Dictionary of Tropical Medicine: for Health Professionals
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Communicable Disease Control
LECTURE NOTES For Nursing Students Communicable Disease Control Mulugeta Alemayehu Hawassa University In collaboration with the Ethiopia Public Health Training Initiative, The Carter Center, the Ethiopia Ministry of Health, and the Ethiopia Ministry of Education 2004 Funded under USAID Cooperative Agreement No. 663-A-00-00-0358-00. Produced in collaboration with the Ethiopia Public Health Training Initiative, The Carter Center, the Ethiopia Ministry of Health, and the Ethiopia Ministry of Education. Important Guidelines for Printing and Photocopying Limited permission is granted free of charge to print or photocopy all pages of this publication for educational, not-for-profit use by health care workers, students or faculty. All copies must retain all author credits and copyright notices included in the original document. Under no circumstances is it permissible to sell or distribute on a commercial basis, or to claim authorship of, copies of material reproduced from this publication. ©2004 by Mulugeta Alemayehu All rights reserved. Except as expressly provided above, no part of this publication may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, recording, or by any information storage and retrieval system, without written permission of the author or authors. This material is intended for educational use only by practicing health care workers or students and faculty in a health care field. Communicable Disease Control Preface This lecture note was written because there is currently no uniformity in the syllabus and, for this course additionally, available textbooks and reference materials for health students are scarce at this level and the depth of coverage in the area of communicable diseases and control in the higher learning health institutions in Ethiopia. -
Iliopsoas Tendonitis/Bursitis Exercises
ILIOPSOAS TENDONITIS / BURSITIS What is the Iliopsoas and Bursa? The iliopsoas is a muscle that runs from your lower back through the pelvis to attach to a small bump (the lesser trochanter) on the top portion of the thighbone near your groin. This muscle has the important job of helping to bend the hip—it helps you to lift your leg when going up and down stairs or to start getting out of a car. A fluid-filled sac (bursa) helps to protect and allow the tendon to glide during these movements. The iliopsoas tendon can become inflamed or overworked during repetitive activities. The tendon can also become irritated after hip replacement surgery. Signs and Symptoms Iliopsoas issues may feel like “a pulled groin muscle”. The main symptom is usually a catch during certain movements such as when trying to put on socks or rising from a seated position. You may find yourself leading with your other leg when going up the stairs to avoid lifting the painful leg. The pain may extend from the groin to the inside of the thigh area. Snapping or clicking within the front of the hip can also be experienced. Do not worry this is not your hip trying to pop out of socket but it is usually the iliopsoas tendon rubbing over the hip joint or pelvis. Treatment Conservative treatment in the form of stretching and strengthening usually helps with the majority of patients with iliopsoas bursitis. This issue is the result of soft tissue inflammation, therefore rest, ice, anti- inflammatory medications, physical therapy exercises, and/or injections are effective treatment options. -
The Textual and Visual Uses of the Literary Motif of Cross-Dressing In
The Textual and Visual Uses of the Literary Motif of Cross-Dressing in Medieval French Literature, 1200–1500 Vanessa Elizabeth Wright Submitted in accordance with the requirements for the degree of PhD in Medieval Studies University of Leeds Institute for Medieval Studies September 2019 2 The candidate confirms that the work submitted is her own and that appropriate credit has been given where reference has been made to the work of others. This copy has been supplied on the understanding that it is copyright material and that no quotation from the thesis may be published without proper acknowledgement. The right of Vanessa Elizabeth Wright to be identified as Author of this work has been asserted by her in accordance with the Copyright, Designs and Patents Act 1988. 