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Paraesophageal Hernia and Intrathoracic Diverticulitis

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Paraesophageal Hernia and Intrathoracic Diverticulitis J Am Board Fam Pract: first published as 10.3122/15572625-13-2-141 on 1 March 2000. Downloaded from Paraesophageal Hernia and Intrathoracic Diverticulitis Michael K. Magill, MD, and Christine Gazak, JI1A, MPH Paraesophageal hernia is a potentially lethal condi­ litis. Although he improved initially, he developed tion that has not been previously discussed in the increasing pain despite continued outpatient treat­ family medicine literature. It accounts for 3.5% to ment with metronidazole and ciprofloxacin. 5 % 1 of all hiatal hernias. Diagnosis of paraesoph­ His medical history was notable for a myocardial ageal hernia can be difficult. Patients are usually infarction approximately 7 years earlier, which had elderly and have symptoms of esophageal reflux or been well tolerated. He had a right bundle branch postprandial pain, bloating, and vomiting. The pa­ block of uncertain duration. He also had a history tient might have a history of hiatal hernia. of deep venous thrombosis for which he had re­ We report here a case of paraesophageal hernia, cently begun taking warfarin. complicated by herniation of colon into the thorax, At admission his respiratory rate was 24/min, with intrathoracic diverticulosis, probable divertic­ pulse 90 beats per minute and regular, blood pres­ ulitis, and myocardial infarction. Family physicians sure 90/60 mm Hg. He was initially alert and ori­ should be aware of the symptoms of paraesophageal ented. His lips were moderately cyanotic, and ox­ hernia so they can promptly diagnose and manage ygen saturation was 94% on 3 L of oxygen by nasal this potentially lethal condition. cannula. His lungs were clear to auscultation, and his heart had no murmurs or gallops. During his abdominal examination, he had normal bowel Case Report sounds and no distention or tenderness to palpita­ A 90-year-old man was admitted to a community tion. hospital complaining of abdominal pain. The pain The initial laboratory evaluation included a was located in the left upper quadrant of the abdo­ white blood cell count of 15,200/JJ.L with a left men, and he described it as steady and "aggravat­ . shift. His international normalized ratio was 1.1. ing." He had no associated fever and did not report His serum creatine kinase was 161 UIL with an MB http://www.jabfm.org/ a history of esophageal reflux-type symptoms, but fraction of 12.5%. An electrocardiogram showed a he did have several episodes of vomiting nonbloody right bundle branch block with evidence of old fluid. There was no associated diarrhea, constipa­ myocardial injury. The chest radiograph showed a tion, melena, or hematochezia. large hiatal hernia and barium retained from a The patient also complained of intermittent, previous radiologic examination in diverticulae poorly characterized pain in his left lower anterior located both above and below the diaphragm thorax, which he said was different from his ab­ (Figure 1). on 25 September 2021 by guest. Protected copyright. dominal pain. He denied exertional chest pain but The patient was presumed to have a paraesoph­ did complain of shortness of breath when walking ageal hernia, recurrent diverticulitis, and new-onset up one flight of stairs. He denied orthopnea, par­ myocardial infarction. The diverticulitis was oxysmal nocturnal dyspnea, and edema. thought to be located in the intrathoracic bowel in He had been released from the hospital 8 days view of the benign findings of the abdominal ex­ earlier after a 4-day admission for nausea, de­ amination. During his hospitalization the patient creased appetite, and weight loss. At that time he showed fluctuating orientation and memory, had been given antibiotics for presumed diverticu- thought to represent dementia. The family re­ quested that no aggressive treatment be performed, and the patient was given antibiotics until his ab­ Submitted, revised, 2 May 1999. dominal pain improved. He did not develop signs From the Department of Family and Preventive Medicine (MKM, CG), University of Utah School of Medicine, Salt of bowel obstruction and was released from the Lake City. Reprints are not available. hospital. ParaesophageaI Hernia 141 J Am Board Fam Pract: first published as 10.3122/15572625-13-2-141 on 1 March 2000. Downloaded from rarer cases of paraesophageal hiatal hernias in which the transverse colon might be involved in­ clude a report of a patient who had a paraesopha­ geal hernia in which herniation of the splenic flex­ ure led to colonic obstruction.6 Another report described a rarer event in which a hernia through the aortic hiatus allowed passage of first the stom­ ach and then the colon through the diaphragm.7 Paraesophageal hiatal hernias can occur, though very seldom, as congenital problems diagnosed in infancy.s They are found predominately as ac­ quired problems in the elderly. Patients might complain of reflux, epigastric pressure or