CASE HISTORY (Scheme)
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1. Intermittent Chest Pain: Angina: • Stable: (Caused By
CVS: 1. Intermittent chest pain: Angina: • Stable: (caused by chronic narrowing in one or more coronary arteries), episodes of pain are precipitated by exertion and may occur more readily when walking in cold or windy weather, after a large meal or while carrying a heavy load; the pain is promptly relieved by rest and/or sublingual glyceryl nitrate (GTN) spray, and typically lasts for less than 10 minutes. • unstable angina (caused by a sudden severe narrowing in a coronary artery), there is usually an abrupt onset or worsening of chest pain episodes that may occur on minimal exertion or at rest. • Retrosternal/ Progressive onset/ increase in intensity over 1–2 minutes/ Constricting, heavy/ Sometimes arm(s), neck, epigastrium/ Associated with breathlessness/ Intermittent, with episodes lasting 2–10 minutes/ Triggered by emotion, exertion, especially if cold, windy/ Relieved by rest, nitrates Mild to moderate. • Aggravated by thyroxine or drug-induced anemia, e.g. aspirin or NSAIDs Esophageal: • Retrosternal or epigastric/ Over 1–2 minutes; can be sudden (spasm)/ C: Gripping, tight or burning/ R: Often to back, sometimes to arms/ A: Heartburn, acid reflux/ T: Intermittent, often at night-time; variable duration/ Lying flat/some foods may trigger/ Not relieved by rest; nitrates sometimes relieve/ Usually mild but esophageal spasm can mimic myocardial infarction. 2. Acute chest pain: MI: • SOCRATES: Retrosternal/ Rapid over a few minutes/ Constricting, heavy/ Often to arm(s), neck, jaw, sometimes epigastrium/ Sweating, nausea, vomiting, breathlessness, feeling of impending death (angor animi)/ Acute presentation; prolonged duration/ ’Stress’ and exercise rare triggers, usually spontaneous/ Not relieved by rest or nitrates/ Usually severe. -
Scoliosis, Alters the Position of the Beat
Br Heart J: first published as 10.1136/hrt.8.3.162 on 1 July 1946. Downloaded from THE HEART IN STERNAL DEPRESSION BY WILLIAM EVANS From the Cardiac Department ofthe London Hospital Received June 25, 1946 The place where the apex beat appears on the chest wall depends as much on the symmetry of the thorax as on the size of the heart. A change in the alignment of the spine, the posterior fulcrum of the thoracic cage, in the form of scoliosis, alters the position of the beat. Local deformity of the ribs which form the walls of the cage will do the same thing. Deformity of the sternum, the anterior fulcrum of the thorax, as a cause of displacement of the apex beat has received less attention. The effects of depression of the sternum (pectus excavatum) on the shape and position of the heart have been studied in sixteen adults examined during the past year. DESCRIPTION OF CASES All sixteen patients had been referred for an explanation of certain signs connected with the heart, with the knowledge that deformity of the chest was present, but without appreciating that the two conditions might be related. In many of them suspicion of heart disease had led to restriction of their physical activities and to a change of design for their future livelihood. The symptoms that had caused the patients to seek medical advice in the first place were http://heart.bmj.com/ TABLE I SUMMARY OF FINDNGS IN 16 HEALTHY SUBJECTS WITH DEPRESSED STERNUM Antero-posterior Radiological findings in anterior view chest measurement _ _ Case Age Sternal in inches No. -
5 Precordial Pulsations
Chapter 5 / Precordial Pulsations 113 5 Precordial Pulsations CONTENTS MECHANICS AND PHYSIOLOGY OF THE NORMAL APICAL IMPULSE PHYSICAL PRINCIPLES GOVERNING THE FORMATION OF THE APICAL IMPULSE NORMAL APICAL IMPULSE AND ITS DETERMINANTS ASSESSMENT OF THE APICAL IMPULSE LEFT PARASTERNAL AND STERNAL MOVEMENTS RIGHT PARASTERNAL MOVEMENT PULSATIONS OVER THE CLAVICULAR HEADS PULSATIONS OVER THE SECOND AND/OR THIRD LEFT INTERCOSTAL SPACES SUBXIPHOID IMPULSE PRACTICAL POINTS IN THE CLINICAL ASSESSMENT OF PRECORDIAL PULSATIONS REFERENCES In this chapter the pulsations of the precordium will be discussed in relation to their identification, the mechanisms of their origin, and their