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Placental Abruption
Placental Abruption Definition: Placental separation, either partial or complete prior to the birth of the fetus Incidence 0.5 – 1% (4). Risk factors include hypertension, smoking, preterm premature rupture of membranes, cocaine abuse, uterine myomas, and previous abruption (5). Diagnosis: Symptoms (may present with any or all of these) o Vaginal bleeding (usually dark and non-clotting). o Abdominal pain and/or back pain varying from intermittent to severe. o Uterine contractions are usually present and may vary from low amplitude/high frequency to hypertonus. o Fetal distress or fetal death. Ultrasound o Adherent retroplacental clot OR may just appear to be a thick placenta o Resolving hematomas become hypoechoic within one week and sonolucent within 2 weeks May be a diagnosis of exclusion if vaginal bleeding and no other identified etiology Consider in differential with uterine irritability on toco and cat II-III tracing, as small proportion present without bleeding Classification: Grade I: Slight vaginal bleeding and some uterine irritability are usually present. Maternal blood pressure, and fibrinogen levels are unaffected. FHR remains normal. Grade II: Mild to moderate vaginal bleeding seen; tetanic contractions may be present. Blood pressure usually normal, but tachycardia may be present. May be postural hypotension. Decreased fibrinogen; with levels below 250 mg percent; may be evidence of fetal distress. Reviewed 01/16/2020 1 Updated 01/16/2020 Grade III: Bleeding is moderate to severe, but may be concealed. Uterus tetanic and painful. Maternal hypotension usually present. Fetal death has occurred. Fibrinogen levels are less then 150 mg percent with thrombocytopenia and coagulation abnormalities. -
NUR 306- MIDWIWERY-II.Pdf
THE CATHOLIC UNIVERSITY OF EASTERN AFRICA P.O. Box 62157 A. M. E. C. E. A 00200 Nairobi - KENYA Telephone: 891601-6 Fax: 254-20-891084 MAIN EXAMINATION E-mail:[email protected] SEPTEMBER-DECEMBER 2020 TRIMESTER SCHOOL OF NURSING REGULAR PROGRAMME NUR/UNUR 306: MIDWIFERY II-LABOUR Date: DECEMBER2020 Duration: 3 Hours INSTRUCTIONS: i. All questions are compulsory ii. Indicate the answers in the answer booklet provided PART -I: MULTIPLE CHOICE QUESTIONS (MCQs) (20 MARKS): Q1.The following are two emergencies that can occur in third stage of labour: a) Cord prolapsed, foetal distress. b) Ruptured uterus ,foetal distress. c) Uterine inversion, cord pulled off. d) Cord round the neck, foetal distress. Q2 A woman in preterm labor at 30 weeks of gestation receives two 12-mg doses of betamethasone intramuscularly. The purpose of this pharmacologic treatment is to: a). Stimulate fetal surfactant production. b). Reduce maternal and fetal tachycardia associated with ritodrine administration. c). Suppress uterine contractions. d). Maintain adequate maternal respiratory effort and ventilation during magnesium sulfate therapy. Q3. Episiotomy should only be considered in case of : a) Breech, vacuum delivery, scarring from genital cutting, fetal distress. b) Primigravida, disturbed woman, genital cutting, obstructed labor. c) Obstructed labor, hypertonic contractions, multigravida, forceps delivery. Cuea/ACD/EXM/APRIL 2020/SCHOOL OF NURSING Page 1 ISO 9001:2015 Certified by the Kenya Bureau of Standards d) Primigravida, hypotonic contractions, fetal distress, genital cutting. Q4.A multiporous woman in labour tells the midwife “I feel like opening the bowels”.How should the midwife respond? a) Allow the woman to use the bedpan. -
Uterine Rupture After Misoprostol Use for Termination of Pregnancy in Second Trimester with Previous Caesarean Section: a Case Report Marjan Ghaemi*
Case Report iMedPub Journals Critical Care Obstetrics and Gynecology 2018 http://www.imedpub.com/ Vol.4 No.3:14 ISSN 2471-9803 DOI: 10.21767/2471-9803.1000167 Uterine Rupture after Misoprostol Use for Termination of Pregnancy in Second Trimester with Previous Caesarean Section: A Case Report Marjan Ghaemi* Yas Hospital, Tehran University of Medical Sciences, Tehran, Iran *Corresponding author: Marjan Ghaemi, Yas Hospital, Tehran University of Medical Sciences, Tehran, Iran, Tel: 0098-912-1967735; E-mail: [email protected] Received date: September 25, 2018; Accepted date: October 16, 2018; Published date: October 22, 2018 Copyright: © 2018 Ghaemi M. