Abdominal Trauma
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Internal Bleeding
Internal bleeding What is internal bleeding? It is a leakage of blood from the blood vessels of the surrounding tissues because of an injury affect the vessels and lead to rupture. Internal bleeding occurs inside the body cavities such as the head, chest, abdomen, or eye, and it is difficult to detect, because the leaked blood cannot be seen, and the person may not feel its occurrence till the symptoms associated with that bleeding start to appear. Note: It should be noted that people who take anticoagulant drugs are more likely to have this bleeding than others. What are symptoms of abdominal internal bleeding? There are many symptoms developed by the patients of internal bleeding in the abdomen or chest as below: • Feeling of pain in the abdomen. • Shortness of breath. • Feeling of chest pain. • Dizziness upon standing. • Bruises around the navel or on both sides of the abdomen. • Nausea, Vomiting. • Blood in urine. • Dark color stool. What are the symptoms of abdominal internal bleeding? Sometimes, internal bleeding may lead to loss of large amounts of blood, and in this case, the patient will have many symptoms, as below: • Accelerated heart beats • Low blood pressure • Skin sweating • General weakness • Feeling lethargic or feeling sleepy When should I go to seek medical care? Internal bleeding is very dangerous and life threatening and you should visit the doctor when experience one of the following cases:: ✓ After exposure to a severe injury, to ensure that there is no internal bleeding. ✓ Feeling severe pain in the abdomen ✓ Feeling acute shortness of breath ✓ feeling dizzy ✓ Seeing a change in vision Note: When these symptoms are noticed, you should go immediately to medical care or you must call the emergency services to avoid death. -
The European Guideline on Management Of
Rossaint et al. Critical Care (2016) 20:100 DOI 10.1186/s13054-016-1265-x RESEARCH Open Access The European guideline on management of major bleeding and coagulopathy following trauma: fourth edition Rolf Rossaint1, Bertil Bouillon2, Vladimir Cerny3,4,5,6, Timothy J. Coats7, Jacques Duranteau8, Enrique Fernández-Mondéjar9, Daniela Filipescu10, Beverley J. Hunt11, Radko Komadina12, Giuseppe Nardi13, Edmund A. M. Neugebauer14, Yves Ozier15, Louis Riddez16, Arthur Schultz17, Jean-Louis Vincent18 and Donat R. Spahn19* Abstract Background: Severe trauma continues to represent a global public health issue and mortality and morbidity in trauma patients remains substantial. A number of initiatives have aimed to provide guidance on the management of trauma patients. This document focuses on the management of major bleeding and coagulopathy following trauma and encourages adaptation of the guiding principles to each local situation and implementation within each institution. Methods: The pan-European, multidisciplinary Task Force for Advanced Bleeding Care in Trauma was founded in 2004 and included representatives of six relevant European professional societies. The group used a structured, evidence-based consensus approach to address scientific queries that served as the basis for each recommendation and supporting rationale. Expert opinion and current clinical practice were also considered, particularly in areas in which randomised clinical trials have not or cannot be performed. Existing recommendations were reconsidered and revised based on new scientific evidence and observed shifts in clinical practice; new recommendations were formulated to reflect current clinical concerns and areas in which new research data have been generated. This guideline represents the fourth edition of a document first published in 2007 and updated in 2010 and 2013. -
A Rare Case of Penetrating Trauma of Frontal Sinus with Anterior Table Fracture Himanshu Raval1*, Mona Bhatt2 and Nihar Gaur3
ISSN: 2643-4474 Raval et al. Neurosurg Cases Rev 2020, 3:046 DOI: 10.23937/2643-4474/1710046 Volume 3 | Issue 2 Neurosurgery - Cases and Reviews Open Access CASE REPORT Case Report: A Rare Case of Penetrating Trauma of Frontal Sinus with Anterior Table Fracture Himanshu Raval1*, Mona Bhatt2 and Nihar Gaur3 1 Department of Neurosurgery, NHL Municipal Medical College, SVP Hospital Campus, Gujarat, India Check for updates 2Medical Officer, CHC Dolasa, Gujarat, India 3GAIMS-GK General Hospital, Gujarat, India *Corresponding author: Dr. Himanshu Raval, Resident, Department of Neurosurgery, NHL Municipal Medical College, SVP Hospital Campus, Elisbridge, Ahmedabad, Gujarat, 380006, India, Tel: 942-955-3329 Abstract Introduction Background: Head injury is common component of any Road traffic accident (RTA) is the most common road traffic accident injury. Injury involving only frontal sinus cause of cranio-facial injury and involvement of frontal is uncommon and unique as its management algorithm is bone fractures are rare and constitute 5-9% of only fa- changing over time with development of radiological modal- ities as well as endoscopic intervention. Frontal sinus inju- cial trauma. The degree of association has been report- ries may range from isolated anterior table fractures causing ed to be 95% with fractures of the anterior table or wall a simple aesthetic deformity to complex fractures involving of the frontal sinuses, 60% with the orbital rims, and the frontal recess, orbits, skull base, and intracranial con- 60% with complex injuries of the naso-orbital-ethmoid tents. Only anterior table injury of frontal sinus is rare in pen- region, 33% with other orbital wall fractures and 27% etrating head injury without underlying brain injury with his- tory of unconsciousness and questionable convulsion which with Le Fort level fractures. -
Skin Injuries – Can We Determine Timing and Mechanism?
