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Perforated Ulcers Shaleen Sathe, MS4 Christina Lebedis, MD CASE HISTORY
Perforated Ulcers Shaleen Sathe, MS4 Christina LeBedis, MD CASE HISTORY 54-year-old male with known history of hypertension presents with 2 days of acute onset abdominal pain, nausea, vomiting, and diarrhea, with periumbilical tenderness and abdominal distention on exam, without guarding or rebound tenderness. Labs, including CBC, CMP, and lipase, were unremarkable in the emergency department. Radiograph Perforated Duodenal Ulcer Radiograph of the chest in the AP projection shows large amount of free air under diaphragm (blue arrows), suggestive of intraperitoneal hollow viscus perforation. CT Perforated Duodenal Ulcer CT of the abdomen in the axial projection (I+, O-), at the level of the inferior liver edge, shows large amount of intraperitoneal free air (blue arrows) in lung window (b), and submucosal edema in the gastric antrum and duodenal bulb (red arrows), suggestive of a diagnosis of perforated bowel, most likely in the region of the duodenum. US Perforated Gastric Ulcer US of the abdomen shows perihepatic fluid (blue arrow) and free fluid in the right paracolic gutter (not shown), concerning for intraperitoneal pathology. Radiograph Perforated Gastric Ulcer Supine radiograph of the abdomen shows multiple air- filled dilated loops of large bowel, with air lucencies on both sides of the sigmoid colon wall (green arrows), consistent with Rigler sign and perforation. CT Perforated Gastric Ulcer CT of the abdomen in the axial (a) and sagittal (b) projections (I+, O-) shows diffuse wall thickening of the gastric body and antrum (green arrows) with an ulcerating lesion along the posterior wall of the stomach (red arrows), and free air tracking adjacent to the stomach (blue arrow), concerning for gastric ulcer perforation. -
Upper Gastrointestinal Endoscopy Prior to Laparoscopic Cholecystectomy: a Clinical Study at a Tertiary Care Centre in Central India
International Surgery Journal K olla V et al. Int Surg J. 2016 May;3(2):637-642 http://www.ijsurgery.com pISSN 2349-3305 | eISSN 2349-2902 DOI: http://dx.doi.org/10.18203/2349-2902.isj20161136 Research Article Upper gastrointestinal endoscopy prior to laparoscopic cholecystectomy: a clinical study at a tertiary care centre in central India Venkatesh Kolla, Neelam Charles*, Sanjay Datey, Devendra Mahor, Anand Gupta, Sanjeev Malhotra Department of General Surgery, SAMCPGI, Indore, MP, India Received: 11 January 2016 Revised: 21 February 2016 Accepted: 29 February 2016 *Correspondence: Dr. Neelam Charles, E-mail: [email protected] Copyright: © the author(s), publisher and licensee Medip Academy. This is an open-access article distributed under the terms of the Creative Commons Attribution Non-Commercial License, which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. ABSTRACT Background: Cholelithiasis is one of the most common problems encountered in surgery. It’s an immense challenge to discriminate between gastrointestinal symptoms due to gall stones or any other causes. These gastrointestinal symptoms have been related to gallstones but causal relationship has not been established yet, which is highly discouraging for the operating surgeon. An objectives of the study was to analyze the use of upper gastrointestinal endoscopy (UGE) as a pre-operative investigative tool in gallstone disease patients presenting with chronic dyspepsia. Methods: This prospective observational study was conducted on 216 patients at Department of Surgery at Aurobindo PG institute. The data collected from the patients included personal information, presenting signs and symptoms, investigations including USG, UGE finding, biopsy reports if present, medications, surgery details, any post-operative complications. -
Helicobacter Pylori (Hp) Infection of the Stomach and Relevant GI Symptoms
