MEDICAL EQUIPMENT for MULTIPLACE HYPERBARIC CHAMBERS Part II: Ventilators
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Airway Pressures and Volutrauma
Airway Pressures and Volutrauma Airway Pressures and Volutrauma: Is Measuring Tracheal Pressure Worth the Hassle? Monitoring airway pressures during mechanical ventilation is a standard of care.1 Sequential recording of airway pressures not only provides information regarding changes in pulmonary impedance but also allows safety parameters to be set. Safety parameters include high- and low-pressure alarms during positive pressure breaths and disconnect alarms. These standards are, of course, based on our experience with volume control ventilation in adults. During pressure control ventilation, monitoring airway pressures remains important, but volume monitoring and alarms are also required. Airway pressures and work of breathing are also important components of derived variables, including airway resistance, static compliance, dynamic compliance, and intrinsic positive end-expiratory pressure (auto-PEEP), measured at the bedside.2 The requisite pressures for these variables include peak inspiratory pressure, inspiratory plateau pressure, expiratory plateau pressure, and change in airway pressure within a breath. Plateau pressures should be measured at periods of zero flow during both volume control and pressure control ventilation. Change in airway pressure should be measured relative to change in volume delivery to the lung (pressure-volume loop) to elucidate work of breathing. See the related study on Page 1179. Evidence that mechanical ventilation can cause and exacerbate acute lung injury has been steadily mounting.3-5 While most of this evidence has originated from laboratory animal studies, recent clinical reports appear to support this concept.6,7 Traditionally, ventilator-induced lung injury brings to mind the clinical picture of tension pneumothorax. Barotrauma (from the root word baro, which means pressure) is typically associated with excessive airway pressures. -
What Chamber Operators Should Know About Ear Barotrauma (And How to Prevent It) Robert Sheffield, CHT and Kevin “Kip” Posey, CHT / June 2018
LEARN.HYPERBARICMEDICINE.COM International ATMO Education What Chamber Operators Should Know About Ear Barotrauma (and How to Prevent It) Robert Sheffield, CHT and Kevin “Kip” Posey, CHT / June 2018 INTRODUCTION Ear barotrauma (i.e. “ear block”, “ear squeeze”) is the most common complication of hyperbaric treatment. It occurs when the pressure in the hyperbaric chamber is greater than the pressure in the middle ear. It is prevented by patient assessment, patient education, and the appropriate actions of the chamber operator. The chamber operator has an important role in preventing ear barotrauma in hyperbaric patients. OBJECTIVES At the conclusion of this article, the reader will be able to: Describe the anatomy of the middle ear Explain the mechanism of ear barotrauma Describe 3 techniques to equalize middle ear pressure ANATOMY OF THE MIDDLE EAR The middle ear is an air space that separates the external ear canal from the inner ear. The eardrum, called the tympanic membrane (TM), vibrates when sound enters the ear canal. The vibration is transmitted to a series of bones in the middle ear. These bones transmit vibration to another membrane (the oval window) that separates the air‐filled middle ear from the fluid‐filled inner ear, where sound is sensed in the cochlea. The nasopharynx is the area behind the nose and above the palate. The Eustachian tubes open into this area. Because of the location of the Eustachian tube openings, the same things that cause nasal congestion (e.g. allergies, upper respiratory infection) can cause swelling around the opening of the Eustachian tubes, making it more difficult to equalize pressure in the middle ear. -
Critical Care in the Monoplace Hyperbaric Chamber