Skin injuries – can we determine timing and mechanism? Jo Tully VFPMS Seminar 2016 What skin injuries do we need to consider? • Bruising • Commonest accidental and inflicted skin injury • Basic principles that can be applied when formulating opinion • Abrasions • Lacerations }we need to be able to tell the difference • Incisions • Stabs/chops • Bite marks – animal v human / inflicted v ‘accidental’ v self-inflicted Our role…. We are often/usually/always asked…………….. • “What type of injury is it?” • “When did this injury occur?” • “How did this injury occur?” • “Was this injury inflicted or accidental?” • IS THIS CHILD ABUSE? • To be able to answer these questions (if we can) we need knowledge of • Anatomy/physiology/healing - injury interpretation • Forces • Mechanisms in relation to development, plausibility • Current evidence Bruising – can we really tell which bruises are caused by abuse? Definitions – bruising • BLUNT FORCE TRAUMA • Bruise =bleeding beneath intact skin due to BFT • Contusion = bruise in deeper tissues • Haematoma - extravasated blood filling a cavity (or potential space). Usually associated with swelling • Petechiae =Pinpoint sized (0.1-2mm) hemorrhages into the skin due to acute rise in venous pressure • medical causes • direct forces • indirect forces Medical Direct Indirect causes mechanical mechanical forces forces Factors affecting development and appearance of a bruise • Properties of impacting object or surface • Force of impact • Duration of impact • Site - properties of body region impacted (blood supply, -
Thoracic and Abdominal Trauma
INJURIESINJURIES TOTO THETHE TRUNKTRUNK THROACIC AND ABDOMINAL INJURIES INITIALINITIAL ASSESSMENTASSESSMENT 1. PRIMARY SURVEY (1. MIN) 2. VITAL FUNCTIONS TREAT LIFE THREATENING FIRST 3. SECONDARY SURVEY 4. DEFINITIVE CARE A.B.C.D.E. LIFELIFE THREATENINGTHREATENING INJURIESINJURIES A. INJURIES TO THE AIRWAYS B. TENSION PTX SUCKING CHEST WOUND MASSIVE HEMOTHORAX FLAIL CHEST C. CARDIAC TAMPONADE MASSIVE HEMOTHORAX LIFELIFE THREATENINGTHREATENING CHESTCHEST INJURIESINJURIES •PNEUMOTHORAX •HEMOTHORAX •PULMONARY CONTUSION •TRACHEBRONCHIAL TREE INJURY •BLUNT CARDIAC INJURY •TRAUMATIC AORTIC INJURY •TRAMATIC DIAPHRAGMATIG INJURY •MEDIASTINAL TRANSVERSING WOUNDS PNEUMOTHORAXPNEUMOTHORAX AIR BETWEEN THE PARIETAL AND VISCERAL PLEURA RIB FRACTURES INJURIES TO THE LUNG INJURIES TO THE AIRWAYS BULLAS IATROGENIG FROM THE RETROPERITONEUM PNEUMOTHORAXPNEUMOTHORAX 1. 2. TENSIONTENSION PNEUMOTHORAXPNEUMOTHORAX ONE WAY VALVE – AIR FROM THE LUNG OR THROUGH THE CHEST WALL INTO THE THORACIC CAVITY CONSEQUENCE: HYPOXIA, BLOCKING OF THE VENOUS INFLOW CHEST PAIN, AIR HUNGER, HYPOTENSION, NECK VEIN DISTENSION, TACHYCARDIA CARDIAC TAMPONADE – NO BREATH SOUNDS IMMEDIATE TREATMENT TENSIONTENSION PNEUMOTHORAXPNEUMOTHORAX TENSION PTX NEEDLE THORACOCENTESIS HEMOTHORAXHEMOTHORAX BLOOD IN THE THORACIC CAVITY LUNG LACERATION RIB FRACTURE INTERCOSTAL VESSEL INJURY ART. MAMMARY INJURY PENETRATING OR BLUNT INJURY HEMOTHORAXHEMOTHORAX 1. ? 2. ! HTXHTX HEMOTHORAXHEMOTHORAX TREATMENT : CHEST TUBE – THORACOTOMY IS RARELY INDICATED THORACOTOMY: 1500 ML / DRAINAGE OR 200 ML/ HOUR -
Anesthesia for Trauma
Anesthesia for Trauma Maribeth Massie, CRNA, MS Staff Nurse Anesthetist, The Johns Hopkins Hospital Assistant Professor/Assistant Program Director Columbia University School of Nursing Program in Nurse Anesthesia OVERVIEW • “It’s not the speed which kills, it’s the sudden stop” Epidemiology of Trauma • ~8% worldwide death rate • Leading cause of death in Americans from 1- 45 years of age • MVC’s leading cause of death • Blunt > penetrating • Often drug abusers, acutely intoxicated, HIV and Hepatitis carriers Epidemiology of Trauma • “Golden Hour” – First hour after injury – 50% of patients die within the first seconds to minutesÆ extent of injuries – 30% of patients die in next few hoursÆ major hemorrhage – Rest may die in weeks Æ sepsis, MOSF Pre-hospital Care • ABC’S – Initial assessment and BLS