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Citation: Ghaemi M (2018) Uterine Rupture after Misoprostol Use for Termination of Pregnancy in Second Trimester with Previous Caesarean Section: A Case Report. Crit Care Obst Gyne Vol.4 No.3:14. cesarean section, hospitalized for severe oligohydramnios with unknown etiology and no history of fluid leakage. In her Abstract previous surgical history, she mentioned laparoscopic cholecystectomy 10 years ago. Considering her medical history Modalities for termination of pregnancy may result in some and normal kidney and bladder in fetal ultrasound, we did not adverse effects especially uterine rupture in women with have proved data for her severe oligohydramnios. Amnio- the previous cesarean section. Here we report uterine infusion was performed to prevent foetal cord compression and rupture in the 23rd week of pregnancy after Misoprostol uses for abortion induction in second pregnancy with the to assess foetal structures. -
Delivery Mode for Prolonged, Obstructed Labour Resulting in Obstetric Fistula: a Etrr Ospective Review of 4396 Women in East and Central Africa
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by eCommons@AKU eCommons@AKU Obstetrics and Gynaecology, East Africa Medical College, East Africa 12-17-2019 Delivery mode for prolonged, obstructed labour resulting in obstetric fistula: a etrr ospective review of 4396 women in East and Central Africa C. J. Ngongo T. J. Raassen L. Lombard J van Roosmalen S. Weyers See next page for additional authors Follow this and additional works at: https://ecommons.aku.edu/eastafrica_fhs_mc_obstet_gynaecol Part of the Obstetrics and Gynecology Commons Authors C. J. Ngongo, T. J. Raassen, L. Lombard, J van Roosmalen, S. Weyers, and Marleen Temmerman DOI: 10.1111/1471-0528.16047 www.bjog.org Delivery mode for prolonged, obstructed labour resulting in obstetric fistula: a retrospective review of 4396 women in East and Central Africa CJ Ngongo,a TJIP Raassen,b L Lombard,c J van Roosmalen,d,e S Weyers,f M Temmermang,h a RTI International, Seattle, WA, USA b Nairobi, Kenya c Cape Town, South Africa d Athena Institute VU University Amsterdam, Amsterdam, The Netherlands e Leiden University Medical Centre, Leiden, The Netherlands f Department of Obstetrics and Gynaecology, Ghent University Hospital, Ghent, Belgium g Centre of Excellence in Women and Child Health, Aga Khan University, Nairobi, Kenya h Faculty of Medicine and Health Science, Ghent University, Ghent, Belgium Correspondence: CJ Ngongo, RTI International, 119 S Main Street, Suite 220, Seattle, WA 98104, USA. Email: [email protected] Accepted 3 December 2019. Objective To evaluate the mode of delivery and stillbirth rates increase occurred at the expense of assisted vaginal delivery over time among women with obstetric fistula. -
Management of Prolonged Decelerations ▲
OBG_1106_Dildy.finalREV 10/24/06 10:05 AM Page 30 OBGMANAGEMENT Gary A. Dildy III, MD OBSTETRIC EMERGENCIES Clinical Professor, Department of Obstetrics and Gynecology, Management of Louisiana State University Health Sciences Center New Orleans prolonged decelerations Director of Site Analysis HCA Perinatal Quality Assurance Some are benign, some are pathologic but reversible, Nashville, Tenn and others are the most feared complications in obstetrics Staff Perinatologist Maternal-Fetal Medicine St. Mark’s Hospital prolonged deceleration may signal ed prolonged decelerations is based on bed- Salt Lake City, Utah danger—or reflect a perfectly nor- side clinical judgment, which inevitably will A mal fetal response to maternal sometimes be imperfect given the unpre- pelvic examination.® BecauseDowden of the Healthwide dictability Media of these decelerations.” range of possibilities, this fetal heart rate pattern justifies close attention. For exam- “Fetal bradycardia” and “prolonged ple,Copyright repetitive Forprolonged personal decelerations use may onlydeceleration” are distinct entities indicate cord compression from oligohy- In general parlance, we often use the terms dramnios. Even more troubling, a pro- “fetal bradycardia” and “prolonged decel- longed deceleration may occur for the first eration” loosely. In practice, we must dif- IN THIS ARTICLE time during the evolution of a profound ferentiate these entities because underlying catastrophe, such as amniotic fluid pathophysiologic mechanisms and clinical 3 FHR patterns: embolism or uterine rupture during vagi- management may differ substantially. What would nal birth after cesarean delivery (VBAC). The problem: Since the introduction In some circumstances, a prolonged decel- of electronic fetal monitoring (EFM) in you do? eration may be the terminus of a progres- the 1960s, numerous descriptions of FHR ❙ Complete heart sion of nonreassuring fetal heart rate patterns have been published, each slight- block (FHR) changes, and becomes the immedi- ly different from the others. -
