Application of the International Classification of Diseases to Dentistry and Stomatology
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Benign Cementoblastoma Associated with an Impacted Mandibular Third Molar – Report of an Unusual Case
Case Report Benign Cementoblastoma Associated with an Impacted Mandibular Third Molar – Report of an Unusual Case Chethana Dinakar1, Vikram Shetty2, Urvashi A. Shetty3, Pushparaja Shetty4, Madhvika Patidar5,* 1,3Senior Lecturer, 4Professor & HOD, Department of Oral Pathology and Microbiology, AB Shetty Memorial Institute of Dental Science, Mangaloge, 2Director & HOD, Nittee Meenakshi Institute of Craniofacial Surgery, Mangalore, 5Senior Lecturer, Department of Oral Pathology and Microbiology, Babu Banarasi Das College of Dental Sciences, Lucknow *Corresponding Author: Email: [email protected] ABSTRACT Cementoblastoma is characterized by the formation of cementum-like tissue in direct connection with the root of a tooth. It is a rare lesion constituting less than 1% of all odontogenic tumors. We report a unique case of a large cementoblastoma attached to the lateral root surface of an impacted permanent mandibular third molar in a 33 year old male patient. The association of cementoblastomas with impacted teeth is a rare finding. Key Words: Odontogenic tumor, Cementoblastoma, Impacted teeth, Third molar, Cementum Access this article online opening limited to approximately 10mm. The swelling Quick Response was firm to hard in consistency and tender on palpation. Code: Website: Lymph nodes were not palpable. www.innovativepublication.com On radiographical examination, it showed a large, well circumscribed radiopaque mass attached to the lateral root surface of impacted permanent right mandibular DOI: 10.5958/2395-6194.2015.00005.3 third molar. The mass displayed a radiolucent area at the other end and was seen occupying almost the entire length of the ramus of mandible. The entire lesion was INTRODUCTION surrounded by a thin, uniform radiolucent line (Fig. -
Glossary for Narrative Writing
Periodontal Assessment and Treatment Planning Gingival description Color: o pink o erythematous o cyanotic o racial pigmentation o metallic pigmentation o uniformity Contour: o recession o clefts o enlarged papillae o cratered papillae o blunted papillae o highly rolled o bulbous o knife-edged o scalloped o stippled Consistency: o firm o edematous o hyperplastic o fibrotic Band of gingiva: o amount o quality o location o treatability Bleeding tendency: o sulcus base, lining o gingival margins Suppuration Sinus tract formation Pocket depths Pseudopockets Frena Pain Other pathology Dental Description Defective restorations: o overhangs o open contacts o poor contours Fractured cusps 1 ww.links2success.biz [email protected] 914-303-6464 Caries Deposits: o Type . plaque . calculus . stain . matera alba o Location . supragingival . subgingival o Severity . mild . moderate . severe Wear facets Percussion sensitivity Tooth vitality Attrition, erosion, abrasion Occlusal plane level Occlusion findings Furcations Mobility Fremitus Radiographic findings Film dates Crown:root ratio Amount of bone loss o horizontal; vertical o localized; generalized Root length and shape Overhangs Bulbous crowns Fenestrations Dehiscences Tooth resorption Retained root tips Impacted teeth Root proximities Tilted teeth Radiolucencies/opacities Etiologic factors Local: o plaque o calculus o overhangs 2 ww.links2success.biz [email protected] 914-303-6464 o orthodontic apparatus o open margins o open contacts o improper -
Dry Socket (Alveolar Osteitis): Incidence, Pathogenesis, Prevention and Management