in trauma – GO TEAM: role of CRNA’s at Maryland Shock Trauma Center • Resuscitation • Reduction of fractures • Extrication of trapped victims • Amputation • Uncooperative patients Initial Management Plan • Airway maintenance with cervical spine protection • Breathing: ventilation and oxygenation • Circulation with hemorrhage control • Disability • Exposure Initial Assessment • Primary Survey: – AIRWAY • ALWAYS ASSUME A CERVICAL SPINE INJURY EXISTS UNTIL PROVEN OTHERWISE • Provide MANUAL IN-LINE NECK STABILIZATION • Jaw-thrust maneuver Initial Assessment • Airway cont’d: – Cervical spine evaluation • Cross table lateral and swimmer’s view Xray • Need to see all seven cervical vertebrae • Only negative CT scan R/O injury Initial Assessment • Cervical -
Wound Classification
Wound Classification Presented by Dr. Karen Zulkowski, D.N.S., RN Montana State University Welcome! Thank you for joining this webinar about how to assess and measure a wound. 2 A Little About Myself… • Associate professor at Montana State University • Executive editor of the Journal of the World Council of Enterstomal Therapists (JWCET) and WCET International Ostomy Guidelines (2014) • Editorial board member of Ostomy Wound Management and Advances in Skin and Wound Care • Legal consultant • Former NPUAP board member 3 Today We Will Talk About • How to assess a wound • How to measure a wound Please make a note of your questions. Your Quality Improvement (QI) Specialists will follow up with you after this webinar to address them. 4 Assessing and Measuring Wounds • You completed a skin assessment and found a wound. • Now you need to determine what type of wound you found. • If it is a pressure ulcer, you need to determine the stage. 5 Assessing and Measuring Wounds This is important because— • Each type of wound has a different etiology. • Treatment may be very different. However— • Not all wounds are clear cut. • The cause may be multifactoral. 6 Types of Wounds • Vascular (arterial, venous, and mixed) • Neuropathic (diabetic) • Moisture-associated dermatitis • Skin tear • Pressure ulcer 7 Mixed Etiologies Many wounds have mixed etiologies. • There may be both venous and arterial insufficiency. • There may be diabetes and pressure characteristics. 8 Moisture-Associated Skin Damage • Also called perineal dermatitis, diaper rash, incontinence-associated dermatitis (often confused with pressure ulcers) • An inflammation of the skin in the perineal area, on and between the buttocks, into the skin folds, and down the inner thighs • Scaling of the skin with papule and vesicle formation: – These may open, with “weeping” of the skin, which exacerbates skin damage. -
Gunshot Wounds
Gunshot Wounds Michael Sirkin, MD Chief, Orthopaedic Trauma Service Assistant Professor, New Jersey Medical School North Jersey Orthopaedic Institute Created March 2004; Reviewed March 2006, August 2010 Ballistics • Most bullets made of lead alloy – High specific gravity • Maximal mass • Less effect of air resistance • Bullet tips – Pointed – Round – Flat – Hollow Ballistics • Low velocity bullets – Made of low melting point lead alloys – If fired from high velocity they melt, 2° to friction • Deform • Change missile ballistics • High velocity bullets – Coated or jacketed with a harder metal – High temperature coating – Less deformity when fired Velocity • Energy = ½ mv2 • Energy increases by the square of the velocity and linearly with the mass • Velocity of missile is the most important factor determining amount of energy and subsequent tissue damage Kinetic Energy of High and Low Velocity Firearms Kinetic Energy of Shotgun Shells Wounding power • Low velocity, less severe – Less than 1000 ft/sec – Less than 230 grams • High velocity, very destructive – Greater than 2000 ft/sec – Weight less than 150 grams • Shotguns, very destructive at close range – About 1200 ft/sec – Weight up to 870 grams Factors that cause tissue damage • Crush and laceration • Secondary missiles • Cavitation • Shock wave Crush and Laceration • Principle mechanism in low velocity gunshot wounds • Material in path is crushed or lacerated • The kinetic energy is dissipated • Increased tissue damage with yaw or tumble – Increased profile – Increased rate of kinetic -
