Updated and Revised List of Journals in Biotechnology Approved by the Board of Studies in Biotechnology 2015 1
Total Page：16
File Type：pdf, Size：1020Kb
Load more
Recommended publications
-
Neurogenic Decisions Require a Cell Cycle Independent Function of The
RESEARCH ARTICLE Neurogenic decisions require a cell cycle independent function of the CDC25B phosphatase Fre´ de´ ric Bonnet1†, Angie Molina1†, Me´ lanie Roussat1, Manon Azais2, Sophie Bel-Vialar1, Jacques Gautrais2, Fabienne Pituello1*, Eric Agius1* 1Centre de Biologie du De´veloppement, Centre de Biologie Inte´grative, Universite´ de Toulouse, CNRS, UPS, Toulouse, France; 2Centre de Recherches sur la Cognition Animale, Centre de Biologie Inte´grative., Universite´ de Toulouse, CNRS, UPS, Toulouse, France Abstract A fundamental issue in developmental biology and in organ homeostasis is understanding the molecular mechanisms governing the balance between stem cell maintenance and differentiation into a specific lineage. Accumulating data suggest that cell cycle dynamics play a major role in the regulation of this balance. Here we show that the G2/M cell cycle regulator CDC25B phosphatase is required in mammals to finely tune neuronal production in the neural tube. We show that in chick neural progenitors, CDC25B activity favors fast nuclei departure from the apical surface in early G1, stimulates neurogenic divisions and promotes neuronal differentiation. We design a mathematical model showing that within a limited period of time, cell cycle length modifications cannot account for changes in the ratio of the mode of division. Using a CDC25B point mutation that cannot interact with CDK, we show that part of CDC25B activity is independent *For correspondence: of its action on the cell cycle. [email protected] (FP); [email protected] (EA) †These authors contributed equally to this work Introduction In multicellular organisms, managing the development, homeostasis and regeneration of tissues Competing interests: The requires the tight control of self-renewal and differentiation of stem/progenitor cells. -
Resume of Kejin Hu
CURRIULUM VITAE Of Kejin Hu PERSONAL INFORMATION Name: Kejin HU Visa status: USA citizen Language(s): English, Chinese Home city: Vestavia Hills, AL, 35226 RANK/TITLE, Assistant Professor Department: Biochemistry and Molecular Genetics Division: UAB Stem Cell Institute Business Address: SHEL 705, 1825 University Boulevard, Birmingham, AL, 35294 Phone: 205-934-4700 (office); 205-876-8693 (home); 205-703-6688 (cell) Fax: 205-975-3335 MEMBERSHIP: ISSCR (international Society for Stem Cell Research), since 2011 Member of Genetics Society of America (GSA, since 2006) EDUCATIONS June, 1999 to May, 2003, PhD, in marine molecular biology at the Department of Zoology, The University of Hong Kong, Hong Kong, China. July, 1995-October, 1997, MPhil, in fungal biochemistry/microbiology in the Hong Kong Polytechnic University. September, 1981-July, 1985, BSc in botany/agronomy at The Central China (Huazhong) Agricultural University, Wuhan, China. TEACHING EXPERIENCE: Advanced Stem Cell/Regenerative medicine, GBSC 709, Since 2014 SCIENTIFIC ACTIVITIES Ad hoc reviewer for the following journals: 1) Stem Cells; 2) Stem Cells and Development; 3) Stem Cell International; 4) Human Immunology; 5) Molecular Biotechnology; 6) Comparative Biochemistry and Physiology; 7) Journal of Heredity; 8) Scientific Reports; 9) Cellular Reprogramming; 10) Cell and Tissue Research; 11) Science Bulletin; 12) Reproduction, Fertility and Development. 1 Grant Reviewer for 1) Medical Research Council (MRC) of the United Kingdom (remote review, 2015); 2) New York Stem Cell Science (panel meeting from 09/28-09/30, 2016); 3) UAB internal grants SCIENTIFIC/ACADEMIC EXPERIENCE 2011 to present, Assistant Professor, Department of Biochemistry and Molecular Genetics, University of Alabama at Birmingham, Birmingham, AL September, 2007 to July, 2011, Research Associate in human iPSC reprogramming and human pluripotent stem cell biology, and their differentiation into blood lineage, Wisconsin National Primate Research Center, University of Wisconsin, Madison, WI. -
