Spontaneous Cardiac Tamponade
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Pericardial Disease and Other Acquired Heart Diseases
Royal Brompton & Harefield NHS Foundation Trust Pericardial disease and other acquired heart diseases Sylvia Krupickova Exam oriented Echocardiography course, 4th November 2016 Normal Pericardium: 2 layers – fibrous - serous – visceral and parietal layer 2 pericardial sinuses – (not continuous with one another): • Transverse sinus – between in front aorta and pulmonary artery and posterior vena cava superior • Oblique sinus - posterior to the heart, with the vena cava inferior on the right side and left pulmonary veins on the left side Normal pericardium is not seen usually on normal echocardiogram, neither the pericardial fluid Acute Pericarditis: • How big is the effusion? (always measure in diastole) • Where is it? (appears first behind the LV) • Is it causing haemodynamic compromise? Small effusion – <10mm, black space posterior to the heart in parasternal short and long axis views, seen only in systole Moderate – 10-20 mm, more than 25 ml in adult, echo free space is all around the heart throughout the cardiac cycle Large – >20 mm, swinging motion of the heart in the pericardial cavity Pericardiocentesis Constrictive pericarditis Constriction of LV filling by pericardium Restriction versus Constriction: Restrictive cardiomyopathy Impaired relaxation of LV Constriction versus Restriction Both have affected left ventricular filling Constriction E´ velocity is normal as there is no impediment to relaxation of the left ventricle. Restriction E´ velocity is low (less than 5 cm/s) due to impaired filling of the ventricle (impaired relaxation) -
Cardiac Tamponade Management Clinical Guideline
Cardiac Tamponade Management Clinical Guideline V1.0 August 2020 Summary Cardiac Tamponade Management Clinical Guideline V1.0 Page 2 of 20 1. Introduction 1.1 Cardiac tamponade is a clinical syndrome caused by the accumulation of fluid, blood, pus, clots or gas in the pericardial space, resulting in reduced ventricular filling and subsequent haemodynamic compromise. This includes a haemodynamic spectrum ranging from incipient or preclinical tamponade (when pericardial pressure equals right atrial pressure but it is lower than left atrial pressure) to haemodynamic shock with significant reduction of stroke volume and blood pressure, the latter representing a life-threatening medical emergency. 1.2 The diagnosis of cardiac tamponade is essentially a clinical diagnosis requiring echocardiographic confirmation of the initial diagnostic suspicion. In most patients, cardiac tamponade should be diagnosed by clinical examination that typically shows elevated systemic venous pressure, tachycardia, muffled heart sounds and paradoxical arterial pulse. Systemic blood pressure may be normal, decreased, or even elevated. Clinical signs may also include decreased electrocardiographic voltage with electrical alternans and an enlarged cardiac silhouette on chest X-ray with slow-accumulating effusions. 1.3 Once a clinical diagnosis of tamponade is suspected, an echocardiogram should be performed without delay. The diagnosis is then confirmed by echocardiographic demonstration of several 2D and Doppler-based findings (i.e. evidence of pericardial effusion with variable cardiac chambers’ compression, abnormal respiratory variation in tricuspid and mitral valve flow velocities, inferior vena cava plethora). 1.4 This should immediately trigger On-call Consultant Cardiologist review in order to stratify the patient risk, identify specific supportive and monitoring requirements and guide the optimal timing and modality of pericardial drainage. -
Cardiac Tamponade And/Or Pericardiocentesis Following Atrial Fibrillation Ablation – National Quality Strategy Domain: Patient Safety
Quality ID #392 (NQF 2474): HRS-12: Cardiac Tamponade and/or Pericardiocentesis Following Atrial Fibrillation Ablation – National Quality Strategy Domain: Patient Safety 2018 OPTIONS FOR INDIVIDUAL MEASURES: REGISTRY ONLY MEASURE TYPE: Outcome DESCRIPTION: Rate of cardiac tamponade and/or pericardiocentesis following atrial fibrillation ablation. This measure is submitted as four rates stratified by age and gender: • Submission Age Criteria 1: Females 18-64 years of age • Submission Age Criteria 2: Males 18-64 years of age • Submission Age Criteria 3: Females 65 years of age and older • Submission Age Criteria 4: Males 65 years of age and older INSTRUCTIONS: This measure is