ICD-10-PCS Reference Manual 08/04/10 Preliminary
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Percutaneous Nephrostomy and Sclerotherapy with 96% Ethanol for the Treatment of Simple Renal Cysts: Pilot Study Mustafa Kadıhasanoğlu1, Mete Kilciler2, Özcan Atahan3
İstanbul Med J 2016; 17: 20-3 Original Article DOI: 10.5152/imj.2016.20981 Percutaneous Nephrostomy and Sclerotherapy with 96% Ethanol for the Treatment of Simple Renal Cysts: Pilot Study Mustafa Kadıhasanoğlu1, Mete Kilciler2, Özcan Atahan3 Introduction: The objectives of this study was to evaluate the safety and efficacy of aspiration with percutaneous nephrostomy tube and sclerotherapy with 96% ethanol for simple renal cyst. Methods: Between 2011-2014, 34 patients with symptomatic renal cysts were included in the study. Mean age was 52.3±4.6 years (range, 39-72 years). The Abstract patients had only flank pain. Procedure was performed with ultrasound guidance and under fluoroscopic control. After puncture with 18 G angiography needle, guide-wire was advanced to the collecting system. An 14Fr nephrostomy catheter was then advanced over the guide wire. After taking cystography 96% ethyl alcohol was injected into the cyst and drained amount 10%. We continued daily to inject same amount of alcohol postoperatively until the drainage was less than 50 mL. 6th months and yearly follow-up were performed with ultrasound.. Results: Percutaneous access was achieved in all patients. Cysts were unilateral, single and with a mean diameter 9.1±3.2 cm (range, 7-16 cm). Median drained volume was 212 mL (200-1600 mL) and median the injected ethanol volume was 54 mL (20- 160 mL). Radiological improvement at the end of the 6th month was amount 94.1% and 91.1% at the end of the 1st year while 83.3% of patients had symptomatic decline. There was no major complication after the procedure. -
ICD-10 Coding Clinic Corner: Accountability Act of 1996 (HIPAA) Is 413.65(A)(2): Diabetes and Osteomyelitis
Issue No. 6 Volume No. 2 September 2015 New and Revised Place of Service TABLE of CONTENTS Codes for OutpatientNote Hospital from Tony New and Revised Place of Joette P. Derricks, MPA, CMPE, CHC, CPC, CSSGB Service Codes for Outpatient Joh Vice President of Regulatory Affairs & Research Hospital ..................................... 1 MiraMed Global Services ERCP with Exchange of a Common Bile Duct Stent .......... 3 Any health insurer subject to the policy. Contractor editing shall Laparoscopic Procedure: An uniform electronic claim transaction treat POS 19 and POS 22 in the Education .................................. 5 and code set standards under the same way. The definition of a Health Insurance Portability and “campus” is found in Title 42 CFR ICD-10 Coding Clinic Corner: Accountability Act of 1996 (HIPAA) is 413.65(a)(2): Diabetes and Osteomyelitis ..... 7 required to make changes to the Campus means the physical Place of Service (POS) code from the Stars of MiraMed ...................... 8 area immediately adjacent to POS code set maintained by the the provider’s main buildings, Are You a Good Auditor? ......... 9 Centers for Medicare and Medicaid other areas and structures that Services (CMS) effective January 1, Coding Case Scenario ............. 11 are not strictly contiguous to 2016. the main buildings but are The POS code set provides setting located within 250 yards of the If you have an article information necessary to main buildings, and any other appropriately pay Medicare and areas determined on an or idea to share for The Medicaid claims. Other payers may individual case basis, by the Code , please submit to: or may not use the POS codes in the CMS regional office, to be part Dr. -
Central Venous Access with Accelerated Seldinger Technique Versus Modified Seldinger Technique
