R Jogi – Basic Ophthalmology, 4Th Edition
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ISSN: 2320-5407 Int
ISSN: 2320-5407 Int. J. Adv. Res. 6(9), 979-984 Journal Homepage: - www.journalijar.com Article DOI: 10.21474/IJAR01/7759 DOI URL: http://dx.doi.org/10.21474/IJAR01/7759 RESEARCH ARTICLE CORNEAL ALTERATION IN EYES WITH PSEUDOEXFOLIATION SYNDROME. Dr. Tania Sadiq1, Prof. Dr. Syed Tariq Qureshi2, Dr. Arshi Nazir3 and Dr Anshulee Sood4. 1. Postgraduate Scholar, Department of ophthalmology government medical college, srinagar. 2. Professor and Head, Department of ophthalmology government medical college, srinagar. 3. Registrar, Department of ophthalmology government medical college, srinagar. 4. Fellow, Department of ophthalmology government medical college, srinagar. …………………………………………………………………………………………………….... Manuscript Info Abstract ……………………. ……………………………………………………………… Manuscript History Background: Pseudoexfoliation syndrome (PXS) is an age-related systemic microfibrillopathy, caused by gradual deposition of Received: 24 July 2018 extracellular grey and white material over various tissues .In Final Accepted: 30 August 2018 pseudoexfoliation eyes, corneal endothelial changes are Published: September 2018 noted.Objective of Our study was to find corneal alterations among Keywords:- patients of Pseudoexfoliation syndrome of Kashmir region. Corneal Endothelium, Corneal Material And Methods:After obtaining the ethical clearance from the Endothelial cell density,Central Corneal institutional ethical committee,150 patients withPseudoexfoliation Thickness, , Pseudoexfoliation . were included in our Descriptive(Observational )study. Thorough ocular evaluation was done and corneal changes were noted including corneal endothelial cell density and Central corneal thickness using NON CONTACT specular microscope .Appropriate statistical tests were used for analyzing data. Results:Pseudoexfoliation was predominantly seen in males . Mean Central corneal thickness in Pseudoexfoliation with glaucoma eyes was(509.6+13.73μ) which was lower when compared with mean central corneal thickness in Pseudoexfoliation without glaucoma eyes (523.5+17.15 μ). -
Ophthalmic Pathologies in Female Subjects with Bilateral Congenital Sensorineural Hearing Loss
Turkish Journal of Medical Sciences Turk J Med Sci (2016) 46: 139-144 http://journals.tubitak.gov.tr/medical/ © TÜBİTAK Research Article doi:10.3906/sag-1411-82 Ophthalmic pathologies in female subjects with bilateral congenital sensorineural hearing loss 1, 2 3 4 5 Mehmet Talay KÖYLÜ *, Gökçen GÖKÇE , Güngor SOBACI , Fahrettin Güven OYSUL , Dorukcan AKINCIOĞLU 1 Department of Ophthalmology, Tatvan Military Hospital, Bitlis, Turkey 2 Department of Ophthalmology, Kayseri Military Hospital, Kayseri, Turkey 3 Department of Ophthalmology, Faculty of Medicine, Hacettepe University, Ankara, Turkey 4 Department of Public Health, Gülhane Military Medical School, Ankara, Turkey 5 Department of Ophthalmology, Gülhane Military Medical School, Ankara, Turkey Received: 15.11.2014 Accepted/Published Online: 24.04.2015 Final Version: 05.01.2016 Background/aim: The high prevalence of ophthalmologic pathologies in hearing-disabled subjects necessitates early screening of other sensory deficits, especially visual function. The aim of this study is to determine the frequency and clinical characteristics of ophthalmic pathologies in patients with congenital bilateral sensorineural hearing loss (SNHL). Materials and methods: This descriptive study is a prospective analysis of 78 young female SNHL subjects who were examined at a tertiary care university hospital with a detailed ophthalmic examination, including electroretinography (ERG) and visual field tests as needed. Results: The mean age was 19.00 ± 1.69 years (range: 15 to 24 years). A total of 39 cases (50%) had at least one ocular pathology. Refractive errors were the leading problem, found in 35 patients (44.9%). Anterior segment examination revealed heterochromia iridis or Waardenburg syndrome in 2 cases (2.56%). -
Vision Screening Training