A Guide to Obstetrical Coding Production of This Document Is Made Possible by Financial Contributions from Health Canada and Provincial and Territorial Governments
ICD-10-CA | CCI A Guide to Obstetrical Coding Production of this document is made possible by financial contributions from Health Canada and provincial and territorial governments. The views expressed herein do not necessarily represent the views of Health Canada or any provincial or territorial government. Unless otherwise indicated, this product uses data provided by Canada’s provinces and territories. All rights reserved. The contents of this publication may be reproduced unaltered, in whole or in part and by any means, solely for non-commercial purposes, provided that the Canadian Institute for Health Information is properly and fully acknowledged as the copyright owner. Any reproduction or use of this publication or its contents for any commercial purpose requires the prior written authorization of the Canadian Institute for Health Information. Reproduction or use that suggests endorsement by, or affiliation with, the Canadian Institute for Health Information is prohibited. For permission or information, please contact CIHI: Canadian Institute for Health Information 495 Richmond Road, Suite 600 Ottawa, Ontario K2A 4H6 Phone: 613-241-7860 Fax: 613-241-8120 www.cihi.ca [email protected] © 2018 Canadian Institute for Health Information Cette publication est aussi disponible en français sous le titre Guide de codification des données en obstétrique. Table of contents About CIHI ................................................................................................................................. 6 Chapter 1: Introduction .............................................................................................................. -
Chapter 9 Risk Factors
Chapter 9 Risk factors 9.1 Introduction The previous chapters have all focused on adverse pregnancy and birth outcomes as risk factors for foetal loss and neonatal death. The present chapter takes the discussion and analysis one layer further to the underlying risk factors for the selected adverse pregnancy and birth outcomes themselves. These risk factors are more remote in the causal chain and include maternal factors, complications of the placenta, cord, and membranes, and birth complications. These have been referred to earlier, in Chapter 3, in the operationalisation of the conceptual model (Figure 3.6). Besides their direct effect on pregnancy/birth outcome, they are believed to affect foetal loss (i.e. spontaneous abortion and stillbirth), both directly and indirectly, and neonatal death indirectly through the intermediate outcomes (i.e. adverse pregnancy and birth outcomes). As became clear in Chapter 3, many different causes and risk factors underlie the adverse pregnancy and birth outcomes, but what appear to be main risk factors? Are these risk factors also important in regions in transition and, more specifically, in Kerala? Moreover, does the relative importance of these risk factors change during the epidemiologic transition? A region’s position within the transition could very well be reflected by its specific combination of underlying risk factors. Factors such as infections and malnutrition are likely to be important during the early of the transition, while others such as diabetes and advanced maternal age may become more significant during later stages. This could lead to a redefinition of the concepts of endogenous and exogenous mortality (see Chapter 2). -
Concurrent Intraoperative Uterine Rupture and Placenta Accreta. Do