Central Venous Access with Accelerated Seldinger Technique versus Modified Seldinger Technique L A N E THAUT , D O , CAPT , M C , USAF SAN ANTONIO MILITARY MEDICAL CENTER CO- AUTHORS: WELLS WEYMOUTH, MD, DANIEL RESCHKE, MD, BRANDEN HUNSAKER,MD Disclosures Expired POWERWAND™ combination device kits were donated by Access Scientific for the express purpose to be used in this study; however, no other contributions were made. We approached Access Scientific to obtain the devices to conduct this study. No financial relationship with Access Scientific. Background Techniques Research and Outcomes Questions Background Air Force SAMMC San Antonio, TX Level 1 Trauma Center 85,000+ patients annually Central Access Common Procedure Approximately 8% of hospitalized patients Multiple indications Large volume fluid or blood product resuscitation Administration of central acting medications Multiple medications simultaneously Trans venous pacing Difficult peripheral access Quick Story What if there was a simpler/quicker way? Intra-osseous? Complications Slower Flow Rate Labs Traditional Central Line Multiple steps/parts Midlines Usually used for extended dwelling lines. Self contained, all in one device. Equivalent and sometimes superior flow rate. Question Will the use of combination devices (midline/POWERWAND™) and the associated accelerated technique reduce the time of CVC placement? VS What is Accelerated Seldinger Technique? Needle, guidewire, dilator, and sheath into one. The device needle is inserted into the target vein under ultrasound guidance and a flash is observed The internal guidewire is then advanced into the vein and snapped into the needle hub. Dilator collar is turned and the dilator and sheath are advanced The dilator hub is disengaged from the needle hub, and the guidewire, dilator, and needle are all removed as a single unit. -
Biology 152 – Brain/Spinal Cord/Ear/Eye Objectives
Biology 152 – Brain/Spinal Cord/Ear/Eye Objectives Items will be identified on a sheep's brain dissection, human brain models, sagittal/coronal sections of human brains in plastic, ear and eye models, and an eye dissection. You will need to learn a proper function for each listed item for the practical. BRAIN REGIONS – learn their names, position in the brain, and functions Meninges – protective tissue layers around the brain and spinal cord Dura mater strong mother, collagenous layer with dural sinuses, protects brain and allows reabsorption of CSF into blood stream Arachnoid arachnoid villi “pooch” into dural sinus to allow CSF loss to blood, holds membrane CSF and allows circulation around brain/spine Pia mater weak mother, holds shape of brain and allows diffusion of nutrients and wastes between tissues and CSF Cerebrum – two hemispheres where all conscious thought occurs L/R Hemispheres dual hard drives that control behavior and store all memory Cerebral cortex thin gray matter (nonmyelinated) layer that stores information Frontal lobe site of voluntary motor control, behavior, and intelligence Parietal lobe site of gustatory (taste) storage, special sense/navigation ability Temporal lobe site of olfactory and auditory memory storage Occipital lobe site of visual memory storage Precentral gyrus primary motor cortex router connecting frontal lobe to muscles Postcentral gyrus primary somatosensory router connecting senses to posterior brain regions Central sulcus low spot in cerebrum dividing all motor from all sensory areas Gyri/sulci -
Neuromuscular Organisation of Mammalian Extraocular Muscles
Rapporter fra Høgskolen i Buskerud nr. 36 RAPPORT RAPPORT Neuromuscular organisation of mammalian extraocular muscles Inga-Britt Kjellevold Haugen (M.Phil) Rapporter fra Høgskolen i Buskerud Nr. 36 Neuromuscular organisation of mammalian extraocular muscles Inga-Britt Kjellevold Haugen (M.Phil) Kongsberg 2002 HiBus publikasjoner kan kopieres fritt og videreformidles til andre interesserte uten avgift. En forutsetning er at navn på utgiver og forfatter(e) angis - og angis korrekt. Det må ikke foretas endringer i verket. ISBN 82-91116-52-0 ISSN 0807-4488 2 CONTENTS 1. PREFACE ...................................................................................................................................... 4 2. ACKNOWLEDGEMENT............................................................................................................. 5 3. INTRODUCTION ......................................................................................................................... 6 4. LITERATURE REVIEW OF MAMMALIAN EXTRAOCULAR MUSCLES .................... 10 4.1 MUSCLE HISTOLOGY .................................................................................................................. 14 4.1.1 Ultrastructure and physiology ............................................................................................. 14 4.1.2 Fibre classification and distribution .................................................................................... 21 4.1.3 Motor innervation ............................................................................................................... -
