalp. The condition most commonly affects young females with blonde hair, but males and those with darker hair colors can be affected. Patients are known to have short, sparse hair that does not need cutting, and hairs are easily and painlessly plucked from the scalp. No known treatment exists for this rare disorder, but many patients improve with age. Case Report neck line. The patient had no notable medical Discussion We present the case of a 27-month-old female history and took no daily medicines. An older Loose anagen syndrome is an uncommon presenting to the clinic with a chief complaint brother and sister had no similar findings. She condition characterized by loosely attached hairs of diffuse hair loss for the last five months. The was growing well and meeting all developmental of the scalp leading to diffuse thinning with poor mother stated that she began finding large clumps milestones. The mother denied any major growth, thus requiring few haircuts. It was first of hair throughout the house, most notably in the traumas, psychologically stressful periods or any described in 1984 by Zaun, who called it “syndrome child’s play area.
    [Show full text]