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Xist
Revealing the Mechanism of Xist-Mediated Silencing
Association of BRCA1 with the Inactive X Chromosome and XIST RNA
Long Non‑Coding Rnas XIST and MALAT1 Hijack the PD‑L1 Regulatory Signaling Pathway in Breast Cancer Subtypes
Non-Coding RNA: X-Chromosome Inactivation Unravelled
Agonists Regulate the Expression of Stemness and Differentiation Genes in Brain Tumour Stem Cells
Reprogramming Capacity of Nanog Is Functionally Conserved in Vertebrates and Resides in a Unique Homeodomain Thorold W
Thoughts About SLC16A2, TSIX and XIST Gene Like Sites in the Human Genome and a Potential Role in Cellular Chromosome Counting Martina Rinčić1, Ivan Y
DNA Hypomethylation Can Activate Xist Expression and Silence X-Linked Genes
XIST RNA and the Mechanism of X Chromosome Inactivation, Kathrin Plath, Susanna Mlynarczyk-Evans, Dmitri A
Lamin B Receptor: Interplay Between Structure, Function and Localization
Dosage Compensation in Mammals: Fine-Tuning the Expression of the X Chromosome
Rnai-Mediated Knockdown of Xist Can Rescue the Impaired Postimplantation Development of Cloned Mouse Embryos
RNF12 Activates Xist and Is Essential for X Chromosome Inactivation
Sex-Specific Silencing of X-Linked Genes by Xist RNA PNAS PLUS
Team Publications Private: Mammalian Developmental Epigenetics
Integrated Analysis of Xist Upregulation and Gene Silencing at the Onset of Random X-Chromosome Inactivation at High Temporal and Allelic Resolution
Xist/Tsix Expression Dynamics During Mouse Peri-Implantation
Molecular Coupling of Tsix Regulation and Pluripotency
Top View
The Long Non-Coding RNA XIST: a New Cornerstone in Carcinogenesis
Epigenetics: X Inactivation by Titration
X Chromosome Inactivation in the Absence of Dicer
LECTURE 13: DOSAGE COMPENSATION Reading: Ch. 12, P
The Transcriptional Foundation of Pluripotency Ian Chambers and Simon R
Xist Incrna Forms Silencing Granules That Induce Heterochromatin
Non-Coding Rnas and Nucleosome Remodeling Complexes: an Intricate Regulatory Relationship
X Chromosome Dosage Compensation
Xist at the Edge: Silencing at the Nuclear Periphery Extends to X Inactivation
Xist Gene Regulation at the Onset of X Inactivation Claire E Senner1 and Neil Brockdorff2
1 High XIST and Low 53BP1 Expression Predict Poor Outcome
In Silico and in Vitro Analysis of Lncrna XIST Reveals a Panel of Possible Lung Cancer Regulators and a Five-Gene Diagnostic Signature
A Gene-By-Gene Mosaic of Dosage Compensation Strategies on the Human X Chromosome Authors: Adrianna K
Characterization of the Promoter Region of the Mouse Xist Gene
Progress Toward Understanding Chromosome Silencing by Xist RNA
Tsix Transcription Across the Xist Gene Alters Chromatin Conformation Without Affecting Xist Transcription: Implications for X-Chromosome Inactivation
Downloaded from Ecosystems in the Mediterranean in a Way That (2016)
Roles of Long Noncoding Rnas in X-Chromosome Inactivation
Dosage Compensation Mechanisms: Evolution Dosage Compensation Mechanisms: Evolution
Mechanistic Concepts in X Inactivation Underlying Dosage Compensation in Mammals
Female Mice Lacking Xist RNA Show Partial Dosage Compensation and Survive to Term
A Mixed Modality Approach Towards Xi Reactivation for Rett Syndrome and Other X-Linked Disorders
Retinoic Acid Accelerates Downregulation of the Xist
High XIST and Low 53BP1 Expression Predict Poor Outcome After High-Dose Alkylating Chemotherapy in Patients with a BRCA1-Like Breast Cancer Philip C
Pluripotency Factor Binding and Tsix Expression Act Synergistically To
Xist Localization and Function: New Insights from Multiple Levels Andrea Cerase1*, Greta Pintacuda2, Anna Tattermusch2 and Philip Avner1,3*
The X-Inactivation Trans-Activator Rnf12 Is Negatively Regulated by Pluripotency Factors in Embryonic Stem Cells
New Insights Into X-Chromosome Reactivation During Reprogramming to Pluripotency
Oct4 to Count 2 Anton Wutz1
X-Inactivation and Epigenetics
Impaired Imprinted X Chromosome Inactivation Is Responsible for the Skewed Sex Ratio Following in Vitro Fertilization
Rnai in X Inactivation: Contrasting Findings on the Role of Interference
Tsix-Mediated Epigenetic Switch of a CTCF-Flanked Region of the Xist Promoter Determines the Xist Transcription Program
Association Between BRCA1 Mutations and Ratio of Female to Male Births in Offspring of Families with Breast Cancer, Ovarian Cancer, Or Both
BRCA1 Supports XIST RNA Concentration on the Inactive X Chromosome