Oral Diagnosis Whether the Migration Process Is Similar to That Occurring in Individuals Without Cleft Lip and Palate
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Parotid Adenoid Cystic Carcinoma: a Case Report and Review of The
ancer C C as & e y Ilson et al., Oncol Cancer Case Rep 2015,1:1 g R o e l p o o c r t n Oncology and Cancer Case O ISSN: 2471-8556 Reports ResearchCase Report Article OpenOpen Access Access Parotid Adenoid Cystic Carcinoma: A Case Report and Review of the Literature Sepúlveda Ilson1*, Frelinghuysen Michael2, Platín Enrique3, Ortega Pablo4 and Delgado Carolina5 1Maxillofacial-Head and Neck Radiologist, ENT-Head and Neck Surgery Service, General Hospital of Concepcion, Chile 2Physician, Radiation Oncologist, Oncology Service, General Hospital of Concepcion, Chile 3Professor of Oral and Maxillofacial Radiology, University of North Carolina School of Dentistry, Chapel Hill, NC, USA 4Physician, Otolaryngologist, ENT-Head and Neck Surgery Service, General Hospital of Concepcion, Chile 5Physician Pathologist, Pathology Department, General Hospital of Concepción, University of Concepcion School of Medicine, Concepcion, Chile Abstract We report on a patient who presented to the ENT service with swelling of the right side of the parotid gland. The swelling had been present for four years. Imaging studies revealed an expansive process confined to the superficial right parotid lobule. The affected area was well delineated with irregular enhancement post intravenous contrast media administration. Surgical biopsy concluded the presence of Adenoid Cystic Carcinoma. The patient was treated with adjuvant radiation therapy and follow up exams confirm there is no evidence of recurrence. Introduction Adenoid cystic carcinoma (ACC) is malignant epithelial tumors that most commonly occur between the 5th and 6th decades of life. It is a slowly growing but highly invasive cancer with a high recurrence rate. This tumor has the propensity for perineural invasion. -
Maxillary Ameloblastoma: a Review with Clinical, Histological
in vivo 34 : 2249-2258 (2020) doi:10.21873/invivo.12035 Review Maxillary Ameloblastoma: A Review With Clinical, Histological and Prognostic Data of a Rare Tumor ZOI EVANGELOU 1, ATHINA ZARACHI 2, JEAN MARC DUMOLLARD 3, MICHEL PEOC’H 3, IOANNIS KOMNOS 2, IOANNIS KASTANIOUDAKIS 2 and GEORGIA KARPATHIOU 1,3 Departments of 1Pathology and Otorhinolaryngology, and 2Head and Neck Surgery, University Hospital of Ioannina, Ioannina, Greece; 3Department of Pathology, University Hospital of Saint-Etienne, Saint-Etienne, France Abstract. Diagnosis of odontogenic tumors can be neoplasms, diagnosis could be straightforward. In locations challenging due to their rarity and diverse morphology, but outside the oral cavity or when rare histological variants are when arising near the tooth, the diagnosis could be found, suspecting the correct diagnosis can be challenging. suspected. When their location is not typical, like inside the This is especially true for maxillary ameloblastomas, which paranasal sinuses, the diagnosis is less easy. Maxillary are rare, possibly leading to low awareness of this neoplasm ameloblastomas are exceedingly rare with only sparse at this location and often show non-classical morphology, information on their epidemiological, histological and genetic thus, rendering its diagnosis more complicated. characteristics. The aim of this report is to thoroughly review Thus, the aim of this review is to define and thoroughly the available literature in order to present the characteristics describe maxillary ameloblastomas based on the available of this tumor. According to available data, maxillary literature after a short introduction in the entity of ameloblastomas can occur in all ages but later than mandible ameloblastoma. ones, and everywhere within the maxillary region without necessarily having direct contact with the teeth. -
