EPA 2012 Prague, Czech Republic 3-6 March 2012

Total Page:16

File Type:pdf, Size:1020Kb

EPA 2012 Prague, Czech Republic 3-6 March 2012 Beyond Diversity Towards Harmony 20th EUROPEAN CONGRESS OF PSYCHIATRY EPA 2012 Prague, Czech Republic 3-6 March 2012 FINAL PROGRAMME 1 CME Accreditation is being applied for 2 20th EUROPEAN CONGRESS OF PSYCHIATRY EPA 2012 FINAL PROGRAMME EPA SECRETARIAT 15, Avenue de la Liberté 67000 Strasbourg, France Tel. + 33 3 8823 9930 Fax + 33 3 8835 2973 [email protected] CONGRESS SECRETARIAT 1-3 rue de Chantepoulet CH 1211 Geneva 1, Switzerland Tel: +41 22 908 04 88 Fax: +41 22 906 91 40 [email protected] I 20th EUROPEAN CONGRESS OF PSYCHIATRY EPA 2012 Prague, Czech Republic 3-6 March 2012 Table of Contents – to be updated 2 Dear Colleagues & Friends, It is our pleasure to welcome you to the 20th European Congress of Psychiatry taking place 3-6 March 2012, in the beautiful city of Prague, Czech Republic. Following our tradition, the cities we visit for the European Congress of Psychiatry have a special relationship or history connected with psychiatry. Prague is no exception with names such as Sigmund Freud, Jan Jánsk , Arnold Pick and Oskar Fischer. EPA will be celebrating its 20th Congress under the motto “Beyond Diversity Towards Harmony”, as a tribute to the EPA’s growing influence in Europe. Congress highlights will include presentations by outstanding European and international experts in psychiatry. This year’s high-quality, multidisciplinary scientific programme provides a full review of the most important aspects of diagnosis and treatment in psychiatry and of the latest achievements in this field. The scientific programme is composed of Plenary and State-of-the-Art Lectures, Pro & Con Debates, Symposia, Workshops, Free Communication Sessions, a dedicated European Early Career Psychiatrists Programme, CME Academia Courses and Sponsored Symposia organised by leading pharmaceutical companies. As part of EPA’s educational activities, webcasting of sessions as well as ePosters will be available after the Congress. We are pleased to announce that Plenary, State of the Art and European Early Career Psychiatrists’ Conversations Sessions will be recorded and broadcasted via the internet after the congress. Webcasts will be posted on the web allowing you to access and refer to these presentations at any time. ePoster presentations will be available on the website after the congress for outstanding Posters and Free Communication Presentations. We welcome you to the 20th European Congress of Psychiatry in the inspiring city of Prague! Patrice Boyer Cyril Höschl EPA President Chair, Local Organising Committee Chair, Scientific Programme Committee 3 4 EPA 2012 20th EUROPEAN CONGRESS OF PSYCHIATRY Prague, Czech Republic 3-6 March 2012 ACKNOWLEDGEMENTS EPA wishes to express its gratitude to the contribution and support of the following healthcare industries PLATINUM SPONSOR GOLD SPONSORS SILVER SPONSORS 5 GENERAL INFORMATION CONGRESS VENUE Prague Congress Centre 5.května 65, 140 21 Prague 4 Czech Republic, European Union www.kcp.cz LANGUAGE English is the official language of the Congress. REGISTRATION DESKS Registration desks are situated on the 1st floor of the Congress venue and will operate at the following hours: Saturday, 3 March 12:00 – 20:30 Sunday, 4 March 07:00 – 18:15 Monday, 5 March 07:30 – 18:15 Tuesday, 6 March 07:30 – 16:45 HOSPITALITY DESK A Hospitality Desk situated in the Registration area, offering local city information to Congress participants will operate at the following hours: Saturday, 3 March 16:00 – 19:00 Sunday, 4 March 09:00 – 17:00 Monday, 5 March 09:00 – 17:00 Tuesday, 6 March 09:00 – 12:30 EPA BOOTH OPENING HOURS The EPA booth is located in the exhibition area and will be open at the following times: Sunday, 4 March 09:00 – 18:00 Monday, 5 March 09:00 – 16:30 Tuesday, 6 March 09:00 – 16:30 NAME BADGE Upon registration you will receive your name badge. You are kindly requested to wear your badge during the entire Congress in order to have access to the Congress halls and exhibition area. 6 ABSTRACT CD-ROM All accepted abstracts are published in an online supplement of the European Psychiatry Journal as well as a CD-Rom published by Elsevier. Please approach the Lundbeck booth located in the Exhibition area to collect your CD-ROM of abstracts. INTERNET ZONE & WIFI Free internet facilities are available to all Congress participants in the exhibition area, during exhibition opening hours. Please do be considerate of fellow participants when using these facilities. MOBILE PHONES Mobile phones must be switched off or muted during all sessions. NOTICE BOARD/MESSAGES Updates and notifications will be posted during the congress on a notice board near the registration area on the 1st floor of the Congress venue. Participants are also welcome to use this space to place messages for colleagues. SMOKING POLICY Smoking is prohibited throughout the congress venue. Please be considerate of the rules of the venue and your fellow participants. LIABILITY AND INSURANCE The Congress Secretariat and Organisers cannot accept liability for personal accidents or loss of or damage to private property of participants. Participants are advised not to leave their personal belongings unattended in session halls and throughout the congress venue. CARING FOR THE ENVIRONMENT . It is our belief that by employing a Green Culture in the workplace we can duplicate this culture and the responsibilities it entails. Organising green congresses all over Europe is a real challenge as green is not only the onsite conduct but the suppliers we are using, the source of the raw materials, the methods of production, management of waste, shipping vs. using local products and fair management of labor throughout the supply chain. 