pathophysiological and clinical significance. Precordial pulsations include the “apical impulse,” left parasternal movement, right parasternal movement, pulsations of the clavicular heads, pulsations over the second left intercostal space, and subxiphoid impulses. MECHANICS AND PHYSIOLOGY OF THE NORMAL APICAL IMPULSE Since during systole the heart contracts, becoming smaller and therefore moving away from the chest wall, why should one feel a systolic outward movement (the apical impulse) at all? Logically speaking there should not be an apical impulse. Several different methods of recording the precordial motion have been used to study the apical impulse going back to the late 19th century (1,2). Among the more modern methods, the notable ones are the recordings of the apexcardiogram (3–17), the impulse cardiogram (18), and the kinetocardiogram (19–21). While apexcardiography records the relative displacement of the chest wall under the transducer pickup device, which is often held by the examiner’s hands, the proponents of the impulse cardiography and kinetocardiography point out that these methods allow the recording of the absolute movement of the chest wall because the pickup device is anchored to a fixed point held 113 114 Cardiac Physical Examination in space away from the chest. -
CARDIOLOGY Section Editors: Dr
2 CARDIOLOGY Section Editors: Dr. Mustafa Toma and Dr. Jason Andrade Aortic Dissection DIFFERENTIAL DIAGNOSIS PATHOPHYSIOLOGY (CONT’D) CARDIAC DEBAKEY—I ¼ ascending and at least aortic arch, MYOCARDIAL—myocardial infarction, angina II ¼ ascending only, III ¼ originates in descending VALVULAR—aortic stenosis, aortic regurgitation and extends proximally or distally PERICARDIAL—pericarditis RISK FACTORS VASCULAR—aortic dissection COMMON—hypertension, age, male RESPIRATORY VASCULITIS—Takayasu arteritis, giant cell arteritis, PARENCHYMAL—pneumonia, cancer rheumatoid arthritis, syphilitic aortitis PLEURAL—pneumothorax, pneumomediasti- COLLAGEN DISORDERS—Marfan syndrome, Ehlers– num, pleural effusion, pleuritis Danlos syndrome, cystic medial necrosis VASCULAR—pulmonary embolism, pulmonary VALVULAR—bicuspid aortic valve, aortic coarcta- hypertension tion, Turner syndrome, aortic valve replacement GI—esophagitis, esophageal cancer, GERD, peptic OTHERS—cocaine, trauma ulcer disease, Boerhaave’s, cholecystitis, pancreatitis CLINICAL FEATURES OTHERS—musculoskeletal, shingles, anxiety RATIONAL CLINICAL EXAMINATION SERIES: DOES THIS PATIENT HAVE AN ACUTE THORACIC PATHOPHYSIOLOGY AORTIC DISSECTION? ANATOMY—layers of aorta include intima, media, LR+ LRÀ and adventitia. Majority of tears found in ascending History aorta right lateral wall where the greatest shear force Hypertension 1.6 0.5 upon the artery wall is produced Sudden chest pain 1.6 0.3 AORTIC TEAR AND EXTENSION—aortic tear may Tearing or ripping pain 1.2–10.8 0.4–0.99 produce -
Signs and Symptoms
Signs and symptoms For the most part, symptoms are related to disturbed bowel functions. Pain first, vomiting next and fever last has been described as classic presentation of acute appendicitis. Pain starts mid abdomen, and except in children below 3 years, tends to localize in right iliac fossa in a few hours. This pain can be elicited through various signs. Signs include localized findings in the right iliac fossa. The abdominal wall becomes very sensitive to gentle pressure (palpation). Also, there is severe pain on suddenly releasing a deep pressure in lower abdomen (rebound tenderness). In case of a retrocecal appendix, however, even deep pressure in the right lower quadrant may fail to elicit tenderness (silent appendix), the reason being that the cecum, distended with gas, prevents the pressure exerted by the palpating hand from reaching the inflamed appendix. Similarly, if the appendix lies entirely within the pelvis, there is usually complete absence of the abdominal rigidity. In such cases, a digital rectal examination elicits tenderness in the rectovesical pouch. Coughing causes point tenderness in this area (McBurney's point) and this is the least painful way to localize the inflamed appendix. If the abdomen on palpation is also involuntarily guarded (rigid), there should be a strong suspicion of peritonitis requiring urgent surgical intervention. Rovsing's sign Continuous deep palpation starting from the left iliac fossa upwards (anti clockwise along the colon) may cause pain in the right iliac fossa, by pushing bowel contents towards the ileocaecal valve and thus increasing pressure around the appendix. This is the Rovsing's sign.