3 Acknowledgements I would like to thank my supervisors Rosalind Brown-Grant, Catherine Batt, and Melanie Brunner for their guidance, support, and for continually encouraging me to push my ideas further. They have been a wonderful team of supervisors and it has been a pleasure to work with them over the past four years. I would like to thank my examiners Emma Cayley and Helen Swift for their helpful comments and feedback on this thesis and for making my viva a positive and productive experience. I gratefully acknowledge the funding that allowed me to undertake this doctoral project. Without the School of History and the Institute for Medieval Studies Postgraduate Research Scholarship, I would not have been able to undertake this study. Trips to archives and academic conferences were made possible by additional bursaries and fellowships from Institute for Medieval Studies, the Royal Historical Society, the Society for the Study of Medieval Languages and Literatures, the Society for Medieval Feminist Scholarship’s Foremothers Fellowship (2018), and the Society for the Study of French History. -
Introduction to Bacteriology and Bacterial Structure/Function
INTRODUCTION TO BACTERIOLOGY AND BACTERIAL STRUCTURE/FUNCTION LEARNING OBJECTIVES To describe historical landmarks of medical microbiology To describe Koch’s Postulates To describe the characteristic structures and chemical nature of cellular constituents that distinguish eukaryotic and prokaryotic cells To describe chemical, structural, and functional components of the bacterial cytoplasmic and outer membranes, cell wall and surface appendages To name the general structures, and polymers that make up bacterial cell walls To explain the differences between gram negative and gram positive cells To describe the chemical composition, function and serological classification as H antigen of bacterial flagella and how they differ from flagella of eucaryotic cells To describe the chemical composition and function of pili To explain the unique chemical composition of bacterial spores To list medically relevant bacteria that form spores To explain the function of spores in terms of chemical and heat resistance To describe characteristics of different types of membrane transport To describe the exact cellular location and serological classification as O antigen of Lipopolysaccharide (LPS) To explain how the structure of LPS confers antigenic specificity and toxicity To describe the exact cellular location of Lipid A To explain the term endotoxin in terms of its chemical composition and location in bacterial cells INTRODUCTION TO BACTERIOLOGY 1. Two main threads in the history of bacteriology: 1) the natural history of bacteria and 2) the contagious nature of infectious diseases, were united in the latter half of the 19th century. During that period many of the bacteria that cause human disease were identified and characterized. 2. Individual bacteria were first observed microscopically by Antony van Leeuwenhoek at the end of the 17th century. -
Medical Terminology Abbreviations Medical Terminology Abbreviations
34 MEDICAL TERMINOLOGY ABBREVIATIONS MEDICAL TERMINOLOGY ABBREVIATIONS The following list contains some of the most common abbreviations found in medical records. Please note that in medical terminology, the capitalization of letters bears significance as to the meaning of certain terms, and is often used to distinguish terms with similar acronyms. @—at A & P—anatomy and physiology ab—abortion abd—abdominal ABG—arterial blood gas a.c.—before meals ac & cl—acetest and clinitest ACLS—advanced cardiac life support AD—right ear ADL—activities of daily living ad lib—as desired adm—admission afeb—afebrile, no fever AFB—acid-fast bacillus AKA—above the knee alb—albumin alt dieb—alternate days (every other day) am—morning AMA—against medical advice amal—amalgam amb—ambulate, walk AMI—acute myocardial infarction amt—amount ANS—automatic nervous system ant—anterior AOx3—alert and oriented to person, time, and place Ap—apical AP—apical pulse approx—approximately aq—aqueous ARDS—acute respiratory distress syndrome AS—left ear ASA—aspirin asap (ASAP)—as soon as possible as tol—as tolerated ATD—admission, transfer, discharge AU—both ears Ax—axillary BE—barium enema bid—twice a day bil, bilateral—both sides BK—below knee BKA—below the knee amputation bl—blood bl wk—blood work BLS—basic life support BM—bowel movement BOW—bag of waters B/P—blood pressure bpm—beats per minute BR—bed rest MEDICAL TERMINOLOGY ABBREVIATIONS 35 BRP—bathroom privileges BS—breath sounds BSI—body substance isolation BSO—bilateral salpingo-oophorectomy BUN—blood, urea, nitrogen -