pain, postprandial vomiting of food, postprandial dys­ pnea, anemia, or symptoms of intermittent volvu­ lus. 9 When the symptoms and signs of paraesoph­ ageal hernia include sudden onset of abdominal pain and vomiting, they are likely a result of stran­ gulation of herniated stomach or bowel, which is a medical emergency. Although paraesophageal hernias account for Figure 1. Chest radiograph showing large hiatal hernia only 5% of all hiatal hernias, they are dangerous and barium located in diverticula above and belqw the because symptoms can be nonspecific, leading to diaphragm. delays in diagnosis and resultant morbidity or mor­ tality. Paraesophageal hernia should be suspected in elderly patients who have symptoms ranging Discussion from heartburn and vague abdominal discomfort to Hiatal hernias occur as three major types: type I, vomiting and severe pain. These patients might the classic sliding hiatal hernia in which the esopha­ have few or no localizing abdominal findings on go gastric junction is located above the diaphragm; physical examination, but they have evidence of lO type IT, in which the esophagogastric junction re­ hiatal hernia on chest radiographs. Wo et al rec­ http://www.jabfm.org/ mains below the diaphragm but the fundus of the ommended that patients with heartburn, plus post­ stomach is prolapsed above the diaphragm; and prandial distress (pain in the epigastrium or chest, type ill, a mixed type in which both the esophago­ dyspnea, nau ea or vomiting during or after eating), gastric junction and some or all the stomach are be suspected of having paraesophageal hernia, es­ above the diaphragm, but in which the esophago­ pecially type ill, rather than simply the more com­ gastric junction remains more caudal than the fun­ mon sliding hiatal hernia (type I). 2 4 on 25 September 2021 by guest. Protected copyright. dus. - Most para esophageal hernias are type ill; The finding of a stomach-type air-fluid level or type IT is relatively uncommon. large-bowel gas pattern within the mediastinum The hernia is due to attenuation of the anterior should further raise suspicion of a paraesophageal and lateral components of the phrenicoesophageal rather than sliding hiatal hernia. The differential fascial complex. The resulting weakness in the diagnosis for mediastinal masses occurring with muscle fibers creates mobility and instability within similar appearance includes pericardial effusion or the hiatus, which allows for herniation into the cyst, bronchogenic cyst, or esophageal diverticu­ mediastinum of the stomach, the transverse colon, lum. 11 Upper gastrointe tinal endoscopy can estab­ and on rare occasions the entire bowel or other lish the diagnosis, but a type ill hernia is often intraabdominal organs, such as the spleen. missed by endoscopy. Barium esophagogram might Listerud and Harkins5 described 11 muscle vari­ be necessary to confirm a type ill hernia.10 ations in the formation of an esophageal hiatus. In Most authors recommend surgical repair of more than 90% of the reported cases, the hiatus is paraesophageal hernias at the time of diagnosis produced from the right crus. Documentation of because of the risk of strangulation and its high 142 JABFP March- April2000 Vol. 13 0.2 J Am Board Fam Pract: first published as 10.3122/15572625-13-2-141 on 1 March 2000. Downloaded from mortality rate. 12 Older case series included large 5. Listerud MD, Harkins HN. Anatomy of the esoph­ proportions of emergency procedures. For exam­ ageal hiatus. Arch Surgery 1958;76:835-42. ple, of 29 patients with paraesophageal hernias re­ 6. Farrell B, Gerard PS, Bryk D. Paraesophageal hernia ported by Hill,13 10 required emergency surgical causing colonic obstruction. J Clin Gastroenterol 1991;13:188-90. repair of the strangulation and obstruction, and 2 of 7. Wagner RB, Barry MJ, Manney MS. Sequential her­ those cases resulted in death. More recent series niation of stomach, then colon through the aortic have described much better elective surgical out­ hiatus. South Med J 1997;90:338-40. comes, particularly those in which repair is accom­ 8. Jawad A, al-Samarrai AI, al-Mofada S, et al. Congen­ plished laparoscopically.9,14-16 Successful emer­ ital para-oesophageal hiatal hernia in infancy. Pedi­ gency laparoscopic repair has also been described.17 atr Surg Int 1998;13(2-3):91-4. In some instances, symptoms have been present for 9. Hawasli A, Zonca S. Laparoscopic repair of para­ many years, and the patient might be unwilling to esophageal hiatal hernia. Am Surg 1998;64:703-10. undergo surgery to correct the condition. Some 10. WO JM, Branum GD, Hunter JG, et aI., Clinical have suggested that the risk of emergency compli­ features of type III (mixed) paraesophageal hernia. Am] Gastroenterol 1996;91:914-6. cations of hiatal hernias might be lower than gen­ 11. Malatino LS, Presicci PF, Bellanuova I, Di Grande erally believed, given the occasional patient in A, Consoli A, Mangiafico RA. Giant paraesophageal whom bowel is only incidentally identified in the hernia in an asymptomatic old man.
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