Clinical and Audiological Features of Ménière's
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Correlations Between Audiogram and Objective Hearing Tests in Sensorineural Hearing Loss
International Tinnitus Journal, Vol. 5, No.2, 107-112 (1999) Correlations Between Audiogram and Objective Hearing Tests in Sensorineural Hearing Loss L. Bishara,1 J. Ben-David,l L. Podoshin,1 M. Fradis,l C.B. Teszler,l H. Pratt,2 T. Shpack,3 H. Feiglin,3 H. Hafner,3 and N. Herlinger2 I Department of Otolaryngology, Head and Neck Surgery, and 3Institute of Audiology, Bnai-Zion Medical Center, and 2Evoked Potentials Laboratory, Technion, Haifa, Israel Abstract: Owing to its subjective nature, behavioral pure-tone audiometry often is an unre liable testing method in uncooperative subjects, and assessing the true hearing threshold be comes difficult. In such cases, objective tests are used for hearing-threshold determination (i.e., auditory brainstem evoked potentials [ABEP] and frequency-specific auditory evoked potentials: slow negative response at 10 msec [SN-1O]). The purpose of this study was to evaluate the correlation between pure-tone audiogram shape and the predictive accuracy of SN-IO and ABEP in normal controls and in patients suf fering from sensorineural hearing loss (SNHL). One-hundred-and-fifty subjects aged 15 to 70, some with normal hearing and the remainder with SNHL, were tested prospectively in a double-blind design. The battery of tests included pure-tone audiometry (air and bone conduction), speech reception threshold, ABEP, and SN- 10. Patients with SNHL were divided into four categories according to audiogram shape (i.e., flat, ascending, descending, and all other shapes). The results showed that ABEP predicts behavioral thresholds at 3 kHz and 4 kHz in cases of high-frequency hearing loss. -
CASE REPORT 48-Year-Old Man
THE PATIENT CASE REPORT 48-year-old man SIGNS & SYMPTOMS – Acute hearing loss, tinnitus, and fullness in the left ear Dennerd Ovando, MD; J. Walter Kutz, MD; Weber test lateralized to the – Sergio Huerta, MD right ear Department of Surgery (Drs. Ovando and Huerta) – Positive Rinne test and and Department of normal tympanometry Otolaryngology (Dr. Kutz), UT Southwestern Medical Center, Dallas; VA North Texas Health Care System, Dallas (Dr. Huerta) Sergio.Huerta@ THE CASE UTSouthwestern.edu The authors reported no A healthy 48-year-old man presented to our otolaryngology clinic with a 2-hour history of potential conflict of interest hearing loss, tinnitus, and fullness in the left ear. He denied any vertigo, nausea, vomiting, relevant to this article. otalgia, or otorrhea. He had noticed signs of a possible upper respiratory infection, including a sore throat and headache, the day before his symptoms started. His medical history was unremarkable. He denied any history of otologic surgery, trauma, or vision problems, and he was not taking any medications. The patient was afebrile on physical examination with a heart rate of 48 beats/min and blood pressure of 117/68 mm Hg. A Weber test performed using a 512-Hz tuning fork lateral- ized to the right ear. A Rinne test showed air conduction was louder than bone conduction in the affected left ear—a normal finding. Tympanometry and otoscopic examination showed the bilateral tympanic membranes were normal. THE DIAGNOSIS Pure tone audiometry showed severe sensorineural hearing loss in the left ear and a poor speech discrimination score. The Weber test confirmed the hearing loss was sensorineu- ral and not conductive, ruling out a middle ear effusion. -
Treatment of Vertebrobasilar Insufficiency– Associated Vertigo with a Fixed Combination of Cinnarizine and Dimenhydrinate
International Tinnitus Journal, Vol. 14, No. 1, 57–67 (2008) Treatment of Vertebrobasilar Insufficiency– Associated Vertigo with a Fixed Combination of Cinnarizine and Dimenhydrinate Volker Otto,1,2 Bernhard Fischer,1,3 Mario Schwarz,4 Wolfgang Baumann,4 and Rudolf Preibisch-Effenberger1 1 Ear, Nose, and Throat Clinic, Otto-von-Guericke University, Magdeburg; 2 Schönebecker Kreiskrankenhaus, Schönebeck; 3 Ear, Nose, and Throat Clinic, Marienhospital Osnabrück; and 4 Department of Clinical Research, Hennig Arzneimittel, Flörsheim am Main, Germany Abstract: Thirty-seven patients suffering from vertigo associated with vertebrobasilar insuf- ficiency participated in our prospective, single-center, double-blind, comparative