AUSTRALIAN OFFICIAL JOURNAL of TRADE MARKS 12 October 2006
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43E Festival International Du Film De La Rochelle Du 26 Juin Au 5 Juillet 2015 Le Puzzle Des Cinémas Du Monde
43e Festival International du Film de La Rochelle du 26 juin au 5 juillet 2015 LE PUZZLE DES CINÉMAS DU MONDE Une fois de plus nous revient l’impossible tâche de synthétiser une édition multiforme, tant par le nombre de films présentés que par les contextes dans lesquels ils ont été conçus. Nous ne pouvons nous résoudre à en sélectionner beaucoup moins, ce n’est pas faute d’essayer, et de toutes manières, un contexte économique plutôt inquiétant nous y contraint ; mais qu’une ou plusieurs pièces essentielles viennent à manquer au puzzle mental dont nous tentons, à l’année, de joindre les pièces irrégulières, et le Festival nous paraîtrait bancal. Finalement, ce qui rassemble tous ces films, qu’ils soient encore matériels ou virtuels (50/50), c’est nous, sélectionneuses au long cours. Nous souhaitons proposer aux spectateurs un panorama généreux de la chose filmique, cohérent, harmonieux, digne, sincère, quoique, la sincérité… Ambitieux aussi car nous aimons plus que tout les cinéastes qui prennent des risques et notre devise secrète pourrait bien être : mieux vaut un bon film raté qu’un mauvais film réussi. Et enfin, il nous plaît que les films se parlent, se rencontrent, s’éclairent les uns les autres et entrent en résonance dans l’esprit du festivalier. En 2015, nous avons procédé à un rééquilibrage géographique vers l’Asie, absente depuis plusieurs éditions de la programmation. Tout d’abord, avec le grand Hou Hsiao-hsien qui en est un digne représentant puisqu’il a tourné non seulement à Taïwan, son île natale mais aussi au Japon, à Hongkong et en Chine. -
Introduction to Nutrigenetics, Nutrigenomics and Epigenetics
Medicine, Nursing and Health Sciences Introduction to Nutrigenetics, Nutrigenomics and Epigenetics Australia n China n India n Italy n Malaysia n South Africa Introduction to Nutrigenetics, Nutrigenomics and Epigenetics The Nutrition Society of Australia (Melbourne), Monash University and MyGene are pleased to invite you to our upcoming ‘Introduction to Nutrigenetics, Nutrigenomics and Epigenetics’ symposium. Please note, numbers will be capped at 100, and delegates need to sign up for their preferred workshop sessions using the link below. Where: Level 5 of The Alfred Centre, Monash University (Go to the 1.00pm Networking coffee/tea Foyer of Lecture Theatre ‘B’ set of lifts in the The Alfred Centre), 99 Commercial Road, 1.30pm Introduction to Nutrigenetics, Michael Fenech (CSIRO) Melbourne. Nutrigenomics and Epigenetics Jeff Craig (MCRI) When: 1-5.15pm, 2.30pm Global trends in genetics: research, Graeme Smith (MyGene) Tuesday 27 August 2013 technology and practice Melissa Adamski (MyGene) RSVP: Brianna McFarlane, [email protected] 3.10pm Tea/coffee break Foyer of Lecture Theatre (by 13 August 2013) 3.30pm Workshop 1 Level 5 breakout room The workshops are a choice of two 4.15pm Changeover time from following topics (click link below to sign up): 4.30pm Workshop 2 Level 5 breakout room Workshop preference: 5.15pm Close http://www.mygene.com.au/nsa- symposium/ 1. Designing genetic research projects (including considering the data, Biographies In 2003-05, Dr Fenech proposed a novel interpreting the data and validation) disease prevention strategy based on Michael Fenech 2. Adding genetic testing to your personalised diagnosis and prevention of current research project Professor Michael Fenech is recognised DNA damage by appropriate diet/life-style internationally for his research in nutritional intervention, which has led to the Genome 3. -
6615 Sofiva Genomics
6615 SOFIVA GENOMICS Chairman Yi-Ning Su 2017.01.04 Company Profile SOFIVA GENOMICS Founded in July, 2012, SOFIVA GENOMICS focuses on clinical needs to develop next-generation testing models in genomic medicine Maternal-Fetal Medicine Genomic Medicine Clinical Medicine We have Asia’s leading genetic testing laboratory to provide comprehensive services to those in need Our Orientation ■ Mothers and fetuses Targets ■ Individuals with genetic diseases ■ Individuals with cancer ■ Healthy individuals ■ Mothers and fetuses: Helping mothers successfully conceive and safely give birth to healthy babies ■ Individuals with genetic diseases: Helping doctors in giving patients swift and