Critical Care in the Monoplace Hyperbaric Critical Care - Monoplace Chamber • 30 minutes, so only key points • Highly suggest critical care medicine is involved • Pitfalls Lindell K. Weaver, MD Intermountain Medical Center Murray, Utah, and • Ventilator and IV issues LDS Hospital Salt Lake City, Utah Key points Critical Care in the Monoplace Chamber • Weaver LK. Operational Use and Patient Care in the Monoplace Chamber. In: • Staff must be certified and experienced Resp Care Clinics of N Am-Hyperbaric Medicine, Part I. Moon R, McIntyre N, eds. Philadelphia, W.B. Saunders Company, March, 1999: 51-92 in CCM • Weaver LK. The treatment of critically ill patients with hyperbaric oxygen therapy. In: Brent J, Wallace KL, Burkhart KK, Phillips SD, and Donovan JW, • Proximity to CCM services (ed). Critical care toxicology: diagnosis and management of the critically poisoned patient. Philadelphia: Elsevier Mosby; 2005:181-187. • Must have study patient in chamber • Weaver, LK. Critical care of patients needing hyperbaric oxygen. In: Thom SR and Neuman T, (ed). The physiology and medicine of hyperbaric oxygen therapy. quickly Philadelphia: Saunders/Elsevier, 2008:117-129. • Weaver LK. Management of critically ill patients in the monoplace hyperbaric chamber. In: Whelan HT, Kindwall E., Hyperbaric Medicine Practice, 4th ed.. • CCM equipment North Palm Beach, Florida: Best, Inc. 2017; 65-95. • Without certain modifications, treating • Gossett WA, Rockswold GL, Rockswold SB, Adkinson CD, Bergman TA, Quickel RR. The safe treatment, monitoring and management -
Consensus Conference, the ECHM
24 Diving and Hyperbaric Medicine Volume 47 No. 1 March 2017 Consensus Conference Tenth European Consensus Conference on Hyperbaric Medicine: recommendations for accepted and non-accepted clinical indications and practice of hyperbaric oxygen treatment Daniel Mathieu, Alessandro Marroni and Jacek Kot Abstract (Mathieu D, Marroni A, Kot J. Tenth European Consensus Conference on Hyperbaric Medicine: recommendations for accepted and non-accepted clinical indications and practice of hyperbaric oxygen treatment. Diving and Hyperbaric Medicine. 2017 March;47(1):24-32.) The tenth European Consensus Conference on Hyperbaric Medicine took place in April 2016, attended by a large delegation of experts from Europe and elsewhere. The focus of the meeting was the revision of the European Committee on Hyperbaric Medicine (ECHM) list of accepted indications for hyperbaric oxygen treatment (HBOT), based on a thorough review of the best available research and evidence-based medicine (EBM). For this scope, the modified GRADE system for evidence analysis, together with the DELPHI system for consensus evaluation, were adopted. The indications for HBOT, including those promulgated by the ECHM previously, were analysed by selected experts, based on an extensive review of the literature and of the available EBM studies. The indications were divided as follows: Type 1, where HBOT is strongly indicated as a primary treatment method, as it is supported by sufficiently strong evidence; Type 2, where HBOT is suggested as it is supported by acceptable levels of evidence; Type 3, where HBOT can be considered as a possible/optional measure, but it is not yet supported by sufficiently strong evidence. For each type, three levels of evidence were considered: A, when the number of randomised controlled trials (RCTs) is considered sufficient; B, when there are some RCTs in favour of the indication and there is ample expert consensus; C, when the conditions do not allow for proper RCTs but there is ample and international expert consensus. -
Clinical Management of Severe Acute Respiratory Infections When Novel Coronavirus Is Suspected: What to Do and What Not to Do
INTERIM GUIDANCE DOCUMENT Clinical management of severe acute respiratory infections when novel coronavirus is suspected: What to do and what not to do Introduction 2 Section 1. Early recognition and management 3 Section 2. Management of severe respiratory distress, hypoxemia and ARDS 6 Section 3. Management of septic shock 8 Section 4. Prevention of complications 9 References 10 Acknowledgements 12 Introduction The emergence of novel coronavirus in 2012 (see http://www.who.int/csr/disease/coronavirus_infections/en/index. html for the latest updates) has presented challenges for clinical management. Pneumonia has been the most common clinical presentation; five patients developed Acute Respira- tory Distress Syndrome (ARDS). Renal failure, pericarditis and disseminated intravascular coagulation (DIC) have also occurred. Our knowledge of the clinical features of coronavirus infection is limited and no virus-specific preven- tion or treatment (e.g. vaccine or antiviral drugs) is available. Thus, this interim guidance document aims to help clinicians with supportive management of patients who have acute respiratory failure and septic shock as a consequence of severe infection. Because other complications have been seen (renal failure, pericarditis, DIC, as above) clinicians should monitor for the development of these and other complications of severe infection and treat them according to local management guidelines. As all confirmed cases reported to date have occurred in adults, this document focuses on the care of adolescents and adults. Paediatric considerations will be added later. This document will be updated as more information becomes available and after the revised Surviving Sepsis Campaign Guidelines are published later this year (1). This document is for clinicians taking care of critically ill patients with severe acute respiratory infec- tion (SARI). -