study. Patients were randomly allocated to treatment with placebo; betahistine (12 mg betahistine dimesylate, one tablet three times daily); or the fixed combination of 20 mg cinnarizine and 40 mg dimen- hydrinate (one tablet three times daily) for 4 weeks. The primary efficacy end point was the decrease of the mean vertigo score (SM), which was based on the patients’ assessments of 12 in- dividual vertigo symptoms after 4 weeks of treatment. Patients treated with the fixed combi- nation showed significantly greater reductions of SM as compared to patients receiving placebo ( p Ͻ .001) or the reference therapy betahistine ( p Ͻ .01). The vestibulospinal parameter lat- eral sway (Unterberger’s test) improved to a significantly greater extent in patients taking the fixed combination as compared to those receiving placebo ( p Ͻ .001). No serious adverse event was reported in any therapy group. The tolerability of the fixed combination was judged as very good or good by 91% (betahistine, 73%; placebo, 82%). In conclusion, the fixed com- bination proved to be statistically more effective than the common antivertiginous drug beta- histine in reducing vertebrobasilar insufficiency–associated vertigo symptoms. -
Multiple Sclerosis Revealed by Intrapontine Axial Lesion of Peripheral Nerves
Central Annals of Otolaryngology and Rhinology Case Report *Corresponding author Sébastien Schmerber, Department of Oto-Rhino- Laryngology, Otology, Neurotology, Auditory implants, Multiple Sclerosis Revealed by Cochlear Implant Centre of the French Alps, University Hospital of Grenoble, CHU A. Michallon, BP 217 38043 Grenoble cedex 09 France, Tel : 33 4 76 76 56 62 ; Fax : Intrapontine Axial Lesion of 33 4 76 76 51 20; Email: Submitted: 11 April 2016 Peripheral Nerves Accepted: 01 August 2016 Published: 03 August 2016 Georges Dumas1, M. Filidoro2, Cindy Colombé1, and Sébastien ISSN: 2379-948X Schmerber1* Copyright 1Department of Otolaryngology-Head and Neck Surgery, Grenoble University Hospital, © 2016 Schmerber et al. France 2Department of ENT, CHR Chambery, France OPEN ACCESS Abstract Keywords • Multiple sclerosis Multiple sclerosis(MS) is most often revealed by motor, sensitive symptoms with • Cranial nerves paresthesia, ocular symptoms, and more seldom by symptoms with rapidly installed • Vertigo hearing loss and vertigo.We report an observation with initialsymptoms of a peripheral • Facial palsyl pathology mimicking a meningo-neuritis. A 22 years old young woman was addressed • hearing loss as anemergency for a sudden peripheral symptomatology associating a sudden right hearing loss with ear fullness,vertigo with vomiting and a right peripheral facial palsy. The patient had a spontaneous nystagmus beating toward the left side suppressed by fixation and sensory neural hearing loss on low frequencies. The Fukuda tests initially deviated toward the right side. The caloric test showed a right hypofunction at 75% and a correlated consistent left preponderance. The head shaking test (HST) and skull vibration induced nystagmus test (SVINT) revealed a left nystagmus. -
Decoding Vestibular Migraine for Easier Diagnosis
International Journal of Otorhinolaryngology and Head and Neck Surgery Vazhipokkil AC et al. Int J Otorhinolaryngol Head Neck Surg. 2019 May;5(3):699-704 http://www.ijorl.com pISSN 2454-5929 | eISSN 2454-5937 DOI: http://dx.doi.org/10.18203/issn.2454-5929.ijohns20191733 Original Research Article Decoding vestibular migraine for easier diagnosis Anoop C. Vazhipokkil*, Ashwini Shenoy Department of ENT, Head and Neck Surgery, Sri Ramachandra Medical College and Research Institute, Chennai, Tamil Nadu, India Received: 02 January 2019 Revised: 22 February 2019 Accepted: 25 February 2019 *Correspondence: Dr. Anoop C. Vazhipokkil, E-mail: [email protected] Copyright: © the author(s), publisher and licensee Medip Academy. This is an open-access article distributed under the terms of the Creative Commons Attribution Non-Commercial License, which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. ABSTRACT Background: Due to the absence of a unified set of diagnostic criteria, vestibular migraine is always an underdiagnosed entity. This study was undertaken to find specific pointers in the diagnostic protocol which can help in diagnosing vestibular migraine and also to assess our treatment module for vestibular migraine. Methods: An elaborate proforma was prepared for evaluating each patient at our vertigo clinic for a time period of two years. A detailed history is followed by general examination, ENT examination, specific tests for eyes, tests for vestibulospinal tract, and also a few more tests such as Doppler, pure tone audiogram, ECHO etc. when the diagnosis was in doubt. A total of 206 patients were evaluated. -