[5] Psoas sign Psoas sign or "Obraztsova's sign" is right lower-quadrant pain that is produced with either the passive extension of the patient's right hip (patient lying on left side, with knee in flexion) or by the patient's active flexion of the right hip while supine. -
Abdominal Examination Positioning
ABDOMINAL EXAMINATION POSITIONING Patients hands remain on his/hers side Legs, straight Head resting on pillow – if neck is flexed, ABD muscles will tense and therefore harder to palpate ABD . INSPECTION AUSCULATION PALPATION PERCUSSION INSPECTION INSPECTION Shape Skin Abnormalities Masses Scars (Previous op's - laproscopy) Signs of Trauma Jaundice Caput Medusae (portal H-T) Ascities (bulging flanks) Spider Navi-Pregnant women Cushings (red-violet) ... Hands + Mouth Clubbing Palmer Erythmea Mouth ulceration Breath (foeter ex ore) ... AUSCULTATION Use stethoscope to listen to all areas Detection of Bowel sounds (Peristalsis/Silent?? = Ileus) If no bowel sounds heard – continue to auscultate up to 3mins in the different areas to determine the absence of bowel sounds Auscultate for BRUITS!!! - Swishing (pathological) sounds over the arteries (eg. Abdominal Aorta) ... PALPATION ALWAYS ASK IF PAIN IS PRESENT BEFORE PALPATING!!! Firstly: Superficial palpation Secondly: Deep where no pain is present. (deep organs) Assessing Muscle Tone: - Guarding = muscles contract when pressure is applied - Ridigity = inidicates peritoneal inflamation - Rebound = Releasing of pressure causing pain ....... MURPHY'S SIGN Indication: - pain in U.R.Quadrant Determines: - cholecystitis (inflam. of gall bladder) - Courvoisier's law – palpable gall bladder, yet painless - cholangitis (inflam. Of bile ducts) ... METHOD Ask patient to breathe out. Gently place your hand below the costal margin on the right side at the mid-clavicular line (location of the gallbladder). Instruct to breathe in. Normally, during inspiration, the abdominal contents are pushed downward as the diaphragm moves down. If the patient stops breathing in (as the gallbladder comes in contact with the examiner's fingers) the patient feels pain with a 'catch' in breath. -
General Medicine - Surgery IV Year
1 General Medicine - Surgery IV year 1. Overal mortality rate in case of acute ESR – 24 mm/hr. Temperature 37,4˚C. Make appendicitis is: the diagnosis? A. 10-20%; A. Appendicular colic; B. 5-10%; B. Appendicular hydrops; C. 0,2-0,8%; C. Appendicular infiltration; D. 1-5%; D. Appendicular abscess; E. 25%. E. Peritonitis. 2. Name the destructive form of appendicitis. 7. A 34-year-old female patient suffered from A. Appendicular colic; abdominal pain week ago; no other B. Superficial; gastrointestinal problems were noted. On C. Appendix hydrops; clinical examination, a mass of about 6 cm D. Phlegmonous; was palpable in the right lower quadrant, E. Catarrhal appendicitis. appeared hard, not reducible and fixed to the parietal muscle. CBC: leucocyts – 3. Koher sign is: 7,5*109/l, ESR – 24 mm/hr. Temperature A. Migration of the pain from the 37,4˚C. Triple antibiotic therapy with epigastrium to the right lower cefotaxime, amikacin and tinidazole was quadrant; very effective. After 10 days no mass in B. Pain in the right lower quadrant; abdominal cavity was palpated. What time C. One time vomiting; term is optimal to perform appendectomy? D. Pain in the right upper quadrant; A. 1 week; E. Pain in the epigastrium. B. 2 weeks; C. 3 month; 4. In cases of appendicular infiltration is D. 1 year; indicated: E. 2 years. A. Laparoscopic appendectomy; B. Concervative treatment; 8. What instrumental method of examination C. Open appendectomy; is the most efficient in case of portal D. Draining; pyelophlebitis? E. Laparotomy. A. Plain abdominal film; B. -
Cardiology 1