to be submitted a minimum of once per performance period for patients with atrial fibrillation ablation performed during the performance period. This measure may be submitted by eligible clinicians who perform the quality actions described in the measure based on the services provided and the measure-specific denominator coding. NOTE: Include only patients that have had atrial fibrillation ablation performed by November 30, 2018, for evaluation of cardiac tamponade and/or pericardiocentesis occurring within 30 days within the performance period. This will allow the evaluation of cardiac tamponade and/or pericardiocentesis complications within the performance period. A minimum of 30 cases is recommended by the measure owner to ensure a volume of data that accurately reflects provider performance; however, this minimum number is not required for purposes of QPP submission. This measure will be calculated with 5 performance rates: 1) Females 18-64 years of age 2) Males 18-64 years of age 3) Females 65 years of age and older 4) Males 65 years of age and older 5) Overall percentage of patients with cardiac tamponade and/or pericardiocentesis occurring within 30 days Eligible clinicians should continue to submit the measure as specified, with no additional steps needed to account for multiple performance rates. -
Cardiac Tamponade As the Initial Manifestation of Severe Hypothyroidism: a Case Report
World Journal of Cardiovascular Diseases, 2012, 2, 321-325 WJCD http://dx.doi.org/10.4236/wjcd.2012.24051 Published Online October 2012 (http://www.SciRP.org/journal/wjcd/) Cardiac tamponade as the initial manifestation of severe hypothyroidism: A case report Ronny Cohen1,2*, Pablo Loarte2,3, Simona Opris2, Brooks Mirrer1,2 1NYU School of Medicine, New York, USA 2Division of Cardiology, Woodhull Medical Center, New York, USA 3Division of Nephrology and Hypertension, Brookdale University Hospital and Medical Center, New York, USA Email: *[email protected] Received 11 May 2012; revised 14 June 2012; accepted 23 June 2012 ABSTRACT incidence of pericardial effusion secondary to hypothy- roidism varies in different studies from 30% to 80% [2]. Background: Hypothyroidism is a commonly seen con- Cardiac tamponade as a complication of hypothyroidism dition. The presence of pericardial effusion with car- is very rare [3]. Until 1992, there were less than 30 cases diac tamponade as initial manifestation of this endoc- described and even more recently there are only few rinological condition is very unusual. Objectives: In cases found in the world literature. This low incidence is hypothyroidism pericardial fluid accumulates slowly, most likely due to slow accumulation of fluid and grad- allowing adaptation and stretching of the pericardial ual pericardial distention [4]. Hypothyroidism is cha- sac, sometimes accommodating a large volume. Case racterized by low metabolic demands and therefore, de- Report: A 39 year-old female presented with chest pain, spite a depressed cardiac contractility and cardiac out- dyspnea and lower extremity edema for 1 day. Brady- put, cardiac function remains sufficient to sustain the cardia, muffled heart sounds and severe hyper- tension workload imposed on the heart. -
Cardiac Tamponade: Emergency Management
Cardiac Tamponade: Emergency Management Subject: Emergency management of cardiac tamponade Policy Number N/A Ratified By: Clinical Guidelines Committee Date Ratified: December 2015 Version: 1.0 Policy Executive Owner: Clinical Director, Medicine, Frailty and Networked Service ICSU Designation of Author: Consultant Cardiologist Name of Assurance Committee: As above Date Issued: December 2015 Review Date: 3 years hence Target Audience: Emergency Department, Medicine, Surgery Key Words: Cardiac Tamponade, Pericardiocentesis 1 Version Control Sheet Version Date Author Status Comment 1.0 Dec Dr David Brull Live New guideline. 2015 (Consultant) Rationale: This guideline has been written Dr Akish Luintel as part of the coordinated response to a (Cardiology recent serious incident. Registrar) This guideline is based on current best practice utilising our links to the Barts Heart Centre where all our Tertiary Cardiology is sent 2 Clinical signs of Tamponade – Management algorithm Clinical Signs of Tamponade 1. Tachycardia, tachypnoea 2. Raised JVP, Hypotension & quiet heart sounds (Beck’s Triad) 3. Pulsus Paradoxus 4. Kussmaul’s Sign 5. Hepatomegaly 6. Pericardial rub Medical Emergency: Organise URGENT Echo Bleep Cardiology on 3038/3096 in hours Out of hours Call Bart’s Heart Centre: Barts Heart Electrophysiology SpR 07810 878 450 Cardiology SpR Interventional 07833 237 316 Bart’s Heart Switchboard 0207 377 7000 Management of Tamponade (Monitor in Intensive Care or Coronary Care) Transfer to Barts for Emergency Pericardiocentesis Treat on-site if patient peri-arrest Supportive Management (as required) Do not delay pericardiocentesis Volume expansion Oxygen Inotropes Positive pressure ventilation should be avoided 3 Criteria for use This is a guideline for the emergency management of patients presenting with cardiac tamponade. -