7 CERTIFICATE OF ATTENDANCE & CONGRESS ACCREDITATION CERTIFICATE OF ATTENDANCE Certificates of attendance will be available at the pre-paid registration stations from 14:00 on Monday, 5 March until the end of sessions on Tuesday, 6 March. Participants are requested to scan the barcode on their name tags at the machine and the certificate will automatically be printed with the participant’s name. Hostesses will be available to assist you. CONGRESS ACCREDITATION The 20th European Congress of Psychiatry is accredited by the European Accreditation Council for Continuing Medical Education (EACCME) to provide the following CME activity for medical specialists. The EACCME is an institution of the European Union of Medical Specialists (UEMS). To learn more about UEMS, please visit www.uems.net The 20th European Congress of Psychiatry is designated for a maximum of 21 hours of European external CME credits. Each medical specialist should claim only those hours of credit that he/she actually spent in the educational activity. EACCME credits are recognised by the American Medical Association towards the Physician’s Recognition Award (PRA). To convert EACCME credit to AMA PRA category 1 credit, contact the AMA. ATTENDANCE TRACKING REPORT Self scanning devices are placed in the registration area. You may approach the scanners and self-scan your badge in order to record your attendance. Only participants who have recorded their attendance will receive a report tracking their daily attendance time-frame at the Congress from the Secretariat after the Congress. CERTIFICATE OF ATTENDANCE AT CME ACADEMIA COURSES CME Academia Courses take place throughout the Congress. Participants of Courses are requested to present their Course ticket at the entrance of the appropriate halls, and complete a Course Evaluation Form at the conclusion of the Course. The complete evaluation forms should be handed to the hostess at the exit of the hall. Certificate of Attendance at CME Academia Courses will be given by the hostess only to participants who submit an Evaluation Form. 8 FINAL PROGRAMME EPA 2012 Scientific Programme Now on your personal Mobile device To access the EPA 2012 Scientific Programme App: http://epa.kenesapp.com EPA Congress http://epa.kenesapp.com/android Information at your finger tips Choose, plan and make the most of your Congress 9 ePOSTERS Following the success of ePosters introduced at EPA2011 held in Vienna for the first time, EPA is pleased to continue this presentation tool to highlight the outstanding posters. Participants and members will be granted access to an advanced ePoster platform, which provides user-friendly access to a large number of selected posters online before, during and after the Congress in an ePoster Archive. The ePoster Archive allows users to browse by poster sections and features educational tools such as searching for specific content, being able to comment on posters and ask questions to the authors. When permitted by the authors, posters will be available for download in PDF format. Abstracts selected for ePoster Presentations will be presented both as a standard poster, as well as in an electronic presentation of about 4 minutes, including 2-3 slides to present the poster. Please refer to the Final Programme for information regarding time and location of the ePoster Presentation Sessions. Free Communication Presentations at the congress will be available as ePosters after the Congress in the ePoster Archive. WEBCASTS EPA is pleased to announce that webcasts from the 20th European Congress of Psychiatry will be recorded to be made available online. Plenary, State-of-the-Art and ECP Conversation Sessions will be recorded and made available after the Congress, offering you up to 11 hours of online educational content. Check the -icon for the sessions to be captured. The webcasts will be made available through the EPA website within 3 weeks after the Congress. 10 INFORMATION FOR PRESENTERS All speakers presenting in sessions are kindly requested to submit their presentations to the Speakers’ Ready Room at least 1 hour before the start of their session. The Speakers’ Ready Room is clearly marked at the Congress venue. If using a PowerPoint (or any other computer) presentation, please note you need to bring it on a CD, a DVD or on a “disk on key” Memory stick (using the USB port in the computer) and load it on one of the Congress’ computers in the Speakers’ Ready Room, at least 1 hour before the start of the session.