United States Patent [191 [11] Patent Number: 5,403,718 Durward Et Al
. US005403718A United States Patent [191 [11] Patent Number: 5,403,718 Durward et al. [451 Date of Patent: * Apr. 4, 1995 [54] METHODS AND ANTIBODIES FOR THE I OTHER PUBLICATIONS IMMUNE CAPTURE AND DETECHON 0F Nowinski et al, Science, 219:637-644 (11 Feb. 1983), BQRRELI-A BURGDORFERI - “Monoclonal Antibodies for Diagnosis of Infectious [76] Inventors: David W. Dorward, 401 N. 7th St; Diseases in Humans”. Tom G. Schwan, 601 S. 5th St.; . E . _ 1 E . Claude F. Garon, 904 Ponderosa Dr., Primary xammer Caro ' Bldwen all of Hamilton, Mont. 59840 [57] ABSTRACT [ * ] Notice; The portion of the term of this patent The invention relates to novel antigens associated with subsequent to Jun, 8, 2010 has been Borrelia burgdorferi which are exported (or shed) in disclaimed. vivo and whose detection is a means of diagnosing _ Lyme disease. The antigens are extracellular membrane [211 App 1' No" 929’172 vesicles and other bioproducts including the major ex [22] Filed: Aug. 11,1992 tracellular protein. The invention further provides anti bodies, monoclonal and/or polyclonal, labeled and/or Related US. Application Data unlabeled, that react with the antigens. The invention [63] Continuation-impart ofSer. No. 485 551 Feb. 27 1990 . relates to a math“ for immune capture of Speci?c mi‘ Pat No_ 5,217,871 ’ ’ ’ ’ , croorganisms for their subsequent cultivation. The in vention is also directed to a method of diagnosing Lyme clci? ------------------ -- GolN 332940 disease by detecting the antigens in a biological sample . ................................ .. , . , taken from a host using the antibodies in conventional 530/3871; 530/388.4; 530/3895 immunoassay formats. -
A Spectral BSSRDF for Shading Human Skin
Eurographics Symposium on Rendering (2006) Tomas Akenine-Möller and Wolfgang Heidrich (Editors) A Spectral BSSRDF for Shading Human Skin Craig Donner and Henrik Wann Jensen† Universtiy of California at San Diego, La Jolla, CA, USA Abstract We present a novel spectral shading model for human skin. Our model accounts for both subsurface and surface scattering, and uses only four parameters to simulate the interaction of light with human skin. The four parameters control the amount of oil, melanin and hemoglobin in the skin, which makes it possible to match specific skin types. Using these parameters we generate custom wavelength dependent diffusion profiles for a two-layer skin model that account for subsurface scattering within the skin. These diffusion profiles are computed using convolved diffusion multipoles, enabling an accurate and rapid simulation of the subsurface scattering of light within skin. We combine the subsurface scattering simulation with a Torrance-Sparrow BRDF model to simulate the interaction of light with an oily layer at the surface of the skin. Our results demonstrate that this four parameter model makes it possible to simulate the range of natural appearance of human skin including African, Asian, and Caucasian skin types. Categories and Subject Descriptors (according to ACM CCS): I.3.7 [Computer Graphics]: Color, shading, shadowing, and texture 1 Introduction Debevec et al. [DHT∗00] measured the reflectance field of human faces, allowing for rendering of skin under varying Simulating the appearance of human skin is a challenging illumination conditions with excellent results. Jensen and problem due to the complex structure of the skin. Further- Buhler [JB02], Hery [Her03] and Weyrich et al. -
Aedes Aegypti (Yellow Fever Mosquito) Fact Sheet