Vision Screening Training Child Health and Disability Prevention (CHDP) Program State of California CMS/CHDP Department of Health Care Services Revised 7/8/2013 Acknowledgements Vision Screening Training Workgroup – comprising Health Educators, Public Health Nurses, and CHDP Medical Consultants Dr. Selim Koseoglu, Pediatric Ophthalmologist Local CHDP Staff 2 Objectives By the end of the training, participants will be able to: Understand the basic anatomy of the eye and the pathway of vision Understand the importance of vision screening Recognize common vision disorders in children Identify the steps of vision screening Describe and implement the CHDP guidelines for referral and follow-up Properly document on the PM 160 vision screening results, referrals and follow-up 3 IMPORTANCE OF VISION SCREENING 4 Why Screen for Vision? Early diagnosis of: ◦ Refractive Errors (Nearsightedness, Farsightedness) ◦ Amblyopia (“lazy eye”) ◦ Strabismus (“crossed eyes”) Early intervention is the key to successful treatment 5 Why Screen for Vision? Vision problems often go undetected because: Young children may not realize they cannot see properly Many eye problems do not cause pain, therefore a child may not complain of discomfort Many eye problems may not be obvious, especially among young children The screening procedure may have been improperly performed 6 Screening vs. Diagnosis Screening Diagnosis 1. Identifies children at 1. Identifies the child’s risk for certain eye eye condition conditions or in need 2. Allows the eye of a professional -
Hereditary Reversion Pigmentation of the Eyelids with Heterochromia of the Iris
874 LEE MASTEN FRANCIS enucleated. The following report on the Other cells were round with hyper- specimen was submitted from the New chromatic nuclei; while scattered thruout York State Institute for the Study of the tumor were large deeply staining Malignant Diseases: cells with one or two nuclei but free from The gross appearance of a cross sec- pigment. There were apparently two tion of the eye shows a tumor lying in types of pigmented cells, the one a large the lower temporal quadrant of the eye, irregular cell with long protoplasmic evidently springing from the choroid processes densely filled with fine yellow- near the margin of the optic disc. This ish granules, evidently chromatophores. tumor measured 15x10 mm. and was The other type of pigmented cell was a slightly nodular irregular ovoid tumor. evidently a tumor cell of the type men- The surface appeared smooth, was dark tioned above but containing fewer gran- gray in color and was of a soft consist- ules than the chromatophores. Thruout ency. The retina was markedly detached the tumor were small areas of hemor- and contained a clear serous fluid. Cross rhage and between the cells could be section of the tumor mass showed a demonstrated here and there, free pig- deeply pigmented homogeneous surface. ment granules. Microscopically, the tumor varied as From this picture, we would make a to the cellular constituents. There were diagnosis of malignant melanoma, fre- areas showing many pigment cells and quently called melanosarcoma, but by other areas almost free from the same. some authorities considered as melanotic The tumor was very vascular showing many fine capillaries around which in carcinoma. -
RETINAL DISORDERS Eye63 (1)
RETINAL DISORDERS Eye63 (1) Retinal Disorders Last updated: May 9, 2019 CENTRAL RETINAL ARTERY OCCLUSION (CRAO) ............................................................................... 1 Pathophysiology & Ophthalmoscopy ............................................................................................... 1 Etiology ............................................................................................................................................ 2 Clinical Features ............................................................................................................................... 2 Diagnosis .......................................................................................................................................... 2 Treatment ......................................................................................................................................... 