Periodontal Disease
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Crowded and Rotated Teeth Crowded Teeth Are Common in Small Breed Dogs, While Crowded and Rotated Premolars Are Typically Seen in Brachycephalic Breeds
1 Crowded and rotated teeth Crowded teeth are common in small breed dogs, while crowded and rotated premolars are typically seen in brachycephalic breeds. To some this might suggest that the propensity for crowded, rotated teeth have a hereditary link. Studies show that the smaller the dog, the larger the teeth are in proportion to the mouth when compared to the teeth and mouths of larger dogs. Rotated and/or crowded conditions can occur in a single tooth, in multiple teeth, or in any combination. It is not uncommon to find crowded mandibular incisors in brachycephalic breeds. (Figure 1). Another common finding in many breeds, but especially in brachycephalics, is maxillary third premolars crowded with maxillary fourth premolars or the mandibular fourth premolars crowded with first molars. The maxillary third premolars and mandibular fourth premolars are usually also rotated in this condition .(Figure 2) Another common condition is incisors crowded together but also against the canine teeth Figure 1: Crowding of the mandibular incisors. Treatment included extracting the right and left mandibular second incisors. Figure 2: Crowded and rotated maxillary third premolars in a brachiocephalic dog 2 Rotation and crowding can cause pain from chronic tooth on tooth contact. This might be compared to the pain that humans experience from a caries that has been overfilled. It is a condition that generally does not result in clinical signs; however, it can be quite painful. The chronic trauma resulting from tooth on tooth contact can lead to tooth non vitality. Rotation and crowding can also result in tooth on soft tissue contact, which can be not only painful but can result in soft tissue defects. -
Long-Term Uncontrolled Hereditary Gingival Fibromatosis: a Case Report
Long-term Uncontrolled Hereditary Gingival Fibromatosis: A Case Report Abstract Hereditary gingival fibromatosis (HGF) is a rare condition characterized by varying degrees of gingival hyperplasia. Gingival fibromatosis usually occurs as an isolated disorder or can be associated with a variety of other syndromes. A 33-year-old male patient who had a generalized severe gingival overgrowth covering two thirds of almost all maxillary and mandibular teeth is reported. A mucoperiosteal flap was performed using interdental and crevicular incisions to remove excess gingival tissues and an internal bevel incision to reflect flaps. The patient was treated 15 years ago in the same clinical facility using the same treatment strategy. There was no recurrence one year following the most recent surgery. Keywords: Gingival hyperplasia, hereditary gingival hyperplasia, HGF, hereditary disease, therapy, mucoperiostal flap Citation: S¸engün D, Hatipog˘lu H, Hatipog˘lu MG. Long-term Uncontrolled Hereditary Gingival Fibromatosis: A Case Report. J Contemp Dent Pract 2007 January;(8)1:090-096. © Seer Publishing 1 The Journal of Contemporary Dental Practice, Volume 8, No. 1, January 1, 2007 Introduction Hereditary gingival fibromatosis (HGF), also Ankara, Turkey with a complaint of recurrent known as elephantiasis gingiva, hereditary generalized gingival overgrowth. The patient gingival hyperplasia, idiopathic fibromatosis, had presented himself for examination at the and hypertrophied gingival, is a rare condition same clinic with the same complaint 15 years (1:750000)1 which can present as an isolated ago. At that time, he was treated with full-mouth disorder or more rarely as a syndrome periodontal surgery after the diagnosis of HGF component.2,3 This condition is characterized by had been made following clinical and histological a slow and progressive enlargement of both the examination (Figures 1 A-B). -
Veterinary Dentistry Extraction
