Anorexia Nervosa: Loss of Contact with Reality? Hallucinations? Delusions? Flattened Affect? Cognitive Deficits? and Social Dysfunction?
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
THE IMPORTANCE of NUTRITION AS the BEST MEDICINE for EATING DISORDERS Carolyn Coker Ross, MD, MPH
DIET AND NUTRITION THE IMPORTANCE OF NUTRITION AS THE BEST MEDICINE FOR EATING DISORDERS Carolyn Coker Ross, MD, MPH ver seven million girls and women groups. Current research demonstrates to 24, and the suicide rate was 75 times and one million boys and men that eating disorder symptoms may be as higher. will suffer from an eating disorder common or more common among certain Medical consequences of eating disor- in their lifetime. Up to 3.7% of ethnic groups (Asians, blacks, and Hispan- ders include arrested sexual maturity and O 6 females will be diagnosed with anorexia ics) when compared with whites. There growth failure in prepubertal patients. nervosa and an estimated 4.2% will have was no difference found in dieting and Many with eating disorders may look and bulimia nervosa.1 The majority of adoles- restraint scores between Asian, Latino, feel deceptively well and may have normal cent patients seen in referral centers fit and white adolescent girls and boys7 and electrograms but are still at high risk for into a third category, “eating disorder not no difference in binging or BED in obese cardiac arrhythmias and sudden death. otherwise specified” or EDNOS and do patients who sought to lose weight with Prolonged amenorrhea is associated with not fit strict criteria for either anorexia or bariatric surgery.8 These changes may be an increased risk of osteopenia and rate of bulimia.2 Nineteen percent of college- related to an extension of cultural ideals in fractures. Neuroimaging studies with com- aged females are bulimic; many go undi- these ethnic populations of what is attrac- puterized tomography (CT) have demon- agnosed until much later. -
Common Signs and Symptoms of Eating Disorders (Anorexia/Bulimia)
Common Signs and Symptoms of Eating Disorders (Anorexia/Bulimia) 1. Dramatic weight loss in a relatively short period of time. 2. Wearing big or baggy clothes or dressing in layers to hide body and/or weight loss. 3. Obsession with calories and fat content of foods. 4. Obsession with continuous exercise. 5. Frequent trips to the bathroom immediately following meals (sometimes accompanied with water running in the bathroom for a long period of time to hide the sound of vomiting). 6. Visible food restriction and self-starvation. 7. Visible bingeing and/or purging. 8. Use or hiding use of diet pills, laxatives, ipecac syrup (can cause immediate death!) or enemas. 9. Isolation. Fear of eating around and with others. 10. Hiding food in strange places (closets, cabinets, suitcases, under the bed) to avoid eating (Anorexia) or to eat at a later time (Bulimia). 11. Flushing uneaten food down the toilet (can cause sewage problems). 12. Vague or secretive eating patterns. 13. Keeping a "food diary" or lists that consists of food and/or behaviors (ie., purging, restricting, calories consumed, exercise, etc.) 14. Pre-occupation or obsession with food, weight (even if “average” weight or thin), and/or cooking. 15. Visiting websites that promote unhealthy ways to lose weight. 16. Reading books about weight loss and eating disorders. 17. Unusual food rituals: shifting the food around on the plate to look eaten; cutting food into tiny pieces; making sure the fork avoids contact with the lips (using teeth to scrap food off the fork or spoon); chewing food and spitting it out, but not swallowing; dropping food into napkin on lap to later throw away. -
Hypokalaemia in a Woman with Eating Disorder
Grand Rounds Vol 11 pages 53–55 Specialities: Acute Medicine; Nephrology; Psychiatry Article Type: Case Report DOI: 10.1102/1470-5206.2011.0013 ß 2011 e-MED Ltd Hypokalaemia in a woman with eating disorder Zachary Z. Brenera, Boris Medvedovskya, James F. Winchestera and Michael Bergmanb aDivision of Nephrology, Department of Medicine, Beth Israel Medical Center, Albert Einstein School of Medicine of Yeshiva University, New York, USA; bDepartment of Medicine, Campus Golda, Rabin Medical Center, Petah-Tikva, Tel-Aviv University, Israel Corresponding address: Dr Zachary Z. Brener, 350 E. 17th St., Division of Nephrology, Beth Israel Medical Center, New York, NY 10003, USA. Email: [email protected] Date