2 BRANCH RETINAL ARTERY OCCLUSION ................................................................................................ 3 CENTRAL RETINAL VEIN OCCLUSION (CRVO) ..................................................................................... 3 Pathophysiology & Etiology ............................................................................................................ 3 Clinical Features ............................................................................................................................... 3 Diagnosis ......................................................................................................................................... -
Strabismus, Amblyopia & Leukocoria
Strabismus, Amblyopia & Leukocoria [ Color index: Important | Notes: F1, F2 | Extra ] EDITING FILE Objectives: ➢ Not given. Done by: Jwaher Alharbi, Farrah Mendoza. Revised by: Rawan Aldhuwayhi Resources: Slides + Notes + 434 team. NOTE: F1& F2 doctors are different, the doctor who gave F2 said she is in the exam committee so focus on her notes Amblyopia ● Definition Decrease in visual acuity of one eye without the presence of an organic cause that explains that decrease in visual acuity. He never complaints of anything and his family never noticed any abnormalities ● Incidence The most common cause of visual loss under 20 years of life (2-4% of the general population) ● How? Cortical ignorance of one eye. This will end up having a lazy eye ● binocular vision It is achieved by the use of the two eyes together so that separate and slightly dissimilar images arising in each eye are appreciated as a single image by the process of fusion. It’s importance 1. Stereopsis 2. Larger field If there is no coordination between the two eyes the person will have double vision and confusion so as a compensatory mechanism for double vision the brain will cause suppression. The visual pathway is a plastic system that continues to develop during childhood until around 6-9 years of age. During this time, the wiring between the retina and visual cortex is still developing. Any visual problem during this critical period, such as a refractive error or strabismus can mess up this developmental wiring, resulting in permanent visual loss that can't be fixed by any corrective means when they are older Why fusion may fail ? 1. -
Genes in Eyecare Geneseyedoc 3 W.M
Genes in Eyecare geneseyedoc 3 W.M. Lyle and T.D. Williams 15 Mar 04 This information has been gathered from several sources; however, the principal source is V. A. McKusick’s Mendelian Inheritance in Man on CD-ROM. Baltimore, Johns Hopkins University Press, 1998. Other sources include McKusick’s, Mendelian Inheritance in Man. Catalogs of Human Genes and Genetic Disorders. Baltimore. Johns Hopkins University Press 1998 (12th edition). http://www.ncbi.nlm.nih.gov/Omim See also S.P.Daiger, L.S. Sullivan, and B.J.F. Rossiter Ret Net http://www.sph.uth.tmc.edu/Retnet disease.htm/. Also E.I. Traboulsi’s, Genetic Diseases of the Eye, New York, Oxford University Press, 1998. And Genetics in Primary Eyecare and Clinical Medicine by M.R. Seashore and R.S.Wappner, Appleton and Lange 1996. M. Ridley’s book Genome published in 2000 by Perennial provides additional information. Ridley estimates that we have 60,000 to 80,000 genes. See also R.M. Henig’s book The Monk in the Garden: The Lost and Found Genius of Gregor Mendel, published by Houghton Mifflin in 2001 which tells about the Father of Genetics. The 3rd edition of F. H. Roy’s book Ocular Syndromes and Systemic Diseases published by Lippincott Williams & Wilkins in 2002 facilitates differential diagnosis. Additional information is provided in D. Pavan-Langston’s Manual of Ocular Diagnosis and Therapy (5th edition) published by Lippincott Williams & Wilkins in 2002. M.A. Foote wrote Basic Human Genetics for Medical Writers in the AMWA Journal 2002;17:7-17. A compilation such as this might suggest that one gene = one disease. -
Blue Light and Your Eyes