Recommended publications
  • History of Alzheimer's Disease
    Print ISSN 1738-1495 / On-line ISSN 2384-0757 Dement Neurocogn Disord 2016;15(4):115-121 / https://doi.org/10.12779/dnd.2016.15.4.115 DND REVIEW History of Alzheimer’s Disease Hyun Duk Yang,1,2 Do Han Kim,1 Sang Bong Lee,3 Linn Derg Young,2,4,5 1Harvard Neurology Clinic, Yongin, Korea 2Brainwise Co. Ltd., Yongin, Korea 3Barun Lab Inc., Yongin, Korea 4Department of Business Administration, Cheongju University, Cheongju, Korea 5Boston Research Institute for Medical Policy, Yongin, Korea As modern society ages rapidly, the number of people with dementia is sharply increasing. Direct medical costs and indirect social costs for dementia patients are also increasing exponentially. However, the lack of social awareness about dementia results in difficulties to dementia patients and their families. So, understanding dementia is the first step to remove or reduce the stigma of dementia patients and promote the health of our community. Alzheimer’s disease is the most common form of dementia. The term, ‘Alzheimer’s disease’ has been used for over 100 years since first used in 1910. With the remarkable growth of science and medical technologies, the techniques for diagnosis and treat- ment of dementia have also improved. Although the effects of the current symptomatic therapy are still limited, dramatic improvement is ex- pected in the future through the continued research on disease modifying strategies at the earlier stage of disease. It is important to look at the past to understand the present and obtain an insight into the future. In this article, we review the etymology and history of dementia and pre- vious modes of recognizing dementia.
    [Show full text]
  • Are the Infectious Roots of Alzheimer's Buried Deep in the Past?
    View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by ZENODO Review Article iMedPub Journals 2017 http://journals.imedpub.com Journal of MPE Molecular Pathological Epidemiology Vol. 3 No. S2: 2 Are the Infectious Roots of Alzheimer’s Dr. Lawrence Broxmeyer Buried Deep in the Past? The N.Y. Institute of Medical Research, New York City, USA Abstract Corresponding author: Dr. Lawrence Broxmeyer, MD Recent literature shows a controversial new push to tie microorganisms to Alzheimer’s disease (AD). Study after study, in which scientists have injected human Alzheimer-diseased brain tissue into mice and other laboratory animals that later [email protected]. developed the disease have left little doubt that Alzheimer’s disease (AD) arises edu from an infectious process. By 2013 Mawanda and Wallace’s “Can Infections Cause Alzheimer’s Disease” struck down some of the commonly entertained pathogens N.Y. Institute of Medical Research, New York for AD such as herpes simplex virus type 1, Chlamydia pneumoniae, and several City, USA. types of spirochetes. Instead they pointed to two prime suspects for Alzheimer’s amyloid-beta deposition: “especially chronic infections like tuberculosis and Tel: 011-718-229-3694 leprosy.” To be sure, it was German neuropathologist Oskar Fischer of the Prague school of Neuropathology, Alzheimer’s great rival, who was the first to suggest that infection might be causative for Alzheimer’s. Fischer’s credentials: he was the co- discoverer of Alzheimer’s disease. His suspected germ was the Streptothrix, today Citation: Broxmeyer L. Are the Infectious Roots classified as Actinomycetes, a rare central nervous system pathogen which at the of Alzheimer’s Buried Deep in the Past? J Mol time was so constantly and consistently mistaken for tuberculosis that Choppen- Path Epidemol.
    [Show full text]
  • Are the Infectious Roots of Alzheimer's Buried Deep in the Past?