accepted for publication 13 April 2011 Abstract Chronic hypokalaemia often remains a diagnostic challenge, especially in young women without hypertension. A concealed diuretic abuse should be suspected, especially in young women with eating disorders. This case describes a woman with chronic hypokalaemia in whom a thorough medical history and proper laboratory tests were essential to early and accurate diagnosis. Keywords Hypokalaemia; eating disorders; diuretics. Introduction Chronic hypokalaemia often remains a diagnostic challenge, especially in young women without hypertension. After the exclusion of the most obvious causes, a concealed diuretic abuse associated with or without surreptitious vomiting and laxative abuse should be suspected, especially in young women concerned with their body image. A conclusive diagnosis may be difficult as such patients often vigorously deny diuretic intake[1]. Also, only a minority of patients with eating disorders (approximately 6%) abuse diuretics[2–4]. This case describes a woman with chronic hypokalaemia in whom a thorough medical history and proper laboratory tests were essential to an early and accurate diagnosis. -
Section 15: Treatment of Eating Disorders
Formulary and Prescribing Guidelines SECTION 15: TREATMENT OF EATING DISORDERS Section 15. Treatment of eating disorders 15.1 Introduction Please review the Trust document “Guidelines for the assessment and treatment of eating disorders” in the CAMHS Operational Policy. When screening for eating disorders one or two simple questions should be considered for use with specific target groups 1. Do you think you have an eating problem? 2. Do you worry excessively about your weight?’ Early detection may be helped by five screening questions using The SCOFF questionnaire. A score of two or more positive answers should raise clinical suspicion and lead to an in depth diagnostic evaluation. 1. Do you ever make yourself Sick because you feel uncomfortably full? 2. Do you worry you have lost Control over how much you eat? 3. Have you recently lost more than One stone in a three month period? 4. Do you believe yourself to be Fat when others say you are too thin? 5. Would you say that Food dominates your life? It is important to take into account that clients with eating disorders can develop Acute Kidney Injury through a variety of mechanisms associated with each condition. Clinicians should be vigilant in the monitoring of physical health especially serum creatinine and levels of hydration.3 15.2 Anorexia nervosa The following would represent a reasonable initial screen for Anorexia Nervosa in primary care if there are no other indications or diagnostic concerns: Full Blood Count, ESR, Urea and Electrolytes, Creatinine, Liver Function Tests, Random Blood Glucose, Urinalysis, ECG (should be considered in all cases and essential if symptoms/signs of compromised cardiac function, bradycardia, electrolyte abnormality and/or BMI less than 15 kg/m2 or equivalent on centile chart). -
ABSTRACT Autism Spectrum Disorder (ASD)
ADLN - Perpustakaan Universitas Airlangga ABSTRACT Autism Spectrum Disorder (ASD) was a disturbance of pervasife development in children characterized by the disturbance and delayed in cognitive, language, behaviour, communication, and social interaction. In the past 10 to 20 years, increased the number of autism currently reached an average of 8 persons among 1000 resident or 1:125. The cause of autism is is yet not found until now. The purpose of this research is analize risk factor of the trigger autism in children. This research used case control design. In the case category (ASD) accounted for 35 children according to record data in special school and the control category as many as 105 children taken from public school. Independent variables were genetic, premature, postmature, antenatal bleeding, maternal age of pregnancy, caesar childbirth, low birth weight (LBW), interval of pregnancy, organic brain syndrome, asphyxia, vaccination, smoking, consumtion of medicine and medicinal herbs, and spontaneous abortion. Whereas dependent variable was ASD incident. The data analysis by calculated odds ratio with 95% CI. The result of the research obtained were genetic, postmature, antenatal bleeding, caesar childbirth, low birth weight, interval of pregnancy, organic brain syndrome, asphyxia, vaccination, smoking, consumtion of medicine and medicinal herbs, spontaneous abortion was not the risk factor of the trigger ASD incident. The result obtained on the variables that significally premature (OR= 7.12 , 95% CI: 2.56<OR<26.07) which means that the children born prematurely had a 7.12 times higher for the onset of autism than children born with normal gestational age and another variable maternal age over 35 years (OR=8.58 , 95% CI: 1.30 <OR< 92.57) which means that the mother was pregnant at the age over 35 years had a 5.58 times higher risk to had children had autism than the mother who become pregnant less than 35 years. -