Name: DOB: PHN/ULI: RHRN: RefMD: Dr. RefMD Fax: RefDate: Date Today: March 8, 2016 CONFIRMATION: Referral Received Refractory TRIAGE CATEGORY: Enhanced Primary Care Pathway REFERRAL STATUS: CLOSED H. PYLORI Dear Dr. , The above-named patIent was referred to GI-CAT for further assessment of refractory Helicobacter pylori (Hp) infection of the stomach and relevant GI symptoms. Based on full review of your referral, It has been determined that management of this patient within the Enhanced Primary Care Pathway is appropriate, without need for specialist consultation at this time. ThIs clInIcal pathway has been developed by the Calgary Zone PrImary Care Network In partnershIp wIth the SectIon of Gastroenterology and Alberta Health ServIces. These local guidelines are based on best available clinical evIdence, and are practical in the prImary care setting. This package includes: 1. Focused summary of Hp relevant to prImary care 2. Summary of 2016 CanadIan AssocIatIon of Gastroenterology Guidelines for treatment of Helicobacter pylori 3. Review of your patient’s Hp treatment hIstory 4. Recommended next-round Hp treatment regImen 5. Checklist for your in-clinic followup of this patient This referral is CLOSED. If you would like to discuss this referral with a Gastroenterologist, call Specialist LINK, a dedicated GI phone consultation service, available 08:00-17:00 weekdays at 403-910-2551 or toll-free 1-855-387-3151. If your patient completes the Enhanced Primary Care Pathway and remains symptomatic or if your patient’s status or symptoms change, a new referral IndIcatIng ‘completed care pathway’ or ‘new InformatIon’ should be faxed to GI Central Access and TrIage at 403-944-6540. -
Gastroscopy and Gastric Photography with the Olympus GTF-A
Gut, 1970, 11, 176-181 Gut: first published as 10.1136/gut.11.2.176 on 1 February 1970. Downloaded from Gastroscopy and gastric photography with the Olympus GTF-A R. COCKEL AND C. F. HAWKINS From Queen Elizabeth Hospital, Birmingham SUMMARY One hundred patients were examined with the Olympus GTF-A fibregastroscope and gastrocamera. The entire stomach was seen in most cases; the technique permitting this is described in detail. The examination was most rewarding in patients with gastric haemor- rhage and when radiology was equivocal. Advantages and disadvantages of the instrument are discussed. http://gut.bmj.com/ Gastroscopy, once referred to as a 'narrow Description of Instrument peeping speciality' (Rogers, 1937), has been transformed by the development of new in- The Olympus GTF-A is 10.2 mm in diameter in struments (Morrissey, Tanaka, and Thorsen, the flexible part and 12.7 mm in diameter at the 1967). Now, instead of requiring long experience rigid tip of 6.5 cm where the camera is enclosed on September 27, 2021 by guest. Protected copyright. and persistent practice, gastroscopy can be (Figure 2). It is better than the Hirschowitz fibre- performed with little difficulty. A major advance scope because of the clearer view due to the was the application of fibreoptics to gastroscopy quality of fibre bundles, increased magnification, by Hirschowitz and his colleagues (Hirschowitz, and a wider angle of viewing which makes orien- Curtiss, Peters, and Pollard, 1958; Hirschowitz, tation easier. The intragastric camera has a photo- 1961; Hirschowitz, Balint, and Fulton, 1962), for graphing angle of 800, including the whole of the the flexible fibrescope reduces the patient's dis- area seen through the fibre bundle, so avoiding comfort and lessens the risk of oesophageal parallax. -
HYDROGEN & METHANE TESTING for IBS and Sugar Intolerances
phone: 1300 122 388 fax: 1300 122 399 www.breathtestlab.com.au PATIENT NAME: .......................................................................................................................................................................................................................... DOB: ........................................................................................................................................................ ADDRESS: ....................................................................................................................................................................................................................................................................................................................................................................................................................................... ...................................................................................................................................................................................................................................................................................................................................................................................................................................................................................... PHONE: ....................................................................................................................................................................... EMAIL: ................................................................................................................................................................................................................................. -
Dietary, Non-Microbial Intervention to Prevent Helicobacter Pylori- Associated Gastric Diseases
Review Article Page 1 of 8 Dietary, non-microbial intervention to prevent Helicobacter pylori- associated gastric diseases Young-Min Han1*, Jong-Min Park1*, Migyeong Jeong1, Jun-Hwan Yoo2, Won-Hee Kim2, Seok-Pyo Shin2, Weon-Jin Ko2, Ki-Baik Hahm1,2 1CHA University Cancer Prevention Research Center, CHA Bio Complex, Seongnam 463-400, Korea; 2Department of Gastroenterology, CHA University Bundang Medical Center, Seongnam 463-712, Korea *These authors contributed equally to this work. Correspondence to: Ki-Baik Hahm, MD, PhD. CHA University Bundang Medical Center, 59 Yatap-ro, Bundang-gu, Seongnam 463-712 and CHA Bio Complex Cancer Prevention Research Center, 335 Pangyo-ro, Gundang-gu, Seongnam 463-400, Korea. Email: [email protected]. Abstract: Since the discovery of Helicobacter pylori (H. pylori) infection as the major cause of gastroduodenal disorders including acute and chronic gastritis, gastroduodenal ulcer, chronic atrophic gastritis, and gastric cancer almost three decades ago, the possibility of preventing these clinical diseases through eradicating H. pylori has been the focus of active research, but soon debate in the scientific community, though eradication opens the feasibility of cancer prevention and the removal of bacteria significantly prevents development or recurrence of peptic ulcer diseases and some clinical diseases, was proposed due to uncertainty in either achievement of complete eradication or inefficacy in cancer prevention with eradication alone. Still its linkage to gastric cancer is incontestable. Since the multiple combination of bacterial factors, environmental insults, and the host immune response that drives the initiation and progression of mucosal atrophy, metaplasia, and dysplasia toward gastric cancer is intervened, simple eradication deemed the feasibility of cancer prevention. -
Dentistry – Surgery IV Year
Dentistry – Surgery IV year Which sign is not associated with peptic ulcer perforation: A 42-year-old male, with previous Sudden onset ulcer history and typical clinical picture of peptic ulcer perforation, on Cloiberg caps examination, in 4 hours after the Positive Blumberg sign beginning of the disease, discomfort in Free air below diaphragm on plain right upper quadrant, heart rate - abdominal film 74/min., mild abdominal wall muscles rigidity, negative Blumberg sign. Free Intolerable abdominal pain air below diaphragm on X-ray abdominal film. What is you diagnosis? Which ethiological factors causes Peptic ulcer recurrence peptic ulcer disease most oftenly? Acute cholecystitis Abdominal trauma, alimentary factor Chronic cholecystitis H. pylori, NSAID's Covered peptic ulcer perforation H. pylori, hyperlipidemia Acute appendicitis (subhepatic Drugs, toxins location) NSAID's, gastrinoma Most oftenly perforated peptic ulcers are located at: A 30-year-old male is operated on for peptic ulcer perforation, in 2,5 hours Posterior wall of antrum after the beginning of the disease. Fundus of stomach Which operation will be most efficient (radical operation for peptic ulcer)? Cardiac part of stomach Simple closure and highly selective Posterior wall of duodenal bulb vagotomy Anterior wall of duodenal bulb Simple closure Simple closure with a Graham patch Penetrated ulcer can cause such using omentum complications: 1). Abdominal abscess; Stomach resection 2). Portal pyelophlebitis; 3). Stomach- organ fistula; 4). Acute pancreatitis; 5). Antrumectomy Bleeding. 1, 2, 3; It has been abandoned as a method to treat ulcer disease 2, 3, 5; 1, 3, 5; A 42-year-old executive has refractory 3, 4, 5; chronic duodenal ulcer disease. -