Cath Lab Essentials: “Pericardial Effusion & Tamponade”
Cath Lab Essentials: “Pericardial effusion & tamponade” Pranav M. Patel, MD, FACC, FSCAI Chief, Division of Cardiology Director, Cardiac Cath Lab & CCU University of California, Irvine Division of Cardiology Acknowledgments No financial disclosures Case A 52-year-old man with a 3-day history of progressively worsening dyspnea on exertion to the point that he is unable to walk more than one block without resting. He has had sharp intermittent pleuritic chest pain and a nonproductive cough. He is taking no medications. Case Temp is 37.7 °C (99.9 °F), blood pressure is 88/44 mm Hg, pulse is 125/min, and respiration rate is 29/min; BMI is 27. Oxygen saturation is 95%. Pulsus paradoxus is 15 mm Hg. JVP is 12 cm H2O. Cardiac examination discloses muffled heart sounds with no rubs. Lung auscultation reveals normal breath sounds and no crackles. There is 2+ pedal edema. ECG-electrical alternans Chest X-ray Question What is the most appropriate treatment? A. Dobutamine to increase BP B. Broad spectrum antibiotics C. Pericardiocentesis D. Surgical pericardiectomy Echocardiogram: RV collapse in diastole Most commonly involves the RV outflow tract (more compressible area of RV) Occurs in early diastole, immediately after closure of the pulmonary valve, at the time of opening of the tricuspid valve When collapse extends form outflow tract to the body of the right ventricle, this is evidence that intrapericardial pressure is elevated more substantially https://www.youtube.com/channel/UCPgiLlKxXci7WX8VrZ9g0wQ FN Delling 2007 Subcostal view FN -
Hemopericardium, Cardiac Tamponade, and Liver Abscess in a Young Male Vitorino Modesto Dos Santos,* Lister Arruda Modesto Dos Santos‡
www.medigraphic.org.mx NCT Clinical case Neumol Cir Torax Vol. 76 - Núm. 4:325-328 Octubre-diciembre 2017 Hemopericardium, cardiac tamponade, and liver abscess in a young male Vitorino Modesto dos Santos,* Lister Arruda Modesto dos Santos‡ *Catholic University Medical Course and Armed Forces Hospital, Brasília-DF, Brazil; ‡State Workers Hospital of São Paulo-SP, Brazil. Word received: 24-VII-2017; accepted: 05-X-2017 ABSTRACT. A previously healthy 18-year-old man was admitted with asthenia, fever, shivering, oppressive chest pain, and orthopnea of three-day duration. He had an infected finger wound and lymphangitis on his forearm, self-medicated with topical ointments unsuccessfully. o He was febrile (39 C), hypotensive, with low SpO2 and oliguria, without peripheral edema, hepatojugular reflux or pericardial friction rub. He suddenly had jugular distention, muffled heart sounds and paradoxical pulse, indicating cardiac tamponade, further confirmed. Erythrocyte sedimentation rate, neutrophil-lymphocyte count ratio, C-reactive protein, and procalcitonin) were elevated. Despite of intensive care he had irreversible cardiac arrest. Autopsy revealed hemopericardium causing death by cardiac tamponade and pulmonary edema, in addition to fibrinous pericarditis, hepatic abscess, and acute tubular necrosis. Methicillin-resistant Staphylococcus aureus (MRSA) was found in tissue and blood samples. Eventual tuberculosis coinfection and pericardial involvement by malignancy were ruled out. The role of autopsy to better understand mechanisms of cardiac tamponade is commented. Key words: Cardiac tamponade, hemopericardium, liver abscess, pericarditis. RESUMEN. Un hombre previamente sano de 18 años ingresó al hospital con astenia, fiebre, temblores, dolor torácico opresivo y ortopnea, con duración de tres días. Presentaba una herida infectada en un dedo de la mano y linfangitis en el antebrazo, automedicadas con ungüentos o tópicos sin mejora. -
Cardiac Tamponade Non Invasive Assessment by Echo