    Review Article iMedPub Journals 2017 http://journals.imedpub.com Journal of MPE Molecular Pathological Epidemiology Vol. 2 No. S2: 2 Are the Infectious Roots of Alzheimer’s Dr. Lawrence Broxmeyer Buried Deep in the Past? The N.Y. Institute of Medical Research, New York City, USA Abstract Corresponding author: Dr. Lawrence Broxmeyer, MD Recent literature shows a controversial new push to tie microorganisms to Alzheimer’s disease (AD). Study after study, in which scientists have injected human Alzheimer-diseased brain tissue into mice and other laboratory animals that later [email protected]. developed the disease have left little doubt that Alzheimer’s disease (AD) arises edu from an infectious process. By 2013 Mawanda and Wallace’s “Can Infections Cause Alzheimer’s Disease” struck down some of the commonly entertained pathogens N.Y. Institute of Medical Research, New York for AD such as herpes simplex virus type 1, Chlamydia pneumoniae, and several City, USA. types of spirochetes. Instead they pointed to two prime suspects for Alzheimer’s amyloid-beta deposition: “especially chronic infections like tuberculosis and Tel: 011-718-229-3694 leprosy.” To be sure, it was German neuropathologist Oskar Fischer of the Prague school of Neuropathology, Alzheimer’s great rival, who was the first to suggest that infection might be causative for Alzheimer’s. Fischer’s credentials: he was the co- discoverer of Alzheimer’s disease. His suspected germ was the Streptothrix, today Citation: Broxmeyer L. Are the Infectious Roots classified as Actinomycetes, a rare central nervous system pathogen which at the of Alzheimer’s Buried Deep in the Past? J Mol time was so constantly and consistently mistaken for tuberculosis that Choppen- Path Epidemol.
    [Show full text]
  • Kraepelin Revisited: Schizophrenia from Degeneration to Failed Regeneration
    Molecular Psychiatry (2015) 20, 671–676 © 2015 Macmillan Publishers Limited All rights reserved 1359-4184/15 www.nature.com/mp REVIEW Kraepelin revisited: schizophrenia from degeneration to failed regeneration P Falkai1, MJ Rossner1,2, TG Schulze3, A Hasan1, MM Brzózka1, B Malchow1, WG Honer4 and A Schmitt1,5 One hundred years after its conceptual definition as ‘Dementia Praecox’ by Emil Kraepelin, schizophrenia is still a serious psychiatric illness that affects young adults and leads to disability in at least half of patients. The key treatment issue is partial or non-response, especially of negative symptoms. The illness is also associated with different degrees of cognitive dysfunction, particularly in verbal and working memory; the resulting functional impairment may lead to unemployment and an inability to maintain stable relationships. Patients’ cognitive dysfunction led Kraepelin to the assumption that schizophrenia is a form of juvenile dementia caused by a degenerative process of the human brain. Postmortem studies and a plethora of imaging studies do not support the notion of a degenerative process, but such a process is supported by the recently published, largest genome-wide association study on schizophrenia. More than a 100 hits were described, converging on pathways that have a significant role in dopamine metabolism in immune modulation, calcium signalling and synaptic plasticity. This review suggests that research should focus on animal models based on risk genes like transcription factor 4 and study the effects of exposure to environmental stressors relevant for schizophrenia. The use of relevant end points like pre-pulse inhibition or cognitive dysfunction will allow us to gain an understanding of the molecular pathways in schizophrenia and consequently result in improved treatment options, especially for the disabling aspects of this illness.
    [Show full text]
  • Clinical Aspects of Alzheimer's Disease
    Clinical Biochemistry 72 (2019) 3–6 Contents lists available at ScienceDirect Clinical Biochemistry journal homepage: www.elsevier.com/locate/clinbiochem Review Clinical aspects of Alzheimer's disease T Martina Zvěřováa,b,c a Department of Psychiatry, First Faculty of Medicine, Charles University in Prague and General University Hospital in Prague, Czech Republic b Department of Psychiatry, First Faculty of Medicine, Charles University in Prague and General University Hospital in Prague, Ke Karlovu 11, 121 08 Prague 2, Czech Republic c Na Homolce Hospital, Prague 5, Czech Republic ARTICLE INFO ABSTRACT Keywords: Alzheimer's disease is a progressive, irreversible, incurable, neurodegenerative illness and the most common of Alzheimer's disease the dementing disorders. It starts usually after 60 years of age and may span 8 to 12 years. The continuous and Dementia slow decline caused by this disease, is characterized by cognitive deterioration, loss of functional independence, Plaques changes in behaviour, and expanding needs for care. In the last three decades, the proteins predominating Tangles neuritic plaques and neurofibrillary tangles have been detected and researched: amyloid-beta protein in the Beta amyloid plaques and hyperphosphorylated tau in the tangles. Alzheimer's disease is now considered a long-term process Tau protein Clinical aspects with a slow progress and with a prolonged development of pathological changes that precedes symptoms by years. AD is becoming one of the most problematic and expensive illness for the civilization, also known as “silent threat”. 1. Introduction variant of AD are rarer and atypical variants of Alzheimer's disease with relative preservation of memory. 1.1. History of Alzheimer ‘disease AD is the most common type of dementia in order of occurrence, accounting for 60–70% of all cases.