See discussions, stats, and author profiles for this publication at: https://www.researchgate.net/publication/273250883 Dry Socket (Alveolar Osteitis): Incidence, Pathogenesis, Prevention and Management Article · January 2013 CITATIONS READS 4 6,266 4 authors, including: Deepak Viswanath Mahesh kumar R krishnadevaraya college of dental sciences krishnadevaraya college of dental sciences 46 PUBLICATIONS 131 CITATIONS 14 PUBLICATIONS 29 CITATIONS SEE PROFILE SEE PROFILE Some of the authors of this publication are also working on these related projects: AID reviews View project Systematic Reviews View project All content following this page was uploaded by Mahesh kumar R on 08 March 2015. The user has requested enhancement of the downloaded file. GirishREVIEW G Gowda ARTICLE et al Dry Socket (Alveolar Osteitis): Incidence, Pathogenesis, Prevention and Management Girish G Gowda, Deepak Viswanath, Mahesh Kumar, DN Umashankar ABSTRACT registered.12-15 The duration varies from 5 to 10 days Alveolar osteitis (AO) is the most common postoperative depending on the severity of the condition. complication after tooth extraction. The pathophysiology, etiology, prevention and treatment of the alveolar osteitis are ETIOLOGY very essential in oral surgery. The aim of this article is to provide a better basis for clinical management of the condition. In The exact etiology of AO is not well understood. Birn addition, the need for identification and elimination of the risk suggested that the etiology of AO is an increased local factors as well as preventive and symptomatic management of fibrinolysis leading to disintegration of the clot. However, the condition are discussed. several local and systemic factors are known to be Keywords: Alveolar osteitis, Localised osteitis, Septic socket, contributing to the etiology of AO. -
Oral Health in Prevalent Types of Ehlers–Danlos Syndromes
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Ghent University Academic Bibliography J Oral Pathol Med (2005) 34: 298–307 ª Blackwell Munksgaard 2005 Æ All rights reserved www.blackwellmunksgaard.com/jopm Oral health in prevalent types of Ehlers–Danlos syndromes Peter J. De Coster1, Luc C. Martens1, Anne De Paepe2 1Department of Paediatric Dentistry, Centre for Special Care, Paecamed Research, Ghent University, Ghent; 2Centre for Medical Genetics, Ghent University Hospital, Ghent, Belgium BACKGROUND: The Ehlers–Danlos syndromes (EDS) Introduction comprise a heterogenous group of heritable disorders of connective tissue, characterized by joint hypermobility, The Ehlers–Danlos syndromes (EDS) comprise a het- skin hyperextensibility and tissue fragility. Most EDS erogenous group of heritable disorders of connective types are caused by mutations in genes encoding different tissue, largely characterized by joint hypermobility, skin types of collagen or enzymes, essential for normal pro- hyperextensibility and tissue fragility (1) (Fig. 1). The cessing of collagen. clinical features, modes of inheritance and molecular METHODS: Oral health was assessed in 31 subjects with bases differ according to the type. EDS are caused by a EDS (16 with hypermobility EDS, nine with classical EDS genetic defect causing an error in the synthesis or and six with vascular EDS), including signs and symptoms processing of collagen types I, III or V. The distribution of temporomandibular disorders (TMD), alterations of and function of these collagen types are displayed in dental hard tissues, oral mucosa and periodontium, and Table 1. At present, two classifications of EDS are was compared with matched controls. -
Recognizing Benign and Malignant Skin Conditions by Claudia Joy Wingo
Protecting Our Shell: Recognizing Benign and Malignant Skin Conditions By Claudia Joy Wingo Learning Outcomes: Participants who attend this presentation should have: • Learned basic terminology, pathophysiology and methods of diagnosis for a variety of skin lesions. • Gained a basic understanding on the difference of appearance between benign and malignant skin lesions. • Acquired specific herbal protocols in reference to skin conditions and lesions. Skin cancer is the most common form of cancer in the United States with more than 2 million cases diagnosed each year. The large majority of these are slow growing, non-melanoma skin cancers (NMSC) but early detection is important to prevent lesion infiltration, disfigurement and possible loss of function as well as recognition of the rarer but more dangerous melanoma lesions (Mahon SM, 2011). As herbalists, naturopaths and integrative health practitioners we pride ourselves on client-centered care, taking the time to do a thorough and extensive client intake. In this role, it is important that we have the basic skills to recognize and distinguish a variety of skin conditions including cutaneous skin lesions. This pictorial presentation seeks to educate herbal and integrative health practitioners on the appearance of both benign and malignant skin lesions and possible herbal recommendations for the prior. We will briefly cover skin physiology and pathophysiology, methods of clinical diagnosis, associated risk factors and identifying features of a variety of common skin lesions. This in turn, will aid the practitioner in knowing when to refer the client on. Herbal protocols for support and prevention of recurrence as well as case studies will be covered. -
