Neuro-Ophthalmology/Orbit 2017-2019
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Fluids Hypertension Syndromes: Migraines, Headaches, Normal Tension Glaucoma, Benign Intracranial Hypertension, Caffeine Intolerance
Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 1 Fluids Hypertension Syndromes: Migraines, Headaches, Normal Tension Glaucoma, Benign Intracranial Hypertension, Caffeine Intolerance. Etiologies, Pathophysiologies and Cure. Author: Leonardo Izecksohn. Medical Doctor, Ophthalmologist, Master of Public Health. We have no financial interest on any medicament, device, or technique described in this e-book. We authorize the free copy and distribution of this e-book for educational purposes. The 1st. edition was written at the year 1996, with 2 pages. There are other editions spread at the Internet. This is the enlarged and revised edition 65-f, updated on May 24, 2016. ISBN 978-85-906664-1-7 DOI: 10.13140/2.1.3074.5602 www.izecksohn.com/leonardo/ [email protected] Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 2 Abstract A – Migraines, Headaches and Fluids Hypertension Syndromes – What are they? - Answer: Migraines and most primary headaches are the aches of the pressure increase in the fluids: - Intraocular Aqueous Humor, - Intracranial Cerebrospinal Fluid, and - Inner ear’s Perilymph and Endolymph. We denominate the fluids’ pressure rises and their consequent migraines, signs, symptoms and sick- nesses as the Fluids Hypertension Syndromes. Migraines and headaches are not sicknesses: they are symptoms of the sicknesses. B – How many Fluids Hypertension Syndromes do exist? - Answer: There are three Fluids Hypertension Syndromes: 1- Ocular, due to raises of the intraocular Aqueous Humor pressure. 2- Cerebrospinal, due to raises of the intracranial Cerebrospinal Fluid pressure. 3- Inner Ears, due to raises of the inner ears' Perilymph and Endolymph pressures. Each patient can present one, two, or all the three Fluids Hypertension Syndromes in the same time. -
ANMC Specialty Clinic Services
Cardiology Dermatology Diabetes Endocrinology Ear, Nose and Throat (ENT) Gastroenterology General Medicine General Surgery HIV/Early Intervention Services Infectious Disease Liver Clinic Neurology Neurosurgery/Comprehensive Pain Management Oncology Ophthalmology Orthopedics Orthopedics – Back and Spine Podiatry Pulmonology Rheumatology Urology Cardiology • Cardiology • Adult transthoracic echocardiography • Ambulatory electrocardiology monitor interpretation • Cardioversion, electrical, elective • Central line placement and venous angiography • ECG interpretation, including signal average ECG • Infusion and management of Gp IIb/IIIa agents and thrombolytic agents and antithrombotic agents • Insertion and management of central venous catheters, pulmonary artery catheters, and arterial lines • Insertion and management of automatic implantable cardiac defibrillators • Insertion of permanent pacemaker, including single/dual chamber and biventricular • Interpretation of results of noninvasive testing relevant to arrhythmia diagnoses and treatment • Hemodynamic monitoring with balloon flotation devices • Non-invasive hemodynamic monitoring • Perform history and physical exam • Pericardiocentesis • Placement of temporary transvenous pacemaker • Pacemaker programming/reprogramming and interrogation • Stress echocardiography (exercise and pharmacologic stress) • Tilt table testing • Transcutaneous external pacemaker placement • Transthoracic 2D echocardiography, Doppler, and color flow Dermatology • Chemical face peels • Cryosurgery • Diagnosis -
Textbook of Ophthalmology, 5Th Edition