    [Show full text]
  • Guestbook Guestbook
    GUESTBOOK GUESTBOOK The story of the psychiatric ward in prague with portraits of famous people book Guest Guestbook A FEW INTRODUCTORYG WORDS uestbook As a young assistant I was one day dashing from one place to another through the corridors of our renowned dean‘ s offi ce. An older colleague stopped me and asked: “You come from that psychiatric ward, you might possibly know – do in- stitutions have a soul?“ I don’t know any longer what my answer was at that time, but I defi nitely did not consider it important in the rush of that day. But today I do know that institutions do have a soul. The founders, the heads, “the core“ employ- ees, but also the patients and important “passers by“ form something, that insepa- rably belongs to each institution and creates its atmosphere. Not long ago I heard this metaphor which is supposed to characterise human- kind nowadays. People are sitting on a train that is always accelerating into a blind curve. Not many passengers know where they are coming from and nobody has a clue where they are going. And that is why it does no harm to think about the roots and traditions which together determine the direction and way of our lives. The psychiatric Clinic at Karlov is one of the most important institutions in- fl uencing the development of its discipline. Modern Czech psychiatry was created in this campus. Daniel Tuke of York, the famous English psychiatrist, visited our Clinic in October 1853. In the Guestbook, he made the following comment: ’Vis- ited the Asylum with much pleasure and admired the great cleanliness and good or- der of the institution.
    [Show full text]
  • V. A. Kral, the Montreal Hebrew Old People's Home, and Benign
    V. A. Kral, the Montreal Hebrew Old People’s Home, and benign senescent forgetfulness Jeremia Heinik To cite this version: Jeremia Heinik. V. A. Kral, the Montreal Hebrew Old People’s Home, and benign senes- cent forgetfulness. History of Psychiatry, SAGE Publications, 2006, 17 (3), pp.313-332. 10.1177/0957154X06061600. hal-00570849 HAL Id: hal-00570849 https://hal.archives-ouvertes.fr/hal-00570849 Submitted on 1 Mar 2011 HAL is a multi-disciplinary open access L’archive ouverte pluridisciplinaire HAL, est archive for the deposit and dissemination of sci- destinée au dépôt et à la diffusion de documents entific research documents, whether they are pub- scientifiques de niveau recherche, publiés ou non, lished or not. The documents may come from émanant des établissements d’enseignement et de teaching and research institutions in France or recherche français ou étrangers, des laboratoires abroad, or from public or private research centers. publics ou privés. HPY 17(3) Heinik 26/7/06 16:02 Page 1 History of Psychiatry, 17(3): 313–332 Copyright © 2006 SAGE Publications (London, Thousand Oaks, CA and New Delhi) www.sagepublications.com [200609] DOI: 10.1177/0957154X06061600 V. A. Kral, the Montreal Hebrew Old People’s Home, and benign senescent forgetfulness JEREMIA HEINIK* Ichilov Hospital and Tel Aviv University The term Benign Senescent Forgetfulness, introduced in 1958 by V. A. Kral, constitutes the origin of the concept of Mild Cognitive Impairment (MCI), a widely studied but controversial entity. The ambiguities surrounding MCI warrant a re-assessment of its historical origin. Any attempt at an in-depth investigation of Kral’s works on that subject should begin with a description of the patient population and professional arena in the Montreal Hebrew Old People’s and Sheltering Home, where Kral was a consultant.