Traumatic Intracranial Aneurysms Due to Penetrating Brain Injury. a Case Report and Suggested Management Guidelines Breck Aaron Jones MD; Alex Patrick Michael MD
Traumatic Intracranial Aneurysms Due to Penetrating Brain Injury. A Case Report and Suggested Management Guidelines Breck Aaron Jones MD; Alex Patrick Michael MD Southern Illinois University School of Medicine Methods Learning Objectives Introduction Angiogram Traumatic intracranial aneurysms A Pubmed search of the literature Identification of traumatic intracranial pertaining to traumatic aneurysms. (TICA) are rare in occurrence and pseudoaneurysms and penetrating Classification of traumatic intracranial equally rare in the literature. Less brain trauma. The literature was aneurysms. than 1% of intracranial aneurysms reviewed for case reports and Treatment and management of are caused by blunt trauma, while management recommendations. traumatic intracranial aneurysms. even fewer are caused by penetrating trauma. Penetrating Results References Traumatic intracranial aneurysm 1.Aarabi B. Management of traumatic aneurysms caused by trauma creates a unique type of high-velocity missile head wounds. Neurosurg Clin N Am. formation is the most commonly aneurysm that does not incorporate Oct 1995;6(4):775-797. described vascular injury after 2.Rao GP, Rao NS, Reddy PK. Technique of removal of an all three vessel wall layers. Because penetrating brain injury. impacted sharp object in a penetrating head injury using the of their rarity, the natural history and Histologically, traumatic aneurysms lever principle. Br J Neurosurg. Dec 1998;12(6):569-571. 3.Vascular complications of penetrating brain injury. J can be described as true management of TICAs are not well Trauma. Aug 2001;51(2 Suppl):S26-28. Angiogram showing traumatic intracranial (incorporating intima, media, defined in the literature. Here we 4.Crompton MR. The pathogenesis of cerebral aneurysms. aneurysm following a gunshot wound to adventitia), false (incorporating one or Brain. -
Pressure Ulcer Staging Cards and Skin Inspection Opportunities.Indd
Pressure Ulcer Staging Pressure Ulcer Staging Suspected Deep Tissue Injury (sDTI): Purple or maroon localized area of discolored Suspected Deep Tissue Injury (sDTI): Purple or maroon localized area of discolored intact skin or blood-fi lled blister due to damage of underlying soft tissue from pressure intact skin or blood-fi lled blister due to damage of underlying soft tissue from pressure and/or shear. The area may be preceded by tissue that is painful, fi rm, mushy, boggy, and/or shear. The area may be preceded by tissue that is painful, fi rm, mushy, boggy, warmer or cooler as compared to adjacent tissue. warmer or cooler as compared to adjacent tissue. Stage 1: Intact skin with non- Stage 1: Intact skin with non- blanchable redness of a localized blanchable redness of a localized area usually over a bony prominence. area usually over a bony prominence. Darkly pigmented skin may not have Darkly pigmented skin may not have visible blanching; its color may differ visible blanching; its color may differ from surrounding area. from surrounding area. Stage 2: Partial thickness loss of Stage 2: Partial thickness loss of dermis presenting as a shallow open dermis presenting as a shallow open ulcer with a red pink wound bed, ulcer with a red pink wound bed, without slough. May also present as without slough. May also present as an intact or open/ruptured serum- an intact or open/ruptured serum- fi lled blister. fi lled blister. Stage 3: Full thickness tissue loss. Stage 3: Full thickness tissue loss. Subcutaneous fat may be visible but Subcutaneous fat may be visible but bone, tendon or muscle are not exposed. -