A Physicochemical Perspective of Aging from Single-Cell Analysis Of
TOOLS AND RESOURCES A physicochemical perspective of aging from single-cell analysis of pH, macromolecular and organellar crowding in yeast Sara N Mouton1, David J Thaller2, Matthew M Crane3, Irina L Rempel1, Owen T Terpstra1, Anton Steen1, Matt Kaeberlein3, C Patrick Lusk2, Arnold J Boersma4*, Liesbeth M Veenhoff1* 1European Research Institute for the Biology of Ageing, University of Groningen, University Medical Center Groningen, Groningen, Netherlands; 2Department of Cell Biology, Yale School of Medicine, New Haven, United States; 3Department of Pathology, School of Medicine, University of Washington, Seattle, United States; 4DWI-Leibniz Institute for Interactive Materials, Aachen, Germany Abstract Cellular aging is a multifactorial process that is characterized by a decline in homeostatic capacity, best described at the molecular level. Physicochemical properties such as pH and macromolecular crowding are essential to all molecular processes in cells and require maintenance. Whether a drift in physicochemical properties contributes to the overall decline of homeostasis in aging is not known. Here, we show that the cytosol of yeast cells acidifies modestly in early aging and sharply after senescence. Using a macromolecular crowding sensor optimized for long-term FRET measurements, we show that crowding is rather stable and that the stability of crowding is a stronger predictor for lifespan than the absolute crowding levels. Additionally, in aged cells, we observe drastic changes in organellar volume, leading to crowding on the *For correspondence: micrometer scale, which we term organellar crowding. Our measurements provide an initial [email protected] framework of physicochemical parameters of replicatively aged yeast cells. (AJB); [email protected] (LMV) Competing interest: See Introduction page 19 Cellular aging is a process of progressive decline in homeostatic capacity (Gems and Partridge, Funding: See page 19 2013; Kirkwood, 2005). -
Autophagy in the Endocrine Glands
A WECKMAN and others Autophagy in the endocrine 52:2 R151–R163 Review glands Autophagy in the endocrine glands Andrea Weckman, Antonio Di Ieva, Fabio Rotondo1, Luis V Syro2, Leon D Ortiz3, Kalman Kovacs1 and Michael D Cusimano Division of Neurosurgery, Department of Surgery, St Michael’s Hospital, University of Toronto, Toronto, Ontario, Canada Correspondence 1Division of Pathology, Department of Laboratory Medicine, St Michael’s Hospital, University of Toronto, Toronto, should be addressed Ontario, Canada to A Di Ieva 2Department of Neurosurgery, Hospital Pablo Tobon Uribe and Clinica Medellin, Medellin, Colombia Email 3Division of Neurooncology, Instituto de Cancerologia, Clinic Las Americas, Medellin, Colombia [email protected] Abstract Autophagy is an important cellular process involving the degradation of intracellular Key Words components. Its regulation is complex and while there are many methods available, there is " autophagy currently no single effective way of detecting and monitoring autophagy. It has several " endocrine glands cellular functions that are conserved throughout the body, as well as a variety of different " crinophagy physiological roles depending on the context of its occurrence in the body. Autophagy is also " endocrine diseases involved in the pathology of a wide range of diseases. Within the endocrine system, autophagy has both its traditional conserved functions and specific functions. In the endocrine glands, autophagy plays a critical role in controlling intracellular hormone levels. In peptide-secreting cells of glands such as the pituitary gland, crinophagy, a specific form of autophagy, targets the secretory granules to control the levels of stored hormone. In steroid-secreting cells of glands such as the testes and adrenal gland, autophagy targets the steroid-producing organelles. -
