LEAD 415 9. REFERENCES Abadin HG, Hibbs BF, Pohl HR. 1997b. Breast-feeding exposure of infants to cadmium, lead, and mercury: A public health viewpoint. Toxicol Ind Health 15(4):1-24. Abadin HG, Wheeler JS, Jones DE, et al. 1997a. A framework to guide public health assessment decisions at lead sites. J Clean Technol Environ Toxicol Occup Med 6:225-237. Abbate C, Buceti R, Munao F, et al. 1995. Neurotoxicity induced by lead levels: An electrophysiological study. Int Arch Occup Environ Health 66:389-392. ACGIH. 1986. Documentation of the threshold limit values and biological exposure indices. 5th ed. Cincinnati, OH: American Conference of Governmental Industrial Hygienists, BEI-19 to BEI-23. ACGIH. 1998. 1998 TLVs and BEIs. Threshold limit values for chemical substances and physical agents. Biological exposure indices. Cincinnati, OH: American Conference of Governmental Industrial Hygienist. ACGIH. 2004. Lead. Threshold limit values for chemical substances and physical agents and biological exposure indices. Cincinnati, OH: American Conference of Governmental Industrial Hygienists. Adebonojo FO. 1974. Hematologic status of urban black children in Philadelphia: Emphasis on the frequency of anemia and elevated blood lead levels. Clin Pediatr 13:874-888. Adhikari N, Sinha N, Narayan R, et al. 2001. Lead-induced cell death in testes of young rats. J Appl Toxicol 21:275-277. Adinolfi M. 1985. The development of the human blood-CSF-brain barrier. Dev Med Child Neurol 27:532-537. Agency for Toxic Substances and Disease Registry. 1989. Decision guide for identifying substance- specific data needs related to toxicological profiles; notice. Fed Regist 54(174):37618-37634. Agency for Toxic Substances and Disease Registry. 1995. Multisite lead and cadmium exposure study with biological markers incorporated. Atlanta, GA: U.S. Department of Health and Human Services, Public Health Service, Agency for Toxic Substances and Disease Registry. Agency for Toxic Substances and Disease Registry. 1997. Public health statement for lead. Atlanta, GA: U.S. Department of Health and Human Services, Public Health Service, Agency for Toxic Substances and Disease Registry. Agency for Toxic Substances and Disease Registry. 2004a. Interaction profile for arsenic, cadmium, chromium, and lead. Atlanta, GA: Agency for Toxic Substances and Disease Registry. http://www.atsdr.cdc.gov/interactionprofiles/ip04.html. July 22, 2005. Agency for Toxic Substances and Disease Registry. 2004b. Interaction profile for lead, manganese, zinc, and copper. Atlanta, GA: Agency for Toxic Substances and Disease Registry. http://www.atsdr.cdc.gov/interactionprofiles/ip06.html. July 22, 2005. _______________________ *Not cited in text LEAD 416 9. REFERENCES Agency for Toxic Substances and Disease Registry. 2006. Interaction profile for chlorpyrifos, lead, mercury, and methylmercury. Atlanta, GA: Agency for Toxic Substances and Disease Registry. http://www.atsdr.cdc.gov/interactionprofiles/IP-11/ip11.pdf. June 14, 2007. Aguilera de Benzo Z, Fraile R, Carrion N, et al. 1989. Determination of lead in whole blood by electrothermal atomization atomic absorption spectrometry using tube and platform atomizers and dilution with Triton X-100. J Anal Atom Spectrom 4:397-400. Ahamed M, Verma S, Kumar A, et al. 2005. Environmental exposure to lead and its correlation with biochemical indices in children. Sci Total Environ 346(1-3):48-55. Ahlgren L, Liden S, Mattson, et al. 1976. X-ray fluorescence analysis of lead in human skeleton in vivo. Scand J Work Environ Health 2:82-86. *Ahmed NS, El-Gendy KS, El-Refaie AK et al. 1987. Assessment of lead toxicity in traffic controllers of Alexandria, Egypt, road intersections. Arch Environ Health 42:92-95. Aiba Y, Ohshiba S, Horiguchi S, et al. 1999. Peripheral hemodynamics evaluated by acceleration plethysmography in workers exposed to lead. Ind Health 37:3-8. Åkesson A, Stal P, Vahter M. 2000. Phlebotomy increases cadmium uptake in hemochromatosis. Environ Health Perspect 108:289-291. Al-Hakkak ZSH, Hamamy HA, Murad AMB, et al. 1986. Chromosome aberrations in workers at a storage battery plant in Iraq. Mutat Res 171:53-60. Al Khayat A, Habibullah J, Koutouby A, et al. 1997b. Correlation between maternal and cord blood lead levels. Int J Environ Health Res 7(4):323-328. Al Khayat A, Menon NS, Alidina MR. 1997a. Acute lead encephalopathy in early infancy-clinical presentation and outcome. Ann Trop Paediatr 17(1):39-44. Al-Modhefer AJA, Bradbury MWB, Simmons TJB. 1991. Observations on the chemical nature of lead in human blood serum. Clin Sci 81:823-829. Al-Neamy FR, Almehdi AM, Alwash R, et al. 2001. Occupational lead exposure and amino acid profiles and liver function tests in industrial workers. Int J Environ Health Res 11(2):181-188. Al-Rashdan A, Heitkemper D, Caruso JA. 1991. Lead speciation by HPLC-ICP-AES and HPLC-ICP­ MS. J Chromatogr Sci 29(3):98-102. Al-Saleh I, Nester M, DeVol E, et al. 