Veterinary Dentistry Extraction Introduction The extraction of teeth in the dog and cat require specific skills. In this chapter the basic removal technique for a single rooted incisor tooth is developed for multi-rooted and canine teeth. Deciduous teeth a nd feline teeth, particularly those affected by odontoclastic resorptive lesions, also require special attention. Good technique requires careful planning. Consider if extraction is necessary, and if so, how is it best accomplished. Review the root morphology and surrounding structures using pre-operative radiographs. Make sure you have all the equipment you need, and plan pre and post-operative management. By the end of this chapter you should be able to: ü Know the indications for extracting a tooth ü Unders tand the differing root morphology of dog and cat teeth ü Be able to select an extraction technique and equipment for any individual tooth ü Know of potential complications and how to deal with them ü Be able to apply appropriate analgesic and other treatment. Indications for Extraction Mobile Teeth Mobile teeth are caused by advanced periodontal disease and bone loss. Crowding of Teeth Retained deciduous canine. Teeth should be considered for extraction when they are interfering with occlusion or crowding others (e.g. supernumerary teeth). Retained Deciduous Teeth Never have two teeth of the same type in the same place at the same time. This is the rule of dental succession. Teeth in the Line of a Fracture Consider extracting any teeth in the line of a fracture of the mandible or maxilla. Teeth Destroyed by Disease Teeth ruined by advanced caries, feline neck lesions etc. -
Feline Dentistry: Cats Are Not Small Dogs Matt Lemmons, DVM, DAVDC Medvet Indianapolis Carmel, IN
Basics for Practitioners: Oral Anatomy and Pathology Matt Lemmons, DVM, DAVDC MedVet Indianapolis Carmel, IN Dentistry is truly a branch of medicine and surgery. A strong knowledge of normal anatomy and pathology is cornerstone to adequate diagnosis and treatment of diseases of the oral cavity. The majority of oral related disease is inflammatory (periodontal disease) or traumatic (fractured teeth, orthopedic injuries) in nature. However other causes are not rare and need to be recognized. The basic dental unit is the tooth and surrounding periodontium. The tooth consists of the crown and root. The crown is covered in enamel and the root by cementum. Deep to the crown and cementum is the dentin. Dentin is a porous hard tissue which continuously grows toward the center of the tooth as long as the tooth is vital. Deep to the dentin is the pulp which consists of nerves, blood vessels, connective tissue, fibroblasts and odontoblasts. The periodontium is composed of the cementum, periodontal ligament, alveolar bone, and gingiva. The periodontal ligament serves to anchor the cementum to the alveolar bone, act as a shock absorber and aid in sensation. The gingiva is attached to the bone (attached gingiva), tooth by connective tissue and the most apical extent is not attached and is known as the free gingiva. The potential space between the free gingiva and tooth and ending apically at the sulcular epithelium is the gingival sulcus. In health this should be less than 3mm in depth in dogs and 1mm in cats. When addressing the teeth and periodontium, directional nomenclature is not similar to directional nomenclature of the rest of the body. -
Dentinal Hypersensitivity: a Review
Dentinal Hypersensitivity: A Review Abstract Dentinal hypersensitivity is generally reported by the patient after experiencing a sharp pain caused by one of several different stimuli. The pain response varies substantially from one person to another. The condition generally involves the facial surfaces of teeth near the cervical aspect and is very common in premolars and canines. The most widely accepted theory of how the pain occurs is Brannstrom’s hydrodynamic theory, fluid movement within the dentinal tubules. The dental professional, using a variety of diagnostic techniques, will discern the condition from other conditions that may cause sensitive teeth. Treatment of the condition can be invasive or non-invasive in nature. The most inexpensive and efficacious first line of treatment for most patients is a dentifrice containing a desensitizing active ingredient such as potassium nitrate and/or stannous fluoride. This review will address the prevalence, diagnosis, and treatment of dentinal hypersensitivity. In addition the home care recommendations will focus on desensitizing dentifrices. Keywords: Dentinal hypersensitivity, hydrodynamic theory, stannous fluoride, potassium nitrate Citation: Walters PA. Dentinal Hypersensitivity: A Review. J Contemp Dent Pract 2005 May;(6)2:107-117. © Seer Publishing 1 The Journal of Contemporary Dental Practice, Volume 6, No. 2, May 15, 2005 Introduction The prevalence of dentinal hypersensitivity Dentifrices and mouth rinses are routinely used has been reported over the years in a variety as a delivery system for therapeutic agents of ways: as greater than 40 million people such as antimicrobials and anti-sensitivity in the U.S. annually1, 14.3% of all dental agents. Therapeutic oral care products are patients2, between 8% and 57% of adult dentate available to assist the patient in the control of population3, and up to 30% of adults at some time dental caries, calculus formation, and dentinal during their lifetime.4 hypersensitivity to name a few. -