    [Show full text]
  • A Century of Confusion in Researching Alzheimer's Disease
    http://ijh.sciedupress.com International Journal of Healthcare 2016, Vol. 2, No. 2 REVIEWS A century of confusion in researching Alzheimer’s disease Mario D. Garrett∗1,2, Ramón Valle1 1School of Social Work, San Diego State University, California, USA 2Department of Psychology, Boğaziçi University, Istanbul, Turkey Received: February 19, 2016 Accepted: April 5, 2016 Online Published: April 14, 2016 DOI: 10.5430/ijh.v2n2p13 URL: http://dx.doi.org/10.5430/ijh.v2n2p13 ABSTRACT More than a century ago Alois Alzheimer published a case study that later evolved into the Amyloid Cascade hypothesis—which assumes that increasing proliferation of plaques and tangles in the brain cause dementia. However, studies involving the removal of plaques—amyloid-β—in patients’ brains resulted in worse cognitive performance, suggesting that plaques cannot solely be the disease. The search then focused on tau misfolded protein. But the evidence is uncertain. This paper suggests a critical history approach to understanding this confusion in Alzheimer’s disease research. Confusion is related to variability in expression of the disease, inaccuracy of clinical diagnostic tools, the relationship to other diseases, and the increasing neurological variance among older adults. The final verdict is that there is an unclear relationship between the biology and the expression of the disease. Alzheimer’s disease may in fact be the expression of another, yet unknown, disease. An often overlooked component in Alzheimer’s disease is white matter in the brain. Although found to be negatively related to dementia and positively related to learning, white matter remains unexamined in most current research. Historical evidence suggests that this was not the case a century ago.
    [Show full text]
  • 2018 Prague Program Book
    International Neuropsychological Society 2018 Mid-Year Meeting Bridging Science and Humanity The International Neuropsychological Society is a multi-disciplinary, non-profit organization dedicated to enhancing communication among the scientific disciplines which contribute to the understanding of – brain-behavior relationships. The Society currently has more than 4700 members throughout the world. July 18 20, 2018, Prague, Czech Republic www.ins2018.org www.ins2018.org FINAL PROGRAMME FLOOR PLAN PRAGUE CONGRESS CENTRE SECOND FLOOR South South Hall 2A Hall 2B Parallel Session Parallel Session Forum Hall Plenary Hall Speakers Ready Room 3 2 Registration 1 Exhibition & Coffee Break area Access to the Open Terrace North Hall Parallel Session Terrace 2A List of Exhibitors: Poster Area 1 Novartis 2 Taylor & Francis 3 INS + Cambridge – 3 – International Neuropsychological Society 2018 Mid-Year Meeting Bridging Science and Humanity, July 18–20, 2018, Prague, Czech Republic TABLE OF CONTENTS WELCOME ADDRESS .........................................................................................................................3 MEETING COMMITTEE .......................................................................................................................4 ABOUT INS ........................................................................................................................................6 ABOUT CENES ...................................................................................................................................8
    [Show full text]
  • Oskar Fischer and the Study of Dementia
    doi:10.1093/brain/awn256 Brain 2009: 132; 1102–1111 | 1102 BRAIN A JOURNAL OF NEUROLOGY OCCASIONAL PAPER Oskar Fischer and the study of dementia Michel Goedert MRC Laboratory of Molecular Biology, Hills Road, Cambridge CB2 0QH, UK Correspondence to: Michel Goedert, MRC Laboratory of Molecular Biology, Hills Road, Cambridge CB2 0QH, UK E-mail: [email protected] The centenary of Alois Alzheimer’s description of the case of Auguste Deter has renewed interest in the early history of dementia research. In his 1907 paper Alzheimer described the presence of plaques and tangles in one case of presenile dementia. In the same year, Oskar Fischer reported neuritic plaques in 12 cases of senile dementia. These were landmark findings in the history of research in dementia because they delineated the clinicopathological entity that is now known as Alzheimer’s disease. Although much has been written about Alzheimer, only little is known about Fischer. The present article discusses Fischer’s work on dementia in the context of his life and time. Keywords: Alzheimer; dementia; Fischer; Kraepelin; presbyophrenia Over the past 2 years, the centenary of Alois Alzheimer’s descrip- had suffered from presenile dementia. Four additional cases of tion of the case of Auguste Deter has been marked repeatedly dementia with plaques and tangles (Bonfiglio, 1908; Perusini, (Jucker et al., 2006; Perry et al., 2006). It seems clear that 1909; Sarteschi, 1909) were known to Kraepelin when he Alzheimer’s name will be linked forever with one of the most was revising his textbook. As discussed before (Amaducci et al., common and feared diseases affecting the elderly population.
    [Show full text]