Concurrent intraoperative uterine rupture and placenta accreta Romanian Journal of Anaesthesia and Intensive Care 2018 Vol 25 No 1, 83-85 CASE REPORT DOI: http://dx.doi.org/10.21454/rjaic.7518.251.acc Concurrent intraoperative uterine rupture and placenta accreta. Do preoperative chronic hypertension, preterm premature rupture of membranes, chorioamnionitis, and placental abruption provide warning to this rare occurrence? M. Anthony Cometa, Scott M. Wasilko, Adam L. Wendling Department of Anesthesiology, Division of Obstetric Anesthesiology, University of Florida College of Medicine, Gainesville, FL, USA Abstract Uterine and placental pathology can be a major cause of morbidity and mortality in the parturient and infant. When presenting alone, placental abruption, uterine rupture, or placenta accreta can result in significant peripartum hemorrhage, requiring aggressive surgical and anesthetic management; however, the presence of multiple concurrent uterine and placental pathologies can result in significant morbidity and mortality. We present the anesthetic management of a parturient who underwent an urgent cesarean delivery for non- reassuring fetal tracing in the setting of chronic hypertension, preterm premature rupture of membranes, and chorioamnionitis who was subsequently found to have placental abruption, uterine rupture, and placenta accreta. Keywords: preterm premature rupture of membranes (PPROM), chorioamnionitis, placental abruption, uterine rupture, placenta accrete, cesarean-hysterectomy Received: 14 March 2018 / Accepted: 30 March 2018 Rom J Anaesth Intensive Care 2018; 25: 83-85 operative bleeding that requires aggressive fluid and blood product management and resuscitation [5]. At present, the literature does not reference the Introduction presentation and anesthetic management of these aforementioned conditions being concurrently present Various placental and uterine pathologies can result in one parturient. -
OBGYN-Study-Guide-1.Pdf
OBSTETRICS PREGNANCY Physiology of Pregnancy: • CO input increases 30-50% (max 20-24 weeks) (mostly due to increase in stroke volume) • SVR anD arterial bp Decreases (likely due to increase in progesterone) o decrease in systolic blood pressure of 5 to 10 mm Hg and in diastolic blood pressure of 10 to 15 mm Hg that nadirs at week 24. • Increase tiDal volume 30-40% and total lung capacity decrease by 5% due to diaphragm • IncreaseD reD blooD cell mass • GI: nausea – due to elevations in estrogen, progesterone, hCG (resolve by 14-16 weeks) • Stomach – prolonged gastric emptying times and decreased GE sphincter tone à reflux • Kidneys increase in size anD ureters dilate during pregnancy à increaseD pyelonephritis • GFR increases by 50% in early pregnancy anD is maintaineD, RAAS increases = increase alDosterone, but no increaseD soDium bc GFR is also increaseD • RBC volume increases by 20-30%, plasma volume increases by 50% à decreased crit (dilutional anemia) • Labor can cause WBC to rise over 20 million • Pregnancy = hypercoagulable state (increase in fibrinogen anD factors VII-X); clotting and bleeding times do not change • Pregnancy = hyperestrogenic state • hCG double 48 hours during early pregnancy and reach peak at 10-12 weeks, decline to reach stead stage after week 15 • placenta produces hCG which maintains corpus luteum in early pregnancy • corpus luteum produces progesterone which maintains enDometrium • increaseD prolactin during pregnancy • elevation in T3 and T4, slight Decrease in TSH early on, but overall euthyroiD state • linea nigra, perineum, anD face skin (melasma) changes • increase carpal tunnel (median nerve compression) • increased caloric need 300cal/day during pregnancy and 500 during breastfeeding • shoulD gain 20-30 lb • increaseD caloric requirements: protein, iron, folate, calcium, other vitamins anD minerals Testing: In a patient with irregular menstrual cycles or unknown date of last menstruation, the last Date of intercourse shoulD be useD as the marker for repeating a urine pregnancy test. -
A Rare Case of Placenta Percreta Causing Uterine Rupture and Massive Haemoperitoneum in 2Nd Trimester: an Obvious Near Miss