Periodontal Health, Gingival Diseases and Conditions 99 Section 1 Periodontal Health
CHAPTER Periodontal Health, Gingival Diseases 6 and Conditions Section 1 Periodontal Health 99 Section 2 Dental Plaque-Induced Gingival Conditions 101 Classification of Plaque-Induced Gingivitis and Modifying Factors Plaque-Induced Gingivitis Modifying Factors of Plaque-Induced Gingivitis Drug-Influenced Gingival Enlargements Section 3 Non–Plaque-Induced Gingival Diseases 111 Description of Selected Disease Disorders Description of Selected Inflammatory and Immune Conditions and Lesions Section 4 Focus on Patients 117 Clinical Patient Care Ethical Dilemma Clinical Application. Examination of the gingiva is part of every patient visit. In this context, a thorough clinical and radiographic assessment of the patient’s gingival tissues provides the dental practitioner with invaluable diagnostic information that is critical to determining the health status of the gingiva. The dental hygienist is often the first member of the dental team to be able to detect the early signs of periodontal disease. In 2017, the American Academy of Periodontology (AAP) and the European Federation of Periodontology (EFP) developed a new worldwide classification scheme for periodontal and peri-implant diseases and conditions. Included in the new classification scheme is the category called “periodontal health, gingival diseases/conditions.” Therefore, this chapter will first review the parameters that define periodontal health. Appreciating what constitutes as periodontal health serves as the basis for the dental provider to have a stronger understanding of the different categories of gingival diseases and conditions that are commonly encountered in clinical practice. Learning Objectives • Define periodontal health and be able to describe the clinical features that are consistent with signs of periodontal health. • List the two major subdivisions of gingival disease as established by the American Academy of Periodontology and the European Federation of Periodontology. -
Third Molar (Wisdom) Teeth
Third molar (wisdom) teeth This information leaflet is for patients who may need to have their third molar (wisdom) teeth removed. It explains why they may need to be removed, what is involved and any risks or complications that there may be. Please take the opportunity to read this leaflet before seeing the surgeon for consultation. The surgeon will explain what treatment is required for you and how these issues may affect you. They will also answer any of your questions. What are wisdom teeth? Third molar (wisdom) teeth are the last teeth to erupt into the mouth. People will normally develop four wisdom teeth: two on each side of the mouth, one on the bottom jaw and one on the top jaw. These would normally erupt between the ages of 18-24 years. Some people can develop less than four wisdom teeth and, occasionally, others can develop more than four. A wisdom tooth can fail to erupt properly into the mouth and can become stuck, either under the gum, or as it pushes through the gum – this is referred to as an impacted wisdom tooth. Sometimes the wisdom tooth will not become impacted and will erupt and function normally. Both impacted and non-impacted wisdom teeth can cause problems for people. Some of these problems can cause symptoms such as pain & swelling, however other wisdom teeth may have no symptoms at all but will still cause problems in the mouth. People often develop problems soon after their wisdom teeth erupt but others may not cause problems until later on in life. -
Desquamative Gingivitis Desquamative Gingivitis
