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G6PC
A Clinical and Molecular Review of Ubiquitous Glucose-6-Phosphatase Deficiency Caused by G6PC3 Mutations Siddharth Banka1,2* and William G Newman1,2
Tissue-Specific Alteration of Metabolic Pathways Influences Glycemic Regulation
G6PC Gene Glucose-6-Phosphatase Catalytic Subunit
Dysregulation of Post-Transcriptional Modification by Copy Number
Pancreatic Islet Beta Cell-Specific Deletion of G6pc2 Reduces Fasting Blood Glucose
Long-Read Sequencing Identified a Causal Structural Variant in An
Exploring Molecular Pathogenesis To
A Seven-Gene Signature Model Predicts Overall Survival in Kidney Renal Clear Cell Carcinoma Ling Chen1, Zijin Xiang2, Xueru Chen2, Xiuting Zhu2 and Xiangdong Peng2*
Mrna Therapy Restores Euglycemia and Prevents Liver Tumors in Murine Model of Glycogen Storage Disease
Glucose-6-Phosphatase: Novel Therapeutic Approaches for Type 2
Glucose 6 Phosphatase, Catalytic Subunit (G6PC) Rabbit Polyclonal Antibody Product Data
Evolutionary History of Glucose-6-Phosphatase Encoding
Induction of Renal and Intestinal Gluconeogenesis by Glucagon
Functional Analysis of Mouse G6pc1 Mutations Using a Novel in Situ
Glucose 6 Phosphatase, Catalytic Subunit (G6PC) (NM 000151) Human Untagged Clone Product Data
GENOME-WIDE ASSOCIATION STUDIES of CHILDHOOD BONE HEALTH by Kelly Amanda Johnson BS, University of Maryland, College Park, 2014
(G6PC) (NM 000151) Human Tagged ORF Clone Product Data
Targeted Deletion of Liver Glucose-6 Phosphatase Mimics Glycogen Storage Disease Type 1A Including Development of Multiple Adenomas
Top View
Susceptibility to Type 2 Diabetes May Be Modulated by Haplotypes in G6PC2,A Target of Positive Selection Nasser M
Analyzing the Distribution of Mutations for Glycogen Storage Disease
Predominance of the C.648G > T G6PC Gene Mutation and Late Complications in Korean Patients with Glycogen Storage Disease
Downloading the Genomic Sequences with Flanking Source of Normal Adult and Fetal Tissues