SCIENCE CITATION INDEX EXPANDED - JOURNAL LIST Total Journals: 8631
SCIENCE CITATION INDEX EXPANDED - JOURNAL LIST Total journals: 8631 1. 4OR-A QUARTERLY JOURNAL OF OPERATIONS RESEARCH 2. AAPG BULLETIN 3. AAPS JOURNAL 4. AAPS PHARMSCITECH 5. AATCC REVIEW 6. ABDOMINAL IMAGING 7. ABHANDLUNGEN AUS DEM MATHEMATISCHEN SEMINAR DER UNIVERSITAT HAMBURG 8. ABSTRACT AND APPLIED ANALYSIS 9. ABSTRACTS OF PAPERS OF THE AMERICAN CHEMICAL SOCIETY 10. ACADEMIC EMERGENCY MEDICINE 11. ACADEMIC MEDICINE 12. ACADEMIC PEDIATRICS 13. ACADEMIC RADIOLOGY 14. ACCOUNTABILITY IN RESEARCH-POLICIES AND QUALITY ASSURANCE 15. ACCOUNTS OF CHEMICAL RESEARCH 16. ACCREDITATION AND QUALITY ASSURANCE 17. ACI MATERIALS JOURNAL 18. ACI STRUCTURAL JOURNAL 19. ACM COMPUTING SURVEYS 20. ACM JOURNAL ON EMERGING TECHNOLOGIES IN COMPUTING SYSTEMS 21. ACM SIGCOMM COMPUTER COMMUNICATION REVIEW 22. ACM SIGPLAN NOTICES 23. ACM TRANSACTIONS ON ALGORITHMS 24. ACM TRANSACTIONS ON APPLIED PERCEPTION 25. ACM TRANSACTIONS ON ARCHITECTURE AND CODE OPTIMIZATION 26. ACM TRANSACTIONS ON AUTONOMOUS AND ADAPTIVE SYSTEMS 27. ACM TRANSACTIONS ON COMPUTATIONAL LOGIC 28. ACM TRANSACTIONS ON COMPUTER SYSTEMS 29. ACM TRANSACTIONS ON COMPUTER-HUMAN INTERACTION 30. ACM TRANSACTIONS ON DATABASE SYSTEMS 31. ACM TRANSACTIONS ON DESIGN AUTOMATION OF ELECTRONIC SYSTEMS 32. ACM TRANSACTIONS ON EMBEDDED COMPUTING SYSTEMS 33. ACM TRANSACTIONS ON GRAPHICS 34. ACM TRANSACTIONS ON INFORMATION AND SYSTEM SECURITY 35. ACM TRANSACTIONS ON INFORMATION SYSTEMS 36. ACM TRANSACTIONS ON INTELLIGENT SYSTEMS AND TECHNOLOGY 37. ACM TRANSACTIONS ON INTERNET TECHNOLOGY 38. ACM TRANSACTIONS ON KNOWLEDGE DISCOVERY FROM DATA 39. ACM TRANSACTIONS ON MATHEMATICAL SOFTWARE 40. ACM TRANSACTIONS ON MODELING AND COMPUTER SIMULATION 41. ACM TRANSACTIONS ON MULTIMEDIA COMPUTING COMMUNICATIONS AND APPLICATIONS 42. ACM TRANSACTIONS ON PROGRAMMING LANGUAGES AND SYSTEMS 43. ACM TRANSACTIONS ON RECONFIGURABLE TECHNOLOGY AND SYSTEMS 44. -
Journal List Emerging Sources Citation Index (Web of Science) 2020
JOURNAL TITLE ISSN eISSN PUBSLISHER NAME PUBLISHER ADDRESS 3C EMPRESA 2254‐3376 2254‐3376 AREA INNOVACION & DESARROLLO C/ELS ALZAMORA NO 17, ALCOY, ALICANTE, SPAIN, 03802 3C TECNOLOGIA 2254‐4143 2254‐4143 3CIENCIAS C/ SANTA ROSA 15, ALCOY, SPAIN, 03802 3C TIC 2254‐6529 2254‐6529 AREA INNOVACION & DESARROLLO C/ELS ALZAMORA NO 17, ALCOY, ALICANTE, SPAIN, 03802 3D RESEARCH 2092‐6731 2092‐6731 SPRINGER HEIDELBERG TIERGARTENSTRASSE 17, HEIDELBERG, GERMANY, D‐69121 3L‐LANGUAGE LINGUISTICS LITERATURE‐THE SOUTHEAST ASIAN JOURNAL OF ENGLISH LANGUAGE STUDIES 0128‐5157 2550‐2247 PENERBIT UNIV KEBANGSAAN MALAYSIA PENERBIT UNIV KEBANGSAAN MALAYSIA, FAC ECONOMICS & MANAGEMENT, BANGI, MALAYSIA, SELANGOR, 43600 452 F‐REVISTA DE TEORIA DE LA LITERATURA Y LITERATURA COMPARADA 2013‐3294 UNIV BARCELONA, FACULTAD FILOLOGIA GRAN VIA DE LES CORTS CATALANES, 585, BARCELONA, SPAIN, 08007 AACA DIGITAL 1988‐5180 1988‐5180 ASOC ARAGONESA CRITICOS ARTE ASOC ARAGONESA CRITICOS ARTE, HUESCA, SPAIN, 00000 AACN ADVANCED CRITICAL CARE 1559‐7768 1559‐7776 AMER ASSOC CRITICAL CARE NURSES 101 COLUMBIA, ALISO VIEJO, USA, CA, 92656 A & A PRACTICE 2325‐7237 2325‐7237 LIPPINCOTT WILLIAMS & WILKINS TWO COMMERCE SQ, 2001 MARKET ST, PHILADELPHIA, USA, PA, 19103 ABAKOS 2316‐9451 2316‐9451 PONTIFICIA UNIV CATOLICA MINAS GERAIS DEPT CIENCIAS BIOLOGICAS, AV DOM JOSE GASPAR 500, CORACAO EUCARISTICO, CEP: 30.535‐610, BELO HORIZONTE, BRAZIL, MG, 00000 ABANICO VETERINARIO 2007‐4204 2007‐4204 SERGIO MARTINEZ GONZALEZ TEZONTLE 171 PEDREGAL SAN JUAN, TEPIC NAYARIT, MEXICO, C P 63164 ABCD‐ARQUIVOS -