Zeroing in on the Cause of Your Patient's Facial Pain
Feras Ghazal, DDS; Mohammed Ahmad, Zeroing in on the cause MD; Hussein Elrawy, DDS; Tamer Said, MD Department of Oral Health of your patient's facial pain (Drs. Ghazal and Elrawy) and Department of Family Medicine/Geriatrics (Drs. Ahmad and Said), The overlapping characteristics of facial pain can make it MetroHealth Medical Center, Cleveland, Ohio difficult to pinpoint the cause. This article, with a handy at-a-glance table, can help. [email protected] The authors reported no potential conflict of interest relevant to this article. acial pain is a common complaint: Up to 22% of adults PracticE in the United States experience orofacial pain during recommendationS F any 6-month period.1 Yet this type of pain can be dif- › Advise patients who have a ficult to diagnose due to the many structures of the face and temporomandibular mouth, pain referral patterns, and insufficient diagnostic tools. disorder that in addition to Specifically, extraoral facial pain can be the result of tem- taking their medication as poromandibular disorders, neuropathic disorders, vascular prescribed, they should limit disorders, or atypical causes, whereas facial pain stemming activities that require moving their jaw, modify their diet, from inside the mouth can have a dental or nondental cause and minimize stress; they (FIGURE). Overlapping characteristics can make it difficult to may require physical therapy distinguish these disorders. To help you to better diagnose and and therapeutic exercises. C manage facial pain, we describe the most common causes and underlying pathological processes. › Consider prescribing a tricyclic antidepressant for patients with persistent idiopathic facial pain. C Extraoral facial pain Extraoral pain refers to the pain that occurs on the face out- 2-15 Strength of recommendation (SoR) side of the oral cavity. -
Benign Fibro-Osseous Lesions Plus…
“Vision is the art of seeing things invisible.” Jonathan Swift 1667 - 1745 Benign Fibro-osseous Lesions Plus… Steven R. Singer, DDS [email protected] 212.305.5674 Benign Fibro-osseous Lesions Fibrous Dysplasia A group of lesions in which normal bone is Localized change in bone metabolism replaced initially by fibrous connective tissue Normal cancellous bone is replaced by Over time, the lesion is infiltrated by osteoid fibrous connective tissue and cementoid tissue The connective tissue contains varying amounts of abnormal bone with irregular This is a benign and idiopathic process trabeculae Trabeculae are randomly oriented. (Remember that normal trabeculae are aligned to respond to stress) Fibrous Dysplasia Fibrous Dysplasia Lesions may be solitary (monostotic) or Fibrous dysplasia is non-hereditary involve more than one bone (polyostotic) Caused by a mutation in a somatic cell. Monostotic form accounts for 70% of all Extent of lesions depends on the timing of cases the mutation. Polyostotic form is more common in the first If the mutation occurs earlier, the disease decade will be more widespread throughout the M=F except in McCune-Albright syndrome, body. An example is McCune-Albright which is almost exclusively found in females Syndrome 1 Fibrous Dysplasia Fibrous Dysplasia McCune-Albright Syndrome • Monostotic and polyostotic forms usually -Almost exclusively begins in the second decade of life females -Polyostotic fibrous • Slow, painless expansion of the jaws dysplasia • Patients may complain of swelling or have -
2021 Follow-Up After Emergency Department Visits for Non-Traumatic Dental Conditions in Adults