Textbook of Ophthalmology Textbook of Ophthalmology 5th Edition HV Nema Former Professor and Head Department of Ophthalmology Institute of Medical Sciences Banaras Hindu University Varanasi India Nitin Nema MS Dip NB Assistant Professor Department of Ophthalmology Sri Aurobindo Institute of Medical Sciences Indore India ® JAYPEE BROTHERS MEDICAL PUBLISHERS (P) LTD. New Delhi • Ahmedabad • Bengaluru • Chennai Hyderabad • Kochi • Kolkata • Lucknow • Mumbai • Nagpur Published by Jitendar P Vij Jaypee Brothers Medical Publishers (P) Ltd B-3 EMCA House, 23/23B Ansari Road, Daryaganj, New Delhi 110 002 I ndia Phones: +91-11-23272143, +91-11-23272703, +91-11-23282021, +91-11-23245672 Rel: +91-11-32558559 Fax: +91-11-23276490 +91-11-23245683 e-mail: [email protected], Visit our website: www.jaypeebrothers.com Branches 2/B, Akruti Society, Jodhpur Gam Road Satellite Ahmedabad 380 015, Phones: +91-79-26926233, Rel: +91-79-32988717 Fax: +91-79-26927094, e-mail: [email protected] 202 Batavia Chambers, 8 Kumara Krupa Road, Kumara Park East Bengaluru 560 001, Phones: +91-80-22285971, +91-80-22382956, 91-80-22372664 Rel: +91-80-32714073, Fax: +91-80-22281761 e-mail: [email protected] 282 IIIrd Floor, Khaleel Shirazi Estate, Fountain Plaza, Pantheon Road Chennai 600 008, Phones: +91-44-28193265, +91-44-28194897 Rel: +91-44-32972089, Fax: +91-44-28193231, e-mail: [email protected] 4-2-1067/1-3, 1st Floor, Balaji Building, Ramkote Cross Road Hyderabad 500 095, Phones: +91-40-66610020, +91-40-24758498 Rel:+91-40-32940929 Fax:+91-40-24758499, e-mail: [email protected] No. 41/3098, B & B1, Kuruvi Building, St. -
Permanent Central Scotoma Caused by Looking at the Sun During an Eclipse, and Complicated by Uniocular, Transi- Ent, Revolving Hemianopsia
PERMANENT CENTRAL SCOTOMA CAUSED BY LOOKING AT THE SUN DURING AN ECLIPSE, AND COMPLICATED BY UNIOCULAR, TRANSI- ENT, REVOLVING HEMIANOPSIA. From Dr. Knapp’s Practice, Reported by Dr. A. DUANE, New York. Reprinted from the Archives of Ophthalmology, Vol. xxiv., No. i, 1895 PERMANENT CENTRAL SCOTOMA CAUSED BY LOOKING AT THE SUN DURING AN ECLIPSE, AND COMPLICATED BY UNIOCULAR, TRANSI- ENT, REVOLVING HEMIANOPSIA. From Dr. Knapp’s Practice, Reported by Dr. A. DUANE, New York, instances of central scotoma after expos- ALTHOUGHure to sunlight are by no means rare, the subjoined case seems worthy ofrecord, because of the persistence of the scotoma twelve years afterwards, and because of the pres- ence of a peculiar hemiopic and scotoma scintil- lans, which apparently was likewise the result of the action of the sun’s rays. The patient, P. W., a man twenty-four years of age, consulted Dr. Knapp on Feb. 5, 1895, and gave the following history: Twelve years previous he had, on the occasion of the transit of Venus, 1 looked directly at the sun through the tube formed by the nearly closed fist. Soon after, he found that when both eyes were open, but not when the left was closed, a greenish cloud hid com- pletely the centre of every object looked at. This had exactly the shape of the illuminated portion of the sun at the time of the transit, i. e., was a circle with a crescentic defect at the upper part corresponding to the spot occupied by the planet at the time. It was then of considerable size, covering an area 5 inches in width when projected upon a surface 15 or 20 inches off. -
Evaluation of Oxidative Stress in Migraine Patients with Visual Aura - the Experience of an Rehabilitation Hospital
Evaluation of oxidative stress in migraine patients with visual aura - the experience of an Rehabilitation Hospital Adriana Bulboaca1,4, Gabriela Dogaru2,4, Mihai Blidaru1, Angelo Bulboaca3,4, Ioana Stanescu3,4 Corresponding author: Gabriela Dogaru, E-mail address: [email protected] Balneo Research Journal DOI: http://dx.doi.org/10.12680/balneo.2018.201 Vol.9, No.3, September 2018 p: 303 –308 1- Department of Pathophysiology, Iuliu Haţieganu University of Medicine and Pharmacy, Cluj-Napoca, Romania 2 -Department of PRM, Iuliu Haţieganu University of Medicine and Pharmacy Cluj-Napoca, Cluj-Napoca, Romania 3 - Department of Neurology, Iuliu Haţieganu University of Medicine and Pharmacy, Cluj-Napoca, Romania 4- Rehabilitation Hospital, Cluj-Napoca, Romania Abstract Background: Although there are previous studies regarding the migraine pathophysiology, the clinical entity of migraine with aura can have an different pathophysiological mechanism compared with migraine without aura. One of the most important mechanism in migraine is represented by increasing of oxidative stress. The aim of this study was to study the levels of two oxidative