The Genetic Code: Dissected International Journal of Biology And
International Journal of Biology and Genetics Research Article The Genetic Code: Dissected BOZEGHA WB* Numeration Science Literature, Development Research Project, Nigeria. Received: 04 January, 2021; Accepted: 08 January, 2021; Published: 15 January, 2021 *Corresponding Author: W.B.Bozegha, Numeration Science Literature, Development Research Project, Foubiri Sabagreia, Kolga, Bayelsa State, Nigeria. E-mail address: [email protected] Abstract Statement of the Problem: By genetic code, it is meant the true genetic code of 24 permutation quadruplets, which is a fabric segmented into quadruplet codons comprising the RNA four bases A,U,G,C (Adenine, Uracil, Guanine, Cytosine). Dissection is the gory experience of frogs and rabbits and the like of animals, when they become specimens, rather prisoners of war, in the hands of biologists and zoologists prosecuting anatomical studies in laboratories. To all intents and purposes the 24 quadruplet genetic code is now made such a specimen, though not in the hands of the traditional practitioners of dissection, but under a Numerationist from the perspective of Numeration as science of number and arrangement. Methodology and Theoretical Orientation: Bodies of the animal specimens under dissection are usually cut open with sharp instruments to expose their entrails for visual access in furtherance of anatomical studies. So the 24 quadruplet genetic code whose body is a segmented fabric or string of the RNA four bases A,U,G,C in diverse sequences called codons subjected to separation of its constituent nucleotide base types, though without cutting instruments, because they are amenable to dichotomization in terms of Purines and Pyrimidines. Findings: The 24 quadruplet genetic code is transformed into a two-winged creature upon dissection. -
Targeting Macrophages As a Candidate for Tissue Regeneration
Macrophages and Tissue Regeneration Zhang et al. Curr. Issues Mol. Biol. (2018) 29: 37-48. caister.com/cimb Snapshot: Targeting Macrophages as a Candidate for Tissue Regeneration Jing Zhang1,2,3, Yang Yang4, Zhi Yang4,5, Tian Li4,5 and Fulin Chen1,2,3* Introduction Macrophages, also known as "big eaters" due to 1Lab of Tissue Engineering, College of Life their phagocytic capacity, were first annotated by Sciences, Northwest University, 229 Taibai North Mechnikov in the late 18th century (Fraga et al., Road, Xi'an, 710069, P. R. China 2017; Tauber, 2003; Wan et al., 2017). They are a 2Provincial Key Laboratory of Biotechnology of specific mononuclear cell group abundant in almost Shaanxi, 229 Taibai North Road, Xi'an, 710069, P. every organ of higher animals. In mammals, R. China macrophages can derive from the yolk sac, fetal 3Key Laboratory of Resource Biology and liver, and bone marrow (Geissmann et al., 2010; Biotechnology in Western China Ministry of Wynn et al., 2013). Their differentiation is Education, 229 Taibai North Road, Xi'an, 710069, P. orchestrated by multiple growth factors (GFs) R. China. (Gordon, 2003; Sica et al., 2012). Tissue-resident 4College of Life Sciences, Northwest University, 229 macrophages, usually originating from the bone Taibai North Road, Xi'an, 710069, P. R. China marrow hematopoietic progenitors, has the ability of 5Department of Biomedical Engineering, The Fourth self-renewing (Geissmann et al., 2010; Jenkins et Military Medical University, 169 Changle West Road, al., 2011; Schulz et al., 2012; Davies et al., 2013). Xi'an 710032, China Generally, they are divided into two phenotypes, classically activated M1 macrophages and * Correspondence: [email protected] alternatively activated M2 macrophages, which refer to the state activated by Th1 and Th2 lymphocytes DOI: https://dx.doi.org/10.21775/cimb.029.037 or with interferon-gamma (INF-γ) and interleukin 4 (IL-4), respectively (Goerdt et al., 1999; Gordon, Abstract 2003; Sica et al., 2012). -
Disentangling Gold Open Access
Forthcoming in Glanzel, W., Moed, H.F., Schmoch U., Thelwall, M. (2018). Springer Handbook of Science and Technology Indicators. Springer Disentangling Gold Open Access Daniel Torres-Salinas*, Nicolas Robinson-Garcia** and Henk F. Moed*** * Universidad de Granada (EC3metrics SL y Medialab-UGR) and Universidad de Navarra, Spain ** School of Public Policy, Georgia Institute of Technology, United States *** Visiting scholar, Universidad de Granada, Spain Abstract This chapter focuses on the analysis of current publication trends in gold Open Access (OA). The purpose of the chapter is to develop a full understanding on country patterns, OA journals characteristics and citation differences between gold OA and non-gold OA publications. For this, we will first review current literature regarding Open Access and its relation with its so-called citation advantage. Starting with a chronological perspective we will describe its development, how different countries are promoting OA publishing, and its effects on the journal publishing industry. We will deepen the analysis by investigating the research output produced by different units of analysis. First, we will focus on the production of countries with a special emphasis on citation and disciplinary differences. A point of interest will be identification of national idiosyncrasies and the relation between OA publication and research of local interest. This will lead to our second unit of analysis, OA journals indexed in Web of Science. Here we will deepen on journals characteristics and publisher types to clearly identify factors which may affect citation differences between OA and traditional journals which may not necessarily be derived from the OA factor. Gold OA publishing is being encouraged in many countries as opposed to Green OA. -
Stem Cells, Influenza, Pit Bulls, Darwin, and More
Editorial Top ten in Journal of Biology in 2009: stem cells, influenza, pit bulls, Darwin, and more Miranda Robertson This is a more or less frivolous look at the top ten most however feel able with reasonable confi dence to reject one accessed articles – of any kind – published in Journal of of the most important objections to the policy, which is that Biology this year. Frivolous, because the validity of any if authors are allowed to opt out of re-review of their revised conclusions drawn from this statistic is undermined by manuscripts reviewers may refuse to referee them. We have many considerations. The number of times an article is ac- had no refusals – although it is impossible to say whether cessed does not measure how many people actually read it, this means reviewers are content with the policy, or sim- nor, for most articles, is it any indication of how much it ply don’t read beyond the fi rst paragraph of the request to will be cited (itself an imperfect measure of importance): where the policy is explained. Probably both. it is most likely to refl ect what people think they want to read about. What else? Fifth most accessed, and again both topical and provocative, is Jonathan Howard’s Opinion on why Darwin Even then, there is the problem that more recently pub- didn’t discover Mendel’s laws [7]; and the two research pa- lished articles have had less time to accrue accesses; al- pers in the top ten, Chan et al. on conservation of gene ex- though by far the highest access rates occur in the fi rst three pression in vertebrate tissues [8] and Puigbo et al. -