Odontogenic Tumors
4/26/20 CONTEMPORARY MANAGEMENT OF ODONTOGENIC TUMORS RUI FERNANDES, DMD, MD,FACS, FRCS(ED) PROFESSOR UNIVERSITY OF FLORIDA COLLEGE OF MEDICINE- JACKSONVILLE 1 2 Benign th 4 Edition Odontogenic 2017 Tumors Malignant 3 4 BENIGN ODONTOGENIC TUMORS BENIGN ODONTOGENIC TUMORS • EPITHELIAL • MESENCHYMAL • AMELOBLASTOMA • ODONTOGENIC MYXOMA • CALCIFYING EPITHELIAL ODONTOGENIC TUMOR • ODONTOGENIC FIBROMA • PINDBORG TUMOR • PERIPHERAL ODONTOGENIC FIBROMA • ADENOMATOID ODONTOGENIC TUMOR • CEMENTOBLASTOMA • SQUAMOUS ODONTOGENIC TUMOR • ODONTOGENIC GHOST CELL TUMOR 5 6 1 4/26/20 BENIGN ODONTOGENIC TUMORS MALIGNANT ODONTOGENIC TUMORS • PRIMARY INTRAOSSEOUS CARCINOMA • MIXED TUMORS • CARCINOMA ARISING IN ODONTOGENIC CYSTS • AMELOBLASTIC FIBROMA / FIBRO-ODONTOMA • AMELOBLASTIC FIBROSARCOMA • ODONTOMA • AMELOBLASTIC SARCOMA • CLEAR CELL ODONTOGENIC CARCINOMA • ODONTOAMELOBLASTOMA • SCLEROSING ODONTOGENIC CARCINOMA New to the Classification • PRIMORDIAL ODONTOGENIC TUMOR New to the Classification • ODONTOGENIC CARCINOSARCOMA 7 8 0.5 Cases per 100,000/year Ameloblastomas 30%-35% Myxoma AOT 3%-4% Each Ameloblastic fibroma CEOT Ghost Cell Tumor 1% Each 9 10 Courtesy of Professor Ademola Olaitan AMELOBLASTOMA • 1% OF ALL CYSTS AND TUMORS • 30%-60% OF ALL ODONTOGENIC TUMORS • 3RD TO 4TH DECADES OF LIFE • NO GENDER PREDILECTION • MANDIBLE 80% • MAXILLA 20% 11 12 2 4/26/20 AMELOBLASTOMA CLASSIFICATION AMELOBLASTOMA HISTOLOGICAL CRITERIA • SOLID OR MULTI-CYSTIC Conventional 2017 • UNICYSTIC 1. PALISADING NUCLEI 2 • PERIPHERAL 2. REVERSE POLARITY 3. VACUOLIZATION OF THE CYTOPLASM 4. HYPERCHROMATISM OF BASAL CELL LAYER 1 3 4 AmeloblAstomA: DelineAtion of eArly histopathologic feAtures of neoplasiA Robert Vickers, Robert Gorlin, CAncer 26:699-710, 1970 13 14 AMELOBLASTOMA CLASSIFICATION OF 3677 CASES AMELOBLASTOMA SLOW GROWTH – RADIOLOGICAL EVIDENCE Unicystic Peripheral 6% 2% Solid 92% ~3 yeArs After enucleAtion of “dentigerous cyst” P.A . -
Oral Pathology Final Exam Review Table Tuanh Le & Enoch Ng, DDS
Oral Pathology Final Exam Review Table TuAnh Le & Enoch Ng, DDS 2014 Bump under tongue: cementoblastoma (50% 1st molar) Ranula (remove lesion and feeding gland) dermoid cyst (neoplasm from 3 germ layers) (surgical removal) cystic teratoma, cyst of blandin nuhn (surgical removal down to muscle, recurrence likely) Multilocular radiolucency: mucoepidermoid carcinoma cherubism ameloblastoma Bump anterior of palate: KOT minor salivary gland tumor odontogenic myxoma nasopalatine duct cyst (surgical removal, rare recurrence) torus palatinus Mixed radiolucencies: 4 P’s (excise for biopsy; curette vigorously!) calcifying odontogenic (Gorlin) cyst o Pyogenic granuloma (vascular; granulation tissue) periapical cemento-osseous dysplasia (nothing) o Peripheral giant cell granuloma (purple-blue lesions) florid cemento-osseous dysplasia (nothing) o Peripheral ossifying fibroma (bone, cartilage/ ossifying material) focal cemento-osseous dysplasia (biopsy then do nothing) o Peripheral fibroma (fibrous ct) Kertocystic Odontogenic Tumor (KOT): unique histology of cyst lining! (see histo notes below); 3 important things: (1) high Multiple bumps on skin: recurrence rate (2) highly aggressive (3) related to Gorlin syndrome Nevoid basal cell carcinoma (Gorlin syndrome) Hyperparathyroidism: excess PTH found via lab test Neurofibromatosis (see notes below) (refer to derm MD, tell family members) mucoepidermoid carcinoma (mixture of mucus-producing and squamous epidermoid cells; most common minor salivary Nevus gland tumor) (get it out!) -