Anorexia/Cachexia Heart Failure Symptom Management Guideline for Adults, Age 19 and Older in British Columbia
Anorexia/Cachexia Heart Failure Symptom Management Guideline For adults, age 19 and older in British Columbia What is anorexia? Anorexia is a syndrome characterized by some or all of the following symptoms: loss of appetite, nausea, early satiety, weakness, fatigue, food aversion, and significant physical and/or psychological symptoms. Causes of anorexia are multifactorial and include fatigue, dyspnea, medication side-effects, nausea, depression, anxiety and sodium restricted diets, which may all be found in patients with heart failure. What is cachexia? Cachexia is a syndrome characterized by severe body weight, fat and muscle loss and increased protein catabolism due to underlying disease. The prevalence of cachexia is 16–42% in the heart failure population and is associated with a 50%, 18 month mortality risk independent of variables such as ejection fraction, age and functional ability. How is cachexia diagnosed? Chronic condition with >5% weight loss in <12 months; or body mass index (BMI) <20kg/m2; and 3 out of 5 additional criteria: 1) Fatigue, 2) Decreased muscle strength, 3) Anorexia, 4) Low muscle mass, 5) Abnormal biochemistry *Blood testing to diagnose cachexia in advanced stages of disease is not advocated. Reminder: Malnutrition also affects prognosis in patients with heart failure and is often found in early transitions of the disease. However this symptom management guideline will focus on the assessment and treatment of anorexia and cachexia. Approach to Managing Anorexia/Cachexia Assessment History: When did weight loss begin? How much weight was lost? Obtain baseline (dry) weight. How is [the patients] appetite? What do they eat or drink on a typical day? How has weight loss affected mood? Ask about: nausea, early satiety, dyspnea, poor oral hygiene, dysphagia, malabsorption, bowel habits. -
Curriculum Vitae Peter Robert Martin Address
CURRICULUM VITAE PETER ROBERT MARTIN ADDRESS: Department of Psychiatry and Behavioral Sciences Vanderbilt Psychiatric Hospital Suite 3035, 1601 23rd Avenue South Nashville, Tennessee 37232-8650 U.S.A. Phone: 615-343-4527 Mobile: 615-364-7175 E-Mail: [email protected] [email protected] https://orcid.org/0000-0003-2292-4741 WEBSITES: https://wag.app.vanderbilt.edu/PublicPage/Faculty/Details/27348 http://www.vanderbilt.edu/ics/ DATE AND PLACE OF BIRTH: September 6, 1949, Budapest, Hungary FAMILY: Married Barbara Ruth Bradford, December 23, 1985 Alexander Bradford Martin, born October 21, 1989 EDUCATION: 1967 - 1971 Honours B.Sc. (Molecular Genetics) McGill University, Montreal, Quebec, Canada 1971 - 1975 M.D., C.M. McGill University, Montreal, Quebec, Canada 1975 - 1976 Resident in Internal Medicine, Sunnybrook Medical Centre, University of Toronto, Toronto, Ontario, Canada 1976 - 1978 Research Fellow in Clinical Pharmacology, Clinical Pharmacology Program, Addiction Research Foundation Clinical Institute - Toronto Western Hospital, University of Toronto 1976 - 1979 M.Sc. (Pharmacology) University of Toronto Dissertation: Intravenous phenobarbital treatment of barbiturate and other hypnosedative withdrawal: A pharmacokinetic approach. 1978 - 1979 Resident in Psychiatry, Affective Disorders Unit, Clarke Institute of Psychiatry, University of Toronto 1979 Resident in Psychiatry, Hospital for Sick Children, University of Toronto 1980 Resident in Psychiatry, Toronto General Hospital, University of Toronto LICENSURE AND CERTIFICATION: The College of Physicians and Surgeons of Ontario (License No. 28907), 1976. The Board of Medical Examiners of the State of Maryland (License No. D26685), 1981. The Board of Medical Examiners of the State of Tennessee (License No. MD17128), 1986. Fellow of the Royal College of Physicians (Canada), Psychiatry, 1981. -