2001. Relationship between blood lead concentrations, intelligence, and academic achievement of Saudi Arabian schoolgirls. Int J Hyg Environ Health 204:165­ 174. Al-Saleh I, Shinwari N, Mashour A, et al. 2005. Is lead considered as a risk factor for high blood pressure during menopause period among Saudi women? Int J Hyg Environ Health 208(5):341-356. Alessio L. 1988. Relationships between “chelatable lead” and the indicators of exposure and effect in current and past occupational life. Sci Total Environ 71:293-299. LEAD 417 9. REFERENCES Alessio L, Bertazzi PA, Monelli O, et al. 1976. Free erythrocyte protoporphyrin as an indicator of the biological effect of lead in adult males: II. Comparison between free erythrocyte protoporphyrin and other indicators of effect. Int Arch Occup Environ Health 37:89-105. Alexander FW, Delves HT. 1981. Blood lead levels during pregnancy. Int Arch Occup Environ Health 48:35-39. Alexander BH, Checkoway H, Costa-Mallen P, et al. 1998b. Interaction of blood lead and δ­ aminolevulinic acid dehydratase genotype on markers of heme synthesis and sperm production in lead smelter workers. Environ Health Perspect 106:213-216. Alexander BH, Checkoway H, Faustman EM, et al. 1998a. Contrasting associations of blood and semen lead concentrations with semen quality among lead smelter workers. Am J Ind Med 34:464-469. Alexander BH, Checkoway H, van Netten C, et al. 1996. Semen quality of men employed at a lead smelter. Occup Environ Med 53:411-416. Alexander FW, Clayton BE, Delves HT. 1974. Mineral and trace-metal balances in children receiving normal and synthetic diets. QJ Med 43:89-111. Allen LB, Siitonen PH, Thompson HC. 1998. Determination of copper, lead, and nickel in edible oils by plasma and furnace atomic spectroscopies. J Am Oil Chem Soc 75(4):477-481. Allen-Gil SM, Gubala CP, Landers DH, et al. 1997. Heavy metal accumulation in sediment and freshwater fish in U.S. arctic lakes. Environ Toxicol Chem 16(4):733-741. Alomran AH, Shleamoon MN. 1988. The influence of chronic lead exposure on lymphocyte proliferative response and immunoglobulin levels in storage battery workers. J Biol Sci Res 19:575-585. Altman PL, Dittmer DS. 1974. Biological handbooks: Biology data book. Vol. III. 2nd ed. Bethesda, MD: Federation of American Societies for Experimental Biology, 1987-2008, 2041. Altmann L, Sveinsson K, Kraemer U, et al. 1998. Visual functions in 6-year-old children in relation to lead and mercury levels. Neurotoxicol Teratol 20(1):9-17. Alvares AP, Kapelner S, Sassa S, et al. 1975. Drug metabolism in normal children, lead-poisoned children, and normal adults. Clin Pharmacol Ther 17:179-183. American Academy of Pediatrics. 1998. Screening for elevated blood lead levels. Policy statement. Committee on environmental health. Pediatrics 101(6):1072-1078. Amitai Y, Graef JW, Brown MJ, et al. 1987. Hazards of deleading homes of children with lead poisoning. Am J Dis Child 141:758-760. Anders E, Bagnell CR, Krigman M, et al. 1982. Influence of dietary protein composition on lead absorption in rats. Bull Environ Contam Toxicol 28:61-67. Andersen ME, Krishnan K. 1994. Relating in vitro to in vivo exposures with physiologically based tissue dosimetry and tissue response models. In: Salem H, ed. Animal test alternatives: Refinement, reduction, replacement. New York: Marcel Dekker, Inc., 9-25. LEAD 418 9. REFERENCES Andersen ME, Clewell HJ III, Gargas ML, et al. 1987. Physiologically based pharmacokinetics and the risk assessment process for methylene chloride. Toxicol Appl Pharmacol 87:185-205. Anderson RJ. 1987. Peripheral nerve conduction velocities and excitability. In: Lowndes HE, ed. Electrophysiology in neurotoxicology, Vol. 11. Piscataway, NJ: Department of Pharmacology and Toxicology, 51-69. Angle CR, Kuntzelman DR. 1989. Increased erythrocyte protoporphyrins and blood lead--a pilot study of childhood growth patterns. J Toxicol Environ Health 26:149-156. Angle CR, McIntire MS. 1978. Low level lead and inhibition of erythrocyte pyrimidine nucleotidase. Environ Res 17:296-302. Angle CR, Manton WI, Stanek KL. 1995. Stable isotope identification of lead sources in preschool children: The Omaha study. Clin Toxicol 33(6):657-662. Angle CR, Marcus A, Cheng I-H, et al. 1984. Omaha childhood blood lead and environmental lead: A linear total exposure model. Environ Res 35:160-170. Angle CR, McIntire MS, Swanson MS, et al. 1982. Erythrocyte nucleotides in children--increased blood lead and cytidine triphosphate. Pediatr Res 16:331-334. Annesi-Maesano I, Pollitt R, King G, et al. 2003. In utero exposure to lead and cord blood total IgE. Is there a connection? Allergy 58:589-594. Anttila A, Heikkila P, Nykyri E, et al. 1996. Risk of nervous system cancer among workers exposed to lead. J Occup Environ Med 38(2):131-136. Anttila A, Heikkila P, Pukkala E, et al. 1995. Excess lung cancer among workers exposed to lead. Scand J Work Environ Health 21:460-469. APHA. 1998. Method 3111.