International Journal of Reproduction, Contraception, Obstetrics and Gynecology Parmar C et al. Int J Reprod Contracept Obstet Gynecol. 2021 Jun;10(6):2495-2497 www.ijrcog.org pISSN 2320-1770 | eISSN 2320-1789 DOI: https://dx.doi.org/10.18203/2320-1770.ijrcog20212201 Case Report A rare case of placenta percreta causing uterine rupture and massive haemoperitoneum in 2nd trimester: an obvious near miss Chirayu Parmar*, Mittal Parmar, Gayatri Desai Department of Gynecology, Kasturba Hospital, SEWA Rural, Jhagadi, Gujarat, India Received: 30 December 2020 Accepted: 05 May 2021 *Correspondence: Dr. Chirayu Parmar, E-mail: [email protected] Copyright: © the author(s), publisher and licensee Medip Academy. This is an open-access article distributed under the terms of the Creative Commons Attribution Non-Commercial License, which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. ABSTRACT Placenta accreta spectrum is very rarely encountered with ruptured uterus and is commonly seen in third trimester of pregnancy. Hereby, a case of placenta percreta with uterine rupture in second trimester of pregnancy is presented. 40 year old women with previous 2 LSCS presented in emergency department with ninteen weeks pregnancy and massive haemoperitoneum. Emergency laprotomy revealed uterine rupture alnong with placenta percreta for which obstetric hysterectomy was done. Although, a rare occurrence, obstetricians should consider patients placenta accreta spectrum in -
Appendix 1. Coding of All Adverse Maternal and Newborn Outcomes
Appendix 1. Coding of All Adverse Maternal and Newborn Outcomes Adverse Outcome Source Variables Coding Preeclampsia Superimposed preeclampsia ICD-10 Pre-existing hypertensive disorder with O11.001- superimposed proteinuria O11.004, O11.009 Preeclampsia ICD-10 Gestational (pregnancy-induced) O13.001- hypertension without significant O13.004, proteinuria (includes gHTN and mild pre- O13.009 eclampsia) Gestational (pregnancy-induced) O14.001- hypertension with significant proteinuria O14.004, O14.009 ICD-10 Unspecified maternal hypertension O16.001- O16.004, O16.009 ICD-10 Eclampsia in pregnancy, labour, O15.001- puerperium, unspecified time O15.909 Gestational diabetes mellitus Gestational diabetes ICD-10 Diabetes mellitus arising in pregnancy O24.801- (gestational) O24.804, O24.809 Gestational diabetes BCPDR Insulin-dependent or non-insulin dependent diabetes mellitus in pregnancy Spontaneous preterm birth <32 weeks Gestational age <32 weeks BCPDR Final estimate of gestational age per BCPDR algorithm Induction of labor BCPDR Labour initiation No PROM ICD-10 Premature rupture of membranes O42.011- O42.909 Indicated preterm birth <37 weeks Schummers L, Hutcheon JA, Bodnar LM, Lieberman E, Himes KP. Risk of adverse pregnancy outcomes by prepregnancy body mass index: a population-based study to inform prepregnancy weight loss counseling. Obstet Gynecol 2014;125. The authors provided this information as a supplement to their article. © Copyright 2014 American College of Obstetricians and Gynecologists. Page 1 of 7 Gestational age <37 weeks -
Early Accreta and Uterine Rupture in the Second Trimester
Open Access Case Report DOI: 10.7759/cureus.2904 Early Accreta and Uterine Rupture in the Second Trimester Joshua A. Ronen 1 , Krystal Castaneda 2 , Sara Y. Sadre 3 1. Internal Medicine, Texas Tech University Health Sciences Center of the Permian Basin, Odessa, USA 2. MS3/Ross University School of Medicine, California Hospital Medical Center, Los Angeles, USA 3. MS4/Ross University School of Medicine, California Hospital Medical Center, Los Angeles, USA Corresponding author: Joshua A. Ronen, [email protected] Abstract The differential diagnosis of third trimester bleeding can range from placenta abruptia to placenta previa to uterine rupture and the placenta accreta spectrum (PAS). However, patients with risk factors such as multiple cesarean sections (c-sections), advanced maternal age (AMA), grand multiparity, and single-layer uterine closure are at greater risk of developing these complications earlier than we would traditionally expect. This case recounts a 38-year-old gravida 6 preterm 3 term 1 abortus 1 live 4 (G6P3114) at 23 weeks and five days gestational age (GA) with a past medical history of preterm pregnancy, pre-eclampsia, chronic abruptia, three previous c-sections, and low-lying placenta who presented to the emergency department (ED) with vaginal bleeding. Initial workup revealed placenta accreta and possible percreta. The patient was placed on intramuscular (IM) corticosteroids in anticipation of preterm delivery. As soon as the patient was stable, she was discharged home. She presented to a different hospital the next day with the same complaints. Imaging was consistent with accreta and her presentation with abruption. During the hospital stay, the patient went into threatened preterm labor (PTL).