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- Mutations Affecting the Secretory COPII Coat Component SEC23B Cause Congenital Dyserythropoietic Anemia Type II
- SEC23B Is Required for the Maintenance of Murine Professional
- Pancreatic SEC23B Deficiency Is Sufficient to Explain the Perinatal
- Functions of the COPII Gene Paralogs SEC23A and SEC23B Are Interchangeable in Vivo
- Autophagy-Associated IL-15 Production Is Involved in the Pathogenesis of Leprosy Type 1 Reaction
- Functions of the COPII Gene Paralogs SEC23A and SEC23B Are Interchangeable in Vivo
- A Common SEC23B Missense Mutation in Congenital Dyserythropoietic Anemia Type II Leads to Growth Restriction and Symptoms of Chronic Pancreatitis in Mice
- Applying Forward Genetic Approaches to Rare Mendelian
- Cox Duke 0066D 14521.Pdf
- RAP-011 Rescues the Disease Phenotype in a Cellular Model of Congenital Dyserythropoietic Anemia Type II by Inhibiting the SMAD2-3 Pathway
- SEC23B Gene Sec23 Homolog B, Coat Complex II Component
- COPII-Mediated Vesicle Formation at a Glance
- SEC23B Is Required for Pancreatic Acinar Cell Function in Adult Mice
- RNA-Sequencing Identifies Dysregulation of the Human Pancreatic Islet Transcriptome
- Non-Canonical Role of Wild-Type SEC23B in the Cellular Stress Response Pathway Lamis Yehia 1, Darren Liu 1,2,Shuaifu 2,3,Pranaviyer 1,6 and Charis Eng 1,2,3,4,5
- Integrated Functional Genomics Approach for the Design of Patient-Individual Antitumor Vaccines1
- Molecular Pathways Involved in the Development of Congenital Erythrocytosis
- Assessment of Genetics Mutations in Genes CDAN1, SEC23B and Dell-15Q22 in Inducate Congenital Dyserythropoietic Anemia Syndrome