Dysphagia, Aspiration, and Choking Jean Herrick, MA, OTR/L Occupational Therapist REACH Clinic DDS Northeast Region
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
General Signs and Symptoms of Abdominal Diseases
General signs and symptoms of abdominal diseases Dr. Förhécz Zsolt Semmelweis University 3rd Department of Internal Medicine Faculty of Medicine, 3rd Year 2018/2019 1st Semester • For descriptive purposes, the abdomen is divided by imaginary lines crossing at the umbilicus, forming the right upper, right lower, left upper, and left lower quadrants. • Another system divides the abdomen into nine sections. Terms for three of them are commonly used: epigastric, umbilical, and hypogastric, or suprapubic Common or Concerning Symptoms • Indigestion or anorexia • Nausea, vomiting, or hematemesis • Abdominal pain • Dysphagia and/or odynophagia • Change in bowel function • Constipation or diarrhea • Jaundice “How is your appetite?” • Anorexia, nausea, vomiting in many gastrointestinal disorders; and – also in pregnancy, – diabetic ketoacidosis, – adrenal insufficiency, – hypercalcemia, – uremia, – liver disease, – emotional states, – adverse drug reactions – Induced but without nausea in anorexia/ bulimia. • Anorexia is a loss or lack of appetite. • Some patients may not actually vomit but raise esophageal or gastric contents in the absence of nausea or retching, called regurgitation. – in esophageal narrowing from stricture or cancer; also with incompetent gastroesophageal sphincter • Ask about any vomitus or regurgitated material and inspect it yourself if possible!!!! – What color is it? – What does the vomitus smell like? – How much has there been? – Ask specifically if it contains any blood and try to determine how much? • Fecal odor – in small bowel obstruction – or gastrocolic fistula • Gastric juice is clear or mucoid. Small amounts of yellowish or greenish bile are common and have no special significance. • Brownish or blackish vomitus with a “coffee- grounds” appearance suggests blood altered by gastric acid. -
Obstructive Sleep Apnea
ObSTruCTIve Sleep ApneA provider’s guide to diagnose and code sleep apnea Sleep apnea is a common disorder that by When reviewing these symptoms it is helpful definition is characterized by a reduction in to clarify the history with the patient’s sleeping normal breathing during hours of sleep, often partner, when available. The most useful symptom related to the collapse of the soft tissues in the for identifying patients with OSA is nocturnal back of the throat. Obstructive sleep apnea (OSA) choking or gasping. Snoring alone is not a is the most common sleeping disorder. It has been diagnostic predictor for OSA. However, the lack diagnosed in 3 to 7% of Americans. It is estimated of snoring and/or presence of apnea reduce the that 20% of the entire American population has not likelihood of an OSA diagnosis. been diagnosed. Quantification of the patient’s perception Independent risk factors for of daytime sleepiness and/or fatigue is an important historical finding. This can be developing OSA include: determined by using the Epworth Sleepiness › Obesity (BMI > 30 kg/m2) Scale (epworthsleepinessscale.com). A score of 10 supports the hypothesis of excessive daytime › African – American race sleepiness, which should prompt the clinician to › Male gender have the patient tested for OSA. › Advancing age › Cranio – facial anomalies The physical examination should focus on: Smoking › 1. Review of the oral airway, specifically: › Controlled substance use and alcohol intake the size of the uvula and tonsils, and › Chronic medical conditions such as: the presence of nasal septal deviation end-stage renal disease, congestive heart failure, 2. -
Osteopathic Approach to the Spleen