Symptomatic Middle Ear and Cranial Sinus Barotraumas As a Complication of Hyperbaric Oxygen Treatment
İst Tıp Fak Derg 2016; 79: 4 KLİNİK ARAŞTIRMA / CLINICAL RESEARCH J Ist Faculty Med 2016; 79: 4 http://dergipark.ulakbim.gov.tr/iuitfd http://www.journals.istanbul.edu.tr/iuitfd SYMPTOMATIC MIDDLE EAR AND CRANIAL SINUS BAROTRAUMAS AS A COMPLICATION OF HYPERBARIC OXYGEN TREATMENT HİPERBARİK OKSİJEN TEDAVİSİ KOMPLİKASYONU: SEMPTOMATİK ORTA KULAK VE KRANYAL SİNUS BAROTRAVMASI Bengüsu MİRASOĞLU*, Aslıcan ÇAKKALKURT*, Maide ÇİMŞİT* ABSTRACT Objective: Hyperbaric oxygen therapy (HBOT) is applied for various diseases. It is generally considered safe but has some benign complications and adverse effects. The most common complication is middle ear barotrauma. The aim of this study was to collect data about middle ear and cranial sinus barotraumas in our department and to evaluate factors affecting the occurrence of barotrauma. Material and methods: Files of patients who had undergone hyperbaric oxygen therapy between June 1st, 2004, and April 30th, 2012, and HBOT log books for the same period were searched for barotraumas. Patients who were intubated and unconscious were excluded. Data about demographics and medical history of conscious patients with barotrauma (BT) were collected and evaluated retrospectively. Results: It was found that over eight years and 23,645 sessions, 39 of a total 896 patients had BT; thus, the general BT incidence of our department was 4.4%. The barotrauma incidence was significantly less in the multiplace chamber (3.1% vs. 8.7%) where a health professional attended the therapies. Most barotraumas were seen during early sessions and were generally mild. A significant accumulation according to treatment indications was not determined. Conclusion: It was thought that the low barotrauma incidence was related to the slow compression rate as well as training patients thoroughly and monitoring them carefully. -
Hyperbaric Oxygen Therapy (HBOT) Final Evidence Report
20, 2012 Health Technology Assessment Hyperbaric Oxygen Therapy (HBOT) for Tissue Damage, Including Wound Care and Treatment of Central Nervous System (CNS) Conditions Final Evidence Report February 15, 2013 Health Technology Assessment Program (HTA) Washington State Health Care Authority PO Box 42712 Olympia, WA 98504-2712 (360) 725-5126 hta.hca.wa.gov [email protected] Hyperbaric Oxygen Therapy (HBOT) for Tissue Damage, Including Wound Care and Treatment of Central Nervous System (CNS) Conditions A Health Technology Assessment Prepared for Washington State Health Care Authority FINAL REPORT – February 15, 2013 Acknowledgement This report was prepared by: Hayes, Inc. 157 S. Broad Street Suite 200 Lansdale, PA 19446 P: 215.855.0615 F: 215.855.5218 This report is intended to provide research assistance and general information only. It is not intended to be used as the sole basis for determining coverage policy or defining treatment protocols or medical modalities, nor should it be construed as providing medical advice regarding treatment of an individual’s specific case. Any decision regarding claims eligibility or benefits, or acquisition or use of a health technology is solely within the discretion of your organization. Hayes, Inc. assumes no responsibility or liability for such decisions. Hayes employees and contractors do not have material, professional, familial, or financial affiliations that create actual or potential conflicts of interest related to the preparation of this report. Prepared by Winifred Hayes, Inc. Page i February -
Respiratory Therapy Pocket Reference