Symptomatic Intracranial Hemorrhage (Sich) and Activase® (Alteplase) Treatment: Data from Pivotal Clinical Trials and Real-World Analyses
Symptomatic intracranial hemorrhage (sICH) and Activase® (alteplase) treatment: Data from pivotal clinical trials and real-world analyses Indication Activase (alteplase) is indicated for the treatment of acute ischemic stroke. Exclude intracranial hemorrhage as the primary cause of stroke signs and symptoms prior to initiation of treatment. Initiate treatment as soon as possible but within 3 hours after symptom onset. Important Safety Information Contraindications Do not administer Activase to treat acute ischemic stroke in the following situations in which the risk of bleeding is greater than the potential benefit: current intracranial hemorrhage (ICH); subarachnoid hemorrhage; active internal bleeding; recent (within 3 months) intracranial or intraspinal surgery or serious head trauma; presence of intracranial conditions that may increase the risk of bleeding (e.g., some neoplasms, arteriovenous malformations, or aneurysms); bleeding diathesis; and current severe uncontrolled hypertension. Please see select Important Safety Information throughout and the attached full Prescribing Information. Data from parts 1 and 2 of the pivotal NINDS trial NINDS was a 2-part randomized trial of Activase® (alteplase) vs placebo for the treatment of acute ischemic stroke. Part 1 (n=291) assessed changes in neurological deficits 24 hours after the onset of stroke. Part 2 (n=333) assessed if treatment with Activase resulted in clinical benefit at 3 months, defined as minimal or no disability using 4 stroke assessments.1 In part 1, median baseline NIHSS score was 14 (min: 1; max: 37) for Activase- and 14 (min: 1; max: 32) for placebo-treated patients. In part 2, median baseline NIHSS score was 14 (min: 2; max: 37) for Activase- and 15 (min: 2; max: 33) for placebo-treated patients. -
Thoracic Gunshot Wound: a Tanmoy Ganguly1, 1 Report of 3 Cases and Review of Sandeep Kumar Kar , Chaitali Sen1, Management Chiranjib Bhattacharya2, Manasij Mitra3
2015 iMedPub Journals Journal of Universal Surgery http://www.imedpub.com Vol. 3 No. 1:2 ISSN 2254-6758 Thoracic Gunshot Wound: A Tanmoy Ganguly1, 1 Report of 3 Cases and Review of Sandeep Kumar Kar , Chaitali Sen1, Management Chiranjib Bhattacharya2, Manasij Mitra3, 1 Department of Cardiac Anesthesiology, Abstract Institute of Postgraduate Medical Thoracic gunshot injury may have variable presentation and the treatment plan Education and Research, Kolkata, India differs. The risk of injury to heart, major blood vessels and the lungs should be 2 Department of Anesthesiology, Institute evaluated in every patient with rapid clinical examination and basic monitoring and of Postgraduate Medical Education and surgery should be considered as early as possible whenever indicated. The authors Research, Kolkata, India present three cases of thoracic gunshot injury with three different presentations, 3 Krisanganj Medical College, Institute of one with vascular injury, one with parenchymal injury and one case with fortunately Postgraduate Medical Education and no life threatening internal injury. The first case, a 52 year male patient presented Research, Kolkata, India with thoracic gunshot with hemothorax and the bullet trajectory passed very near to the vital structures without injuring them. The second case presented with 2 hours history of thoracic gunshot wound with severe hemodynamic instability. Corresponding author: Sandeep Kumar Surgical exploration revealed an arterial bleeding from within the left lung. The Kar, Assistant Professor third case presented with post gunshot open pneumothorax. All three cases managed successfully with resuscitation and thoracotomy. Preoperative on table fluoroscopy was used for localisation of bullet. [email protected] Keywords: Horacic trauma, Gunshot injury, Traumatic pneumothorax, Emergency thoracotomy, Fluoroscopy.