The Duodenal Ulcer Is the Chronic Recurrent Disease Which Is Characterized by Ulcer Defect on a Mucosa of the Duodenum
Definition Peptic ulcer of a stomach and duodenum (ICD 10: K25-26)– chronic disease with polycyclic behaviour, which chracterized by secretoric, motoric and trophic modifications this organs and development ulcerous defects of its mucose. Contrary to general belief, more peptic ulcers arise in the duodenum, than in the stomach. About 4% of stomach ulcers are caused by a malignant tumor, so multiple biopsies are needed to make sure, duodenal ulcers are generally benign. Gastric ulcer The gastric ulcer is a chronic disease. The main features of peptic ulcer is the presence of ulcer defect in the mucosa. It forms the basis of pathology of many gastroenterological diseases. Such phenomenon is explained by not only considerable distribution of disease but also the dangerous complications which always accompany gastric ulcers. Etiology and pathogenesis Frequency of morbidity of the peptic ulcer among the adult population is about 4 %. It is more frequently seen patients in age group between 50 - 60 years. Disturbance between the factors of aggression and defense mechanism of mucosa leads to peptic ulcer. Etiology and pathogenesis Aggression factors: hydrochloric acid (HCl), pepsin, reverse diffusion of ions of hydrogen (H+), products of lipid hyperoxidizing. Aggressive factors (hydrochloric acid and pepsin) Parietal cells secrete hydrogen ions in concentration which is three times as high as the concentration of hydrogen ions present in the blood. Each secreted hydrogen ion combines with a chlorine ion. The final step in the secretion of hydrogen ions is performed by means of the proton pump which includes the specific K+, H+-ATPase occuring on the membranes of microvilli. -
Demographic, Chemical, and Helicobacter Pylori Positivity
Hindawi Canadian Journal of Gastroenterology and Hepatology Volume 2021, Article ID 3351352, 8 pages https://doi.org/10.1155/2021/3351352 Research Article Demographic, Chemical, and Helicobacter pylori Positivity Assessment in Different Types of Gallstones and the Bile in a Random Sample of Cholecystectomied Iranian Patients with Cholelithiasis Mohammad Bagher Jahantab ,1 Amir Abbas Safaripour ,1 Sajad Hassanzadeh ,2 and Mohammad Javad Yavari Barhaghtalab 1 1Department of General Surgery, Shahid Beheshti Hospital, Yasuj University of Medical Sciences, Yasuj, Iran 2Department of Internal Medicine, Imam Sajjad Hospital, Yasuj University of Medical Sciences, Yasuj, Iran Correspondence should be addressed to Mohammad Javad Yavari Barhaghtalab; [email protected] Received 6 May 2021; Revised 30 July 2021; Accepted 4 August 2021; Published 10 August 2021 Academic Editor: Masanao Nakamura Copyright © 2021 Mohammad Bagher Jahantab et al. *is is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Background. *e occurrence of stones in the gallbladder or common bile duct and the symptoms and complications they cause is called gallstone disease. *e symptoms of gallstone disease range from mild, nonspecific symptoms to a severe right quadrant abdominal pain. Characteristics of gallstone types in an Iranian population have not been well studied before and there are very limited studies on the demographic pattern of stone types in our country, so this study is one of the first studies on its kind on the epidemiology of gallstone types in Iran. As information on chemical components of the stone will help in the management and prevention of gallstones, in this study, we aimed to do chemical component analysis of gallstones including cholesterol, bilirubin, and calcium. -
The Consensus of Integrative Diagnosis and Treatment of Acute Pancreatitis-2017