Cardiology 1 SINGLE BEST ANSWER (SBA) a. Sick sinus syndrome b. First-degree AV block QUESTIONS c. Mobitz type 1 block d. Mobitz type 2 block 1. A 19-year-old university rower presents for the pre- e. Complete heart block Oxford–Cambridge boat race medical evaluation. He is healthy and has no significant medical history. 5. A 28-year-old man with no past medical history However, his brother died suddenly during football and not on medications presents to the emergency practice at age 15. Which one of the following is the department with palpitations for several hours and most likely cause of the brother’s death? was found to have supraventricular tachycardia. a. Aortic stenosis Carotid massage was attempted without success. b. Congenital long QT syndrome What is the treatment of choice to stop the attack? c. Congenital short QT syndrome a. Intravenous (IV) lignocaine d. Hypertrophic cardiomyopathy (HCM) b. IV digoxin e. Wolff–Parkinson–White syndrome c. IV amiodarone d. IV adenosine 2. A 65-year-old man presents to the heart failure e. IV quinidine outpatient clinic with increased shortness of breath and swollen ankles. On examination his pulse was 6. A 75-year-old cigarette smoker with known ischaemic 100 beats/min, blood pressure 100/60 mmHg heart disease and a history of cardiac failure presents and jugular venous pressure (JVP) 10 cm water. + to the emergency department with a 6-hour history of The patient currently takes furosemide 40 mg BD, increasing dyspnoea. His ECG shows a narrow complex spironolactone 12.5 mg, bisoprolol 2.5 mg OD and regular tachycardia with a rate of 160 beats/min. -
Abdominal Examination
ABDOMINAL EXAMINATION Dr. Ahmed Al Sarkhy Associate professor of paediatrics , KSUMC, KSU REMEMBER BEFORE STARTING... ALWAYS Introduce your self Take permission Wash your hands GASTROINTESTINAL EXAMINATION VS. ABDOMINAL EXAM General examination General inspection (ABCDE) VS Abdominal examination Growth parameters Inspection Hands and arms Palpation Face, eyes and mouth Percussion Neck Auscultation Lower limbs GENERAL INSPECTION (ABCDE) A: APPEARANCE; well, ill, irritable, toxic B; BODY BUILT: (weight, height, waist circumference) BREATHING: resp. Distress, grunting, wheezing C: COLOUR: pale, jaundice, cyanosis D: DEHYDRATION/DYSMORPHIC FEATHURES E: EXTENSIONS: Ox tubes, IV lines, cardiac monitors Signs of dehydration HANDS Nails Clubbing Koilonychia Leuconychia Palmar erythema Dupuytren’s contractures Hepatic flap HANDS Palmar erythema Dupuytren’s contractures ARMS Spider naevi (telangiectatic lesions) Bruising Wasting Scratch marks (chronic cholestasis) FACE, EYES … Conjuctival pallor (anaemia) Sclera: jaundice Cornea: Kaiser Fleischer’s rings (Wilson’s disease) Xanthelasma (chronic cholestasis) MOUTH Breath (fetor hepaticus, DKA) Lips Angular stomatitis Cheilitis Ulceration Peutz-Jeghers syndrome Gums Gingivitis, bleeding Candida albicans Pigmentation Tongue Atrophic glossitis (B12, FA def) Furring NECK AND CHEST Cervical lymphadenopathy Left supraclavicular fossa (Virchov’s node=lymphoma) Gynaecomastia Spider nevi NLOWER LIMBS VASCULAITIS EDEMA (pitting, non-pitting) ABDOMINAL -
Ministry of Health of Ukraine Kharkiv National Medical University
Ministry of Health of Ukraine Kharkiv National Medical University PHYSICAL METHODS OF CARDIOVASCULAR SYSTEM EXAMINATION. INQUIRY AND GENERAL INSPECTION OF THE PATIENTS WITH CARDIOVASCULAR PATHOLOGY. INSPECTION AND PALPATION OF PRECORDIAL AREA Methodical instructions for students Рекомендовано Ученым советом ХНМУ Протокол №__от_______2017 г. Kharkiv KhNMU 2017 Physical methods of cardiovascular system examination. Inquiry and general inspection of the patients with cardiovascular pathology. Inspection and palpation of precordial area / Authors: Т.V. Ashcheulova, O.M. Kovalyova, O.V. Honchar. – Kharkiv: KhNMU, 2016. – 16 с. Authors: Т.V. Ashcheulova O.M. Kovalyova O.V. Honchar INQUIRY OF A PATIENT WITH CARDIOVASCULAR PATHOLOGY The main complaints in patients with cardiovascular disease include: 1. Dyspnea, asthma attacks 2. Pain in the heart region 3. Palpitations 4. Intermissions of heart beats 5. Swelling of the lower extremities and accumulation of fluid in cavities 6. Cough, hemoptysis 7. Dyspepsia 8. Asthenovegetative disorders: weakness, fatigue, decline in