Blue Light and Your Eyes What is blue light? 211 West Wacker Drive, Suite 1700 Sunlight is made up of red, orange, yellow, green, blue, indigo and Chicago, Illinois 60606 violet light. When combined, it becomes the white light we see. 800.331.2020 Each of these has a different energy and wavelength. Rays on the PreventBlindness.org red end have longer wavelengths and less energy. On the other end, blue rays have shorter wavelengths and more energy. Light that looks white can have a large blue component, which can expose the eye to a higher amount of wavelength from the blue end of the spectrum. Where are you exposed to blue light? The largest source of blue light is sunlight. In addition, there are many other sources: • Fluorescent light • CFL (compact fluorescent light) bulbs • LED light • Flat screen LED televisions • Computer monitors, smart phones, and tablet screens Blue light exposure you receive from screens is small compared to the amount of exposure from the sun. And yet, there is concern over the long-term effects of screen exposure because of the close proximity of the screens and the length of time spent looking at them. According to a recent NEI-funded study, children’s eyes absorb more blue light than adults from digital device screens. This publication is copyrighted. This sheet may be reproduced—unaltered in hard print (photocopied) for educational purposes only. The Prevent Blindness name, logo, telephone number and copyright information may not be omitted. Electronic reproduction, other reprint, excerption or use is not permitted without written consent. -
Cataract Surgery and Diplopia
…AFTER CATARACT SURGERY Lionel Kowal RVEEH Melbourne חננו מאתך דעה בינה והשכל DISTORTION Everything in my talk is distorted by selection bias I don‟t do cataract surgery. I don‟t see the numerous happy pts that you produce I see a small Array of pts with imperfect outcomes that may /not be due to the cataract surgery DIPLOPIA AFTER CATARACT SURGERY ‘Old’ reasons ‘New’ reasons : Normal or near- normal muscle function: usually ≥1 ‘minor’ stresses on sensory & motor fusion Inf Rectus contracture after -- Anisometropia: Monovision & -caine damage Aniseikonia Other muscles damaged by -caine Metamorphopsia 2ary to macular disease Incidental 4ths and occult Minor acquired motility changes Graves‟ Ophthalmopathy of the elderly: Sagging eye uncovered by cataract surgery muscles Old, Rare & largely forgotten Other sensory issues: Big Amblyopia: fixation switch difference in contrast between Hiemann Bielschowsky images, large field defects. „OLD‟ REASONS : -CAINE TOXICITY IS IT A PERI- OR RETRO- BULBAR? If you add an EMG monitor to your injecting needle, whether you think you are doing a Retro- or Peri- Bulbar, you are IN the inf rectus ~ ½ of the time* *Elsas, Scott „OLD‟ REASONS : -CAINE TOXICITY CAN BE ANY MUSCLE, USU IR, ESP. LIR Day 1: LIR paresis : left hyper, restricted L depression, diplopia : everyone anxious ≤1% Day 7-10: diplopia goes : everyone happy Week 2+: LIR fibrosis begins - diplopia returns : left hypo, vertical & torsional diplopia, restricted L elevation: everyone upset 0.1-0.2% Hardly ever gets better Spontaneous recovery from inferior rectus contracture (consecutive hypotropia) following local anesthetic injury. Sutherland S, Kowal L. Binocul Vis Strabismus Q. -
Eyemed Blue Light
BLUE LIGHT: FREQUENTLY ASKED QUESTIONS From ZZZs to disease, the blue light battle is on It’s indisputable: our eyes are overexposed to digital devices like never before. And in the background hides potentially harmful blue light that may affect our sleep, or even cause long-term vision issues. But, here’s some good news — you can act now to potentially minimize vision issues later with advanced lens filtering technology formulated to guard your eyes. Q: WHAT IS BLUE LIGHT? A: Blue light is a natural part of the light spectrum visible to the human eye; it can come from fluorescent lighting, electronic screens, and of course, the sun. By day, blue light can be associated with boosted mood and attention, but by night, it can be a culprit of interrupted sleep. 