Cardiac Tamponade Non Invasive Assessment by Echo Fahmi Othman, MD Cardiology Consultant Non-invasive lab, Heart hospital, Hamad Medical Corporation Declaration of interest • I have nothing to declare Objectives •Introduction •Why Echo is important in cardiac tamponade. • Take home messages Introduction: • Cardiac tamponade is a life-threatening, slow or rapid compression of the heart due to the pericardial accumulation of fluid, pus, blood, clots or gas as a result of inflammation, trauma, rupture of the heart or aortic dissection. • Can be classified based on the: • Onset to (acute, subacute) or (chronic if more than three months). • The size mild (<10 mm), moderate (10–20 mm) or large (>20 mm) • Distribution (circumferential or loculated) • 10-50 ml of pericardial fluid is normally present. • 100 ml of pericardial fluid is enough to cause circumferential effusion. • 300-600 ml of non hemorrhagic pericardial fluid can cause tamponade. Pressure/volume curve of the pericardium with fast accumulating pericardial fluid leading to cardiac tamponade with a smaller volume (A) compared with the slowly accumulating pericardial fluid reaching cardiac tamponade only after larger volumes (B Feb 2018 August 2018 Why Echo is Important in Cardiac Tamponade? •To make the diagnosis •For triage •To guide and monitor the pericardiocentesis Diagnosis Symptoms Of Cardiac Tamponade Sensitivity of the physical examination in the diagnosis of cardiac tamponade Signs of cardiac tamponade Pulsus paradoxus Pulsus paradoxus Sensitivity Of The ECG In The Diagnosis Of Cardiac Tamponade Sensitivity Of The Chest Radiograph In The Diagnosis Of Cardiac Tamponade Echocardiographic Signs Of Cardiac Tamponade Echocardiography in Cardiac Tamponade • Chamber collapse • Doppler Signs of Increased Ventricular Interdependence • Inferior Vena Cava Plethora Chamber collapse • The lower pressure cardiac chambers (atria) are affected before the higher pressure cardiac chambers (ventricles). -
Cardiac Tamponadeafterthrombolysis
Postgrad Med J: first published as 10.1136/pgmj.70.824.455 on 1 June 1994. Downloaded from Postgrad Med J (1994) 70, 455 - 456 © The Fellowship of Postgraduate Medicine, 1994 Cardiac tamponade after thrombolysis T.D. Heymann and W. Culling Department ofMedicine, Kingston Hospital, Kingston upon Thames, Surrey KT2 7QB, UK Summary: Thrombolysis has been very effective in reducing the morbidity and mortality from acute myocardial infarction. Serious adverse events are not uncommon, however. We describe a case in which a haemopericardium and tamponadedeveloped in a patient with a history ofrecurrent idiopathicpericarditis and to whom streptokinase had been administered following a suspected myocardial infarction. The case highlights the need for caution in the administration of thrombolytics to patients with a documented history of pericarditis. Introduction Thrombolysis is the treatment of choice in acute On the following morning the patient's pain myocardial infarction although serious adverse became much more severe. She was dyspnoeic and effects are well recognized and not uncommon.' nauseated. Though examination revealed no objec- Cerebrovascular accidents and gastrointestinal tive deterioration in the patient's clinical state her bleeding are the commoner life-threatening prob- electrocardiogram revealed new T wave inversion lems described. Little attention has been paid to in the anterior leads. Concerned that she was another potentially fatal haemorrhagic complica- suffering a myocardial infarction, streptokinase tion, that of cardiac tamponade. We report a case was administered. Two hours later the patient of tamponade following the administration of became hypotensive. She had a tachycardia, an streptokinase to a patient presenting with severe elevated jugular venous pressure and quiet heart central chest pain. -
Pericardial Disease: Tamponade and Constriction
STATE-OF-THE-ART ECHOCARDIOGRAPHY Pericardial Disease: Tamponade and Constriction FEBRUARY 16, 2016 Sanjiv J. Shah, MD, FASE Associate Professor of Medicine Director, Heart Failure with Preserved EF Program Division of Cardiology, Department of Medicine Northwestern University Feinberg School of Medicine [email protected] N O R T H W E S T E R N U N I V E R S I T Y F E I N B E R G S C H O O L O F M E D I C I N E Questions • In a patient with pericardial effusion, how can I diagnose tamponade (i.e., who needs an urgent pericardiocentesis?) • Why is there no Kussmaul sign in tamponade? Why is there loss of Y-descent? • What are the echo clues to constriction? • Why is septal > lateral e’ in constriction? • In constriction, why does hepatic vein flow reversal increase