DOI: 10.5772/intechopen.69268 Provisional chapter Chapter 1 Desquamative Gingivitis Desquamative Gingivitis Hiroyasu Endo, Terry D. Rees, Hideo Niwa, HiroyasuKayo Kuyama, Endo, Morio Terry D.Iijima, Rees, Ryuuichi Hideo Niwa, KayoImamura, Kuyama, Takao Morio Kato, Iijima, Kenji Doi,Ryuuichi Hirotsugu Imamura, TakaoYamamoto Kato, and Kenji Takanori Doi, Hirotsugu Ito Yamamoto and TakanoriAdditional information Ito is available at the end of the chapter Additional information is available at the end of the chapter http://dx.doi.org/10.5772/intechopen.69268 Abstract Desquamative gingivitis (DG) is characterized by erythematous, epithelial desquama‐ tion, erosion of the gingival epithelium, and blister formation on the gingiva. DG is a clinical feature of a variety of diseases or disorders. Most cases of DG are associated with mucocutaneous diseases, the most common ones being lichen planus, mucous membrane pemphigoid, and pemphigus vulgaris. Proper diagnosis of the underlying cause is important because the prognosis varies, depending on the disease. This chapter presents the underlying etiology that is most commonly associated with DG. The current literature on the diagnostic and management modalities of patients with DG is reviewed. Keywords: gingival diseases/pemphigus/pemphigoid, benign mucous membrane/lichen planus, oral/hypersensitivity/autoimmune diseases 1. Introduction Manifestations of desquamative gingivitis (DG) include erythematous gingiva, epithelial des‐ quamation, and erosion of the gingival epithelium, as well as blister formation on the gingiva [1, 2] (Figure 1). The DG lesions may be localized or generalized and may extend into the alveolar mucosa. Similar lesions are often found on the buccal mucosa, tongue, and palate in the oral cavity. The signs of DG are clearly different from those of dental plaque‐induced gingivitis. -
Periodontal Disease)
Patient Fact Sheet Gum Disease (Periodontal Disease) It can often go unnoticed – until it’s too late. While you may not think periodontal (gum) disease affects you, 75 percent of adults over the age of 35 show signs and symptoms. In fact, periodontal disease is the leading cause of tooth loss in adults. Why? Because it occurs at an age when cavities are usually a thing of the past and the initial symptoms often go unnoticed. Recent studies have also shown a possible link between periodontal disease and heart disease. One theory in support of this is that the bacteria that cause periodontal disease enter the bloodstream and promote blood clots and narrowing of the arteries that cause heart attacks. It has also been shown that if a woman develops severe periodontal disease during pregnancy, she is more likely to give birth to a low birth weight infant. Research also shows periodontal disease is linked to many other health problems, as well. What is gum (periodontal) If I have no symptoms, how do I How can I prevent periodontal disease? What causes it? know if I have gum disease? disease? Periodontal disease, or gum disease, is a bacterial Periodontal disease can be easily detected by your • Brush your teeth twice a day with a soft-bristled infection of the gums, ligaments and bone that support general dentist or a periodontist (a specialist in toothbrush. Hold the brush at a 45-degree angle to the teeth and anchor them in the jaw. The bacteria, periodontal diseases) during regular dental the gum line and gently clean where the gums meet which act mainly on certain carbohydrates in our diets, examinations.Therefore, regular checkups, ideally every your teeth. -
Lance Canines: an Illustrated Exploration I Would Like to Discuss a Recent Case of a Young Sheltie with a Couple of Dental Problems
Lance Canines: An Illustrated Exploration I would like to discuss a recent case of a young sheltie with a couple of dental problems. The first problem was a common one in shelties and is variably called lance canines, rostrally displaced maxillary canines or mesially displaced maxillary canines. Whatever name you choose, the problem is that the permanent maxillary canine teeth erupt pointing in the wrong direction. To understand a problem, you must first understand normal so I will review the normal relationship of the canine triad. The canine triad is composed of the maxillary lateral incisor and the canine teeth on one side and is depicted in Figure 1. In this picture, you can see that the crowns of the canine teeth are basically vertical. There is a large space between the maxillary lateral incisor and the maxillary canine and this space is known as a diastema. When the mouth is closed, the mandibular canine crown resides in the centre of the diastema so that it does not contact either of the other teeth in the triad. Figure 1: The normal canine triad. In affected shelties, one or both of the maxillary canines is malpositioned so that it is lying more horizontally. As such, the crown crosses the diastema and blocks the mandibular canine out as in Figure 2. Now on closure, the mandibular canine contacts the maxillary canine and is often forced to tip labially. Owners notice this because the mandibular canine then starts to catch on the upper lip. There is variability in the degree to which the maxillary canine is malpositioned. -