Osteopathic approach to the spleen Luc Peeters and Grégoire Lason 1. Introduction the first 3 years to 4 - 6 times the birth size. The position therefore progressively becomes more lateral in place of The spleen is an organ that is all too often neglected in the original epigastric position. The spleen is found pos- the clinic, most likely because conditions of the spleen do tero-latero-superior from the stomach, its arterial supply is not tend to present a defined clinical picture. Furthermore, via the splenic artery and the left gastroepiploic artery it has long been thought that the spleen, like the tonsils, is (Figure 2). The venous drainage is via the splenic vein an organ that is superfluous in the adult. into the portal vein (Figure 2). The spleen is actually the largest lymphoid organ in the body and is implicated within the blood circulation. In the foetus it is an organ involved in haematogenesis while in the adult it produces lymphocytes. The spleen is for the blood what the lymph nodes are for the lymphatic system. The spleen also purifies and filters the blood by removing dead cells and foreign materials out of the circulation The function of red blood cell reserve is also essential for the maintenance of human activity. Osteopaths often identify splenic congestion under the influence of poor diaphragm function. Some of the symptoms that can be associated with dysfunction of the spleen are: Figure 2 – Position and vascularisation of the spleen Anaemia in children Disorders of blood development Gingivitis, painful and bleeding gums Swollen, painful tongue, dysphagia and glossitis Fatigue, hyperirritability and restlessness due to the anaemia Vertigo and tinnitus Frequent colds and infections due to decreased resis- tance Thrombocytosis Tension headaches The spleen is also considered an important organ by the osteopath as it plays a role in the immunity, the reaction of the circulation and oxygen transport during effort as well as in regulation of the blood pressure. -
Hemoptysis in Children
R E V I E W A R T I C L E Hemoptysis in Children G S GAUDE From Department of Pulmonary Medicine, JN Medical College, Belgaum, Karnataka, India. Correspondence to: Dr G S Gaude, Professor and Head, Department of Pulmonary Medicine, J N Medical College, Belgaum 590 010, Karnataka, India. [email protected] Received: November, 11, 2008; Initial review: May, 8, 2009; Accepted: July 27, 2009. Context: Pulmonary hemorrhage and hemoptysis are uncommon in childhood, and the frequency with which they are encountered by the pediatrician depends largely on the special interests of the center to which the child is referred. Diagnosis and management of hemoptysis in this age group requires knowledge and skill in the causes and management of this infrequently occurring potentially life-threatening condition. Evidence acquisition: We reviewed the causes and treatment options for hemoptysis in the pediatric patient using Medline and Pubmed. Results: A focused physical examination can lead to the diagnosis of hemoptysis in most of the cases. In children, lower respiratory tract infection and foreign body aspiration are common causes. Chest radiographs often aid in diagnosis and assist in using two complementary diagnostic procedures, fiberoptic bronchoscopy and high-resolution computed tomography. The goals of management are threefold: bleeding cessation, aspiration prevention, and treatment of the underlying cause. Mild hemoptysis often is caused by an infection that can be managed on an outpatient basis with close monitoring. Massive hemoptysis may require additional therapeutic options such as therapeutic bronchoscopy, angiography with embolization, and surgical intervention such as resection or revascularization. Conclusions: Hemoptysis in the pediatric patient requires prompt and thorough evaluation and treatment. -
17 Nutrition for Patients with Upper Gastrointestinal Disorders 403
84542_ch17.qxd 7/16/09 6:35 PM Page 402 Nutrition for Patients with Upper 17 Gastrointestinal Disorders TRUE FALSE 1 People who have nausea should avoid liquids with meals. 2 Thin liquids, such as clear juices and clear broths, are usually the easiest items to swallow for patients with dysphagia. 3 All patients with dysphagia are given solid foods in pureed form. 4 In people with GERD, the severity of the pain reflects the extent of esophageal damage. 5 High-fat meals may trigger symptoms of GERD. 6 People with esophagitis may benefit from avoiding spicy or acidic foods. 7 Alcohol stimulates gastric acid secretion. 8 A bland diet promotes healing of peptic ulcers. 9 People with dumping syndrome should avoid sweets and sugars. 