stress molecules: nitric oxide (NO) and malondialdehyde (MDA) in migraine with visual aura compared with migraine without aura. Material and Method: a Control group (healthy volunteers) of 37 patients and 58 patient with migraine divided in Group 1 (migraine with visual aura) and Group 2 (migraine without aura) were taken in the study. All the patient were assessed regarding the age, body mass index, blood pressure, basal glycaemia, smoking/non-smoking status, C reactive protein and fibrinogen. Visual aura was assessed regarding transitive negative visual symptoms or positive visual symptoms. Oxidative status was assessed by measurements of the plasma levels of NO and MDA. -
Acute Visual Loss
425 Acute Visual Loss ShirleyH.Wray,MD,PhD,FRCP1 1 Department of Neurology, Massachusetts General Hospital, Boston, Address for correspondence ShirleyH.Wray,MD,PhD,FRCP, Massachusetts Department of Neurology, Massachusetts General Hospital, 55 Fruit St, Boston 02114, MA (e-mail: [email protected]). Semin Neurol 2016;36:425–432. Abstract Acute visual loss is a frightening experience, a common ophthalmic emergency, and a diagnostic challenge. In this review, the author focusses on the diagnosis of transient Keywords monocular blindness and visual loss due to infarction of the retina and/or the optic nerve ► ocular stroke —the ocular parallel of cerebral stroke. Illustrative Case the left supraclinoid internal carotid artery (ICA) just proximal to the origin of the left posterior communicating artery. Day 1: The patient is a 75-year-old ophthalmologist who Day 22: The patient consulted a neurovascular surgeon experienced an acute transient “white out” of her vision in who obtained a head and neck computed tomographic her left eye lasting for 20 minutes. She had no accompanying angiogram that showed that the ICA aneurysm was symptoms. At this time, the patient was concerned and unchanged in size and morphology from the previous anxious that the white out of vision was an attack of transient exam. No other aneurysm was seen. The surgeon reviewed monocular blindness—a transient ischemic attack that can all the imaging studies with the patient and reassured her herald stroke. that there was minimal risk of rupture of the aneurysm. Day 4: She asked her ophthalmology fellow to examine her eye including intraocular pressure, dilated funduscopy, and Special Explanatory Note automated (Humphrey) visual fields. -
Orbit Ossifying Fibroma – Case Report and Literature Review
www.surgicalneurologyint.com Surgical Neurology International Editor-in-Chief: Nancy E. Epstein, MD, NYU Winthrop Hospital, Mineola, NY, USA. SNI: General Neurosurgery Editor Eric Nussbaum, MD National Brain Aneurysm and Tumor Center, Twin Cities, MN, USA Open Access Case Report Orbit ossifying fibroma – Case report and literature review Nicollas Rabelo1, Vinicius Trindade Gomes da Silva1, Marcelo Prudente do Espírito Santo1, Davi Solla1, Dan Zimelewicz Oberman2, Bruno Sisnando da Costa1, Fernando Pereira Frassetto3, Manoel Jacobsen Teixeira1, Eberval Gadelha Figueiredo1 Departments of 1Neurosurgery, 3Pathology, University of São Paulo, São Paulo, 2Department of Neurosurgery, Air Force Galeão Hospital, Rio de Janeiro, Brazil. E-mail: Nicollas Rabelo - [email protected]; Vinicius Trindade Gomes da Silva - [email protected]; Marcelo Prudente do Espírito Santo - [email protected]; Davi Solla - [email protected]; Dan Zimelewicz Oberman - [email protected]; Bruno Sisnando da Costa - [email protected]; Fernando Pereira Frassetto - [email protected]; Manoel Jacobsen Teixiera - [email protected]; *Eberval Gadelha Figueiredo - [email protected] ABSTRACT Background: Ossifying fibroma (OF) is benign bone lesions, most frequent in young children, more common in the maxillary sinus and mandible (75–89%), the pathogenesis of the tumor is not clear, there are many subtypes of OF. is paper aims to report an OF a case and literature review. Case Description: Male, 19 years old, with a progressive history proptosis since 2012, diagnosed as a right *Corresponding author: supraorbital lesion at an external service and assigned to conservative management. en, he evolved with double Eberval Gadelha Figueiredo, vision, which worsened in February of 2018, associated with a moderate headache. On admission: proptosis Av. -