Hereditary Gingival Fibromatosis, Inherited Disease, Gingivectomy
Clinical Practice 2014, 3(1): 7-10 DOI: 10.5923/j.cp.20140301.03 Hereditary Gingival Fibromatosis - A Case Report Anand Kishore1,*, Vivek Srivastava2, Ajeeta Meenawat2, Ambrish Kaushal3 1King George Medical College, Lucknow 2BBD College of dental sciences 3Chandra Dental College & Hospital Abstract Hereditary gingival fibromatosis is characterized by a slow benign enlargement of gingival tissue. It causes teeth being partially or totally covered by enlarged gingiva, causing esthetic and functional problems. It is usually transmitted both as autosomal dominant trait and autosomal recessive inheritance although sporadic cases are commonly reported. This paper reports three cases of gingival fibromatosis out of which one was in a 15 year old girl treated with convectional gingivectomy. Keywords Hereditary gingival fibromatosis, Inherited disease, Gingivectomy having the gingival enlargement before the patient’s birth 1. Introduction and she got operated in the village government hospital. No further relevant medical history was present. Hereditary gingival fibromatosis (HGF) or Idiopathic gingival fibromatosis is a rare, benign, asymptomatic, non-hemorrhagic and non-exudative proliferative fibrous lesion of gingival tissue occurring equally among men and women, in both arches with varying intensity in individuals within the same family [1]. It occurs as an autosomal dominant condition although recessive form also does occur. Consanguinity seems to increase the risk of autosomal dominant inheritance. It affects the marginal gingival, attached gingival and interdental papilla presenting as pink, non-hemorrhagic and have a firm, fibrotic consistency [2]. It also shows a generalized firm nodular enlargement with smooth to stippled surfaces and minimal tendency to bleed. Figure 1. Gingival enlargement However, in some cases the enlargement can be so firm and dense that it feels like bone on palpation [3]. -
Oral Diseases Associated with Human Herpes Viruses: Aetiology, Clinical Features, Diagnosis and Management
www.sada.co.za / SADJ Vol 71 No. 6 CLINICAL REVIEW < 253 Oral diseases associated with human herpes viruses: aetiology, clinical features, diagnosis and management SADJ July 2016, Vol 71 no 6 p253 - p259 R Ballyram1, NH Wood2, RAG Khammissa3, J Lemmer4, L Feller5 ABSTRACT Human herpesviruses (HHVs) are very prevalent DNA ACRONYMS viruses that can cause a variety of orofacial diseases. EM: erythema multiforme Typically they are highly infectious, are contracted early in HHV: human herpes virus life, and following primary infection, usually persist in a latent form. Primary oral infections are often subclinical, but may PCR: polymerase chain reaction be symptomatic as in the case of herpes simplex virus- HSV, HHV-1: herpes simplex virus induced primary herpetic gingivostomatitis. Reactivation VZV, HHV-3: varicella-zoster virus of the latent forms may result in various conditions: herpes EBV, HHV-4: Epstein-Barr virus simplex virus (HSV) can cause recurrent herpetic orolabial CMV, HHV-5: cytomegalovirus lesions; varicella zoster virus (VZV) can cause herpes zoster; Epstein-Barr virus (EBV) can cause oral hairy Key words: herpes simplex virus, human herpes virus-8, leukoplakia; and reactivation of HHV-8 can cause Kaposi varicella zoster virus, Epstein-Barr virus, recurrent herpes sarcoma. In immunocompromised subjects, infections labialis, recurrent intraoral herpetic ulcers, treatment, val- with human herpesviruses are more extensive and aciclovir, aciclovir, famcicylovir. severe than in immunocompetent subjects. HSV and VZV infections are treated with nucleoside analogues aciclovir, valaciclovir, famciclovir and penciclovir. These agents INTRODucTION have few side effects and are effective when started The human herpesvirus (HHV) family comprises a diverse early in the course of the disease.