STATE OF CALIFORNIA-HEALTH AND HUMAN SERVICES AGENCY California Department of Public Health Division of Communicable Disease Control Aedes aegypti (Yellow Fever Mosquito) Fact Sheet What is the Aedes aegypti mosquito? Aedes aegypti, also known as the “yellow fever mosquito”, is an invasive mosquito; it is not native to California. This black and white striped mosquito bites people and animals during the day. Why are we concerned about the Aedes aegypti mosquito in California? This mosquito is an aggressive day biting mosquito and has the potential to transmit several viruses, including dengue, chikungunya, and yellow fever. However, none of these viruses are currently known to be transmitted within California. The eggs of Aedes aegypti have the ability to survive being dry for long periods of time which allows eggs to be easily spread to new locations. Where do Aedes aegypti mosquitoes lay their eggs? Female mosquitoes lay their eggs in small artificial or natural containers that hold water. Containers can include dishes under potted plants, bird baths, ornamental fountains, tin cans, or discarded tires. Even a small amount of standing water can produce mosquitoes. What is the life cycle of the Aedes aegypti mosquito? About three days after feeding on blood, the female lays her eggs inside a container just above the water line. Eggs are laid over a period of several days, are resistant to drying, and can survive for periods of six or more months. When the container is refilled with water, the eggs hatch into larvae. The entire life cycle (i.e., from egg to adult) can occur in as little as 7-8 days. -
Isolation and Growth of Adult Human Epidermal Keratinocytes in Cell Culture
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Elsevier - Publisher Connector CITATION CLASSIC 0022-202X/78/7102-0157$02.00/0 THE JOURNAL OF INVESTIGATIVE DERMATOLOGY, 71:157–162, 1978 Vol. 71, No. 2 Copyright & 1978 by The Williams & Wilkins Co. PrintedinU.S.A. Isolation and Growth of Adult Human Epidermal Keratinocytes in Cell Culture SU-CHIN LIU,PH.D., AND MARVIN KARASEK,PH.D. Humanepidermalkeratinocytesmaybeisolatedinhighyieldfrom 0.1 mm keratotome sections of adult skin by short-term trypsinrelease.Whenplatedonacollagen-coatedplasticsurfaceor on a collagen gel, keratinocytes attach with high efficiencies (470%) and form confluent, stratified cultures. Cell populations of predominantly basal cells produce proliferative primary cell cultures while populations of basal cells and malpighian cells result in nonproliferative primary cultures. Both nonproliferative and proliferative primary cultures may be subcultured on collagen gels following dispersion by trypsin and EDTA. Methotrexate strongly inhibits proliferative keratinocytes at low concentrations (0.1 mg/ml) but has no cytotoxic effect on non- proliferative cells. L-serine and dexamethasone increase the multiplication rate of both primary and subcultured human keratinocytes. The ability to isolate and to grow human epidermal keratinocytes from Preparation of Collagen Surfaces both normal and diseased human skin in sufficient quantities for Acid soluble collagen is extracted and purified from adult rabbit skin as described biochemical and genetic studies has been a long-range goal of many previously [10]. Three types of culture surfaces are prepared on 35-mm plastic investigators. Although keratinocytes may be obtained from postem- Petri dishes: (a) collagen-coated, (b) thin gel, and (c) 2-mm collagen gel. -
Anorexia/Cachexia Heart Failure Symptom Management Guideline for Adults, Age 19 and Older in British Columbia
Anorexia/Cachexia Heart Failure Symptom Management Guideline For adults, age 19 and older in British Columbia What is anorexia? Anorexia is a syndrome characterized by some or all of the following symptoms: loss of appetite, nausea, early satiety, weakness, fatigue, food aversion, and significant physical and/or psychological symptoms. Causes of anorexia are multifactorial and include fatigue, dyspnea, medication side-effects, nausea, depression, anxiety and sodium restricted diets, which may all be found in patients with heart failure. What is cachexia? Cachexia is a syndrome characterized by severe body weight, fat and muscle loss and increased protein catabolism due to underlying disease. The prevalence of cachexia is 16–42% in the heart failure population and is associated with a 50%, 18 month mortality risk independent of variables such as ejection fraction, age and functional ability. How is cachexia diagnosed? Chronic condition with >5% weight loss in <12 months; or body mass index (BMI) <20kg/m2; and 3 out of 5 additional criteria: 1) Fatigue, 2) Decreased muscle strength, 3) Anorexia, 4) Low muscle mass, 5) Abnormal biochemistry *Blood testing to diagnose cachexia in advanced stages of disease is not advocated. Reminder: Malnutrition also affects prognosis in patients with heart failure and is often found in early transitions of the disease. However this symptom management guideline will focus on the assessment and treatment of anorexia and cachexia. Approach to Managing Anorexia/Cachexia Assessment History: When did weight loss begin? How much weight was lost? Obtain baseline (dry) weight. How is [the patients] appetite? What do they eat or drink on a typical day? How has weight loss affected mood? Ask about: nausea, early satiety, dyspnea, poor oral hygiene, dysphagia, malabsorption, bowel habits. -
Fever / Sepsis
Fever / Sepsis History Signs and Symptoms Differential · Age · Warm · Infections / Sepsis · Duration of fever · Flushed · Cancer / Tumors / Lymphomas · Severity of fever · Sweaty · Medication or drug reaction · Past medical history · Chills/Rigors · Connective tissue disease · Medications Associated Symptoms · Arthritis · Immunocompromised (transplant, (Helpful to localize source) · Vasculitis HIV, diabetes, cancer) · myalgias, cough, chest pain, · Hyperthyroidism · Environmental exposure headache, dysuria, abdominal pain, · Heat Stroke · Last acetaminophen or ibuprofen mental status changes, rash · Meningitis Adult Contact, Droplet, and Airborne Precautions Temperature Measurement Procedure B / if available Pediatric General Section Protocols IV Procedure IO Procedure I P If indicated If indicated Temperature NO Greater than 100.4 F YES (38 C) If Suspected infection ? B then proceed to Protocol 72A otherwise Proceed to Protocol Exit to 72A Appropriate Protocol Pearls · Recommended Exam: Mental Status, Skin, HEENT, Neck, Heart, Lungs, Abdomen, Back, Extremities, Neuro · Febrile seizures are more likely in children with a history of febrile seizures and with a rapid elevation in temperature. · Patients with a history of liver failure should not receive acetaminophen. · Droplet precautions include standard PPE plus a standard surgical mask for providers who accompany patients in the back of the ambulance and a surgical mask or NRB O2 mask for the patient. This level of precaution should be utilized when influenza, meningitis, mumps, streptococcal pharyngitis, and other illnesses spread via large particle droplets are suspected. A patient with a potentially infectious rash should be treated with droplet precautions. · Airborne precautions include standard PPE plus utilization of a gown, change of gloves after every patient contact, and strict hand washing precautions. This level of precaution is utilized when multi-drug resistant organisms (e.g. -
Pubic Lice/Crabs Fact Sheet
Pubic Lice/Crabs Fact Sheet What are pubic lice/crabs? Also called crab lice or "crabs," pubic lice are parasitic insects found primarily in the pubic or genital area of humans. Pubic lice infestation is found worldwide and occurs in all races, ethnic groups, and levels of society. How do pubic lice spread? People with pubic lice are said to be “infested” not “infected.” Pubic lice usually are spread through sexual contact and are most common in adults. Pubic lice found on children may be a sign of sexual exposure or abuse. Occasionally, pubic lice may be spread by close personal contact or contact with articles such as clothing, bed linens, or towels that have been used by an infested person. Persons infested with pubic lice should be examined for the presence of other sexually transmitted diseases. What are the signs and symptoms of public lice? Signs and symptoms of public lice/crabs include itching in the genital area or presence of nits or crawling pubic lice. How long does it take for symptoms to appear? To understand this, we need to understand the life cycle. Pubic lice have three forms: the egg (also called a nit), the nymph, and the adult. Nit: Nits are lice eggs. They are hard to see. Nits attach to the hair shaft. They are oval and usually yellow to white. Pubic lice nits take about 6–10 days to hatch. Nymph: A nymph is an immature louse that hatches from the nit (egg). A nymph looks like an adult pubic louse but it is smaller.