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NIPBL
Topologically Associating Domain Boundaries Are Commonly
The Mutational Landscape of Myeloid Leukaemia in Down Syndrome
Gene Regulation by Cohesin in Cancer: Is the Ring an Unexpected Party to Proliferation?
Cohesin Mutations in Cancer: Emerging Therapeutic Targets
NIPBL Mutations and Genetic Heterogeneity in Cornelia De Lange
Principles of Genome Folding Into Topologically Associating Domains Quentin Szabo, Frederic Bantignies, Giacomo Cavalli
Table of Contents Chapter 1: Introduction
Cytogenetics
Cornelia De Lange Syndrome with NIPBL Gene Mutation: a Case Report
Transcriptional Regulators Are Upregulated in the Substantia Nigra
Characterization of the NIPBL Protein Associated with Cornelia De Lange Syndrome
NIPBL: a New Player in Myeloid Cell Ferrata Storti Foundation Differentiation
Cohesin Components Stag1 and Stag2 Differentially Influence Haematopoietic Mesoderm Development in Zebrafish Embryos
Functional Genomics of Cohesin Acetyltransferases in Human Cells
The Cohesin Loader NIPBL Interacts with Pre-Ribosomal RNA and Treacle to Regulate
Product Description SALSA® MLPA® Probemix P141-A4 NIPBL-1 & P142
(Nipbl) Haploinsufficiency on Genome-Wide Cohesin Binding and Target Gene Expression: Modeling Cornelia De Lange Syndrome Daniel A
The Cohesin-Associated Protein Wapal Is Required for Proper Polycomb-Mediated Gene Silencing
Top View
1 Diagnosing Cornelia De Lange Syndrome and Related
NIPBL Gene NIPBL, Cohesin Loading Factor
NIPBL Polyclonal Antibody Drosophila Melanogaster Nipped-B Gene Product and Fungal Scc2-Type Sister Chromatid Cohesion Proteins
Intragenic and Large NIPBL Rearrangements Revealed by MLPA in Cornelia De Lange Patients
The Genome-Wide Impact of Nipblb Loss-Of-Function on Zebrafish Gene
Supplemental Material.Pdf
BRD4 Interacts with NIPBL and BRD4 Is Mutated in a Cornelia De
De Novo Megakaryoblastic Leukemia
Genome-Wide CNV Investigation Suggests a Role for Cadherin, Wnt, and P53 Pathways in Primary Open-Angle Glaucoma Valeria Lo Faro1,2, Jacoline B
Promoterless Transposon Mutagenesis Drives Solid Cancers Via Tumor Suppressor Inactivation
Large Genomic Rearrangements in NIPBL Are Infrequent in Cornelia De Lange Syndrome
Low Tolerance for Transcriptional Variation at Cohesin Genes Is
On the Existence and Functionality of Topologically Associating Domains
NIPBL Rearrangements in Cornelia De Lange Syndrome: Evidence for Replicative Mechanism and Genotype–Phenotype Correlation
The Changing Scenario of Non-Down Syndrome Acute Megakaryoblastic
Cornelia De Lange Syndrome Genetics
Down Syndrome-Like Acute Megakaryoblastic Leukemia in A
The Cohesin Complex and Its Roles in Chromosome Biology