10 Pernicious anemia is a potential complication of gastric surgery. UPON COMPLETION OF THIS CHAPTER, YOU WILL BE ABLE TO ● Give examples of ways to promote eating in people with anorexia. ● Describe nutrition interventions that may help maximize intake in people who have nausea. ● Compare the three levels of solid food textures included in the National Dysphagia Diet. ● Compare the four liquid consistencies included in the National Dysphagia Diet. ● Plan a menu appropriate for someone with GERD. ● Teach a patient about role of nutrition therapy in the treatment of peptic ulcer disease. ● Give examples of nutrition therapy recommendations for people experiencing dumping syndrome. utrition therapy is used in the treatment of many digestive system disorders. For many disorders, diet merely plays a supportive role in alleviating symptoms rather than alter- ing the course of the disease. -
Dysphagia - Pathophysiology of Swallowing Dysfunction, Symptoms, Diagnosis and Treatment
ISSN: 2572-4193 Philipsen. J Otolaryngol Rhinol 2019, 5:063 DOI: 10.23937/2572-4193.1510063 Volume 5 | Issue 3 Journal of Open Access Otolaryngology and Rhinology REVIEW ARTICLE Dysphagia - Pathophysiology of Swallowing Dysfunction, Symptoms, Diagnosis and Treatment * Bahareh Bakhshaie Philipsen Check for updates Department of Otorhinolaryngology-Head and Neck Surgery, Odense University Hospital, Denmark *Corresponding author: Dr. Bahareh Bakhshaie Philipsen, Department of Otorhinolaryngology-Head and Neck Surgery, Odense University Hospital, Sdr. Boulevard 29, 5000 Odense C, Denmark, Tel: +45 31329298, Fax: +45 66192615 the vocal folds adduct to prevent aspiration. The esoph- Abstract ageal phase is completely involuntary and consists of Difficulty swallowing is called dysphagia. There is a wide peristaltic waves [2]. range of potential causes of dysphagia. Because there are many reasons why dysphagia can occur, treatment Dysphagia is classified into the following major depends on the underlying cause. Thorough examination types: is important, and implementation of a treatment strategy should be based on evaluation by a multidisciplinary team. 1. Oropharyngeal dysphagia In this article, we will describe the mechanism of swallowing, the pathophysiology of swallowing dysfunction and different 2. Esophageal dysphagia causes of dysphagia, along with signs and symptoms asso- 3. Complex neuromuscular disorders ciated with dysphagia, diagnosis, and potential treatments. 4. Functional dysphagia Keywords Pathophysiology Dysphagia, Deglutition, Deglutition disorders, FEES, Video- fluoroscopy Swallowing is a complex process and many distur- bances in oropharyngeal and esophageal physiology including neurologic deficits, obstruction, fibrosis, struc- Introduction tural damage or congenital and developmental condi- Dysphagia is derived from the Greek phagein, means tions can result in dysphagia. Breathing difficulties can “to eat” [1]. -
Abdominal Pain - Gastroesophageal Reflux Disease
ACS/ASE Medical Student Core Curriculum Abdominal Pain - Gastroesophageal Reflux Disease ABDOMINAL PAIN - GASTROESOPHAGEAL REFLUX DISEASE Epidemiology and Pathophysiology Gastroesophageal reflux disease (GERD) is one of the most commonly encountered benign foregut disorders. Approximately 20-40% of adults in the United States experience chronic GERD symptoms, and these rates are rising rapidly. GERD is the most common gastrointestinal-related disorder that is managed in outpatient primary care clinics. GERD is defined as a condition which develops when stomach contents reflux into the esophagus causing bothersome symptoms and/or complications. Mechanical failure of the antireflux mechanism is considered the cause of GERD. Mechanical failure can be secondary to functional defects of the lower esophageal sphincter or anatomic defects that result from a hiatal or paraesophageal hernia. These defects can include widening of the diaphragmatic hiatus, disturbance of the angle of His, loss of the gastroesophageal flap valve, displacement of lower esophageal sphincter into the chest, and/or failure of the phrenoesophageal membrane. Symptoms, however, can be accentuated by a variety of factors including dietary habits, eating behaviors, obesity, pregnancy, medications, delayed