The Ocular Signs of Migraine.1
THE OCULAR SIGNS OF MIGRAINE.1 BY E. R. Chambers, F.R.C.S., Ophthalmic Surgeon, Bristol Royal Infirmary. most constan an Visual phenomena form one of the Gowers pom s striking features in migraine attacks. a func lona out that the of migraine are prodromata ac so is the hea disturbance in the sensory organ, and a coarse which succeeds. By the headache is implied or, disturbance consisting of pain, often intense, w 1 s the other hand, slight and of short duration, occur alone, not emg in some cases the prodromata followed by headache. These premonitory symptoms, sense o not include The although sensory, do pain. sometimes loss, sight is very often involved, by partia of its or general, sometimes by an elaborate display higher crude functions, in light, colour or apparent motion. the we Of the processes which induce premonitions a have only hypothesis to rest on ; there is clearly cerebra disturbance of function in certain parts of the the subse hemisphere, probably the cortex, which is in function quent seat of the pain. The disturbance to e is apparently crude, and seems as it were ripp condition, through the centre, leaving an inhibited that cerebra which quickly passes away. It is possible cause o anaemia due to vaso-motor spasm is the A 1 aper read at a Meeting of the Bristol Medico-Chirurgical Society, held at the University of Bristol on January 13th, 1926. 29 Mr. E. R. Chambers these prodromal symptoms, and in support of this is the fact that the retinal arteries are markedly contracted during the prodromal stage. -
Migraine Aura Without Headache Donald M
B rief Reports Migraine Aura Without Headache Donald M. Pedersen, PA-C, PhD, William M. Wilson, PhD, George L. White, Jr, PA-C PhD Richard T. Murdock, MS/HSA, and Kathleen B. Digre, MD Salt Lake City, Utah Migraine is described as a familial disorder characterized to the disturbance described. The patient also denies by recurrent headaches that are variable in intensity, antecedent trauma or emotional stress. The episodes arc frequency, and duration.1 Attacks are usually unilateral reportedly always similar in nature, with an expanding but can also be bilateral and accompanied by throbbing scintillating scotoma and without subsequent headache, pain, photophobia, phonophobia, nausea, and vomiting. The patient’s first episode, his m ost recent episode, and Some migraines are preceded by, or are associated with, “a few” of the others have occurred after a 60-minutc neurological and mood disturbances. All of the above exercise period, which he performs consistently as a mat characteristics, however, are not necessarily present in ter of his daily routine. There is a history of myopia, for each attack, nor in each patient.2 correction o f which soft contact lenses are used, and It has been suggested that die prevalence of mi numerous vitreous floaters have been reported. The pa graine is probably markedly underestimated. Estimates tient takes no medication, has no history of illicit drug range from 10% to 34% of the general population, with use, and is otherwise healthy except for a history of mild some authors reporting both age-related and sex-related seasonal allergic rhinitis. There is a family history of differences. -
Federal Air Surgeon's Medical Bulletin • Vol
Federal Air Surgeon’s Medical Bulletin Aviation Safety Through Aerospace Medicine For FAA Aviation Medical Examiners, Office of Aerospace Medicine Personnel, Flight Standards Inspectors, and Other Aviation Professionals. Vol. 52, No. 2 2014-2 CONTENTS Federal Air Surgeon’s Medical Bulletin NEW AEROSPacE MEDICINE LEADERS ANNOUNCED -- -- -- -- -- -- -- -- -- -- 2 From the Office of Aerospace Medicine Library of Congress ISSN 1545-1518 QUESTIONS AND ANSWERS -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- -- 2 FROM THE FEDERAL AIR SURGEON’S PERSPECTIVE-- -- -- -- -- -- -- -- -- -- -- 3 Federal Air Surgeon ECG NORMAL VARIANT