Pulmonary Physiology Volume Control Pressure Control Pressure Support Respiratory Therapy “AC” Assist Control; AC-VC, ~CMV (controlled mandatory Measure of static lung compliance. If in AC-VC, perform a.k.a. a.k.a. AC-PC; Assist Control Pressure Control; ~CMV-PC a.k.a PS (~BiPAP). Spontaneous: Pressure-present inspiratory pause (when there is no flow, there is no effect ventilation = all modes with RR and fixed Ti) PPlateau of Resistance; Pplat@Palv); or set Pause Time ~0.5s; RR, Pinsp, PEEP, FiO2, Flow Trigger, rise time, I:E (set Pocket Reference RR, Vt, PEEP, FiO2, Flow Trigger, Flow pattern, I:E (either Settings Pinsp, PEEP, FiO2, Flow Trigger, Rise time Target: < 30, Optimal: ~ 25 Settings directly or by inspiratory time Ti) Settings directly or via peak flow, Ti settings) Decreasing Ramp (potentially more physiologic) PIP: Total inspiratory work by vent; Reflects resistance & - Decreasing Ramp (potentially more physiologic) Card design by Respiratory care providers from: Square wave/constant vs Decreasing Ramp (potentially Flow Determined by: 1) PS level, 2) R, Rise Time ( rise time ® PPeak inspiratory compliance; Normal ~20 cmH20 (@8cc/kg and adult ETT); - Peak Flow determined by 1) Pinsp level, 2) R, 3)Ti (shorter Flow more physiologic) ¯ peak flow and 3.) pt effort Resp failure 30-40 (low VT use); Concern if >40. Flow = more flow), 4) pressure rise time (¯ Rise Time ® Peak v 0.9 Flow), 5) pt effort ( effort ® peak flow) Pplat-PEEP: tidal stress (lung injury & mortality risk). Target Determined by set RR, Vt, & Flow Pattern (i.e. for any set I:E Determined by patient effort & flow termination (“Esens” – PDriving peak flow, Square (¯ Ti) & Ramp ( Ti); Normal Ti: 1-1.5s; see below “Breath Termination”) < 15 cmH2O. -
Contraindications and Relative Contraindications, and Complications That May Occur with And/Or During HBOT
HYPERBARIC OXYGEN THERAPY INDICATIONS, CONTRAINDICTIONS AND COMPLICATIONS Policy: This policy lists accepted conditions or indications for insurance reimbursement for Hyperbaric oxygen therapy (HBOT), contraindications and relative contraindications, and complications that may occur with and/or during HBOT. Additional information is provided regarding drug therapy with HBOT. 1.0 HYPERBARIC OXYGEN THERAPY (HBOT) ACCEPTED CONDITIONS FOR INSURANCE REIMBURSEMENT: 1.1 The following list includes the currently accepted conditions: A. Air or gas embolism B. Carbon monoxide/cyanide poisoning and smoke inhalation C. Crush injury/traumatic ischemia D. Decompression sickness E. Enhancement in healing in selected problem wounds F. Exceptional anemia resulting for blood loss G. Gas Gangrene(clostridial) H. Necrotizing soft tissue infections I. Refractory osteomyelitis J. Comprised skin grafts/flaps K. Radiation tissue damage L. Thermal burns 2.0 ABSOLUTE CONTRAINDICATIONS 2.1 Untreated pneumothorax A. Surgical relief of the pneumothorax before the HBOT treatment, if possible, removes the obstacle to treatment. 3.0 RELATIVE CONTRAINDICATIONS-- “Conditions in which caution must sometimes be observed but which are not necessarily a contraindication to HBOT.” (Kindwall, 1995) 3.1 History of spontaneous pneumothorax 3.2 Severe sinus infection 3.3 Upper respiratory infection 3.4 Asymptomatic pulmonary lesions on chest x-ray 3.5 Uncontrollable high fever (greater than 39C) 3.6 History of chest or ear surgery 3.7 Congenital spherocytosis 3.8 Any anemia or -
Renal Function in Hyperbaric Environment
APPLIED HUMAN SCIENCE Journal of Physiological Anthropology Renal Function in Hyperbaric Environment Yang Saeng Park1), John R. Claybaugh2), Keizo Shiraki3) and Motohiko Mohri4) 1) Kosin Medical College, Korea 2) Tripler Army Medical Center, USA 3) University of Occupational and Environmental Health 4) Japan Marine Science and Technology Center Abstract. During mixed gas saturation diving (to 3– diuresis. This topic has been reviewed previously by Hong 49.5 ATA) daily urine flow increases by about 500 ml/ (1975), Hong et al. (1983; 1995), Hong and Claybaugh day, with no changes in fluid intake and glomerular (1989), Shiraki (1987), and Sagawa et al. (1996). filtration rate. The diuresis is accompanied by a significant decrease in urine osmolality and increase in excretion Characteristics of Hyperbaric Diuresis of such solutes as urea, K+, Na+, Ca2+ and inorganic phosphate (Pi). The fall in urine osmolality is mainly Fig. 1 depicts time courses of urine flow in subjects due to a reduction of free water reabsorption which is exposed to 31 ATA He-O2 atmosphere determined in associated with a suppression of insensible water loss three saturation dives, Seadragon IV (Nakayama et al., and the attendant inhibition of antidiuretic