Received: 27 October 2018 DOI: 10.1111/jebm.12342 GUIDELINE The consensus of integrative diagnosis and treatment of acute pancreatitis-2017 Junxiang Li Jing Chen Wenfu Tang Digestive Disease Committee, Chinese Association of Integrative Medicine Abstract Correspondence Acute pancreatitis (AP) is one of the most common acute abdominal diseases. The digestive Wenfu Tang,Digestive Disease Committee, disease committee, Chinese Association of Integrative Medicine, released Integrated traditional Chinese Association of Integrative Medicine. Chinese and Western medicine for diagnosis and treatment of acute pancreatitis in 2010.1 Since then, Department of Integrative Medicine, West China Hospital, Sichuan University, Chengdu 610041, further studies and great progress have been made by domestic and foreign counterparts from China. the perspective of both Chinese and Western medicine in AP, including the classification, fluid Email: [email protected] resuscitation, organ function maintenance, surgery intervention, enteral nutrition (EN), and The translators note: The consensus of Inte- syndrome differentiation and treatment. It is necessary to update the consensus on diagnosis and grative diagnosis and treatment of Acute treatment of integrated Chinese and Western medicine to meet clinical needs. Therefore, the Pancreatitis-2017 (Chinese version) has been published on Chinese Journal of Integrated Tra- 2012 Revision of the Atlanta Classification Standard (RAC) by the International AP Consensus,2 ditional and Western Medicine on Digestion, the 2013 -
Patient Medical History Page One
Patient Medical History Page One Date: ______/______/______ Patient Name: ________________________________________________________ DOB: ______/______/______ Age: ____________Height: ________ inches Weight: _______ lbs Race:__________________ Gender: M F Marital Status: Single Married Divorced Widowed Occupation: ________________________ Primary Care Physician: ________________________________Referring Physician: ___________________________________ Sleep Complaint: _________________________________________________________________________________________ _______________________________________________________________________________________________________ Past Medical History Please answer all questions to the best of your ability. Do you now or have you ever had : Tuberculosis (TB) Yes No Lung Disease Yes No Cancer Yes No Thyroid Disease Yes No High Blood Pressure Yes No Stomach Disease Yes No Diabetes (Blood sugar high or low) Yes No Intestinal Disease Yes No Heart Attack Yes No Peptic Ulcer Yes No Other Heart Disease Yes No Liver Disease Yes No Kidney Disease Yes No Seizures Yes No Please explain all “Yes” answers: ________________________________________________________________________________________________________ ________________________________________________________________________________________________________ Habits Do you now or have you ever used: 1. Tobacco ( smoke cigarettes, chew tobacco, etc.) Yes No If yes, indicate amount per day: ___________________ If yes, how long ? _______ years Have you quit? Yes No If yes, when? -
Syphilis of the Stomach and Intestines
kohn: sythilis of the stomach and intestines 685 The aridity immediately after a meal is nil, but steadily increases dunng digestion, and is less in the very young than in the older. Un a barley water diet free hydrochloric acid appears in the stomach in a few minutes, but on a milk diet it does not show itself for an hour or more, due to the fact that the casein absorbs it or combines with it in some way, and the free acid does not appear until the casein has taken up all required for its complete digestion The free acid appears later in disease than in health, due to the in¬ creased amount of food in the stomach and to the slower secretion of the aad; in cases of pylorospasm the acidity is increased. Opinions differ as to the occurrence of lactic and volatile fatty acids, but these probably do not occur in healthy breast-fed infants, while in those ill or on cows’ milk they are fairly common. Part of the acidity is probably due to a fat-splitting enzyme in the infant’s stomach. Pepsin is present at all ages and in all kinds of health, and acts in the infant stomach though to a less degree than in the adult. I he peptic digestion goes on to the stage of peptones, but not beyond that, ihe fact that the stomach contents will not digest fibrin in the thermostat is due to the fact that all the hydrochloric acid is combined with the casein, and while the protein with which it is combined, will be acted upon by the pepsin, a foreign protein with¬ out the addition of more acid mil resist the enzyme Rennin occurs in the stomach after the first few weeks of life- whether during the first week is a moot question.