Head and Neck Pathology Traditional Prognostic Factors
314A ANNUAL MEETING ABSTRACTS Design: 421 archived cases of EC(1995-2007) were reviewed and TMAs prepared Conclusions: Positive GATA3 staining is seen in all vulvar PDs. GATA3 staining is as per established procedures. ERCC1 and RRM1 Immunofl uorescence stains were generally retained in the invasive component associated with vulvar PDs. GATA3 is combined with Automated Quantitative Analysis to assess their expression. The average more sensitive than GCDFP15 for vulvar PDs. Vulvar PDs only rarely express ER and of triplicate core expression was used to determine high and low score cutoff points PR. Vulvar PD should be added to the GATA3+/GCDFP15+ tumor list. using log-rank test on overall survival(OS). Association between expression profi les and clinicopathological parameters was tested using Fisher’s exact test. The independent prognostic value of ERCC1 and RRM1 was tested using Cox model adjusted for Head and Neck Pathology traditional prognostic factors. Results: 304(72%) type-I EC cases and 117(38%) type-II EC cases were identifi ed. Caucasian women had higher proportion of type-I tumors(p<0.001) while elderly women 1297 Subclassification of Perineural Invasion in Oral Squamous Cell were more likely to have type-II tumors (p<0.001). ERCC1 and RRM1 expression was Carcinoma: Prognostic Implications observed in 80% of tumors (336 cases 335 cases,respectively). Kaplan Meier curves K Aivazian, H Low, K Gao, JR Clark, R Gupta. Royal Prince Alfred Hospital, Sydney, showed statistically signifi cant difference in OS between low and high expression of New South Wales, Australia; Royal Prince Alfred Hospital, Sydney, Australia; Sydney ERCC1 and RRM1. -
Tumors of Teeth and Jaws: Pathology of Tumor-Like Conditions of the Dentoalveolar System
TAHER MOHAMMAD MOHAMMADI TUMORS OF TEETH AND JAWS: PATHOLOGY OF TUMOR-LIKE CONDITIONS OF THE DENTOALVEOLAR SYSTEM Minsk BSMU 2018 МИНИСТЕРСТВО ЗДРАВООХРАНЕНИЯ РЕСПУБЛИКИ БЕЛАРУСЬ БЕЛОРУССКИЙ ГОСУДАРСТВЕННЫЙ МЕДИЦИНСКИЙ УНИВЕРСИТЕТ КАФЕДРА ПАТОЛОГИЧЕСКОЙ АНАТОМИИ ТАХЕР МОХАММАД МОХАММАДИ ОПУХОЛИ ЗУБОЧЕЛЮСТНОЙ СИСТЕМЫ: ПАТОЛОГИЧЕСКАЯ АНАТОМИЯ ОПУХОЛЕПОДОБНЫХ ПРОЦЕССОВ ЗУБОЧЕЛЮСТНОЙ СИСТЕМЫ TUMORS OF TEETH AND JAWS: PATHOLOGY OF TUMOR-LIKE CONDITIONS OF THE DENTOALVEOLAR SYSTEM Учебно-методическое пособие Минск БГМУ 2018 2 УДК 616.314-006-091(075.8)-054.6 ББК 616-091я73 М86 Рекомендовано Научно-методическим советом университета в качестве учебно-методического пособия 21.02.2018 г., протокол № 6 Р е ц е н з е н т ы: д-р мед. наук, проф., зав. каф. морфологии человека С. Л. Кабак; канд. мед. наук, доц., зав. 1-й каф. терапевтической стоматологии Л. А. Казеко Мохаммади, Т. М. М86 Опухоли зубочелюстной системы: патологическая анатомия опухолеподобных процессов зубочелюстной системы = Tumors of teeth and jaws: pathology of tumor- like conditions of the dentoalveolar system : учебно-методическое пособие / Т. М. Мохаммади. – Минск : БГМУ, 2018. – 35 с. ISBN 978-985-567-981-4. Описываются морфологические изменения, а также клинико-радиологические проявления опухолеподобных процессов зубочелюстной системы, что необходимо для морфологической постановки диагноза. Предназначено для студентов медицинского факультета иностранных учащихся, обучаю- щихся на английском языке. УДК 616.314-006-091(075.8)-054.6 ББК 616-091я73 ISBN 978-985-567-981-4 © Мохаммади Т. М., 2018 © УО «Белорусский государственный медицинский университет», 2018 3 INTRODUCTION Tumor-like processes (benign processes) in the jaws represent a wide spectrum of conditions, many of which have cystic form. Tumor-like processes can be diagnosed with intraoral and panoramic radiography. -