Peripheral Venous Malformations with a Dominant Outflow Vein: Results of Ethanol Embolization
CASE REPORT Peripheral Venous Malformations with a Dominant Outflow Vein: Results of Ethanol Embolization Hadi Rokni-Yazdi1, Mahsa Ghajarzadeh2, Amir Hossein Keyvan3, Mohammad Javad Namavar3, and Sepehr Azizi3 1 Advanced Diagnostic and Interventional Radiology Research Center (ADIR), Imaging Medical Center, Imam Hospital, Tehran University of Medical Sciences, Tehran, Iran 2 Brain and Spinal Injury Repair Research Center, Tehran University of Medical Sciences, Tehran, Iran 3 Department of Infectious Diseases, Imam Hospital, Tehran University of Medical Sciences, Tehran, Iran Received: 11 Nov. 2013; Accepted: 29 Mar. 2014 Abstract- Venous malformations are the most common form of symptomatic vascular malformations. VM s could classify into low-flow lesions (VMs) and high-flow lesions (AVMs). For low-flow venous lesions, direct percutaneous puncture with injection of sclerosing agents (sclerotherapy) has been described as a successful therapy. In this article, we want to introduce a patient who treated with ethanol sclerotherapy for VM located in the right flank. The patients were a 35-year-old man with right flank mass, skin discoloration and hemorrhagic foci. Color Doppler ultrasonography showed low flow vascular malformation while Magnetic Resonance Imaging (MRI) showed that the mass contained fat tissue with branching tubular signal void structures inside. The draining vein was first coiled via tortuous venous malformation vessels access and then VM was embolized.Under ultrasonographic guide, direct puncture of one branches of venous malformation was performed, and contrast media were injected. The patient underwent the sclerotherapy every month for four consecutive months. The patient was followed up for a year, and clinical examination revealed 40-50% size reduction of the lesion while no bleeding was detected from the lesion during the follow-up period. -
Entrapment Neuropathy of the Central Nervous System. Part II. Cranial
Entrapment neuropathy of the Cranial nerves central nervous system. Part II. Cranial nerves 1-IV, VI-VIII, XII HAROLD I. MAGOUN, D.O., F.A.A.O. Denver, Colorado This article, the second in a series, significance because of possible embarrassment considers specific examples of by adjacent structures in that area. The same entrapment neuropathy. It discusses entrapment can occur en route to their desti- nation. sources of malfunction of the olfactory nerves ranging from the The first cranial nerve relatively rare anosmia to the common The olfactory nerves (I) arise from the nasal chronic nasal drip. The frequency of mucosa and send about twenty central proces- ocular defects in the population today ses through the cribriform plate of the ethmoid bone to the inferior surface of the olfactory attests to the vulnerability of the optic bulb. They are concerned only with the sense nerves. Certain areas traversed by of smell. Many normal people have difficulty in each oculomotor nerve are pointed out identifying definite odors although they can as potential trouble spots. It is seen perceive them. This is not of real concern. The how the trochlear nerves are subject total loss of smell, or anosmia, is the significant to tension, pressure, or stress from abnormality. It may be due to a considerable variety of causes from arteriosclerosis to tu- trauma to various bony components morous growths but there is another cause of the skull. Finally, structural which is not usually considered. influences on the abducens, facial, The cribriform plate fits within the ethmoid acoustic, and hypoglossal nerves notch between the orbital plates of the frontal are explored. -
CME Anatomy of Aging Face