accurate diagnoses and treatments Service Orientation ■ Individuals with cancer: Facilitating the detection, prognosis, and tracking of cancer so that doctors can treat and monitor their patients more accurately ■ Healthy individuals: Grasping their genetic health and risk of contracting disease and early planning for health promotion ■ Now: Technology and platform combining biochemical detection + gene chip Technical + next-generation sequencing to provide customers with comprehensive Development testing services ■Future: Development of comprehensive and customized testing using a single sample and a single platform ■ Business promotion: Joint promotion and hospitals, introduction to medical personnel, onsite recommendations, promotion at foreign Marketing and domestic conferences/expos Channels ■ PR promotion: online/Facebook marketing, literature and posters to -
DVD Profiler
101 Dalmatians II: Patch's London Adventure Animation Family Comedy2003 74 minG Coll.# 1 C Barry Bostwick, Jason Alexander, The endearing tale of Disney's animated classic '101 Dalmatians' continues in the delightful, all-new movie, '101 Dalmatians II: Patch's London A Martin Short, Bobby Lockwood, Adventure'. It's a fun-filled adventure fresh with irresistible original music and loveable new characters, voiced by Jason Alexander, Martin Short and S Susan Blakeslee, Samuel West, Barry Bostwick. Maurice LaMarche, Jeff Bennett, T D.Jim Kammerud P. Carolyn Bates C. W. Garrett K. SchiffM. Geoff Foster 102 Dalmatians Family 2000 100 min G Coll.# 2 C Eric Idle, Glenn Close, Gerard Get ready for outrageous fun in Disney's '102 Dalmatians'. It's a brand-new, hilarious adventure, starring the audacious Oddball, the spotless A Depardieu, Ioan Gruffudd, Alice Dalmatian puppy on a search for her rightful spots, and Waddlesworth, the wisecracking, delusional macaw who thinks he's a Rottweiler. Barking S Evans, Tim McInnerny, Ben mad, this unlikely duo leads a posse of puppies on a mission to outfox the wildly wicked, ever-scheming Cruella De Vil. Filled with chases, close Crompton, Carol MacReady, Ian calls, hilarious antics and thrilling escapes all the way from London through the streets of Paris - and a Parisian bakery - this adventure-packed tale T D.Kevin Lima P. Edward S. Feldman C. Adrian BiddleW. Dodie SmithM. David Newman 16 Blocks: Widescreen Edition Action Suspense/Thriller Drama 2005 102 min PG-13 Coll.# 390 C Bruce Willis, Mos Def, David From 'Lethal Weapon' director Richard Donner comes "a hard-to-beat thriller" (Gene Shalit, 'Today'/NBC-TV). -
A Murine Atlas of Age-Related Changes in Intercellular Communication Inferred with the Package Scdiffcom
bioRxiv preprint doi: https://doi.org/10.1101/2021.08.13.456238; this version posted August 15, 2021. The copyright holder for this preprint (which was not certified by peer review) is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under aCC-BY 4.0 International license. scAgeCom: a murine atlas of age-related changes in intercellular communication inferred with the package scDiffCom Cyril Lagger 1,*, Eugen Ursu 2,*, Anaïs Equey 3, Roberto A. Avelar 1, Angela O. Pisco 4, Robi Tacutu 2, João Pedro de Magalhães 1,# 1. Integrative Genomics of Ageing Group, Institute of Life Course and Medical Sciences, University of Liverpool, Liverpool, UK 2. Systems Biology of Aging Group, Institute of Biochemistry of the Romanian Academy, Bucharest, Romania 3. Department of Medicine, Karolinska Institutet, Stockholm, Sweden 4. Chan Zuckerberg Biohub, San Francisco, California, USA *These authors contributed equally #Corresponding author’s email: [email protected] Abstract Dysregulation of intercellular communication is a well-established hallmark of aging. To better understand how this process contributes to the aging phenotype, we built scAgeCom, a comprehensive atlas presenting how cell-type to cell-type interactions vary with age in 23 mouse tissues. We first created an R package, scDiffCom, designed to perform differential intercellular communication analysis between two conditions of interest in any mouse or human single-cell RNA-seq dataset. The package relies on its own list of curated ligand-receptor interactions compiled from seven established studies. We applied this tool to single-cell transcriptomics data from the Tabula Muris Senis consortium and the Calico murine aging cell atlas. -
Circulating Exosomes Potentiate Tumor Malignant Properties in a Mouse Model of Chronic Sleep Fragmentation
