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NMNAT3
Nicotinamide Adenine Dinucleotide Is Transported Into Mammalian
Metabolism and Biochemical Properties of Nicotinamide
Nicotinamide Mononucleotide Preserves Mitochondrial Function and Increases Survival in Hemorrhagic Shock
Nmnat3 Is Dispensable in Mitochondrial NAD Level Maintenance in Vivo
NMNAT3 Improves Mitochondrial Function and Enhances Bmscs Anti-Oxidative Stress Through the NAD+-Sirt3 Pathway
Potential Therapeutic Benefit of NAD+ Supplementation for Glaucoma And
Targeting Diet and Exercise for Neuroprotection and Neurorecovery in Glaucoma
Interplay Between Compartmentalized NAD+ Synthesis and Consumption: a Focus on the PARP Family
Muscle NAD+ Depletion and Serpina3n As Molecular Determinants of Murine Cancer Cachexia : the Effects of Blocking Myostatin and Activins
[Frontiers in Bioscience 14, 410-431, January 1, 2009] 410 the NMN/Namn Adenylyltransferase (NMNAT) Protein Family Corinna Lau
Upregulation of Mitochondrial NAD+ Levels Impairs the Clonogenicity of SSEA1+ Glioblastoma Tumor-Initiating Cells
SARM1 Depletion Rescues NMNAT1 Dependent Photoreceptor Cell Death and Retinal Degeneration
Ep 3006040 A1
Subcellular Compartmentalization of NAD+ and Its Role in Cancer: a Serenade of Metabolic Melodies
Advances in NAD-Lowering Agents for Cancer Treatment
Programmed Axon Degeneration: from Mouse to Mechanism to Medicine
Metabolomic Profiles of Induced Pluripotent Stem Cells Derived From
Therapeutic Potential of NAD-Boosting Molecules: the in Vivo Evidence
Top View
NMNAT3 Is Protective Against the Effects of Neonatal Cerebral Hypoxia-Ischemia Rafael Galindo Washington University School of Medicine in St
Role of Nadгѕ in Regulating Cellular and Metabolic Signaling Pathways
NMNAT3 Is Involved in the Protective Effect of SIRT3 in Ang II-Induced Cardiac Hypertrophy
Methods and Compositions for Treating Neuropathies
Stimulation of Nicotinamide Adenine Dinucleotide Biosynthetic Pathways Delays Axonal Degeneration After Axotomy
A Rise in NAD Precursor Nicotinamide Mononucleotide (NMN) After Injury Promotes Axon Degeneration
NAD+ Metabolism: Pathophysiologic Mechanisms and Therapeutic Potential
Chronic Depletion of Subcellular NAD Pools Reveals Their Interconnectivity and a Buffering Function of Mitochondria