๑). ความรู้วิทยาศาสตร์การแพทย์พื้นฐานประยุกต์ (Correlated Basic Medical Science) ทางโสต ศอ นาสิกวิทยา และระบบที่เกี่ยวข้อง
ภาคผนวก ๑ เนือ้ หาของการฝึ กอบรม/หลกั สูตร ๑). ความรู้วิทยาศาสตร์การแพทย์พื้นฐานประยุกต์ (Correlated basic medical science) ทางโสต ศอ นาสิกวิทยา และระบบที่เกี่ยวข้อง ๑. Anatomy and physiology of hearing ๒. Basic audiology ๓. Anatomy and physiology of vestibular system ๔. Medication in ear disease ๕. Anatomy and physiology of the nose and paranasal sinus ๖. Basic immunology and immunotherapy ๗. Antihistamine, intranasal steroid and related drug ๘. Snoring and sleep disorder: basic ๙. Anatomy of the neck ๑๐. Voice: anatomy, physiology and test ๑๑. Wound healing and physiology of flap ๑๒. Radiotherapy in head and neck cancer ๑๓. Chemotherapy In head and neck cancer ๑๔. Anesthesia and pain management ๑๕. Basic radiologic imaging ๑๖. Antibiotic: pharmacology and application ๑๗. Common contagious disease in clinical practice ๑๘. Nutrition: evaluation and management ๑๙. Laser; basic principle and application ๒๐. Medical law and ethic in clinical practice ๒). โรคหรือภาวะของผู้ป่วย แบ่งเป็น แพทย์ประจ ำบ้ำนต้องสำมำรถให้กำรวินิจฉัย ดูแลรักษำ และฟื้นฟู หรือให้ค ำแนะน ำเพื่อส่งต่อ ได้ ในโรคทำงหูคอจมูกฯ ต่อไปนี้ ระดับที่ ๑ โรคหรือภำวะที่พบบ่อย ซึ่งแพทย์ประจ ำบ้ำนสำมำรถเรียนรู้ได้จำกผู้ป่วยโดยตรง Symptom and sing Epistaxis (R040) Cough (R05) Stridor (R061) Mouth breathing (R065) Sneezing (R067) Snoring (R0683) Pain in throat (R070) Asphyxia (R0901) Hypoxemia (R09.2) Nasal congestion (R0981) Postnasal drip (R0982) Dysphagia (R13) Halitosis (R196) Neck mass (R221) Facial weakness (R29810) speech and voice (R47-R49) localized enlarged lymph nodes (R590) -
DIAGNOSIS ICD-9 ICD-10 a Abscess
2701 North Decatur Road Decatur, GA 30033 ● P: (404) 501-7445 ● F: (404) 501-7460 www.atlantaoralpathology.com Steve Budnick, DDS Susan Muller, DMD, MS DIAGNOSIS ICD-9 ICD-10 A Abscess -oral 528.5 K12.2 -jaw 526.4 M27.2 -skin, neck 682.1 L02.01 Acute and/or chronic inflammation -jaw 526.4 M27.2 -oral soft tissue 528.00 K12.2 Amalgam tattoo 709.09 L81.8 Ameloblastoma-mandible 213.1 D16.5 -maxilla/skull 213.0 D16.4 B Benign neoplasm of lip D10.0 Benign neoplasm of tongue D10.1 Bengin neoplasm of parotid D11.0 Benign neoplasm of other major salivary gland D11.7 Benign neoplasm of major salivary gland, unspecified D11.9 Benign neoplasm floor of mouth D10.2 Benign neoplasm, unspecified mouth D10.30 Benign neoplasm, other parts of mouth D10.39 Benign neoplasm of tonsil D10.4 Benign neoplasm-middle ear and nasal sinus D14.0 Benign fibro-osseous lesion -mandible 213.1 D16.5 -maxilla 213.0 D16.4 C Caliber persistent artery 747.6 I77.9 Candidiasis B37.9 Central giant cell granuloma M27.1 Chronic osteomyelitis 526.4 M27.2 Chronic sialadenitis 527.2 K11.23 Cyst of undetermined origin 526.2 M27.40 Cysts, bone{aneurysmal,hemorrhagic) 526.2 M27.49 D Dental follicle (enlarged) 526.9 M27.0 Dentigerous cyst 526.0 K09.0 Dental granuloma 526.4 M27.2 Developmental cyst NOS 526.1 K09.0 Dysplasia – mid 528.79 K13.29 -moderate 528.79 K13.29 -severe 528.79 K13.29 E Epidermoid cyst – mouth 528.4 K09.9 - skin 706.2 L72.0 Epulis fissuratum 528.9 K13.70 Eruption cyst 526.0 K09.0 Erythema migrans 529.1 K14.1 Erythema multiforme 695.1 L51.8 Exostosis 526.81 M27.8 F -
Practical Insights in Oral Pathology