www.impactjournals.com/oncotarget/ Oncotarget, Vol. 7, No. 34 Research Paper Circulating exosomes potentiate tumor malignant properties in a mouse model of chronic sleep fragmentation Abdelnaby Khalyfa1, Isaac Almendros1,4, Alex Gileles-Hillel1, Mahzad Akbarpour1, Wojciech Trzepizur1, Babak Mokhlesi2, Lei Huang3, Jorge Andrade3, Ramon Farré4, David Gozal1 1Section of Pediatric Sleep Medicine, Department of Pediatrics, Pritzker School of Medicine, Biological Sciences Division, The University of Chicago, Chicago, IL, USA 2Department of Medicine, Section of Pulmonary and Critical Care, Sleep Disorders Center, The University of Chicago, Chicago, IL, USA 3Center for Research Informatics, The University of Chicago, Chicago, IL, USA 4Unitat de Biofísica i Bioenginyeria, Facultat de Medicina, Universitat de Barcelona-Institut Investigacions Biomediques August Pi Sunyer-CIBER Enfermedades Respiratorias, Barcelona, Spain Correspondence to: David Gozal, email: [email protected] Keywords: sleep fragmentation, obstructive sleep apnea, tumors, microenvironment, cancer biology Received: April 16, 2016 Accepted: June 30, 2016 Published: July 13, 2016 ABSTRACT Background: Chronic sleep fragmentation (SF) increases cancer aggressiveness in mice. Exosomes exhibit pleiotropic biological functions, including immune regulatory functions, antigen presentation, intracellular communication and inter- cellular transfer of RNA and proteins. We hypothesized that SF-induced alterations in biosynthesis and cargo of plasma exosomes may affect tumor cell properties. Results: SF-derived exosomes increased tumor cell proliferation (~13%), migration (~2.3-fold) and extravasation (~10%) when compared to exosomes from SC-exposed mice. Similarly, Pre exosomes from OSA patients significantly enhanced proliferation and migration of human adenocarcinoma cells compared to Post. SF- exosomal cargo revealed 3 discrete differentially expressed miRNAs, and exploration of potential mRNA targets in TC1 tumor cells uncovered 132 differentially expressed genes that encode for multiple cancer-related pathways. -
Tricks of the Light
Tricks of the Light: A Study of the Cinematographic Style of the Émigré Cinematographer Eugen Schüfftan Submitted by Tomas Rhys Williams to the University of Exeter as a thesis for the degree of Doctor of Philosophy in Film In October 2011 This thesis is available for Library use on the understanding that it is copyright material and that no quotation from the thesis may be published without proper acknowledgement. I certify that all material in this thesis which is not my own work has been identified and that no material has previously been submitted and approved for the award of a degree by this or any other University. Signature: ………………………………………………………….. 1 Abstract The aim of this thesis is to explore the overlooked technical role of cinematography, by discussing its artistic effects. I intend to examine the career of a single cinematographer, in order to demonstrate whether a dinstinctive cinematographic style may be defined. The task of this thesis is therefore to define that cinematographer’s style and trace its development across the course of a career. The subject that I shall employ in order to achieve this is the émigré cinematographer Eugen Schüfftan, who is perhaps most famous for his invention ‘The Schüfftan Process’ in the 1920s, but who subsequently had a 40 year career acting as a cinematographer. During this time Schüfftan worked throughout Europe and America, shooting films that included Menschen am Sonntag (Robert Siodmak et al, 1929), Le Quai des brumes (Marcel Carné, 1938), Hitler’s Madman (Douglas Sirk, 1942), Les Yeux sans visage (Georges Franju, 1959) and The Hustler (Robert Rossen, 1961). -
Lifelong Restriction of Dietary Branched-Chain Amino Acids Has Sex-Specific Benefits for Frailty and Life Span in Mice