gastric emptying, altered esophageal mucosal resistance, and/or impaired esophageal clearance. Signs and Symptoms Typical GERD symptoms include heartburn, regurgitation, dysphagia, excessive eructation, and epigastric pain. Patients can also present with extra-esophageal symptoms including cough, hoarse voice, sore throat, and/or globus. GERD can present with a wide spectrum of disease severity ranging from mild, intermittent symptoms to severe, daily symptoms with associated esophageal and/or airway damage. For example, severe GERD can contribute to shortness of breath, worsening asthma, and/or recurrent aspiration pneumonia. -
High Risk Percutaneous Endoscopic Gastrostomy Tubes: Issues to Consider
NUTRITIONINFLAMMATORY ISSUES BOWEL IN GASTROENTEROLOGY, DISEASE: A PRACTICAL SERIES APPROACH, #105 SERIES #73 Carol Rees Parrish, M.S., R.D., Series Editor High Risk Percutaneous Endoscopic Gastrostomy Tubes: Issues to Consider Iris Vance Neeral Shah Percutaneous endoscopy gastrostomy (PEG) tubes are a valuable tool for providing long- term enteral nutrition or gastric decompression; certain circumstances that complicate PEG placement warrant novel approaches and merit review and discussion. Ascites and portal hypertension with varices have been associated with poorer outcomes. Bleeding is one of the most common serious complications affecting approximately 2.5% of all procedures. This article will review what evidence exists in these high risk scenarios and attempt to provide more clarity when considering these challenging clinical circumstances. INTRODUCTION ince the first Percutaneous Endoscopic has been found by multiple authors to portend a poor Gastrostomy tube was placed in 1979 (1), they prognosis in PEG placement (3,4, 5,6,7,8). This review Shave become an invaluable tool for providing will endeavor to provide more clarity when considering long-term enteral nutrition (EN) and are commonly used these challenging clinical circumstances. in patients with dysphagia following stroke, disabling motor neuron diseases such as multiple sclerosis and Ascites & Gastric Varices amyotrophic lateral sclerosis, and in those with head The presence of ascites is frequently viewed as a and neck cancer.They are also used for patients with relative, if not absolute, contraindication to PEG prolonged mechanical intubation, as well as gastric placement. Ascites adds technical difficulties and the decompression in those with severe gastroparesis, risk for potential complications (see Table 1). -
Silent Reflux (Also Called LPR Or EOR)
Silent reflux (also called LPR or EOR) This leaflet explains what your condition is, why it happens, what the symptoms are and how it can be managed. If there is anything you don’t understand or if you have any further questions please talk to your doctor or nurse. What is silent reflux? Everyone has juices in the stomach which are acidic and digest and break down food. At the top of the stomach there is a muscular valve which closes to prevent food and stomach juices escaping upwards into the gullet. If this muscular valve (oesophageal sphincter) does not work very well, the stomach juices can leak backwards into the gullet, causing reflux or symptoms of indigestion (heartburn). However, in some people, small amounts of stomach juice can spill even further back into the back of your throat, affecting the throat lining and your voice box (larynx) and causing irritation and hoarseness. This is known as laryngo pharyngeal reflux (LPR) or extra oesophageal reflux (EOR). Its common name is 'silent reflux' because many people do not experience any of the classic symptoms of heartburn or indigestion. Silent reflux can occur during the day or night, even if a person hasn't eaten anything. Usually, however, silent reflux occurs at night. What are the symptoms of silent reflux? The most common symptoms are: • A sensation of food sticking or a feeling of a lump in the throat. • A hoarse, tight or 'croaky' voice. • Frequent throat clearing. • Difficulty swallowing (especially tablets or solid foods). • A sore, dry and sensitive throat. • Occasional unpleasant "acid" or "bilious" taste at the back of the mouth. -
Muscle Tension Dysphagia?