Activation of Stem Cells in Hepatic Diseases
Edinburgh Research Explorer Activation of stem cells in hepatic diseases Citation for published version: Bird, TG, Lorenzini, S & Forbes, SJ 2008, 'Activation of stem cells in hepatic diseases', Cell and tissue research, vol. 331, no. 1, pp. 283-300. https://doi.org/10.1007/s00441-007-0542-z Digital Object Identifier (DOI): 10.1007/s00441-007-0542-z Link: Link to publication record in Edinburgh Research Explorer Document Version: Peer reviewed version Published In: Cell and tissue research General rights Copyright for the publications made accessible via the Edinburgh Research Explorer is retained by the author(s) and / or other copyright owners and it is a condition of accessing these publications that users recognise and abide by the legal requirements associated with these rights. Take down policy The University of Edinburgh has made every reasonable effort to ensure that Edinburgh Research Explorer content complies with UK legislation. If you believe that the public display of this file breaches copyright please contact [email protected] providing details, and we will remove access to the work immediately and investigate your claim. Download date: 06. Oct. 2021 Europe PMC Funders Group Author Manuscript Cell Tissue Res. Author manuscript; available in PMC 2011 February 06. Published in final edited form as: Cell Tissue Res. 2008 January ; 331(1): 283–300. doi:10.1007/s00441-007-0542-z. Europe PMC Funders Author Manuscripts Activation of stem cells in hepatic diseases T. G. Bird, S. Lorenzini, and S. J. Forbes MRC/University of Edinburgh Centre for Inflammation Research, The Queen’s Medical Research Institute, University of Edinburgh, 47 Little France Crescent, Edinburgh EH16 4TJ, UK Abstract The liver has enormous regenerative capacity. -
Journal of Biology Celebrates Its Fifth Anniversary Biomedcentral
BioMed Central Editorial Journal of Biology celebrates its fifth anniversary Published: 29 June 2007 Journal of Biology 2007, 6:5 The electronic version of this article is the complete one and can be found online at http://jbiol.com/content/6/3/5 © 2007 BioMed Central Ltd Five years ago this month, Journal of have been cited and accessed. Publish- an article by Mark Noble and col- Biology was launched under the guid- ing on average only every two months leagues provided evidence that brain ance of Editor-in-Chief Martin Raff has its perils, however: both authors cells are susceptible to chemotherapy and Editor Theodora Bloom as the and readers told us that they’d be [10]; the article has been downloaded premier biology journal of the open happier to see a journal that wasn’t so more than 5,000 times from the access publisher BioMed Central, the very selective and published more Journal of Biology site with a flurry of publisher of Genome Biology and the often. The journal is now planning to media interest. BMC series of journals. As we cele- build on its success in publishing Of course, for many authors, brate Journal of Biology’s birthday, we high-quality articles and is striving to readers and institutions there is one take this opportunity to reflect on the increase the rate of publication, while measure that matters above all others first five years during which the maintaining a very high standard. in evaluating a journal: the impact journal has published articles of factor, as determined by The Thomson exceptional interest across the full Corporation (ISI).