ARTICLES https://doi.org/10.1038/s43587-020-00006-2 Lifelong restriction of dietary branched-chain amino acids has sex-specific benefits for frailty and life span in mice Nicole E. Richardson 1,2,3, Elizabeth N. Konon1,2, Haley S. Schuster1,2, Alexis T. Mitchell1,2, Colin Boyle1,2, Allison C. Rodgers1, Megan Finke1,2, Lexington R. Haider1,2, Deyang Yu1,2, Victoria Flores1,2,4, Heidi H. Pak1,2,4, Soha Ahmad1,2, Sareyah Ahmed1,2, Abigail Radcliff1,2, Jessica Wu1,2, Elizabeth M. Williams1,2, Lovina Abdi1,2, Dawn S. Sherman1,2, Timothy A. Hacker1 and Dudley W. Lamming 1,2,3,4,5 ✉ Protein-restricted diets promote health and longevity in many species. While the precise components of a protein-restricted diet that mediate the beneficial effects to longevity have not been defined, we recently showed that many metabolic effects of protein restriction can be attributed to reduced dietary levels of the branched-chain amino acids (BCAAs) leucine, isoleucine and valine. Here, we demonstrate that restricting dietary BCAAs increases the survival of two different progeroid mouse mod- els, delays frailty and promotes the metabolic health of wild-type C57BL/6J mice when started in midlife, and leads to a 30% increase in life span and a reduction in frailty in male, but not female, wild-type mice when they undergo lifelong feeding. Our results demonstrate that restricting dietary BCAAs can increase health span and longevity in mice and suggest that reducing dietary BCAAs may hold potential as a translatable intervention to promote healthy aging. ietary interventions can robustly promote organismal both Drosophila and rodents5,19–22. -
Nmscutsgo? Lit Was a Larger Problem Than Just New York Ity; It Is Encountered by Most Older Merican Cities," She Explained
'Leap Into Faith \..Page 3 US PoslagePAID Bronx. New York Permit No. 7608 Non-profit Org. Thursday, October 1,1981 Volume 63 IRPHAM UNIVERSITY, NEW YORK Number 21 Bellamy On Mayor And Big Apple by Maryellen Gordon New York is number one, in both the od things and in screwing things up," narked Carol Bellamy, City Council esident Tuesday, in an appearance1 spon- ied by the Young Democrats. Approximately 8 5 people attended the entheld in Keating 1st, where Bellamy also ited that because of the financial im- jvements the City has undergone, she rates pKoch's politics a B-, 'Ed Koch can validly run on the financial nation," she confirmed, "however that lyincludes this primary, not the one three arsfrom now." Bellamy further added that energy and enthusiasm are Koch's other isilive points. On the negative side, she ited racial problems and education. "One n't'pander' to groups. It is unnecessary to len one's mouth all the time," she ex- lined. While Koch rates himself an 'A' for his ndling of the city's education system, llamy gives him a 'C, citing the high rate truancy and the high number of school ings. "We still have enormous problems re." 11975, the City was virtually bankrupt e to a number of factors, Bellamy stated. nmsCutsGo? lit was a larger problem than just New York ity; it is encountered by most older merican cities," she explained. She cited , by Mark Dillon \" ' ;• '<\Jr irious problems with the city's infrastruc- . Sweeping changes in,fe,derai student aid programs were The changes will affect.the amount of money available foi\ and the outflux of industries to signed into law August 13 by President Reagaitas part of a student aid and the ability of students to obtain fund&'fprv Mhern states that built-up to the '75 fiscal series of budget cms designed tp'help the nation1^ economy, higher education. -
Dissecting the Genetic Etiology of Lupus at ETS1 Locus
Dissecting the Genetic Etiology of Lupus at ETS1 Locus A dissertation submitted to the Graduate School of the University of Cincinnati in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Immunobiology of the College of Medicine 2017 by Xiaoming Lu B.S. Sun Yat-sen University, P.R. China June 2011 Dissertation Committee: John B. Harley, MD, PhD Harinder Singh, PhD Leah C. Kottyan, PhD Matthew T. Weirauch, PhD Kasper Hoebe, PhD Lili Ding, PhD i Abstract Systemic lupus erythematosus (SLE) is a complex autoimmune disease with strong evidence for genetics factor involvement. Genome-wide association studies have identified 84 risk loci associated with SLE. However, the specific genotype-dependent (allelic) molecular mechanisms connecting these lupus-genetic risk loci to immunological dysregulation are mostly still unidentified. ~ 90% of these loci contain variants that are non-coding, and are thus likely to act by impacting subtle, comparatively hard to predict mechanisms controlling gene expression. Here, we developed a strategic approach to prioritize non-coding variants, and screen them for their function. This approach involves computational prioritization using functional genomic databases followed by experimental analysis of differential binding of transcription factors (TFs) to risk and non-risk alleles. For both electrophoretic mobility shift assay (EMSA) and DNA affinity precipitation assay (DAPA) analysis of genetic variants, a synthetic DNA oligonucleotide (oligo) is used to identify factors in the nuclear lysate of disease or phenotype-relevant cells. This strategic approach was then used for investigating SLE association at ETS1 locus. Genetic variants at chromosomal region 11q23.3, near the gene ETS1, have been associated with systemic lupus erythematosus (SLE), or lupus, in independent cohorts of Asian ancestry. -