Bedside Neuro-Otological Examination and Interpretation of Commonly
J Neurol Neurosurg Psychiatry: first published as 10.1136/jnnp.2004.054478 on 24 November 2004. Downloaded from BEDSIDE NEURO-OTOLOGICAL EXAMINATION AND INTERPRETATION iv32 OF COMMONLY USED INVESTIGATIONS RDavies J Neurol Neurosurg Psychiatry 2004;75(Suppl IV):iv32–iv44. doi: 10.1136/jnnp.2004.054478 he assessment of the patient with a neuro-otological problem is not a complex task if approached in a logical manner. It is best addressed by taking a comprehensive history, by a Tphysical examination that is directed towards detecting abnormalities of eye movements and abnormalities of gait, and also towards identifying any associated otological or neurological problems. This examination needs to be mindful of the factors that can compromise the value of the signs elicited, and the range of investigative techniques available. The majority of patients that present with neuro-otological symptoms do not have a space occupying lesion and the over reliance on imaging techniques is likely to miss more common conditions, such as benign paroxysmal positional vertigo (BPPV), or the failure to compensate following an acute unilateral labyrinthine event. The role of the neuro-otologist is to identify the site of the lesion, gather information that may lead to an aetiological diagnosis, and from there, to formulate a management plan. c BACKGROUND Balance is maintained through the integration at the brainstem level of information from the vestibular end organs, and the visual and proprioceptive sensory modalities. This processing takes place in the vestibular nuclei, with modulating influences from higher centres including the cerebellum, the extrapyramidal system, the cerebral cortex, and the contiguous reticular formation (fig 1). -
Balance Disorders in Children
Balance disorders in children Prof. Dr. Balasubramanian Thiagarajan (drtbalu) Reassurance plays a vital role Introduction 8% of children 1. Young children don’t usually complain of in the age group of 1-15 vertigo years 2. History can be elusive experienced 3. Diagnosis can be elusive vertigo 4. Other than middle ear disease & congenital or hereditary sensorineural conditions excluded migraine is the condition associated with giddiness 5. Posterior fossa diseases should be considered in older children Cinnarizine If appropriate can be used antimigraine treatments are effective Maturation of the vestibular system Phylogenetically vestibular At 4 months of age the baby system is older than auditory. Otic capsule can tilt its Each stage in development is in develops early in head to keep advance of auditory system gestation between it vertical 4th – 12th week of intrauterine life Vestibular system is the Vestibular first sensory nerve system to myelinates by develop. Full 16 weeks term babies demonstrate doll’s eye response By 24 weeks there is a primitive vestibulo- ocular reflex present Moro reflex is present in normal child Cochlear duct at birth has two and half coils by 50 mm stage Semicircular canals are formed from the utricular portion of otic vesicle by 30 mm stage After Birth Normal ENG values for canal paresis and directional preponderance calculations are 1. Maturation of vestibulospinal & wider than those seen in adults. vestibulo ocular reflexes continues and are maximal at 6-12 months of age Maximum slow phase velocity 2. Bithermal caloric responses can be readings are often similar to demonstrated in 9 month old babies those in adults 3. -
MEDICINE TODAY Audiometry -~60