What is Muscle Tension Dysphagia? Jenny Erpenbeck, MS CCC-SLP Division of Laryngology Department of Otolaryngology – Head and Neck Surgery Case Study – “Dan” • 28 y/o male referred by PCP • 8 months of progressive dysphagia after a URI • Symptoms: – Solids sticking in the back of his mouth – Feels as if he “cannot swallow” – Describes swallowing as effortful • Current diet: puree and thin liquids • 25-35 lbs weight loss since onset • No pneumonia Work Up • TNE and EGD: WNL • Questionnaires: – EAT 10: 35/40 – VHI 10: 0/40 • Laryngostroboscopy: WNL, supraglottic compression during phonation • MBSS… What Now? • Normal work up revealing no organic etiology for swallowing issues • But…huge impact of dysphagia symptoms on oral intake and quality of life Functional dysphagia, swallowing phobia, psychogenic dysphagia, somatic dysphagia? But there’s a problem with these terms… • They reflect the lack of mechanistic understanding and clear etiology • As a result, receiving care is difficult – No clear disciplinary home – No standards of care and guidelines – Patients feel dismissed and distance themselves from the label Psychogenic Dysphagia and Globus: Reevaluation of 23 Patients (Ravich et al, 1989) • Documented an explanation for symptoms in 15 patients (65%) • “Once attribution of symptoms to psychogenic causes is made, the diagnosis is rarely reconsidered” Muscle Tension Dysphagia (MTDg): Symptomology and Theoretical Framework (Kang 2016) • Case series with chart review • N = 67 • Common symptoms: – Difficulty swallowing solids (37%) – Throat -
Tolerability and Product Properties of a Gum-Containing Thickener in Patients with Dysphagia Linda Killeen1,Bsc,Mirianlansink2, Phd & Dea Schröder3,Bsc
FEATURE Tolerability and Product Properties of a Gum-Containing Thickener in Patients With Dysphagia Linda Killeen1,BSc,MirianLansink2, PhD & Dea Schröder3,BSc Abstract Purpose: The aim of the study was to determine the gastrointestinal (GI) tolerability of drinks and foods thickened with a gum- containing thickener compared to a starch-based thickener in patients with dysphagia. Design: A randomized, double-blind, controlled, parallel group study. Methods: Subjects started with a 3-day run-in period on a starch-based thickener and continued with a 14-day intervention on either the starch-based or gum-containing thickener. GI tolerance parameters were recorded at baseline and for three consecutive days in both weeks. Product properties were studied using a feedback questionnaire from carers. Findings: Incidence and intensity of GI symptoms was low and not significantly different between groups. Carers indicated that starch-thickened drinks became significantly thinner with time compared to gum-containing thickened drinks (p =.029). Conclusions and Clinical Relevance: No differences in GI tolerance parameters between groups were observed. We hypothesize that use of the gum-containing thickener is preferred to a starch-based thickener due to the stability of its viscosity during consumption. Key words: Gastrointestinal tolerability; dysphagia; tara gum; humans. Introduction accident (Martino et al., 2005), up to 82% of patients with Parkinson’s disease (Kalf, de Swart, Bloem, & Eating and drinking are an important part of life, not only Munneke, 2011), more than 35% of patients with head out of necessity but also because they are enjoyable social and neck diseases (García-Peris et al., 2007), between activities (Ekberg, Hamdy, Woisard, Wuttge-Hannig, & 13% and 57% of individuals with established dementia Ortega, 2002). -
Patient & Family Handbook
Immune Deficiency Foundation Patient & Family Handbook For Primary Immunodeficiency Diseases This book contains general medical information which cannot be applied safely to any individual case. Medical knowledge and practice can change rapidly. Therefore, this book should not be used as a substitute for professional medical advice. SIXTH EDITION COPYRIGHT 1987, 1993, 2001, 2007, 2013, 2019 IMMUNE DEFICIENCY FOUNDATION Copyright 2019 by Immune Deficiency Foundation, USA. Readers may redistribute this article to other individuals for non-commercial use, provided that the text, html codes, and this notice remain intact and unaltered in any way. The Immune Deficiency Foundation Patient & Family Handbook may not be resold, reprinted or redistributed for compensation of any kind without prior written permission from the Immune Deficiency Foundation. If you have any questions about permission, please contact: Immune Deficiency Foundation, 110 West Road, Suite 300, Towson, MD 21204, USA; or by telephone at 800-296-4433. Immune Deficiency Foundation Patient & Family Handbook For Primary Immunodeficiency Diseases 6th Edition The development of this publication was supported by Shire, now Takeda. 110 West Road, Suite 300 Towson, MD 21204 800.296.4433 www.primaryimmune.org [email protected] Editors Mark Ballow, MD Jennifer Heimall, MD Elena Perez, MD, PhD M. Elizabeth Younger, Executive Editor Children’s Hospital of Philadelphia Allergy Associates of the CRNP, PhD University of South Florida Palm Beaches Johns Hopkins University Jennifer Leiding,