Critical Physiological and Pathological Functions of Forkhead Box O Tumor Suppressors
DISCOVERIES (2013, Oct-Dec), 1(1): e5 DOI: 10.15190/d.2013.5 FOXO proteins control critical cellular decisions and cellular fate REVIEW Article Critical physiological and pathological functions of Forkhead Box O tumor suppressors Georgiana R. Dumitrascu1 and Octavian Bucur2,* 1“Victor Babes” National Institute of Pathology and Biomedical Sciences, Bucharest, Romania; 2Department of Pathology, Harvard Medical School and Beth Israel Deaconess Medical Center, Boston, MA, USA; Correspondence to: Dr. Octavian Bucur, Department of Pathology, Harvard Medical School and Beth Israel Deaconess Medical Center, 330 Brookline Ave, Boston, MA, 02215, USA. Phone: +1 617 667 4314; E-mail: [email protected]. Citation: Dumitrascu GR, Bucur O. Critical physiological and pathological functions of Forkhead Box O tumor suppressors. Discoveries 2013, Oct-Dec; 1(1): e5. DOI: 10.15190/d.2013.5 ABSTRACT death (Puma); Bcl-2/adenovirus E1B 19 kDa-interacting The Forkhead box, subclass O (FOXO) proteins protein (bNIP3); PTEN – induced kinase 1 (Pink1); are critical transcription factors, ubiquitously peroxisome proliferative activated receptor-gama expressed in the human body. These proteins are (PGC1); glucose-6-phosphatase (G6Pase); phosphor- characterized by a remarkable functional diversity, enol-pyruvate carboxykinase (PEPCK); Agouti-related protein (Agrp); neuropeptide Y (NpY); pro-opiomelan- being involved in cell cycle arrest, apoptosis, ocortin (POMC); glycogenolytic gene glucose-6- oxidative detoxification, DNA damage repair, stem phosphatase (G6pc); manganese superoxide dismutase cell maintenance, cell differentiation, cell (MnSOD); CBP/p300-interacting trans-activator with metabolism, angiogenesis, cardiac development, ED-rich tail 2 (Cited 2); mixed lineage leukemia aging and others. In addition, FOXO have critical (MLL); L-selectin (CD62L); activation-induced cytidine implications in both normal and cancer stem cell deaminase (AID); biology. -
Laszloandrew-2000-Everyframearembrandt.Pdf
EVERY FRAME A REMBRANDT ART AND PRACTICE OF CINEMATOGRAPHY Andrew Laszlo, A.S.C. with additional material by Andrew Quicke Focal Press An Imprint of Elsevier Boston Oxford Auckland Johannesburg Melbourne New Delhi Focal Press is an imprint of Elsevier Copyright © 2000 Butterworth-Heinemann (Si A member of the Reed Elsevier group All rights reserved. All trademarks found herein are property of their respective owners. No part of this publication may be reproduced, stored in a retrieval system, or transmitted in any form or by any means, electronic, mechanical, photo copying, recording, or otherwise, without the prior written permission of the publisher. Permissions may be sought directly from Elsevier's Science and Technology Rights Department in Oxford, UK. Phone: (44) 1865 843830, Fax: (44) 1865 853333, e-mail: [email protected]. You may also complete your request on-line via the Elsevier homepage: http://www.elsevier.com by selecting "Customer Support" and then "Obtaining Permissions". 0 Recognizing the importance of preserving what has been written, Elsevier prints its books on acid-free paper whenever possible. GLOBAL E*sev*er suPPorts me efforts of American Forests and the Global ReLeaf REPEAT program in its campaign for the betterment of trees, forests, and our environment. Library of Congress Cataloging-in-Publication Data Laszlo, Andrew, 1926- Every frame a Rembrandt: art and practice of cinematography / Andrew Laszlo, with Andrew Quicke. p. cm. ISBN-13: 978-0-240-80399-9 ISBN-10: 0-240-80399-X (pbk. : alk. paper) 1. Cinematography. I. Quicke, Andrew. II. Title. TR850.L38 2000 778.5—dc21 ISBN-13: 978-0-240-80399-9 99-058083 ISBN-10: 0-240-80399-X British Library Cataloguing-in-Publication Data A catalogue record for this book is available from the British Library.