PRACTICAL INSIGHTS IN ORAL PATHOLOGY Kirk Y. Hirata, MD January 13, 2017 ROAD TO THE PODIUM? • 1985-90: LLUSM • 1990-94: Anatomic and Clinical Pathology Residency, UH John A. Burns School of Medicine • 1994-95: Hematopathology Fellowship, Scripps Clinic, San Diego • July 1995: HPL - new business, niche? ORAL PATHOLOGY • outpatient biopsies, some were from dentists • s/o inflammation, “benign odontogenic cyst”, etc • no service to general dentists or oral surgeons • wife was a dentist, residency at QMC 1990-91 • idea? ORAL PATHOLOGY • telephone calls • lunches (marketing) • textbooks • courses, including microscopy • began to acquire cases • QMC dental resident teaching once a month AFTER 21 YEARS • established myself in the community as an “oral pathologist” • QMC Dental Residency Program has been recognized • 7TH edition of Jordan (1999) • UCSF consultation service I feel fortunate to have joined this group of outstanding dermato- pathologists. I believe that my training, experience and expertise in oral and maxillofacial pathology expands the scope and breadth of services that we are able to offer the medical and dental community for their diagnostic pathology needs. I initially trained as a dentist at the University of Toronto that was followed by an internship at the Toronto Western Hospital (now the University Health Network). Following training in anatomic pathology I completed a residency in oral and maxillofacial pathology under the direction of Dr. Jim Main. I also completed a fellowship in oral medicine and then a Master of Science degree in oral pathology. I was fortunate to be able to train with Professor Paul Speight at the University of London were I was awarded a PhD degree in Experimental Pathology. -
Oral Medicine and Radiology
4A.4.2 SYLLABUS ( Including Teaching Hours.) MUST KNOW 1.Oral medicine and diagnostic AIDS: Section A-Diagnostic Methods 06 HRS (1) Definition and importance of Diagnosis and various types of diagnosis (2) Method of clinical examinations. (a) General Physical examination by inspection. (b) Oro-facial region by inspection, palpation and other means (c) To train the students about the importance, role, use of saliva and techniques of diagnosis of saliva as part of oral disease (d) Examination of lesions like swellings, ulcers, erosions, sinus, fistula, growths, pigmented lesions, white and red patches (e) Examination of lymph nodes (3) Investigations (a) Biopsy and exfoliative cytology (b) Hematological, Microbiological and other tests and investigations necessary for diagnosis and prognosis Section B- Diagnosis, Differential Diagnosis 04 HRS (1) Teeth: Developmental abnormalities, causes of destruction of teeth and their sequelae and discoloration of teeth (2) Inflamation – Injury, infection and sperad of infection,fascial space infections, osteoradionecrosis. (3) Temparomandibular joint: Developmental abnormalities of the condyle. Rheumatoid arthritis, Osteoarthritis, Subluxation and luxation. (4) Periodontal diseases: Gingival hyperplasia, gingivitis, periodontitis, pyogenic granuloma (5) Common cysts and Tumors: CYSTS: Cysts of soft tissue: Mucocele and Ranula 07 HRS Cysts of bone: Odontogenic and nonodontogenic. TUMORS: Soft Tissue: Epithelial: Papilloma, Carcinoma, Melanoma Connective tissue: Fibroma, Lipoma, Fibrosarcoma Vascular: Haemangioma, Lymphangioma Nerve Tissue: Neurofibroma, Traumatic Neuroma, Neurofibromatosis Salivary Glands: Pleomorphic adenoma, Adenocarcinoma, Warthin’s Tumor, Adenoid cystic carcinoma. (6) Teeth: Developmental abnormalities, causes of destruction of teeth and their sequelae and discoloration of teeth (7) Inflamation – Injury, infection and sperad of infection,fascial space infections, osteoradionecrosis. (8) Temparomandibular joint: Developmental abnormalities of the condyle. -