DQA Measure EDF-A-A Effective January 1, 2021 **Please read the DQA Measures User Guide prior to implementing this measure.** DQA Measure Specifications: Administrative Claims-Based Measures Follow-up after Emergency Department Visits for Non-Traumatic Dental Conditions in Adults Description: The percentage of ambulatory care sensitive non-traumatic dental condition emergency department visits among adults aged 18 years and older in the reporting period for which the member visited a dentist within (a) 7 days and (b) 30 days of the ED visit Numerators: Number of ambulatory care sensitive non-traumatic dental condition ED visits in the reporting period for which the member visited a dentist within (a) 7 days (NUM1) and (b) 30 days (NUM2) of the ED visit Denominator: Number of ambulatory care sensitive non-traumatic dental condition ED visits in the reporting period Rates: NUM1/DEN and NUM2/DEN Rationale: The use of emergency departments (EDs) for non-traumatic dental conditions has been a growing public health concern across the United States (US)1,2,3,4,5,6,7,8 with over 2 million visits occurring in 2015.9 The majority of ED visits are semi-urgent (53.8%) or non-urgent (23.9%)10, which can be better managed in an ambulatory care setting. Dental care in an ED setting is not definitive with limited care continuity that ultimately leads to poor oral health outcomes.11,12,13 This process of care measure can be used to assess if the patient had timely follow-up with a dentist for more definitive care. References: 1. -
June 18, 2013 8:30 Am – 11:30 Am
Tuesday – June 18, 2013 8:30 am – 11:30 am Poster Abstracts – Tuesday, June 18, 2013 #1 ORAL LESIONS AS THE PRESENTING MANIFESTATION OF CROHN'S DISEASE V Woo, E Herschaft, J Wang U of Nevada, Las Vegas Crohn’s disease (CD) is an immune-mediated disorder of the gastrointestinal tract which together with ulcerative colitis, comprise the two major types of inflammatory bowel disease (IBD). The underlying etiology has been attributed to defects in mucosal immunity and the intestinal epithelial barrier in a genetically susceptible host, resulting in an inappropriate inflammatory response to intestinal microbes. The lesions of CD can affect any region of the alimentary tract as well as extraintestinal sites such as the skin, joints and eyes. The most common presenting symptoms are periumbilical pain and diarrhea associated with fevers, malaise and anemia. Oral involvement has been termed oral CD and may manifest as lip swelling, cobblestoned mucosa, mucogingivitis and linear ulcerations and fissures. Oral lesions may precede gastrointestinal involvement and can serve as early markers of CD. We describe a 6-year-old male who presented for evaluation of multifocal gingival erythema and swellings. His medical history was unremarkable for gastrointestinal disorders or distress. Histopathologic examination showed multiple well-formed granulomas that were negative for special stains and foreign body material. A diagnosis of granulomatous gingivitis was rendered. The patient was advised to seek consultation with a pediatric gastroenterologist and following colonoscopy, was diagnosed with early stage CD. Timely recognition of the oral manifestations of CD is critical because only a minority of patients will continue to exhibit CD-specific oral lesions at follow-up. -
High Frequency of Allelic Loss in Dysplastic Lichenoid Lesions
0023-6837/00/8002-233$03.00/0 LABORATORY INVESTIGATION Vol. 80, No. 2, p. 233, 2000 Copyright © 2000 by The United States and Canadian Academy of Pathology, Inc. Printed in U.S.A. High Frequency of Allelic Loss in Dysplastic Lichenoid Lesions Lewei Zhang, Xing Cheng, Yong-hua Li, Catherine Poh, Tao Zeng, Robert Priddy, John Lovas, Paul Freedman, Tom Daley, and Miriam P. Rosin Faculty of Dentistry (LZ, Y-HL, CP, RP), University of British Columbia, and BC Cancer Research Centre (MPR), Cancer Control Unit, Vancouver, British Columbia, School of Kinesiology (XC, TZ, MPR), Simon Fraser University, Burnaby, British Columbia, Faculty of Dentistry (JL), Dalhousie University, Halifax, Nova Scotia, and Department of Pathology (TD), University of Western Ontario, London, Ontario, Canada; and The New York Hospital Medical Center of Queens (PF), Flushing, New York