hormone 1981), Seadragon VI (Shiraki et al., 1987), and New (ADH) system. The increase in urea excretion may be Seatopia (Sagawa et al., 1990), conducted in Japan associated with a reduction of urea reabsorption at the Marine Science and Technology Center (JAMSTEC). The collecting duct as a consequence of ADH suppression. daily urine flow increased rapidly upon compression to a The rise in K+ excretion is due to a facilitated K+ secretion value 700–1000 ml/day above the predive level, then it at the distal tubule as a result of increased aldosterone, dropped off slightly to a steady level of approximately 500 urine flow and excretion of impermeable anions such ml/day above the predive level. -
Subacute Normobaric Oxygen and Hyperbaric Oxygen Therapy in Drowning, Reversal of Brain Volume Loss: a Case Report
[Downloaded free from http://www.medgasres.com on Monday, July 3, 2017, IP: 12.22.86.35] CASE REPORT Subacute normobaric oxygen and hyperbaric oxygen therapy in drowning, reversal of brain volume loss: a case report Paul G. Harch1, *, Edward F. Fogarty2 1 Department of Medicine, Section of Emergency Medicine, University Medical Center, Louisiana State University School of Medicine, New Orleans, LA, USA 2 Department of Radiology, University of North Dakota School of Medicine, Bismarck, ND, USA *Correspondence to: Paul G. Harch, M.D., [email protected] or [email protected]. orcid: 0000-0001-7329-0078 (Paul G. Harch) Abstract A 2-year-old girl experienced cardiac arrest after cold water drowning. Magnetic resonance imaging (MRI) showed deep gray mat- ter injury on day 4 and cerebral atrophy with gray and white matter loss on day 32. Patient had no speech, gait, or responsiveness to commands on day 48 at hospital discharge. She received normobaric 100% oxygen treatment (2 L/minute for 45 minutes by nasal cannula, twice/day) since day 56 and then hyperbaric oxygen treatment (HBOT) at 1.3 atmosphere absolute (131.7 kPa) air/45 minutes, 5 days/week for 40 sessions since day 79; visually apparent and/or physical examination-documented neurological improvement oc- curred upon initiating each therapy. After HBOT, the patient had normal speech and cognition, assisted gait, residual fine motor and temperament deficits. MRI at 5 months after injury and 27 days after HBOT showed near-normalization of ventricles and reversal of atrophy. Subacute normobaric oxygen and HBOT were able to restore drowning-induced cortical gray matter and white matter loss, as documented by sequential MRI, and simultaneous neurological function, as documented by video and physical examinations. -
Hyperbaric Oxygen Therapy Effectively Treats Long-Term Damage from Radiation Therapy
Hyperbaric oxygen therapy effectively treats long-term damage from radiation therapy HBOT is last hope for many patients “For the subset of patients who suffer from late effects of radiation exposure, hyperbaric oxygen therapy is often the only treatment than can prevent irreversible bone or tissue loss or enable them to undergo life-improving reconstructive procedures such as breast or facial surgeries,” explains Susan Sprau, M.D., Medical Director of UCLA Hyperbaric Medicine. “By offering this therapy, we are able to provide a better quality of life to patients who have already survived devastating illnesses.” Late side effects from More than 11 million people living in the U.S. today have been diagnosed with radiotherapy result from scarring cancer, and about half of them have received radiation therapy (radiotherapy). and narrowing of the blood While improved radiotherapy techniques have increased treatment precision and vessels within the treatment area, reduced side effects caused by radiotherapy, the high doses of radiation used to which may lead to inadequate kill cancer cells may still cause long-term damage to nearby healthy cells in some blood supply and cause necrosis of normal tissues and bones. patients. By helping the blood carry more oxygen to affected areas, hyperbaric Hyperbaric oxygen therapy oxygen therapy (HBOT) has been proven effective for these patients. (HBOT) helps blood carry more oxygen to affected areas and Long-term side effects stimulates growth of new blood vessels by exposing patients to For most cancer patients who experience negative effects from radiotherapy, the pure oxygen within a sealed side effects are short-term and appear within six months of their last exposure chamber set at greater than the to radiation.