Odontogenic Cysts, Odontogenic Tumors, Fibroosseous, and Giant Cell Lesions of the Jaws Joseph A
Odontogenic Cysts, Odontogenic Tumors, Fibroosseous, and Giant Cell Lesions of the Jaws Joseph A. Regezi, D.D.S., M.S. Oral Pathology and Pathology, Department of Stomatology, University of California, San Francisco, San Francisco, California ologic correlation in assessing these lesions is of Odontogenic cysts that can be problematic because particular importance. Central giant cell granuloma of recurrence and/or aggressive growth include is a relatively common jaw lesion of young adults odontogenic keratocyst (OKC), calcifying odonto- that has an unpredictable behavior. Microscopic di- genic cyst, and the recently described glandular agnosis is relatively straightforward; however, this odontogenic cyst. The OKC has significant growth lesion continues to be somewhat controversial be- capacity and recurrence potential and is occasion- cause of its disputed classification (reactive versus ally indicative of the nevoid basal cell carcinoma neoplastic) and because of its management (surgical syndrome. There is also an orthokeratinized vari- versus. medical). Its relationship to giant cell tumor of ant, the orthokeratinized odontogenic cyst, which is long bone remains undetermined. less aggressive and is not syndrome associated. Ghost cell keratinization, which typifies the calcify- KEY WORDS: Ameloblastoma, CEOT, Fibrous dys- ing odontogenic cyst, can be seen in solid lesions plasia, Giant cell granuloma, Odontogenic kerato- that have now been designated odontogenic ghost cyst, Odontogenic myxoma, Odontogenic tumors. cell tumor. The glandular odontogenic cyst contains Mod Pathol 2002;15(3):331–341 mucous cells and ductlike structures that may mimic central mucoepidermoid carcinoma. Several The jaws are host to a wide variety of cysts and odontogenic tumors may provide diagnostic chal- neoplasms, due in large part to the tissues involved lenges, particularly the cystic ameloblastoma. -
Lesions Associated with Impacted Tooth
Review Article Lesions Associated with Impacted Tooth Dr Radha Baral,1 Dr Bidhata Ojha,2 Dr Dipshikha Bajracharya3 1,2Lecturer, 3Associate Professor, ABSTRACT Department of Oral Pathology, Impacted teeth have been associated with various pathological conditions such as cysts, Kantipur Dental College, Kathmandu, tumors, pericoronitis, periodontitis, and pathological root resorption. This review is intended to Nepal introduce lesions associated with the impacted tooth. From the literature review following 12 lesions was found to be associated with impacted tooth which are dentigerous cyst, odontogenic Corresponding Author keratocyst, calcifying odontogenic cyst, central mucoepidermoid carcinoma, unicystic Dr Radha Baral ameloblastoma, calcifying epithelial odontogenic tumor, adenomatoid odontogenic tumor, Email: [email protected] squamous odontogenic tumor, ameloblastic fibroma, ameloblastic fibro odontoma, odontoma, central odontogenic fibroma. These entities can be considered in differential diagnosis when Citation clinicians encounter a lesion in intimate association with impacted tooth thus helping in Baral R, Ojha B, Bajracharya D. Lesions formulation of diagnosis and to develop an appropriate treatment plan. Associated with Impacted Tooth. J Kantipur Dent Coll. 2020;1(1):25-31. Keywords: associated lesions, impacted tooth, unerupted tooth INTRODUCTION papers broadly suitable to the topic were found. We ultimately integrated 33 articles that were closely related Peterson characterized impacted teeth as those teeth that to the