9 November 196S Schizophrenia-Freeman MEDIALSHRNAL 373 In the Salford comprehensive community mental health ser- FURTHER READING vice vulnerable cases of schizophrenia have been treated with Bennett, D. H., New Aspects of the Mental Health Services, ed. H. L. Frceman and J. Farndale, 1967. Oxford. this preparation for nearly two years and experience has been Brown, G. W., Bone, M., Dalison, B., and Wing, J. K., Schizophrenia gained in over 100 cases. This confirms results from elsewhere and Social Care, 1966. London. Br Med J: first published as 10.1136/bmj.4.5627.373 on 9 November 1968. Downloaded from Kinross-Wright, J., and Charalampous, K. D., Int. 7. Neuropsychiat., that it represents an important step forward in the community 1965, 1, 66. management of schizophrenia. The injections may be given in Psychiatric Hospital Care, ed. H. L. Freeman, 1965. London. Treatment of Mental Disorders in the Community, ed. G. R. Daniel and hospital clinics, at general practitioners' surgeries, or by nurses 1968. London. at the patients' homes. An interested family doctor can H. L. Freeman, certainly make a big contribution to the community care of his schizophrenic patients by undertaking these injections, since it is possible to do a rapid check on the mental state B.M.J. Publications at the same time, or perhaps receive a report from an The following are available from the Publishing Manager, B.M.A. accompanying relative. It may also be necessary to issue House, Tavistock Square, London W.C.1. The prices include regular prescriptions for antiparkinsonian drugs, since side- postage. effects are fairly common, at least in the early stages of the The New Gcneral Practice .. -
56-Questions for Your Audiologist
56 Tips for Home or School Questions For Your Audiologist By: Jill Grattan, Nevada Dual Sensory Impairment Project March, 2011 1. What is my child’s hearing loss in each ear? 2. What is the type of hearing loss my child has (e.g., conductive, sensorineural, mixed)? 3. What type of sounds and noises will he/she have difficulty hearing? 4. Will his/her hearing be affected by noisy environments and background noise (e.g., will he/she hear less in a class- room or restaurant)? 5. What, if any, medical condition does my child have? 6. Does my child have a progressive/degenerative condition? 6a. If yes, how rapidly should one expect changes to occur? 6b. What behaviors might I observe that indicate a change in my child’s hearing? 7. How often should my child visit an audiologist to check his/her hearing? 8. What suggestions do you have for the teacher working with my child? 9. What information should be shared with the people who interact with my child? 10. What assistive listening devices might benefit my child? 11. What adaptations do you think my child might need in the educational setting or at home? 12. What should be expected in terms of daily functioning (e.g., strain, headaches, frustration, etc.)? Screening Questions 1. What does the ‘newborn hearing screening test’ actually screen for? 1a. Can my child pass this test and still be hearing impaired? 2. Tests related to hearing and functioning of the ear: • Impedance testing - Tympanogram; Acoustic Reflex Test • Behavioral Testing - Behavioral Audiome- try; Pure-Tone Audiometry or Pure-Tone • Otoacoustic Emissions Testing (OAEs) Air Conduction Testing; Pure-Tone Bone • Auditory Brainstem Response (ABR) Conduction Testing; Visual Reinforce- • Speech Audiometry - Speech Awareness Threshold (SAT) or ment Audiometry (VRA); Conditioned Speech Detection Threshold (SDT); Speech Reception Thresh- Play Audiometry old or Speech Recognition Threshold (SRT) 3. -
Audiometric Test Procedures
Audiometric Test Procedures 101 This information is meant to help you better understand the various test procedures as well as some of the terms you might see on an audiometric report. By Larry Medwetsky individual could, in fact, exhibit nor- In the previous issue of Hearing mal hearing acuity across these three Loss Magazine, I provided an over- Anyone who has ever had their frequencies, yet, exhibit a significant view concerning hearing threshold hearing tested should know how hearing loss in the higher frequencies results as recorded on the audiogram to read the audiogram, but that’s (3000-8000 Hz). Thus, it is important and an explanation of the pure-tone easier said than done. Hopefully, to examine the SRT in the context of audiogram. In this article, I will after reading this article you will the other audiometric test findings. describe various test procedures have a greater understanding of the Speech Awareness Threshold that are typically administered in principles discussed and use your (SAT): an audiometric evaluation and what knowledge going forward—be it in Compound words are pre- information the tests provide. reviewing hearing test results you sented, the goal being to determine already have or when discussing your the softest level one can detect the Audiometric Test Procedures results at your next hearing test. presence of words. This test is often Pure-tone Audiometry: Tones of used when an individual’s hearing loss different frequencies are presented; the is so great that the person is unable goal is to find the softest sound level relatively flat hearing losses, and the to recognize/repeat the words, yet is which one can hear (threshold) the average of 500 and 1000 Hz for those aware that words have been presented. -