LIST - - - - - - - - - - - - - - - - - - - - - - - - - 4 James R. Fraser, MD, MPH AVIATION MEDIcal EXAMINER INFORMATION LINKS - - - - - - - - - - - - 4 Editor NEW PROTOCOL FOR DIABETES MEDIcaTIONS -- -- -- -- -- -- -- -- -- -- -- -- -- -- 5 Michael E. Wayda, BS OAM PHYSICIANS ON CALL, PART 4- - - - - - - - - - - - - - - - - - - - 6 The Federal Air Surgeon’s Medical COARCTATION OF THE AORTA (CASE REPORT) -- -- -- -- -- -- -- -- -- -- -- -- -- -- 8 Bulletin is published quarterly for aviation medical examiners and CEREBRAL CAVERNOUS MALFORMATION (CASE REPORT)-- -- -- -- -- -- 10 others interested in aviation safety ADENOID CYSTIC CARCINOMA (CASE REPORT)- -- -- -- -- -- -- -- -- -- -- -- -- 12 and aviation medicine. The Bulletin is prepared by the FAA’s Civil CAMI’S POSTMORTEM AVIATION TOXICOLOGY COLLOQUIUM - - - - 13 Aerospace Medical Institute, with (PHANTOM) LETTERS TO THE EDITOR -- -- -- -- -
Transient Visual Loss – Valerie Biousse, MD and Nancy J
Transient Visual Loss – Valerie Biousse, MD and Nancy J. Newman, MD Biousse V, Nahab F, Newman NJ. Management of Acute Retinal Ischemia: Follow the Guidelines! Ophthalmology. 2018 Oct;125(10):1597-1607 Acute retinal arterial ischemia, including vascular transient monocular vision loss (TMVL) and branch (BRAO) and central retinal arterial occlusions (CRAO), are ocular and systemic emergencies requiring immediate diagnosis and treatment. Guidelines recommend the combination of urgent brain magnetic resonance imaging with diffusion-weighted imaging, vascular imaging, and clinical assessment to identify TMVL, BRAO, and CRAO patients at highest risk for recurrent stroke, facilitating early preventive treatments to reduce the risk of subsequent stroke and cardiovascular events. Because the risk of stroke is maximum within the first few days after the onset of visual loss, prompt diagnosis and triage are mandatory. Eye care professionals must make a rapid and accurate diagnosis and recognize the need for timely expert intervention by immediately referring patients with acute retinal arterial ischemia to specialized stroke centers without attempting to perform any further testing themselves. The development of local networks prompting collaboration among optometrists, ophthalmologists, and stroke neurologists should facilitate such evaluations, whether in a rapid-access transient ischemic attack clinic, in an emergency department-observation unit, or with hospitalization, depending on local resources. Copyright © 2018 American Academy of Ophthalmology. -
Pediatric Dermatology- Pigmented Lesions
Pediatric Dermatology- Pigmented Lesions OPTI-West/Western University of Health Sciences- Silver Falls Dermatology Presenters: Bryce Lynn Desmond, DO; Ben Perry, DO Contributions from: Lauren Boudreaux, DO; Stephanie Howerter, DO; Collin Blattner, DO; Karsten Johnson, DO Disclosures • We have no financial or conflicts of interest to report Melanocyte Basic Science • Neural crest origin • Migrate to epidermis, dermis, leptomeninges, retina, choroid, iris, mucous membrane epithelium, inner ear, cochlea, vestibular system • Embryology • First appearance at the end of the 1st trimester • Able to synthesize melanin at the beginning of the 2nd trimester • Ratio of melanocytes to basal cells is 1:10 in skin and 1:4 in hair • Equal numbers of melanocytes across different races • Type, number, size, dispersion, and degree of melanization of the melanosomes determines pigmentation Nevus of Ota • A.k.a. Nevus Fuscocoeruleus Ophthalmomaxillaris • Onset at birth (50-60%) or 2nd decade • Larger than mongolian spot, does not typically regress spontaneously • Often first 2 branches of trigeminal nerve • Other involved sites include ipsilateral sclera (~66%), tympanum (55%), nasal mucosa (30%). • ~50 cases of melanoma reported • Reported rates of malignant transformation, 0.5%-25% in Asian populations • Ocular melanoma of choroid, orbit, chiasma, meninges have been observed in patients with clinical ocular hyperpigmentation. • Acquired variation seen in primarily Chinese or Japanese adults is called Hori’s nevus • Tx: Q-switched ruby, alexandrite, and