with expiration but JVP goes up with inspiration (+Kussmaul)? Cardiac tamponade Case presentation • 52-year-old woman with malaise, CP » Low-grade fever, malaise, fatigue x 2 weeks » CP x 1 week, pleuritic, worse when supine, better when sitting forward, positional » No lightheadedness, dizziness, orthopnea, PND, syncope, or palpitations • No prior medical history, no medications Case presentation (continued) • Laboratory work-up: » ANA, rheumatoid factor: normal » Chem panel, CBC: normal » PPD: nonreactive; HIV: negative • Echocardiogram: normal • Prescribed NSAIDs • Symptoms resolved within 2-3 days Case presentation (continued) • 2 weeks later: recurrence of malaise, fatigue, low grade fevers, pleuritic CP; +SOB/dizziness • Physical exam: » 99.8, 98/80, 110, 22, -
Getting to the Heart of Back and Shoulder Pain Kathleen Bradbury-Golas, DNP, APN, NP-C, ACNS-BC Theresa M
LWW/AENJ TME200069 April 26, 2010 11:12 Char Count= 0 Advanced Emergency Nursing Journal Vol. 32, No. 2, pp. 127–134 Copyright c 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins Cases OF NOTE Column Editor: Theresa M. Campo, DNP, RN, APN, NP-C Getting to the Heart of Back and Shoulder Pain Kathleen Bradbury-Golas, DNP, APN, NP-C, ACNS-BC Theresa M. Campo, DNP, RN, APN, NP-C Anthony Chiccarine, DO, FACEP Abstract A healthy male presents to the emergency department with common musculoskeletal complaints that have shown no improvement after 10 days of conservative management. The emergency department provider notes a tachycardia and the patient confirms new onset shortness of breath for 1 day. After a comprehensive workup, the patient is admitted to the hospital. The purpose of this case presentation is to provide advanced practice nurses with information on the manifestations of what is initially felt to be musculoskeletal complaints. This article also emphasizes the need for an astute review of this patient’s “triage” vital signs and other presenting signs and symptoms that will assist the advanced practice nurse in making an accurate diagnosis so as to provide appropriate patient management. Key words: echocardiography, pericarditis, pericardial effusion, pericardial tamponade, tachycardia USCULOSKELETAL COMPLAINTS different diagnosis than one would expect are commonly seen in the quick with back and shoulder pain. Mcare (QC) and emergency care setting. Having a keen awareness of not only CASE FOR REVIEW the patient’s complaint but also the vital signs and other presenting signs and symptoms are A 39-year-old man presented to the emer- of extreme importance. -
Cardiac Tamponade
Otterbein University Digital Commons @ Otterbein Nursing Student Class Projects (Formerly MSN) Student Research & Creative Work Summer 2015 Cardiac Tamponade Ashley Miner Otterbein University, [email protected] Follow this and additional works at: https://digitalcommons.otterbein.edu/stu_msn Part of the Cardiovascular Diseases Commons, Medical Pathology Commons, and the Nursing Commons Recommended Citation Miner, Ashley, "Cardiac Tamponade" (2015). Nursing Student Class Projects (Formerly MSN). 74. https://digitalcommons.otterbein.edu/stu_msn/74 This Project is brought to you for free and open access by the Student Research & Creative Work at Digital Commons @ Otterbein. It has been accepted for inclusion in Nursing Student Class Projects (Formerly MSN) by an authorized administrator of Digital Commons @ Otterbein. For more information, please contact [email protected]. Cardiac Tamponade Ashley Miner, RN, BS Otterbein University, Westerville, Ohio Introduction Pathophysiological Processes Research Topic References References- cont. “Cardiac tamponade (CT) is a life- threatening condition and a medical emergency Implications for Nursing What is the Topic? characterized by pathologic accumulation of fluid in the pericardial sac that compresses The topic the author chose to research Underlying Significance of they myocardium, prevents adequate cardiac filling, and reduces cardiac output” (Schub Care is cardiac tamponade. Cardiac tamponade & Boling, 2015, p.1). Fluid builds up in the pericardial sac, which then causes increased Chandraratna, P. N., Mohar, D. S., & occurs when fluid builds up in the Implications for nursing care include close Schub, T., & Boling, B. (2015, March Pathophysiology Pathophysiology cardiac pressure and leads to compression of the heart. Cardiac tamponade results from Sidarous, P. F. (2014). Role of pericardial cavity, which is the cavity that monitoring of the patient.