SUMMARY: Oral lichen planus (OLP) is a common mucosal condition that is considered premalignant by some, whereas others argue that only lichenoid lesions with epithelial dysplasia are at risk of progressing into oral carcinoma. A recent study from this laboratory used microsatellite analysis to evaluate OLP for loss of heterozygosity (LOH) at loci on three chromosomal arms (3p, 9p, and 17p) (Am J Path 1997;Vol151:Page323-Page327). Loss on these arms is a common event in oral epithelial dysplasia and has been associated with risk of progression of oral leukoplakia to cancer. The data showed that, although dysplastic epithelium demonstrated a high frequency of LOH (40% for mild dysplasia), a significantly lower frequency of LOH was noted in OLP (6%), which is even lower than that in hyperplasia (14%). -
White Lesions of the Oral Cavity and Derive a Differential Diagnosis Four for Various White Lesions
2014 self-study course four course The Ohio State University College of Dentistry is a recognized provider for ADA, CERP, and AGD Fellowship, Mastership and Maintenance credit. ADA CERP is a service of the American Dental Association to assist dental professionals in identifying quality providers of continuing dental education. ADA CERP does not approve or endorse individual courses or instructors, nor does it imply acceptance of credit hours by boards of dentistry. Concerns or complaints about a CE provider may be directed to the provider or to ADA CERP at www.ada.org/goto/cerp. The Ohio State University College of Dentistry is approved by the Ohio State Dental Board as a permanent sponsor of continuing dental education ABOUT this FREQUENTLY asked COURSE… QUESTIONS… Q: Who can earn FREE CE credits? . READ the MATERIALS. Read and review the course materials. A: EVERYONE - All dental professionals in your office may earn free CE contact . COMPLETE the TEST. Answer the credits. Each person must read the eight question test. A total of 6/8 course materials and submit an questions must be answered correctly online answer form independently. for credit. us . SUBMIT the ANSWER FORM Q: What if I did not receive a ONLINE. You MUST submit your confirmation ID? answers ONLINE at: A: Once you have fully completed your p h o n e http://dent.osu.edu/sterilization/ce answer form and click “submit” you will be directed to a page with a . RECORD or PRINT THE 614-292-6737 unique confirmation ID. CONFIRMATION ID This unique ID is displayed upon successful submission Q: Where can I find my SMS number? of your answer form. -
Spectrum of PEX1 and PEX6 Variants in Heimler Syndrome
European Journal of Human Genetics (2016) 24, 1565–1571 Official Journal of The European Society of Human Genetics www.nature.com/ejhg ARTICLE Spectrum of PEX1 and PEX6 variants in Heimler syndrome Claire EL Smith1, James A Poulter1, Alex V Levin2,3,4, Jenina E Capasso4, Susan Price5, Tamar Ben-Yosef6, Reuven Sharony7, William G Newman8,9, Roger C Shore10, Steven J Brookes10, Alan J Mighell1,11,12 and Chris F Inglehearn*,1,12 Heimler syndrome (HS) consists of recessively inherited sensorineural hearing loss, amelogenesis imperfecta (AI) and nail abnormalities, with or without visual defects. Recently HS was shown to result from hypomorphic mutations in PEX1 or PEX6,both previously implicated in Zellweger Syndrome Spectrum Disorders (ZSSD). ZSSD are a group of conditions consisting of craniofacial and neurological abnormalities, sensory defects and multi-organ dysfunction. The finding of HS-causing mutations in PEX1 and PEX6 shows that HS represents the mild end of the ZSSD spectrum, though these conditions were previously thought to be distinct nosological entities. Here, we present six further HS families, five with PEX6 variants and one with PEX1 variants, and show the patterns of Pex1, Pex14 and Pex6 immunoreactivity in the mouse retina. While Ratbi et al. found more HS-causing mutations in PEX1 than in PEX6, as is the case for ZSSD, in this cohort PEX6 variants predominate, suggesting both genes play a significant role in HS. The PEX6 variant c.1802G4A, p.(R601Q), reported previously in compound heterozygous state in one HS and three ZSSD cases, was found in compound heterozygous state in three HS families.