The Functional Hearing Inventory
THE FUNCTIONAL HEARING INVENTORY: CRITERION-RELATED VALIDITY AND INTERRATER RELIABILITY by PAMELA M. BROADSTON, B.S., M.A. A DISSERTATION IN SPECIAL EDUCATION Submitted to the Graduate Faculty of Texas Tech University in Partial Fulfillment of the Requirements for the Degree of DOCTOR OF EDUCATION Approved December, 2003 Copyright 2003, Pamela M. Broadston ACKNOWLEDGEMENTS First and foremost, I thank my Lord, Jesus Christ for opening the door that provided the opportunity for me to obtain this degree. Without His almighty love and endless grace, I would never have achieved this milestone. This milestone could also never have been achieved without the love and support of my family. I cannot proceed without first acknowledging them: to my parents who provided constant love and support throughout this entire endeavor; to my brother Bob, without his financial support I would probably still be working on my master's degrees one class at a time; to my sister, who allowed me to vent and provided sound advice during trying times; to my baby brother, Jeff, thanks for believing in me. I most gratefully thank my dissertation committee for their wisdom, support, and constructive criticism. Their dedication and skilled instruction were vital to the completion of this project. They include: Dr. Carol Layton who provided me with her expertise and guidance in diagnostics and assessment, Dr. Nora Griffm-Shirley who got me hooked on O&M, and Dr. Robert Kennedy who patiently explained and re-explained statistics, time and time again. Last but not least, I want to thank my chair. Dr. Roseanna Davidson, for providing the resources and opportunities that enhanced my doctoral studies and for her expertise and guidance into the field of deafblindness. -
Clinical Policy: Central Auditory Processing Disorder Reference Number: HNCA.CP.MP.375 Effective Date: 10/07 Coding Implications Last Review Date: 3/21 Revision Log
Clinical Policy: Central Auditory Processing Disorder Reference Number: HNCA.CP.MP.375 Effective Date: 10/07 Coding Implications Last Review Date: 3/21 Revision Log See Important Reminder at the end of this policy for important regulatory and legal information. Description Central auditory processing disorder (CAPD), also known as auditory processing disorder (APD), refers to the efficiency and effectiveness by which the central nervous system (CNS) utilizes auditory information in the perceptual processing of auditory information. The diagnosis, management, and even the existence of an auditory-specific perceptual deficit are controversial. Policy/Criteria I. It is the policy of Health Net of California that diagnostic testing and therapy for the management of central auditory processing disorder are considered investigational due to lack of scientific evidence to support the validity of any diagnostic tests and treatment. Background According to the American Speech-Language Hearing Association (ASHA), central auditory processing disorder (CAPD), also known as auditory processing disorder (APD), refers to difficulties in the perceptual processing of auditory information in the CNS as demonstrated by poor performance in one or more of the skills noted above. CAPD It is a complex and heterogeneous group of auditory-specific disorders usually associated with a range of listening and learning deficits. Children or adults suspected of CAPD may exhibit a variety of listening and related complaints such as difficulty understanding speech in noisy environments, following directions, and discriminating (or telling the difference between) similar-sounding speech sounds. The child may have difficulty with spelling, reading, and understanding information presented verbally in a classroom. Some individuals may also have behavioral, emotional or social difficulties.