Published online: 2020-01-15 Free full text on www.ijps.org CME Anatomy of aging face Rakesh Khazanchi, Aditya Aggarwal, Manoj Johar1 Department of Plastic and Cosmetic Surgery, Sir Ganga Ram Hospital, New Delhi - 110 060, 1Fortis Hospital, Noida, UP, India Address for correspondence: Dr. Rakesh Khazanchi, Department of Plastic and Cosmetic Surgery, Sir Ganga Ram Hospital, New Delhi - 110 060, India. E-mail: [email protected] ejuvenation of the face is evolving into a common deposition in regions of body called ‘depots’ procedure in India. This may be attempted by f) Fascial and ligament laxity Reither surgical or non surgical means. Surgical g) Shrinkage of glandular tissue (Salivary glands) rejuvenation of face includes a large variety of procedures h) Skeletal resorption to revert the changes of aging. In the past, face lift operation was done to simply lift the sagging skin rather Facial soft tissues are arranged in concentric layers. than shaping the face. However it often ended up in Skin is the outermost layer and then the basic building giving the patient an ‘operated on’ look producing tight blocks-fat, superficial fascia also known as superficial appearing face. The surgeons have now learnt that aging musculoaponeurotic system (SMAS), deep fascia and the process is a complex process that involves soft tissues as periosteum that covers the facial skeleton. Interspersed well as skeleton of face and is not just sagging of skin. in these layers are vessels, nerves, facial muscles and Therefore in order to get a good result after surgical retaining ligaments. Knowledge of these layers allows facial rejuvenation, it is paramount to understand these the surgeon to dissect in a given anatomic plane without anatomical structures and the effect of aging process on damaging important structures. -
CHAPTER 8 Face, Scalp, Skull, Cranial Cavity, and Orbit
228 CHAPTER 8 Face, Scalp, Skull, Cranial Cavity, and Orbit MUSCLES OF FACIAL EXPRESSION Dural Venous Sinuses Not in the Subendocranial Occipitofrontalis Space More About the Epicranial Aponeurosis and the Cerebral Veins Subcutaneous Layer of the Scalp Emissary Veins Orbicularis Oculi CLINICAL SIGNIFICANCE OF EMISSARY VEINS Zygomaticus Major CAVERNOUS SINUS THROMBOSIS Orbicularis Oris Cranial Arachnoid and Pia Mentalis Vertebral Artery Within the Cranial Cavity Buccinator Internal Carotid Artery Within the Cranial Cavity Platysma Circle of Willis The Absence of Veins Accompanying the PAROTID GLAND Intracranial Parts of the Vertebral and Internal Carotid Arteries FACIAL ARTERY THE INTRACRANIAL PORTION OF THE TRANSVERSE FACIAL ARTERY TRIGEMINAL NERVE ( C.N. V) AND FACIAL VEIN MECKEL’S CAVE (CAVUM TRIGEMINALE) FACIAL NERVE ORBITAL CAVITY AND EYE EYELIDS Bony Orbit Conjunctival Sac Extraocular Fat and Fascia Eyelashes Anulus Tendineus and Compartmentalization of The Fibrous "Skeleton" of an Eyelid -- Composed the Superior Orbital Fissure of a Tarsus and an Orbital Septum Periorbita THE SKULL Muscles of the Oculomotor, Trochlear, and Development of the Neurocranium Abducens Somitomeres Cartilaginous Portion of the Neurocranium--the The Lateral, Superior, Inferior, and Medial Recti Cranial Base of the Eye Membranous Portion of the Neurocranium--Sides Superior Oblique and Top of the Braincase Levator Palpebrae Superioris SUTURAL FUSION, BOTH NORMAL AND OTHERWISE Inferior Oblique Development of the Face Actions and Functions of Extraocular Muscles Growth of Two Special Skull Structures--the Levator Palpebrae Superioris Mastoid Process and the Tympanic Bone Movements of the Eyeball Functions of the Recti and Obliques TEETH Ophthalmic Artery Ophthalmic Veins CRANIAL CAVITY Oculomotor Nerve – C.N. III Posterior Cranial Fossa CLINICAL CONSIDERATIONS Middle Cranial Fossa Trochlear Nerve – C.N. -
Practice Parameter for the Use of Ultrasound to Guide Vascular Access Procedures
PRACTICE GUIDELINES AIUM Practice Parameter for the Use of Ultrasound to Guide Vascular Access Procedures I. Introduction he clinical aspects of this parameter were developed collaboratively among the AIUM and other organizations whose members use ultrasound for guidance in vascular T “ ” access procedures (see Acknowledgments ). Recommendations for practitioner requirements, the written request for the examination, procedure documentation, and quality control vary among the organizations and are addressed by each separately. This parameter has been developed by and for clinicians from diverse specialties and practitioner levels who perform vascular access. While vascular access may be performed using external landmarks, point-of-care ultrasound is now increasingly available.1 Appropriately used, ultrasound guidance for vascular access has been shown to improve success rates while reducing iatrogenic injury, the number of needle passes, and infection rates.2 Addition- ally, it may improve patient comfort and satisfaction. This parameter is intended to be evidence based when possi- ble and to include selected references of importance, but it is not meant to be a comprehensive or rigorous literature