PAVOL JOZEF ŠAFARIK UNIVERSITY in KOŠICE Dissociative Amnesia: a Clinical and Theoretical Reconsideration DEGREE THESIS
PAVOL JOZEF ŠAFARIK UNIVERSITY IN KOŠICE FACULTY OF MEDICINE Dissociative amnesia: a clinical and theoretical reconsideration Paulo Alexandre Rocha Simão DEGREE THESIS Košice 2017 PAVOL JOZEF ŠAFARIK UNIVERSITY IN KOŠICE FACULTY OF MEDICINE FIRST DEPARTMENT OF PSYCHIATRY Dissociative amnesia: a clinical and theoretical reconsideration Paulo Alexandre Rocha Simão DEGREE THESIS Thesis supervisor: Mgr. MUDr. Jozef Dragašek, PhD., MHA Košice 2017 Analytical sheet Author Paulo Alexandre Rocha Simão Thesis title Dissociative amnesia: a clinical and theoretical reconsideration Language of the thesis English Type of thesis Degree thesis Number of pages 89 Academic degree M.D. University Pavol Jozef Šafárik University in Košice Faculty Faculty of Medicine Department/Institute Department of Psychiatry Study branch General Medicine Study programme General Medicine City Košice Thesis supervisor Mgr. MUDr. Jozef Dragašek, PhD., MHA Date of submission 06/2017 Date of defence 09/2017 Key words Dissociative amnesia, dissociative fugue, dissociative identity disorder Thesis title in the Disociatívna amnézia: klinické a teoretické prehodnotenie Slovak language Key words in the Disociatívna amnézia, disociatívna fuga, disociatívna porucha identity Slovak language Abstract in the English language Dissociative amnesia is a one of the most intriguing, misdiagnosed conditions in the psychiatric world. Dissociative amnesia is related to other dissociative disorders, such as dissociative identity disorder and dissociative fugue. Its clinical features are known -
Social Cognition in Eating Disorders: Encoding and Representational Processes in Binging and Purging Patients
RESEARCH ARTICLE Social Cognition in Eating Disorders: Encoding and Representational Processes in Binging and Purging Patients Lily Rothschild-Yakar1,2*, Zohar Eviatar2, Adi Shamia2 & Eitan Gur3 1Safra Children’s Hospital, Sheba Medical Center, Tel Hashomer, Israel 2Department of Psychology, University of Haifa, Israel 3Sheba Medical Center, Tel Hashomer, Israel Abstract Objective: The present study investigates social cognition impairments in 29 women with bingeing/purging spectrum eating disorders (ED) compared to 27 healthy controls. Method: Measures were used to examine encoding and representational processes in relation to affect perception and affect attribution, as well as the ability to recognize mental causality in social relationships. Results: ED patients failed to correctly encode causality in interpersonal relations, exhibited deficits in their ability to ascribe behaviour to mental states, and showed a greater tendency to attribute negative affects in interpersonal relationships. Stepwise regression analyses suggested that ED symptoms could account for deficits in the recognition of causality in interpersonal relations. Conclusions: In addition to addressing ED symptoms, social cognition deficits should be addressed in the psychological treatment of EDs. Copyright # 2010 John Wiley & Sons, Ltd and Eating Disorders Association. Keywords eating disorders; social cognition; mentalization *Correspondence Lily Rothschild-Yakar, PhD, Department of Psychology, University of Haifa, Haifa 31905, Israel. Tel: 972-3-649-9563; Fax: 974-4-824-0966. Email: [email protected] Published online 29 July 2010 in Wiley Online Library (wileyonlinelibrary.com) DOI: 10.1002/erv.1013 cognition in AN (restricting type), suggesting similarities Introduction with autistic spectrum disorders. They suggested that Dysfunctional social cognition may play an important deficits in social cognition may be central to the role in eating disorders (ED). -
Dsm-5 Diagnostic Criteria for Eating Disorders Anorexia Nervosa