performance Dyspnea is a painful feeling of lack of air, one of the symptoms of heart failure, predominantly is of inspiratory type and can be associated with physical activity (in the early stages of compensation) or occur at rest (a sign of severe cardiac decompensation). It is a compensatory responsive activation of the respiratory center in case of congestion and decreased blood flow in larger and small circulation due to reduced myocardial contractility. Dyspnea is typical for heart failure on the background of valvular heart disease (especially mitral valve pathology), ischemic heart disease (angina pectoris, myocardial infarction, cardiosclerosis, arrhythmias and heart blockages), essential and symptomatic hypertension (due to chronic kidney disease, pheochromocytoma, Cushing's disease, primary aldosteronism etc.). -
Abdominal Pain Part II
Abdominal Pain Part II Jassin M. Jouria, MD Dr. Jassin M. Jouria is a medical doctor, professor of academic medicine, and medical author. He graduated from Ross University School of Medicine and has completed his clinical clerkship training in various teaching hospitals throughout New York, including King’s County Hospital Center and Brookdale Medical Center, among others. Dr. Jouria has passed all USMLE medical board exams, and has served as a test prep tutor and instructor for Kaplan. He has developed several medical courses and curricula for a variety of educational institutions. Dr. Jouria has also served on multiple levels in the academic field including faculty member and Department Chair. Dr. Jouria continues to serves as a Subject Matter Expert for several continuing education organizations covering multiple basic medical sciences. He has also developed several continuing medical education courses covering various topics in clinical medicine. Recently, Dr. Jouria has been contracted by the University of Miami/Jackson Memorial Hospital’s Department of Surgery to develop an e- module training series for trauma patient management. Dr. Jouria is currently authoring an academic textbook on Human Anatomy & Physiology. ABSTRACT Abdominal pain is one of the most common complaints that patients make to medical professionals, and it has a wide array of causes, ranging from very simple to complex. Although many cases of abdominal pain turn out to be minor constipation or gastroenteritis, there are more serious causes that need to be ruled out. An accurate patient medical history, family medical history, laboratory work and imaging are important to make an accurate diagnosis. -
The Cardiovascular Examination
CHAPTER 1 The Cardiovascular Examination KEY POINTS • The cardiovascular examination lends itself to a systematic approach. • The examination should be thorough but should be directed by the history to areas likely to be relevant. • Certain cardiovascular signs are quite sensitive and specific. • When the examination is well performed, many unnecessary investigations can be avoided. CASE 1 SCENARIO: TARA WITH 3. Pick up the patient’s hand. Feel the radial pulse. DYSPNOEA Inspect the patient’s hands for clubbing. Demon- strate Schamroth’s sign (Fig. 1.1). If there is no 34-year-old Tara was referred to the hospital by her general clubbing, opposition of the index finger (nail to practitioner. She presented with increasing dyspnoea for the nail) demonstrates a diamond shape; in clubbing last 2 weeks. She has found it difficult to lie flat in bed and this space is lost. Also look for the peripheral has been waking up frequently feeling breathless. She also stigmata of infective endocarditis. Splinter haemor- has a dry cough and has felt extremely tired for weeks. She rhages are common (and are usually caused by also has high fever with a shake. She is an intravenous drug trauma), whereas Osler’s nodes and Janeway lesions user and her general practitioner (GP) found a loud murmur (Fig. 1.2) are rare. Look quickly, but carefully, at on auscultation. each nail bed, otherwise it is easy to miss key signs. Please examine the cardiovascular system. Note the presence of an intravenous cannula and, if an infusion is running, look at the bag to see The cardiovascular system should be examined in what it is.