review, as this has been accomplished elsewhere.3 The intent of this document is to highlight appropriate evidence while also providing a practical, real-world expert consensus from clinicians with diverse back- grounds on the best use and techniques for incorporating ultra- sound into vascular access procedures with the ultimate goal of improving the care of our patients. II. Indications/Contraindications Ultrasound should be used to aid in central venous, peripheral venous, and arterial access procedures.4 When used appropriately by qualified personnel, there are no absolute contraindications to using ultrasound as a procedural adjunct for vascular guidance. -
Botulinum Toxin to Improve Lower Facial Symmetry in Facial Nerve Palsy SA Sadiq Et Al 1432
Eye (2012) 26, 1431–1436 & 2012 Macmillan Publishers Limited All rights reserved 0950-222X/12 www.nature.com/eye 1;2 3 4 Botulinum toxin to SA Sadiq , S Khwaja and SR Saeed CLINICAL STUDY improve lower facial symmetry in facial nerve palsy Abstract Introduction In long-standing facial palsy, Introduction muscles on the normal side overcontract In facial palsy, paralysis of muscles on the causing difficulty in articulation, eating, affected side of the face results in loss of drinking, cosmetic embarrassment, and forehead creases, loss of the nasolabial fold, psychological effects as patients lack lagophthalmos, brow droop, and drooping of confidence in public. the corner of the mouth. In contrast, muscles on Methods We injected botulinum toxin A the unaffected side of the face no longer have (BTXA) into the normal contralateral smile opposing forces.1 This may cause difficulty in muscles to weaken them and restore symmetry 1Manchester Royal Eye articulation, eating, drinking, and is often to both active and passive movements by Hospital, Manchester, UK cosmetically unacceptable to patients because of neutralising these overacting muscles. asymmetry, especially when speaking, smiling, 2 Results A total of 14 patients received BTXA The University of and laughing. There are significant Manchester, Manchester (79% women, median age 47 years, average psychological effects as patients lack the Academic Health Science length of palsy 8 years). They were all difficult confidence to carry out many daily activities in Centre, Central Manchester cases graded between 2 and 6 (average grade 3 Foundation Trust, public, such as appearing in photographs. House–Brackmann). All 14 patients reported Manchester, UK Although management is difficult, there are a improved facial symmetry with BTXA (dose range of reanimation options available. -
Computed Tomography of the Buccomasseteric Region: 1
605 Computed Tomography of the Buccomasseteric Region: 1. Anatomy Ira F. Braun 1 The differential diagnosis to consider in a patient presenting with a buccomasseteric James C. Hoffman, Jr. 1 region mass is rather lengthy. Precise preoperative localization of the mass and a determination of its extent and, it is hoped, histology will provide a most useful guide to the head and neck surgeon operating in this anatomically complex region. Part 1 of this article describes the computed tomographic anatomy of this region, while part 2 discusses pathologic changes. The clinical value of computed tomography as an imaging method for this region is emphasized. The differential diagnosis to consider in a patient with a mass in the buccomas seteric region, which may either be developmental, inflammatory, or neoplastic, comprises a rather lengthy list. The anatomic complexity of this region, defined arbitrarily by the soft tissue and bony structures including and surrounding the masseter muscle, excluding the parotid gland, makes the accurate anatomic diagnosis of masses in this region imperative if severe functional and cosmetic defects or even death are to be avoided during treatment. An initial crucial clinical pathoanatomic distinction is to classify the mass as extra- or intraparotid. Batsakis [1] recommends that every mass localized to the cheek region be considered a parotid tumor until proven otherwise. Precise clinical localization, however, is often exceedingly difficult. Obviously, further diagnosis and subsequent therapy is greatly facilitated once this differentiation is made. Computed tomography (CT), with its superior spatial and contrast resolution, has been shown to be an effective imaging method for the evaluation of disorders of the head and neck.