DSM-5 DIAGNOSTIC CRITERIA FOR EATING DISORDERS ANOREXIA NERVOSA DIAGNOSTIC CRITERIA To be diagnosed with anorexia nervosa according to the DSM-5, the following criteria must be met: 1. Restriction of energy intaKe relative to requirements leading to a significantly low body weight in the context of age, sex, developmental trajectory, and physical health. 2. Intense fear of gaining weight or becoming fat, even though underweight. 3. Disturbance in the way in which one's body weight or shape is experienced, undue influence of body weight or shape on self-evaluation, or denial of the seriousness of the current low body weight. Even if all the DSM-5 criteria for anorexia are not met, a serious eating disorder can still be present. Atypical anorexia includes those individuals who meet the criteria for anorexia but who are not underweight despite significant weight loss. Research studies have not found a difference in the medical and psychological impacts of anorexia and atypical anorexia. BULIMIA NERVOSA DIAGNOSTIC CRITERIA According to the DSM-5, the official diagnostic criteria for bulimia nervosa are: • Recurrent episodes of binge eating. An episode of binge eating is characterized by both of the following: o Eating, in a discrete period of time (e.g. within any 2-hour period), an amount of food that is definitely larger than most people would eat during a similar period of time and under similar circumstances. o A sense of lacK of control over eating during the episode (e.g. a feeling that one cannot stop eating or control what or how much one is eating). -
Cancer Cachexia and Fatigue
CME Palliative care Cancer cachexia and mechanisms (Fig 1). The cachectic Other cachectic factors patient is analogous to an accelerating Cachexia can occur in the absence of car running out of petrol. The anorexia anorexia, suggesting that catabolic fatigue component of cancer cachexia reduces mediators produced by tumour or host fuel supply (by ca 300–500 kcal/day) cells are involved in the cancer cachexia whilst accelerated metabolic cycling Grant D Stewart BSc(Hons) MBChB MRCS(Ed), process.9 Experimental cachexia models drives hypermetabolism (by ca Surgical Research Fellow suggest pro-inflammatory cytokines, 100–200 kcal/day). There are also the Richard JE Skipworth BSc(Hons) MBChB such as tumour necrosis factor- , inter- direct catabolic effects of muscle proteol- α MRCS(Ed), Surgical Research Fellow leukin (IL)-6, IL-1 and interferon- , can ysis and lipolysis. These changes underlie γ Kenneth CH Fearon MBChB(Hons) MD all play a role. Activation of the neuro- a key paradox of cachexia: whilst meta- FRCS(Glas) FRCS(Ed) FRCS(Eng), Professor of endocrine stress response is also thought bolic rate may be increased, overall (or Surgical Oncology to be important. Potential mediators total) energy expenditure is decreased Department of Clinical and Surgical Sciences include increased adrenergic activity, ele- due to a fall in physical activity.7 (Surgery), University of Edinburgh, Royal vated cortisol, low insulin and increased Infirmary, Edinburgh activity of the renin-angiotensin system.1 Anorexia With regard to tumour-specific Clin Med 2006;6:140–3 The anorexia component of cancer cachectic factors, proteolysis-inducing cachexia has both a neurohumoral mech- factor (PIF) is produced by tumours and anism due to disturbance of the central excreted in the urine of patients with Background physiological mechanisms controlling cancer cachexia. -
Cancer Anorexia Cachexia Syndrome (CACS)
Cancer Anorexia Cachexia Syndrome (CACS) Amanda Werner, RN, BSN Supportive Care and Vitality Clinics Moffitt Cancer Center Objectives Define CACS Identify contributing factors in CACS Describe the effects of CACS on patient outcomes 58 year old Male Lung Cancer No appetite Significant weight loss: muscle and fat Decreased physical ability and function Family forcing food Patient socially withdrawn Cancer Anorexia Cachexia Syndrome (CACS) Multifactoral syndrome Negative protein and energy balance Ongoing loss of skeletal muscle mass (with/without loss of fat mass) Leads to progressive functional impairment Underlying Mechanisms Symptoms of CACS Poor appetite Involuntary weight loss Increased fatigue Loss of physical strength Cachexia is NOT… Starvation Fully reversed by conventional nutritional support or artificial nutrition Intentional Negative Outcomes Treatment ◦ Poor tolerance to treatment options ◦ Not eligible for treatment due to performance status Physical ◦ Decreased function and ability to complete ADLs Psychosocial ◦ Decreased quality of life ◦ Altered body image ◦ Source of patient/family emotional distress and conflict PATIENTS & CAREGIVERS NEED SUPPORT TO COPE WITH THE DISTRESS OF CACHEXIA 10 Prevalence Varies by tumor type Under recognized ◦ ½ of cancer patients have cachexia ◦ Approx. 30% die from cachexia Under treated ◦ Condition could be present in an obese patient CACS Nutritional Impact Primary Cachexia CACS direct impact on nutrition Secondary Cachexia Impact of cancer & treatment Tertiary