1 Q: HOW DOES BLUE LIGHT INTERRUPT SLEEP? A: Researchers know that exposure to light at night suppresses the secretion of melatonin, a hormone that tells us when it is time to sleep. And an extended lack of deep sleep has been linked to depression and a decline in the body’s ability to fight off certain diseases. 1 Q: CAN BLUE LIGHT EXPOSURE CAUSE LONG-TERM DAMAGE TO MY EYESIGHT? A: In addition to disrupting sleep, blue light has been found to contribute to retinal stress, which could lead to an early onset of age-related macular degeneration (AMD).2 Macular degeneration deteriorates healthy cells within the macula, creating a loss of central vision that may impact reading, writing, driving, color perception and other cognitive functions. In serious cases, blindness can occur. -
Causes of Heterochromia Iridis with Special Reference to Paralysis Of
CAUSES OF HETEROCHROMIA IRIDIS WITH SPECIAL REFER- ENCE TO PARALYSIS OF THE CERVICAL SYMPATHETIC. F. PHINIZY CALHOUN, M. D. ATLANTA, GA. This abstract of a candidate's thesis presented for membership in the American Ophthal- mological Society, includes the reports of cases, a general review of the literature of the sub- ject, the results of experiments, and histologic observations on the effect of extirpation of the cervical sympathetic in the rab'bit, the conclusions reached from the investigation, and a bib- liography. That curious condition which con- thinks that the word hetcrochromia sists in a difference in the pigmentation should apply to those cases in which of the two eyes, is regarded by the parts of the same iris have different casual observer as a play or caprice of colors. In those cases where a cycli- nature. This phenomenon has for cen- tis accompanies the iris decoloration, turies been noted, and was called hcte- Butler8 uses the term "heterochromic roglaucus by Aristotle1. One who cyclitis," but the "Chronic Cyclitis seriously studies the subject, is at once with Decoloration of the Iris" as de- impressed with the complexity of the scribed by Fuchs" undoubtedly gives a situation, and soon learns that nature more accurate description of the dis- plays a comparatively small part in its ease, notwithstanding its long title. causation. It is however only within The commonly accepted and most uni- a comparatively recent time that the versally used term Hetcrochromia Iri- pathologic aspect has been considered, dis exactly expresses and implies the and in this discussion I especially wish picture from its derivation (irtpoa to draw attention to that part played other, xpw/xa) color. -
Spontaneous Rupture of the Anterior Capsule of a Hypermature Lens
127 SINGAPORE MEDICAL JOURNAL Vol. 4, No. 3. September, 1963. SPONTANEOUS RUPTURE OF THE ANTERIOR CAPSULE OF A HYPERMATURE LENS By Arthur Lim Siew Ming, F.R.C.S. (Eng.), D.O. (Lond.) (Department of Ophthalmology, General Hospital, Singapore) As early as 1764, Morgagni, the famous She was admitted for removal of the nucleus, anatomist wrote in his book "The seats and and was given local steroids and i % pilocar- causes of disease" about hypermature cataracts pine eye drops to the left eye every 6 hours. in the elderly. This is a type of hypermature The eye was examined daily and no change cataract which is still known by his name-the or increased tension was seen. Morgagnian cataract. On 24th. January, the nucleus was removed In 1900, Gifford described four cases of hy- through a superior limbal section with a von permature cataract resulting in "spontaneous Greafe's cataract knife extending for about 3 cure". Of these, only one retained vision and of the circumference of the limbus, after a pre - the other three were blinded by secondary glau- placed suture was inserted. coma. Since this description there were scatter- A small peripheral iridotomy was done and ed reports in the ophthalmic literature of spon- the nucleus of the lens removed without diffi- taneous rupture of the anterior capsule of the culty with a vectis, which was inserted behind hypermature cataract, with expulsion of the the nucleus and lifted it gently against the pos- contents of the lens into the anterior chamber. terior surface of the cornea in order to However, lens induced uveitis and secondary manoeuvre it out through the section.